Log in

Relevant bibliographies by topics / CD62 / Journal articles

To see the other types of publications on this topic, follow the link: CD62.

Journal articles on the topic 'CD62'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'CD62.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Pelikan, Zdenek. "Expression of Surface Markers on the Blood Cells during the Delayed Asthmatic Response to Allergen Challenge." Allergy & Rhinology 5, no. 2 (2014): ar.2014.5.0087. http://dx.doi.org/10.2500/ar.2014.5.0087.

Full text

Abstract:

Patients with bronchial asthma develop various types of asthmatic response to bronchial challenge with allergen, such as immediate/early asthmatic response (IAR), late asthmatic response (LAR) or delayed asthmatic response (DYAR), because of different immunologic mechanisms. The DYAR, occurring between 24 and 56 hours after the bronchial allergen challenge (p < 0.01), differs from IAR and LAR in clinical as well as immunologic features. This study investigates the expression of CD molecules (markers) on the surface of particular cell populations in the peripheral blood and their changes dur

APA, Harvard, Vancouver, ISO, and other styles

2

Metzelaar, Marcel J., Henk-Jan Schuurman, Harry F. G. Heijnen, Jan J. Sixma, and H. Karel Nieuwenhuis. "Biochemical and immunohistochemical characteristics of CD62 and CD63 monoclonal antibodies." Virchows Archiv B Cell Pathology Including Molecular Pathology 61, no. 1 (1992): 269–77. http://dx.doi.org/10.1007/bf02890428.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Kehrel, Beate, Sonja Wierwille, Kenneth J. Clemetson, et al. "Glycoprotein VI Is a Major Collagen Receptor for Platelet Activation: It Recognizes the Platelet-Activating Quaternary Structure of Collagen, Whereas CD36, Glycoprotein IIb/IIIa, and von Willebrand Factor Do Not." Blood 91, no. 2 (1998): 491–99. http://dx.doi.org/10.1182/blood.v91.2.491.

Full text

Abstract:

Abstract Simple collagen-related peptides (CRPs) containing a repeat Gly-Pro-Hyp sequence are highly potent platelet agonists. Like collagen, they must exhibit tertiary (triple-helical) and quaternary (polymeric) structure to activate platelets. Platelet signaling events induced by the peptides are the same as most of those induced by collagen. The peptides do not recognize the α2β1 integrin. To identify the signaling receptor involved, we have evaluated the response to the CRP, Gly-Lys-Hyp(Gly-Pro-Hyp)10-Gly-Lys-Hyp-Gly of platelets with defined functional deficiencies. These studies exclude

APA, Harvard, Vancouver, ISO, and other styles

4

Tan, Kiat, Muzahir Tayebjee, Indran Davagnanam, Mark Moss, Gregory Lip, and Andrew Blann. "Soluble CD40L in peripheral artery disease." Thrombosis and Haemostasis 93, no. 03 (2005): 578–83. http://dx.doi.org/10.1160/th04-09-0586.

Full text

Abstract:

SummaryAlthough soluble CD40L (sCD40L, possibly derived from platelets and pro-inflammatory in vitro) may be implicated in thrombosis and haemostasis, there are little data in peripheral artery disease (PAD). We hypothesised the following: (a) that sCD40L relates to the clinical severity of PAD; and (b) that peripheral artery angioplasty acutely raises sCD40L levels. sCD40L was compared to established platelet markers soluble P selectin, platelet microparticles and platelet surface expression of CD62 and CD63. We recruited 36 healthy controls, 33 patients with intermittent claudication (IC), a

APA, Harvard, Vancouver, ISO, and other styles

5

Ritesh, P., S. Pahuja, J. Chavez, et al. "Correlation of surface expression of CD11b or CD32 in polymorphonuclear cells (PMNs) and CD69 in natural killer cells (NK) with progression-free survival (PFS) following chemoimmunotherapy with rituximab and liposomal doxorubicin (LD) in patients (pts) with relapsed or refractory B-cell lymphoma." Journal of Clinical Oncology 27, no. 15_suppl (2009): 8583. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.8583.

Full text

Abstract:

8583 Antibody dependent cellular citotoxicity (ADCC) play a significant role in rituximab's anti-tumor activity. FcγRIIIa polymorphisms have been associated with clinical responses to rituximab. The predictive value of FcγRIIIa polymorphisms is lost when rituximab is combined with chemotherapy. Alternative assays to assess the immune system had not been studied in patients treated with rituximab plus chemotherapy. To this end, we prospectively studied the pre-treatment quality and function of PMNs and NK cells from pts with refractory/relapsed B-cell lymphomas in a Phase I/II trial. Forty-two

APA, Harvard, Vancouver, ISO, and other styles

6

Nesterova, I. V., V. V. Malinovskaya, S. V. Khaydukov, D. L. Nguyen Thi, G. A. Chudilova, and L. V. Lomtatidze. "DIFFERENTIATED EFFECTS OF GLUCOSAMINYLMURAMILDIPEPTIDE ON THE NONTRANSFORMED AND EXPERIMENTALLY TRANSFORMED PHENOTYPE OF CD62L+CD63+CD66d+ NEUTROPHILIC GRANULOCYTES IN CONVENTIONALLY HEALTHY PEOPLE." Medical Immunology (Russia) 20, no. 6 (2018): 847–54. http://dx.doi.org/10.15789/1563-0625-2018-6-847-854.

Full text

Abstract:

Modern studies have shown a high plasticity and phenotypic diversity of neutrophilic granulocytes (NG) provided by different receptors, which are diagnostic markers for the functional capacity of the cell in the course of their activities. We investigated NG from peripheral blood, obtained from healthy people of both sexes aged from 26 to 66 years. Evaluation of the neutrophil membrane receptor expression was carried out by flow cytometry. The relative amount of neutrophilic granulocytes expressing membrane CD62L, CD63, CD66d receptors and the intensity of their expression were determined acco

APA, Harvard, Vancouver, ISO, and other styles

7

Brignole-Baudouin, Françoise, Patrick Philip, Isabelle Sudaka, and Jacques Bayle. "HOW CD62-CD63 PLATELET-ACTIVATED ANTIGENS MAY HE EXPRESSED IN GLANZMANN'S THROMBASTEHNIA?" Biology of the Cell 79, no. 3 (1993): 288. http://dx.doi.org/10.1016/0248-4900(93)90218-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Saving, Kay L., and Peggy E. Mankin. "CD62 Expression During Thrombus Formation." Journal of Pediatric Hematology/Oncology 25, no. 3 (2003): 266–69. http://dx.doi.org/10.1097/00043426-200303000-00017.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Leytin, Valery, David J. Allen, Adam Gwozdz, Marie B. Garvey та John J. Freedman. "Role of P-Selectin and GPIbα in the Fast and Delayed Clearance of Transfused Platelets." Blood 104, № 11 (2004): 3625. http://dx.doi.org/10.1182/blood.v104.11.3625.3625.

Full text

Abstract:

Abstract Platelets become activated during preparation and storage of platelet concentrates (PCs) for transfusion. Flow cytometric assays of platelet activation can be employed for quantifying in vitro quality of PCs. It remains, however, unclear whether the level of in vitro platelet activation in stored PCs correlates with in vivo survival of the platelets after transfusion. Platelet surface glycoprotein (GP) Ibα and P-selectin (CD62) can be involved in regulation of posttransfusion PC clearance, mediating adhesive interactions of platelets with counter-receptors on leukocytes and endothelia

APA, Harvard, Vancouver, ISO, and other styles

10

Flaumenhaft, Robert, James R. Dilks, Sunita R. Patel, Giannoula Klement, and Joseph E. Italiano. "Megakaryocyte-Derived Microparticles: Mechanism of Formation and Circulation in Plasma." Blood 108, no. 11 (2006): 1507. http://dx.doi.org/10.1182/blood.v108.11.1507.1507.

Full text

Abstract:

Abstract Platelet microparticles are submicron membrane vesicles expressing platelet markers and are a normal constituent of circulating blood. Several studies have demonstrated positive correlations between thrombotic states and increased platelet microparticle levels. Yet the physiology of these microparticles is poorly understood. Specifically, the origin of circulating platelet microparticles is not known. Platelet microparticles are routinely formed in vitro following exposure of platelets to pharmacologic concentrations of platelet agonists such as the combination of thrombin and collage

APA, Harvard, Vancouver, ISO, and other styles

11

Stok, U., A. Shephard, S. Cucnik, S. Sodin-Šemrl, and P. Zigon. "AB0080 ALTERED CONCENTRATIONS OF DIFFERENT SMALL EXTRACELLULAR VESICLE POPULATIONS IN PLASMA OF PATIENTS WITH ANTIPHOSPHOLIPID SYNDROME." Annals of the Rheumatic Diseases 80, Suppl 1 (2021): 1069.3–1070. http://dx.doi.org/10.1136/annrheumdis-2021-eular.1371.

Full text

Abstract:

Background:Antiphospholipid syndrome (APS) is a systemic autoimmune disorder characterized by thrombosis, obstetric complications, and the presence of antiphospholipid antibodies (aPL) that cause endothelial injury and thrombophilia [1]. Extracellular vesicles (EVs) are involved in various thrombotic disorders [2], including APS [3, 4], and therefore may influence the prothrombotic status of APS patients. One of the hallmarks of activated endothelium is the expression of adhesion molecules, such as ICAM-1 (CD54) and E-selectin (CD62E), that play a key function in the interactions with leukocyt

APA, Harvard, Vancouver, ISO, and other styles

12

Karawajczyk, Malgorzata, Lena Douhan Håkansson, Miklos Lipcsey, et al. "High expression of neutrophil and monocyte CD64 with simultaneous lack of upregulation of adhesion receptors CD11b, CD162, CD15, CD65 on neutrophils in severe COVID-19." Therapeutic Advances in Infectious Disease 8 (January 2021): 204993612110340. http://dx.doi.org/10.1177/20499361211034065.

Full text

Abstract:

Background and Aims: The pronounced neutrophilia observed in patients with coronavirus disease 2019 (COVID-19) infections suggests a role for these leukocytes in the pathology of the disease. Monocyte and neutrophil expression of CD64 and CD11b have been reported as early biomarkers to detect infections. The aim of this study was to study the expression of receptors for IgG (CD64) and adhesion molecules (CD11b, CD15s, CD65, CD162, CD66b) on neutrophils and monocytes in patients with severe COVID-19 after admission to an intensive care unit (ICU). Methods: The expression of receptors was analyz

APA, Harvard, Vancouver, ISO, and other styles

13

Baudouin-Brignole, J. Bayle, A. Gog, F. "PMA induces platelet activation of specific antigens (CD62/CD63) in GpIIb-IIIa deficient platelets from Glanzmann's thrombasthenia." Platelets 8, no. 6 (1997): 391–96. http://dx.doi.org/10.1080/09537109777078.

Full text

APA, Harvard, Vancouver, ISO, and other styles

14

Cahill, M. R., M. G. Macey, and A. C. Newland. "Fixation with formaldehyde induces expression of activation dependent platelet membrane glycoproteins, P selectin (CD62) and GP53 (CD63)." British Journal of Haematology 84, no. 3 (1993): 527–29. http://dx.doi.org/10.1111/j.1365-2141.1993.tb03112.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Spangenberg, Peter, Helge Redlich, Iris Bergmann, Wolfgang Lösche, Matthias Götzrath, and Beate Kehrel. "The Platelet Glycoprotein IIb/IIIa Complex Is lnvolved in the Adhesion of Activated Platelets to Leukocytes." Thrombosis and Haemostasis 70, no. 03 (1993): 514–21. http://dx.doi.org/10.1055/s-0038-1649615.

Full text

Abstract:

SummaryThe adhesion of activated platelets to leukocytes (rosette formation) seems to be mediated by CD62 on platelets and its counterreceptor (CD 15 or a sialic acid-containing glycoprotein) on polymorphonuclear leukocytes (PMNL). However, neither treatment of platelets with an anti-CD62 antibody or fucoidan nor treatment of PMNL with anti-CD15 antibody or neuraminidase are able to inhibit completely the adhesion. Therefore, we have studied the platelet GPIIb/IIIa complex (CD41a) for its involvement in the adhesion of activated platelets to PMNL. The following evidences point to a participati

APA, Harvard, Vancouver, ISO, and other styles

16

Divers, SG, K. Kannan, RM Stewart, et al. "Quantitation of CD62, soluble CD62, and lysosome-associated membrane proteins 1 and 2 for evaluation of the quality of stored platelet concentrates." Transfusion 35, no. 4 (1995): 292–97. http://dx.doi.org/10.1046/j.1537-2995.1995.35495216076.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Ribeiro, Daniel, Stephanie Laufs, Eike B. Buss, W. Jens Zeller, Anthony D. Ho, and Stefan Fruehauf. "Functional Activity of In Vivo Primed Granulocytes: A Comparative Study." Blood 104, no. 11 (2004): 3818. http://dx.doi.org/10.1182/blood.v104.11.3818.3818.

Full text

Abstract:

Abstract Recombinant granulocyte colony-stimulating factor (G-CSF) has been widely used in the treatment of chemotherapy-induced neutropenia as well as in mobilization of peripheral blood stem cells in context with autologous bone marrow transplantation. Recombinant G-CSF expressed in E.coli (Filgrastim) and G-CSF expressed in CHO-cells (Lenograstim), are in clinical use. Here we study the effects of the different G-CSF on functional activity of granulocytes including chemotaxis, oxidative burst and antigen expression. Granulocytes were obtained from patients with hematological malignancies be

APA, Harvard, Vancouver, ISO, and other styles

18

Xu, Heping, Ayyakkannu Manivannan, Isabel Crane, Rosemary Dawson, and Janet Liversidge. "Critical but divergent roles for CD62L and CD44 in directing blood monocyte trafficking in vivo during inflammation." Blood 112, no. 4 (2008): 1166–74. http://dx.doi.org/10.1182/blood-2007-06-098327.

Full text

Abstract:

Abstract Using noninvasive in vivo imaging and experimental autoimmune uveoretinitis as a model, we show for the first time that the mechanisms controlling blood monocyte recirculation through peripheral and lymphoid tissues alter during inflammation. The recirculation of monocytes in mice with ocular inflammation but not controls was found to depend on the selectin CD62-ligand (CD62L) and on CD44. Not only was rolling efficiency ablated or markedly reduced in antibody-treated mice, but most of the labeled monocytes also disappeared from the circulation within seconds, anti-CD44–treated monocy

APA, Harvard, Vancouver, ISO, and other styles

19

Kalmarova, K., E. Kurca, V. Nosal, et al. "Measurement of platelet p-selectin expression by flow cytometry in patients with acute ischemic stroke." Acta Medica Martiniana 18, no. 1 (2018): 14–20. http://dx.doi.org/10.2478/acm-2018-0002.

Full text

Abstract:

AbstractAims: The aim of this study was to asses the platelet activation in the acute phase of ischemic stroke and transient ischemic attack (TIA) by defining p-selectin (CD62) expression by flow cytometry in vivo – without stimulation with agonists. We also studied whether antiplatelet therapy supresses the levels of baseline p-selectin expression and verified if there is a correlation between platelet CD62 expression and the type of ischemic stroke.Methods: We determined the expression of platelet surface p-selectin using whole-blood flow cytometry within the first 48-hours after onset of ce

APA, Harvard, Vancouver, ISO, and other styles

20

Aruffo, Alejandro, Waldemar Kolanus, Gerd Walz, Pam Fredman, and Brian Seed. "CD62/P-selectin recognition of myeloid and tumor cell sulfatides." Cell 67, no. 1 (1991): 35–44. http://dx.doi.org/10.1016/0092-8674(91)90570-o.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

Meguri, Yusuke, Takeru Asano, Takanori Yoshioka, et al. "Host Immune Status Determines the Effects of Therapeutic Interleukin-2 Administration: Enhancement of GVL or Induction of Tolerance?" Blood 124, no. 21 (2014): 2431. http://dx.doi.org/10.1182/blood.v124.21.2431.2431.

Full text

Abstract:

Abstract Interleukin-2 (IL-2) has a central role in immune tolerance thorough maintaining the homeostasis of CD4+CD25+Foxp3+ regulatory T cell (Treg). We recently reported that administration of low-dose IL-2 could preferentially enhance Treg in vivo and suppress clinical manifestations of chronic GVHD after allogeneic hematopoietic stem cell transplantation (NEJM2011). On the other hand, IL-2 is also necessary for the development of cytotoxic T cell function and has been used for the systemic immune therapy to amplify anti-tumor immunity. Thus, IL-2 administration after transplantation can in

APA, Harvard, Vancouver, ISO, and other styles

22

Janowska-Wieczorek, Anna, Marcin Majka, Jacek Kijowski, et al. "Platelet-derived microparticles bind to hematopoietic stem/progenitor cells and enhance their engraftment." Blood 98, no. 10 (2001): 3143–49. http://dx.doi.org/10.1182/blood.v98.10.3143.h8003143_3143_3149.

Full text

Abstract:

Because human CD34+ and murine Sca-1+hematopoietic stem–progenitor cells (HSPCs) express platelet-binding sialomucin P-selectin (CD162) and integrin Mac-1 (CD11b–CD18) antigen, it was inferred that these cells might interact with platelets. As a result of this interaction, microparticles derived from platelets (PMPs) may transfer many platelet antigens (CD41, CD61, CD62, CXCR4, PAR-1) to the surfaces of HSPCs. To determine the biologic significance of the presence of PMPs on human CD34+ and murine Sca-1+ cells, their expressions on mobilized peripheral blood (mPB) and on nonmobilized PB- and b

APA, Harvard, Vancouver, ISO, and other styles

23

Remon, J., N. Abedallaa, E. Taranchon-Clermont, et al. "CD52, CD22, CD26, EG5 and IGF-1R expression in thymic malignancies." Lung Cancer 108 (June 2017): 168–72. http://dx.doi.org/10.1016/j.lungcan.2017.03.019.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Berens, Christina, Jens Müller, Heiko Rühl, Johannes Oldenburg, and Bernd Pötzsch. "A Novel Method for Flow Cytometric Quantification of Human Platelet Membrane Glycoproteins in Citrated Whole Blood." Blood 134, Supplement_1 (2019): 2367. http://dx.doi.org/10.1182/blood-2019-124646.

Full text

Abstract:

Introduction: Diagnosis of platelet receptor defects can be confirmed by flow cytometry using a panel of monoclonal antibodies. The conventional method utilizing platelet rich plasma (PRP) has many limitations such as time-consuming preparation, requirement of a large sample volume or limitations in samples from patients with thrombocytopenia or abnormally large platelets. In order to overcome these difficulties a novel method was developed using a second antibody directed against an erythrocyte protein which allows differentiation between platelets and erythrocytes in citrated whole blood (CW

APA, Harvard, Vancouver, ISO, and other styles

25

Bierling, Philippe, Ali Bettaieb, Patricia Fromont, Marina Favrin and, and Najib Duedari. "Anti-GMP140 (CD62) autoantibody in a patient with autoimmune thrombocytopenic purpura." British Journal of Haematology 87, no. 3 (1994): 631–33. http://dx.doi.org/10.1111/j.1365-2141.1994.tb08327.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Pedigo, M., T. Wun, and T. Paglieroni. "Removal by white cell-reduction filters of activated platelets expressing CD62." Transfusion 33, no. 11 (1993): 930–35. http://dx.doi.org/10.1046/j.1537-2995.1993.331194082385.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

AMMAR, TAMESHWAR, and Barry Tabakin. "THE EFFECTS OF AMRINONE AND MILRINONE ON THE EXPRESSION OF CD62." Anesthesia & Analgesia 80, Supplement (1995): SCA22. http://dx.doi.org/10.1213/00000539-199504001-00022.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Janowska-Wieczorek, Anna, Marcin Majka, Jacek Kijowski, et al. "Platelet-derived microparticles bind to hematopoietic stem/progenitor cells and enhance their engraftment." Blood 98, no. 10 (2001): 3143–49. http://dx.doi.org/10.1182/blood.v98.10.3143.

Full text

Abstract:

Abstract Because human CD34+ and murine Sca-1+hematopoietic stem–progenitor cells (HSPCs) express platelet-binding sialomucin P-selectin (CD162) and integrin Mac-1 (CD11b–CD18) antigen, it was inferred that these cells might interact with platelets. As a result of this interaction, microparticles derived from platelets (PMPs) may transfer many platelet antigens (CD41, CD61, CD62, CXCR4, PAR-1) to the surfaces of HSPCs. To determine the biologic significance of the presence of PMPs on human CD34+ and murine Sca-1+ cells, their expressions on mobilized peripheral blood (mPB) and on nonmobilized

APA, Harvard, Vancouver, ISO, and other styles

29

Bajnok, Anna, Maria Ivanova, János Rigó, and Gergely Toldi. "The Distribution of Activation Markers and Selectins on Peripheral T Lymphocytes in Preeclampsia." Mediators of Inflammation 2017 (2017): 1–7. http://dx.doi.org/10.1155/2017/8045161.

Full text

Abstract:

Introduction. Impaired maternal immune tolerance resulting in systemic inflammation plays a pivotal role in the pathogenesis of preeclampsia. Phenotypical changes of monocytes and neutrophil granulocytes have already been studied in preeclampsia, and some studies also included T lymphocyte activation markers; however, the results are controversial and a comprehensive analysis of activation markers is lacking. The characteristics of cellular adhesion molecules in preeclampsia are yet to be described.Material and Methods. Peripheral blood samples of 18 preeclamptic patients and 20 healthy pregna

APA, Harvard, Vancouver, ISO, and other styles

30

Callan, M. F. C., L. Tan, N. Annels, et al. "Direct Visualization of Antigen-specific CD8+T Cells during the Primary Immune Response to Epstein-Barr Virus In Vivo." Journal of Experimental Medicine 187, no. 9 (1998): 1395–402. http://dx.doi.org/10.1084/jem.187.9.1395.

Full text

Abstract:

Primary infection with virus can stimulate a vigorous cytotoxic T cell response. The magnitude of the antigen-specific component versus the bystander component of a primary T cell response remains controversial. In this study, we have used tetrameric major histocompatibility complex–peptide complexes to directly visualize antigen-specific cluster of differentration (CD)8+ T cells during the primary immune response to Epstein-Barr virus (EBV) infection in humans. We show that massive expansion of activated, antigen-specific T cells occurs during the primary response to this virus. In one indivi

APA, Harvard, Vancouver, ISO, and other styles

31

Rødland, Ernst Kristian, Thor Ueland, Turid M. Pedersen, et al. "Activation of Platelets by Aspergillus fumigatus and Potential Role of Platelets in the Immunopathogenesis of Aspergillosis." Infection and Immunity 78, no. 3 (2009): 1269–75. http://dx.doi.org/10.1128/iai.01091-09.

Full text

Abstract:

ABSTRACT Aspergillus fumigatus is the most frequent cause of invasive mold infections worldwide. Platelets contribute to inflammation and promote thrombosis, characteristically seen in aspergillosis, and might be involved both in antifungal defense and in the histopathological process. In the experiments reported here, in vitro activation of platelets by conidia, swollen conidia, and hyphae from A. fumigatus was assessed by flow cytometry and enzyme immunoassays. THP-1 monocytes and human monocytes with and without platelets were cultured with hyphae from A. fumigatus, and the release of inter

APA, Harvard, Vancouver, ISO, and other styles

32

Xianghong, G., C. Guanping, Y. Fenghua, and W. Jiayin. "Changes in platelet functional parameters and CD62 P expression in liver cirrhosis." African Health Sciences 13, no. 4 (2014): 1079. http://dx.doi.org/10.4314/ahs.v13i4.31.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Bajorath, Juergen, Diane Hollenbaugh, Gordon King, et al. "CD62/P-Selectin Binding Sites for Myeloid Cells and Sulfatides Are Overlapping." Biochemistry 33, no. 6 (1994): 1332–39. http://dx.doi.org/10.1021/bi00172a007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Simon, M., S. Hluchý, Ľ. Horovská, J. Antalíková, and J. Čuboň. "Immunohistochemical localization of adhesion molecules (CD62 and CD18) in the mammary gland of dairy cows." Czech Journal of Animal Science 52, No. 4 (2008): 88–95. http://dx.doi.org/10.17221/2272-cjas.

Full text

Abstract:

Localization of the L-selectin (CD62L) and &beta;2-integrin (CD18) bearing cells in different tissues of the bovine mammary gland was examined. Five dairy cows of Holstein-Friesian breed in the middle of their second and third lactation cycle were used in the study. Blood, milk and udder tissue samples were collected from each cow to estimate the milk somatic cell count (SCC) and bacteriological infection of the mammary gland. The expression of CD62L and CD18 on blood cells, milk cells and parenchymal tissues of udder, F&uuml;rstenberg&rsquo;s rosette and the transverse section of

APA, Harvard, Vancouver, ISO, and other styles

35

Corinaldesi, Giorgio. "Platelet Activation in Cardiovascular Disease." Blood 118, no. 21 (2011): 5242. http://dx.doi.org/10.1182/blood.v118.21.5242.5242.

Full text

Abstract:

Abstract 5242 Platelets play a pivotal role in the pathogenesis of coronary artery disease (CAD). Among a number of platelet integrin receptors ITGA2B and ITGB3 appear to be fundamental mediators of platelet aggregation as they interact with fibrinogen, VWF, fibronectin, vitronectin, and thrombospondin; for these major properties these two receptors are risk-determining factors for increased platelet thrombogenicity, enhancement of leukocytes trafficking and activation by CD11a/CD18 (LFA-1) and CD11b/CD18 (MAC-1), thus leading to CAD. Receptor clustering promote cell adhesion by increasing the

APA, Harvard, Vancouver, ISO, and other styles

36

Kipnis, Andre, Scott Irwin, Angelo A. Izzo, Randall J. Basaraba, and Ian M. Orme. "Memory T Lymphocytes Generated by Mycobacterium bovis BCG Vaccination Reside within a CD4 CD44lo CD62 Ligandhi Population." Infection and Immunity 73, no. 11 (2005): 7759–64. http://dx.doi.org/10.1128/iai.73.11.7759-7764.2005.

Full text

Abstract:

ABSTRACT In the lungs of mice vaccinated with Mycobacterium bovis BCG, there was an accumulation of CD4 cells expressing the activated effector phenotype CD44hi CD62 ligandlo (CD62Llo) which were capable of secreting gamma interferon. Upon cell transfer, however, cells expressing a resting/naïve phenotype (CD44lo CD62Lhi) were capable of protecting the recipients from a virulent challenge infection, suggesting the emergence of T-cell memory from within this subset.

APA, Harvard, Vancouver, ISO, and other styles

37

Hujacova, Andrea, Jan Sirc, Kristyna Pekarkova, et al. "Large Platelet and Endothelial Extracellular Vesicles in Cord Blood of Preterm Newborns: Correlation with the Presence of Hemolysis." Diagnostics 11, no. 8 (2021): 1316. http://dx.doi.org/10.3390/diagnostics11081316.

Full text

Abstract:

Different biomarkers are investigated to detect the causes of severe complications in preterm infants. Extracellular vesicles (EVs) are recognized as an important part of cell-to-cell communication, and their increased levels were reported in numerous pathological states. We aimed to increase our knowledge about the incidence of platelet and endothelial EVs in cord blood of preterm newborns using conventional flow cytometry. The presence of platelet (CD36+CD41+), activated platelet (CD41+CD62+), and endothelial (CD31+CD105+) EVs was analyzed. Immune electron microscopy was used to confirm the

APA, Harvard, Vancouver, ISO, and other styles

38

Girtovitis, Fotios I. F., Vasilis Papagiannis, Ioannis Klonizakis, et al. "The Expression of Platelet Membrane Glycoproteins in Patients with Myelodysplastic Syndrome (MDS)." Blood 104, no. 11 (2004): 4730. http://dx.doi.org/10.1182/blood.v104.11.4730.4730.

Full text

Abstract:

Abstract The receptors of platelet membrane have a significant function. Most of them are glycoproteins and play a major role in the adhesion and aggregation of platelets, in a way that any change of their quantity or quality will lead to functional defects of the second. Aim of this study is to determine the qualitative expression of platelet membrane glycoproteins among patients (pts) with MDS, as a reflection of the various defects that characterize those syndromes. Materials and methods: 24 pts were studied, 15 male and 9 female with mean age 73.2 ± 13,4 years old, who suffered from all of

APA, Harvard, Vancouver, ISO, and other styles

39

Hollenbaugh, Diane, Jurgen Bajorath, Ronald Stenkamp, and Alejandro Aruffo. "Interaction of P-selectin (CD62) and its cellular ligand: Analysis of critical residues." Biochemistry 32, no. 12 (1993): 2960–66. http://dx.doi.org/10.1021/bi00063a006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Fu, Shuang, Adam C. Yopp, Xia Mao, et al. "CD4+ CD25+ CD62+ T-Regulatory Cell Subset Has Optimal Suppressive and Proliferative Potential." American Journal of Transplantation 4, no. 1 (2004): 65–78. http://dx.doi.org/10.1046/j.1600-6143.2003.00293.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Moore, K. L., N. L. Stults, S. Diaz, et al. "Identification of a specific glycoprotein ligand for P-selectin (CD62) on myeloid cells." Journal of Cell Biology 118, no. 2 (1992): 445–56. http://dx.doi.org/10.1083/jcb.118.2.445.

Full text

Abstract:

P-selectin (CD62, GMP-140, PADGEM), a Ca(2+)-dependent lectin on activated platelets and endothelium, functions as a receptor for myeloid cells by interacting with sialylated, fucosylated lactosaminoglycans. P-selectin binds to a limited number of protease-sensitive sites on myeloid cells, but the protein(s) that carry the glycans recognized by P-selectin are unknown. Blotting of neutrophil or HL-60 cell membrane extracts with [125I]P-selectin and affinity chromatography of [3H]glucosamine-labeled HL-60 cell extracts were used to identify P-selectin ligands. A major ligand was identified with

APA, Harvard, Vancouver, ISO, and other styles

42

Kirchmaier, C. M., Dagmar Westrup, J. Graff, et al. "Ex vivo/In vitro Interaction between ASA, Clopidogrel and GPIIb/IIIa-Inhibitors." Hämostaseologie 19, no. 03 (1999): 112–14. http://dx.doi.org/10.1055/s-0038-1660392.

Full text

Abstract:

SummaryWe report on an in vivo interaction study between aspirin (ASA) and Clopidogrel in healthy male volunteers and on an in-vitro interaction of abciximab and the peptidomimetic GPIIb/IIIa-inhibitor SR121566A with blood from subjects of the in vivo-study. Ten healthy male volunteers were randomly assigned to two groups (N = 5). Group 1 started with ASA and group 2 with Clopidogrel. From day 4 to 8 subjects of both groups received combined treatment with ASA and Clopidogrel. Blood from volunteers was spiked with abciximab or SR121566A. Inhibitory effects of ASA and Clopidogrel on collagen- o

APA, Harvard, Vancouver, ISO, and other styles

43

Powell, Daniel J., Mark E. Dudley, Paul F. Robbins, and Steven A. Rosenberg. "Transition of late-stage effector T cells to CD27+ CD28+ tumor-reactive effector memory T cells in humans after adoptive cell transfer therapy." Blood 105, no. 1 (2005): 241–50. http://dx.doi.org/10.1182/blood-2004-06-2482.

Full text

Abstract:

Abstract In humans, the pathways of memory T-cell differentiation remain poorly defined. Recently, adoptive cell transfer (ACT) of tumor-reactive T lymphocytes to metastatic melanoma patients after nonmyeloablative chemotherapy has resulted in persistence of functional, tumor-reactive lymphocytes, regression of disease, and induction of melanocyte-directed autoimmunity in some responding patients. In the current study, longitudinal phenotypic analysis was performed on melanoma antigen-specific CD8+ T cells during their transition from in vitro cultured effector cells to long-term persistent me

APA, Harvard, Vancouver, ISO, and other styles

44

Coito, A. J., G. D. Shaw, L. Meng, et al. "CD62–PSGL-1 interactions regulate cytokine chemokine and apoptotic networks in cardiac allograft recipients." Transplantation Proceedings 34, no. 5 (2002): 1463–64. http://dx.doi.org/10.1016/s0041-1345(02)02931-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Graff, J., D. Andries, M. Elsner, et al. "Platelet CD62 expression and PDGFAB secretion in patients undergoing PTCA and treatment with abciximab." British Journal of Clinical Pharmacology 51, no. 6 (2001): 577–82. http://dx.doi.org/10.1046/j.1365-2125.2001.01392.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Komsa-Penkova, Regina, Svetla J. Todinova, Tonya D. Andreeva, et al. "Alterations in Platelet Activity and Elastic Modulus of Healthy Subjects, Carriers of G20210A Polymorphism in the Prothrombin Gene." Journal of Biomedical and Clinical Research 9, no. 1 (2016): 72–79. http://dx.doi.org/10.1515/jbcr-2016-0011.

Full text

Abstract:

Summary Platelet activation is a complex process in which platelet reorganization takes place associated with changes in the cell shape, topology, membrane elasticity and microparticle production. The aim of this study was to investigate the changes/aberrations in the platelet activity, elasticity and morphology in healthy subjects, carriers of A allele of prothrombin G20210A polymorphism. Blood samples from 18 healthy subjects were used for platelet analysis by force-mode atomic force microscopy. Restriction analysis was used to investigate the carriage of G20210A polymorphism in the prothrom

APA, Harvard, Vancouver, ISO, and other styles

47

Reeves, R. Keith, Jacqueline Gillis, Fay E. Wong, Yi Yu, Michelle Connole, and R. Paul Johnson. "CD16− natural killer cells: enrichment in mucosal and secondary lymphoid tissues and altered function during chronic SIV infection." Blood 115, no. 22 (2010): 4439–46. http://dx.doi.org/10.1182/blood-2010-01-265595.

Full text

Abstract:

Abstract Natural killer (NK) cells contribute to control of HIV/SIV infection. We defined macaque NK-cell subsets based on expression of CD56 and CD16 and found their distribution to be highly disparate. CD16+ NK cells predominated in peripheral blood, whereas most mucosal NK cells were CD56+, and lymph nodes contained both CD56+ and CD16−CD56− (double-negative [DN]) subsets. Functional profiles were also distinct among subsets—CD16+ NK cells expressed high levels of cytolytic molecules, and CD56+ NK cells were predominantly cytokine-secreting cells, whereas DN NK possessed both functions. In

APA, Harvard, Vancouver, ISO, and other styles

48

Morgan, Doris A., Monika Jost, Patrick Bender, Kelly Mayo, and Andres Kriete. "Microarray Analysis of Thrombopoietin Induced Lineage Commitment of Bi-Potential Cell Line HU-3." Blood 108, no. 11 (2006): 4198. http://dx.doi.org/10.1182/blood.v108.11.4198.4198.

Full text

Abstract:

Abstract HU-3 is a cytokine dependent cell line derived from a patient with megakaryoblastic leukemia. The cells have a strong megakaryocytic phenotype when cultured in GM-CSF and undergo terminal erythropoiesis when switched to stem cell factor and erythropoietin. (Morgan, 1997). HU-3 cells are equally responsive to thrombopoietin (TPO), but no longer can be induced into the erythroid pathway nor do they advance along the megakaryocyte differentiation pathway. Microarray analysis of the bi-potential and unipotential cells using arrays with genome-wide coverage and bioinformatics software iden

APA, Harvard, Vancouver, ISO, and other styles

49

Boudjeltia, Karim Zouaoui, Patrick Durez, Didier Oberweis, et al. "Effects of raloxifene treatment on the phenotype of blood monocytes." Canadian Journal of Physiology and Pharmacology 88, no. 5 (2010): 601–5. http://dx.doi.org/10.1139/y10-002.

Full text

Abstract:

Raloxifene (RLX), a selective oestrogen receptor modulator, has oestrogen-agonist effects on bone, lipoproteins, and homocysteine and oestrogen-antagonist activity in the breast and uterus, positioning it as a potential drug for long-term prevention of coronary heart disease in postmenopausal women. To further evaluate its influence on cardiovascular risk factors, we studied the effects of 60 mg/day RLX on serum lipid levels, inflammatory (high-sensitivity C-reactive protein, and coagulation (fibrinogen) markers, monocytes, and fibrinolysis in 15 healthy postmenopausal women. Markers were meas

APA, Harvard, Vancouver, ISO, and other styles

50

Lopez-Vilchez, Irene, Maribel Diaz-Ricart, Berta Fuste, Ana Galan, James White, and Gines Escolar. "Tissue factor-enriched vesicles are taken up by platelets and induce platelet aggregation in the presence of factor VIIa." Thrombosis and Haemostasis 97, no. 02 (2007): 202–11. http://dx.doi.org/10.1160/th06-04-0216.

Full text

Abstract:

SummaryWe investigated the interactions of vesicles containing human tissue factor (TF) with platelets and evaluated responses induced by rFVIIa using standard aggregometry, ultrastructural and flow-cytometry techniques. Washed platelets were exposed to a preparation of placental human TF (pTF) or to a relipidated formulation of recombinant humanTF (rTF). Under stirring conditions, pTF induced reversible aggregation with platelets returning to their resting state after 5 minutes. This reversible response to pTF was partially inhibited by antibodies against CD62-P, but not by antithrombin agent

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!