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Dissertations / Theses on the topic 'Cataplexy'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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1

Rovere, Heloisa Helena Dal. "Narcolepsia: avaliação da qualidade de vida e impacto social." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-11022015-153022/.

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Narcolepsia é uma condição neurológica crônica, o principal sintoma é a sonolência diurna excessiva, associada a cataplexia, a paralisia do sono e as alucinações hipnagógicas. Paciente com narcolepsia apresenta dificuldade em manter a atenção e vigilância nas tarefas rotineiras e monótonas, com riscos de acidentes acarretando um sério prejuízo e impacto social nas suas relações de trabalho e sócio-familiares e na percepção da qualidade de vida.Foram avaliados 40 pacientes ( 28 mulheres e 12 homens) com idade média de 42 anos. O presente trabalho teve como objetivo: a) avaliar a percepção da QV
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2

Newman, Janice. "Pupillometric assessment of excessive daytime sleepiness in narcolepsy-cataplexy." Thesis, University of Ottawa (Canada), 1991. http://hdl.handle.net/10393/7692.

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Ten untreated patients with narcolepsy-cataplexy and ten age and sex matched normals between the ages of 20 and 71 underwent pupillometric analyses immediately prior to each of five Multiple Sleep Latency Test (MSLT) sessions. Although narcoleptics were sleepier in terms of both their Stanford Sleepiness Scale (SSS) ratings and their latencies to sleep onset, the baseline pupil diameter, pupillary light reflex and pupillary orienting response did not differentiate between groups. Narcoleptics did, however, exhibit a significantly greater frequency of spontaneous oscillations in the dark-adapte
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3

Mullington, Janet. "Sleepiness and daytime sleep in narcolepsy-cataplexy: Chronobiological, napping and performance aspects." Thesis, University of Ottawa (Canada), 1994. http://hdl.handle.net/10393/10317.

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This dissertation deals with the chronobiology of sleep and sleepiness in narcolepsy-cataplexy. The text consists of a series of papers: three research papers, and a theoretical paper, with a technical paper appended. The first research paper presents an ambulatory EEG study in which subjects were free to go about their routine home activities, wearing the portable recorder for 24 hours. The timing and duration of sleep episodes were calculated relative to nocturnal midsleep time. Results demonstrated that the most frequent timing of naps was about 1-1.5 h in advance of that found for normal h
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4

Oka, Yasunori. "Narcolepsy without cataplexy: 2 subtypes based on CSF hypocretin-1/orexin-A findings." Kyoto University, 2008. http://hdl.handle.net/2433/124226.

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5

Cresswell, Paul William. "Analysis and modelling of excessive daytime sleepiness in narcolepsy cataplexy syndrome and myotonic dystrophy." Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445955.

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6

Roman, Alexis. "La narcolepsie de type 1 : une pathologie du sommeil paradoxal ?" Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1329/document.

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La narcolepsie de type 1 (NT1) est une maladie neurologique rare caractérisée par une hypersomnolence diurne et des cataplexies - pertes de tonus musculaire pendant l'éveil provoqué par une émotion forte. Chez l'homme, la NT1 est due à la mort spécifique et postnatale des neurones à orexine (Orex) promoteurs de l'éveil, et est considérée comme une pathologie de l'éveil. Toutefois, les observations cliniques suggèrent une dérégulation du sommeil paradoxal (SP) dans cette pathologie. Les patients NT1 ont une latence d'apparition du SP très courte et de fréquents endormissements en SP. De plus, l
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7

Clément, Olivier. "L'hypothalamus latéral contiendrait le générateur principal du sommeil paradoxal : arguments neuroanatomiques et pharmacologiques chez le rat." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00739636.

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Les mécanismes neurologiques responsables du déclenchement et de l'homéostasie du sommeil, et du sommeil paradoxal (SP) en particulier, sont l'objet d'un nombre toujours plus important d'études du fait notamment de l'attention croissante portée aux pathologies associées. Les travaux rapportés dans cette thèse s'inscrivent parfaitement dans cette dynamique puisqu'ils ont pour objectif de mieux caractériser les populations neuronales mises en jeu dans la régulation du SP ainsi que leurs interactions. Dans cette optique, nous avons combiné différentes approches techniques complémentaires à savoir
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8

Mahé, Julien Grimaud Nicole. "Narcolepsie-cataplexie avancées physiopathologiques et thérapeutiques /." [S.l.] : [s.n.], 2005. http://theses.univ-nantes.fr/thesemed/PHmahe.pdf.

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9

THUMEREL, JEAN-LUC. "Narcolepsie cataplexie et metabolisme de la serotonine." Amiens, 1991. http://www.theses.fr/1991AMIEM079.

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10

Bernard-Valnet, Raphaël. "Etude des processus auto-immuns dans la narcolepsie avec cataplexie." Thesis, Toulouse 3, 2015. http://www.theses.fr/2015TOU30307.

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La narcolepsie avec cataplexie est un trouble du sommeil rare et handicapant. Cette affection est induite par la destruction spécifique des neurones produisant l'orexine et localisés dans l'hypothalamus latéral. Cette pathologie est associée à des facteurs génétiques, environnementaux et biologiques, qui pointent vers une origine auto-immune. Par ailleurs, en 2009, la campagne de vaccination contre le virus grippal H1N1, utilisant le vaccin Pandemrix(r), a aboutit à une augmentation drastique du nombre de cas de narcolepsie en Europe. Les modèles animaux actuels de narcolepsie ne permettent pa
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11

DEL, BANO JEAN-PASCAL. "Retentissement socio-professionnel de la narcolepsie-cataplexie : resultats d'une enquete nationale." Aix-Marseille 2, 1992. http://www.theses.fr/1992AIX20914.

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12

Vidal, Corinne. "Les formes symptomatiques du syndrome de gelineau : etude critique de la litterature a propos d'une observation clinique personnelle." Clermont-Ferrand 1, 1990. http://www.theses.fr/1990CLF13018.

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13

Chétrit, Magali. "Activité mentale au cours du sommeil et à l'endormissement dans la narcolepsie-cataplexie." Montpellier 1, 1996. http://www.theses.fr/1996MON1T008.

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14

RUBIN, DELABROYE BRIGITTE. "Narcolepsie-cataplexie chez l'enfant : a propos de quatre observations, revue de la litterature." Reims, 1993. http://www.theses.fr/1993REIMM003.

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15

Dauvilliers, Yves. "Effets de l'hypocrétine, des facteurs génétiques et de l'âge sur les états de veille et de sommeil : apports de la narcolepsie." Montpellier 1, 2004. http://www.theses.fr/2004MON1T014.

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La complexite de la regulation de la veille et du sommeil normal et pathologique implique la presence de facteurs environnementaux et genetiques qui commencent a etre apprehendes pour ces derniers. La narcolepsie est une maladie modele pour comprendre la dysregulation de ces mecanismes en recherchant notamment des facteurs genetiques predisposant a cette affection et a la severite de son phenotype. Nous avons entrepris une etude sur le role de l'hypocretine-1 dans differentes formes cliniques d'hypersomnie. Des taux bas d'hypocretine-1 dans le liquide cephalo-rachidien s'averent tres specifiqu
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16

Le, Gallou Anne. "Evaluation de la somnolence chez les parents du premier degré de sujets narcoleptiques." Montpellier 1, 1991. http://www.theses.fr/1991MON11194.

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17

Ohayon, Maurice Moyses. "Étude épidémiologique comparative de la somnolence diurne." Lyon 1, 1997. http://www.theses.fr/1997LYO1T352.

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18

Woods, Caroline. "The role of specific central amygdala neurons in emotionally-triggered cataplexy." Thesis, 2018. https://hdl.handle.net/2144/30881.

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Narcolepsy is a neurological disorder characterized by a person’s inability to regulate sleep-wake cycles. Excessive daytime sleepiness and cataplexy are prominent symptoms of narcolepsy. Cataplexy is partial to full body muscle atonia usually brought on by the person with narcolepsy experiencing a positive emotion. Some features of cataplexy resemble those of REM sleep, including similar brain activity and muscle atonia. The neuronal circuit that produces cataplexy has yet to be determined. The similarities between REM sleep and cataplexy support the hypothesis that cataplexy is the result of
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19

Burgess, Christian R. "Role of the Catecholamine and Limbic Systems in Narcolepsy/Cataplexy." Thesis, 2012. http://hdl.handle.net/1807/43377.

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In this thesis I investigated the neural circuits that trigger cataplexy in mice. Specifically, I first addressed the theory that cataplexy is a REM sleep disorder. I then investigated a role for the noradrenergic and dopaminergic systems in murine cataplexy. Finally, I addressed the role of the amygdala in triggering cataplexy. From this work several specific conclusions can be drawn: 1. Cataplexy does not share a common executive mechanism with REM sleep, although the two may share a common mechanism that generates muscle atonia. Muscle tone during REM sleep and cataplexy is similar, how
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20

Tse, Gavin. "Characterizing a Role for Dopamine on Sleep and Cataplexy in Narcoleptic Mice." Thesis, 2008. http://hdl.handle.net/1807/11171.

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Narcolepsy is a disabling sleep disorder that is characterized by persistent sleepiness, and cataplexy – an involuntary loss of waking muscle tone. Cataplexy and narcolepsy are caused by the loss of hypocretin containing neurons in the hypothalamus. However, it is hypothesized that dopamine is also involved in sleep and motor control and plays a role in cataplexy. This study investigated how manipulating dopamine affected sleep and cataplexy in narcoleptic mice devoid of hypocretin. We used d-amphetamine to increase endogenous dopamine levels and quinpirole (D2 agonist) to agonize D2 receptor
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21

Sanghera, Karan Paul. "Pharmacogenetic Inhibition of the Subcoeruleus Region Influences REM Sleep and Cataplexy in Narcoleptic Mice." Thesis, 2013. http://hdl.handle.net/1807/42909.

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Introduction: Cataplexy - the sudden involuntary loss of skeletal muscle tone – is a defining feature of narcolepsy. The current study aimed to determine if cataplexy is influenced by direct manipulation of REM sleep circuitry. We did this by pharmacogenetically inhibiting the REM sleep center, subcoeruleus (Sub-C). Methods: Inhibitory DREADD (hM4D-Gi) was bilaterally targeted to the Sub-C in hypocretin knockout mice (n=7). Intraperitoneal administration of clozapine-n-oxide was used to inhibit Sub-C cells expressing hM4D-Gi. Electrophysiological and behavioral criteria were used to cha
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22

Begovic, Jelena. "Activity patterns of central amygdala neurons in a mouse model of narcolepsy." Thesis, 2019. https://hdl.handle.net/2144/36300.

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Narcolepsy is a disorder of unstable wake and sleep states caused by the lack of orexin neurons which degenerate most likely as a consequence of an autoimmune process. The state instability of narcolepsy includes rapid eye movement (REM) sleep intruding into wake in the form of dream-like hallucinations and cataplexy, muscle paralysis (atonia) much like occurs in REM sleep. In mice lacking orexin peptides, cataplexy is also observed with similar presentation as in humans of muscle paralysis during wakefulness which is often triggered by positive emotions. Prior research showed that the activat
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23

Šusta, Marek. "Brain Activation Sequences." Doctoral thesis, 2017. http://www.nusl.cz/ntk/nusl-354434.

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Brain Activation Sequences Abstract INTRODUCTION: This research goes beyond the EEG source localization up to the field of brain connectivity in an attempt to create software tool that eases diagnostic procedures in selected nosologic units by discriminating between patients and healthy controls. METHODS: Experiment 1 - a group of 26 adult patients (14 male, 12 female) suffering from NC and 10 adult controls (5 male, 5 female) participated in the experiment. The experiment contained audio recordings designed to trigger laughter in participants during the EEG recording. Experiment 2 - twenty ei
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24

Willie, Jon Timothy. "Lessons from sleepy mice : narcolepsy and the Orexin neuropeptide system." 2005. http://edissertations.library.swmed.edu/pdf/WillieJ042905/WillieJon.pdf.

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25

Viegas, Ana Isabel Loureiro 1988. "Descrição de uma população narcoléptica portuguesa." Master's thesis, 2015. http://hdl.handle.net/10451/23768.

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Tese de mestrado, Ciências do Sono, Faculdade de Medicina, Universidade de Lisboa, 2015<br>Introdução A Narcolepsia é uma perturbação do sono crónica caracterizada por sonolência diurna excessiva e manifestações do sono REM na vigília, nomeadamente, a cataplexia, a paralisia do sono e as alucinações hipnagógicas e hipnopômpicas (Kadotani H., Faraco J. & Mignot E., 1998). O seu diagnóstico envolve a realização de exames laboratoriais de Medicina do Sono, nomeadamente, a Polissonografia do Sono (PSG) e o Teste de Latências Múltiplas do Sono (TLMS), e/ou de técnicas como a Tipagem do HLA e o do
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