Relevant bibliographies by topics / Cataplexy

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Cataplexy'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Cataplexy"

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Reddy, Abhishek, and Mary Halsey Maddox. "1231 Slurred Speech- Unusual Presentation of Cataplexy in Childhood Narcolepsy." Sleep 43, Supplement_1 (2020): A470. http://dx.doi.org/10.1093/sleep/zsaa056.1225.

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Abstract Introduction Narcolepsy, a chronic neurological disorder of excessive daytime sleepiness, cataplexy, sleep paralysis, and hypnagogic/hypnopompic hallucinations, frequently presents in late childhood/early adolescence. Cataplexy, the most specific symptom, presents as transient loss of muscle tone causing weakness. Approximately 80% of children with narcolepsy present with cataplexy1. Report of Case Eight year old African American boy presented to sleep clinic with concerns for excessive daytime sleepiness and slurred speech. Teachers initially referred to speech therapy because of slu
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Coelho, Fernando Morgadinho Santos, Márcia Pradella-Hallinan, Gabriela Rodrigues Alves, et al. "A study of T CD4, CD8 and B lymphocytes in narcoleptic patients." Arquivos de Neuro-Psiquiatria 65, no. 2b (2007): 423–27. http://dx.doi.org/10.1590/s0004-282x2007000300011.

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Narcolepsy is characterized by excessive daytime sleep and cataplexy. Little is known about the possible difference in pathophysiology between patients with or without cataplexy. OBJECTIVE: To quantify T CD4, T CD8 and B lymphocytes in subgroups of patients with narcolepsy and the presence or absence of the HLA-DQB1*0602 allele between groups. METHOD: Our study was prospective and controlled (transversal) with 22 narcoleptic patients and 23 health control subjects. Patients underwent an all-night polysomnographic recording (PSG) and a multiple sleep latency Test (MSLT). The histocompatibility
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Reading, Paul. "Cataplexy." Practical Neurology 19, no. 1 (2018): 21–27. http://dx.doi.org/10.1136/practneurol-2018-002001.

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Remarkably and almost invariably, the clinical phenomenon of cataplexy results from the loss of around 40 000 hypocretin-containing neurones in the lateral hypothalamus in the context of narcolepsy type 1. Cataplexy reflects the dysregulation of rapid-eye-movement (REM) sleep, such that REM-sleep atonia intrudes inappropriately into wakefulness as brief episodes of either focal or total paralysis of voluntary muscle. The semiology of cataplexy differs between adults and children. A defining and enigmatic aspect is that certain emotional stimuli usually trigger the episodes. Cataplexy can be th
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Cao, Michelle, and Christian Guilleminault. "Cataplexy." Scholarpedia 3, no. 1 (2008): 3317. http://dx.doi.org/10.4249/scholarpedia.3317.

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Siegel, J. M., K. S. Tomaszewski, H. Fahringer, G. Cave, T. Kilduff, and W. C. Dement. "Heart rate and blood pressure changes during sleep-waking cycles and cataplexy in narcoleptic dogs." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 1 (1989): H111—H119. http://dx.doi.org/10.1152/ajpheart.1989.256.1.h111.

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Cataplexy is the abrupt loss of muscle tone experienced by narcoleptics. It is usually precipitated by strong emotions or athletic activity. It has been hypothesized that cardiovascular variables have a role in the triggering of cataplexy. In the present study, we have utilized the narcoleptic canine model to directly investigate changes in heart rate and blood pressure in relation to cataplectic episodes. We found that heart rate increased 18% on average in the 20 s preceding cataplexy onset and then fell during cataplexy. Thus, from a cardiovascular standpoint, cataplexy can be subdivided in
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Egel, Robert T., Anthony Lee, Thomas Bump, and Alexander Javois. "Isolated Cataplexy in the Differential Diagnosis of Drop Attacks: A Case of Successful Clinical Diagnosis and Treatment." Case Reports in Neurological Medicine 2012 (2012): 1–4. http://dx.doi.org/10.1155/2012/757586.

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Drop attacks are sudden spontaneous falls that are not accompanied by alteration of consciousness and are followed by immediate recovery. Cataplexy, which is usually associated with narcolepsy, is one of the causes of drop attacks. We report a patient with the rare condition of cataplexy without associated narcolepsy (isolated cataplexy). Isolated cataplexy should be included in the differential diagnosis when a patient presents with recurrent drop attacks and normal diagnostic test results.
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Mahoney, C. E., W. Zhao, A. Coffey, C. Woods, D. Kroeger, and T. Scammell. "0005 Cataplexy Triggered by Social Cues: A Role for Oxytocin in the Amygdala." Sleep 43, Supplement_1 (2020): A2. http://dx.doi.org/10.1093/sleep/zsaa056.004.

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Abstract Introduction People with narcolepsy type 1 report that cataplexy is triggered most often by positive social experiences such as laughing with friends, yet the mechanisms through which social interaction promotes cataplexy are unknown. We hypothesize a subpopulation of central amygdala neurons that are sensitive to the prosocial neuropeptide, oxytocin (CeAOTR), respond to positive valence and trigger cataplexy. Methods We have used in vivo calcium imaging, chemogenetic and optogenetic approaches to characterize the activity pattern of these neurons and to manipulate their activity stat
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Dauvilliers, Y., N. Foldvary-Schaefer, R. K. Bogan, et al. "0753 Cataplexy-Free Days in a Phase 3, Placebo-Controlled, Double-Blind, Randomized Withdrawal Study of JZP-258 in Adults With Narcolepsy With Cataplexy." Sleep 43, Supplement_1 (2020): A286. http://dx.doi.org/10.1093/sleep/zsaa056.749.

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Abstract Introduction Sodium oxybate (SXB) is a standard of care for the treatment of cataplexy and excessive daytime sleepiness in narcolepsy. JZP-258 is an oxybate product candidate with 92% less sodium. This analysis evaluated cataplexy-free days/week, as a measure of treatment impact, in a placebo-controlled randomized withdrawal study of JZP-258 treatment in patients with narcolepsy. Methods Treatment for cataplexy at study entry included 1) SXB (SXB-only); 2) SXB plus other anticataplectics (SXB+other); 3) anticataplectics other than SXB (other anticataplectics); or 4) cataplexy treatmen
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Coelho, Fernando Morgadinho Santos, Márcia Pradella-Hallinan, Mario Predazzoli Neto, Lia Rita Azeredo Bittencourt, and Sérgio Tufik. "Prevalence of the HLA-DQB1*0602 allele in narcolepsy and idiopathic hypersomnia patients seen at a sleep disorders outpatient unit in São Paulo." Revista Brasileira de Psiquiatria 31, no. 1 (2009): 10–14. http://dx.doi.org/10.1590/s1516-44462009000100004.

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OBJECTIVE: Narcolepsy (with and without cataplexy) and idiopathic hypersomnia, are disorders with common features but with different HLA-DQB1*0602 allele prevalence. The present study describes the prevalence of HLA-DQB1*0602 allele in narcoleptics with and without cataplexy and in patients with idiopathic hypersomnia. METHOD: Subjects comprised 68 patients who were diagnosed for narcolepsy or idiopathic hypersomnia and 23 healthy controls according to the International Classification of Sleep Disorders-2. Subjects comprised 43 patients with narcolepsy and cataplexy, 11 patients with narcoleps
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Lebedeva, A. V., N. V. Pantina, A. P. Efimenko, et al. "Differential diagnosis of cataplexy and generalized epilepsy. Case report." Medical alphabet, no. 12 (August 12, 2024): 20–23. http://dx.doi.org/10.33667/2078-5631-2024-12-20-23.

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Cataplexy is rare disease, the main clinical features of which are loss of muscle tone without loss of consciousness, associated with specific factors such as laughter, and less often fear, anger or crying. The differential diagnosis of epileptic seizures and cataplexy is often difficult because of similar clinical features. Establishing the diagnosis of cataplexy is important because in most cases it is associated with narcolepsy; in addition, misinterpretation of cataplexy can lead to hyperdiagnosis of epilepsy or wrong efficiency mark of antiepileptic drugs. Clinical and instrumental examin
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Dissertations / Theses on the topic "Cataplexy"

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Rovere, Heloisa Helena Dal. "Narcolepsia: avaliação da qualidade de vida e impacto social." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-11022015-153022/.

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Narcolepsia é uma condição neurológica crônica, o principal sintoma é a sonolência diurna excessiva, associada a cataplexia, a paralisia do sono e as alucinações hipnagógicas. Paciente com narcolepsia apresenta dificuldade em manter a atenção e vigilância nas tarefas rotineiras e monótonas, com riscos de acidentes acarretando um sério prejuízo e impacto social nas suas relações de trabalho e sócio-familiares e na percepção da qualidade de vida.Foram avaliados 40 pacientes ( 28 mulheres e 12 homens) com idade média de 42 anos. O presente trabalho teve como objetivo: a) avaliar a percepção da QV
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Newman, Janice. "Pupillometric assessment of excessive daytime sleepiness in narcolepsy-cataplexy." Thesis, University of Ottawa (Canada), 1991. http://hdl.handle.net/10393/7692.

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Ten untreated patients with narcolepsy-cataplexy and ten age and sex matched normals between the ages of 20 and 71 underwent pupillometric analyses immediately prior to each of five Multiple Sleep Latency Test (MSLT) sessions. Although narcoleptics were sleepier in terms of both their Stanford Sleepiness Scale (SSS) ratings and their latencies to sleep onset, the baseline pupil diameter, pupillary light reflex and pupillary orienting response did not differentiate between groups. Narcoleptics did, however, exhibit a significantly greater frequency of spontaneous oscillations in the dark-adapte
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Mullington, Janet. "Sleepiness and daytime sleep in narcolepsy-cataplexy: Chronobiological, napping and performance aspects." Thesis, University of Ottawa (Canada), 1994. http://hdl.handle.net/10393/10317.

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This dissertation deals with the chronobiology of sleep and sleepiness in narcolepsy-cataplexy. The text consists of a series of papers: three research papers, and a theoretical paper, with a technical paper appended. The first research paper presents an ambulatory EEG study in which subjects were free to go about their routine home activities, wearing the portable recorder for 24 hours. The timing and duration of sleep episodes were calculated relative to nocturnal midsleep time. Results demonstrated that the most frequent timing of naps was about 1-1.5 h in advance of that found for normal h
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Oka, Yasunori. "Narcolepsy without cataplexy: 2 subtypes based on CSF hypocretin-1/orexin-A findings." Kyoto University, 2008. http://hdl.handle.net/2433/124226.

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Cresswell, Paul William. "Analysis and modelling of excessive daytime sleepiness in narcolepsy cataplexy syndrome and myotonic dystrophy." Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445955.

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Roman, Alexis. "La narcolepsie de type 1 : une pathologie du sommeil paradoxal ?" Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1329/document.

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La narcolepsie de type 1 (NT1) est une maladie neurologique rare caractérisée par une hypersomnolence diurne et des cataplexies - pertes de tonus musculaire pendant l'éveil provoqué par une émotion forte. Chez l'homme, la NT1 est due à la mort spécifique et postnatale des neurones à orexine (Orex) promoteurs de l'éveil, et est considérée comme une pathologie de l'éveil. Toutefois, les observations cliniques suggèrent une dérégulation du sommeil paradoxal (SP) dans cette pathologie. Les patients NT1 ont une latence d'apparition du SP très courte et de fréquents endormissements en SP. De plus, l
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Clément, Olivier. "L'hypothalamus latéral contiendrait le générateur principal du sommeil paradoxal : arguments neuroanatomiques et pharmacologiques chez le rat." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00739636.

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Les mécanismes neurologiques responsables du déclenchement et de l'homéostasie du sommeil, et du sommeil paradoxal (SP) en particulier, sont l'objet d'un nombre toujours plus important d'études du fait notamment de l'attention croissante portée aux pathologies associées. Les travaux rapportés dans cette thèse s'inscrivent parfaitement dans cette dynamique puisqu'ils ont pour objectif de mieux caractériser les populations neuronales mises en jeu dans la régulation du SP ainsi que leurs interactions. Dans cette optique, nous avons combiné différentes approches techniques complémentaires à savoir
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Mahé, Julien Grimaud Nicole. "Narcolepsie-cataplexie avancées physiopathologiques et thérapeutiques /." [S.l.] : [s.n.], 2005. http://theses.univ-nantes.fr/thesemed/PHmahe.pdf.

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THUMEREL, JEAN-LUC. "Narcolepsie cataplexie et metabolisme de la serotonine." Amiens, 1991. http://www.theses.fr/1991AMIEM079.

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Bernard-Valnet, Raphaël. "Etude des processus auto-immuns dans la narcolepsie avec cataplexie." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30307.

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La narcolepsie avec cataplexie est un trouble du sommeil rare et handicapant. Cette affection est induite par la destruction spécifique des neurones produisant l'orexine et localisés dans l'hypothalamus latéral. Cette pathologie est associée à des facteurs génétiques, environnementaux et biologiques, qui pointent vers une origine auto-immune. Par ailleurs, en 2009, la campagne de vaccination contre le virus grippal H1N1, utilisant le vaccin Pandemrix(r), a aboutit à une augmentation drastique du nombre de cas de narcolepsie en Europe. Les modèles animaux actuels de narcolepsie ne permettent pa
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Books on the topic "Cataplexy"

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Lammers, Gert Jan. Narcolepsy with cataplexy. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0014.

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Narcolepsy with cataplexy is caused by disturbed cerebral hypocretin (also called orexin) transmission. It results in impaired physiological boundaries of wake and sleep stages and their specific components, leading to clinical symptoms such as excessive daytime sleepiness (EDS), impaired sustained attention, disturbed nocturnal sleep, cataplexy, and hypnagogic hallucinations. This chapter discusses the consequences for daily life of the disorder, the diagnostic challenges, particularly the interpretation of the results of the multiple sleep latency test (MSLT), the presumed cause and pathophy
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Narcoplexic. Rolling A Path : : My Own Narcolepsy, Skateboarding, Cataplexy and Lifestyle. Narcoplexic, 2014.

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Luppi, Pierre-Hervé, Olivier Clément, Christelle Peyron, and Patrice Fort. Neurobiology of REM sleep. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0003.

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REM (paradoxical) sleep is a state characterized by rapid eye movements, EEG activation, and muscle atonia. REM sleep behavior disorder (RBD) is a parasomnia characterized by loss of muscle atonia during REM sleep. Cataplexy, a key symptom of narcolepsy, is a striking sudden episode of muscle weakness comparable to REM sleep atonia triggered by emotions during wakefulness. This chapter presents recent results on the neuronal network responsible for REM sleep and explores hypotheses explaining RBD and cataplexy. RBD could be due to a specific degeneration of glutamatergic neurons responsible fo
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Doghramji, Karl. Commonly Encountered Sleep Disorders. Edited by Anthony J. Bazzan and Daniel A. Monti. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190690557.003.0015.

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Complaints related to sleep and wakefulness are some of the most commonly encountered in clinical settings. This chapter reviews specific sleep disorders including insomnia disorder, hypersomnolence disorder, narcolepsy, obstructive sleep apnea hypopnea syndrome, central sleep apnea syndrome and selected parasomnias (nonrapid eye movement sleep arousal disorders and rapid eye movement sleep behavior disorder). These disorders are some of the best characterized and commonly comorbid with other medical and psychiatric disorders. Their defining characteristics, diagnostic modalities, and treatmen
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Calandra-Buonaura, Giovanna, and Pietro Cortelli. Autonomic dysfunction and sleep disorders. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0029.

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Autonomic dysfunctions are frequently associated with sleep disorders, as the autonomic nervous system and sleep are closely related from anatomical, physiological, and neurochemical points of view. The autonomic dysfunctions described in this chapter may result from a common pathogenetic mechanism that affects both the autonomic and the sleep functions, as in fatal familial insomnia, or from a prevalent expression of a primary disorder of autonomic regulation during sleep, as in congenital central hypoventilation syndrome. Alternatively, the autonomic dysfunction may be mainly caused by the s
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Book chapters on the topic "Cataplexy"

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Toftness, Alexander R. "Narcolepsy (and Cataplexy)." In Incredible Consequences of Brain Injury. Routledge, 2022. http://dx.doi.org/10.4324/9781003276937-37.

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Overeem, Sebastiaan. "The Clinical Features of Cataplexy." In Narcolepsy. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8390-9_26.

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Dauvilliers, Yves, and Régis Lopez. "Parasomnias in Narcolepsy with Cataplexy." In Narcolepsy. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8390-9_27.

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Sinton, Christopher M. "Narcolepsy-Cataplexy in the Rats." In Rodent Model as Tools in Ethical Biomedical Research. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-11578-8_23.

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Shahid, Azmeh, Kate Wilkinson, Shai Marcu, and Colin M. Shapiro. "Cataplexy Emotional Trigger Questionnaire (CETQ)." In STOP, THAT and One Hundred Other Sleep Scales. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9893-4_15.

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Nishino, Seiji, Masatoshi Sato, Mari Matsumura, and Takashi Kanbayashi. "Narcolepsy–Cataplexy Syndrome and Symptomatic Hypersomnia." In Sleep Medicine. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2089-1_26.

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Sakurai, Takeshi, Masashi Yanagisawa, and Michihiro Mieda. "Rodent Models of Human Narcolepsy-cataplexy." In Hypocretins. Springer US, 2005. http://dx.doi.org/10.1007/0-387-25446-3_3.

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Mieda, Michihiro, and Masashi Yanagisawa. "Rodent Models of Human Narcolepsy-Cataplexy." In The Orexin/Hypocretin System. Humana Press, 2006. http://dx.doi.org/10.1385/1-59259-950-8:255.

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Ponz, Aurélie, and Sophie Schwartz. "Humor Processing in Human Narcolepsy with Cataplexy." In Narcolepsy. Springer New York, 2008. http://dx.doi.org/10.1007/978-1-4419-0854-4_11.

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Dauvilliers, Yves, and Giuseppe Plazzi. "Diurnal and Nocturnal Sleep in Narcolepsy with Cataplexy." In Narcolepsy. Springer New York, 2008. http://dx.doi.org/10.1007/978-1-4419-0854-4_7.

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Conference papers on the topic "Cataplexy"

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Bartolini, Ilaria, and Andrea Di Luzio. "Towards Automatic Recognition of Narcolepsy with Cataplexy." In the 15th International Conference. ACM Press, 2017. http://dx.doi.org/10.1145/3151848.3151875.

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Augustine, Kurt E., Bruce M. Cameron, Jon J. Camp, Lois E. Krahn, and Richard A. Robb. "Virtual-reality-based system for controlled study of cataplexy." In Medical Imaging 2002, edited by Seong K. Mun. SPIE, 2002. http://dx.doi.org/10.1117/12.466950.

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Acho, M., and D. Burman. "Narcolepsy with Cataplexy in a Patient with Prior Cranial Irradiation." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3486.

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"TREATMENT-INDUCED MANIA IN A PATIENT WITH NARCOLEPSY WITH CATAPLEXY." In 8th World Congress of the World Association of Dual Disorders (WADD) and the 26th Congress of the Spanish Society of Dual Disorders SEPD. SEPD/WADD, 2024. http://dx.doi.org/10.17579/abstractbookdualdisorders-p-034.

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Leppmeier, L. "“Weak with Laughter”: Cataplexy as a Hint for Early Diagnosis of Niemann-Pick Type C (NPC)?" In Abstracts of the 46th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1739713.

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Bogan, Richard, Nancy Foldvary-Schaefer, Roman Skowronski, Abby Chen, and Michael Thorpy. "Long-term Safety During a Clinical Trial of Low-Sodium Oxybate in Participants With Narcolepsy With Cataplexy (P5-13.006)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000202310.

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