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Relevant bibliographies by topics / CD8a+ Dendritic cells

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Journal articles

Academic literature on the topic 'CD8a+ Dendritic cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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Journal articles on the topic "CD8a+ Dendritic cells"

1

Zarnani, Amir-Hassan, Seyed-Mohammad Moazzeni, Fazel Shokri, Mojdeh Salehnia, and Mahmood Jeddi-Tehrani. "Kinetics of murine decidual dendritic cells." Reproduction 133, no. 1 (2007): 275–83. http://dx.doi.org/10.1530/rep.1.01232.

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Dendritic cells (DCs) are professional antigen presenting cells (APC) capable of induction of primary immune responses as well as immunologic tolerance. Myeloid and lymphoid subsets of murine DCs are able to shift cytokine responses of T cells toward Th2 and Th1 profiles respectively. Thus, DCs would be suitable candidates to mediate the balance of maternal immune responses to conception. We analyzed pregnancy-related variations in uterus and splenic DCs in a murine model. C57BL/6-mated Balb/c female mice with vaginal plugs were scarified at early, middle, and late pregnancy. Frozen sections o

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2

Jiang, Y., L. Ye, Y. Cui, et al. "Effects of Lactobacillus rhamnosus GG on the maturation and differentiation of dendritic cells in rotavirus-infected mice." Beneficial Microbes 8, no. 4 (2017): 645–56. http://dx.doi.org/10.3920/bm2016.0157.

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Rotavirus-related diarrhoea is considered one of the most important diseases in field animal production. In addition to the classic vaccine strategy, a number of studies have utilised probiotics, such as Lactobacillus rhamnosus GG (LGG), to prevent rotavirus-induced diarrhoea. Although it has been partially revealed that Toll-like receptors (TLRs) are involved in the LGG-mediated protection against rotavirus infection, the details of the underlying immunologic mechanisms remain largely unknown. In this study, three-to-four-week-old female BALB/c mice were divided into three groups and orally a

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3

Song, Joo Hye, Sun Young Chang, Bayasi Guleng, et al. "Tu1918 Intestinal Dendritic Cells Survey Circulatory Antigens Prior to Induction of Regulatory CD8a/ T Cells." Gastroenterology 142, no. 5 (2012): S—878. http://dx.doi.org/10.1016/s0016-5085(12)63405-2.

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4

Ma, Qing, Dan Li, Roza Nurieva, et al. "Reduced GVHD in C3-Deficient Mice Is Associated with a Decrease in Donor Th1/Th17 Polarization and Recipient Th1- Driven Dendritic Cell Activation." Blood 118, no. 21 (2011): 308. http://dx.doi.org/10.1182/blood.v118.21.308.308.

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Abstract Abstract 308 Graft-vs-host disease (GVHD) is an alloimmune response after allogeneic hematopoietic stem transplantation (HSCT) mediated by donor T cells against antigen presented by recipient dendritic cells. Our studies with a murine model of GVHD indicate that the complement system regulates the alloimmune response leading to acute GVHD. We used the disparity in MHC class I and II antigens between BALB/c (H-2d) as donors and either wild-type (WT) or complement deficient C57BL/6 (H-2b) as recipients. We found that mice deficient in the central component of the complement system (C3−/

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5

Broadley, S., A. Plaumann, A. Seidlmeier, et al. "Complement- and platelet-mediated shuttling of blood-borne bacteria to CD8a+ dendritic cells." Molecular Immunology 56, no. 3 (2013): 266. http://dx.doi.org/10.1016/j.molimm.2013.05.079.

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6

Broadley, Steven, Patricia Graef, Ann Plaumann, et al. "Complement- and platelet-mediated shuttling of blood-borne bacteria to CD8A+ dendritic cells." Immunobiology 217, no. 11 (2012): 1160. http://dx.doi.org/10.1016/j.imbio.2012.08.088.

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7

Chen, Xiufen, Dominick Fosco, Douglas E. Kline, and Justin Kline. "Calreticulin Promotes Immunity Against Acute Myeloid Leukemia Cells in Vivo." Blood 124, no. 21 (2014): 996. http://dx.doi.org/10.1182/blood.v124.21.996.996.

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Abstract Pre-apoptotic cancer cells release internalized calreticulin (CRT) to their surface prior to death, which acts as an ‘eat-me’ signal to local phagocytes. Chemotherapy and irradiation, which can induce immunogenic cell death through CRT translocation, can also result in local and/or systemic immune suppression in the host. To bypass the requirement of exposing the host to chemotherapy to induce translocation of CRT to the cell surface, murine acute myeloid leukemia (AML) cells (C1498), were engineered to constitutively express cell surface CRT (C1498.CRT). Vector control C1498 or C1498

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8

Yoshimura, Hideaki, Masaaki Hotta, Atsushi Satake, and Shosaku Nomura. "GM-CSF Therapy Expands Regulatory T Cells and Protects Against Chronic Graft Versus Host Disease." Blood 128, no. 22 (2016): 2151. http://dx.doi.org/10.1182/blood.v128.22.2151.2151.

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Abstract Regulatory T cells (Tregs) possess the ability to suppress chronic graft-versus-host disease (cGVHD). Hence, the in vivo expansion of Tregs can be used as therapy against cGvHD. In addition to IL-2, Tregs require TCR and costimulatory signals from antigen presenting cells such as dendritic cells (DCs) for their optimal proliferation. Both fms-like tyrosine kinase 3 ligand (FLT3L) and granulocyte-macrophage colony stimulation factor (GM-CSF) induce the development of DCs and promote the proliferation of Tregs in a DC-dependent manner. GM-CSF preferentially increases CD11c+CD8a- DCs, wh

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9

Misaki, K., A. Morinobu, J. Saegusa, K. Nishimura, and S. Kumagai. "AB0169 Trichostatin a induces CD8A positive tolerogenic dendritic cells and regulatory T cells in SKG mice, and ameliorates the severe arthritis." Annals of the Rheumatic Diseases 71, Suppl 3 (2013): 647.6–647. http://dx.doi.org/10.1136/annrheumdis-2012-eular.169.

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10

Shreedhar, Vijay K., Brian L. Kelsall, and Marian R. Neutra. "Cholera Toxin Induces Migration of Dendritic Cells from the Subepithelial Dome Region to T- and B-Cell Areas of Peyer's Patches." Infection and Immunity 71, no. 1 (2003): 504–9. http://dx.doi.org/10.1128/iai.71.1.504-509.2003.

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ABSTRACT Intestinal M cells deliver macromolecules, particles, and pathogens into the subepithelial dome (SED) region of Peyer's patch mucosa, an area rich in dendritic cells (DCs). We tested whether uptake of the mucosal adjuvant cholera toxin (CT) or live Salmonella bacteria can induce DC migration within Peyer's patches. Virus-sized, fluorescent polystyrene microparticles were efficiently transported by M cells and ingested by CD11c+, CD11b−, and CD8a− DCs in the SED region. DCs loaded with microparticles remained in the SED for up to 14 days. CT (but not the CT B subunit) and live attenuat

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