Relevant bibliographies by topics / Cel Hypoxia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cel Hypoxia'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 July 2025

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Journal articles on the topic "Cel Hypoxia"

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Guo, Zihan, Yunlan Ding, Mengmeng Wang, Qing Zhai, Jiyong Liu, and Qiong Du. "Comparing the Differences in Adverse Events among Chimeric Antigen Receptor T-Cell Therapies: A Real-World Pharmacovigilance Study." Pharmaceuticals 17, no. 8 (2024): 1025. http://dx.doi.org/10.3390/ph17081025.

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In this study, we compared the similarities and differences in adverse events (AEs) among CAR T-cell products through signal mining via the FDA Adverse Event Reporting System (FAERS) and identified unknown AEs to provide a reference for safe clinical medication. Data from the FAERS database spanning from the fourth quarter of 2017 to the first quarter of 2024 were extracted. Signals were identified using the reporting odds ratio (ROR) method and the Medicines and Healthcare Products Regulatory Agency (MHRA) method. A total of 11,386 AE reports related to six CAR T-cell products were selected.
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Locke, Frederick L., Justin Chou, Saran Vardhanabhuti, et al. "Association of pretreatment (preTx) tumor characteristics and clinical outcomes following second-line (2L) axicabtagene ciloleucel (axi-cel) versus standard of care (SOC) in patients (pts) with relapsed/refractory (R/R) large B-cell lymphoma (LBCL)." Journal of Clinical Oncology 40, no. 16_suppl (2022): 7565. http://dx.doi.org/10.1200/jco.2022.40.16_suppl.7565.

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7565 Background: The Phase 3 randomized ZUMA-7 trial in 2L R/R LBCL showed axi-cel superiority to SOC (salvage chemotherapy and HDT-ASCT) in event-free survival (EFS; hazard ratio [HR], 0.398; P<.0001; Locke et al. N Eng J Med. 2021). We report results of exploratory analyses of tumor characteristics, including preTx tumor burden (TB), tissue hypoxia-related lactate dehydrogenase (LDH) level, and tumor microenvironment (TME). Methods: TB was calculated as the sum of product diameters of ≤6 reference lesions (Locke et al. Blood Adv. 2020). Serum LDH was assessed. PreTx tumor samples were ass
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Locke, Frederick L., Justin Chou, Saran Vardhanabhuti, et al. "Association of pretreatment (preTx) tumor characteristics and clinical outcomes following second-line (2L) axicabtagene ciloleucel (axi-cel) versus standard of care (SOC) in patients (pts) with relapsed/refractory (R/R) large B-cell lymphoma (LBCL)." Journal of Clinical Oncology 40, no. 16_suppl (2022): 7565. http://dx.doi.org/10.1200/jco.2022.40.16_suppl.7565.

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7565 Background: The Phase 3 randomized ZUMA-7 trial in 2L R/R LBCL showed axi-cel superiority to SOC (salvage chemotherapy and HDT-ASCT) in event-free survival (EFS; hazard ratio [HR], 0.398; P<.0001; Locke et al. N Eng J Med. 2021). We report results of exploratory analyses of tumor characteristics, including preTx tumor burden (TB), tissue hypoxia-related lactate dehydrogenase (LDH) level, and tumor microenvironment (TME). Methods: TB was calculated as the sum of product diameters of ≤6 reference lesions (Locke et al. Blood Adv. 2020). Serum LDH was assessed. PreTx tumor samples were ass
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Ho, A. S., X. Huang, H. Cao, A. C. Koong, and Q. T. Le. "Detection of circulating hypoxia-regulated miR-210 in pancreatic adenocarcinoma patients." Journal of Clinical Oncology 27, no. 15_suppl (2009): 4624. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.4624.

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4624 Background: MicroRNAs (miRs) are small non-coding transcripts involved in many cellular mechanisms, including tumorigenesis. miR-210, in particular, has been shown to be induced by hypoxia, over-expressed in several different cancers, and correlated with adverse outcomes in breast cancer. Moreover, since pancreatic adenocarcinomas have been previously shown to be extremely hypoxic, we hypothesized that miR-210 may be elevated in the plasma of these patients compared to non-cancer controls. Here, we compared the circulating plasma levels of miR-210 in pancreatic cancer patients and control
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Douiev, Liza, Chaya Miller, Shmuel Ruppo, Hadar Benyamini, Bassam Abu-Libdeh та Ann Saada. "Upregulation of COX4-2 via HIF-1α in Mitochondrial COX4-1 Deficiency". Cells 10, № 2 (2021): 452. http://dx.doi.org/10.3390/cells10020452.

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Cytochrome-c-oxidase (COX) subunit 4 (COX4) plays important roles in the function, assembly and regulation of COX (mitochondrial respiratory complex 4), the terminal electron acceptor of the oxidative phosphorylation (OXPHOS) system. The principal COX4 isoform, COX4-1, is expressed in all tissues, whereas COX4-2 is mainly expressed in the lungs, or under hypoxia and other stress conditions. We have previously described a patient with a COX4-1 defect with a relatively mild presentation compared to other primary COX deficiencies, and hypothesized that this could be the result of a compensatory u
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Storti, Paola, Irma Airoldi, Marina Bolzoni та ін. "Hypoxia-Inducible Factor (HIF)-1α Is a Therapeutic Target in Myeloma-Induced Angiogenesis". Blood 118, № 21 (2011): 3927. http://dx.doi.org/10.1182/blood.v118.21.3927.3927.

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Abstract Abstract 3927 Hypoxia-inducible factor (HIF)-1α is a critical trigger and regulator of tumor associated angiogenesis. It has been previously reported that bone marrow (BM) microenvironment is hypoxic in multiple myeloma (MM) patients and that HIF-1α is overexpressed by CD138+ MM cells and modulates the transcriptional and pro-angiogenic profiles of MM cells. The potential role of HIF-1α as a therapeutic target in MM is under investigation. To deepen this issue, in this study we explored the effect of a stable HIF-1α inhibition in MM cells on cell proliferation, survival and on MM-indu
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Lee, Sohyeon, Yoonyoung Kim, and Eun Seong Lee. "Hypoxia-Responsive Azobenzene-Linked Hyaluronate Dot Particles for Photodynamic Tumor Therapy." Pharmaceutics 14, no. 5 (2022): 928. http://dx.doi.org/10.3390/pharmaceutics14050928.

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In this study, we developed ultra-small hyaluronate dot particles that selectively release phototoxic drugs into a hypoxic tumor microenvironment. Here, the water-soluble hyaluronate dot (dHA) was covalently conjugated with 4,4′-azodianiline (Azo, as a hypoxia-sensitive linker) and Ce6 (as a photodynamic antitumor agent), producing dHA particles with cleavable Azo bond and Ce6 (dHA-Azo-Ce6). Importantly, the inactive Ce6 (self-quenched state) in the dHA-Azo-Ce6 particles was switched to the active Ce6 (dequenched state) via the Azo linker (–N=N–) cleavage in a hypoxic environment. In vitro stu
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Upadhyay, Laxmi, Muhammad Shan Ul Abedin, Stephen Yu, et al. "Managing CAR T-Cell Toxicity: Impact of Steroid Prophylaxis on Toxicity and Outcomes." Blood 144, Supplement 1 (2024): 7246. https://doi.org/10.1182/blood-2024-210573.

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Introduction: Chimeric antigen receptor (CAR) T-cell therapy has shown promising effects in treating relapsed/refractory hematological malignancies, significantly improving outcomes. However, CAR T-cell therapy is associated with side effects like cytokine release syndrome (CRS) and immune effector cell-associated neurotoxicity syndrome (ICANS). The incidence of CRS ranges from 41% to 94%, and ICANS from 21% to 64%, with grade 3 or higher CRS ranging from 1% to 24%. Prophylactic use of steroids has shown promising results in decreasing the incidence of CRS and ICANS in the ZUMA-1 cohort 6, inc
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Gurevich Shapiro, Anna, Eitan Winter, Pascale Zwicky, et al. "Single Cell Analysis of Pre-Treatment Peripheral Immune Composition Can Predict CAR-T Outcomes in Patients with Diffuse Large B-Cell Lymphoma." Blood 144, Supplement 1 (2024): 4788. https://doi.org/10.1182/blood-2024-203893.

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Background: Chimeric Antigen Receptor (CAR)-T cell therapy has revolutionized the treatment landscape for relapsed/refractory Diffuse Large B-cell Lymphoma (DLBCL). However, CAR-T failure remains a challenge, and an effective pre-treatment prognostic score is yet to be established. Aim: To gain a deeper understanding of the cellular and molecular determinants of response to CAR-T treatment by applying single cell multiomics, which will facilitate early response prediction, address resistance mechanisms, and refine patient selection criteria. This is particularly crucial with the introduction o
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Wei, Guijie, Jianhua Chen, Ziqi Jing, et al. "Glucose transporter 1 (GLUT1)-targeting and hypoxia-activated mitochondria-specific chemo-thermal therapy via a glycosylated poly(amido amine)/celastrol (PAMAM/Cel) complex." Journal of Colloid and Interface Science 608 (February 2022): 1355–65. http://dx.doi.org/10.1016/j.jcis.2021.10.129.

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Dissertations / Theses on the topic "Cel Hypoxia"

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Ljungkvist, Anna. "Imaging the tumor microenvironment : the dynamics and modification of hypoxia." Doctoral thesis, Umeå : Univ, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-106.

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Lawrentschuk, Nathan Leo. "Hypoxia and angiogenesis in renal cell carcinoma." Connect to thesis, 2009. http://repository.unimelb.edu.au/10187/6790.

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Hypoxia is one of the hallmarks of cancer. It was first postulated to occur in solid tumours by Thomlinson and Gray in 1955.1 The presence of hypoxia has been demonstrated in different types of solid tumours.2 Intratumoral hypoxia is caused by the lack of functional blood vessels in proliferating tumour tissue, resulting in low intratumoral oxygen concentrations. If hypoxia is severe or prolonged, cell death occurs.3 Malignant cells can undergo genetic and adaptive changes that allow them to escape from dying of oxygen deprivation. These changes are associated with a more aggressive malignant
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Lester, Robin D. "Hypoxia activated cell signaling receptors in cancer." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p3297526.

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Thesis (Ph. D.)--University of California, San Diego, 2008.<br>Title from first page of PDF file (viewed April 28, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references (p. 114-134).
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Schioppa, Tiziana. "Effects of tumour hypoxia on cell migration." Thesis, Open University, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434200.

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Cell adaptation to hypoxia requires activation of transcriptional programs that coordinate expression of genes involved in oxygen delivery (via- angiogenesis) and metabolic adaptation (via glycolisis). During migration and invasion of normal and pathological tissues, cells may encounter different oxygen levels, due to poor or altered vascularization, and recent evidence has suggested that chemotaxis is a cell function which may be affected by oxygen availability. This thesis describes how oxygen avaibility is a determinant parameter in the setting of chemotactic responsiveness to Stromal-Deriv
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Ledaki, Ioanna I. "Heterogeneity of tumour response to hypoxia : carbonic anhydrase IX induction defines a subpopulation of hypoxic cells with stem cell properties and drug resistance." Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:757a8e79-b20d-496c-b69b-4d6a3b7b56e3.

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Carbonic anhydrase IX (CA9) is strongly induced by hypoxia and its overexpression is associated with poor therapeutic outcome in cancer. The function of CAIX is to catalyze the reversible hydration of CO2 to bicarbonate and a proton. This helps hypoxic tumours to maintain a more neutral intracellular pH (pH<sub>i</sub>) promoting survival, but produces a more acidic extracellular (pH<sub>e</sub>) which promotes invasion and metastasis. Recent evidence has expanded on the role of hypoxia and CAIX by relating them to stem cell niches. In this study, taking advantage of the transmembrane location
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Nilsson, Ingrid. "Hypoxia, PDGF and VEGF in Vascular Development." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6894.

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Bedessem, Baptiste. "Contributions à l'étude de la réponse moléculaire à l'hypoxie : Modélisation mathématique et expérimentations sur cellules FUCCI." Thesis, Université Grenoble Alpes (ComUE), 2015. http://www.theses.fr/2015GREAS024/document.

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Les effets biologiques de l'hypoxie sont très étudiés aujourd'hui, principalement en raison du rôle crucial que jouent les conditions d'oxygénation dans le développement des cancers.Depuis plusieurs années, une littérature foisonnante tente ainsi de décrire les multiples aspects de la réponse moléculaire, cellulaire et physiologique à l'hypoxie. La complexité des voies de signalisation impliquées et la diversité de leurs effets cellulaires rendent la tâche délicate. Cet état de fait se reflète dans la pluralité des méthodes utilisées, depuis les simulations numériques jusqu'aux approches expér
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Lidgren, Anders. "Hypoxia inducible factor-1α in renal cell carcinoma". Doctoral thesis, Umeå universitet, Kirurgisk och perioperativ vetenskap, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1462.

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Hypoxia Inducible Factor-1α in Renal Cell Carcinoma Departments of Surgical and Perioperative Sciences, Urology and Andrology; Radiation Sciences, Oncology; Medical Biosciences, Pathology; and Medical Biosciences, Clinical Chemistry, Umeå University, Umeå, Sweden Background: Renal cell carcinoma (RCC) accounts for approximately 2-3% of all human cancers. A distinguished feature of RCC is vascularisation and among the three dominating RCC types conventional RCC (cRCC) generally is more vascularised than papillary RCC (pRCC) and chromophobe RCC (chRCC). Angiogenesis is a critical step in tumour
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Milani, Manuela. "Cell stress response and hypoxia in breast cancer." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:74d3bf91-9888-4e9e-b5e1-7d5d2d476174.

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During severe hypoxia (<0.01% oxygen) the protein folding machinery becomes dysfunctional, resulting in the accumulation of unfolded proteins with consequent endoplasmic reticulum (ER) stress and activation of the unfolded protein response (UPR) and autophagy, a process involved in the physiological turnover of cytoplasmic components. The link between the UPR and autophagy is not clearly defined. The aim of this thesis is to investigate the role of the induction of UPR under severe hypoxia in tumour survival and resistance to therapy. The results of this research suggest that the activating tr
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Ibegbu, Augustine. "The effects of hypoxia on neuronal cell signalling." Thesis, Queen Margaret University, 2009. https://eresearch.qmu.ac.uk/handle/20.500.12289/7367.

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Hypoxia adversely affects cells and tissues, and neuronal cells in particular have been shown to be more susceptible to the injurious effects of hypoxia i.e. they may begin to die when oxygen supply is reduced or completely eliminated. Cannabinoid (CB1) receptor and opioid (μ, δ and κ) receptor agonists have been shown to elicit several central nervous system (CNS) effects, mediated via G protein-coupled receptors. The aim of the research presented in this thesis was to study the effect of hypoxia on neuronal cell signalling and the consequent neuroprotectant effects of cannabinoid and opioid
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Books on the topic "Cel Hypoxia"

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Chiarotto, James Anthony. Hypoxia-induced upregulation of VEGF mRNA in cervical cancer cell lines. National Library of Canada, 1998.

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1947-, Haddad Gabriel G., and Lister George 1947-, eds. Tissue oxygen deprivation: From molecular to integrated function. M. Dekker, 1996.

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Sukhamay, Lahiri, Cherniack Neil S, Fitzgerald Robert S. 1931-, American Physiological Society (1887- ), and Federation of American Societies for Experimental Biology., eds. Response and adaptation to hypoxia: Organ to organelle. Published for the American Physiological Society by Oxford University Press, 1991.

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Wobma, Holly Michelle. Interferon-gamma/Hypoxia Primed Mesenchymal Stem Cells for an Improved Immunosuppressive Cell Therapy. [publisher not identified], 2018.

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Low, Eddy Wai-Mang. Reactions toward the synthesis of minor-groove binding hypoxic cell sensitizers. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Jamie, Goode, Chadwick Derek, Novartis Foundation, and Symposium on the Tumour Microenvironment: Causes and Consequences of Hypoxia and Acidity (2000 : London, England), eds. The tumour microenvironment: Causes and consequences of hypoxia and acidity. Wiley, 2001.

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Cheng, Phillip Ming-Da. Relationship between modulation of Glomus Cell K+ current and hypoxia transduction in the rat carotid body. s.n.], 1997.

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Jean-Marc, Pequignot, ed. Chemoreception: From cellular signaling to functional plasticity. Kluwer Academic/Plenum Publishers, 2003.

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Koumenis, Constantinos, Amato Giaccia, and Ester Hammond. Tumor Microenvironment and Cellular Stress: Signaling, Metabolism, Imaging, and Therapeutic Targets. Springer, 2013.

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Koumenis, Constantinos, Amato Giaccia, and Ester Hammond. Tumor Microenvironment and Cellular Stress: Signaling, Metabolism, Imaging, and Therapeutic Targets. Springer, 2016.

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Book chapters on the topic "Cel Hypoxia"

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Feron, Olivier. "Hypoxia in Head and Neck Cancer: Current Relevance." In Critical Issues in Head and Neck Oncology. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-84539-0_13.

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Abstract Hypoxia is a common characteristic of solid tumors, typically resulting from abnormal and inefficient tumor vasculature that fails to meet the high oxygen demands of rapidly dividing tumor cells. While healthy tissues generally maintain oxygen levels around 5%, most tumors exhibit median oxygen levels below 1%, a condition known to cause radioresistance. This observation has led to efforts to reverse tumor hypoxia or, alternatively, to exploit the hypoxic environment to ensure selective cytotoxicity of drugs, particularly in the context of head and neck squamous cell carcinoma (HNSCC)
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Bonny, Christophe. "Blocking Stress Signaling Pathways with Cell Permeable Peptides." In Hypoxia and Exercise. Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-34817-9_12.

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Ray, Suman Kumar, and Sukhes Mukherjee. "Hypoxia and Regulation of Cancer Cell Stemness." In Hypoxia and Tumor Microenvironment. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-1016-7_8.

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Schmierer, Bernhard. "Cell Cycle Signaling, Hypoxia." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_45.

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Pathak, Tanusha, Suman Kumar Ray, and Sukhes Mukherjee. "Metabolic Regulation of CAR-T Cell Function by Hypoxic Tumor." In Hypoxia and Tumor Microenvironment. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-1016-7_5.

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Harrison, George H., and Jeremy Wright. "Hypoxic Cell Radiosensitizers." In Cancer Management in Man. Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-2536-6_13.

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Coleman, C. Norman. "Hypoxic Cell Sensitizers." In Concomitant Continuous Infusion Chemotherapy and Radiation. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84186-6_8.

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West, John B. "Acclimatization and Adaptation: Organ to Cell." In Response and Adaptation to Hypoxia. Springer New York, 1991. http://dx.doi.org/10.1007/978-1-4614-7574-3_16.

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Mukherjee, Moubani, Bude Venkataswamy, Maynak Pal, and Mithun Roy. "Strategic Application of Bio-reductive Metalloprodrugs for Cancer Cell-Selective Anticancer Therapy." In Hypoxia and Tumor Microenvironment. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-1016-7_10.

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Masuda, Seiji, Sung-Kwon Moon, Taiho Kambe, Masaya Nagao, and Ryuzo Sasaki. "Hypoxia-Induced Production of Recombinant Erythropoietin Using Hypoxia-Response Enhancer." In Animal Cell Technology: Basic & Applied Aspects. Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-017-0728-2_25.

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Conference papers on the topic "Cel Hypoxia"

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Zhang, Chi, Karsten Mohn, Bin Dong, Shivam Mahapatra, and Seohee Ma. "Coherent Raman Scattering Spectroscopy and Microscopy for Biological Studies." In Optical Molecular Probes, Imaging and Drug Delivery. Optica Publishing Group, 2025. https://doi.org/10.1364/omp.2025.otu4e.2.

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Here, the pulse-picking method for nonlinear optical imaging modalities, including coherent anti-Stokes Raman scattering, is introduced. Furthermore, recent studies employing coherent Raman microscopy to investigate cancer cell metabolism under hypoxic conditions are discussed.
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Кечерюкова, Тахмина Мажитовна, Анна Александровна Шульга, and Анна Сергеевна Гончарова. "IN VIVO STUDY THE ACTION OF HYPOXIC CONDITIONS ON THE PROLIFERATIVE POTENTIAL OF LIVER CANCER CELLS." In Фундаментальные и прикладные исследования. Актуальные проблемы и достижения: сборник статей XXII всероссийской (национальной) научной конференции (Санкт-Петербург, Октябрь 2023). Crossref, 2023. http://dx.doi.org/10.37539/231004.2023.23.56.004.

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Гипоксия играет важную роль во многих биологических процессах, включая пролиферацию клеток. В этом исследовании были определены уровни экспрессии маркера пролиферации Ki-67 в тканях опухоли печени с гипоксическими условиями и без гипоксии. Hypoxia play critical roles in various biological processes, icluding cell proliferation. This study expression of the proliferation marker Ki-67 was measured in liver tumor tissues with hypoxic conditions and without hypoxia.
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Amorim, Ísis Salviano Soares de, Priscyanne Barreto Siqueira, Mariana Moreno de Sousa Rodrigues, and Andre Luiz Mencalha. "GENOMIC AND CLINICAL DATA ANALYSIS OF APE1 PROTEIN, BREAST CANCER STEM CELL PHENOTYPE, AND HYPOXIC TUMOR MICROENVIRONMENT." In Brazilian Breast Cancer Symposium 2022. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s2004.

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Introduction: Breast cancer (BC) is a heterogeneous disease at cellular and molecular levels. BC tumors present a cellular subpopulation of breast cancer stem cells (BCSCs) linked with tumor initiation and progression, recurrence, and therapeutic failure. The BCSC is preferentially found in hypoxic areas of the tumor, which are common features of BC and are significantly associated with worse prognosis. Although hypoxia activates an aggressive BCSC phenotype, the proteins that perform this molecular crossroad are still unknown. Therefore, finding proteins that performed this crossing would hel
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Das, Saikat. "Low dose radiation and chemotherapy significantly reduces hypoxic cell population in locally advanced cervix cancer-results of a phase II study." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685259.

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Introduction: Tumor hypoxia is one of the major causes of high incidence of treatment failures to chemoradiation which is the standard of care in locally advanced cervical cancer. The necessity of newer treatment options that can circumvent hypoxia is highly relevant in this group. Use of low dose radiation to enhance the efficacy of cell cycle specific chemotherapy by mechanism of chemopotentiation is one of the elegant approaches reported in the literature. We have already published the feasibility, efficacy and tolerance of low dose radiation and chemotherapy in neoadjuvant setting in cervi
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Oppegard, Shawn C., and David T. Eddington. "Modulation of Oxygen Tensions via Microfabricated Devices." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53093.

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Oxygen is a key modulator of many cellular pathways and plays an important role in a number of cellular behaviors. The hypoxic inducible factor 1α (HIF-1α) is often considered the master regulator of the cellular response to oxygen tension (1). HIF-1α is a transcription factor involved in angiogenesis, glucose transport and glycolysis, apoptosis, migration, and differentiation, among many other functions (2). Unfortunately devices permitting in vitro oxygen modulation fail to meet the needs of biomedical research due to the inability to effectively mimic conditions found in vivo. The gold stan
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Svirskaya, A. V., M. A. Yakauleva, D. B. Nizheharodava, and M. M. Zafranskaya. "EFFECT OF HYPOXIA ON IMMUNOMODULATORY PROPERTIES OF MULTIPOTENT MESENCHYMAL STROMAL CELLS." In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-94-97.

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This study characterizes the effect of hypoxia on human multipotent mesenchymal stromal cells ability to modulate mitogen-stimulated proliferation of peripheral blood mononuclear cells, what can be used for optimization of biomedical cell products protocols using for cell therapy of pathological conditions accompanied by hypoxia.
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Luigi, Varesio, Paolo Fardin, Rogier Versteeg, Fabiola Blengio, Annalisa Barla, and Maria Carla Bosco. "Abstract 2002: Cell reprogramming by hypoxia." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2002.

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Leahy, Rachel, Weiling Xu, Suzy A. A. Comhair, and Serpil C. Erzurum. "Hypoxia In Airway Epithelial Cell Differentiation." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5115.

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Gao, S., M. Emin, R. Shah, A. Jimenez, and S. Jelic. "Intermittent Hypoxia Disrupts Endothelial Cell Cholesterol Trafficking." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4702.

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te Boekhorst, Veronika, Liying Jiang, Steffi Lehmann, Alba Zuidema, and Peter Friedl. "Abstract 5062: Hypoxia-induced amoeboid cancer cell migration." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-5062.

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Reports on the topic "Cel Hypoxia"

1

Nielsen, T. B., and J. L. Kidwell. Cell Biology of Hypoxia, 1996. Defense Technical Information Center, 1996. http://dx.doi.org/10.21236/ada340589.

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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desatura
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Anderson, Donald M., Lorraine C. Backer, Keith Bouma-Gregson, et al. Harmful Algal Research & Response: A National Environmental Science Strategy (HARRNESS), 2024-2034. Woods Hole Oceanographic Institution, 2024. http://dx.doi.org/10.1575/1912/69773.

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Harmful and toxic algal blooms (HABs) are a well-established and severe threat to human health, economies, and marine and freshwater ecosystems on all coasts of the United States and its inland waters. HABs can comprise microalgae, cyanobacteria, and macroalgae (seaweeds). Their impacts, intensity, and geographic range have increased over past decades due to both human-induced and natural changes. In this report, HABs refers to both marine algal and freshwater cyanobacterial events. This Harmful Algal Research and Response: A National Environmental Science Strategy (HARRNESS) 2024-2034 plan bu
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