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Journal articles on the topic 'Catamenial epilepsy'

Author: Grafiati

Published: 4 June 2025

Last updated: 23 June 2025

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1

Husna, Machlusil, I. Wayan Arsana Wiyasa, Ria Damayanti, Syah Sembung Wasiso, Fahimma Fahimma, and Kartika Agustina. "DEALING WITH UNCONTROLLED SEIZURE IN CATAMENIAL EPILEPSY: A CASE REPORT." MNJ (Malang Neurology Journal) 9, no. 1 (2022): 73–75. http://dx.doi.org/10.21776/ub.mnj.2023.009.01.16.

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Catamenial epilepsy refers to a seizure pattern that coincides with the menstrual period. Patients with catamenial epilepsy often leads to intractable epilepsy and may have an adverse impact on quality of life. Several medications are the therapeutic options, however, there is uncertainty regarding the best treatment, and these medications often fail to control the seizure. This makes catamenial epilepsy categorized as pharmaco-resistant epilepsy. This paper reported a case of perimenstrual catamenial epilepsy with an uncontrolled seizure problem. Seizures still occur with optimal management, and thereby clinicians must continue to re-evaluate clinical conditions and treatment selection to achieve optimal management. Personalized-based treatment should be considered in catamenial epilepsy management. This report discusses the challenges of catamenial epilepsy, understanding the catamenial process, and dealing with the problem with a practical personalized approach.

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2

Rose, Susan R., and Douglas F. Rose. "Catamenial Epilepsy." Endocrinologist 8, no. 5 (1998): 353–58. http://dx.doi.org/10.1097/00019616-199809000-00007.

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3

Chia, Kee Vui, and Robert John Scarffe. "Catamenial Epilepsy." Australian and New Zealand Journal of Obstetrics and Gynaecology 33, no. 1 (1993): 93–94. http://dx.doi.org/10.1111/j.1479-828x.1993.tb02067.x.

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4

Roca, Catherine, Lilian Gonsalves, and Linda D. Bradley. "Catamenial Epilepsy." Psychosomatics 35, no. 5 (1994): 507–8. http://dx.doi.org/10.1016/s0033-3182(94)71751-5.

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5

Sujin, D., T. Samyuktha, P. Ram Mohan Reddy, R. Vinola, S. Saran Kumar, and D. Hepcy Kalarani. "Catamenial epilepsy." International Journal of Pharmacognosy and Pharmaceutical Sciences 4, no. 1 (2022): 83–85. http://dx.doi.org/10.33545/27067009.2022.v4.i1a.101.

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6

French, Jacqueline A. "Catamenial Epilepsy: The Elusive Condition." Epilepsy Currents 5, no. 3 (2005): 113–14. http://dx.doi.org/10.1111/j.1535-7511.2005.05301.x.

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Frequency of Catamenial Seizure Exacerbation in Women with Localization-related Epilepsy Herzog AG, Harden CL, Liporace J, Pennell P, Schomer DL, Sperling M, Fowler K, Nikolov B, Shuman S, Newman M Ann Neurol 2004;56:431–434 This investigation assessed the frequency of catamenial epilepsy in 87 women who charted seizures and menses during three cycles. Catamenial epilepsy designation was made if two of three cycles showed at least one of three previously defined catamenial patterns. Among ovulatory cycles, average daily seizure frequency was significantly greater during the perimenstrual and preovulatory phases. Among anovulatory cycles, average daily seizure frequency was substantially less during the midfollicular phase than during the remainder of the cycle. Overall, 39.1% of the women had catamenial epilepsy.

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7

&NA;. "Managing catamenial epilepsy." Inpharma Weekly &NA;, no. 984 (1995): 4. http://dx.doi.org/10.2165/00128413-199509840-00005.

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8

Reddy, Doodipala S. "Perimenstrual Catamenial Epilepsy." Women's Health 3, no. 2 (2007): 195–206. http://dx.doi.org/10.2217/17455057.3.2.195.

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9

Karlov, V. A., P. N. Vlasov, and T. S. Eligulashvili. "Dependence of spectral power of EEG rhythms on menstrual cycle phases in women suffering from epilepsy." Neurology Bulletin XXV, no. 1-2 (1993): 62–66. http://dx.doi.org/10.17816/nb105950.

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The effect of menstrual cycle on EEG parameters is investigated in norm, in epilepsy becoming acute in menstrual and perimenstrual periods (catamenial), and in epilepsy whose exacerbation is not connected with menstrual cycle. The ten dencies to the decrease of spectral power in cycle luteal phase in healthy women and women suffering from epilepsy are mentioned. The primary role of spectrum low-frequency components and total power level in neurodynamics of catamenial epilepsy is shown.

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10

Reddy, DS. "Pharmacotherapy of catamenial epilepsy." Indian Journal of Pharmacology 37, no. 5 (2005): 288. http://dx.doi.org/10.4103/0253-7613.16851.

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11

Verrotti, Alberto, Melissa Laus, Giangennaro Coppola, Pasquale Parisi, Angelika Mohn, and Francesco Chiarelli. "Catamenial epilepsy: hormonal aspects." Gynecological Endocrinology 26, no. 11 (2010): 783–90. http://dx.doi.org/10.3109/09513590.2010.490606.

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12

Reddy, D. S. "Pharmacology of catamenial epilepsy." Methods and Findings in Experimental and Clinical Pharmacology 26, no. 7 (2004): 547. http://dx.doi.org/10.1358/mf.2004.26.7.863737.

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13

Bauer, J. "Catamenial Epilepsy and Menopause." Epilepsia 42, no. 4 (2001): 572–74. http://dx.doi.org/10.1046/j.1528-1157.2001.042004572.x.

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14

Lundberg, Po. "Catamenial epilepsy: A review." Cephalalgia 17, no. 20_suppl (1997): 42–45. http://dx.doi.org/10.1177/0333102497017s2013.

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15

Gilad, Ronit, Menachem Sadeh, Abraham Rapoport, Ron Dabby, and Yair Lampl. "Lamotrigine and catamenial epilepsy." Seizure 17, no. 6 (2008): 531–34. http://dx.doi.org/10.1016/j.seizure.2008.02.001.

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16

Reid, B. A., and K. F. Gangar. "Catamenial epilepsy and goserelin." Lancet 339, no. 8787 (1992): 253. http://dx.doi.org/10.1016/0140-6736(92)90066-c.

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17

Haider, Yasser, and DavidB Barnett. "Catamenial epilepsy and goserelin." Lancet 338, no. 8781 (1991): 1530. http://dx.doi.org/10.1016/0140-6736(91)92354-5.

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18

Felton, Elizabeth A., Bobbie J. Henry-Barron, Amanda K. Jan, et al. "The Feasibility and Tolerability of Medium Chain Triglycerides in Women with a Catamenial Seizure Pattern on the Modified Atkins Diet." Nutrients 13, no. 7 (2021): 2261. http://dx.doi.org/10.3390/nu13072261.

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Ketogenic diet therapy (KDT), particularly modified Atkins diet (MAD), is increasingly recognized as a treatment for adults with epilepsy. Women with epilepsy (WWE) comprise 50% of people with epilepsy and approximately one in three have catamenial epilepsy. The purpose of this study was to determine whether adding a medium chain triglyceride emulsion to MAD to target catamenial seizures was feasible and well-tolerated. This was a prospective two-center study of pre-menopausal WWE with a catamenial seizure pattern on MAD. After a 1-month baseline interval with no changes in treatment, participants consumed betaquik® (Vitaflo International Ltd.) for 10 days each menstrual cycle starting 2 days prior to and encompassing the primary catamenial seizure pattern for five cycles. Participants recorded seizures, ketones, and menses, and completed surveys measuring tolerability. Sixteen women aged 20–50 years (mean 32) were enrolled and 13 (81.2%) completed the study. There was 100% adherence for consuming betaquik® in the women who completed the study and overall intervention adherence rate including the participants that dropped out was 81.2%. The most common side effects attributed to MAD alone prior to starting betaquik® were constipation and nausea, whereas abdominal pain, diarrhea, and nausea were reported after adding betaquik®. The high adherence rate and acceptable tolerability of betaquik® shows feasibility for future studies evaluating KDT-based treatments for catamenial seizures.

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19

Martínez Ramírez, Mauricio Andrés, Sandra Milena Sanchez Gutiérrez, Yuly Natalia Guzmán Yara, et al. "Treatment approach to a patient with catamenial epilepsy. Case report." Case reports 8, no. 1 (2022): 105–15. http://dx.doi.org/10.15446/cr.v8n1.91649.

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Introduction: Catamenial epilepsy refers to the worsening or exacerbation of seizures due to hormonal changes during the menstrual cycle. It is thought to be secondary to the neuroactive properties of endogenous steroid hormones and the natural cyclic variation in their serum levels throughout the menstrual cycle. Case presentation: A 31-year-old female patient from Bogotá (Colombia) was admitted to the emergency department due to an episode of tonic-clonic seizure associated with the menstrual period. Since the onset of the seizures was related to menstruation (every 28 days), it was established that the patient had structural focal epilepsy with catamenial features. Advantages of medical vs. surgical treatment were discussed during a multidisciplinary medical board and it was decided to start pharmacological treatment with progestogens, which resulted in complete remission of the seizures as established during a follow-up visit. Conclusions: Catamenial epilepsy should be considered as a cause of epilepsy refractory to antiepileptic medications. Furthermore, it should be approached from a multidisciplinary perspective and its management should be focused on improving the patients’ quality of life.

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20

Alcantara-Boquiren, Marie Janice, and Almaira Pagayao. "Progesterone in Treatment of Catamenial Epilepsy." Fertility & Reproduction 01, no. 04 (2019): 169–79. http://dx.doi.org/10.1142/s2661318219500191.

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Catamenial epilepsy refers to the temporal correlation of seizure exacerbations with the menstrual cycle. Changes in the serum estradiol and progesterone are implicated in this phenomenon, which makes management of epilepsy in women more challenging. This is a case of a 24-year-old nulligravid who was referred to a reproductive medicine specialist due to refractory focal seizures despite antiepileptic pharmacotherapy. EEG confirmed neurologic dysfunction but clinical history, physical examination, cranial CT and MRI ruled out structural, vascular, traumatic, and infectious causes of seizure. Adjunctive hormonal treatment was given, which improved patient symptoms. This report aims to discuss catamenial epilepsy as to its pathophysiology, diagnosis, and management. Better understanding of this disease entity will help address treatment difficulties in epilepsy in female patients and the associated management issues.

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21

Herkes, Geoffrey K. "Drug Treatment of Catamenial Epilepsy." CNS Drugs 3, no. 4 (1995): 260–66. http://dx.doi.org/10.2165/00023210-199503040-00003.

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22

Reddy, Doodipala Samba. "Neuroendocrine aspects of catamenial epilepsy." Hormones and Behavior 63, no. 2 (2013): 254–66. http://dx.doi.org/10.1016/j.yhbeh.2012.04.016.

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23

Kumar, N., M. Behari, G. K. Ahuja, and B. L. Jailkhani. "Phenytoin Levels in Catamenial Epilepsy." Epilepsia 29, no. 2 (1988): 155–58. http://dx.doi.org/10.1111/j.1528-1157.1988.tb04412.x.

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24

Duncan, Susan, Carol L. Read, and Martin J. Brodie. "How Common Is Catamenial Epilepsy?" Epilepsia 34, no. 5 (1993): 827–31. http://dx.doi.org/10.1111/j.1528-1157.1993.tb02097.x.

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25

Herzog, Andrew G., Pavel Klein, and Bernard J. Rand. "Three Patterns of Catamenial Epilepsy." Epilepsia 38, no. 10 (1997): 1082–88. http://dx.doi.org/10.1111/j.1528-1157.1997.tb01197.x.

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26

Mehta, Dhruvi, Yashi Patel, and Monit Shah. "Genethormonal Outlook of Epilepsy: A Case Report Associated with Catamenial Epilepsy." International Journal of Research and Review 10, no. 8 (2023): 437–48. http://dx.doi.org/10.52403/ijrr.20230855.

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Epilepsy can be considered as unprovoked seizures within 24 hours. From decades, seizures shrouded even when CNS was not studied. With the help of case study, we have tried to point out a hypothetical point correlating genetically acquired epilepsy and catamenial epilepsy. So far, studies have been conducted on the influence of female sex hormones upon differently oriented electrical charges in the brain and related complications. The present article describes genetic variations and hormonal changes as two sides of the disease condition. We have tried to explain the mechanism of QARS gene mutation from previously researched data and an evidence-based study of the same has been done for a better understanding of the reader. The article has aimed discussion upon etiological factors including female sex hormones, thyroid hormones, insulin, prolactin, alteration in bone metabolism, adrenal hormones, and genetic epilepsy for better apprehension of pathological conditions. Keywords: Catamenial epilepsy, genetic epilepsy, adrenal hormones, unprovoked seizures

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27

Choi, Hyunmi, Kamil Detyniecki, Carl Bazil, et al. "Development and validation of a predictive model of drug-resistant genetic generalized epilepsy." Neurology 95, no. 15 (2020): e2150-e2160. http://dx.doi.org/10.1212/wnl.0000000000010597.

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ObjectiveTo develop and validate a clinical prediction model for antiepileptic drug (AED)–resistant genetic generalized epilepsy (GGE).MethodWe performed a case-control study of patients with and without drug-resistant GGE, nested within ongoing longitudinal observational studies of AED response at 2 tertiary epilepsy centers. Using a validation dataset, we tested the predictive performance of 3 candidate models, developed from a training dataset. We then tested the candidate models' predictive ability on an external testing dataset.ResultsOf 5,189 patients in the ongoing longitudinal study, 121 met criteria for AED-resistant GGE and 468 met criteria for AED-responsive GGE. There were 66 patients with GGE in the external dataset, of whom 17 were cases. Catamenial epilepsy, history of a psychiatric condition, and seizure types were strongly related with drug-resistant GGE case status. Compared to women without catamenial epilepsy, women with catamenial epilepsy had about a fourfold increased risk for AED resistance. The calibration of 3 models, assessing the agreement between observed outcomes and predictions, was adequate. Discriminative ability, as measured with area under the receiver operating characteristic curve (AUC), ranged from 0.58 to 0.65.ConclusionCatamenial epilepsy, history of a psychiatric condition, and the seizure type combination of generalized tonic clonic, myoclonic, and absence seizures are negative prognostic factors of drug-resistant GGE. The AUC of 0.6 is not consistent with truly effective separation of the groups, suggesting other unmeasured variables may need to be considered in future studies to improve predictability.

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28

Lukić, Stevo. "CATAMENIAL EPILEPSY - UPDATE ON PRACTICAL MANAGEMENT." Acta Medica Medianae 57, no. 4 (2018): 117–21. http://dx.doi.org/10.5633/amm.2018.0416.

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29

Foldvary-Schaefer, N., and T. Falcone. "Catamenial epilepsy: Pathophysiology, diagnosis, and management." Neurology 61, Issue 6, Supplement 2 (2003): S2—S15. http://dx.doi.org/10.1212/wnl.61.6_suppl_2.s2.

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30

Poblano, Adrián, Reyes Haro, and Gerson Angel-Alavez. "Catamenial-sleep epilepsy: A case report." Sleep and Biological Rhythms 11, no. 2 (2013): 139–42. http://dx.doi.org/10.1111/sbr.12014.

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31

Lim, Li-Ling, Nancy Foldvary, Ed Mascha, and Julia Lee. "Acetazolamide in Women with Catamenial Epilepsy." Epilepsia 42, no. 6 (2001): 746–49. http://dx.doi.org/10.1046/j.1528-1157.2001.33600.x.

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32

Murri, L., and R. Galli. "Catamenial epilepsy, progesterone and its metabolites." Cephalalgia 17, no. 20_suppl (1997): 46–47. http://dx.doi.org/10.1177/0333102497017s2014.

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This study was undertaken to determine plasma allopregnanolone (3alpha-hydroxy-5alpha-pregnan-20-one, A-PREG) and pregnanolone (3alpha-hydroxy-5beta-pregnan-20-one, PREG) concentrations in the luteal phase (22nd to 24th day of the ovarian cycle) in 15 women with partial epilepsy and catamenial exacerbation of the seizures in the intercritical phase and in 15 healthy women matched for age in order to determine if an impaired metabolism of 3alpha-hydroxymetabolites of progesterone (P) occurs in this convulsive disorder. No significant differences between the two groups were found with respect to the mean plasmatic A-PREG and PREG levels. A significant correlation was found instead between P and A-PREG or PREG concentrations.

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33

Reddy, Doodipala S., and Michael A. Rogawski. "Neurosteroid replacement therapy for catamenial epilepsy." Neurotherapeutics 6, no. 2 (2009): 392–401. http://dx.doi.org/10.1016/j.nurt.2009.01.006.

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34

Reddy, Doodipala S. "Role of neurosteroids in catamenial epilepsy." Epilepsy Research 62, no. 2-3 (2004): 99–118. http://dx.doi.org/10.1016/j.eplepsyres.2004.09.003.

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35

S. Gulnara, Rakhimbaeva, Sagatov R. Dilshod, and Nadjimitdinov A. Saidkamol. "New perspectives in the course of treatment of catamenial epilepsy." International Journal of Psychosocial Rehabilitation 24, no. 04 (2020): 2280–85. http://dx.doi.org/10.37200/ijpr/v24i4/pr201338.

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36

Yusupova, Dilnoza, and Gulnora Rakhimbaeva. "Genetic markers of catamenial epilepsy in women." Journal of the Neurological Sciences 429 (October 2021): 118265. http://dx.doi.org/10.1016/j.jns.2021.118265.

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37

Reddy, Doodipala S., Hee-Yong Kim, and Michael A. Rogawski. "Neurosteroid Withdrawal Model of Perimenstrual Catamenial Epilepsy." Epilepsia 42, no. 3 (2002): 328–36. http://dx.doi.org/10.1046/j.1528-1157.2001.10100.x.

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38

Herzog, Andrew G. "Catamenial epilepsy: Definition, prevalence pathophysiology and treatment." Seizure 17, no. 2 (2008): 151–59. http://dx.doi.org/10.1016/j.seizure.2007.11.014.

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39

Cagnetti, C., S. Lattanzi, N. Foschi, L. Provinciali, and M. Silvestrini. "Seizure course during pregnancy in catamenial epilepsy." Neurology 83, no. 4 (2014): 339–44. http://dx.doi.org/10.1212/wnl.0000000000000619.

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40

Scharfman, Helen E. "Insight into Molecular Mechanisms of Catamenial Epilepsy." Epilepsy Currents 3, no. 3 (2003): 86–88. http://dx.doi.org/10.1046/j.1535-7597.2003.03305.x.

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41

Herkes, G. K., M. J. Eadie, F. Sharbrough, and T. Moyer. "Patterns of seizure occurence in catamenial epilepsy." Epilepsy Research 15, no. 1 (1993): 47–52. http://dx.doi.org/10.1016/0920-1211(93)90008-u.

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42

Yusupova, Dilnoza, and Fakhmitdin Muratov. "Optimization of treatment of catamenial epilepsy in women." Journal of the Neurological Sciences 429 (October 2021): 119166. http://dx.doi.org/10.1016/j.jns.2021.119166.

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43

Pack, Alison M. "Having Catamenial Epilepsy Equals Fewer Seizures in Pregnancy." Epilepsy Currents 15, no. 3 (2015): 124–25. http://dx.doi.org/10.5698/1535-7597-15.3.124.

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44

Ichida, M., A. Gomi, N. Hiranouchi, et al. "A case of cerebral endometriosis causing catamenial epilepsy." Neurology 43, no. 12 (1993): 2708. http://dx.doi.org/10.1212/wnl.43.12.2708.

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45

Reddy, Doodipala Samba, Jordan Gould, and O. Gangisetty. "A Mouse Kindling Model of Perimenstrual Catamenial Epilepsy." Journal of Pharmacology and Experimental Therapeutics 341, no. 3 (2012): 784–93. http://dx.doi.org/10.1124/jpet.112.192377.

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46

Odintsova, G., W. Khachatryan, and V. Guzeva. "Catamenial epilepsy as a predictor of drug resistance." Journal of the Neurological Sciences 357 (October 2015): e153-e154. http://dx.doi.org/10.1016/j.jns.2015.08.519.

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47

Sadeghi, MaedehMirmohamad, Jafar Mehvari, Mohammad Zare, Mojtaba Akbari, and Mohammadreza Najafi. "Progesterone therapy in women with intractable catamenial epilepsy." Advanced Biomedical Research 2, no. 1 (2013): 8. http://dx.doi.org/10.4103/2277-9175.107974.

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48

Bonuccellia, Ubaldo, Gian B. Melis, Anna M. Paoletti, Piero Fioretti, Luigi Murri, and Alberto Muratorio. "Unbalanced progesterone and estradiol secretion in catamenial epilepsy." Epilepsy Research 3, no. 2 (1989): 100–106. http://dx.doi.org/10.1016/0920-1211(89)90037-5.

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49

Veliog;lu, SK, and M. Qqouml;zmenoglu. "Migraine-Related Seizures in an Epileptic Population." Cephalalgia 19, no. 9 (1999): 797–801. http://dx.doi.org/10.1046/j.1468-2982.1999.1909797.x.

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A relationship between epilepsy and migraine has long been postulated, but the nature of this interaction is still debated. We studied adult patients with epilepsy and investigated the relationship between migraine and epilepsy. Fourteen percent ( n=412) of adult patients with seizures were identified with a diagnosis of migraine. We also found a direct relationship between migraine and epilepsy (a migraine-induced epilepsy) in 1.7% (seven patients) of the patients with seizures. Patients were at increased risk for both conditions if they had migraine with aura and catamenial epilepsy. The seizure began during or shortly after the migraine aura in all of the cases and preceded the headache. Three of four patients who were refractory to management with antiepileptic drugs using either mono or combination therapy improved seizure control with combination antimigraine and antiepileptic drugs.

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50

Mairajuddin, Samar Iltaf, Raheel Muneer Ahmed Channa, and Abubaker Abdul Rahman Shaffi Al Madani. "The menstrual-epilepsy nexus: exploring the relationship between menstrual cycle and epileptic seizures in women." Romanian Journal of Neurology 24, no. 1 (2025): 66–72. https://doi.org/10.37897/rjn.2025.1.11.

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Background. The term “catamenial epilepsy” (CE) describes a pattern of seizures that worsen during specific menstrual cycle phases, with hormonal fluctuations, particularly in progesterone and estrogen, believed to affect neuronal excitability. CE presents a significant challenge for women with epilepsy, as it can complicate treatment and increase seizure frequency. The precise mechanisms behind this hormonal influence on seizure activity remain unclear. Understanding how different menstrual stages impact seizure patterns is critical for improving treatment strategies and quality of life for women with epilepsy. The study aimed to assess the connection between seizure activity in women with epilepsy and menstrual cycle phases, investigating associations between specific phases and seizure frequency. It also sought to determine the prevalence of drug-resistant epilepsy (DRE) in women with catamenial epilepsy (CE) and examine whether CE is more strongly associated with treatment resistance compared to women without cyclical changes. Methods. This systematic review aimed to determine the prevalence of drug-resistant epilepsy (DRE) in women with CE, explore hormonal mechanisms, and assess the relationship between seizure activity and menstrual cycle phases. A comprehensive search of Google Scholar, Cochrane Library, and PubMed identified 1,245 articles, with 45 meeting the inclusion criteria. Results. The findings revealed significant differences in seizure frequency between menstrual phases. The follicular phase had the highest seizure frequency (24%), while the menstrual phase had the lowest (4%) (OR 3.78, 95% CI 2.45–5.82, p < 0.001). Women with CE were nearly four times more likely to have DRE than women without cyclical seizure patterns. Conclusions. The study highlights the need for individualized treatment plans that consider hormonal influences on seizure patterns. Further research into the mechanisms of CE and its impact on treatment strategies is necessary. Integrating hormonal effects into treatment options for women with cyclical seizure fluctuations could improve outcomes.

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