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Journal articles on the topic 'Catalepsy'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 June 2025

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1

Andreeva, Tatiana V., Fedor E. Gusev, Nadezhda A. Sinyakova, et al. "An Analysis of Genetic Predisposition to Hereditary Catalepsy in a Mouse Model of Neuropsychiatric Disorders Using Whole-Genome Sequencing Data." Acta Naturae 15, no. 1 (2023): 26–30. http://dx.doi.org/10.32607/actanaturae.11875.

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Catalepsy is a behavioral condition that is associated with severe psychopathologies, including schizophrenia, depression, and Parkinsons disease. In some mouse strains, catalepsy can be induced by pinching the skin at the scruff of the neck. The main locus of hereditary catalepsy in mice has recently been linked to the 105115 Mb fragment of mouse chromosome 13 by QTL analysis. We performed whole-genome sequencing of catalepsy-resistant and catalepsy-prone mouse strains in order to pinpoint the putative candidate genes related to hereditary catalepsy in mice. We remapped the previously describ

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2

Perkin, G. D. "Catalepsy." Journal of Neurology, Neurosurgery & Psychiatry 59, no. 1 (1995): 86. http://dx.doi.org/10.1136/jnnp.59.1.86.

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3

de Moraes Santos Corrêa, Érica, Gustavo Christofoletti, and Albert Schiaveto de Souza. "Effects of Intracerebral Aminophylline Dosing on Catalepsy and Gait in an Animal Model of Parkinson’s Disease." International Journal of Molecular Sciences 25, no. 10 (2024): 5191. http://dx.doi.org/10.3390/ijms25105191.

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Parkinson’s disease (PD) is a progressive disorder characterized by the apoptosis of dopaminergic neurons in the basal ganglia. This study explored the potential effects of aminophylline, a non-selective adenosine A1 and A2A receptor antagonist, on catalepsy and gait in a haloperidol-induced PD model. Sixty adult male Swiss mice were surgically implanted with guide cannulas that targeted the basal ganglia. After seven days, the mice received intraperitoneal injections of either haloperidol (experimental group, PD-induced model) or saline solution (control group, non-PD-induced model), followed

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4

Kamlekar, Sandeep Kumar, and Sangita Gupta. "Effect of methonolic extract of Vitex negundo on haloperidol induced catalepsy in albino mice." International Journal of Basic & Clinical Pharmacology 9, no. 4 (2020): 621. http://dx.doi.org/10.18203/2319-2003.ijbcp20201188.

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Background: Plants are being used in traditional medicine since history of mankind. The knowledge of these medicinal plants has accrued in the course of many centuries leading to medicinal systems in India such as Ayurveda, Unani and Siddha. Objective: In the present study, we evaluated the anticataleptic efficacy of Vitex negundo, a polyherbal formulation in haloperidol induced catalepsy in mice.Methods: Five groups (n=6) of male albino mice were used in the study. Catalepsy was induced by i.p. administration of haloperidol (1 mg/kg). The degree of catalepsy (cataleptic score) was measured as

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Saposnik, G., J. A. Bueri, R. C. Rey, and R. E. P. Sica. "Catalepsy after stroke." Neurology 53, no. 5 (1999): 1132. http://dx.doi.org/10.1212/wnl.53.5.1132.

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6

Bogousslavsky, J., J. Ghika, G. Saposnik, J. A. Bueri, R. C. Rey, and R. E. P. Sica. "Catalepsy after stroke." Neurology 54, no. 8 (2000): 1711–12. http://dx.doi.org/10.1212/wnl.54.8.1711.

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7

Kulikov, A. V. "Hereditary Catalepsy: Genetic and Molecular Mechanisms of Catalepsy in Mice." Russian Journal of Genetics 40, no. 6 (2004): 631–37. http://dx.doi.org/10.1023/b:ruge.0000033310.22993.92.

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8

Rayam, Sriharsha, Babasaheb Laxmansing Kudagi, Madhavulu Buchineni, Rama Mohan Pathapati, and Mallikarjuna Rao Immidisetty. "Assessment of Morus alba (mulberry) leaves extract for anti-psychotic effect in rats." International Journal of Basic & Clinical Pharmacology 8, no. 9 (2019): 2130. http://dx.doi.org/10.18203/2319-2003.ijbcp20194126.

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Background: Morus alba commonly known as white mulberry has been widely cultivated to feed silkworms. This widely grown plant has been in use by tribals of this country for ailments such as asthma, cough, bronchitis, edema, insomnia, wound healing, diabetes, influenza, eye infections and nose bleeds. Various parts of morus alba linn are used as an cardioprotective, hepatoprotective anti-inflammatory, hypoglycemic, free radical scavenging activity and neuro-protective agent. In this study, anti-psychotic property of M. alba leaves extract (MAE) was evaluated by Haloperidol induced catalepsy mod

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9

Kore, Padmaja, Akash Gaikwad, Auradha G. More, Shivanjali Shinde, Rutuja Sawkar, and Poonam Inamdar. "Antihistaminic effects of Azadirachta indica leaves in laboratory animals." International Journal of Ayurvedic Medicine 13, no. 2 (2022): 334–37. http://dx.doi.org/10.47552/ijam.v13i2.2559.

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Azadirachta indica (Meliaceae) leaves have been traditionally used in the management of asthma and the current study was undertaken to scientifically validate the benefits of plant as an antihistaminic agent using the suitable animal model. The agents with antihistaminic properties are known to be good antiasthmatic agents; hence, in the current research work, the antihistaminic activity of an ethanolic extract of Azadirachta indica leaves (at a dose of 250 mg/kg, i.p.) was evaluated using haloperidol-induced catalepsy and clonidine-induced catalepsy in laboratory rats. The results showed that

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10

N. Kale, Ramdas, and Ravindra Y. Patil. "Effect of Solanum melongena Linn. Leaves Extracts on Clonidine Induced Catalepsy and Milk Induced Leukocytosis and Eosinophilia in Mice." Indo Global Journal of Pharmaceutical Sciences 11, no. 02 (2021): 110–14. http://dx.doi.org/10.35652/igjps.2021.112005.

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Objective: To study the Effect of Solanum melongena Linn. leaves extracts on Clonidine induced catalepsy and milk induced Leukocytosis and Eosinophilia in mice. Methods: Methanolic and chloroform extracts of Solanum melongena Linn. leaves were prepared. Preliminary phytochemical screening of the prepared extracts was carried out. Effects of prepared extracts were studied on Clonidine induced catalepsy and milk induced leukocytosis and eosinophila in mice. Results: Maximum duration of catalepsy was observed at 90 min after the clonidine administration. There was significant inhibition (p˂0.05)

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11

Dergachev, Vladimir D., Ekaterina E. Yakovleva, Maria A. Brusina, Evgeny R. Bychkov, Levon B. Piotrovskiy, and Petr D. Shabanov. "Investigation of antiparkinsonian activity of new imidazole-4,5-dicarboxylic acid derivatives on the experimental model of catalepsy." Research Results in Pharmacology 9, no. 1 (2023): 41–47. http://dx.doi.org/10.18413/rrpharmacology.9.10006.

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Introduction: To study the antiparkinsonian activity of new ligands of the glutamate NMDA receptor complex – 1,2–substituted imidazole-4,5-dicarboxylic acids – on an experimental model of catalepsy caused by haloperidolintraabdominal injections in rats. Materials and methods: The experiments were performed on Wistar rats weighing 300-350 g, obtained from the Rappolovo nursery of the Russian Academy of Medical Sciences (Leningrad Region). The animals were kept in standard plastic cages in vivarium conditions with free access to water and food at a temperature of 22±2 °C and in the experiment we

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Adonina, Svetlana, Ekaterina Bazhenova, and Darya Bazovkina. "Effect of Short Photoperiod on Behavior and Brain Plasticity in Mice Differing in Predisposition to Catalepsy: The Role of BDNF and Serotonin System." International Journal of Molecular Sciences 25, no. 5 (2024): 2469. http://dx.doi.org/10.3390/ijms25052469.

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Seasonal affective disorder is characterized by depression during fall/winter as a result of shorter daylight. Catalepsy is a syndrome of some grave mental diseases. Both the neurotransmitter serotonin (5-HT) and brain-derived neurotrophic factor (BDNF) are involved in the pathophysiological mechanisms underlying catalepsy and depressive disorders. The aim was to compare the response of behavior and brain plasticity to photoperiod alterations in catalepsy-resistant C57BL/6J and catalepsy-prone CBA/Lac male mice. Mice of both strains were exposed for six weeks to standard-day (14 h light/10 h d

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Sneha, Nawale 1. *. P. Pranusha1 K. Padma Priya1 M. Ganga Raju 2. "EVALUATION OF ANTI CATALEPTIC ACTIVITY FOR METHANOLIC EXTRACT OF AERVA LANATA WHOLE PLANT." Journal of Pharma Research 8, no. 7 (2019): 520–25. https://doi.org/10.5281/zenodo.3365637.

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<strong><em>ABSTRACT</em></strong> <strong><em>T</em></strong><em>he present study was done to evaluate the anti-cataleptic activity for methanolic extract of </em><em>Aerva lanata</em><em> whole plant (MEAL) in haloperidol induced catalepsy (HIC) and clonidine induced catalepsy (CIC) in mice. Catalepsy was induced by administering haloperidol (1.0 mg/kg bd.wt, i.p.) and clonidine (0.5 mg/kg, bd.wt, </em><em>s.c.</em><em>) daily for 21 days. The anti-cataleptic effect of </em><em>Aerva lanata</em><em> was evaluated by standard bar test, locomotor activity, rota rod test, and cook’s pole

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14

Bhattacharyya, Bhudev, Sujan K. Bala, and Nirmal C. Sukul. "Differentiation of potencies of Agaricus muscarius by experimental catalepsy." British Homeopathic Journal 76, no. 03 (1987): 122–25. http://dx.doi.org/10.1016/s0007-0785(87)80057-1.

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Abstract Agaricus m., administered orally to rats subjected to restraint to induce catalepsy, enhanced the cataleptic state. The higher the potency the longer its duration of peak action and the longer did it take to reach the peak effect. The action of atropine sulphate which diminishes catalepsy, was suppressed by Agaricus m. The degree of suppresion increased with the increase in potency of Agaricus m. Since restraint-induced catalepsy is mediated by cholinergic-dopaminergic interactions in the brain, Agaricus m. is thought to produce its effect by influencing those systems. The work provid

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15

Sabat, Prasant Kumar. "Evaluation of Antiasthamic Activity of Electrohomeopathy Formulation Pettorale on Experimental Animals." Journal of Drug Delivery and Therapeutics 11, no. 6 (2021): 121–26. http://dx.doi.org/10.22270/jddt.v11i6.5165.

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Objective: The current study was planned to evaluate the antiasthamatic effect of Electrohomeopathic medicine Pettorale in various experimental models. Methods: The antiasthmatic activity of Electrohomeopathic medicine Pettorale was studied on different experimental animals like histamine induced bronchospasm in guinea pig, haloperidol induced catalepsy in rats, egg albumin induced paw anaphylaxis in rats and milk induced leukocytosis in mice. Conclusion: Preliminary phytochemical screening has revealed the presence of alkaloids, glycosides, carbohydrates, amino acids, proteins, st

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16

Wolf, Harald, Ulrich Bässler, Roland Spieß, and Rolf Kittmann. "The femur–tibia control system in a proscopiid (Caelifera, Orthoptera): a test for assumptions on the functional basis and evolution of twig mimesis in stick insects." Journal of Experimental Biology 204, no. 22 (2001): 3815–28. http://dx.doi.org/10.1242/jeb.204.22.3815.

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SUMMARY The extremely slow return movements observed in stick insects (phasmids) after imposed changes in posture are termed catalepsy. In the literature, catalepsy is treated as a behavioural component of the twig mimesis observed in walking stick insects. It is produced by the high gain of the velocity-sensitive component of the relevant joint control systems and by the non-linear dependency of its time constant on movement velocity. The high gain, in turn, causes the system to work close to instability, and this may have driven the evolution of gain control mechanisms. Although these statem

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17

Bansode, Mira T., V. J. Chaware, and V. K. Redasani. "Evaluation of anticataleptic activity of Hydroxytyrosol on Haloperidol induced Catalepsy in Experimental Animal." Asian Journal of Pharmaceutical Research and Development 9, no. 4 (2021): 75–80. http://dx.doi.org/10.22270/ajprd.v9i4.990.

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Catalepsy is a symptom resulting from problems with the nervous system, and causes muscular rigidity. People with the symptoms may also be less sensitive to touch and have a decreased sensitivity to pain. Catalepsy generally causes people to be unresponsive to speech. It is similar to catatonia, a condition marked by strange movements, lack of movement, and/or general non responsiveness. However, it typically has an underlying physiological cause and does not cause stereotyped movements.

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Mangrulkar, Shubhada, and Dinesh Chaple. "Pharmacological assessments of polyphenolic extract of Cymbopogon citratus leaves in rodent model of parkinson’s disease." Journal of Drug Delivery and Therapeutics 9, no. 2 (2019): 311–15. http://dx.doi.org/10.22270/jddt.v9i2.2414.

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Parkinson's disease (PD) is amongst the most common age-related neurodegenerative disorders. Natural compounds, especially the polyphenols have gained great interest as potential therapeutic agents in recent research. Thus, the present study was designed to evaluate the effect of polyphenolic extract of Cymbopogon citratus (C. Citratus) leaves in animal models of PD. In the given study PD was induced by administration of reserpine (5 mg/kg/day, i.p for 5 consecutive days), haloperidol (1 mg/kg, i.p.), in experimental animals. The symptoms of PD such as tremors, akinesia, rigidity and catalepsy

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19

Rakhimboev, S. D., Z. I. Sanoev, T. T. Khamroev, et al. "SCREENING STUDY OF NEUROTROPIC PROPERTIES OF NEW TRIAZOLE DERIVATIVE." Oriental Journal of Medicine and Pharmacology 02, no. 04 (2022): 12–20. http://dx.doi.org/10.37547/supsci-ojmp-02-04-02.

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In this article, as a continuation of scientific research on the study of the neurological activity of newly synthesized compounds, the results of studies of the neutropic activity of the 1,2,4 triazole derivative are presented. At the same time, the motor and search activity of compound D-378 was studied in the study conditions, the effect on M-cholinergic receptors in the arecoline tremor method, the effect on catalepsy with haloperidol, and anxiolytic activity. Studies have shown that the compound D-378 exhibits m-cholinomimetic and anxiolytic activity, and also increases the motor and sear

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Ferré, S., T. Guix, G. Prat, F. Jane, and M. Casas. "Is experimental catalepsy properly measured?" Pharmacology Biochemistry and Behavior 35, no. 4 (1990): 753–57. http://dx.doi.org/10.1016/0091-3057(90)90354-k.

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21

Guarner, Mireia, Andrée Rochfort, and Josep Vilaseca. "DIAGNOSING DEATH AND RESURRECTING THE MYTH OR REALITY OF CATALEPSY." Medical Science Pulse 14, SUPPLEMENT 1 (2020): 1–8. http://dx.doi.org/10.5604/01.3001.0014.6943.

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Death is medically described as the irreversible loss of essential characteristics and requires irreversible loss of capacity for consciousness and capacity to breathe. Death certification has legal, ethical, philosophical, biological, cultural, and religious characteristics and consequences. Its diagnosis is based on the best available scientific evidence: somatic, circulatory and/or neurological criteria. Even if we accept the determination of death is “scientific” and a set of criteria for its diagnosis have been established, death diagnosis still differs among countries. It is for this rea

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Sayyed, Mateen, Khan Mohib, N. Devanna, and Aziz-ur-Rahman. "Role of methanolic extract of Hemidesmus indicus in haloperidol-induced parkinsonism in albino mice-A preliminary study." International Journal of Advances in Pharmacy Medicine and Bioallied Sciences 1, no. 1 (2013): 2–6. https://doi.org/10.5281/zenodo.6982860.

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Objective: To evaluate & establish the antiparkinsonian activity of methanolic extract of Hemidesmus indicus (MEHI) against haloperidol-induced parkinsonism in Swiss albino mice. Materials & Methods: In the present study the MEHI was prepared by successive solvent extraction (Soxhlet apparatus). Two doses i.e. 75 & 100 mg/kg of MEHI were evaluated for antiparkinsonian activity in haloperidol-induced parkinsonism (catalepsy) in albino mice. Benzhexol was used as standard drug (positive control). Results: Pre-treatment of test animals with Hemidesmus indicus (75 & 100 mg/kg i.p)

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Sharma, Amit K., Sparsh Gupta, Ranjan K. Patel, and Neeta Wardhan. "Haloperidol-induced parkinsonism is attenuated by varenicline in mice." Journal of Basic and Clinical Physiology and Pharmacology 29, no. 4 (2018): 395–401. http://dx.doi.org/10.1515/jbcpp-2017-0107.

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Abstract Background Parkinson’s disease (PD) is a neurodegenerative disorder of the central nervous system (CNS). However, there is no known drug to stop/slow down this neurodegeneration. Varenicline is an anti-smoking drug and has the potential to prevent neurodegeneration. Thus, the present study was designed to evaluate the effect of varenicline in animal models of PD. Methods Levodopa and haloperidol were administered in doses of 30 and 1 mg/kg, intraperitoneally (i.p.), respectively. Group 1 was administered haloperidol; groups 2, 3 and 4 were administered haloperidol along with varenicli

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Bhalsinge, Rahul Raghunath, Rajbhoj Satkar, Sayan Das, Shraddha Yadav, Shah A. S., and P. S. Worlikar. "Role of nicotine in depression through dopaminergic mechanism." International Journal of Basic & Clinical Pharmacology 6, no. 4 (2017): 864. http://dx.doi.org/10.18203/2319-2003.ijbcp20171094.

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Background: There are interesting reports in the literature indicating relationship of smoking /nicotine and depression. Smokers use nicotine to treat depression. The objectives of present study were to evaluate the role of nicotine in depression through Dopaminergic mechanism by using haloperidol induced catalepsy model in rats and to estimate Dopamine level in brain of depressed rats after nicotine and imipramine.Methods: Dopaminergic activity was evaluated in haloperidol induced catalepsy in rats. Levels of dopamine in normal as well as in depressed rats brain was estimated using fluorimetr

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Pathan, Saniya Feroz, and Imtiyaz Ansari. "EVALUATION OF NEUROPHARMACOLOGICAL ACTIVITY OF MEDICINAL PLANT." Asian Journal of Pharmaceutical and Clinical Research 11, no. 11 (2018): 162. http://dx.doi.org/10.22159/ajpcr.2018.v11i11.27505.

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Objective: The objective of the study is to evaluate the antiparkinsonian activity of hydroalcoholic extract of the seeds of Canavalia gladiata (HECG) in zebrafish and Swiss albino mice.Materials and Methods: Catalepsy was induced in zebrafish by exposing them to haloperidol solution. Treatment groups were exposed to bromocriptine and HECG, 30 min before haloperidol exposure at the dose of 2, 5, and 10 μg/mL. Latency to travel from one fixed point to another, time spent near the bottom of the tank, and complete cataleptic time were evaluated to assess behavioral changes. In mice, catalepsy was

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Skripka, M. O., D. V. Maltsev, A. A. Spasov, and M. A. Perfiliev. "PHARMACOLOGICAL ANALYSIS OF THE MECHANISMS OF ACTION OF A OUINOXALINE DERIVATIVE WITH ANXIOLYTIC PROPERTIES." Journal of Volgograd State Medical University 19, no. 3 (2022): 134–40. http://dx.doi.org/10.19163/1994-9480-2022-19-3-134-140.

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The interactions of compound ZDM-81 with some CNS neurotransmitter systems were studied using in vivo methods to evaluate the interaction of the compound with dopaminergic (catalepsy, induced with haloperidol), adrenergic (clonidine hypothermia), GABAergic (test of interaction with flumazenil) and serotonergic (5-hydroxytryptophan hyperkinesis) systems. It was shown that the ZDM-81 compound is able to prolong and potentiate the effects of 5-hydroxytryptophan, reduces catalepsy caused by haloperidol, does not affect the hypothermic effect of clonidine, and the effect of the ZDM-81 compound is b

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27

Amoateng, Patrick, Dorcas Osei-Safo, Kennedy Kwami Edem Kukuia, et al. "Psychotropic Effects of an Alcoholic Extract from the Leaves of Albizia zygia (Leguminosae-Mimosoideae)." Evidence-Based Complementary and Alternative Medicine 2017 (2017): 1–12. http://dx.doi.org/10.1155/2017/9297808.

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Background. Albizia zygia is used in Ghanaian traditional medicine for the management of mental disorders. The present study tested the hypothesis that an extract of the leaves of Albizia zygia (AZE) may possess antipsychotic and antidepressant properties. Method. The novelty- and apomorphine-induced locomotor and rearing behaviours of AZE in mice were explored in an open-field observational test system. The effects of AZE in apomorphine-induced cage climbing test, extract-induced catalepsy, and haloperidol-induced catalepsy on mice were also investigated. Lastly, the forced swimming and tail

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Bais, Souravh, N. S. Gill, and Nitan Kumar. "Neuroprotective Effect of Juniperus communis on Chlorpromazine Induced Parkinson Disease in Animal Model." Chinese Journal of Biology 2015 (March 15, 2015): 1–7. http://dx.doi.org/10.1155/2015/542542.

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We evaluated anti-Parkinson’s activity of methanolic extract of Juniperus communis (MEJC) leaves in chlorpromazine (CPZ) induced experimental animal model. In this study effects of Juniperus communis (100 and 200 mg/kg, i.p.) were studied using various behavior parameters like catalepsy (bar test), muscle rigidity (rotarod test), and locomotor activity (actophotometer) and its effect on neurochemical parameters (TBARS, GSH, nitrite, and total protein) in rats. The experiment was designed, by giving chlorpromazine (3 mg/kg, i.p.) for 21 days to induce Parkinson’s disease-like symptoms. Chlorpro

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Ihme, Hannah, Rainer K. W. Schwarting, and Liana Melo-Thomas. "Low frequency deep brain stimulation in the inferior colliculus ameliorates haloperidol-induced catalepsy and reduces anxiety in rats." PLOS ONE 15, no. 12 (2020): e0243438. http://dx.doi.org/10.1371/journal.pone.0243438.

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Deep brain stimulation (DBS) of the colliculus inferior (IC) improves haloperidol-induced catalepsy and induces paradoxal kinesia in rats. Since the IC is part of the brain aversive system, DBS of this structure has long been related to aversive behavior in rats limiting its clinical use. This study aimed to improve intracollicular DBS parameters in order to avoid anxiogenic side effects while preserving motor improvements in rats. Catalepsy was induced by systemic haloperidol (0.5mg/kg) and after 60 min the bar test was performed during which a given rat received continuous (5 min, with or wi

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Janati, A. Bruce, Naif AlGhasab, Sulaman Almesned, and Mohammed Alharbi. "Ictal catatonia associated with segmental catalepsy." Romanian Journal of Neurology 19, no. 2 (2020): 123–26. http://dx.doi.org/10.37897/rjn.2020.2.12.

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Iancu, O. D., P. Darakjian, B. Malmanger, N. A. R. Walter, S. McWeeney, and R. Hitzemann. "Gene networks and haloperidol-induced catalepsy." Genes, Brain and Behavior 11, no. 1 (2011): 29–37. http://dx.doi.org/10.1111/j.1601-183x.2011.00736.x.

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Neal-Beliveau, B. S., J. N. Joyce, and I. Lucki. "Serotonergic involvement in haloperidol-induced catalepsy." Journal of Pharmacology and Experimental Therapeutics 265, no. 1 (1993): 207–17. https://doi.org/10.1016/s0022-3565(25)38100-0.

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Wadenberg, Marie-Louise, and Philip Seeman. "Clozapine pre-treatment enhances raclopride catalepsy." European Journal of Pharmacology 377, no. 1 (1999): R1—R2. http://dx.doi.org/10.1016/s0014-2999(99)00406-9.

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Klemm, W. R. "The catalepsy of blocked dopaminergic receptors." Psychopharmacology 111, no. 2 (1993): 251–53. http://dx.doi.org/10.1007/bf02245532.

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Saver, Jeffrey L., Penny Greenstein, Michael Ronthal, and M. Marsel Mesulam. "Asymmetric catalepsy after right hemisphere stroke." Movement Disorders 8, no. 1 (1993): 69–73. http://dx.doi.org/10.1002/mds.870080113.

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36

Kelly, P. H., and S. L. Hartgraves. "Mesencephalic reticular site for cholinergically-induced catalepsy and for antichol inergic reversal of neuroleptic-induced catalepsy." Behavioural Brain Research 16, no. 2-3 (1985): 212–13. http://dx.doi.org/10.1016/0166-4328(85)90128-7.

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Santos, José Eduardo Tanus dos, and João Batista T. Rocha. "Tolerância às Metilxantinas e interações com o Sistema Dopaminérgico: possíveis implicações clínicas." Ciência e Natura 16, no. 16 (1994): 113. http://dx.doi.org/10.5902/2179460x26502.

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Recent studies have suggested that methylxanthines could change neuroleptics efficiency, since haloperidol or chlorpromazine-induced catalepsy is reversed by methylxanthines. However, these studies were made with acute administration of the drugs, disregarding the possible development of tolerance to methylxanthines. In the present mini-review of the literature it was observed that few studies investigated the tolerance to methylxanthines, furthermore there is some controversy between them. In our laboratory we found no tolerance to caffeine in relation to the etfect on the rat locomotor activ

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Kommu, Sandhya, Madhu Chandaka, Keerthana Sri Pabbineedi, et al. "Phytochemical screening and central nervous system activity of ethanolic extract of phoenix dactylifera." Journal of Modern Techniques in Biology and Allied Sciences 1, no. 1 (2024): 4–9. https://doi.org/10.70604/jmtbas.v1i1.8.

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Herbal medicine has a long and storied history that includes Indigenous peoples, the Chinese, the Egyptians, and the Greeks. These cultures have a long history of using plants for medicinal purposes, and they have preserved this knowledge via both written records and oral traditions. The antipsychotic activity was assessed using catalepsy caused by haloperidol. When compared to the usual medication (haloperidol), the ethanolic extracts of Phoenix Dactylifera (400 mg/kg & 800 mg/kg) caused dose-dependent catalepsy in rats. The Forced Swim Test is used to assess anxiolytic action. This exper

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Suchitra D, Kilimozhi D, Vishwanath B A, and Venkatesan Natarajan. "Isolation and Characterization of Anti Psychotic Compound From Cassia Occidentalis Leaf." International Journal of Research in Pharmaceutical Sciences 12, no. 1 (2021): 292–98. http://dx.doi.org/10.26452/ijrps.v12i1.4088.

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The aim of the current work was mainly to find the neuroleptic effects of responsible compounds isolated from Cassia occidentalis leaves, on condiontioning avoidance and catalepsy induced psychotic wistar rats. Psychosis was induced by condiontioning avoidance and catalepsy induced in albino wistar rats. An isolated fraction of the ethanolic extract of Cassia occidentalis was ingested orally at 100 mg/kg, p.o dose. Haloperidol was considered as standard neuroleptic drug (10 mg/kg, p.o.). The isolated fractions from the column chromatography showing for the higher neuroleptic activity that faci

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Antwi-Adjei, Meshack, Kofi Oduro Yeboah, James Oppong-Kyekyeku, and Newman Osafo. "Inflammation Modulating Activity of the Hydroethanol Stem Bark Extract of Bombax costatum in Murine Models." Scientifica 2022 (August 22, 2022): 1–10. http://dx.doi.org/10.1155/2022/6882147.

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Bombax costatum (Bombacaceae) is traditionally used as a decoction of the leaves, stem, and root to treat headaches, fever, and oedema that may be associated with inflammatory conditions. Thus, the aim of this study was to evaluate the effect of 70%v/v ethanolic extract of the stem bark of Bombax costatum on acute and chronic inflammation. The effect of Bombax costatum extract (10, 50, 100 mg kg−1, p.o) was studied in prostaglandin E2-induced paw oedema in Sprague–Dawley rats (n = 5). Subsequently, the effect of the extract on clonidine and haloperidol-induced catalepsy was also investigated i

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Kalkman, H. O., V. Neumann, and M. D. Tricklebank. "Clozapine inhibits catalepsy induced by olanzapine and loxapine, but prolongs catalepsy induced by SCH 23390 in rats." Naunyn-Schmiedeberg's Archives of Pharmacology 355, no. 3 (1997): 361–64. http://dx.doi.org/10.1007/pl00004955.

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Bagewadi, Harish G., Rajeshwari ., Patil Banderao V., and Syed Hasan Zahid. "Rotarod test and Catalepsy bar test: behavioral testing and neuromodulation of Aloe vera in MPTP induced Parkinson’s disease animal model." International Journal of Basic & Clinical Pharmacology 7, no. 3 (2018): 494. http://dx.doi.org/10.18203/2319-2003.ijbcp20180663.

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Background: Aloe vera (Family Liliaceae) has been used for the treatment of diabetes, skin disorders and as anti-inflammatory agent. Objectives: behavioral testing of antiparkinsonian effect of Aloe vera in MPTP induced animal model.Methods: Rotarod test and Catalepsy bar test were used for behavioral assessment. Assessment of oxidative stress was done in the striatal region of the brain by reduced glutathione (GSH) measurement.Results: A. vera (200 and 400mg/kg, p.o.) was found to significantly increase the retention time in rota rod test and significantly decrease the latency period in catal

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Baturina, M. V., and E. V. Beyer. "Study of autoimmune mechanisms of neuroleptics action." Glavvrač (Chief Medical Officer), no. 8 (July 25, 2022): 49–50. http://dx.doi.org/10.33920/med-03-2208-16.

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The authors set a goal to evaluate the severity of haloperidol catalepsy depending on the level of autoantibodies to dopamine receptors and dopamine in laboratory rats that were subjected to long-term administration of neuroleptics.

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Mishra, Pooja Solanki, Sapna More, Ashutosh Tiwari, and Savita Vyas. "Evaluation of the effect of piperine per se and its interaction with ondansetron on haloperidol induced catalepsy in Albino mice." International Journal of Basic & Clinical Pharmacology 7, no. 6 (2018): 1119. http://dx.doi.org/10.18203/2319-2003.ijbcp20182092.

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Background: This study aims to evaluate the per se effect of piperine and its interaction with ondansetron on haloperidol induced catalepsy in swiss albino mice.Methods: The piperine crystals were separated from crude extract of Piper nigrum. Catalepsy was induced by haloperidol (1mg/kg, i.p.). Control group received 2% gum acacia (10ml/kg), standard group ondansetron (0.5mg/kg), test group piperine (10mg/kg) and combination group ondansetron plus piperine (0.5mg/kg + 10mg/kg), per oral, respectively. In acute study, drugs were administered only once, one hour prior to the haloperidol administ

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Raj, Chanchal N., A. Balasubramaniam, and Sayyed Nadeem. "Effect of various extracts of Tabernaemontana divaricata on haloperidol induced catalepsy in rats." International Current Pharmaceutical Journal 3, no. 3 (2014): 240–42. http://dx.doi.org/10.3329/icpj.v3i3.17891.

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Parkinsons disease (PD) is one of the neurodegenerative diseases with selective loss of dopamine neurons of the substantia nigra pars compacta. In the present study, anti-cataleptic activity of Tabernaemontana divaricata leaves extracts viz. aqueous and ethanolic at different doses (50, 100 and 150 mg/kg i.p.) were studied using haloperidol (1 mg/kg, i.p.) induced catalepsy in rats which is a useful animal model for screening drugs for Parkinsons disease. Both the extracts were found to reduce catalepsy significantly (P<0.001) as compared to the haloperidol treated rats showing greater ef

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Orlando, Biagio, Alessandra Morano, Emanuele Cerulli Irelli, Martina Fanella, Anna Teresa Giallonardo, and Carlo Di Bonaventura. "Unilateral catalepsy in non-convulsive status epilepticus." Epileptic Disorders 23, no. 5 (2021): 760–64. http://dx.doi.org/10.1684/epd.2021.1323.

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Naidu, P. S., and S. K. Kulkarni. "Quercetin, a bioflavonoid, reverses haloperidol-induced catalepsy." Methods and Findings in Experimental and Clinical Pharmacology 26, no. 5 (2004): 323. http://dx.doi.org/10.1358/mf.2004.26.5.831321.

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Fukutake, Toshio, Keizo Hirayama, and Takayuki Komatsu. "Transient Unilateral Catalepsy and Right Parietal Damage." Psychiatry and Clinical Neurosciences 47, no. 3 (1993): 647–50. http://dx.doi.org/10.1111/j.1440-1819.1993.tb01812.x.

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Weitzenhoffer, André M. "Catalepsy Tests: What Do They Tell Us?" International Journal of Clinical and Experimental Hypnosis 44, no. 4 (1996): 307–23. http://dx.doi.org/10.1080/00207149608416095.

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Kulikov, A. V., D. V. Bazovkina, E. M. Kondaurova, and N. K. Popova. "Genetic structure of hereditary catalepsy in mice." Genes, Brain and Behavior 7, no. 4 (2008): 506–12. http://dx.doi.org/10.1111/j.1601-183x.2008.00387.x.

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