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Books on the topic 'Cataboliti'

Author: Grafiati

Published: 1 April 2023

Last updated: 31 July 2025

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1

Suzuki, Koichi, and Judith S. Bond, eds. Intracellular Protein Catabolism. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0335-0.

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1924-, Suzuki Koichi, Bond Judith S, and International Symposium on Intracellular Protein Catabolism (10th : 1994 : Tokyo, Japan), eds. Intracellular protein catabolism. Plenum Press, 1996.

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3

Revhaug, Arthur, ed. Acute Catabolic State. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-48801-6.

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4

1950-, Revhaug A., ed. Acute catabolic state. Springer, 1996.

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5

Rojas, A. E. Carvajal de. Carbon catabolism in streptomyces venezuelae. UMIST, 1995.

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6

McVitty, Rosalind Shirley. In vitro studies of folate catabolism. The Author], 1997.

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7

Beck, Susan Anne. Catabolic factors in tumour-induced cachexia. Aston University. Department of Pharmaceutical Sciences, 1989.

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8

Floderus, Eugenie. Aminopeptidases and arginine catabolism in oral streptococci. Karolinska Institute, Dept. of Oral Microbiology], 1990.

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9

Floderus, Eugenie. Aminopeptidases and arginine catabolism in oral straptococci. Kongl. Carolinska Medico Chirurgiska Institutet, 1990.

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10

International Symposium on Intracellular Protein Catabolism (6th 1986 Büchenberg (Magdeburg, Germany)). Intracellular protein catabolism: Abstracts of the 6th symposium. Edited by Aurich H, Kirschke Heidrun, Wiederanders Bernd, Proteolysis Group in Halle, and Biochemische Gesellschaft der Deutschen Demokratischen Republik. Martin-Luther-Universität Halle-Wittenberg, 1986.

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11

Jensen, Elmo. Regulation of lipoprotein catabolism in cultured rat hepatocytes. [s.n.], 1988.

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12

Jensen, Mogens Henning. The catabolism of purine nucleotides in the human organism. [s.n.], 1986.

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13

Engel, Norbert G. Chlorophyll catabolism in algae and higher plants: A chemical approach. Department of Chemistry, Universität Freiburg (Schwiez), 2001.

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14

A, Khairallah Edward, Bond Judith S, and Bird John W. C, eds. Intracellular protein catabolism: Proceedings of the Fifth International Symposium on Intracellular Protein Catabolism, held May 29-June 2, 1984, at the Airlie Conference Center, Airlie, Virginia. A.R. Liss, 1985.

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15

Koosha, Homa. Investigations into the expression of catabolic Gene derived from Pseudomonas and Azotobacter. Brunel University, 1986.

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16

Joshi, Harshad. Investigation of the catabolic plasmid involvement in naphthalene-degrading strains of Pseudomonas spp.. University of Wolverhampton, 1995.

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17

Al-Bader, Dhia A. Investigating the role of the oxidative pentose phosphate pathway as the major route of carbohydrate catabolism in the cyanobacterium Synechocystis sp. PCC 6803. typescript, 1999.

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18

Koichi Suzuki Judith S. Bond. Intracellular Protein Catabolism. Springer US, 2011.

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19

Suzuki, Koichi, and Judith S. Bond. Intracellular Protein Catabolism. Springer London, Limited, 2012.

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20

Bond, Judith S. Intracellular Protein Catabolism. Island Press, 1996.

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21

Revhaug, Arthur. Acute Catabolic State. Springer Berlin / Heidelberg, 2012.

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22

Acute Catabolic State. Springer, 2012.

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23

Revhaug, Arthur. Acute Catabolic State. Springer London, Limited, 2012.

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24

Turk, Vito. Intracellular Protein Catabolism II. Springer London, Limited, 2012.

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25

Turk, Vito. Intracellular Protein Catabolism II. Springer, 2012.

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26

KHAIRALLAH, EA. Khairallah Intracellular Protein Catabolism. John Wiley & Sons Inc, 1985.

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27

Dutz, H., A. Graffi, and R. Baumann. 3rd Symposium Intracellular Protein Catabolism. de Gruyter GmbH, Walter, 2022.

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28

Catabolic Diseases ; Nutritional Aspects and Diet Implications. Adhyayan Publishers & Distributors, 2005.

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29

Mottram, Linda-Jayne, and Gavin G. Lavery. The metabolic and nutritional response to critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0202.

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The metabolic response to critical illness is complex and affects every body system. The first phase of this response is characterized by increased hypothalamic pituitary activity and resistance (decreased response) to effector hormones in many target tissues. Cytokines released in the early stages of such illness may be important as they appear to stimulate the hypothalamic pituitary axis directly as part of this ‘stress response’. This phase is considered ‘adaptive’ (helpful), increasing the availability of glucose, free fatty acids, and amino acids as substrates for vital organs. However, i

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30

Sentchilo, Vladimir. Molecular aspects of catabolic gene evolution in pseudomonads. 2003.

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31

Leveau, Johannes Henricus Josephus. Bacterial genetics of catabolic adaptation to chloroaromatic compounds. 1998.

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32

Egreteau, Pierre-Yves, and Jean-Michel Boles. Assessing nutritional status in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0204.

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Decreased nutrient intake, increased body requirements, and/or altered nutrient utilization are frequently combined in critically-ill patients. The initial nutritional status and the extent of the disease-related catabolism are the main risk factors for nutrition- related complications. Many complications are related to protein energy malnutrition, which is frequent in the ICU setting. Assessing nutritional status pursues several different goals. Nutritional assessment is required for patients presenting with clinical evidence of malnutrition, with chronic diseases, with acute conditions accom

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33

Leyn, P. De. Patterns and Site of Adenosine Triphosphate Catabolism in Lung Tissue. Leuven University Press, 1993.

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34

Casaer, Michael P., and Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0032.

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Malnutrition in cardiac and critical illness is associated with a compromised clinical outcome. The aim of nutrition therapy is to prevent these complications and particularly to attenuate lean tissue wasting and the loss of muscle force and of physical function. During the last decade, several well-powered randomized controlled nutrition trials have been performed. Their results challenge the existing nutrition practices in critically ill patients. Enhancing the nutritional intake and the administration of specialized formulations failed to evoke clinical benefit. Some interventions even prov

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35

Casaer, Michael P., and Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0032_update_001.

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Malnutrition in cardiac and critical illness is associated with a compromised clinical outcome. The aim of nutrition therapy is to prevent these complications and particularly to attenuate lean tissue wasting and the loss of muscle force and of physical function. During the last decade, several well-powered randomized controlled nutrition trials have been performed. Their results challenge the existing nutrition practices in critically ill patients. Enhancing the nutritional intake and the administration of specialized formulations failed to evoke clinical benefit. Some interventions even prov

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36

Casaer, Michael P., and Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0032_update_002.

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Abstract:

Malnutrition in cardiac and critical illness is associated with a compromised clinical outcome. The aim of nutrition therapy is to prevent these complications and particularly to attenuate lean tissue wasting and the loss of muscle force and of physical function. During the last decade, several well-powered randomized controlled nutrition trials have been performed. Their results challenge the existing nutrition practices in critically ill patients. Enhancing the nutritional intake and the administration of specialized formulations failed to evoke clinical benefit. Some interventions even prov

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37

Perkins, Edward Joseph. The molecular biology of the halogenated aromatic catabolic plasmid pJP4. 1987.

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38

Revhaug, A. Acute Catabolic State (Update in Intensive Care and Emergency Medicine). Springer-Verlag, 1995.

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39

Intracellular protein catabolism: Proceedings of the Fifth International Symposium on Intracellular Protein Catabolism, held May 29-June 2, 1984, at the ... in clinical and biological research). A.R. Liss, 1985.

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40

Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of nutrition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0201.

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Energy is derived from three major categories of macronutrient—carbohydrate, lipid, and protein. While energy requirements to maintain stable weight can be estimated, it is uncertain if meeting these energy requirements improves outcomes in the critically ill. In health, excess energy is stored via non-oxidative metabolism and during periods of inadequate energy delivery catabolism of storage products occurs. Both storing and using the stores cost energy, each may require up to quarter of energy contained in stored nutrient. Excess carbohydrate stored as glycogen is easily available, albeit in

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41

The catabolism of glucagon-like peptidea 2: A novel intestinal growth factor. National Library of Canada, 1998.

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42

Kraus, Jennifer. Nutrient exchange in the Rhizobium-legume symbiosis: Glutamate catabolism by Rhizobium meliloti. 1987.

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43

Sheehan, Patrick J. Starvation effects on the physiology and morphology of catabolic Pseudomonas putida isolates. 1991.

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44

Christianson, Alan. Catabolism Reset Diet : : Repair Your Liver, Stop Storing Fat, and Lose Weight Naturally. Independently Published, 2022.

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45

Guo, Chao. Concentration of genes involved in aromatic catabolism in fuel oil contaminated and noncontaminated soils. 1994.

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46

Metabolism, Structure and Function of Plant Tetrapyrroles: Introduction, Microbial and Eukaryotic Chlorophyll Synthesis and Catabolism. Elsevier, 2019. http://dx.doi.org/10.1016/s0065-2296(19)x0002-6.

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47

Grimm, Bernhard. Metabolism, Structure and Function of Plant Tetrapyrroles: Introduction, Microbial and Eukaryotic Chlorophyll Synthesis and Catabolism. Elsevier Science & Technology Books, 2019.

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48

Krunic, Nancy. Prostaglandin transport and catabolism in the choroid plexus during perinatal and postnatal development in sheep. 1999.

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49

Rabier, Daniel. Amino Acids. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0083.

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Amino acids present in the different biological fluids belong to two groups: the protein group, with the 21 classical amino acids constituting the backbone of the protein, and the nonprotein group, appearing in different metabolic pathways as intermediate metabolites. It is important to know and to be able to recognize the latter, as they are the markers of many inherited metabolic diseases. Three kinds of pathways must be considered: the catabolic pathways, the synthesis pathways, and the transport pathways. A disorder on a catabolic pathway induces an increase of all metabolites upstream and

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50

Turner, C. Catabolic Starvation : The Microbiota Diet: The Real Reason You Can't Lose Weight & How to Fix It. Independently Published, 2019.

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