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Journal articles on the topic 'Cardiomyopathie dilatée'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 July 2024

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1

Miftah, Fatima Azzahra, Jamal Kheyi, Hicham Bouzelmat, Ali Chaib, and Abdelhamid Moustaghfir. "Une cardiomyopathie dilatée réversible." La Presse Médicale 42, no. 9 (September 2013): 1292–94. http://dx.doi.org/10.1016/j.lpm.2012.10.025.

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2

Yousfi, C. "Cardiomyopathie dilatée et dystrophie." Annales d'Endocrinologie 76, no. 4 (September 2015): 430–31. http://dx.doi.org/10.1016/j.ando.2015.07.427.

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3

Bounaga, A., Y. Djellas, N. Kattan, M. A. Beaudoin, P. Gilet, L. Meillet, F. Schillo, and S. Borot. "Cardiomyopathie dilatée sur hypocalcémie sévère chronique." Annales d'Endocrinologie 76, no. 4 (September 2015): 488. http://dx.doi.org/10.1016/j.ando.2015.07.630.

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4

Yayehd, K., T. Tcherou, W. D. Ziga, S. Pessinaba, Y. T. Abena, M. M. Matelbe, N. W. N'da, E. K. Togbossi, S. Baragou, and F. Damorou. "Profil du patient africain porteur d’une cardiomyopathie dilatée dans un hôpital universitaire de Lomé." Journal de la Recherche Scientifique de l’Université de Lomé 26, no. 1 (April 18, 2024): 81–89. http://dx.doi.org/10.4314/jrsul.v26i1.15.

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Introduction : La cardiomyopathie dilatée (CMD) représente une forme lésionnelle grave de l’insuffisance cardiaque. Les objectifs de cette étude étaient de décrire les caractéristiques épidémiologiques, cliniques, paracliniques et évolutifs des patients admis pour cardiomyopathie dilatée au CHU Campus de Lomé. Matériels et méthodes : Nous avons réalisé une étude transversale, de janvier 2010 à décembre 2015 dans le service d’hospitalisation de cardiologie du Centre Hospitalier Universitaire Campus de Lomé. Les patients étaient inclus s’ils présentaient une insuffisance cardiaque clinique et un ventricule gauche dilaté avec une fraction d’éjection ventriculaire gauche (FEVG) inférieure à 50%. Résultats : La prévalence de la CMD était de 7%. L’âge moyen était de 55,4±15,9 ans. Les principaux facteurs de risque étaient l’hypertension artérielle (63,2% des hommes contre 52,6% des femmes, p = 0,004), le diabète (10,7%) et le tabac (1,4%). La FEVG moyenne était de 32,7±7,6%. Les étiologies étaient dominées par les cardiopathies hypertensives (69,2% des hommes contre 49,6% des femmes, p = 0,0006), la cardiomyopathie du péripartum (15,2%), et les causes idiopathiques (13,7%). Les complications hospitalières étaient la fibrillation atriale (6,2%) et le thrombus intracavitaire (10,3%). Il y avait 2,7% de décès hospitaliers. Conclusion : Le patient présentant une CMD était majoritairement un homme hypertendu de la cinquantaine, ou une femme dans le péripartum ; ces groupes constituent vraisemblablement des classes prioritaires sur lesquelles devront être focalisés des programmes thérapeutiques et de prévention. Background: Dilated cardiomyopathy (DCM) is a serious lesional cause of heart failure. The aim of this study was to describe the epidemiological and clinical characteristics and the hospital outcome of patients admitted for DCM in a university hospital of Lomé. Materials and methods: We carried out a crosssectional study from January 2010 to December 2015 in the cardiology hospitalization department of the University Hospital Center Campus of Lomé. Patients were included if they had clinical signs of heart failure with a dilated left ventricle associated with a left ventricular ejection fraction less than 50% at echocardiography. Results: The prevalence of DCM was 7%. Mean age was 55.4±15.9 years. Main cardiovascular risk factors were high blood pressure (63.2% of men versus 52.6% of women, p = 0.004), diabetes (10 .7%) and tobacco (1.4%). Mean left ventricular ejection fraction was 32.7±7.6%. Main etiologies were hypertensive heart disease (69.2% of men versus 49.6% of women, p = 0.0006), peripartum cardiomyopathy (15.2%), and idiopathic causes (13.7%). Hospital complications were atrial fibrillation (6.2%) and intracavitary thrombosis (10.3%). There were 2.7% of in-hospital deaths. Conclusion: The patient presenting with DCM was mainly a hypertensive male in his fifties, or a woman in the peripartum period; These groups probably constitute priority classes on which therapeutic and prevention programs should be focused.

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5

Bennaoui, F., I. Ait Sab, A. Bourrahouat, and M. Sbihi. "Cardiomyopathie dilatée révélant une arthrite juvénile idiopathique." Journal de Pédiatrie et de Puériculture 24, no. 1 (January 2011): 20–23. http://dx.doi.org/10.1016/j.jpp.2010.10.004.

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6

Cuny, Ch, J. Ch Eicher, F. Andre, Y. Morvan, B. Chauffert, B. Lorcerie, F. Martin, and P. Louis. "Cardiomyopathie dilatée révélant une dermatomyosite. Attitude thérapeutique." La Revue de Médecine Interne 13, no. 6 (May 1992): S179. http://dx.doi.org/10.1016/s0248-8663(05)81651-0.

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7

Juillière, Y. "Traitement médical de la cardiomyopathie dilatée idiopathique." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2009, no. 176 (March 2009): 14–17. http://dx.doi.org/10.1016/s1261-694x(09)72488-5.

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8

Marchand, L., B. Segrestin, and G. Raverot. "Cardiomyopathie dilatée révélatrice d’une maladie de Cushing." Annales d'Endocrinologie 75, no. 5-6 (October 2014): 426–27. http://dx.doi.org/10.1016/j.ando.2014.07.524.

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9

Deharo, Jean-Claude, and Hassan Hage. "Tachycardies ventriculaires non soutenues et cardiomyopathie dilatée primitive." Archives des Maladies du Coeur et des Vaisseaux - Pratique 2004, no. 132 (October 2004): 29–31. http://dx.doi.org/10.1016/s1261-694x(04)73439-2.

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10

Benzarouel, D., K. Hasni, H. Ashab, and M. El Hattaoui. "Une cause réversible de la cardiomyopathie dilatée : l’hypocalcémie." Annales de Cardiologie et d'Angéiologie 63, no. 2 (April 2014): 102–6. http://dx.doi.org/10.1016/j.ancard.2012.04.003.

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11

Soulé, N., T. Pérez, K. Abou Ezzi, F. Paoli, R. Froissart, A. Chantepie, and F. Labarthe. "P274 - Cardiomyopathie dilatée révélant une maladie de Pompe infantile." Archives de Pédiatrie 17, no. 6 (June 2010): 118–19. http://dx.doi.org/10.1016/s0929-693x(10)70672-2.

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12

Cénac, A., A. Djibo, and L. Djangnikpo. "La cardiomyopathie dilatée péripartum. Un modèle de maladie polyfactorielle?" La Revue de Médecine Interne 14, no. 10 (January 1993): 1033. http://dx.doi.org/10.1016/s0248-8663(05)80150-x.

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13

Said, Yosra, Wafa Fehri, Dhaker Lahidheb, Senda Trabelsi, Habib Haouala, and Taoufik Najjar. "Maladie cœliaque et cardiomyopathie dilatée idiopathique : une association fortuite ?" La Presse Médicale 39, no. 10 (October 2010): 1099–100. http://dx.doi.org/10.1016/j.lpm.2010.05.014.

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14

Ghrairi, N., S. Chine, H. Bouakkez, A. Dahmouni, I. Nahdi, S. Kachboura, and S. Yalaoui. "Anomalies sérologiques de la maladie cœliaque et cardiomyopathie dilatée." Immuno-analyse & Biologie Spécialisée 27, no. 4 (August 2012): 191–94. http://dx.doi.org/10.1016/j.immbio.2012.05.005.

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15

Mahjoub, S., S. Mehri, F. Ourda, R. Boussaada, B. Zouari, and S. Ben Arab. "Étude épidémiologique de la cardiomyopathie dilatée idiopathique en Tunisie." Annales de Cardiologie et d'Angéiologie 60, no. 4 (August 2011): 202–6. http://dx.doi.org/10.1016/j.ancard.2011.04.006.

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16

Moog, S., T. Cuny, M. Klein, G. Weryha, M. Le Chatton, M. Agopiantz, V. Pascal-Vigneron, and J. Chatelin. "Insuffisance antéhypophysaire et cardiomyopathie dilatée : vers un dépistage systématique ?" Annales d'Endocrinologie 76, no. 4 (September 2015): 402. http://dx.doi.org/10.1016/j.ando.2015.07.325.

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17

Amghar, F., H. Mrini, A. Daoudi, I. Elwadeh, H. Iraqi, and A. Chraibi. "Cardiomyopathie dilatée révélant une hypothyroïdie (à propos d’un cas)." Annales d'Endocrinologie 73, no. 4 (September 2012): 319. http://dx.doi.org/10.1016/j.ando.2012.07.294.

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18

Wahbi, K. "Cardiomyopathie dilatée : pourquoi et quand penser à une laminopathie ?" Archives des Maladies du Coeur et des Vaisseaux - Pratique 2016, no. 248 (May 2016): 11–15. http://dx.doi.org/10.1016/j.amcp.2016.03.003.

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19

THOMAS, Anne. "LA CARDIOMYOPATHIE DILATÉE DU CHIEN : RÉSULTATS DE L’ÉTUDE EUROPEENNE LUPA." Bulletin de l'Académie vétérinaire de France, no. 1-4 (2013): 240. http://dx.doi.org/10.4267/2042/51804.

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20

El Guendouz, F., T. El Hankari, and G. Belmejdoub. "Cardiomyopathie dilatée et hypoparathyroïdie : réversibilité incomplète après correction de l’hypocalcémie." Annales d'Endocrinologie 74, no. 4 (September 2013): 293. http://dx.doi.org/10.1016/j.ando.2013.07.167.

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21

Belmrhar, N., Y. Ghanimi, K. El Ghazouani Bekkali, and A. Chraibi. "Une cardiomyopathie dilatée révélant une acromégalie à propos d’un cas." Annales d'Endocrinologie 76, no. 4 (September 2015): 407–8. http://dx.doi.org/10.1016/j.ando.2015.07.346.

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22

Gilgenkrantz, H. "Cardiomyopathie dilatée ou hypertrophique du hamster : une mutation pour deux phénotypes." médecine/sciences 14, no. 4 (1998): 519. http://dx.doi.org/10.4267/10608/1085.

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23

Amara, M. E., E. Villard, and M. Komajda. "Mise au point sur la génétique de la cardiomyopathie dilatée familiale." Annales de Cardiologie et d'Angéiologie 54, no. 3 (June 2005): 151–56. http://dx.doi.org/10.1016/j.ancard.2004.09.005.

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24

Brembilla-Perrot, B., O. Marçon, F. Chometon, L. Groben, O. Claudon, A. Terrier de la Chaise, P. Louis, et al. "Signification de la fibrillation auriculaire permanente dans la cardiomyopathie dilatée primitive." Annales de Cardiologie et d'Angéiologie 56, no. 3 (June 2007): 107–10. http://dx.doi.org/10.1016/j.ancard.2007.02.003.

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25

Baragou, S., M. Pio, S. Di Bernardo, T. Boulos Ksontini, S. Jiekak Dommange, L. Bonafe, E. Meijboom, and N. Sekarski. "Une cause de cardiomyopathie dilatée chez l’enfant : le déficit primaire en carnitine." Annales de Cardiologie et d'Angéiologie 63, no. 2 (April 2014): 107–10. http://dx.doi.org/10.1016/j.ancard.2011.12.006.

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26

Bouznad, N., G. El Mghari, N. El Ansari, and M. El Hattaoui. "Cardiomyopathie dilatée : une complication rare de l’hypocalcémie. À propos de deux cas." Annales d'Endocrinologie 75, no. 5-6 (October 2014): 353. http://dx.doi.org/10.1016/j.ando.2014.07.278.

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27

Prats, J. Salvia, J. Rohmer, M. Merlant, C. Faucon, J. El Khalifa, C. Picard-Dahan, V. Descamps, and N. Abdelhedi-Mekki. "Cardiomyopathie dilatée sévère associée à une hyperéosinophilie dans une dermatite atopique sous dupilumab." Annales de Dermatologie et de Vénéréologie - FMC 3, no. 8 (December 2023): A322—A323. http://dx.doi.org/10.1016/j.fander.2023.09.557.

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28

ROUSSELOT, Jean François. "DÉPISTAGE DE LA CARDIOMYOPATHIE DILATÉE ET DE LA FIBRILLATION ATRIALE CHEZ LE LÉVRIER IRLANDAIS." Bulletin de l'Académie vétérinaire de France, no. 1-4 (2013): 234. http://dx.doi.org/10.4267/2042/51803.

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29

Constans, J., S. Sire, C. Sergeant, M. Simonoff, and JM Ragnaud. "Cardiomyopathie dilatée et déficit en sélénium au cours du sida. À propos d'un cas." La Revue de Médecine Interne 18, no. 8 (August 1997): 642–45. http://dx.doi.org/10.1016/s0248-8663(97)82466-6.

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30

Vandenhende, M. A., F. Bonnet, L. Sailler, S. Bouillot, P. Morlat, and J. Beylot. "Cardiomyopathie dilatée et toxicité musculaire des hypolipémiants : une maladie de Becker de révélation tardive." La Revue de Médecine Interne 26, no. 12 (December 2005): 977–79. http://dx.doi.org/10.1016/j.revmed.2005.08.012.

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31

Elallouchi, Y., M. Laghmari, M. Lmejjati, H. Ghannane, K. Aniba, and S. Aït Benali. "Accident vasculaire cérébral révélateur d’une cardiomyopathie dilatée primitive chez un nourrisson de 10 mois." Neurochirurgie 63, no. 1 (March 2017): 55. http://dx.doi.org/10.1016/j.neuchi.2016.11.068.

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32

Merlet, N., E. Lavazais-Blancou, B. Rozec, and C. Gauthier. "H025 Les récepteurs bêta3-adrénergiques cardiaques comme nouvelle cible thérapeutique de la cardiomyopathie dilatée." Archives of Cardiovascular Diseases 102 (March 2009): S81. http://dx.doi.org/10.1016/s1875-2136(09)72324-1.

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33

Paule, Philippe, Manuela Oliver, Jean-Marie Gil, Edward Lightburne, Jean Miltgen, and Laurent Fourcade. "Syndrome de Lemierre compliqué d’un purpura vasculaire chez un patient ayant une cardiomyopathie dilatée." La Presse Médicale 39, no. 7-8 (July 2010): 842–45. http://dx.doi.org/10.1016/j.lpm.2009.12.009.

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34

El Belghiti, M. R., K. Aniba, M. Bouskraoui, and S. Fadouach. "Accident vasculaire cérébral révélateur d'une cardiomyopathie dilatée primitive chez un nourrisson de 10 mois." Archives de Pédiatrie 12, no. 7 (July 2005): 1166–67. http://dx.doi.org/10.1016/j.arcped.2005.04.071.

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35

Deibener, J., C. De Chillou, K. Angioi, T. Maalouf, and P. Kaminsky. "Cardiomyopathie dilatée et panuvéite bilatérale révélant une maladie de Lyme.Revue générale à propos d’un cas." La Revue de Médecine Interne 22, no. 1 (January 2001): 65–69. http://dx.doi.org/10.1016/s0248-8663(00)00287-3.

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36

Lepoivre, T., E. Legendre, and M. Pinaud. "Anesthésie pour césarienne chez une patiente atteinte d’une maladie de McArdle et d’une cardiomyopathie dilatée familiale." Annales Françaises d'Anesthésie et de Réanimation 21, no. 6 (June 2002): 517–20. http://dx.doi.org/10.1016/s0750-7658(02)00645-7.

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37

Mandric, C. G., A. G. Dimitriu, and I. Miron. "SFCE-P12 – Cancérologie – La cardiomyopathie dilatée : complication sévère du traitement avec des anthracyclines dans les maladies malignes chez l’enfant." Archives de Pédiatrie 15, no. 5 (June 2008): 1010. http://dx.doi.org/10.1016/s0929-693x(08)72354-6.

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38

Selton-Suty, C., C. Dumitrescu, L. Mock, R. Piquemal, B. Popovic, A. Zanutto, A. Codreanu, M. Nippert, and Y. Juillière. "Évaluation de l'asynchronisme ventriculaire par échocardiographie Doppler tissulaire chez les patients atteints d'une cardiomyopathie dilatée idiopathique à QRS fins." Annales de Cardiologie et d'Angéiologie 53, no. 4 (July 2004): 162–66. http://dx.doi.org/10.1016/j.ancard.2004.02.013.

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39

Brogly, N., E. Guasch, L. Puertas, E. Alsina, T. López, and F. Gilsanz. "Défaillance cardiaque aiguë associée à une cardiomyopathie dilatée en post-partum précoce : traitement par ballon de contrepulsion aortique et lévosimendan." Annales Françaises d'Anesthésie et de Réanimation 29, no. 11 (November 2010): 807–10. http://dx.doi.org/10.1016/j.annfar.2010.08.008.

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40

Wakabayashi, Y., A. Baba, M. Akaishi, T. Yoshikawa, and H. Tomoike. "Plasmaphérèse d’immuno-adsorption en utilisant une colonne immobilisée par le tryptophane pour la cardiomyopathie dilatée : étude randomisée, prospective et multicentrique pour évaluer son efficacité et sa sécurité." Néphrologie & Thérapeutique 11, no. 5 (September 2015): 401. http://dx.doi.org/10.1016/j.nephro.2015.07.405.

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41

Veksler, V. I., I. Murat, and R. Ventura-Clapier. "Creatine kinase and mechanical and mitochondrial functions in hereditary and diabetic cardiomyopathies." Canadian Journal of Physiology and Pharmacology 69, no. 6 (June 1, 1991): 852–58. http://dx.doi.org/10.1139/y91-129.

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To determine whether the development of cardiomyopathies is associated with alterations in creatine kinase function, the functional properties of cardiac contractile apparatus and mitochondria were studied in two different models of cardiomyopathies, the Syrian hamster (hereditary dilated cardiomyopathy, strain UM-X7.1, 200 days old) and the diabetic rat (4–6 weeks after injection of streptozotocin) using ventricular skinned fibers. After Triton X-100 treatment, the hereditary cardiomyopathic fibers demonstrated decreased maximal calcium-activated tension and unchanged calcium sensitivity, whereas fibers from diabetic hearts exhibited unchanged maximal tension and increased calcium sensitivity, when compared with their respective controls. In both cases myofibrillar creatine kinase appeared unchanged. The functional properties of total tissue mitochondria were evaluated using saponin-skinned fibers. Coupling between oxidation and phosphorylation was not altered in cardiomyopathies. Respiration rate (per unit of tissue dry weight) was normal in hereditary cardiomyopathy but was considerably lower in diabetic fibers compared with control fibers. In both models of cardiomyopathies, creatine-stimulated respiration was significantly lower than in controls, thus indicating the depression of functional activity of mitochondrial creatine kinase.Key words: diabetes, hereditary cardiomyopathy, myofibrils, mitochondria, creatine kinase.

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42

Malčić, Ivan, Marija Jelušić, Hrvoje Kniewald, Nina Barišić, Dražen Jelašić, and Jadranka Božikov. "Epidemiology of cardiomyopathies in children and adolescents: a retrospective study over the last 10 years." Cardiology in the Young 12, no. 3 (May 2002): 253–59. http://dx.doi.org/10.1017/s1047951102000550.

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We conducted a retrospective study at the Department of Paediatric Cardiology of the University Hospital Centre Rebro, Zagreb, over the period from 1988 to 1998, so as to assess the epidemiology of childhood cardiomyopathies. The patients were categorized according to the guidelines of the Task Force on Cardiomyopathies of the World Health Organization and the International Society and Federation of Cardiology. We identified 121 infants, children and adolescents as having cardiomyopathy, giving an average occurrence for all cardiomyopathies of 38.81 for each 10,000 patients examined in our outpatient clinics for paediatric cardiology. Of the patients, 50 were female (41.3%) and 71 were male (58.7%). The cardiomyopathy was of the dilated variant in 52 patients (42.9%), with 43 patients (35.5%) having hypertrophic cardiomyopathy, and 6 patients (4.8%) identified with restrictive cardiomyopathy. We encountered no patients with arrhythmogenic right ventricular cardiomyopathy. In nine patients (7.4%), it proved impossible to classify the cardiomyopathy. We placed 11 patients (9.0%) in the group of specific cardiomyopathies. Most of those with dilated cardiomyopathy had been diagnosed prior to the age of 3 years (RR 1.9, 95% CI 1.4–2.47). There were no statistically significant differences in the incidences of dilated as compared to hypertrophic cardiomyopathy (Z 0.923, p = 0.1779), but we encountered a significantly lower occurrence of restrictive cardiomyopathy (Z 6.044, p < 0.001). Of those with hypertrophic cardiomyopathy, 15 patients (34.8%) had the asymmetric variant, while 28 patients (65.2%) exhibited the concentric form. During the period of follow-up, 10 patients died, 4 with dilated cardiomyopathy, 4 with hypertrophic cardiomyopathy, 1 with restrictive cardiomyopathy, and 1 with a specific cardiomyopathy. We encountered 12 (9.9%) patients who, besides cardiomyopathies, also suffered from neuromuscular disorders. Most of these had dilated cardiomyopathy. Mitochondrial disorders, in contrast, were more frequently found in patients with hypertrophic cardiomyopathy.

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43

Morel, N., K. Levesque, A. Maltret, G. Baron, M. Hamidou, P. Orquevaux, J. C. Piette, et al. "Facteurs associés à la survie des fœtus avec bloc auriculo-ventriculaire auto-immun et facteurs associés à la survenue d’une cardiomyopathie dilatée tardive : 214 cas du registre français des lupus néonataux." La Revue de Médecine Interne 36 (June 2015): A43. http://dx.doi.org/10.1016/j.revmed.2015.03.272.

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44

Shumkova, Monika, Kiril Karamfiloff, Raya Ivanova, Kristina Stoyanova, and Dobrin Vassilev. "Survival of patients with cardiomyopathies." Bulgarian Cardiology 27, no. 4 (December 31, 2021): 7–15. http://dx.doi.org/10.3897/bgcardio.27.e76672.

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Cardiomyopathies are a heterogeneous group of diseases. The main pathogenetic mechanism is myocardial damage due to genetic mutations. Cardiomyopathies are one of the leading causes of heart failure, sudden cardiac death, and life-threatening arrhythmias. Certain factors associated with poor prognosis determined the prognosis in this group of patients. Survival in different types of cardiomyopathies depends on the time of diagnosis and initial treatment. The types of cardiomyopathies discussed in this review are hypertrophic cardiomyopathy, dilative cardiomyopathy, restrictive cardiomyopathy, left ventricle non-compaction, and arrhythmogenic right ventricular cardiomyopathy.

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45

Bradic, Zeljko, Branislava Ivanovic, Dejan Markovic, Dusica Simic, Radmilo Jankovic, and Nevena Kalezic. "Preoperative preparation of patients with cardiomyopathies in non-cardiac surgery." Acta chirurgica Iugoslavica 58, no. 2 (2011): 39–43. http://dx.doi.org/10.2298/aci1102039b.

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Cardiomyopathies are myocardial diseases in which there is structural and functional disorder of the heart muscle, in the absence of coronary artery disease, hypertension, valvular disease and congenital heart disease. Cardiomyopathies are grouped into specific morphological and functional phenotypes: dilated cardiomyopathy, hypertrophic cardiomyopathy, restrictive cardiomyopathy, arrhythmogenic right ventricular cardiomyopathy and unclassified cardiomyopathies. Patients with dilated and hypertrophic cardiomypathy are prone to the development of congestive heart failure in the perioperative period. Also, patients with hypertrophic and arrhythmogenic right ventricular cardiomyopathy are prone to arrhythmias in the perioperative period. Preoperative evaluation includes history, physical examination, ECG, chest radiography, complete blood count, electrolytes, creatinine, glomerular filtration rate, glucose, liver enzymes, urin analysis, BNP and echocardiographic evaluation of left ventricular function. Drug therapy should be optimized and continued preoperatively. Surgery should be delayed (unless urgent) in patients with decompensated or untreated cardiomyopathy. Preoperative evaluation requires integrated multidisciplinary approach of anesthesiologists, cardiologist and surgeons.

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46

Sinagra, Gianfranco, Perry M. Elliott, and Marco Merlo. "Dilated cardiomyopathy: so many cardiomyopathies!" European Heart Journal 41, no. 39 (December 23, 2019): 3784–86. http://dx.doi.org/10.1093/eurheartj/ehz908.

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47

Ciarambino, Tiziana, Giovanni Menna, Gennaro Sansone, and Mauro Giordano. "Cardiomyopathies: An Overview." International Journal of Molecular Sciences 22, no. 14 (July 19, 2021): 7722. http://dx.doi.org/10.3390/ijms22147722.

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Background: Cardiomyopathies are a heterogeneous group of pathologies characterized by structural and functional alterations of the heart. Aims: The purpose of this narrative review is to focus on the most important cardiomyopathies and their epidemiology, diagnosis, and management. Methods: Clinical trials were identified by Pubmed until 30 March 2021. The search keywords were “cardiomyopathies, sudden cardiac arrest, dilated cardiomyopathy (DCM), hypertrophic cardiomyopathy (HCM), restrictive cardiomyopathy, arrhythmogenic cardiomyopathy (ARCV), takotsubo syndrome”. Results: Hypertrophic cardiomyopathy (HCM) is the most common primary cardiomyopathy, with a prevalence of 1:500 persons. Dilated cardiomyopathy (DCM) has a prevalence of 1:2500 and is the leading indication for heart transplantation. Restrictive cardiomyopathy (RCM) is the least common of the major cardiomyopathies, representing 2% to 5% of cases. Arrhythmogenic cardiomyopathy (ARCV) is a pathology characterized by the substitution of the myocardium by fibrofatty tissue. Takotsubo cardiomyopathy is defined as an abrupt onset of left ventricular dysfunction in response to severe emotional or physiologic stress. Conclusion: In particular, it has been reported that HCM is the most important cause of sudden death on the athletic field in the United States. It is needless to say how important it is to know which changes in the heart due to physical activity are normal, and when they are pathological.

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Schaufelberger, Maria. "Cardiomyopathy and pregnancy." Heart 105, no. 20 (July 15, 2019): 1543–51. http://dx.doi.org/10.1136/heartjnl-2018-313476.

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Cardiomyopathy is a group of disorders in which the heart muscle is structurally and functionally abnormal in the absence of other diseases that could cause observed myocardial abnormality. The most common cardiomyopathies are hypertrophic and dilated cardiomyopathy. Rare types are arrhythmogenic right ventricular, restrictive, Takotsubo and left ventricular non-compaction cardiomyopathies. This review of cardiomyopathies in pregnancy shows that peripartum cardiomyopathy is the most common cardiomyopathy in pregnancy. Peripartum cardiomyopathy develops most frequently in the month before or after partum, whereas dilated cardiomyopathy often is known already or develops in the second trimester. Mortality in peripartum cardiomyopathy varies from <2% to 50%. Few reports on dilated cardiomyopathy and pregnancy exist, with only a limited number of patients. Ventricular arrhythmias, heart failure, stroke and death are found in 39%–60% of high-risk patients. However, patients with modest left ventricular dysfunction and good functional class tolerated pregnancy well. Previous studies on >700 pregnancies in 500 women with hypertrophic cardiomyopathy showed that prognosis was generally good, even though three deaths were reported in high-risk patients. Complications include different types of supraventricular and ventricular arrhythmias, heart failure and ischaemic stroke. Recent studies on 200 pregnancies in 100 women with arrhythmogenic right ventricular cardiomyopathy have reported symptoms, including heart failure in 18%–33% of pregnancies. Ventricular tachycardia was found in 0%–33% of patients and syncope in one patient. Information on rare cardiomyopathies is sparse and only presented in case reports. Close monitoring by multidisciplinary teams in referral centres that counsel patients before conception and follow them throughout gestation is recommended.

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Schulz, Lukas P., and Annina S. Vischer. "Cardiomyopathies in the Clinical Practice – an Overview." Praxis 111, no. 11 (August 2022): 623–31. http://dx.doi.org/10.1024/1661-8157/a003912.

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Abstract. Cardiomyopathies are myocardial disorders with a structurally and functionally abnormal heart muscle. In this review, we describe pathophysiological aspects, clinical presentation, diagnosis, risk stratification and therapeutical concepts of the three most common forms of cardiomyopathy: hypertrophic cardiomyopathy (HCM), dilated cardiomyopathy (DCM), and arrhythmogenic cardiomyopathy (ACM).

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Hänselmann, A., C. Veltmann, J. Bauersachs, and D. Berliner. "Dilated cardiomyopathies and non-compaction cardiomyopathy." Herz 45, no. 3 (February 27, 2020): 212–20. http://dx.doi.org/10.1007/s00059-020-04903-5.

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