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Relevant bibliographies by topics / Cardiovascular system Obesity Chinese

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Dissertations / Theses

Academic literature on the topic 'Cardiovascular system Obesity Chinese'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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Dissertations / Theses on the topic "Cardiovascular system Obesity Chinese"

1

King, Karen. "Anthropometric indices and cardiovascular risk factors in a Chinese population." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23424680.

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2

Rider, Oliver J. "The effect of obesity and weight loss on cardiovascular system." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504513.

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3

Johns, David James. "Dietary patterns and cardiovascular disease in severe obesity." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610554.

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4

King, Karen, and 金穎和. "Anthropometric indices and cardiovascular risk factors in a Chinese population." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31970199.

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5

Appannah, Geeta. "Dietary patterns, obesity and cardiovascular risk factors in young people." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648138.

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6

Jones, Jenni. "The prevalence of heart-healthy menu items in restaurants of West Virginia." Morgantown, W. Va. : [West Virginia University Libraries], 2002. http://etd.wvu.edu/templates/showETD.cfm?recnum=2585.

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Thesis (M.S.)--West Virginia University, 2002.<br>Title from document title page. Document formatted into pages; contains v, 43 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 27).

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7

Claflin, Kristin Elizabeth. "The brain renin-angiotensin system in metabolic and cardiovascular regulation." Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/2196.

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Leptin acts within the brain to increase resting metabolic rate (RMR) and blood pressure (BP). The renin-angiotensin system (RAS) elicits similar effects in the brain, as reviewed in chapter 1, and it has previously been shown that central angiotensin II type 1 (AT1) receptors are required for leptin-mediated inductions in sympathetic nerve activity to the brown adipose tissue. Thus, we hypothesize that the brain RAS mediates the metabolic effects of leptin. To investigate the interaction between the RAS and leptin, we generated the AT1ALepR-KO mouse which lacks the AT1A receptor in leptin-sen

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8

Conwell, Louise Sonia. "Insulin resistance and cardiovascular risk factors in obese children and adolescents /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19066.pdf.

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9

Thompson, Simon R. "Genetic variation within the IL-18 system and its association with cardiovascular disease and obesity." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/14259/.

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Background and Aims: The interleukin 18 (IL-18) system consists of a proinflammatory cytokine (IL-18), a naturally occurring inhibitor (IL-18BP), and a dimeric receptor. IL-18 has been implicated in many autoimmune conditions, with elevated plasma levels predicting future risk of heart disease in healthy individuals. IL-18 also appears to have metabolic consequences, with IL18 -/- mice having higher bodyweight, attributable to increased feeding. The central hypothesis of this work was that genetic variation within both IL-18 and IL-18BP, that influenced their circulating levels, would be assoc

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10

Yang, Bo, and 杨波. "Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47869641.

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Obesity is closely related to many medical complications such as type 2 diabetes, hypertension and heart failure. Obesity and other factors, including elevated blood glucose levels, hypertension, and dyslipidemia, constitute a constellation of symptoms known as the metabolic syndrome, which are the risk factors for coronary artery disease. Lipocalin-2 is a pro-inflammatory adipokine causally involved in the development of obesity-associated metabolic and cardiovascular diseases. Recent clinical and experimental evidences demonstrate an association between augmented circulating lipocali

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