Relevant bibliographies by topics / Cardiovascular system Obesity Chinese

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Cardiovascular system Obesity Chinese'

Author: Grafiati
Published: 4 June 2021
Last updated: 14 February 2022

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Journal articles on the topic "Cardiovascular system Obesity Chinese"

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Gao, Luying, Linjie Wang, Hongbo Yang, Hui Pan, Fengying Gong, and Huijuan Zhu. "MC4R Single Nucleotide Polymorphisms Were Associated with Metabolically Healthy and Unhealthy Obesity in Chinese Northern Han Populations." International Journal of Endocrinology 2019 (November 6, 2019): 1–9. http://dx.doi.org/10.1155/2019/4328909.

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Melanocortin-4 receptor (MC4R) has been reported to be associated with the risk of obesity, and metabolically unhealthy obese (MUHO) patients tend to have a greater risk of cardiovascular complications than metabolically healthy obese (MHO) patients. Therefore, we aimed to study single nucleotide polymorphisms (SNPs) in the MC4R gene associated with metabolically healthy and unhealthy obesity in Chinese Northern Han populations. A total of 1100 Chinese Northern Han subjects were recruited and divided into four groups according to the criteria of the Adult Treatment Panel-III (ATP-III) and World Health Organization (WHO): MUHO (n = 300), MHO (n = 196), metabolic unhealthy normal weight (MUH-NW) (n = 303), and metabolic healthy normal weight (MH-NW) (n = 301). DNA samples were extracted, and six SNPs of the MC4R gene, including rs2331841, rs656710, rs17782313, rs571312, rs12970134, and rs11872992, were genotyped with the matrix-assisted laser desorption ionization time-of-flight mass spectrometry (MALDI-TOF MS) method. Among the six SNPs of the MC4R gene, rs2331841 (A/G) was the most significant and could account for 0.9% of obesity etiology. Compared with the normal weight group, rs2331841 of the MC4R gene was associated with obesity (P=0.032). The obesity risk of subjects with the AG genotype in the rs2331841 site was 82% higher than the risk of those with the GG genotype (β = 0.60, OR = 1.82, P=0.030). After adjusting for sex and age, the frequency of the A allele in the rs2331841 site was higher in the MUHO group than in the MH-NW group (27.9% vs. 21.1%, respectively, OR = 1.49, 95% CI 1.14–1.96, P=0.005) and in the MUHO group than in the MHO group (27.9% vs. 22.3%, respectively, OR = 1.39, 95% CI 1.02–1.92, P=0.039). Among the three genotypes of rs2331841, the subjects with the AA/AG genotype had higher diastolic blood pressure (DBP) than those with the GG genotype. Our data first suggest that SNPs in the rs2331841 site of the MC4R gene are closely related to obesity and its related metabolic disorders in Chinese Northern Han populations. The participants with an A allele of rs2331841 had a higher risk of obesity and MUHO than other participants.
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Raja-Khan, Nazia, Elisabet Stener-Victorin, XiaoKe Wu, and Richard S. Legro. "The physiological basis of complementary and alternative medicines for polycystic ovary syndrome." American Journal of Physiology-Endocrinology and Metabolism 301, no. 1 (July 2011): E1—E10. http://dx.doi.org/10.1152/ajpendo.00667.2010.

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Polycystic ovary syndrome (PCOS) is a common endocrine disorder that is characterized by chronic hyperandrogenic anovulation leading to symptoms of hirsutism, acne, irregular menses, and infertility. Multiple metabolic and cardiovascular risk factors are associated with PCOS, including insulin resistance, obesity, type 2 diabetes, hypertension, inflammation, and subclinical atherosclerosis. However, current treatments for PCOS are only moderately effective at controlling symptoms and preventing complications. This article describes how the physiological effects of major complementary and alternative medicine (CAM) treatments could reduce the severity of PCOS and its complications. Acupuncture reduces hyperandrogenism and improves menstrual frequency in PCOS. Acupuncture's clinical effects are mediated via activation of somatic afferent nerves innervating the skin and muscle, which, via modulation of the activity in the somatic and autonomic nervous system, may modulate endocrine and metabolic functions in PCOS. Chinese herbal medicines and dietary supplements may also exert beneficial physiological effects in PCOS, but there is minimal evidence that these CAM treatments are safe and effective. Mindfulness has not been investigated in PCOS, but it has been shown to reduce psychological distress and exert positive effects on the central and autonomic nervous systems, hypothalamic-pituitary-adrenal axis, and immune system, leading to reductions in blood pressure, glucose, and inflammation. In conclusion, CAM treatments may have beneficial endocrine, cardiometabolic, and reproductive effects in PCOS. However, most studies of CAM treatments for PCOS are small, nonrandomized, or uncontrolled. Future well-designed studies are needed to further evaluate the safety, effectiveness, and mechanisms of CAM treatments for PCOS.
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Bhatt, Kinal Paresh, Jonathan Quinonez, Abhinav Patel, Mehrie Patel, Abdifitah Mohamed, Muhammad Ali Aziz, Deepa Venkataramani, et al. "COVID-19 Pathophysiology and Clinical Effects on Multiple Organ Systems - A Narrative Review." International Annals of Science 10, no. 1 (June 12, 2021): 151–67. http://dx.doi.org/10.21467/ias.10.1.151-167.

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Patients with comorbidities including Hypertension (HTN), Diabetes Mellitus (DM), Chronic Obstructive Pulmonary Disease (COPD), Asthma, Obesity, Cardiovascular Disease (CVD), Chronic Kidney Disease (CKD), and those who are immunocompromised are prone to more severe complications of COVID-19 and a higher rate of hospitalizations. In the United States, around 94% of COVID-19 deaths had an average of 2.6 additional conditions or causes per death. In a summary report published by the Chinese Centre for Disease Control and Prevention of 72,314 cases, case-fatality rate was elevated among those with preexisting comorbid conditions—10.5% for cardiovascular disease, 7.3% for diabetes, 6.3% for chronic respiratory disease, 6.0% for HTN, and 5.6% for cancer. The COVID-19 pandemic continues to threaten people and healthcare systems globally and therefore the global economy. Currently, there is no cure or vaccine for COVID-19 and there is an urgent need to develop target therapies as we continue to learn more about this novel virus. Without therapeutic interventions, much of how we contain the viral spread is prevention through mitigation strategies (social distancing, face masks, supportive care). Early suspicion of COVID-19 symptoms with radiological and laboratory assessments may play a major role in preventing severity of the COVID-19. With this literature review we aim to provide review of pathophysiology of Severe Acute Respiratory Syndrome Coronavirus-2 (SARS-CoV-2) and its clinical effects on multiple organ systems.
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Hong, Jungha, and Nazim S. Gruda. "The Potential of Introduction of Asian Vegetables in Europe." Horticulturae 6, no. 3 (July 3, 2020): 38. http://dx.doi.org/10.3390/horticulturae6030038.

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Increasing longevity, along with an aging population in Europe, has caused serious concerns about diet-related chronic diseases such as obesity, diabetes, cardiovascular diseases, and certain cancers. As recently noted during the coronavirus pandemic, regular exercise and a robust immune system complemented by adequate consumption of fruit and vegetables are recommended due to their known health benefits. Although the volume of fresh vegetable consumption in the EU is barely growing, demand for diversified, nutritious, and exotic vegetables has been increasing. Therefore, the European market for fresh Asian vegetables is expected to expand across the EU member states, and the introduction of new vegetables has enormous potential. We conducted this review to address the high number and wide range of Asian vegetable species with a commercial potential for introduction into the current European vegetable market. Many of them have not received any attention yet. Four Asian vegetables: (1) Korean ginseng sprout, (2) Korean cabbage, (3) Coastal hog fennel and (4) Japanese (Chinese or Korean) angelica tree, are further discussed. All of these vegetables possess several health benefits, are increasingly in demand, are easy to cultivate, and align with current trends of the European vegetable market, e.g., vegetables having a unique taste, higher value, are decorative and small. Introducing Asian vegetables will enhance the diversity of nutritious horticultural products in Europe, associated with all their respective consumption benefits. Future research on the Asian vegetable market within Europe is needed. In addition, experimental studies of Asian vegetables under practical conditions for their production in different European environments are required. Economic, social, and ecological aspects also ought to be considered.
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Verbovoy, Andrey F., A. V. Pashentseva, and L. A. Sharonova. "Obesity and cardiovascular system." Clinical Medicine (Russian Journal) 95, no. 1 (March 13, 2017): 31–35. http://dx.doi.org/10.18821/0023-2149-2017-95-1-31-35.

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Obesity is a serious medico-social and economic problem. Increased body mass index is associated with a rise in the general mortality rate and mortality due to cardiovascular diseases. In this article, modern views of the influence of obesity on the cardiovascular system are considered.
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Lee, Z. S. K., J. A. J. H. Critchley, G. T. C. Ko, P. J. Anderson, G. N. Thomas, R. P. Young, T. Y. K. Chan, C. S. Cockram, B. Tomlinson, and J. C. N. Chan. "Obesity and cardiovascular risk factors in Hong Kong Chinese." Obesity Reviews 3, no. 3 (August 2002): 173–82. http://dx.doi.org/10.1046/j.1467-789x.2002.00066.x.

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Chair, S. Y., K. C. Choi, T. F. D. Lee, M. L. E. Wong, W. H. C. Chan, W. H. J. Sit, and W. K. W. So. "P069 Central Obesity among Chinese Patients with Heart Disease." International Journal of Cardiology 147 (March 2011): S29. http://dx.doi.org/10.1016/s0167-5273(11)70095-x.

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Csige, Imre, Dóra Ujvárosy, Zoltán Szabó, István Lőrincz, György Paragh, Mariann Harangi, and Sándor Somodi. "The Impact of Obesity on the Cardiovascular System." Journal of Diabetes Research 2018 (November 4, 2018): 1–12. http://dx.doi.org/10.1155/2018/3407306.

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Obesity is a growing health problem worldwide. It is associated with an increased cardiovascular risk on the one hand of obesity itself and on the other hand of associated medical conditions (hypertension, diabetes, insulin resistance, and sleep apnoea syndrome). Obesity has an important role in atherosclerosis and coronary artery disease. Obesity leads to structural and functional changes of the heart, which causes heart failure. The altered myocardial structure increases the risk of atrial fibrillation and sudden cardiac death. However, obesity also has a protective effect on the clinical outcome of underlying cardiovascular disease, the phenomenon called obesity paradox. The improved cardiac imaging techniques allow the early detection of altered structure and function of the heart in obese patients. In this review, we attempt to summarize the relationship between obesity and cardiovascular diseases and outline the underlying mechanisms. The demonstrated new techniques of cardiac diagnostic procedures allow for the early detection and treatment of subclinical medical conditions and, therefore, the prevention of cardiovascular events.
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Cheng, Tsung O. "Fast food, automobiles, television and obesity epidemic in Chinese children." International Journal of Cardiology 98, no. 1 (January 2005): 173–74. http://dx.doi.org/10.1016/j.ijcard.2004.08.019.

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&NA;. "F-14 Thematic Poster - Obesity and the Cardiovascular System." Medicine & Science in Sports & Exercise 46 (May 2014): 712–14. http://dx.doi.org/10.1249/01.mss.0000451253.72444.1b.

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Dissertations / Theses on the topic "Cardiovascular system Obesity Chinese"

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King, Karen. "Anthropometric indices and cardiovascular risk factors in a Chinese population." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23424680.

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Rider, Oliver J. "The effect of obesity and weight loss on cardiovascular system." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504513.

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Johns, David James. "Dietary patterns and cardiovascular disease in severe obesity." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610554.

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King, Karen, and 金穎和. "Anthropometric indices and cardiovascular risk factors in a Chinese population." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31970199.

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Appannah, Geeta. "Dietary patterns, obesity and cardiovascular risk factors in young people." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648138.

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Jones, Jenni. "The prevalence of heart-healthy menu items in restaurants of West Virginia." Morgantown, W. Va. : [West Virginia University Libraries], 2002. http://etd.wvu.edu/templates/showETD.cfm?recnum=2585.

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Thesis (M.S.)--West Virginia University, 2002.
Title from document title page. Document formatted into pages; contains v, 43 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references (p. 27).
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Claflin, Kristin Elizabeth. "The brain renin-angiotensin system in metabolic and cardiovascular regulation." Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/2196.

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Leptin acts within the brain to increase resting metabolic rate (RMR) and blood pressure (BP). The renin-angiotensin system (RAS) elicits similar effects in the brain, as reviewed in chapter 1, and it has previously been shown that central angiotensin II type 1 (AT1) receptors are required for leptin-mediated inductions in sympathetic nerve activity to the brown adipose tissue. Thus, we hypothesize that the brain RAS mediates the metabolic effects of leptin. To investigate the interaction between the RAS and leptin, we generated the AT1ALepR-KO mouse which lacks the AT1A receptor in leptin-sensitive cells. In chapter 2, we demonstrated that stimulation of RMR by DOCA-salt and high fat diet requires AT1A receptors in leptin receptor-expressing cells and that these cells expressing both AT1A and the leptin receptor appear to be agouti related-peptide (AgRP) neurons. In chapter 3, we investigated the role of AT1A specifically in AgRP neurons by utilizing AT1AAgRP-KO mice. Similar to AT1ALepR-KO mice, AT1AAgRP-KO mice exhibited deficits in BAT SNA responses to leptin and induction of RMR by alpha melanocyte stimulating hormone. In chapter 4, we utilized a novel transgenic mouse model to demonstrate that microglia do not express the AT1A receptor under chow or high fat diet fed conditions. Taken together, we conclude that a subset of AgRP neurons, which express both the leptin receptor and the AT1A receptor, are critical for the control of sympathetic nerve activity and ultimately RMR.
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Conwell, Louise Sonia. "Insulin resistance and cardiovascular risk factors in obese children and adolescents /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19066.pdf.

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Thompson, Simon R. "Genetic variation within the IL-18 system and its association with cardiovascular disease and obesity." Thesis, University College London (University of London), 2009. http://discovery.ucl.ac.uk/14259/.

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Background and Aims: The interleukin 18 (IL-18) system consists of a proinflammatory cytokine (IL-18), a naturally occurring inhibitor (IL-18BP), and a dimeric receptor. IL-18 has been implicated in many autoimmune conditions, with elevated plasma levels predicting future risk of heart disease in healthy individuals. IL-18 also appears to have metabolic consequences, with IL18 -/- mice having higher bodyweight, attributable to increased feeding. The central hypothesis of this work was that genetic variation within both IL-18 and IL-18BP, that influenced their circulating levels, would be associated with individual risk for both heart disease and obesity. This was tested by measuring IL-18 levels in several study groups and accessing their correlation with numerous single nucleotide polymorphisms (SNPs) (chosen using a tagging SNP (tSNP) methodology) within both genes. Any effects (either single SNP or haplotypic) were then assessed directly using quantitative PCR (QPCR). Results: For both IL-18 and IL-18BP, tSNP sets were chosen that captured greater than 90% of the common genetic variation seen in a representative European Caucasian sample. Their association with levels of IL-18 and interleukin 18 binding protein (IL-18BP) were assessed in healthy and diseased individuals; genetic variation in IL-18 only was associated with differences in plasma IL-18 (and free IL-18 (fIL-18)) levels in all cohorts. Carriage of the rare allele of IL-18-5848T>C was associated with, on average across all study groups, 44% higher IL-18 levels than TT homozygotes. There was also a haplotypic effect, with a common haplotype - hGTATA (frequency of 20%) - being associated with 30% lower IL-18 levels. The effect was consistently of greater magnitude in diseased than healthy individuals. Despite IL-18 levels being elevated in those individuals who went on to suffer a myocardial infarction (MI) over 15 yr of follow-up (277.6 pg/ml vs. 239.6 pg/ml, p=0.05), there was no significant difference in genotype, or haplotype, frequencies in those who had heart disease compared to those who did not. No consistent association between BMI/obesity and IL-18 levels was observed, however, in two of the study groups genetic variation in IL-18 was associated with significant differences in body mass index (BMI). Overall, hGTATA was associated with a 9% higher BMI, equating to a 7 kg greater body weight for an average male. In in vitro studies of IL-18 expression in healthy, male hGTATA carriers, no significant difference in IL-18 messenger RNA (mRNA) concentrations were observed when compared to hGCATA controls. Conclusion: The data presented here disagrees with previously published results, showing no role for variation within IL-18 in establishing heart disease risk. However there are novel findings that suggest it may play a role in weight control. Given that these effects appear not to be mediated through plasma IL-18 levels, the identification of individuals through genetic testing may be especially relevant.
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Yang, Bo, and 杨波. "Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47869641.

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Obesity is closely related to many medical complications such as type 2 diabetes, hypertension and heart failure. Obesity and other factors, including elevated blood glucose levels, hypertension, and dyslipidemia, constitute a constellation of symptoms known as the metabolic syndrome, which are the risk factors for coronary artery disease. Lipocalin-2 is a pro-inflammatory adipokine causally involved in the development of obesity-associated metabolic and cardiovascular diseases. Recent clinical and experimental evidences demonstrate an association between augmented circulating lipocalin-2 and cardiac dysfunction. However, little is known about the detailed roles of lipocalin-2 in regulating pathophysiological functions of the heart. The present study was designed to compare the heart functions of mice with normal (WT) or deficient lipocalin-2 (Lcn2-KO) expression and to examine the molecular mechanisms underlying lipocalin-2-mediated deteriorated effects in hearts. Echocardiographic analysis revealed that the myocardial contractile function was significantly improved in hearts of Lcn2-KO mice, under both standard chow and high fat diet conditions. The heart function before and after I/R injury (20-min of global ischemia followed by 60-min of reperfusion) was assessed using the Langendorff perfusion system. Compared with WT littermates, hearts from Lcn2-KO mice showed improved functional recovery and reduced infarct size following I/R. These phenomena can be observed in mice under both standard chow and high fat feeding conditions. Under baseline condition, the mitochondrial function of hearts from Lcn2-KO mice was significantly enhanced, as demonstrated by biochemical analysis of respiratory chain activity, markers of biogenesis and oxidative stress, as well as electron microscopic investigation of the mitochondrial ultrastructure. Acute or chronic administration of lipocalin-2 impaired cardiac functional recovery to I/R and dampened the mitochondrial function in hearts of Lcn2-KO mice. These effects were associated with an extensive modification of the fatty acyl chain compositions of intracellular phospholipids. In particular, lipocalin-2 facilitated the redistribution of linoleic acid (C18:2) among different types of phospholipid, including cardiolipin, which is exclusively located in the mitochondria inner membrane. The direct effects of lipocalin-2 on both H9c2 and NCM cells were also examined. TUNEL assay and flow cytometry analysis demonstrated that lipocalin-2 treatment promoted apoptosis in cardiomyocytes. Lipocalin-2 induced an early phase of phosphatidylserine exposure, followed by Bax-translocation and caspase-3 cleavage. The results collectively suggested that lipocalin-2 initiated the intrinsic mitochondria-mediated apoptotic pathway. In the hearts of Lcn2-KO mice, significantly reduced number of apoptotic cells was observed after I/R injury. In conclusion, lacking of lipocalin-2 improved heart function recovery during I/R injury via mitochondrial function restoration, phospholipids remodeling, and inhibition of cardiomyocytes apoptosis.
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Pharmacology and Pharmacy
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Books on the topic "Cardiovascular system Obesity Chinese"

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Obesity and cardiovascular disease. Oxford: Oxford University Press, 2009.

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Iacobellis, Gianluca. Obesity and cardiovascular disease. Oxford: Oxford University Press, 2009.

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Iacobellis, Gianluca. Obesity and cardiovascular disease. Oxford: Oxford University Press, 2009.

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M, Clifton Peter, and CSIRO (Australia), eds. The CSIRO healthy heart program. Camberwell, Vic: Penguin, 2008.

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Carlot-Tary, Maturine. 1998 Vanuatu non-communicable disease survey report. Nouméa, New Caledonia: Secretariat of the Pacific Community, 2000.

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Zhong yi xin nao bing xue. Beijing Shi: Zhongguo yi yao ke ji chu ban she, 2000.

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Nutritional and metabolic bases of cardiovascular disease. Chichester, West Sussex: Wiley-Blackwell, 2011.

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Association, American Heart, ed. Metabolic risk for cardiovascular disease. Dallas, TX: American Heart Association, 2010.

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C, Somogyi J., and Trichopoulou A, eds. Scientific evidence for dietary targets in Europe: 22nd symposium of the Group of European Nutritionists on the scientific evidence for dietary targets in Europe, Athens, April 9-11, 1984. Basel: Karger, 1986.

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Zhong yao ying xiang xin nao xue guan xi tong huo xing cheng fen: Active components on cardiocerebro-vascular system in TCM. Beijing: Ke xue chu ban she, 2012.

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Book chapters on the topic "Cardiovascular system Obesity Chinese"

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Berra, Cesare C. F., and Mariluce Barrasso. "Thyroid Function and Effects on Cardiovascular System." In Thyroid, Obesity and Metabolism, 221–39. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80267-7_13.

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Das, Sabyasachi. "Effect of Obesity in the Cardiovascular System." In Obesity and its Impact on Health, 67–90. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6408-0_6.

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Engeli, Stefan. "Role of the Adipose-Tissue Renin-Angiotensin-Aldosterone System in Obesity and Obesity-Associated Hypertension." In Nutritional and Metabolic Bases of Cardiovascular Disease, 266–72. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444318456.ch34.

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Maurice, Donald H. "Adaptive and Mal-Adaptive Signaling in Cells of the Cardiovascular System: Effect of Obesity-Associated Peptides on Human Blood Platelet Activation." In NATO Science for Peace and Security Series A: Chemistry and Biology, 185–89. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6513-9_15.

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"Effects of Obesity on the Cardiovascular System." In Handbook of Obesity, 841–60. CRC Press, 2003. http://dx.doi.org/10.3109/9780203913376-38.

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Al-Shura, Anika Niambi. "Cardiovascular system." In Mechanisms of Action in Disease and Recovery in Integrative Cardiovascular Chinese Medicine, 13–20. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-817578-1.00003-3.

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Iacobellis, Gianluca. "Introduction: redefinition of the relationship between obesity and the cardiovascular system." In Obesity and Cardiovascular Disease, 1–4. Oxford University Press, 2009. http://dx.doi.org/10.1093/med/9780199549320.003.0001.

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Pacher, Pál, and George Kunos. "The Endocannabinoid System and Cardiovascular Disease." In Abdominal Obesity and the Endocannabinoid System, 179–83. CRC Press, 2008. http://dx.doi.org/10.3109/9781420060850-25.

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"Abdominal Obesity, Metabolic Syndrome and Risk of Cardiovascular Disease." In Abdominal Obesity and the Endocannabinoid System, 1–9. CRC Press, 2008. http://dx.doi.org/10.3109/9781420060850-2.

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Chow, Wing-Sun, Michele AM Yuen, Annette WK Tso, Bernard MY Cheung, Lawrence SC Law, Su-Vui Lo, and Karen SL Lam. "C-Reactive Proteinvs.Other Obesity-Related Biomarkers in the Prediction of Cardiovascular Risk: A 6-Year Prospective Study in Hong Kong Chinese." In CLINICAL - Obesity: Causes & Consequences, P3–410—P3–410. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part3.p35.p3-410.

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Conference papers on the topic "Cardiovascular system Obesity Chinese"

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Ruan, Runxue, Muqing Deng, and Cong Wang. "Implementation of a flexible and extensible clinical data management system for cardiovascular disease." In 2016 35th Chinese Control Conference (CCC). IEEE, 2016. http://dx.doi.org/10.1109/chicc.2016.7554849.

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Dodson, Reuben B., Kendall S. Hunter, and Virginia L. Ferguson. "Elastic Properties of the Human Umbilical Cord in Preeclampsia." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53673.

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Maternal diseases of pregnancy have been found to detrimentally affect the fetal circulatory system, with consequences lasting well into adulthood. In 1995, Barker introduced the idea that major disorders of adult life (including coronary heart disease, hypertension, stroke and diabetes) arise not only through an interaction between factors in our lifestyle, such as a high-fat diet, obesity and smoking, and a genetically determined susceptibility but also through development in utero [1]. Epidemiological evidence continues to support the notion that adult cardiovascular disease (CVD) has fetal origins [2–5].
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Singal, Ashish, Clarence Ojo, and Rumi Faizer. "Characterization of Pulsatility and Temperature Profile During Reactive Hyperemic Response." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6805.

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Patients with peripheral arterial disease (PAD) have compromised blood flow to their extremities as a result of arterial narrowing. PAD is often associated with impairment in endothelial function which is exaggerated by injury from processes related to cardiovascular risk factors such as ageing, hypertension, hyperlipidemia, diabetes, smoking, and obesity [1]. Furthermore, patients with diabetes often have calcified arteries making standard non-invasive testing non diagnostic [2]. With increase in diabetes prevalence and concomitant PAD, a new non-invasive assessment method of arterial function that has the potential to reflect both arterial tone and response to ischemia reperfusion may be valuable. We have developed a peripheral arterial tonometry (PAT) system (previously described, [3]) that is capable of measuring pulsatility in peripheral digits. We complemented our system with simultaneous peripheral temperature measurements that could not only add value in understanding PAD, but also aid in clinical diagnoses. In this investigation, we characterized our system on healthy individuals before using it on patients suffering from arterial disease in future investigations.
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Wang, Wei, Hamada A. Aboubakr, James Vang, Victor Brenk, Sagar M. Goyal, and James Collins. "Nanomagnetic Biosensor for the Detection of Porcine Interferon Gamma." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3375.

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Abstract:
Due to the anatomical and physiological similarities to humans that include similar heart size, flow rate, skin, liver enzymes and bone healing, porcine models as a powerful investigational platform have been widely used in research areas such as diabetes, obesity and islet transplantation [1]. The advantages of relative low cost, ease in handling and comparatively short period of breeding time may make swine provide a promising solution to the shortage of human donors and difficulty in isolating purified islets from adult human in future. Porcine cytokines play a significant role in innate immunity, apoptosis, angiogenesis, cell growth and differentiation. They are involved in cellular responses, maintenance of homeostasis, and disease states such as inflammatory disease, cardiovascular disease, and cancer. Thus, the technologies to analyze the expression of cytokines are developed rapidly and are still hot topics. The traditional approach for cytokine detection and quantification is the use of an enzyme-linked immunosorbent assay (ELISA). However, its inability to do multiplex test calls for more robust detection system. Biochip-based assay for the detection of biological agents using giant magnetoresistive (GMR) sensors and magnetic nanoparticles have emerged recently [2, 3]. It is proved that the nanomagnetic biosensor technology has advantages of low cost, high sensitivity, multiplexity, and real-time signal readout. The integration of GMR biosensor and use of weak magnetic fields allow to eventually realize point-of-care and portability. In addition, interferon gamma (IFNγ) is one of the most important porcine cytokines, and is associated with a number of autoinflammatory and autoimmune diseases. In this work, IFNγ is selected as a model target for the detection of porcine cytokine using nanomagnetic GMR biosensor.
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