Relevant bibliographies by topics / CCR5

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'CCR5'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "CCR5"

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Korbecki, Jan, Klaudyna Kojder, Katarzyna Barczak, et al. "Hypoxia Alters the Expression of CC Chemokines and CC Chemokine Receptors in a Tumor–A Literature Review." International Journal of Molecular Sciences 21, no. 16 (2020): 5647. http://dx.doi.org/10.3390/ijms21165647.

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Hypoxia, i.e., oxygen deficiency condition, is one of the most important factors promoting the growth of tumors. Since its effect on the chemokine system is crucial in understanding the changes in the recruitment of cells to a tumor niche, in this review we have gathered all the available data about the impact of hypoxia on β chemokines. In the introduction, we present the chronic (continuous, non-interrupted) and cycling (intermittent, transient) hypoxia together with the mechanisms of activation of hypoxia inducible factors (HIF-1 and HIF-2) and NF-κB. Then we describe the effect of hypoxia
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Princen, Katrien, Sigrid Hatse, Kurt Vermeire, et al. "Inhibition of Human Immunodeficiency Virus Replication by a Dual CCR5/CXCR4 Antagonist." Journal of Virology 78, no. 23 (2004): 12996–3006. http://dx.doi.org/10.1128/jvi.78.23.12996-13006.2004.

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ABSTRACT Here we report that the N-pyridinylmethyl cyclam analog AMD3451 has antiviral activity against a wide variety of R5, R5/X4, and X4 strains of human immunodeficiency virus type 1 (HIV-1) and HIV-2 (50% inhibitory concentration [IC50] ranging from 1.2 to 26.5 μM) in various T-cell lines, CCR5- or CXCR4-transfected cells, peripheral blood mononuclear cells (PBMCs), and monocytes/macrophages. AMD3451 also inhibited R5, R5/X4, and X4 HIV-1 primary clinical isolates in PBMCs (IC50, 1.8 to 7.3 μM). A PCR-based viral entry assay revealed that AMD3451 blocks R5 and X4 HIV-1 infection at the vi
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Zhang, Yi-jun, Tatjana Dragic, Yunzhen Cao, et al. "Use of Coreceptors Other Than CCR5 by Non-Syncytium-Inducing Adult and Pediatric Isolates of Human Immunodeficiency Virus Type 1 Is Rare In Vitro." Journal of Virology 72, no. 11 (1998): 9337–44. http://dx.doi.org/10.1128/jvi.72.11.9337-9344.1998.

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ABSTRACT We have tested a panel of pediatric and adult human immunodeficiency virus type 1 (HIV-1) primary isolates for the ability to employ the following proteins as coreceptors during viral entry: CCR1, CCR2b, CCR3, CCR4, CCR5, CCR8, CXCR4, Bonzo, BOB, GPR1, V28, US28, and APJ. Most non-syncytium-inducing isolates could utilize only CCR5. All syncytium-inducing viruses used CXCR4, some also employed V28, and one (DH123) used CCR8 and APJ as well. A longitudinal series of HIV-1 subtype B isolates from an infected infant and its mother utilized Bonzo efficiently, as well as CCR5. The maternal
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Yoon, Sunjoo, Ju Hyun Lee, Minsu Kang, et al. "Abstract 5951: The correlation between single nucleotide polymorphism of chemokine receptor and ligand and infiltrating immune cells on tumor microenvironment of gastric cancer." Cancer Research 83, no. 7_Supplement (2023): 5951. http://dx.doi.org/10.1158/1538-7445.am2023-5951.

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Abstract Objective: Chemokines play roles in many normal biological processes, such as hematopoietic cell genesis and leukocyte migration and homing. Chemokines could be responsible for eliciting local accumulation of immune cells within tumor microenvironment. In this study, we examine if genetic variations in chemokine receptor and ligand are associated with infiltrations of immune cells on tumor microenvironment in gastric cancer. Methods: In 356 gastric cancer patients who received curative surgery followed by adjuvant chemotherapy, total 105 single nucleotide polymorphisms (SNP) in a chem
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Taylor, James R., Katherine C. Kimbrell, Robert Scoggins, Marie Delaney, Lijun Wu, and David Camerini. "Expression and Function of Chemokine Receptors on Human Thymocytes: Implications for Infection by Human Immunodeficiency Virus Type 1." Journal of Virology 75, no. 18 (2001): 8752–60. http://dx.doi.org/10.1128/jvi.75.18.8752-8760.2001.

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ABSTRACT The presence or absence of the receptor CD4 and the coreceptors CCR5 and CXCR4 restrict the cell tropism of human immunodeficiency virus type 1 (HIV-1). Despite the importance of thymic infection by HIV-1, conflicting reports regarding the expression of HIV-1 coreceptors on human thymocytes have not been resolved. We assayed the expression and function of the major HIV-1 coreceptors, CCR5 and CXCR4, as well as CCR4 and CCR7 as controls, on human thymocytes. We detected CCR5 on 2.5% of thymocytes, CXCR4 on 53% of the cells, and CCR4 on 16% and CCR7 on 11% of human thymocytes. Moreover,
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Mazur, Grzegorz, Emilia Jaskula, Ilona Kryczek, et al. "Gene Expression for Chemokine Receptors Influences Survival of Non-Hodgkin Lymphoma Patients." Blood 116, no. 21 (2010): 3103. http://dx.doi.org/10.1182/blood.v116.21.3103.3103.

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Abstract Abstract 3103 Non-Hodgkin's lymphoma (nHL) represent heterogenous group of lymphoid malignancies derived from B and T lymphocytes, NK cells or histiocytes. Most of lymphomas are B-cell origin. Lymphoma cells can migrate to other organs and their migration could be linked to chemokines and their receptors. Chemokine receptors are expressed by many cell populations, including lymphoid cells, and their main function is lymphocytes. Chemokine receptors guide lymphocytes homing, chemotaxis, adhesion and interplaying between immunologic system response cells. They are also responsible for c
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Ren, Han-Yun, Meng Wang, Xiang-Juan Ma, Yu-Jun Dong, Zhi-Xiang Qiu, and Wei Liu. "Differential Regulation Of Chemokine Receptor Expressions On T Lymphocyte Subsets In Healthy Donors After Mobilization With Rhg-CSF and Its Correlation With Acute GvHD." Blood 122, no. 21 (2013): 3296. http://dx.doi.org/10.1182/blood.v122.21.3296.3296.

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Abstract Introduction This study is aimed to investigate chemokine receptors (CCR5, CCR6, CCR7, CCR9, CXCR3 and CCR2) expression on T cell subsets in healthy donors after mobilization with recombinant human granulocyte colony-stimulating factor (rhG-CSF) and analyze its correlation with acute graft-versus-host disease (aGVHD) and to understand the possible mechanisms underlying rhG-CSF-induced immune tolerance. Methods Sixty-eight healthy donor and their recipient pairs of family donor allogeneic hematopoietic stem cell transplantation (allo-HSCT) were included in this study. The expressions o
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Kim, Chang H., Jeeho Lee, and Seung G. Kang. "Developmental and antigen-driven switches in the trafficking receptors of FoxP3+ regulatory T cells (99.2)." Journal of Immunology 178, no. 1_Supplement (2007): S194. http://dx.doi.org/10.4049/jimmunol.178.supp.99.2.

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Abstract FoxP3+ regulatory T cells play important roles in immune regulation and tolerance. There is an increasing body of evidence that the migration ability of FoxP3+ T cells is important for their regulatory functions at effector tissue sites. We investigated the two different trafficking receptor switches of FoxP3+ T cells occurring in the thymus and secondary lymphoid tissues. The first trafficking receptor switch in the thymus is developmentally programmed: Precursors of FoxP3+ cells undergo the first trafficking receptor switch from CCR8/CCR9 to CXCR4 and then finally to CCR7, generatin
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Paula Costa, Guilherme de, Laís Roquete Lopes, Maria Cláudia da Silva, et al. "Doxycycline and Benznidazole Reduce the Profile of Th1, Th2, and Th17 Chemokines and Chemokine Receptors in Cardiac Tissue from ChronicTrypanosoma cruzi-Infected Dogs." Mediators of Inflammation 2016 (2016): 1–11. http://dx.doi.org/10.1155/2016/3694714.

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Chemokines (CKs) and chemokine receptors (CKR) promote leukocyte recruitment into cardiac tissue infected by theTrypanosoma cruzi. This study investigated the long-term treatment with subantimicrobial doses of doxycycline (Dox) in association, or not, with benznidazole (Bz) on the expression of CK and CKR in cardiac tissue. Thirty mongrel dogs were infected, or not, with the Berenice-78 strain ofT. cruziand grouped according their treatments: (i) two months after infection, Dox (50 mg/kg) 2x/day for 12 months; (ii) nine months after infection, Bz (3,5 mg/kg) 2x/day for 60 days; (iii) Dox + Bz;
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Zvejniece, Laura, Svetlana Kozireva, Zanna Rudevica, et al. "Expression of the Chemokine Receptor CCR1 in Burkitt Lymphoma Cell Lines Is Linked to the CD10-Negative Cell Phenotype and Co-Expression of the EBV Latent Genes EBNA2, LMP1, and LMP2." International Journal of Molecular Sciences 23, no. 7 (2022): 3434. http://dx.doi.org/10.3390/ijms23073434.

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Chemokines and their receptors regulate the migration of immune cells and the dissemination of cancer cells. CCR1, CCR2, CCR3, and CCR5 all belong to a single protein homology cluster and respond to the same inflammatory chemokines. We previously reported that CCR1 and CCR2B are induced upon Epstein-Barr virus (EBV) infection of B cells in vitro. EBV is present in almost all cases of endemic Burkitt lymphoma (BL); however, the contribution of EBV in the pathogenesis of the disease is not fully understood. Here, we analyzed the relation of the expression of CCR1, CCR2, CCR3, and CCR5, the EBV D
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Dissertations / Theses on the topic "CCR5"

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Schauren, Juliana da Silveira. "Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2013. http://hdl.handle.net/10183/78127.

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O Lúpus Eritematoso Sistêmico (LES) é uma doença autoimune inflamatória crônica que possui uma etiopatogênese complexa. Diversos fatores participam da patogênese da doença, dentre eles alterações no balanço de citocinas e quimiocinas. As quimiocinas e seus receptores são fundamentais na regulação da migração de leucócitos durante a inflamação e acredita-se que elas possam ter um papel importante na patogênese de doenças autoimunes, inclusive no LES. Diversos estudos abordaram o papel de quimiocinas e seus receptores no LES, porém, principalmente se tratando dos receptores de quimiocinas CCR5 e
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Sax, Michael John. "The CCL5-CCR5 Axis in Breast Cancer." Thesis, Griffith University, 2015. http://hdl.handle.net/10072/365646.

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Cancer is one of the leading underlying causes of death worldwide. Despite decades of research, cancer is predicted to be the leading cause of death by the year 2020. Anti angiogenic therapies that target the key signalling molecule, vascular endothelial growth factor (VEGF) have shown promise in the treatment of some solid tumours, resulting in increased progression-free survival times in patients. However, there are several significant problems with current anti-angiogenic therapies, such as the phenomenon of resistance. Tumours can be divided into those that are intrinsically nonresponsive
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Sato, Wakiro. "Human Th17 Cells Are Identified as Bearing CCR2+CCR5- Phenotype." Kyoto University, 2008. http://hdl.handle.net/2433/124335.

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John, Bangan. "Association Among CCR5 Genotypes, CCR5 Expression, And In Vitro HIV Infection." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1365888090.

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Sohy, Denis. "Etude de la dimérisation des récepteurs aux chimiokines CCR2, CCR5 et CXCR4." Doctoral thesis, Universite Libre de Bruxelles, 2008. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210282.

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La dimérisation des récepteurs couplés aux protéines G est un nouveau concept apparu dans la littérature au cours des quelques années qui ont précédé le début de notre travail. Bien qu’il soit clairement établi que les récepteurs sont capables de former des homo et des hétérodimères, les conséquences fonctionnelles de telles interactions demeurent souvent peu claires. Dans une étude précédente, le laboratoire d’accueil a montré que les récepteurs aux chimiokines CCR2 et CCR5 forment des homo et des hétérodimères de manière constitutive et identifié une coopérativité négative de liaison de natu
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Longden, James. "Quantitative approaches to the study of the trafficking of CCR1 and CCR5." Thesis, University of Nottingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.437076.

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Zambra, Francis Maria Báo. "Influência dos genes CCR2 e CCR5 em hiperplasia e câncer de próstata." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/69706.

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A hiperplasia prostática benigna (HPB) e o câncer de próstata (CaP) são duas condições crônicas muito comuns em homens com idade avançada e têm sido relacionadas a processos inflamatórios. As quimiocinas são reconhecidas como mediadores críticos da resposta inflamatória por regular a migração das células imunológicas através da ativação de receptores de quimiocinas na superfície destas células. As quimiocinas estão relacionadas à patogênese tumoral, embora não seja claro de que modo afetam a progressão tumoral humana. O objetivo desse estudo foi investigar a associação de dois polimorfismos de
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Fish, Richard James. "RANTES derivatives and CCR5." Thesis, University of York, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.369362.

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Simonis, Christopher. "Molekulare Klonierung, stabile Transfektion und funktionelle Expression der murinen Chemokinrezeptoren Ccr2 und Ccr5." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-99950.

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Manin, Graziele Zenaro. "Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21052014-153321/.

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A doença dos legionários consiste em uma broncopneumonia severa e atípica, que acomete de 2 a 7% das pessoas infectadas com Legionella spp e que apresenta taxa de mortalidade que varia de 5 a 30%, sendo considerada uma importante causa de morbidade e mortalidade mundial. A patologia causada pela espécie L. pneumophila tem sido amplamente estudada em modelos experimentais e suas características clínicas foram extensivamente descritas. No entanto, este modelo não representa adequadamente a doença que acomete seres humanos, pois L. pneumophila não é letal aos camundongos como é para humanos. Rece
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Books on the topic "CCR5"

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Eiter, Viktoria. Entwicklung monoklonaler Antikörper gegen die humanen Chemokinrezeptoren CCR5 und CCR2. [s.n.], 1999.

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Masters, Jennefer. Myxoma virus induced activation of CC-chemokine receptor 5 (CCR5). National Library of Canada, 2000.

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National Institute of Allergy and Infectious Diseases (U.S.) and National Institutes of Health (U. S.), eds. People who lack a cell surface protein called CCR5 are highly resistant to infection by HIV but may be at increased risk of developing West Nile virus. National Institutes of Health, 2006.

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R, Rogelet Keri, ed. Adult CCRN certification review. Jones and Bartlett Publishers, 2009.

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R, Rogelet Keri, ed. Pediatric CCRN certification review. Jones & Bartlett Learning, 2012.

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Brorsen, Ann J. Adult CCRN certification review. 2nd ed. Jones & Bartlett Learning, 2014.

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Bhat, B. V. Rajarama. Cocycles of CCR flows. American Mathematical Society, 2001.

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1948-, Ashmore Wendy, and Knapp Arthur Bernard, eds. Archaeologies of landscape: Contemporary perspectives. Blackwell, 1999.

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Gallagher, A. M. Attitudes to higher education: Report to CCRU and DENI. Centre for Research on Higher Education, 1996.

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Horgan, Carmel Mary Teresa. Studies on Mitozolomide (CCRG 81010), a new antineoplastic agent. University of Aston.Department of Pharmacy, 1985.

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Book chapters on the topic "CCR5"

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Hütter, Gero. "CCR5." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101567.

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Hütter, Gero. "CCR5." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101567-1.

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Mir, Manzoor Ahmad, Aamina Manzoor, and Nusrat Jan. "CCL5/CCR5 Axis in Cancer." In Cytokine and Chemokine Networks in Cancer. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4657-0_8.

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Soria, Gali, and Adit Ben-Baruch. "The CCL5/CCR5 Axis in Cancer." In Chemokine Receptors in Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-267-4_7.

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Yasuda, Nobuyoshi. "CCR5 Receptor Antagonist." In The Art of Process Chemistry. Wiley-VCH Verlag GmbH & Co. KGaA, 2010. http://dx.doi.org/10.1002/9783527633562.ch2.

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Pryde, David C., and Christopher G. Barber. "CCR5 Antagonists in HIV." In Methods and Principles in Medicinal Chemistry. Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527631995.ch10.

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Junker, Anna, Artur Kamil Kokornaczyk, Ann Kathrin Strunz, and Bernhard Wünsch. "Selective and Dual Targeting of CCR2 and CCR5 Receptors: A Current Overview." In Topics in Medicinal Chemistry. Springer International Publishing, 2014. http://dx.doi.org/10.1007/7355_2014_40.

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Cornu, Tatjana I., Claudio Mussolino, Kristie Bloom, and Toni Cathomen. "Editing CCR5: A Novel Approach to HIV Gene Therapy." In Advances in Experimental Medicine and Biology. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2432-5_6.

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Hütter, Gero. "Transplantation of CCR5-Δ32/Δ32 Stem Cells May Cure HIV Infection." In Stem Cells and Cancer Stem Cells, Volume 13. Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-7233-4_3.

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Zhong, Yanchuan, Yunmeng Peng, and Yiheng Cai. "Spatial Distribution and Determinants of Convalescence Climate Tourist Attraions in Sichuan, China." In Lecture Notes in Civil Engineering. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-97-8401-1_8.

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AbstractThis study analyses the distribution pattern, directionality, and regional aggregation characteristics of W-CCR, S-CCR, and Y-CCR in Sichuan Province, using POI (Point of Interest) data from 538 Convalescent Climate Resorts (CCR) and daily data from 156 national meteorological stations from 1991 to 2020. The factors that influence the spatial distribution of CCR are discussed from the perspectives of climate, natural environment, and human society. The data suggests that CCR is concentrated along the southern edge of the basin, the eastern side of the Hengduan Mountains at the border o
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Conference papers on the topic "CCR5"

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Wadman, Grant, Irina Kufareva, John Dawson, et al. "Molecular Mechanisms of Antagonist Selectivity Against CCR2 and CCR5." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.569.131403.

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Wang, Hao, Nok Him Fung, Christian Aloe, et al. "CCR2/CCR5-dependent Lyve1lowMHCIIhigh interstitial macrophages regulate airway inflammation in asthma." In ERS Lung Science Conference 2025 abstracts. European Respiratory Society, 2025. https://doi.org/10.1183/23120541.lsc-2025.yi4.

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Hsieh, Chia-Ling, Che-Ming Liu, and Shian-Ying Sung. "Abstract 1559: CCR5-CCRL2 regulates CCL5-induced organ specific metastasis: thein vitroandin vivostudies." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1559.

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Abid, S., E. Marcos, A. Parpaleix, et al. "CCR2- and CCR5-Mediated Crosstalk Between Macrophages and Pulmonary-Artery Smooth Muscle Cells Drives Pulmonary Hypertension." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4410.

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Parpaleix, Aurelien, Shariq Abid, Elisabeth Marcos, et al. "Targeting CCR2 and CCR5 to inhibit macrophage/pulmonary artery smooth muscle cells cross-talk in pulmonary hypertension." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa3066.

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Chang, Hui-Wen, Hui-Ju Li, Chia-Ling Hsieh, and Shian-Ying Sung. "Abstract 302: CCR5 and CCRL2 accommodating responses to CCL5 induces directional cancer cells migration and organ specific metastasis." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-302.

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Agarwal, Alpna, Eron Oronsaye, Arthur Nadas, Susan Zolla-Pazner, and Timothy Cardozo. "CCR5/CXCR4 discriminating sites in the HIV-1 gp120 core." In the 2nd ACM Conference. ACM Press, 2011. http://dx.doi.org/10.1145/2147805.2147888.

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Zhao, W., RL Toonkel, M. Sole, AC Borczuk, and CA Powell. "Lung Adenocarcinoma Invasion by TGFBRII Deficient Cells Requires RANTES/CCR5." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5133.

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Halama, Niels, Inka Zoernig, Anna Berthel, et al. "Abstract 3021: CCR5 inhibition: macrophage repolarization therapy for colorectal cancer." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3021.

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Silva, Thauane, Gabrielle Vale, André Ferreira, et al. "Modulation of host antiviral restriction factors by ccr5 and cxcr4 ligands." In IV International Symposium on Immunobiologicals & VII Seminário Anual Científico e Tecnológico. Instituto de Tecnologia em Imunobiológicos, 2019. http://dx.doi.org/10.35259/isi.sact.2019_32700.

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Reports on the topic "CCR5"

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liu, shisi, haiying zhou, lu chen, et al. Meta-analysis of MnSOD rs4880, CCR5 rs1799987 polymorphisms associated with diabetic nephropathy risk. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.9.0001.

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Chen, Liangyu, Shi Yin, Yunqing Hou, et al. The therapeutic effect of inhibition of CCR5 on animal models of stroke or traumatic brain injury: A Systematic review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.1.0026.

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Adair, M. The CCRS Quicklook Swath Browser. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2000. http://dx.doi.org/10.4095/219632.

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Adair, M. The CCRS Quicklook Swath Browser. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2000. http://dx.doi.org/10.4095/219646.

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Cyr, I., and Th Toutin. RADARSAT-1 Stereo Advisor on CCRS Web. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2001. http://dx.doi.org/10.4095/219705.

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Graham, D. F., A. N. Rencz, and V. H. Singhroy. GSC - CCRS Storefront Project, conclusions from selected projects. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1994. http://dx.doi.org/10.4095/193706.

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Vachon, P. W., A. L. Gray, and K. E. Mattar. RADARSAT and ERS Repeat-Pass SAR Interferometry at CCRS. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1998. http://dx.doi.org/10.4095/219176.

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Zou, Chenghui, Weng Zhang, Mao Li, Dan He, Yujie Han, and Mao Lu. A meta-analysis of association between CCL5、CCL11、CCL17 polymorphisms and AD. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0148.

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Abstract:
Review question / Objective: At present, many studies on the association between CCL5、CCL11、CCL17 polymorphisms and atopic dermatitis（AD）are inconsistent. We conducted this meta-analysis of Case control trial to evaluate the association between CCL5、CCL11、CCL17 polymorphisms and atopic dermatitis（AD). Condition being studied: Since the discovery of cytokines, and in particular the role of chemokines in the progression of AD, many clinical studies have been carried out around the world to explore the association of AD with chemokine polymorphism. However, the quality, type and conclusions of st
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Bentz, Dale P., Xiuping Feng, Claus-Jochen Haecker, and Paul E. Stutzman. Analysis of CCRL proficiency cements 135 and 136 using CEMHYD3D. National Institute of Standards and Technology, 2000. http://dx.doi.org/10.6028/nist.ir.6545.

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Wolfe, S. A. Permafrost science at ESS: a workshop on GSC/CCRS scientific opportunities. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 2010. http://dx.doi.org/10.4095/263373.

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