Relevant bibliographies by topics / Catfishes – Parasitic diseases – Immunological aspects

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  2. Dissertations / Theses
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Academic literature on the topic 'Catfishes – Parasitic diseases – Immunological aspects'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Catfishes – Parasitic diseases – Immunological aspects"

1

Kalkanov, I., and Iv Dinev. "PATHOMORPHOLOGICAL ASPECTS OF RELEVANT GASTROINTESTINAL DISEASES IN NEWBORN AND GROWING CALVES." Trakia Journal of Sciences 17, no. 4 (2019): 413–20. http://dx.doi.org/10.15547/tjs.2019.04.019.

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The emphasis of this literature overview is placed on the etiology, epidemiology, specific gross and microscopic pathological changes of the commonest gastrointestinal diseases in newborn and growing calves in dairy and beef farms. Neonatal diarrhea in calves is a multifactorial disease accompanied with high morbidity and mortality rates. It incurs considerable economic losses for cattle industry worldwide. The gastroenteritis in calves is most commonly of infectious or parasitic etiology and the most frequently involved enteropathogens are Cryptosporidium parvum, bovine rotaviruses (GRA BRV), bovine coronaviruses (BCoV), mucosal disease virus - bovine viral diarrhoea (BVDV) and Escherichia coli K99 (F5). Affected calves are from 24th hours to 30 days of age, and the appearance of the disease is largely dependent on the immunological status of newborn calves, the amount of ingested colostrum, whether dams are vaccinated or not during the dry period, and the conditions of housing of newborns. The specific gross and microscopic lesions of the gastrointestinal tract and of some parenchymal organs are essential for the diagnostics and differential diagnostics of reviewed diseases.
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Rodrigues-Silva, Rosângela, Hércules Moura, Cerusa Dreyer, and Luiz Rey. "Human pulmonary dirofilariasis: a review." Revista do Instituto de Medicina Tropical de São Paulo 37, no. 6 (December 1995): 523–30. http://dx.doi.org/10.1590/s0036-46651995000600009.

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The authors presented a detailed summary of the geographical distribution, clinical and pathological aspects of human pulmonary dirofilariasis. Although benign, this zoonosis, of which Dirofilaria immitis is the major etiological agent, represents a medical problem since it produces symptoms which may be confused with neoplasia and thus may subject patients to unnecessary thoracic surgery. Of 229 cases cited in the literature, only 17 were reported in Brazil, despite the existence of highly favorable conditions for the transmission of this infection in man. Thus it may well be that this parasitic infection remains underdiagnosed. Finally, the importance of a differential diagnosis between dirofilariasis and pulmonary neoplasia is emphasized in cases where there is a solitary subpleural nodule ("coin lesion") present. In addition, the development and improvement of modern immunological diagnostic techniques are essential to distinguish this benign disease from other pathological conditions and thus avoid unneccessary surgery. These techniques may reveal the true prevalence of this parasitic infection in our environment.
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Velavan, TP, and Olusola Ojurongbe. "Regulatory T Cells and Parasites." Journal of Biomedicine and Biotechnology 2011 (2011): 1–8. http://dx.doi.org/10.1155/2011/520940.

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Human host encounters a wide array of parasites; however, the crucial aspect is the failure of the host immune system to clear these parasites despite antigen recognition. In the recent past, a new immunological concept has emerged, which provides a framework to better understand several aspects of host susceptibility to parasitic infection. It is widely believed that parasites are able to modulate the magnitude of effector responses by inducing regulatory T cell (Tregs) population and several studies have investigated whether this cell population plays a role in balancing protective immunity and pathogenesis during parasite infection. This review discusses the several mechanism of Treg-mediated immunosuppression in the human host and focuses on the functional role of Tregs and regulatory gene polymorphisms in infectious diseases.
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Leal-Silva, Thaís, Flaviane Vieira-Santos, Fabrício Marcus Silva Oliveira, Luiza de Lima Silva Padrão, Lucas Kraemer, Pablo Hemanoel da Paixão Matias, Camila de Almeida Lopes, et al. "Detrimental role of IL-33/ST2 pathway sustaining a chronic eosinophil-dependent Th2 inflammatory response, tissue damage and parasite burden during Toxocara canis infection in mice." PLOS Neglected Tropical Diseases 15, no. 7 (July 29, 2021): e0009639. http://dx.doi.org/10.1371/journal.pntd.0009639.

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Toxocariasis is a neglected disease that affects people around the world. Humans become infected by accidental ingestion of eggs containing Toxocara canis infective larvae, which upon reaching the intestine, hatch, penetrate the mucosa and migrate to various tissues such as liver, lungs and brain. Studies have indicated that Th2 response is the main immune defense mechanism against toxocariasis, however, there are still few studies related to this response, mainly the IL-33/ST2 pathway. Some studies have reported an increase in IL-33 during helminth infections, including T. canis. By binding to its ST2 receptor, IL-33 stimulating the Th2 polarized immune cell and cytokine responses. Thus, we aimed to investigate the role of the IL-33/ST2 pathway in the context of T. canis larval migration and the immunological and pathophysiological aspects of the infection in the liver, lungs and brain from Wild-Type (WT) BALB/c background and genetically deficient mice for the ST2 receptor (ST2-/-). The most important findings revealed that the IL-33/ST2 pathway is involved in eosinophilia, hepatic and cerebral parasitic burden, and induces the formation of granulomas related to tissue damage and pulmonary dysfunction. However, ST2-/- mice, the immune response was skewed to Th1/Th17 type than Th2, that enhanced the control of parasite burden related to IgG2a levels, tissue macrophages infiltration and reduced lung dysfunction. Collectively, our results demonstrate that the Th2 immune response triggered by IL-33/ST2 pathway mediates susceptibility to T. canis, related to parasitic burden, eosinophilia and granuloma formation in which consequently contributes to tissue inflammation and injury.
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Dissertations / Theses on the topic "Catfishes – Parasitic diseases – Immunological aspects"

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McCartney, Jerald Barton. "Studies on Ichthyophthirius multifiliis and the immune system of Ictalurus punctatus with emphasis on early detection of disease, chemotherapeutic agents and production of biological reagents." 1985. http://hdl.handle.net/2097/27487.

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Lefcort, Hugh G. "The effects of parasites on host behavior : who benefits?" Thesis, 1993. http://hdl.handle.net/1957/36151.

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Some parasites may modify the behavior of their hosts. Altered behaviors may: 1) benefit the host in that they defend against the pathogen, 2) benefit the pathogen and represent manipulations of the host response, and 3) benefit neither the host or the pathogen and simply be a product of the host response to infection. In this thesis I examine four host/parasite systems. For each system, I explore host/parasite behavioral interactions, and examine them with regard to selective pressures that may be acting on both the host and the parasite. I test the Hamilton and Zuk hypothese in 26 species of lizards. I find an inverse relationship between a lizard species' brightness and parasite prevalence. My result lend credence to criticisms of the Hamilton and Zuk Hypothesis. If infection does occur, animals may alter their behavior to impair the growth and reproduction of the parasite. To test this prediction, I examine behavioral thermoregulation in two strains of the snail Biomphalaria glabrata, one resistant to, and one susceptible to, the parasite Schistosoma mansoni. The preferred temperature of infected snails drops five weeks after exposure to the parasite. I propose the hypothesis that pathogen-induced host defense responses result in altered host behaviors and enhanced predation. In particular, I examine the effects of the acute phase response (a physiological response whose symptoms include fever, reduced activity and malaise) on antipredatory behavior in bullfrog (Rana catesbeiana) tadpoles. This host response is associated with the preliminary stages of infection with many pathogens yet its behavioral effects have received little attention. I find that the stereotypical effects of the acute phase response can lead to increased predation. I suggest that altered behaviors may afford some parasites a potential pathway to their next host. I examine the behavioral effects of a yeast, Candida spp., a single-host parasite species in its natural host, the red-legged frog (Rana aurora). Infected tadpoles exhibit the same behavioral modifications that are noted in bacteria injected bullfrog tadpoles. These results suggest that some altered behaviors may occur due to a host response to infection and not due to parasitic manipulation.
Graduation date: 1993
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3

Stilger, Krista L. "Identification of TgElp3 as an essential, tail-anchored mitochondrial lysine acetyltransferase in the protozoan pathogen toxoplasma gondii." Thesis, 2014. http://hdl.handle.net/1805/4660.

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Indiana University-Purdue University Indianapolis (IUPUI)
Toxoplasma gondii, a single-celled eukaryotic pathogen, has infected one-third of the world’s population and is the causative agent of toxoplasmosis. The disease primarily affects immunocompromised individuals such as AIDS, cancer, and transplant patients. The parasites can infect any nucleated cell in warm-blooded vertebrates, but because they preferentially target CNS, heart, and ocular tissue, manifestations of infection often include encephalitis, myocarditis, and a host of neurological and ocular disorders. Toxoplasma can also be transmitted congenitally by a mother who becomes infected for the first time during pregnancy, which may result in spontaneous abortion or birth defects in the child. Unfortunately, the therapy currently available for treating toxoplasmosis exhibits serious side effects and can cause severe allergic reactions. Therefore, there is a desperate need to identify novel drug targets for developing more effective, less toxic treatments. The regulation of proteins via lysine acetylation, a reversible post-translational modification, has previously been validated as a promising avenue for drug development. Lysine acetyltransferases (KATs) are responsible for the acetylation of hundreds of proteins throughout prokaryotic and eukaryotic cells. In Toxoplasma, we identified a KAT that exhibits homology to Elongator protein 3 (TgElp3), the catalytic component of a transcriptional elongation complex. TgElp3 contains the highly conserved radical S-adenosylmethionine and KAT domains but also possesses a unique C-terminal transmembrane domain (TMD). Interestingly, we found that the TMD anchors TgElp3 in the outer mitochondrial membrane (OMM) such that the catalytic domains are oriented towards the cytosol. Our results uncovered the first tail-anchored mitochondrial KAT reported for any species to date. We also discovered a shortened form of Elp3 present in mouse mitochondria, suggesting that Elp3 functions beyond transcriptional elongation across eukaryotes. Furthermore, we established that TgElp3 is essential for parasite viability and that its OMM localization is important for its function, highlighting its value as a potential target for future drug development.
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Books on the topic "Catfishes – Parasitic diseases – Immunological aspects"

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Leopoldina-Meeting Parasitismus, Immunreaktionen bei Parasitosen (1991 Halle an der Saale, Germany). Leopoldina-Meeting Parasitismus, Immunreaktionen bei Parasitosen: Vom 25. bis 26. Oktober 1991 in Halle (Saale). Halle, Saale: Deutsche Akademie der Naturforscher Leopoldina, 1992.

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2

Wakelin, Derek. Immunity to parasites: How parasitic infections are controlled. 2nd ed. Cambridge: Cambridge University Press, 1996.

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3

Garraud, Olivier. Update in tropical immunology. Kerala, India: Research Signpost, 2005.

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4

1936-, Wang Ching Chung, and American Association for the Advancement of Science., eds. Molecular & immunological aspects of parasitism. Washington, D.C: American Association for the Advancement of Science, 1991.

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Kenneth, Walls, and Schantz Peter M, eds. Immunodiagnosis of parasitic diseases. Orlando: Academic Press, 1986.

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6

S, Warren Kenneth, ed. Immunology and molecular biology of parasitic infections. 3rd ed. Boston: Blackwell Scientific Publications, 1993.

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7

Felipe, Kierszenbaum, ed. Parasitic infections and the immune system. San Diego: Academic Press, Inc., 1994.

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8

J, Wyler David, ed. Modern parasite biology: Cellular, immunological, and molecular aspects. New York: Freeman, 1990.

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9

J, Wyler David, ed. Modern parasite biology: Cellular, immunological, and molecular aspects. New York: W.H. Freeman, 1990.

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10

Lefcort, Hugh G. The effects of parasites on host behavior: Who benefits? 1993.

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