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1

Peplow, Philip, James Adams, and Tim Young, eds. Cardiovascular and Metabolic Disease. Cambridge: Royal Society of Chemistry, 2015. http://dx.doi.org/10.1039/9781782622390.

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2

Metabolic syndrome and cardiovascular disease. Philadelphia, PA: Saunders/Elsevier, 2007.

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3

Levine, T. Barry. Metabolic syndrome and cardiovascular disease. Philadelphia, PA: Saunders, 2006.

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4

Association, American Heart, ed. Metabolic risk for cardiovascular disease. Dallas, TX: American Heart Association, 2010.

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5

Levine, T. Barry. Metabolic syndrome and cardiovascular disease. 2nd ed. Chichester, West Sussex: Wiley-Blackwell, 2013.

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6

Levine, T. Barry, and Arlene B. Levine. Metabolic Syndrome and Cardiovascular Disease. Oxford, UK: Blackwell Publishing Ltd., 2012. http://dx.doi.org/10.1002/9781118480045.

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7

Levine, T. Barry. Metabolic syndrome and cardiovascular disease. 2nd ed. Chichester, West Sussex: Wiley-Blackwell, 2013.

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8

Nutritional and metabolic bases of cardiovascular disease. Chichester, West Sussex: Wiley-Blackwell, 2011.

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9

Mancini, Mario, José M. Ordovas, Gabriele Riccardi, Paolo Rubba, and Pasquale Strazzullo, eds. Nutritional and Metabolic Bases of Cardiovascular Disease. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444318456.

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10

1933-, Simopoulos Artemis P., ed. Nutrition and fitness: Obesity, the metabolic syndrome, cardiovascular disease, and cancer. Basel: Karger, 2005.

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11

Granata, Riccarda, and Jørgen Isgaard. Cardiovascular issues in endocrinology. Basel: Karger, 2014.

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12

Jiang, Xian-Cheng, ed. Lipid Transfer in Lipoprotein Metabolism and Cardiovascular Disease. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6082-8.

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13

Marín-García, José. Mitochondria and their role in cardiovascular disease. New York: Springer, 2013.

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14

Obstructive sleep apnea in adults: Relationship with cardiovascular and metabolic disorders. Basel: Karger, 2011.

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15

International Meeting on Atherosclerosis and Cardiovascular Diseases. (7th 1989 Bologna, Italy). Atherosclerosis and cardiovascular disease: 7° international meeting, Bologna, September 1989. Edited by Descovich G. C. Bologna: Editrice Compositori, 1989.

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16

Cholesterol: Interactions with testosterone and cortisol in cardiovascular diseases. Berlin: Springer-Verlag, 1987.

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17

J, Halpern M., ed. Lipid metabolism and its pathology. New York: Plenum Press, 1985.

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18

J, Pownall Henry, ed. Manual of lipid disorders: Reducing the risk for coronary heart disease. 3rd ed. Philadelphia: Lippincott Williams & Wilkins, 2002.

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19

J, Pownall Henry, ed. Manual of lipid disorders: Reducing the risk of coronary heart disease. 2nd ed. Baltimore: Williams & Wilkins, 1999.

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20

E, Beamish Robert, Panagia Vincenzo, Dhalla Naranjan S, University of Manitoba, and Cardiology Symposium (3rd : 1986 : Winnipeg, Man.), eds. Pharmacological aspects of heart disease: Proceedings of an International Symposium on Heart Metabolism in Health and Disease and the Third Annual Cardiology Symposium of the University of Manitoba, July 8-11, 1986, Winnipeg, Canada. Boston: Nijhoff, 1987.

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21

L, Hess Michael, ed. Free radicals, cardiovascular dysfunction, and protection strategies. Austin: R.G. Landes Co., 1994.

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22

Statusseminar in the Framework of the German-Israeli Cooperation in Medical Research (2nd 1989 Reisenburg, Germany). Preventive cardiology: Diagnostic and metabolic aspects of cardiovascular research : selected papers presented at the 2nd Statusseminar in the Framework of the German-Israeli Cooperation in Medical Research, Reisenburg, 22-23 May 1989. München: Urban & Schwarzenberg, 1990.

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23

J, Halpern M., ed. Lipid metabolism and its pathology: Proceedings of the IVth International Colloquium on Lipid Metabolism and its Pathology, Lisbon, 18-22 November 1984. Amsterdam: Excerpta Medica, 1986.

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24

Gotto, Antonio M. Manual of lipid disorders. Baltimore: Williams & Wilkins, 1992.

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25

Maria, Cancela Carral Jose, and Varela Martinez Silvia, eds. Aerobic exercise in special populations. Hauppauge, N.Y: Nova Science, 2009.

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26

Levine, T. Barry, and Arlene Bradley Levine. Metabolic Syndrome and Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2012.

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27

Levine, T. Barry, and Arlene Bradley Levine. Metabolic Syndrome and Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2012.

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28

Eckel, Robert H. Metabolic Risk for Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2011.

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29

Eckel, Robert H. Metabolic Risk for Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2010.

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30

Krentz, Andrew J., and Nathan D. Wong, eds. Metabolic Syndrome and Cardiovascular Disease. CRC Press, 2006. http://dx.doi.org/10.3109/9781420020601.

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31

Metabolic Syndrome and Cardiovascular Disease. Elsevier, 2006. http://dx.doi.org/10.1016/b978-1-4160-2545-0.x5001-8.

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32

Eckel, Robert H. Metabolic Risk for Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2011.

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33

Mancini, Mario, Jose M. Ordovas, Gabrielle Riccardi, Paolo Rubba, and Pasquale Strazzullo. Nutritional and Metabolic Bases of Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2011.

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34

Mancini, Mario, Jose M. Ordovas, Gabrielle Riccardi, Paolo Rubba, and Pasquale Strazzullo. Nutritional and Metabolic Bases of Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2011.

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35

Mancini, Mario, Jose M. Ordovas, Gabrielle Riccardi, Paolo Rubba, and Pasquale Strazzullo. Nutritional and Metabolic Bases of Cardiovascular Disease. Wiley & Sons, Incorporated, John, 2010.

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36

(Editor), Andrew Krentz, and Nathan Wong (Editor), eds. Metabolic Syndrome and Cardiovascular Disease: Epidemiology, Assessment, and Management. Informa Healthcare, 2006.

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37

Metabolic syndrome and cardiovascular disease: Epidemiology, assessment, and management. New York, NY: Informa Healthcare, 2006.

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38

J, Krentz Andrew, and Wong Nathan D, eds. Metabolic syndrome and cardiovascular disease: Epidemiology, assessment, and management. New York: Informa Healthcare, 2007.

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39

Adams, James, Tim Young, Diler Aslan, and Shahrad Taheri. Cardiovascular and Metabolic Disease: Scientific Discoveries and New Therapies. Royal Society of Chemistry, The, 2015.

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40

(Editor), Andrew Krentz, and Nathan Wong (Editor), eds. Metabolic Syndrome and Cardiovascular Disease: Epidemiology, Assessment, and Management. Informa Healthcare, 2007.

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41

M, Forte Trudy, ed. Hormonal, metabolic, and cellular influences on cardiovascular disease in women. Armonk, NY: Futura Pub., 1997.

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42

Pereira, Luis F., Harold W. Goforth, Esteban Martínez, Joseph Z. Lux, Maria Ferrara, and Michael P. Mullen. Cardiovascular Disease, Metabolic Complications and Lipodystrophy in Persons with HIV. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0046.

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The introduction of effective antiretroviral therapy has contributed to a dramatic reduction in HIV-related mortality. As patients live longer, evidence suggests an increased incidence of cardiovascular disease in persons with HIV over that among individuals who do not have HIV, thus early detection and treatment of multimorbidities and modifiable cardiovascular disease risk factors particularly in persons with HIV are needed. Several mechanisms have been proposed to explain the increased risk of cardiovascular disease, including the virus itself, antiretroviral therapy, and traditional risks factors. This chapter discusses detection and treatment of cardiovascular disease in persons with HIV, as well as metabolic complications involved, including dyslipidemia, insulin resistance, and lactic acidosis. The pathogenesis and management of HIV-associated lipodystrophy as well as its psychosocial impact are also addressed.
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43

The cardiovascular and metabolic responses of men with cardiovascular disease to aqua dynamic exercise. 1990.

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44

The cardiovascular and metabolic responses of men with cardiovascular disease to aqua dynamic exercise. 1992.

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45

(Editor), Ezra A. Amsterdam, Denise D., M.d. Barnard (Editor), Barry H. Greenberg (Editor), Scott M. Grundy (Editor), and Philip M., M.d. Liebson (Editor), eds. Contemporary Cardiovascular Continuum. Handbooks in Health Care Company, 2006.

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46

Simopoulos, A. P., ed. Nutrition and Fitness: Obesity, the Metabolic Syndrome, Cardiovascular Disease, and Cancer. S. Karger AG, 2005. http://dx.doi.org/10.1159/isbn.978-3-318-01228-6.

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47

Z, Fisman Enrique, and Tenenbaum Alexander, eds. Cardiovascular diabetology: Clinical, metabolic and inflammatory facets. Basel: Karger, 2008.

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48

Z, Fisman Enrique, and Tenenbaum Alexander, eds. Cardiovascular diabetology: Clinical, metabolic and inflammatory facets. Basel: Karger, 2008.

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49

Elder, Grahame J. Metabolic bone disease after renal transplantation. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0288.

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Patients who undergo kidney transplantation have laboratory, bone, and soft tissue abnormalities that characterize chronic kidney disease mineral and bone disorder (CKD-MBD). After successful transplantation, abnormal values of parathyroid hormone, fibroblast growth factor 23, calcium, phosphate, vitamin D sterols, and sex hormones generally improve, but abnormalities often persist. Cardiovascular risk remains high and is influenced by prevalent vascular calcification, and fracture risk increases due to a combination of abnormal bone ‘quality’, compounded by immunosuppressive drugs and reductions in bone mineral density. Patients with well managed CKD-MBD before transplantation generally have a smoother post-transplant course, and it is useful to assess patients soon after transplantation for risk factors relevant to the general population and to patients with CKD. Targeted laboratory assessment, bone densitometry, and X-ray of the spine are useful for guiding therapy to minimize post-transplant effects of CKD-MBD. To reduce fracture risk, general measures include glucocorticoid dose minimization, attaining adequate 25(OH)D levels, and maintaining calcium and phosphate values in the normal range. Calcitriol or its analogues and antiresorptive agents such as bisphosphonates may protect bone from glucocorticoid effects and ongoing hyperparathyroidism, but the efficacy of these therapies to reduce fractures is unproven. Alternate therapies with fewer data include denosumab, strontium ranelate, teriparatide, oestrogen or testosterone hormone replacement therapy, tibolone, selective oestrogen receptor modulators, and cinacalcet. Parathyroidectomy may be necessary, but is generally avoided within the first post-transplant year. A schema is presented in this chapter that aims to minimize harm when allocating therapy.
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50

(Editor), E. Z. Fisman, and A. Tenenbaum (Editor), eds. Cardiovascular Diabetology: Clinical, Metabolic and Inflammatory Facets (Advances in Cardiology). S Karger Pub, 2008.

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