Academic literature on the topic 'Causes of maxillary sinus hypoplasia'

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Journal articles on the topic "Causes of maxillary sinus hypoplasia"

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Orhan Soylemez, Umut Percem, and Basak Atalay. "Investigation of the accessory maxillary ostium: a congenital variation or acquired defect?" Dentomaxillofacial Radiology 50, no. 6 (2021): 20200575. http://dx.doi.org/10.1259/dmfr.20200575.

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Objective: We sought to determine whether an accessory maxillary ostium (AMO) is a congenital or acquired condition and we investigated concomitant sinus pathologies associated with this structure. Methods: Paranasal sinus CT examinations of individuals aged ≥13 years and <13 years were compared retrospectively. In total, 552 sinuses of 276 patients aged ≥13 years (Group 1) and 284 maxillary sinuses of 142 children aged <13 years (Group 2) were evaluated. Patients were classified as AMO-positive or -negative. The following features were evaluated in Group 1: AMO presence, mucus retention cysts, mucosal thickening, sinusitis of the maxillary sinus, nasal septum deviation, concha hypertrophy, concha bullosa, primary ostium obstruction, uncinate process atelectasis, paradox concha, Agger nasi and Haller cells, and sinus hypoplasia. The sizes and locations of AMOs were also evaluated. The presence of an AMO and sinusitis were evaluated in Group 2. Results: AMOs were detected in 122 sinuses in Group 1. In the AMO-positive group, sinusitis, mucosal thickening, and primary ostium obstruction were significantly more common than in the AMO-negative group (p < 0.00001). Statistically significant associations were not observed between AMO presence and other parameters. AMOs were present in two sinuses in Group 2. Conclusion: Our results suggest that AMOs are acquired defects caused by sinus diseases. The rare occurrence of these structures in patients aged <13 years suggests that they may be a perforation or secondary drainage pathway in patients with sinusitis or primary ostium obstruction.
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Uluyol, S., İ. B. Arslan, A. Demir, G. C. Mercan, O. Dogan, and İ. Çukurova. "The role of the uncinate process in sinusitis aetiology: isolated agenesis versus maxillary sinus hypoplasia." Journal of Laryngology & Otology 129, no. 5 (2015): 458–61. http://dx.doi.org/10.1017/s0022215115000821.

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AbstractObjective:This study aimed to evaluate the clinical significance of maxillary sinus hypoplasia and isolated agenesis of the uncinate process in sinusitis aetiology.Methods:Three patients with isolated agenesis of the uncinate process and 27 patients with 43 maxillary sinus hypoplasia variations were recruited. The frequencies of sinusitis episodes and radiological findings were compared between patient subgroups.Results:In all, 23 type I maxillary sinus hypoplasia, 13 type II maxillary sinus hypoplasia and 7 type III maxillary sinus hypoplasia variations were detected. Patients with isolated agenesis of the uncinate process underwent antibiotic treatment an average of 7 times per year, whereas those with types I, II and III maxillary sinus hypoplasia were treated 1.57, 3.22, and 5.75 times per year, respectively, over a 5-year period. The antibiotic treatment frequency for patients with isolated agenesis of the uncinate process was significantly higher than for those with types I and II maxillary sinus hypoplasia.Conclusion:Isolated agenesis of the uncinate process seems to play a stronger role than types I and II maxillary sinus hypoplasia in the pathophysiology of chronic sinusitis.
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Khanduri, Sachin, Sumit Agrawal, Saakshi Chhabra, and Swati Goyal. "Bilateral Maxillary Sinus Hypoplasia." Case Reports in Radiology 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/148940.

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Maxillary sinus hypoplasia (MSH) is an uncommon abnormality of paranasal sinuses noted in clinical practice. Computed tomography (CT) scan helps in diagnosing the anomaly along with any anatomical variation that may be associated with it. MSH is usually associated with other anomalies like uncinate process hypoplasia. Three types of MSH have been described. Type 1 MSH shows mild maxillary sinus hypoplasia, type 2 shows significant sinus hypoplasia with narrowed infundibular passage and hypoplastic or absent uncinate process, and type 3 is cleft like maxillary sinus hypoplasia with absent uncinate process. CT and endoscopic examination usually complement each other in diagnosing MSH.
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Alanazi, Hani. "Bilateral maxillary sinus hypoplasia." Majmaah Journal of Health Sciences 12, no. 1 (2024): 182. http://dx.doi.org/10.5455/mjhs.2024.01.017.

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Maxillary sinus hypoplasia is an uncommon condition characterized by underdevelopment of the maxillary sinus antrum. Typically, it is accompanied by other anomalies, such as abnormalities in the uncinate process and infundibular passage. We present a rare case of bilateral maxillary sinus hypoplasia in a 22-year-old, symptomatic patient who didn’t respond to maximum medical therapy. Combining a detailed history, endoscopic examination, and CT scan are crucial in diagnosing sinus diseases, particularly in MSH cases.
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Srivastava, Saurabh, Mohd Shakeel, and Prasoon Varshney. "Maxillary Sinus Hypoplasia—A Not-so-uncommon Clinical Entity: A Review." An International Journal Clinical Rhinology 9, no. 1 (2016): 43–45. http://dx.doi.org/10.5005/jp-journals-10013-1263.

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ABSTRACT Bilateral sinus maxillary sinus hypoplasia is a rare anomaly. Radiological diagnosis of maxillary sinus hypoplasia is important for diagnosis to prevent recurrent rhinosinusitis and complications during endoscopic sinus surgery. A high index of suspicion is required by outpatient department examination for proper clinical evaluation and further management of these patients. How to cite this article Bhargava A, Khanduri S, Shakeel M, Srivastava S, Varshney P. Maxillary Sinus Hypoplasia-A Not-so-uncommon Clinical Entity: A Review. Clin Rhinol An Int J 2016;9(1):43-45.
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Perez, Alexandre, Vincent Lenoir, and Tommaso Lombardi. "Cystic-like maxillary sinus hypoplasia." BMJ Case Reports 15, no. 4 (2022): e249659. http://dx.doi.org/10.1136/bcr-2022-249659.

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Shcherbakov, D. A., A. V. Simonov, V. V. Kokareva, A. S. Krotova, and T. Yu Malysheva. "Definition of maxillary sinus hypoplasia." Rossiiskaya rinologiya 28, no. 1 (2020): 32. http://dx.doi.org/10.17116/rosrino20202801132.

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Price, Daniel L., and Oren Friedman. "Facial asymmetry in maxillary sinus hypoplasia." International Journal of Pediatric Otorhinolaryngology 71, no. 10 (2007): 1627–30. http://dx.doi.org/10.1016/j.ijporl.2007.06.014.

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Wake, M., L. Shankar, M. Hawke, and S. Takeno. "Maxillary Sinus Hypoplasia, Embryology, and Radiology." Archives of Otolaryngology - Head and Neck Surgery 119, no. 12 (1993): 1353–57. http://dx.doi.org/10.1001/archotol.1993.01880240091011.

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Ozcan, Kursat Murat, Omer Hizli, Zeynep Alev Sarisoy, Hakan Ulusoy, and Guven Yildirim. "Coexistence of frontal sinus hypoplasia with maxillary sinus hypoplasia: a radiological study." European Archives of Oto-Rhino-Laryngology 275, no. 4 (2018): 931–35. http://dx.doi.org/10.1007/s00405-018-4892-9.

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Book chapters on the topic "Causes of maxillary sinus hypoplasia"

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Newman, Lawrence C., Morris Levin, Rashmi B. Halker Singh, and Rebecca L. Michael. "Sinus Headache." In Headache and Facial Pain, edited by Lawrence C. Newman, Morris Levin, Rashmi B. Halker Singh, and Rebecca L. Michael. Oxford University Press, 2022. http://dx.doi.org/10.1093/med/9780190842130.003.0002.

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Migraine and other primary headache types are often misdiagnosed as “sinus headache” because of their sometimes midfrontal location and nasal/sinus symptoms. However, there are some rhinological and sinus conditions, such as inadequately treated sinusitis, that do lead to head and/or facial pain. Imaging can help but can also be misleading because some degree of paranasal sinus pathology is common. Imaging findings suggestive of sinus-related headaches include filling opacity in one or more sinus cavities; air–fluid levels in maxillary, ethmoid, frontal, or sphenoid sinuses; and evidence of bony changes involving the surrounding osseous structures. A more controversial cause of rhinological head pain—the “nasal contact point headache”—may be underdiagnosed. The causes and best approaches to diagnosing headache due to rhinosinusitis and related conditions are discussed in this chapter.
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Wong, Agnes. "Nuclear and Infranuclear Ocular Motor Disorders." In Eye Movement Disorders. Oxford University Press, 2008. http://dx.doi.org/10.1093/oso/9780195324266.003.0021.

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Binocular diplopia is usually caused by strabismus, whereas monocular diplopia is usually caused by ocular diseases. Incomitant diplopia is usually caused by an acquired strabismus resulting from abnormal innervation or mechanical restriction. The oculomotor (third) nerve: ■ Innervates the medial rectus, superior rectus, inferior rectus, inferior oblique, and levator palpebrae muscles ■ Carries parasympathetic fibers to the iris sphincter and the ciliary body. ■ Common causes of third nerve palsy: Adults: aneurysms, vascular disease (including ischemia, diabetes, hypertension, and inflammatory arteritis), trauma, migraine Children: birth trauma, accidental trauma, neonatal hypoxia, migraine The third nerve originates from the oculomotor nucleus complex, which lies at the ventral border of the periaqueductal gray matter in the midbrain. The nerve fascicle passes ventrally through the medial longitudinal fasciculus, the tegmentum, the red nucleus, and the substantia nigra, and finally emerges from the cerebral peduncle to form the oculomotor nerve trunk, which lies between the superior cerebellar and posterior cerebral arteries. The nerve then passes through the subarachnoid space, running beneath the free edge of the tentorium. It continues lateral to the posterior communicating artery and below the temporal lobe uncus, where it runs over the petroclinoid ligament. It pierces the dura mater at the top of the clivus to enter the cavernous sinus. Within the cavernous sinus, the nerve runs along the lateral wall of the sinus together with the trochlear nerve and the ophthalmic (V1) and maxillary (V2) divisions of the trigeminal nerve. As it leaves the cavernous sinus, it divides into the superior and inferior divisions, which pass through the superior orbital fissure, and enters the orbit within the annulus of Zinn. Within the orbit, the smaller superior division runs lateral to the optic nerve and ophthalmic artery and supplies the superior rectus and levator palpebrae muscles. The larger inferior division supplies the medial rectus, inferior rectus, and inferior oblique muscles, as well as the iris sphincter and ciliary body.
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