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Relevant bibliographies by topics / CD44 molecule (Indian blood group)

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Journal articles

Academic literature on the topic 'CD44 molecule (Indian blood group)'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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Journal articles on the topic "CD44 molecule (Indian blood group)"

1

Abdelmonem, M., N. Ngo, W. Cai, et al. "Racial Disparity in Antibody Against A rare High Prevalence Antigen; (Anti-Inb)." American Journal of Clinical Pathology 160, Supplement_1 (2023): S108. http://dx.doi.org/10.1093/ajcp/aqad150.237.

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Abstract Introduction/Objective Introduction: Ina antigen is very rare except in Iranian and Arab populations (10.6% and 11.8%, respectively). In 1973, Badkere et al. identified this antigen in about 3% of Indians from Bombay. Inb is a very high-prevalence antigen. It is prevalent in 99.9% of Caucasians and blacks, 96% of Indians and South Asians, and 90% of Iranians and Arabs. The Indian blood group system also has two other very high prevalence antigens: INFI and INJA. Indian blood group resides in CD44 glycoprotein that binds to hyaluronate of extracellular matrix. CD44 is an adhesion molec

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2

Kushwaha, Prem Prakash, Shiv Verma, Shashank Kumar, and Sanjay Gupta. "Role of prostate cancer stem-like cells in the development of antiandrogen resistance." Cancer Drug Resistance 5, no. 2 (2022): 459–71. http://dx.doi.org/10.20517/cdr.2022.07.

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Androgen deprivation therapy (ADT) is the standard of care treatment for advance stage prostate cancer. Treatment with ADT develops resistance in multiple ways leading to the development of castration-resistant prostate cancer (CRPC). Present research establishes that prostate cancer stem-like cells (CSCs) play a central role in the development of treatment resistance followed by disease progression. Prostate CSCs are capable of self-renewal, differentiation, and regenerating tumor heterogeneity. The stemness properties in prostate CSCs arise due to various factors such as androgen receptor mu

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3

Hale, L. P., K. H. Singer, and B. F. Haynes. "CD44 antibody against In(Lu)-related p80, lymphocyte-homing receptor molecule inhibits the binding of human erythrocytes to T cells." Journal of Immunology 143, no. 12 (1989): 3944–48. http://dx.doi.org/10.4049/jimmunol.143.12.3944.

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Abstract The cluster of differentiation 44 (CD44) Ag system, originally described in brain and mature T cells, has been subsequently shown to be identical with the human lymphocyte homing-receptor defined by the Hermes-1 antibody and to be involved in T cell/endothelial cell interactions in synovium, mucosa, and lymph node. CD44 is also present on human E. On E, CD44 has been shown to be regulated by the In(Lu) dominant inhibitor gene and to express the Ina and Inb blood group Ag. Because human E have been shown to interact with human T cells via CD2 on T cells and LFA-3 on human E, we have st

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Viana, Denise, Victor Andrade, Marcelo Salgado, Carolina Vasconcelos, and Leuridan Torres. "Abstract PO4-24-10: Prognostic role of SOX2 and STAT3 expression on circulating T lymphocytes and CD44+/CD24neg cells in the locally advanced and metastatic breast cancer." Cancer Research 84, no. 9_Supplement (2024): PO4–24–10—PO4–24–10. http://dx.doi.org/10.1158/1538-7445.sabcs23-po4-24-10.

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Abstract Introduction: Breast cancer (BC) is associated with a continuous increase in incidence, with high mortality rates in several countries. There are four immunophenotypes of BC, luminal A, luminal B, triple negative (TN), and HER2+. Some proteins in tumor cells and the tumor microenvironment are part of tumorigenesis and metastases. The CD44 molecule is considered an important marker of the inflammatory response. CD44 is present in all leukocytes and on the surface of tumor stem cells, involving tumor invasion and metastasis. Some authors have demonstrated that the signal transducer and

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5

Ning, Xueqin, Xiaolei Wei, Bingyuan Chen, et al. "CD44 Expression in Different Plasma Cell Diseases." Blood 138, Supplement 1 (2021): 4754. http://dx.doi.org/10.1182/blood-2021-146780.

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Abstract CD44 expression in different plasma cell diseases Introduction Myeloma is a genetically complex disease which develops via a multistep process whereby plasma cells are driven towards malignancy through the accumulation of genetic "hits" over time. This multistep process permits myeloma to have four recognisable clinical stages including monoclonal gammopathy of undetermined significance (MGUS) , smouldering multiple myeloma (SMM) , multiple myeloma (MM) and plasma cell leukemia (PCL). CD44 as a complex adhesion molecule, is highly expressed in a variety of solid tumors and involved in

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6

Guazzone, V. A., B. Denduchis, and L. Lustig. "Involvement of CD44 in leukocyte recruitment to the rat testis in experimental autoimmune orchitis." Reproduction 129, no. 5 (2005): 603–9. http://dx.doi.org/10.1530/rep.1.00329.

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Experimental autoimmune orchitis (EAO) is characterized by an interstitial mononuclear cell infiltrate and a severe lesion of the seminiferous tubules with germ cells that undergo apoptosis and sloughing. The aim of this study was to determine the role of CD44 in testicular leukocyte recruitment in EAO. The biological functions of CD44 have been attributed to the generation of a functionally active hyaluronan-binding phenotype. Orchitis was induced in Sprague–Dawley adult rats by active immunization with an emulsion of testicular homogenate and complete Freund’s adjuvant using Bordetella pertu

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7

Poole, Joyce, Louise Tilley, Nicole Warke, et al. "Two missense mutations in the CD44 gene encode two new antigens of the Indian blood group system." Transfusion 47, no. 7 (2007): 1306–11. http://dx.doi.org/10.1111/j.1537-2995.2007.01275.x.

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8

De Rossi, G., D. Zarcone, F. Mauro, et al. "Adhesion molecule expression on B-cell chronic lymphocytic leukemia cells: malignant cell phenotypes define distinct disease subsets." Blood 81, no. 10 (1993): 2679–87. http://dx.doi.org/10.1182/blood.v81.10.2679.2679.

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Abstract Expression of surface adhesion molecules of the Ig superfamily (CD54 and CD58), of the integrin family (beta 1, beta 2, and beta 3 chains), of the selectin family (L-selectin), and of the lymphocyte homing receptor (CD44) was analyzed on B-cell chronic lymphocytic leukemia (B- CLL) cells from 74 patients. The aim of the study was the definition of phenotypically distinct disease subsets and the correlation of adhesion molecule phenotypes with clinical parameters. Expression of CD58 on B- CLL cells defined more advanced disease stages. In comparison with beta chain-positive cases, pati

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9

De Rossi, G., D. Zarcone, F. Mauro, et al. "Adhesion molecule expression on B-cell chronic lymphocytic leukemia cells: malignant cell phenotypes define distinct disease subsets." Blood 81, no. 10 (1993): 2679–87. http://dx.doi.org/10.1182/blood.v81.10.2679.bloodjournal81102679.

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Expression of surface adhesion molecules of the Ig superfamily (CD54 and CD58), of the integrin family (beta 1, beta 2, and beta 3 chains), of the selectin family (L-selectin), and of the lymphocyte homing receptor (CD44) was analyzed on B-cell chronic lymphocytic leukemia (B- CLL) cells from 74 patients. The aim of the study was the definition of phenotypically distinct disease subsets and the correlation of adhesion molecule phenotypes with clinical parameters. Expression of CD58 on B- CLL cells defined more advanced disease stages. In comparison with beta chain-positive cases, patients whos

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10

Kanjee, Usheer, Christof Grüring, Mudit Chaand, et al. "CRISPR/Cas9 knockouts reveal genetic interaction between strain-transcendent erythrocyte determinants ofPlasmodium falciparuminvasion." Proceedings of the National Academy of Sciences 114, no. 44 (2017): E9356—E9365. http://dx.doi.org/10.1073/pnas.1711310114.

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During malaria blood-stage infections,Plasmodiumparasites interact with the RBC surface to enable invasion followed by intracellular proliferation. Critical factors involved in invasion have been identified using biochemical and genetic approaches including specific knockdowns of genes of interest from primary CD34+hematopoietic stem cells (cRBCs). Here we report the development of a robust in vitro culture system to produce RBCs that allow the generation of gene knockouts via CRISPR/Cas9 using the immortal JK-1 erythroleukemia line. JK-1 cells spontaneously differentiate, generating cells at

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