Relevant bibliographies by topics / Catecholamines Physiological effect

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  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Catecholamines Physiological effect'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 January 2023

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Journal articles on the topic "Catecholamines Physiological effect"

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Barnea, Eytan R., Rina Perlman, Hassan Fakih, Tova Bick, Shahar Kol, and Zeev Hochberg. "The role of catecholamines in estradiol and progesterone secretion by cultured explants and cells of human term placentae." Acta Endocrinologica 121, no. 6 (December 1989): 767–72. http://dx.doi.org/10.1530/acta.0.1210767.

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Abstract. The effects of physiological concentrations of the native catecholamines norepinephrine and epinephrine upon term placental hormonal function were examined by measuring estradiol and progesterone secretion by organ and cell culture systems. Results show that, in explants, both catecholamines caused a significant increase in the secretion of both sex steroids, p < 0.05. Estradiol secretion was blocked by the alpha and beta adrenergic receptors antagonists, phenoxybenzamine and propranolol, respectively, p < 0.05. Norepinephrine but not epinephrine dependent progesterone secretio
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Haas, M., and T. J. McManus. "Effect of norepinephrine on swelling-induced potassium transport in duck red cells. Evidence against a volume-regulatory decrease under physiological conditions." Journal of General Physiology 85, no. 5 (May 1, 1985): 649–67. http://dx.doi.org/10.1085/jgp.85.5.649.

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Duck red cells exhibit specific volume-sensitive ion transport processes that are inhibited by furosemide, but not by ouabain. Swelling cells in a hypotonic synthetic medium activates a chloride-dependent, but sodium-independent, potassium transport. Shrinking cells in a hypertonic synthetic medium stimulates an electrically neutral co-transport of [Na + K + 2 Cl] with an associated 1:1 K/K (or K/Rb) exchange. These shrinkage-induced modes can also be activated in both hypo- and hypertonic solutions by beta-adrenergic catecholamines (e.g., norepinephrine). Freshly drawn cells spontaneously shr
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Keating, Damien J., and Chen Chen. "Activin A stimulates catecholamine secretion from rat adrenal chromaffin cells: a new physiological mechanism." Journal of Endocrinology 186, no. 2 (August 2005): R1—R5. http://dx.doi.org/10.1677/joe.1.06301.

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Activin A is a member of the transforming growth factor-β family and has known roles in the adrenal cortex, from which activin A is secreted. We aimed to find whether activin A induces secretion of catecholamines from chromaffin cells of the adrenal medulla, which neighbours the adrenal cortex in vivo. Using carbon fibre amperometry, we were able to measure catecholamine secretion in real-time from single chromaffin cells dissociated from the rat adrenal medulla. Activin A stimulated catecholamine secretion in a rapid and dose-dependent manner from chromaffin cells. This effect was fully rever
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Mayerhofer, A., RW Steger, G. Gow, and A. Bartke. "Catecholamines stimulate testicular testosterone release of the immature golden hamster via interaction with alpha- and beta-adrenergic receptors." Acta Endocrinologica 127, no. 6 (December 1992): 526–30. http://dx.doi.org/10.1530/acta.0.1270526.

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Several lines of evidence suggest that catecholamines are involved in the regulation of the development of the testis. We have therefore investigated the ability of testicular parenchyma (decapsulated pieces of testes) from 18 to 20-day-old golden hamsters to respond to catecholaminergic stimuli in vitro. Norepinephrine and epinephrine, as well as the beta-receptor agonist isoproterenol and the alpha-adrenoreceptor agonist phenylephrine were able to significantly stimulate testicular testosterone production. Dopamine and serotonin were not effective. The stimulatory action of norepinephrine on
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Fulop, Tiberiu, and Corey Smith. "Physiological stimulation regulates the exocytic mode through calcium activation of protein kinase C in mouse chromaffin cells." Biochemical Journal 399, no. 1 (September 13, 2006): 111–19. http://dx.doi.org/10.1042/bj20060654.

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Adrenal medullary chromaffin cells release catecholamines and neuropeptides in an activity-dependent manner controlled by the sympathetic nervous system. Under basal sympathetic tone, catecholamines are preferentially secreted. During acute stress, increased sympathetic firing evokes release of both catecholamines as well as neuropeptides. Both signalling molecules are co-packaged in the same large dense core granules, thus release of neuropeptide transmitters must be regulated after granule fusion with the cell surface. Previous work has indicated this may be achieved through a size-exclusion
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Perry, S. F., R. Fritsche, and S. Thomas. "STORAGE AND RELEASE OF CATECHOLAMINES FROM THE CHROMAFFIN TISSUE OF THE ATLANTIC HAGFISH MYXINE GLUTINOSA." Journal of Experimental Biology 183, no. 1 (October 1, 1993): 165–84. http://dx.doi.org/10.1242/jeb.183.1.165.

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A variety of in vivo and in situ experiments were performed on the Atlantic hagfish (Myxine glutinosa) (i) to characterize the levels of circulating catecholamines during acute stresses, including hypoxia, anoxia or physical disturbance (air-exposure), and (ii) to evaluate the potential mechanisms of catecholamine release from the major sites of storage, the systemic heart and posterior cardinal vein (PCV). Adrenaline and noradrenaline were stored at roughly equivalent concentrations (approximately 20 microgram g-1) in cardiac tissue, whereas noradrenaline was the predominant catecholamine sto
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Fritsche, R., S. G. Reid, S. Thomas, and S. F. Perry. "SEROTONIN-MEDIATED RELEASE OF CATECHOLAMINES IN THE RAINBOW TROUT ONCORHYNCHUS MYKISS." Journal of Experimental Biology 178, no. 1 (May 1, 1993): 191–204. http://dx.doi.org/10.1242/jeb.178.1.191.

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The effects of serotonin (5-hydroxytryptamine; 5-HT) on catecholamine release from chromaffin tissue were investigated in the rainbow trout (Oncorhynchus mykiss) in vivo and in situ. Intra-arterial injections of serotonin in vivo caused dose-dependent (50–250 nmol kg-1) increases in both plasma noradrenaline and adrenaline levels. Pre-treatment of fish with the serotonergic receptor antagonist methysergide did not abolish these increases. An in situ saline-perfused head kidney preparation was developed and validated to study the potential direct effect of serotonin on catecholamine release. Th
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Maggs, D. G., and I. A. Macdonald. "Physiological and Symptomatic Responses to Postural Change in Non-Diabetic Subjects during Hypoglycaemia." Clinical Science 87, no. 2 (August 1, 1994): 193–99. http://dx.doi.org/10.1042/cs0870193.

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1. Insulin-induced hypoglycaemia is characterized by an autonomic disturbance which produces some of the symptoms of hypoglycaemia. How an additional autonomic stress like postural change may alter physiological responses and symptoms of hypoglycaemia is not known. In 10 healthy male subjects (mean age 24 years) we observed physiological and symptomatic responses to postural change during acute (20 min) and prolonged (60 min) hyperinsulinaemic (60 m-units min−1 m−2) hypoglycaemia (2.5 mmol/l) and euglycaemia (4.5 mmol/l), and placebo control (saline). 2. In all studies standing increased plasm
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Mader, S. L., C. L. Downing, and E. Van Lunteren. "Effect of age and hypoxia on beta-adrenergic receptors in rat heart." Journal of Applied Physiology 71, no. 6 (December 1, 1991): 2094–98. http://dx.doi.org/10.1152/jappl.1991.71.6.2094.

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Previous reports suggest that hypoxia downregulates cardiac beta-adrenergic receptors from young rats. Because aging alters response to stress, we hypothesized an age-related alteration in the response to hypoxia. Male Fischer-344 rats, aged 3 and 20 mo, were divided into control and hypoxic groups. The hypoxic rats were exposed to hypobaric hypoxia (0.5 atm) for 3 wk. After hypoxic exposure, body weight decreased, hematocrit increased, right ventricular weight increased, and left ventricular weight decreased in all animals. beta-Adrenergic receptor density declined after hypoxic exposure in t
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Pinz, Ilka, and Hans-O. Pörtner. "Metabolic costs induced by lactate in the toad Bufo marinus: new mechanism behind oxygen debt?" Journal of Applied Physiology 94, no. 3 (March 1, 2003): 1177–85. http://dx.doi.org/10.1152/japplphysiol.00131.2002.

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The mechanism of an increase in metabolic rate induced by lactate was investigated in the toad Bufo marinus. Oxygen consumption (V˙o 2) was analyzed in fully aerobic animals under hypoxic conditions (7% O2 in air), accompanied by measurements of catecholamines in the plasma, and was measured in isolated hepatocytes in vitro under normoxia by using specific inhibitors of lactate proton symport [α-cyano-4-hydroxycinnamate (CHC)] and sodium proton exchange (EIPA). The rise in metabolic rate in vivo can be elicited by infusions of hyperosmotic (previous findings) or isosmotic sodium lactate soluti
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Dissertations / Theses on the topic "Catecholamines Physiological effect"

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Klipp, Robert Carl. "Catecholamine Interactions with the Cardiac Ryanodine Receptor." PDXScholar, 2013. https://pdxscholar.library.pdx.edu/open_access_etds/1439.

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The cardiac ryanodine receptor (RyR2) is a Ca2+ ion channel found in the sarcoplasmic reticulum (SR), an intracellular membranous Ca2+ storage system. It is well known that a destabilization of RyR2 can lead to a Ca2+ flux out of the SR, which results in an overload of intracellular Ca2+; this can also lead to arrhythmias and heart failure. The catecholamines play a large role in the regulation of RyR2; stimulation of the Beta-adrenergic receptor on the cell membrane can lead to a hyperphosphorylation of RyR2, making it more leaky to Ca2+. We have previously shown that strong electron donors w
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St, John-Allan Katherine Marie. "The effects of repeated daily cocaine administration on rat cardiac norepinephrine content." Scholarly Commons, 1990. https://scholarlycommons.pacific.edu/uop_etds/2199.

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Cocaine, the principal biologically active alkaloid of Erythroxylon coca, acts as both a local anesthetic agent and an indirect-acting sympathomimetic. These two very different activities make research on and subsequent interpretation of cocaine's multiplicity of varied effects difficult. Not surprisingly, investigations into the central effects of cocaine have yielded mixed results. However, there is a certain body of evidence which points to the euphoric and self-administrating properties of cocaine as appearing to involve the acute activation of central dopamine neuronal systems . With chro
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Scobie, David Roger. "Short term effects of stress hormones on cell division rate in wool follicles : a thesis submitted in fulfilment of the requirements for the degree of Doctor of Philosophy." Title page, abstract and contents only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phs421.pdf.

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Includes bibliographical references (leaves 183-207) A local intradermal technique using colchicine to estimate cell division rate in wool follicles is refined and used throughout the thesis. Statistical methods used to analyse data obtained with this method are described and discussed. The implications of the findings are of great significance to research into the influence of physiological changes on wool production, and suggest experiments should be conducted under controlled environmental conditions, with a minimum of stress imposed on the animals.
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Anderson, Dawn E. "Effects of caffeine on the metabolic and catecholamine responses to exercise in 5 and 28p0sC environments." Virtual Press, 1992. http://liblink.bsu.edu/uhtbin/catkey/833465.

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The influence of caffeine on the metabolic and catecholamine responses to mild exercise in a cold and a warm environment was studied in eight healthy males. The subjects performed 60 minutes of cycling at 50% VO2max in a cold environment (5°C and 70% relative humidity) and a warm environment (28°C and 50% relative humidity) 30 minutes after ingesting caffeine (5mg/kg body weight) or placebo (dextrose). Caffeine ingestion prior to exercise in the warm environment resulted in increased plasma epinephrine, with no effect on plasma norepinephrine. Neither lipid nor carbohydrate metabolism was alte
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Lacombe, A. M. A. "Effects of circulating catecholamines on diving in ducks (Anas platyrhynchos)." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/30724.

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Plasma catecholamines have been measured in chronically adrenalectomised (ADX) ducks, in chronically adrenal denervated ducks (DNX), in their respective sham-operated controls (SH-adx, SH-dnx) as well as in intact ducks after 3 minutes forced submergence. The results showed that 100% of the plasma Epinephrine (EP) and 40 to 80% of plasma Norepinephrine (NE) released during the dive came from the adrenal glands. 20 to 60% of plasma NE came from endings of the autonomic vascular sympathetic nerves which are strongly stimulated during diving. Adrenal catecholamines were released by nerve activat
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Ye, Xuemin. "The effect of water pH on swimming performance, blood pH, red cell pH, ion concentrations and catecholamine concentrations in plasma, and gill potential in rainbow trout (Salmo gairdneri)." Thesis, University of British Columbia, 1986. http://hdl.handle.net/2429/26676.

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The effect of transferring fish from water at pH 7.0 to either more acid or more alkaline conditions was to reduce the maximum critical velocity of the fish. In water of pH 4.0, 5.0, and 10.0, the maximum critical velocity was only 54.5%, 66.5%, and 61% respectively of that recorded for fish in the water of pH 7.0. Thus, both acid and alkaline conditions in the water reduce the aerobic swimming capacity of trout. Exposure to acid conditions increased mucus secretion and this was associated with an increase in coughing and breathing frequency in resting fish. Coughing rate increased from 41/hr
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Can, Adem 1977. "Effects of experience and novelty on sexual behavior and associated neuronal activity in male Japanese quail." 2008. http://hdl.handle.net/2152/17780.

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In many behavioral paradigms, repeated exposures to a particular stimulus or event results in lower immediate early gene (IEG) expression. First, it was investigated if a similar reduction in IEG expression in the brain areas controlling male sexual behaviors would be observed after repeated copulation experiences in male Japanese quail. The results showed that IEG expression, as assessed by egr-1 immunoreactivity, did not increase in the POM, the BST, or the PAG after a copulation episode in highly sexually experienced subjects. One possibility was that the pattern of initial elevation of neu
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Young, Jennifer Charity. "Supplemental vitamin B-6 and endurance exercise effects on plasma catecholamines of trained male cyclists." Thesis, 1996. http://hdl.handle.net/1957/27440.

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This study examined the effect of vitamin B-6 supplementation and exhaustive submaximal exercise on plasma catecholamine concentrations, and the relationship between plasma catecholamines and fuel use, heart rate and oxygen consumption. Five trained men (age= 18-35 years; V0₂max=53 ml 0₂/kg/min.) participated in two controlled dietary periods that were identical except for the addition of 20 mg/d pyridoxine (PN) supplementation during the second period. On the seventh morning of each period, fasted subjects exercised to exhaustion on a cycle ergometer at 74.5% ± 7.8 V0₂max. Blood was drawn pre
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Scobie, David Roger. "Short term effects of stress hormones on cell division rate in wool follicles / by David Roger Scobie." Thesis, 1992. http://hdl.handle.net/2440/21634.

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Includes bibliographical references (leaves 183-207)<br>ix, 207 leaves : ill. (some col.) ; 30 cm.<br>A local intradermal technique using colchicine to estimate cell division rate in wool follicles is refined and used throughout the thesis. Statistical methods used to analyse data obtained with this method are described and discussed. The implications of the findings are of great significance to research into the influence of physiological changes on wool production, and suggest experiments should be conducted under controlled environmental conditions, with a minimum of stress imposed on the a
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Hikoi, Hirotaka. "The effects of opioid receptor antagonism on plasma catecholamines and fat metabolism during prolonged exercise above or below lactate threshold in males." Thesis, 1999. http://hdl.handle.net/1957/33383.

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Books on the topic "Catecholamines Physiological effect"

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Nira, Ben-Jonathan, Bahr Janice M, and Weiner Richard I, eds. Catecholamines as hormone regulators. New York: Raven Press, 1985.

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International Symposium on Catecholamines and Other Neurotransmitters in Stress (7th 1999 Smolenice, Slovakia). Stress: Neural, endocrine, and molecular studies. London: Taylor & Francis, 2002.

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Stress, catecholamines, and cardiovascular disease. New York: Oxford University Press, 1995.

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Annica, Dahlström, Belmaker Robert H, and Sandler Merton, eds. Progress in catecholamine research: Proceedings of the Sixth International Catecholamine Symposium, held in Jerusalem, Israel, June 14-19, 1987. New York: Liss, 1988.

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The catecholaminergic innervation of the rat amygdala. Berlin: Springer, 1998.

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Vaudry, Hubert. Phylogenetic aspects of neuropeptides: From invertebrates to humans. Edited by New York Academy of Sciences. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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International, Symposium on Catecholamines and Other Neurotransmitters in Stress (6th 1995 Smolenice Slovakia). Stress: Molecular genetic and neurobiological advances : proceedings of the Sixth International Symposium on Catecholamines and Other Neurotransmitters in Stress, Smolenice Castle, Slovakia, June 19-24 1995. Australia: Harwood Academic Publishers, 1996.

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International Symposium on Catecholamines and Other Neurotransmitters in Stress (5th 1991 Smolenice, Slovakia). Stress: Neuroendocrine and molecular approaches : proceedings of the Fifth International Symposium on Catecholamines and Other Neurotransmitters in Stress, Smolenice Castle, Czechoslovakia, 24-29 June, 1991. Philadelphia: Gordon and Breach Science Publishers, 1992.

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International Symposium on Catecholamines and Other Neurotransmitters in Stress (9th 2007 Smolenice Castle, Slovakia). Stress, neurotransmitters, and hormones: Neuroendocrine and genetic mechanisms. Edited by Kvetňanský Richard, Ústav experimentálnej endokrinólogie (Slovenská akadémia vied), and National Institutes of Health (U.S.). Malden: Wiley-Blackwell, 2008.

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P, Nijkamp F., Wied David de 1925-, and Jong W. de, eds. Hypertension, brain catecholamines and peptides. Elsevier, 1989.

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Book chapters on the topic "Catecholamines Physiological effect"

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Kayabekir, Murat. "Neurophysiology of Basic Molecules Affecting Sleep and Wakefulness Mechanisms, Fundamentals of Sleep Pharmacology." In Sleep Medicine and the Evolution of Contemporary Sleep Pharmacotherapy [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.100166.

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As part of the biological rhythm, the human brain has a healthy functioning with the ability to differentiate between day and night hours in any given day (sleep rhythm, life rhythm). From the control of hormone levels to muscle tonus, from the regulation of respiratory rate to the content of our thoughts, sleep has an impact on all bodily and cognitive functions. It is not surprising to see such effects of sleep on the body as it leads to significant changes in the electrical activity of the brain in general. Electrical signal changes in the brain (sleep-wakefulness rhythm) are regulated by neurohormonal molecules and their receptors in the body. Neurotransmitters that control sleep and wakefulness can be listed as “Glutamate, Acetylcholine, Histamine, Norepinephrine and GABA”. Main hormones are: Melatonin, Corticotropin Releasing Hormone (CRH), cortisol, prolactin, Growth Hormone (GH), Insulin like Growth Factor (IGF-1, Somatomedin-C), Follicle Stimulating Hormone (FSH) and Luteinizing Hormone (LH), progesterone, estrogen, testosterone, catecholamines, leptin and neuropeptide Y″. The effects of pharmacological agents on sleep and wakefulness cycles are materialized through the following molecules and their receptors: Hypnotics (GABA A agonists, benzodiazepines, gabapentin, tiagabine), sedative antidepressants (tricyclic antidepressants, trazadone, mitrazapine), antihistamines, medications used for the treatment of sleeplessness (melatonin and melatonin analogues), amphetamine (most commonly used stimulant), secretion of monoamines (dopamine), non-amphetamine stimulants used in the treatment of hypersomnia and narcolepsy (modafinil, bupropion, selegiline, caffeine) and other substances (alcohol, nicotine, anesthetics). To the extent we can conceptualize the physiological mechanisms of these basic molecules listed above and the regions they affect, we can appreciate the effects of these substances on sleep physiology and sleep disorders.
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Conference papers on the topic "Catecholamines Physiological effect"

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Kirenskaya, A. V., M. A. Gruden, V. Yu Novototsky-Vlasov, A. M. Ryabova, and Z. I. Storozheva. "EFFECT OF VAL158MET CATECHOLAMINE O-METHYLTRANSFERASE GENE POLYMORPHISM ON INTERHEMISPHERIC ASYMMETRY IN ANTISACCADE TASK." In MODERN PROBLEMS IN SYSTEMIC REGULATION OF PHYSIOLOGICAL FUNCTIONS. NPG Publishing, 2019. http://dx.doi.org/10.24108/5-2019-confnf-38.

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