Relevant bibliographies by topics / Carotid endarterectomy

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Carotid endarterectomy'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Carotid endarterectomy"

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AbuRahma, Ali F., Tucker G. Jennings, John T. Wulu, Lisa Tarakji, and Patrick A. Robinson. "Redo Carotid Endarterectomy Versus Primary Carotid Endarterectomy." Stroke 32, suppl_1 (January 2001): 332. http://dx.doi.org/10.1161/str.32.suppl_1.332-a.

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90 Background/Purpose: Several authorities have recently advocated carotid stenting for recurrent carotid stenosis because of the perception that redo surgery carries a higher complication rate than primary carotid endarterectomy (CEA). This study will compare early and late results of reoperations versus primary CEA. Patient Poplulation and Methods: All redo operations for recurrent carotid stenosis performed during a recent 7-year period by a single vascular surgeon were compared with primary CEA. Since all redo CEAs were done using polytetrafluoroethylene (PTFE) or vein patch closure, only primary CEAs using the same patching were analyzed. A Kaplan Meier life-table analysis was used to estimate stroke-free survival rates and freedom from ≥50% recurrent stenosis. Results: Out of 510 primary CEAs, 265 had PTFE or vein patch closure. One hundred twenty-four reoperations using PTFE or vein patch closure were done during the same period. Both groups had similar demographic characteristics. Indications for reoperations and primary CEAs were symptomatic stenosis in 78% and 58%, and asymptomatic ≥80% stenosis in 22% and 42%, respectively (p<0.001). The 30-day perioperative stroke and transient ischemic attack rates for reoperation and primary CEA were 4.8% versus 0.8% (p=0.015) and 4% versus 1.1%, respectively, with no perioperative deaths in either group. Cranial nerve injury was noted in 17% in reoperation patients versus 5.3% in primary CEA patients, however most of these were transient (p<0.001). The mean hospital stay was 1.8 days for reoperation versus 1.6 days for primary CEA. The cumulative stroke-free survival and freedom from ≥50% recurrent stenosis rates for reoperation at 1, 3, and 5 years were 96%, 91%, 82%, and 98%, 96%, 95%, respectively; and 94%, 92%, 91% and 98%, 96%, 96%, respectively for primary CEA (no statistically significant differences). Conclusions: Reoperation carries higher perioperative stroke and cranial nerve injury rates than primary CEA. However, redo operations are durable and have stroke-free survival rates that are similar to primary CEA. These considerations should be kept in mind when recommending carotid stenting versus reoperation.
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AbuRahma, Ali F., Tucker G. Jennings, John T. Wulu, Lisa Tarakji, and Patrick A. Robinson. "Redo Carotid Endarterectomy Versus Primary Carotid Endarterectomy." Stroke 32, no. 12 (December 2001): 2787–92. http://dx.doi.org/10.1161/hs1201.099649.

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Bick, Carol, and Chris Imray. "Carotid endarterectomy." Nursing Standard 16, no. 3 (October 3, 2001): 47–53. http://dx.doi.org/10.7748/ns2001.10.16.3.47.c3092.

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Ingall, Timothy J., David W. Dodick, and Richard S. Zimmerman. "Carotid endarterectomy." Postgraduate Medicine 107, no. 6 (May 2000): 97–109. http://dx.doi.org/10.3810/pgm.2000.5.15.1093.

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Diaz, Fernando G., and Ghaus M. Malik. "Carotid Endarterectomy." JBNC - JORNAL BRASILEIRO DE NEUROCIRURGIA 4, no. 2-3 (December 22, 2017): 46–54. http://dx.doi.org/10.22290/jbnc.v4i2-3.103.

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Carotid endarterectomy should be considered for patients with symptoms of focal cerebral ischemia, when it can be performed with a combined morbidity and mortality below the annual risk of stroke (5%). The experience with 815 carotid endartectomies perforformed from 1979 to 1992 is presented. There were 530 (65%) men and 285 (35%) women of ages from 34 to 82 (median 65); risk factors included diabetes mellitus 196 (24%), hypertension 554 (68%), and smoking 570 (70%). Clini¬cal presentation consisted of transient ischemic attacks 464 (57%), cerebral infarction with minimal neurological residual 228 (28%), stroke in evolution 2 (0.2%), and asymptomatic stenosis 121 (15%). By Sundts classification of medical risk the groups were: grade I, 106 (13%); grade II, 350 (43%; grade III, 357 (44%); grade IV, 2 (0.2%). All patients received endotracheal anesthesia. Thiopental (3-5 mg/kg) and lidocaine (1 mg/kg) were given for induction and at 15 minutes intervals during carotid cross-clamping. Intraluminal shunts were used in 14 (2%). A conventional (open) endarterectomy was performed in 379 (46%) and a limited endarterectomy (closed) in 436 (54%). Complications included 8 (1%) deaths, 24 (3%) developed a major neurological deficit that persisted, 24 (3%) had perioperative TIAs which resolved completely. Of the patients with preoperative neurological deficits, 32 (4%) recovered. Therefore, at one month after surgery, 782 (96%) were either as well or better than pre-operatively. Of 483 (59%) postoperative angiograms, 40 (5%) showed an internal carotid artery occlusion. Six of these patients developed an immediate postoperative cerebral infarction and one died. Non-neurologic complicalions were: cardiac 40 (5%), peripherail nerve 24 (3%), and local wound problems 16 (2%). A carotid endarterectomy can be performed safely when it is done with meticulous attention to detail and consistent surgical technique.
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Tippett, Troy M., Alton B. Sisco, and Charles E. Chapleau. "Carotid endarterectomy." Journal of Neurosurgery 63, no. 3 (September 1985): 387–89. http://dx.doi.org/10.3171/jns.1985.63.3.0387.

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✓ The authors have reviewed 150 consecutive carotid endarterectomies in 140 patients performed by three neurosurgeons. These were performed in two small community hospitals. There was an overall mortality rate of 1.3%; major or minor stroke was seen in 2.7% of patients and transient neurological dysfunction in 2.7%. Preoperative symptoms included major or minor stroke in 39.3% of patients and transient neurological dysfunction in 43.3%; 17.3% of patients were asymptomatic. The patients were continuously monitored intraoperatively with electroencephalography. There were two operative deaths, both related to myocardial infarction and both on the 2nd postoperative day. These statistics appear to compare favorably with those of series reported by major institutions. The average number of carotid endarterectomies per surgeon per year was 10. These were performed over a 7-year period (October, 1976, through November, 1983). Previous series have implied the need for higher frequency in performing the procedure to assure low morbidity and mortality rates. This series appears to offer evidence to the contrary. A key to these results has been that in 148 of the 150 operations, the primary surgeon has been assisted by one of the other two neurosurgeons. This affords the primary surgeon the benefit of excellent technical assistance, and also broadens the experience of the assisting surgeon, thereby allowing maximum experience from the small volume of cases.
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Lynfield, Joshua. "Carotid Endarterectomy." Annals of Internal Medicine 111, no. 5 (September 1, 1989): 443. http://dx.doi.org/10.7326/0003-4819-111-5-443.

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Cebul, Randall D. "Carotid Endarterectomy." Annals of Internal Medicine 111, no. 8 (October 15, 1989): 660. http://dx.doi.org/10.7326/0003-4819-111-8-660.

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Lederle, Frank A. "Carotid Endarterectomy." Annals of Internal Medicine 112, no. 5 (March 1, 1990): 383. http://dx.doi.org/10.7326/0003-4819-112-5-383.

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Loftus, Christopher M. "Carotid endarterectomy." Postgraduate Medicine 82, no. 5 (October 1987): 241–48. http://dx.doi.org/10.1080/00325481.1987.11700011.

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Dissertations / Theses on the topic "Carotid endarterectomy"

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Gaunt, Michael E. "Assessment of carotid endarterectomy." Thesis, University of Leicester, 1995. http://hdl.handle.net/2381/34353.

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A major cause of perioperative strokes during carotid endarterectomy is intraoperative embolisation. Previous studies have identified that intraoperative embolisation can be detected by monitoring with transcranial Doppler sonography (TCD). However, these studies were unable to demonstrate a convincing link between embolisation and the development of neurological deficits and therefore, the clinical relevance of these TCD detected emboli remained uncertain. This study aimed to accurately determine the incidence and clinical relevance of TCD detected intraoperative embolisation during carotid endarterectomy. To do this 100 consecutive patients undergoing carotid endarterectomy were monitored with TCD. To assess the clinical impact of intraoperative embolisation all patients underwent the following pre- and postoperative assessments; neurological and cognitive function; retinal fundoscopy and automated visual fields; CT and MRI brain scans. During analysis of the intraoperative TCD recordings the operation was divided into its constituent stages and for each stage the number and character of emboli were determined. It was found that the majority of intraoperative emboli were characteristic of air and not associated with an adverse clinical outcome. However, emboli occurring during the dissection and recovery stages of the operation were characteristic of particulate emboli and associated with the development of neurological and cognitive deficits. In particular, gross, persistent particulate embolisation during the recovery phase of the operation heralded early carotid artery thrombosis and was associated with the development of major neurological deficits. The TCD detection of particulate emboli occurred before the development of neurological signs and with early operative intervention to correct the defect, neurological deficit could be avoided. This finding represents an important new clinical application of TCD monitoring and provides direct clinical evidence for the role of platelet emboli in the aetiology of stroke. The second part of the study was concerned with comparing methods of quality control to detect technical defects which may lead to embolisation. The techniques compared were Angioscopy, B-mode ultrasound, continuous wave Doppler and TCD. Angioscopy detected major technical errors in 12 patients (4 intimal flaps, thrombus in 8). TCD detected shunt malfunction in 13 patients (2 potentially serious) in addition to particulate embolisation detected during dissection and recovery. Continuous wave Doppler and B-mode ultrasound images were technically inadequate in 9 and 24 patients respectively and neither technique altered clinical management. Therefore it was concluded, that a combination of TCD monitoring and completion angioscopy provided the maximum yield in terms of diagnosing technical error and establishing the cause of perioperative morbidity and mortality.
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Lennard, Nicola S. "Quality control for carotid endarterectomy." Thesis, University of Leicester, 2004. http://hdl.handle.net/2381/29469.

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The aims of this study are to assess whether the introduction of a rigorous quality control method could produce a sustained reduction in the intraoperative stroke rate in this unit and whether it was feasible and practical to implement such a programme. The second part of this study will assess the incidence of sustained embolisation in the early post-operative period and investigate whether the antiplatelet agent Dextran 40 can help stop this embolisation, potentially preventing carotid artery thrombosis.;A prospective audit of all patients undergoing carotid endarterectomy was performed. The ability to monitor intraoperatively with TCD and perform completion angioscopy was assessed, as was the impact that these quality control techniques had on influencing the surgery. Patients were monitored postoperatively with TCD and any patient who developed sustained embolisation was commenced on an infusion of Dextran 40.;91% had continuous intraoperative TCD monitoring and 94% underwent successful completion angioscopy, a technical error was identified in 5% of angioscopic assessments. The intraoperative stroke rate was 0% during this study. Postoperative monitoring revealed that 5% of patients develop significant embolisation following CEA, Dextran 40 appeared to stop this embolisation. The overall 30-day stroke or death rate following CEA has fallen from 6% prior to 1992 to 2.2% in 1998.;It is possible to implement a quality control programme for CEA and this has been associated with a fall in the overall 30-day death and any stroke rate.
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Wong, John Hoi-Ying. "A regional performance of carotid endarterectomy." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq21224.pdf.

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Webster, Sally E. "Asprin's effectiveness decreases during carotid endarterectomy." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/29538.

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Carotid Endarterectomy (CEA) is a well-established operation which reduces the risk of stroke in patients with atherosclerotic stenosis of the internal carotid artery. Paradoxically, the operation itself carries a risk of peri-operative stroke. This thesis comprises four sections: 1. Intravenous Dextran 40 solution has been shown to reduce the risk of post-operative stroke in patients with high numbers of post-operative emboli. It was hypothesised that Dextran applied locally to the operative field would be as effective as preventing emboli, but have a lower incidence of systemic complications. A randomised trial was carried out comparing Dextran 40 irrigation with conventional Heparinised Saline solution. Post-operative emboli were quantified and compared. 2. Previous work on platelet function during CEA showed an incidental significant increase in aggregation in response to arachidonic acid (the substrate for the Cyclo-oxygenase (COX) pathway) by the end of the operation. Aggregometry performed during CEA showed that the anti-platelet effect of aspirin was significantly reduced during and after surgery. This effect occurred within three minutes of the administration of intravenous unfractionated heparin (UFH) and had never previously been described. It contradicted all knowledge of aspirin's mechanism of action (irreversible acetylation of the Ser-529 residue of COX). 3. Further in vitro, ex vivo and ELISA studies were performed to determine the mechanisms behind the changes in platelet aggregation. 4. Evidence suggests that Low Molecular Weight Heparin (LMWH) causes less platelet activation than UFH. The final part of this thesis describes a pilot randomised trial comparing the effects of LMWH and UFH on the anti-platelet effect of aspirin. This reduction in aspirin's efficacy may be important, not just for patients undergoing CEA, but also may contribute to risk of thrombo-embolic complications in patients undergoing other vascular interventional procedures (surgery, angioplasty, stenting).
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Newman, Jeremy Edward. "Incidence and mechanism of post carotid endarterectomy hypertension." Thesis, University of Leicester, 2014. http://hdl.handle.net/2381/37193.

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Post-endarterectomy hypertension (PEH) is associated with intracranial haemorrhage (ICH), hyperperfusion syndrome stroke and cardiac complications. Whilst well recognised, its pathophysiology is poorly understood. It was hypothesised that pre-operative poorly controlled blood pressure, baroreceptor dysfunction and impairment of cerebral autoregulation might be associated with PEH. Our aim was to investigate these and other pre-operative clinical variables which may be predictive of those who suffer PEH. 106 patients undergoing carotid endarterectomy (CEA) underwent investigations to evaluate the pathophysiology of PEH including; 24-hour ambulatory BP, central aortic BP, baroreceptor sensitivity (BRS), cerebral autoregulation and transcranial Doppler (TCD) measurement of middle cerebral artery blood flow velocity (MCAV); Clinical details, BP readings from the ward, induction of anaesthesia and during surgery, mode of anaesthesia, vasoactive medications and MCAV changes following flow restoration. Patients with PEH (defined as systolic pressure (SBP) > 170mmHg + no symptoms or > 160mmHg with headache/seizure/deficit) were treated according to Unit guidelines. 40/106 required treatment for PEH (26 in recovery, 27 on the ward), while 7 had surges in SBP > 200mmHg on the ward. PEH (recovery/ward) was not associated with pre-operative patient characteristics or TCD variables and was not associated with impaired autoregulation (autoregulation was better preserved in PEH patients (ARI 4.3 +/-1.4 vs. ARI 3.5 +/-1.6 (p=0.03)). PEH was significantly associated with; (i) higher pre-operative BP (peak SBP > 170mmHg = 59% prevalence); (ii) peak SBP > 170mmHg before induction of anaesthesia (61% prevalence) and (iii) impaired BRS (3.4 +/- 1.7ms/mmHg vs. 5.3 +/-2.8ms/mmHg, p=0.001). Length of stay was significantly increased in PEH patients (p < 0.001), while three patients with temporary headache/seizure/deficit and one with delayed ICH required treatment for PEH (p=0.02). PEH was associated with pre-operative poorly controlled BP and impairment of baroreceptor sensitivity. Cerebral autoregulation was better preserved in those who suffered PEH. Within the time constraints of carrying out surgery in the hyper acute period, it is neither achievable nor advisable to aggressively optimise blood pressure prior to surgery. For now the optimal management remains an uncompromising approach to treating PEH.
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Zierler, Brenda Kaye. "Utilization of carotid endarterectomy before and after the North American symptomatic carotid endarterectomy trial (NASCET) report : effects of clinical research results on the care of patients with carotid artery disease /." Thesis, Connect to this title online; UW restricted, 1996. http://hdl.handle.net/1773/7289.

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McMahon, Gregory Scott. "The role of heparin in thromboembolic complications following carotid endarterectomy." Thesis, University of Leicester, 2011. http://hdl.handle.net/2381/9638.

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The importance of platelets emerged from a local research programme, which aimed to reduce the stroke risk associated with carotid endarterectomy (CEA). It had been demonstrated that intra-operative heparinisation induced a transient reversal of aspirin inhibition; platelets were able to aggregate in response to arachidonic acid (AA). It was hypothesized that intra-operative anticoagulation with intravenous low molecular weight heparin (LMWH) instead of unfractionated heparin (UFH) might be associated with a reduction in pleiotropic platelet effects, and that this would result in a reduction of post-CEA embolization, a surrogate marker for stroke risk. A randomized controlled trial recruited 183 patients; 91 randomized to receive standard intra-operative anticoagulation with 5000IU UFH, and; 92 who received 2500IU LMWH intravenously. Studies conducted in sub-populations aimed to investigate the platelet aggregatory responses to AA and adenosine diphosphate (ADP) and the platelet pathways that were active (plasma and serum were assayed for the stable products of platelet metabolism; thromboxane (TXB2) from the cyclo-oxygenase-1 (COX-1) pathway and 12- hydroxyeicosatretraenoic acid (12-HETE) from the 12-lipoxygenase (12-LOX) pathway). To determine how heparin might interact with the platelet, lipase activity, the presence of heparin antibodies and anti-factorXa (FXa) activity were studied. Increases in platelet aggregation to AA and ADP were observed 3 minutes after heparinisation. In response to AA, these increases were similar for both UFH and LMWH, but patients who received UFH demonstrated significantly greater aggregation in response to ADP. Whilst there was no increase in the production of TXB2, there was a significant increase in the generation of 12-HETE. The increase in platelet response was associated with anti-FXa activity, but not with lipase or heparin antibody activity. The intra-operative substitution of LMWH for UFH was associated with a significant reduction in the risk of patients experiencing high-rate embolization post-operatively, and there is an argument for the re-evaluation of anticoagulation during CEA.
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Magee, Timothy Raymond. "The role of bilateral transcranial doppler sonography in carotid endarterectomy." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322523.

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Senaratne, Jawaharlal W. B. "An investigation into genetic and environmental influences on and treatment of end-stage atherosclerotic arterial disease." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365465.

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Chapman, Gordon. "Feasibility of early cerebral haemodynamic testing in patients undergoing carotid endarterectomy." Thesis, University of Leeds, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.446439.

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Books on the topic "Carotid endarterectomy"

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Handelsman, Harry. Carotid endarterectomy. Rockville, MD: U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1990.

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Carotid endarterectomy: Principles and technique. 2nd ed. New York: Informa Healthcare, 2007.

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Loftus, Christopher M. Carotid endarterectomy: Principles and technique. St. Louis, Mo: Quality Medical Pub., 1995.

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L, Browse Norman, Mansfield Averil O, and Bishop C. C. R, eds. Carotid endarterectomy: A practical guide. Oxford: Butterworth-Heinemann, 1997.

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Jenkins, L. C. Anaesthetic management of carotid endarterectomy. London: Lloyd-Luke (Medical Books), 1987.

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Bederson, Joshua B. Treatment of carotid disease: A practitioner's manual. Park Ridge, Ill: The American Association of Neurological Surgeons, 1998.

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Handbook of carotid artery surgery: Facts and figures. Boca Raton, Fla: CRC Press, 1989.

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MD, Henry Michel, ed. Angioplasty and stenting of the carotid and supra-aortic trunks. London: Martin Dunitz, 2004.

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Rosenberg, Norman. CRC handbook of carotid artery surgery: Facts and figures. 2nd ed. Boca Raton, FL: CRC Press, 1994.

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Solomon, Neil Andrew. The risk of carotid endarterectomy in the elderly: An application of claims based research. [New Haven: s.n.], 1990.

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Book chapters on the topic "Carotid endarterectomy"

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Singer, Robert J., Imad Khan, and Brandon Root. "Carotid Endarterectomy Part 2: Endarterectomy." In Carotid Endarterectomy. Touch Surgery Simulations, 2014. http://dx.doi.org/10.18556/touchsurgery/2014.s0027/p2.

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Singer, Robert J., Imad Khan, and Brandon Root. "Carotid Endarterectomy Part 1: Patient Preparation and Opening." In Carotid Endarterectomy. Touch Surgery Simulations, 2014. http://dx.doi.org/10.18556/touchsurgery/2014.s0027/p1.

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Singer, Robert J., Imad Khan, and Brandon Root. "Carotid Endarterectomy Part 3: Closure." In Carotid Endarterectomy. Touch Surgery Simulations, 2014. http://dx.doi.org/10.18556/touchsurgery/2014.s0027/p3.

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"6 Benign Arteriogram-Bad Ulceration." In Carotid Endarterectomy, 84–85. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-10.

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"74 Tying Together—Evacuation of Air—Right Carotid Exposure." In Carotid Endarterectomy, 268–69. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-100.

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"75 Blunt Needle to Evacuate Air and Debris as Final Step-Patch Graph." In Carotid Endarterectomy, 270–71. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-101.

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"76 Sequence of Clamp Removal at Completion of Arteriotomy." In Carotid Endarterectomy, 272–73. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-102.

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"77 Doppler Examination of Repair—Left Carotid Exposure." In Carotid Endarterectomy, 274–75. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-103.

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"78 Completed Dry Repair Without and With Hemashield Patch Graft." In Carotid Endarterectomy, 276–83. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-104.

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"79 Y-Shaped Suture Line-No Patch Graft." In Carotid Endarterectomy, 284–85. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016277-105.

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Conference papers on the topic "Carotid endarterectomy"

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Silva, Tibério Alves da, Ana Carolina Soares de Lira, Bárbara Letícia Barreto Ramos Aragão, and Luciana Karla, Dayanna Grazielle Maia Viana. "Carotid endarterectomy as the treatment of choice for clearing the internal carotid artery in transitional ischemic attacks." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.303.

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Introduction: TIAs are ischemia, caused by stenosis of the carotid and vertebral arteries. Those who have a TIA are at risk of ischemic stroke and myocardial infarction, with carotid endarterectomy being an intervention. Objective: To analyze the benefits of carotid endarterectomy using drugs. Methods: Literature review, in bases such as PUBMED, MEDLINE, descriptors: “Endarterectomy”, “Ischemic Attack”, with operator “AND” and “OR”. Those with two descriptors were selected in the summary and date between 2010-2020, English / Portuguese language, resulting in: 17 articles. Results: The internal carotid artery (ICA) is located in the neck as a branch of the common carotid artery, being one of its branches the middle cerebral artery (MCA), the main artery affected in strokes and TIAs. Thus, ACI ischemia causes a risk of thrombosis in MCA, the treatment of carotid stenosis requires drugs to prevent atheroma, as well as antiplatelet drugs to reduce embolic events¹. In some cases, carotid endarterectomy or carotid stent implantation is complementary. Therefore, patients with TIA or stroke, who have “transient, fluctuating or persistent unilateral motor weakness or speech disorder or eye symptoms”, should undergo endarterectomy if they have moderate-severe stenosis of the extracranial internal carotid artery in the first days of presentation². Thus, endarterectomy is the treatment of choice and stenting should only be offered to symptomatic patients. Conclusion: Therefore, endarterectomy has been shown to be safe for patients with internal carotid artery stenosis, indicating the prevalence in relation to the stent.
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Meek, A. C., P. Jarvis, R. A. Harper, and C. N. McCollum. "PATCH ANGIOPLASTY INCREASES PLATELET DEPOSITION FOLLOWING CAROTID ENDARTERECTOMY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643953.

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Patch angioplasty with vein or Dacron may be required to prevent narrowing when the arteriotomy is closed following carotid endarterectomy. We studied the effect of such patches on intraluminal thrombus formation by measuring radiolabelled platelet uptake in patients following simple closure and patch angioplasty.Following unilateral carotid endarterectomy in 33 patients, the arteriotomy was closed by direct suture in 23, Dacron patch in 6 and saphenous vein patch in 4. Autologous 111In-platelets were infused on the second postoperative day and platelet uptake over the carotid measured on daily gamma camera images for 3 days. Radioactivity over the operated and contralateral carotids were compared as the Carotid Uptake Ratio.Mean (±sem) counts per gamma camera cell at 24 hours in the operated carotid of 43.6±3.0 were consistently higher than the reference artery of 35.4±2.4 (p<0.001). The overall carotid uptake ratio was 1.21±0.04 with that of 1.41±0.07 in patch angioplasty significantly higher than 1.14±0.04 found following standard arteriotomy closure (p<0.01). All 10 patch angioplasties were easily visible as "hot spots" on gamma camera compared to only 11 out of 23 with simple closure but there was no significant difference between platelet uptake with vein or Dacron patches with ratios of 1.47±0.35 and 1.37±0.15 respectively (p>0.5).The greater local platelet accumulation with patch angioplasty may be due to turbulence from excessive widening of the vessel combined with the thrombogenic surface following endarterectomy. Until the significance of platelet accumulation is established platelet inhibitory therapy should be considered when patch closure is unavoidable.
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Meek, A. C., P. Jarvis, C. M. Backhouse, CN McCollum, and RM Greenhalgh. "PLATELET DEPOSITION AFTER CAROTID ENDARTERECTOMY DECREASES WITH TIME." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643480.

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Platelets are deposited on the exposed media following carotid endarterectomy and will continue to accumulate until neointima covers this thrombogenic surface. Radiolabelled platelet uptake was measured to assess the time to intimal repair.Autologous llllndium labelled platelets were infused 2 days and 2 months postoperatively in 10 patients undergoing unilateral carotid endarterectomy. Platelet accumulation was measured daily by gamma camera images counting radioactivity over the operated artery and comparing it to the contralateral side as Carotid Uptake Ratio (CUR).Mean (±sem) counts per gamma camera cell over the operated side at 24 hours were 46.3± 4.3 compared to 38.6± 3.9 on the unoperated side (p<0.001). At 2 months this difference had disappeared with counts of 38.8± 3.2 and 39.1± 3.2 over the operated and reference arteries respectively. Early postoperative CUR at 1.22± 0.04 was significantly higher than 1.01± 0.06 at 2 months which equates to no radiolabelled platelet uptake (p< 0.01). Radioiabellea platelet uptake was visible on 8 of the 10 early scans, but this was seen in only 2 patients at 2 months, both of whom had a persistently high CUR indicating continued platelet accumulation at that time.Early postoperative platelet deposition decreases in the weeks following carotid endarterectomy presumably due to the development of a neointima. Those cases with persistently high platelet accumulation may have luminal thrombus which could lead intimal hyperplasia and restenosis.
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Gibbs, A., and I. Hendrickson. "Unilateral Reversible Posterior Leukoencephalopathy Syndrome After Carotid Endarterectomy." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6662.

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Barbera, Gaetano La, Serena Pisanello, Paola Wiesel, Luca Cimoli, Roberto Prunella, Mariapia Prontera, Angelica Tinelli, Giovanni Boero, Fabrizio Valentino, and Francesco Talarico. "Carotid Endarterectomy (CEA) In Urgently Admitted Symptomatic Patients." In 70th International Congress of the European Society for Cardiovascular and Endovascular Surgery and 7th International Meeting on Aortic Diseases. Thieme Medical Publishers, Inc., 2022. http://dx.doi.org/10.1055/s-0042-1750968.

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Lynch, A. G., and M. T. Walsh. "Hemodynamic Compromise During Carotid Angioplasty and Stenting." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53441.

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Modern surgical treatment of arterial disease is moving towards minimally invasive procedures, as the benefits are numerous. However, one area that is resisting this trend is the treatment of carotid artery disease. For the past number of decades carotid endarterectomy surgery has been referred to as the “gold standard” in the treatment of carotid artery disease. However, in recent year’s carotid angioplasty and stenting (CAS) has emerged to challenge carotid endarterectomy surgery (CES) as a viable alternative for the prevention of strokes. However uptake of this procedure has been hindered due to the peri-operative complications associated with the treatment. During this procedure blood flow in one of the internal carotid arteries supplying blood to the brain is interrupted for a period of time. However, it has been shown that not all patients can accommodate this interruption. Qureshi et al. suggests that ischemic neurological deficits occur in 3 to 13% of patients as a result of hemodynamic compromise.
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Boekhoven, Renate W., Marcel C. M. Rutten, Marc R. H. M. van Sambeek, and Frans N. van de Vosse. "In Vitro Three Dimensional Imaging of Human Carotid Atherosclerotic Plaques Using Ultrasonography." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53463.

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Ruptured atherosclerotic plaques in the carotid artery are the main cause of stroke (70–80%). To prevent it, carotid endarterectomy is the procedure of choice in patients with a recent symptomatic 70–99% stenosis. Today, the selection of candidates is based on stenosis size only. However, endarterectomy is beneficial for only 1 out of 6 patients [1], the patients with unstable plaques (Fig. 1). Knowledge of mechanical properties of different components in the atherosclerotic arteries is important, because it will allow the identification of plaque stability at an early stage.
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Benger, M., N. Mansoor, S. Sciacca, J. Siddiqui, P. Balasundaram, N. Kandasamy, T. Booth, and J. Lynch. "P63 Carotid stenting versus carotid endarterectomy for symptomatic carotid web: a systematic review and meta-analysis." In ESMINT Abstracts. BMA House, Tavistock Square, London, WC1H 9JR: BMJ Publishing Group Ltd., 2022. http://dx.doi.org/10.1136/neurintsurg-2022-esmint.84.

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Shang, Yu, Ran Cheng, Lixin Dong, Sibu P. Saha, and Guoqiang Yu. "Diffuse Optical Detection of Cerebral Ischemia During Carotid Endarterectomy." In Biomedical Optics. Washington, D.C.: OSA, 2010. http://dx.doi.org/10.1364/biomed.2010.bsud78.

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Pedapati, V., K. Du, A. Mina, A. Bradley, J. Espino, K. Batmanghelich, P. Thirumala, and S. Visweswaran. "Quantitative EEG Changes in Carotid Endarterectomy Correlated with Ischemia." In 2022 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2022. http://dx.doi.org/10.1109/spmb55497.2022.10014953.

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Reports on the topic "Carotid endarterectomy"

1

Singer, Robert, Imad Khan, and Brandon Root. Carotid Endarterectomy. Touch Surgery Simulations, July 2014. http://dx.doi.org/10.18556/touchsurgery/2014.s0027.

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Wu, Songlin, Chen Jia, Yu Zhang, Ran Wang, Ting Ma, Le Zhang, Changxi Ju, et al. Endarterectomy Versus Carotid Stenting for Asymptomatic Carotid Artery Stenosis: An overview of systematic reviews and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, February 2023. http://dx.doi.org/10.37766/inplasy2023.2.0038.

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Li, Wenkui, Rong Deng, Li Li, Chuyue Wu, Lina Zhang, and Shengli Chen. Comparison of perioperative safety of carotid artery stenting and endarterectomy for the Treatment of Carotid Artery Stenosis: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0149.

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