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Books on the topic 'Cavity wall'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Arora, S. K. The spacing of wall ties in cavity walls. Watford: Building Research Establishment, 1986.

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2

Large, K. R. Design of a novel cavity wall blow moulding process. Manchester: UMIST, 1990.

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3

Quirouette, R. L. Testing rainscreen wall and window systems: The cavity excitation method. Ottawa: Canada Mortgage and Housing Corporation, 1996.

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4

Bell, Paul. Heat and moisture transfer through cavity wall constructions under simulated winter conditions. Salford: Universityof Salford, 1986.

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5

Ford, R. W. Improved standards of insulation in cavity walls with an outer leaf of facing brickwork. Windsor: Brick Development Association, 1990.

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6

Institution, British Standards. Thermal insulation of cavity walls using man-made mineral fibre batts (slabs). London: B.S.I., 1986.

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7

Office, Energy Efficiency. Energy efficiency in new housing: Detailing for designers and professionals external cavity walls. London: Department of the Environment, 1993.

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8

Office, Energy Efficiency. Energy efficiency in new housing: Site practice for tradesmen external walls injected cavity insulation. London: Department of the Environment, 1993.

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9

Ford, R. W. Improved standards of insulation in cavity walls with an outer leaf of facing brickwork. Windsor: Brick Development Association, 1990.

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10

Burkholder, R. J. Electromagnetic fields backscattered from an S-shaped inlet cavity with an absorber coating on its inner walls. Columbus, Ohio: The Ohio State University, 1987.

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11

Institution, British Standards. British standard specification for urea-formaldehyde (UF) foam systems suitable for thermal insulation of cavity walls with masonry or concrete innerand outer leaves. London: British Standards Institution, 1985.

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12

Institution, British Standards. British standard code of practice for thermal insulation of cavity walls: (with masonry or concrete inner and outer leaves) by filling with urea-formaldehyde (UF) foam systems. London: British Standards Institution, 1985.

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13

Great Britain. Energy Efficiency Office. and Building Research Energy Conservation SupportUnit., eds. Cavity wall insulation in existing housing. [UK]: Energy Efficiency Office, 1993.

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14

Establishment, Building Research, ed. Choosing between cavity, internal and external wall insulation. Garston: Building Research Establishment, 1990.

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15

Great Britain. Energy Efficiency Office. and Building Research Establishment. Energy Conservation Support Unit., eds. Cavity wall insulation: Unlocking the potential in existing dwellings. [UK]: Energy Efficiency Office, 1995.

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16

Cavity wall insulation in existing dwellings: Urea formaldehyde foam. [UK]: Energy Efficiency Office, 1993.

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17

Great Britain. Department of the Environment., ed. Cavity wall insulation in existing dwellings: Mineral wool insulation. [London]: Department of the Environment, 1996.

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18

Cavity Wall Tie Failure (The Estates Gazette Professional Guides). Estates Gazette, 1990.

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19

Chiumello, Davide, and Cristina Mietto. Pathophysiology of pleural cavity disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0123.

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The pleural cavity is normally a virtual space that is essential to guarantee the mechanical coupling between the lung and the chest wall. The volume of pleural liquid is determined by the equilibrium of fluid turnover. The determinants of this balance are the Starling forces, the lymphatic drainage, and the active trans-membrane transport. When fluid or air accumulate inside the pleural cavity, pleural pressure rises to atmospheric level causing the lung to collapse while the chest wall to expand. The displacement is not equally distributed between lung and chest wall, because it depends upon their own compliance. Pneumothorax and pleural effusion are common diseases in critically-ill patients. Pneumothorax is divided in two groups based upon the aetiological mechanism—spontaneous and traumatic. Pleural effusion is classified as transudates or exudates, mainly based on protein content; this classification comprises different pathological mechanisms beneath the two kind of pleural effusion.

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20

Heat Transfer to the Inclined Trailing Wall of an Open Cavity. Storming Media, 1998.

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21

Mckie, Andrew, and Ian Skeate. A Practical Guide to Running Housing Disrepair and Cavity Wall Claims: 2nd Edition. Law Brief Publishing, 2018.

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22

Effects of cavity dimensions, boundary layer, and temperature on cavity noise with emphasis on benchmark data to validate computational aerocoustic codes. Hampton, Va: National Aeronautics and Space Administration, Langley Research Center, 1995.

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23

Sluce, A. J. A. Installing cavity wall insulation in existing housing: A demonstration by the Bournville Village Trust. Great Britain, Energy Efficiency Office, 1990.

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24

Wilkes, N. S. The Prediction of Turbulent Three-dimensional Fluid Flow in a Rectangular Cavity with a Moving Wall. AEA Technology Plc, 1986.

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25

Great Britain. Energy Efficiency Office., ed. Energy efficient refurbishment of low rise cavity wall housing: Case studies with guidance for social housing landlords and contractors. Energy Efficiency Office, 1995.

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26

Anderson, Robert H., Nigel A. Brown, Simon D. Bamforth, Bill Chaudhry, Deborah J. Henderson, and Timothy J. Mohun. Development of the outflow tract. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0023.

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The definitive cardiac outflow tracts have three components: the intra-pericardial arterial trunks, the arterial roots, and the ventricular outflow tracts. Improved correlations between normal development and cardiac malformations can be obtained by analysing the developing outflow tract in tripartite fashion with proximal, intermediate, and distal components. When first seen, the walls of the entire outflow tract express myocardial markers. With ongoing development, the distal border regresses away from the edges of the pericardial cavity. Subsequently, the distal outflow tract becomes the intra-pericardial arterial trunks, with a protrusion from the dorsal wall of the aortic sac forming the aortopulmonary septum. The arterial valves form in the intermediate part of the outflow tract. The proximal part eventually becomes transformed into the ventricular outflow tracts, with muscularization of the proximal cushions producing the right ventricular infundibulum. This approach provides rational explanations for the congenital lesions involving the different parts of the outflow tracts.

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27

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Gastrointestinal infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0016.

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This chapter covers oesophagitis (which is inflammation of the oesophagus), peptic ulcer disease, infectious diarrhoea (including dysentery and enteric or typhoid fever), cholera, Clostridium difficile diarrhoea, acute cholecystitis which is an inflammation of the gall bladder, acute cholangitis (characterized by fever, jaundice, and abdominal pain), pancreatitis (which is inflammation of the pancreas), primary and secondary peritonitis (which is infection of the peritoneal cavity), peritoneal dialysis peritonitis, diverticulitis (sac-like protrusions of the colonic wall), intra-abdominal abscess, liver abscess, and acute and chronic hepatitis (which are inflammation of the liver).

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28

Harrison, Mark. Abdomen. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198765875.003.0004.

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This chapter describes the anatomy of the abdomen as it applies to Emergency Medicine, and in particular the Primary FRCEM examination. The chapter outlines the key details of regions, muscles, functions, blood supply, nerve supply, surface markings, and relations of the abdominal wall and cavity, inguinal region, testis, epididymis and spermatic cord, peritoneum, gastrointestinal tract, liver and biliary tract, pancreas, spleen, kidneys, ureters and bladder, pelvis, prostate, reproductive systems, and genital regions. This chapter is laid out exactly following the RCEM syllabus, to allow easy reference and consolidation of learning.

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29

Replacement Ties in Cavity Walls. Construction Industry Research & Information Association (CIRIA), 1988.

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30

Association, Aggregate Concrete Block, ed. Cavity insulated walls: Specifiers guide. Leicester: Aggregate Concrete Block Association, etc, 1987.

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31

Lancellotti, Patrizio, and Bernard Cosyns. Assessment of the Left Ventricular Systolic Function. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0004.

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Evaluation of ventricular systolic function and cavity dimensions is an essential part of the echocardiographic examination. Treatment strategy and decisionmaking for a patient’s condition is affected by systolic function. Echocardiography plays a major in monitoring the effects of therapy. Appropriate knowledge about how to assess left ventricular size, shape and function is thus crucial. This chapter demonstrates left chamber quantification through various measurements of left ventricular size and dimensions, left ventricular mass, left ventricularglobal function, regional wall motion, left ventricular segmentation, global left ventricular remodelling, and left atrial measurements. Techniques, advantages, and limitations of different methods and echocardiographic examinations are given throughout.

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32

British Steel Corporation. General Steels Commercial Division. and British Constructional Steelwork Association, eds. Steelwork corrosion protection guide: Cavity walls. Redcar: British Steel Corporation, BSC General Steels Commercial Division, 1986.

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33

1958-, Wang Ru, ed. Behaviour of masonry cavity walls under vertical eccentric loads. Edmonton, Alta., Canada: Dept. of Civil Engineering, University of Alberta, 1996.

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34

Hens, Hugo S. L. Performance Based Building Design 1: From below Grade Construction to Cavity Walls. Ernst & Sohn Verlag fur Architektur und Technische, Wilhelm, 2012.

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35

Hens, Hugo S. L. Performance Based Building Design 1: From below Grade Construction to Cavity Walls. Wiley & Sons, Limited, John, 2012.

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36

Hens, Hugo S. L. Performance Based Building Design 1: From below Grade Construction to Cavity Walls. Ernst & Sohn Verlag fur Architektur und Technische, Wilhelm, 2012.

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37

Hens, Hugo S. L. Performance Based Building Design 1: From below Grade Construction to Cavity Walls. Ernst & Sohn Verlag fur Architektur und Technische, Wilhelm, 2012.

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38

Great Britain. Energy efficiency Office. and Building Research EnergyConservation Support Unit., eds. Energy Efficiency in new housing: Detailing for designers and building professionals : external cavity walls. [UK]: Energy Efficiency Office, 1993.

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39

Great Britain. Energy Efficiency Office. and BuildingResearch Energy Conservation Support Unit., eds. Energy efficiency in new housing: Site practice for tradesmen : external walls: injected cavity insulation. [UK]: Energy efficiency Office, 1993.

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40

Zoccali, Carmine, Davide Bolignano, and Francesca Mallamaci. Left ventricular hypertrophy in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0107_update_001.

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Alterations in left ventricular (LV) mass and geometry and LV dysfunction increase in prevalence from stage 2 to stage 5 in CKD. Nuclear magnetic resonance is the most accurate and precise technique for measuring LV mass and function in patients with heart disease. Quantitative echocardiography is still the most frequently used means of evaluating abnormalities in LV mass and function in CKD. Anatomically, myocardial hypertrophy can be classified as concentric or eccentric. In concentric hypertrophy, the muscular component of the LV (LV wall) predominates over the cavity component (LV volume). Due to the higher thickness and myocardial fibrosis in patients with concentric LVH, ventricular compliance is reduced and the end-diastolic volume is small and insufficient to maintain cardiac output under varying physiological demands (diastolic dysfunction). In those with eccentric hypertrophy, tensile stress elongates myocardiocytes and increases LV end-diastolic volume. The LV walls are relatively thinner and with reduced ability to contract (systolic dysfunction). LVH prevalence increases stepwisely as renal function deteriorates and 70–80% of patients with kidney failure present with established LVH which is of the concentric type in the majority. Volume overload and severe anaemia are, on the other hand, the major drivers of eccentric LVH. Even though LVH may regress after renal transplantation, the prevalence of LVH after transplantation remains close to that found in dialysis patients and a functioning renal graft should not be seen as a guarantee of LVH regression. The vast majority of studies on cardiomyopathy in CKD are observational in nature and the number of controlled clinical trials in these patients is very small. Beta-blockers (carvedilol) and angiotensin receptors blockers improve LV performance and reduce mortality in kidney failure patients with LV dysfunction. Although current guidelines recommend implantable cardioverter-defibrillators in patients with ejection fraction less than 30%, mild to moderate symptoms of heart failure, and a life expectancy of more than 1 year, these devices are rarely offered to eligible CKD patients. Conversion to nocturnal dialysis and to frequent dialysis schedules produces a marked improvement in LVH in patients on dialysis. More frequent and/or longer dialysis are recommended in dialysis patients with asymptomatic or symptomatic LV disorders if the organizational and financial resources are available.

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41

Rider, Jennifer R., Paul Brennan, and Pagona Lagiou. Oral and Pharyngeal Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0007.

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This chapter covers cancer of the oral cavity and the oropharynx, which includes the base of the tongue, soft palate, tonsils, and back and side walls of the throat. Many important risk factors for oral and oropharyngeal cancer have been identified, and in 2007 the World Health Organization determined there was sufficient evidence to include human papilloma virus (HPV) type 16 as a cause of these cancers. Tobacco and alcohol remain important modifiable risk factors, but the increasing incidence of HPV-associated tumors is now evident. While these tumors are more amenable to treatment than HPV-negative tumors, they are still a source of considerable morbidity and mortality. Moreover, the lack of a precursor lesion and limited data on efficacy of the HPV vaccine in preventing oral HPV infection are barriers to primary and secondary prevention efforts. Dietary patterns high in fruits and vegetables and low in meats may confer some protection.

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