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Journal articles on the topic 'Celiaki'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Konieczny, Sebastian, Ewa Lange, and Jolanta Krusiec. "Wpływ diet eliminacyjnych na jakość życia osób z wybranymi chorobami autoimmunologicznymi." Kosmos 68, no. 2 (July 8, 2019): 215–26. http://dx.doi.org/10.36921/kos.2019_2541.

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Złożoność etiopatogenezy schorzeń autoimmunologicznych, w tym celiakii i choroby Hashimoto oraz brak skutecznej farmakoterapii powoduje, że istotnym czynnikiem poprawy stanu zdrowia i komfortu życia oraz zmniejszenia rozwoju powikłań może być odpowiednia dietoterapia. W badaniach własnych zaobserwowano, że jedna trzecia osób z celiakią oprócz wyłączenia zbóż glutenowych eliminowała z diety produkty mleczne. Większość osób z chorobą Hashimoto wyłączyła z diety zboża glutenowe, a ponad połowa również produkty mleczne i soję. Największą poprawę stanu zdrowia, w tym szczególnie zmniejszenie występowania nudności, wzdęć, dolegliwości bólowych oraz zwiększenie uczucia komfortu obserwowały osoby z celiakią eliminujące oprócz zbóż glutenowych: mleko i jego przetwory, orzechy i soję. Podobnie największe zmiany samopoczucia związanego z objawami choroby Hashimoto, w tym szczególnie zmniejszenie nadwrażliwości na temperaturę, uczucia suchości skóry oraz poprawę samopoczucia deklarowały osoby eliminujące zboża glutenowe, produkty mleczne, orzechy arachidowe i warzywa psiankowate. Leczenie celiakii oparte na diecie bezglutenowej połączone z eliminacją orzechów i soi może przynieść największe korzyści związane z poprawą stanu zdrowia i jakości życia, a eliminacja zbóż glutenowych i orzeszków ziemnych może wspomagać poprawę jakości życia osób z chorobą Hashimoto. Słowa kluczowe: dieta eliminacyjna, Hashimoto, celiakia

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2

Capilla, A., E. Donat, D. Planelles, C. Espinós, C. Ribes-Koninckx, and F. Palau. "Genetic analyses of celiac disease in a Spanish population confirm association with CELIAC3 but not with CELIAC4." Tissue Antigens 70, no. 4 (October 2007): 324–29. http://dx.doi.org/10.1111/j.1399-0039.2007.00899.x.

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3

Bureš, Jan. "History of celiac disease." Vnitřní lékařství 64, no. 6 (June 1, 2018): 600–601. http://dx.doi.org/10.36290/vnl.2018.083.

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4

Vacková, Zuzana. "Celiac disease in adults." Vnitřní lékařství 66, no. 2 (April 2, 2020): 116–20. http://dx.doi.org/10.36290/vnl.2020.019.

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5

MIKULÍKOVÁ, Renata, Zdeněk SVOBODA, Karolína BENEŠOVÁ, and Sylvie BĚLÁKOVÁ. "Beer and celiac disease." Kvasny Prumysl 59, no. 10-11 (October 1, 2013): 321–23. http://dx.doi.org/10.18832/kp2013035.

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6

Hoffmanová, Iva, Daniel Sánchez, and Helena Tlaskalová-Hogenová. "Diagnostic pitfalls of celiac disease." Vnitřní lékařství 65, no. 1 (January 1, 2019): 24–29. http://dx.doi.org/10.36290/vnl.2019.006.

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7

Karásková, Eva, Maria Velgáňová-Véghová, and Miloš Geryk. "Celiac disease in a family." Pediatrie pro praxi 22, no. 2 (April 29, 2021): 135–38. http://dx.doi.org/10.36290/ped.2021.026.

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8

Atalar, O., and S. Yilmaz. "The branches of the arteria celiaca in the porcupine (Hystrix cristata)." Veterinární Medicína 49, No. 2 (March 29, 2012): 52–56. http://dx.doi.org/10.17221/5675-vetmed.

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This study is aimed at an investigation of the A. celiaca and its branches in the porcupine. Nine adult porcupines (5 males, 4 females) were injected a coloured latex mixture from the aortic arch for the demonstration of the arteria celiaca. The results indicated that the A. celiaca gave off the A. phrenica caudalis after approximately 1 cm from its origin. The A. celiaca was divided into two branches as the A. lienalis and a. hepatica. The A. hepatica was a continuity of celiac artery. The ramus pancreaticus, which was the most important artery for pancreas vascularization, arose from the A. lienalis. The ramus gastrolienalis and the Aa. gastricae breves were observed. The first branch of the A. hepatica was the A. gastrica dextra. The thickest branch of hepatic artery was the A. gastrica sinistra that separated two branches: the ramus visceralis and the ramus parietalis. In summary, in the present study the branches of the A. celiaca in porcupines were studied for the first time. The results of this study may contribute to the data in this area of science.

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9

Strocchi, A., G. Corazza, J. Furne, C. Fine, A. Di Sario, G. Gasbarrini, and M. D. Levitt. "Measurements of the jejunal unstirred layer in normal subjects and patients with celiac disease." American Journal of Physiology-Gastrointestinal and Liver Physiology 270, no. 3 (March 1, 1996): G487—G491. http://dx.doi.org/10.1152/ajpgi.1996.270.3.g487.

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Normal intestinal absorption of nutrients requires efficient luminal mixing to deliver solute to the brush border. Lacking such mixing, the buildup of thick unstirred layers over the mucosa markedly retards absorption of rapidly transported compounds. Using a technique based on the kinetics of maltose hydrolysis, we measured the unstirred layer thickness of the jejunum of normal subjects and patients with celiac disease, as well as that of the normal rat. The jejunum of humans and rats was perfused with varying maltose concentrations, and the apparent Michaelis constant (Km) and maximal velocity (Vmax) of maltose hydrolysis were determined from double-reciprocal plots. The true Km of intestinal maltase was determined on mucosal biopsies. Unstirred layer thickness was calculated from the in vivo Vmax and apparent Km and the in vitro Km of maltase. The average unstirred layer thickness of 11 celiac patients (170 micron) was seven times greater than that of 3 controls (25 micron). The unstirred layer of each celiac exceeded that of the controls. A variety of factors could account for the less efficient luminal stirring observed in celiacs. Although speculative, villous contractility could be an important stirring mechanism that would be absent in celiacs with villous atrophy. This speculation was supported by the finding of a relatively thick unstirred layer (mean: 106 micron) in rats, an animal that lacks villous contractility. Because any increase in unstirred layer slows transport of rapidly absorbed compounds, poor stirring appears to represent a previously unrecognized defect that could contribute to malabsorption in celiac disease and, perhaps, in other intestinal disorders.

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10

Trandafir, Laura Mihaela, Eugen Cîrdeiu, Carmen Oltean, Doina Mihăilă, and Dana-Teodora Anton-Păduraru. "POLIMORFISMUL MANIFESTĂRILOR CLINICE ÎN BOALA CELIACĂ LA COPIL." Romanian Journal of Pediatrics 64, no. 4 (December 31, 2015): 467–71. http://dx.doi.org/10.37897/rjp.2015.4.24.

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Boala celiacă (BC) reprezintă o afecţiune sistemică autoimună produsă prin sensibilizarea la gluten la pacienţii cu predispoziţie genetică. Manifestările clinice ale BC sunt extrem de variate, de la sindromul tipic de malabsorbţie intestinală (caracterizat prin diaree cronică, meteorism abdominal şi malnutriţie) la simptome atipice care pot afecta orice sistem sau organ: constipaţie cronică, hepatocitoliză persistentă, anemie refractară la tratamentul cu fier, dureri abdominale recurente, afecţiuni neurologice, defect al smalţului dentar. Autorii prezintă patru pacienţi de vârstă pediatrică diagnosticaţi cu diferite forme atipice de boală celiacă: forma cu dureri abdominale recurente, cu sindrom de hepatocitoliză şi forma cu constipaţie cronică, toate însoţite de falimentul creşterii şi un caz de diabet zaharat tip I asociat cu boală celiacă. În concluzie, este necesară cunoaşterea tuturor formelor clinice ale bolii celiace de către medicul pediatru, gastroenterolog şi medicul de familie pentru a diagnostica boala la vârsta copilăriei şi, astfel, să prevină complicaţiile pe termen lung, respectiv osteoporoza, infertilitatea si limfomul intestinal.

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11

Waszczuk, Ewa, and Julia Jawny. "Endocrine disorders and coeliac disease." Gastroenterology Review 4 (2011): 209–12. http://dx.doi.org/10.5114/pg.2011.24302.

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12

Bureš, Jan. "Celiac disease in 2018." Vnitřní lékařství 64, no. 6 (June 1, 2018): 602–10. http://dx.doi.org/10.36290/vnl.2018.084.

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13

Martins, Rita de Cássia Azevedo, Lenora Gandolfi, Inês Cristina Modelli, Rodrigo Coutinho de Almeida, Luiz Claudio Castro, and Riccardo Pratesi. "Serologic screening and genetic testing among brazilian patients with celiac disease and their first degree relatives." Arquivos de Gastroenterologia 47, no. 3 (September 2010): 257–62. http://dx.doi.org/10.1590/s0004-28032010000300009.

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CONTEXT: Celiac disease susceptibility has been shown to be associated with the HLA alleles DQA1*0501 and DQB1*0201 (together encoding the DQ2 heterodimer) that are present in practically all celiac disease patients. The DQ8 heterodimer (coded by DQA1*03-DQB1*0302), which is carried on a DRB1*04 (DR4) haplotype, is commonly encoded for by the few celiacs who do not carry the DQ2 heterodimer. Only a few celiac disease patients have been reported without these known risk alleles. OBJECTIVE: To assess the prevalence of celiac disease in a group of first degree relatives of celiac patients, and the frequency of HLA predisposing alleles both in the group of celiac patients and in their first degree relatives, identifying those first degree relatives who would need further screening for celiac disease. METHODS: Ninety celiac disease patients and 207 first degree relatives underwent serologic screening for celiac disease (endomysial and transglutaminase antibodies) followed by intestinal biopsy in positive patients. The HLA-DQA1*0501, DQB1*0201 and DRB1*04 frequencies of celiac patients and their first degree relatives were determined utilizing the PCR method. RESULTS: All the celiac disease patients (n = 90) with the exception of one (1.1%) carried at least one of the alleles investigated. Altogether 11 (5.3%) of the investigated first degree relatives did not carry any of the alleles studied. Fourteen (6.7%) new cases of celiac disease were found among the 207 celiac disease patients first degree relatives. CONCLUSIONS: Considering the cost-benefit of the HLA typing of all the first degree relatives of celiac patients, their HLA status should be decided on an individual basis, taking account of their profile and preferences, and the existence of other medical conditions.

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14

Gaiani, Federica, Sara Graziano, Fatma Boukid, Barbara Prandi, Lorena Bottarelli, Amelia Barilli, Arnaldo Dossena, et al. "The Diverse Potential of Gluten from Different Durum Wheat Varieties in Triggering Celiac Disease: A Multilevel In Vitro, Ex Vivo and In Vivo Approach." Nutrients 12, no. 11 (November 20, 2020): 3566. http://dx.doi.org/10.3390/nu12113566.

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The reasons behind the increasing prevalence of celiac disease (CD) worldwide are still not fully understood. This study adopted a multilevel approach (in vitro, ex vivo, in vivo) to assess the potential of gluten from different wheat varieties in triggering CD. Peptides triggering CD were identified and quantified in mixtures generated from simulated gastrointestinal digestion of wheat varieties (n = 82). Multivariate statistics enabled the discrimination of varieties generating low impact on CD (e.g., Saragolla) and high impact (e.g., Cappelli). Enrolled subjects (n = 46) were: 19 healthy subjects included in the control group; 27 celiac patients enrolled for the in vivo phase. Celiacs were divided into a gluten-free diet group (CD-GFD), and a GFD with Saragolla-based pasta group (CD-Sar). The diet was followed for 3 months. Data were compared between CD-Sar and CD-GFD before and after the experimental diet, demonstrating a limited ability of Saragolla to trigger immunity, although not comparable to a GFD. Ex vivo studies showed that Saragolla and Cappelli activated immune responses, although with great variability among patients. The diverse potential of durum wheat varieties in triggering CD immune response was demonstrated. Saragolla is not indicated for celiacs, yet it has a limited potential to trigger adverse immune response.

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15

Rukunuzzaman, Md, ASM Bazlul Karim, SM Baqui Billah, Md Atiar Rahman, Md Mahbubul Islam, and Mohammed Kamal. "Celiac Disease in Bangladesh Two Case Reports." Bangladesh Journal of Child Health 37, no. 1 (June 18, 2013): 45–48. http://dx.doi.org/10.3329/bjch.v37i1.15351.

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Celiae disease is an immunological disorder precipitated by gluten in genetically susceptible persons. Its prevalence is not known in Bangladesh because of unavailability of its screening test. There is diversity in the presentation of celiac disease. Two children of 5 and 8 years of age who were diagnosed as celiac disease are reported here. One presented typically with chronic diarrhoea & growth failure. Another child presented with features of chronic liver disease. In both the cases IgA tTGA were positive and duodenal biopsy showed villous atrophy. After diagnosis, both the patients were kept on gluten free diet (GFD). After six months of GFD, IgA tTGA came down to normal in both the cases. They were then given gluten containing diet again & after few months IgA tTGA again raised in both the cases. Thereafter the cases were finally diagnosed as celiac disease and were advised life long gluten free diet. Celiac disease is not uncommon in Bangladesh and screening test should be done to diagnose or rule out celiac disease when there is a suspicion. DOI: http://dx.doi.org/10.3329/bjch.v37i1.15351 BANGLADESH J CHILD HEALTH 2013; VOL 37 (1) : 45-48

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16

Packová, Barbora, Michal Šenkyřík, and Radek Kroupa. "Celiac disease - diangostics, therapy, risk of cancer." Onkologie 12, no. 6 (December 15, 2018): 293–96. http://dx.doi.org/10.36290/xon.2018.053.

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17

Latta, Jiří, and Jana Povová. "Celiac disease in elderly - diagnostics, comorbidity, therapy." Interní medicína pro praxi 18, no. 1 (February 1, 2016): 33–34. http://dx.doi.org/10.36290/int.2016.008.

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18

Bojkovic, Gradimir, Zorica Caparevic, Vesna Ilic, Dragos Stojanovic, Djordje Lalosevic, and Mirjana Stojanovic. "Celiac disease - case report." Medical review 55, no. 11-12 (2002): 532–34. http://dx.doi.org/10.2298/mpns0212532b.

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Introduction Celiac disease (nontropical sprue, gluten-sensitive enteropathy, chronic intestinal malabsorption disorder) is caused by gluten intolerance. This hereditary disorder is caused by sensitivity to gliadin. Because the body's own immune system causes the damage, celiac disease is considered to be an autoimmune disorder. However, it is also classified as a disease of malabsorption because nutrients are not absorbed. When people with celiac disease eat foods containing gluten, their immune system responds by damaging the small intestine. Specifically, tiny finger-like protrusions, called villi, on the lining of the small intestine are lost. The diagnosis is suspected on the basis of symptoms and signs, enhanced by laboratory and x-ray studies, and confirmed by biopsy revealing flat mucosa and subsequent clinical and histologic improvement on a gluten-free diet. Gluten must be excluded from diet. Supplementary vitamins, minerals and hematinics may be given depending on deficiency. Case report This is a case report of a 23-year old female patient with a mineralization defect (osteomalacia) and secondary osteoporosis caused by long-time unrecognized celiac disease. The patient had many symptoms: short stature, steatorrhea, anemia, weight loss and chronic bone pain. Laboratory and x-ray studies and jejunal biopsy revealed a chronic intestinal malabsorption disorder caused by gluten intolerance. Gluten-free diet and supplementary vitamins, minerals and hematinics were included with apparent clinical remission. Discussion and Conclusion Some people with celiac disease may not have symptoms. The undamaged part of their small intestine is able to absorb enough nutrients to prevent symptoms. However, people without symptoms are still at risk for complications of celiac disease. Biopsy of the small intestine is the best way to diagnose celiac disease. Decreased bone density (osteoporosis and osteomalacia) is a serious problem for celiacs. If calcium is not absorbed, due to small intestinal damage caused by untreated celiac disease, bones are not as dense. The only treatment for celiac disease is gluten-free diet, that is, avoiding all foods that contain gluten. For most people, this diet stops symptoms, heals the existing intestinal damage, and prevents further damage.

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Blagojevic, Milos, Bogomir Prokic, and Dejana Cupic-Miladinovic. "A. Hepatica in European ground squirrel (Citellus Citellus) compared to other experimental animals." Veterinarski glasnik 70, no. 1-2 (2016): 31–39. http://dx.doi.org/10.2298/vetgl1602031b.

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European ground squirrel is the only representative of its genus in Serbia. It is used as experimental animal in microbioogy, parasitology, pharmacology and immunology. The objective of this work was to investigate a part of cardiovascular system of ground squirrel so in that way to contribute to a better knowledge of this animal body structure and accordingly to comparative anatomy in general. The investigation included 6 ground squirrels, of both gender, body weight 200-300 grams. For obtaining the liver arterial vascularization, after exsanguination of the animal, contrast mass of gelatin coloured with tempera was injected into abdominal aorta (Aorta abdominalis). After injecting, the blood vessels were prepared and photographed. In ground squirrel A. celiaca is odd, larger vessel that exits the abdominal aorta. It is divided into three branches: A. lienalis, A. gastrica sinistra and A. hepatica. A. hepatica is divided into A. hepatica propria and A. gastroduodenalis. A. hepatica propria further gives A. cystica, Rami cardiaci and small branches for Lnn. portales. A. gastroduodenalis is divided into A. pancreaticoduodenalis and A. gastroepiploica dextra. A. celiaca in nutria and rat is an odd artery, divided into A. lienalis, A. gastrica sinistra and A. hepatica. In rabbits, celiac artery (A. celiaca) is divided into A. lienalis and short trunk from which A. gastrica sinistra and A. Hepatica emerge. A. celiaca in golden hamster does not exist in the form of tripus coeliacus (A. lienalis, A. gastrica sinistra and A. hepatica), but from A. celiaca it is firstly separated A. hepatica, and then short trunk from which A. gastrica sinistra and A. Lienalis emerge. In guinea-pig, from abdominal aorta a joint tree branches off into A. celiaca and A. mesenterica cranialis (Truncus celiacomesentericus). Based on the above mentioned results, it can be concluded that A. celiaca in European ground squirrel, nutria and rat branches from abdominal aorta as a separate blood vessel. In these animals A. celiaca branches are: A. lienalis, A. gastrica sinistra and A. hepatica. <br><br><font color="red"><b> This article has been corrected. Link to the correction <u><a href="http://dx.doi.org/10.2298/VETGL1702141E">10.2298/VETGL1702141E</a><u></b></font>

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Kocsis, D., G. Veres, E. Jocsak, Z. Toth, A. Csontos, P. Miheller, Z. Tulassay, and M. Juhasz. "P160 Prevalence of IBD among celiacs in our celiac centre." Journal of Crohn's and Colitis 8 (February 2014): S129. http://dx.doi.org/10.1016/s1873-9946(14)60282-8.

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Dobrzycka, Anna, and Iwona Wilk. "Celiac disease: Definition, diagnosis, symptoms, and methods of treatment." Nursing and Public Health 10, no. 4 (December 22, 2020): 1–8. http://dx.doi.org/10.17219/pzp/125436.

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Antanavičiūtė, Kamilė, and Rūta Šarkūnaitė. "CELIAKIJOS IR 1 TIPO CUKRINIO DIABETO RYŠYS." Health Sciences 31, no. 4 (July 1, 2021): 128–31. http://dx.doi.org/10.35988/sm-hs.2021.139.

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Celiakija – tai autoimuninis plonųjų žarnų uždegimas, sukeltas Th-1 limfocitų hiperreakcijos į kviečiuose, rugiuose bei miežiuose esantį baltymą gliadiną bei lemiantis gleivinės gaurelių atrofiją, kriptų hiperplaziją ir malabsorbciją. Nustatyta, jog sergantys šia liga yra linkę sirgti 1 tipo cukriniu diabetu (CD), nes abi ligas sieja bendra genetinė kilmė. Pasaulyje celiakijos dažnis 1 tipo CD sergančiųjų populiacijoje yra nuo 5 iki 16 kartų didesnis, nei bendrojoje. Dažnai celiakija sergant 1 tipo CD yra asimptominė, tačiau, jos negydant, stebima bloga glikemijos kontrolė, didesnė kardiovaskulinių ligų rizika, didėja diabeto komplikacijų pasireiškimo rizika jaunesniame amžiuje. Šiuo metu nėra bendrosios nuomonės dėl celiakijos diagnozavimo ir gydymo taktikos sergant 1 tipo CD. Pagrindinis celiakijos gydymo būdas bendrojoje populiacijoje yra ilgalaikė begliutenė dieta, todėl šis gydymas rekomenduojamas ir sergantiesiems abiem minėtomis ligomis, tačiau laikantis šios dietos, sergantieji 1 tipo CD turi būti prižiūrimi gydytojų endokrinologo ir dietologo. Tyrimo tikslas − išanalizuoti mokslinėje literatūroje aprašytą celiakijos ir 1 tipo cukrinio diabeto ryšį. Tyrimo metodika. Atlikta sisteminė literatūros apžvalga, naudojantis PubMed, ScienceDirect duomenų bazėmis, nagrinėjant straipsnius apie celiakinę ligą ir 1 tipo cukrinį diabetą. Atrinkti ir išanalizuoti 23 straipsniai. Rezultatai. Atlikti tyrimai patvirtina didesnį celiakijos pasireiškimo dažnį tarp sergančiųjų 1 tipo CD, lyginant su sveikąja populiacija.

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Szczerba, Edyta, Aleksandra Kozłowska, and Aneta Nitsch-Osuch. "Dietary treatment of Coleiac Disease (CD) coexisting with Type 1 Diabetes Mellitus (T1DM)." Medycyna Ogólna i Nauki o Zdrowiu 24, no. 3 (September 27, 2018): 166–71. http://dx.doi.org/10.26444/monz/94718.

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De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (January 1, 2018): 129–30. http://dx.doi.org/10.15446/.v66n1.68105.

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Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las formas no clásicas y silentes son cada vez más frecuentes y se caracterizan por un amplio abanico de signos y síntomas como osteoporosis, hipovitaminosis D, anemia ferropénica, dermatitis herpetiforme, ataxia, polineuropatía sensitiva periférica, migrañas y alteración de las pruebas de función hepática (2).

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De Benedictis-Serrano, Ginno Alessandro, Laura Contreras-Lugo, Génesys Córdova-Rivas, and Carlos Miguel Ríos-González. "El esprúe tropical, una enfermedad olvidada como diagnóstico diferencial de la enfermedad celiaca." Revista de la Facultad de Medicina 66, no. 1 (January 1, 2018): 129–30. http://dx.doi.org/10.15446/revfacmed.v66n1.68105.

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Estimado editor, Por medio de la presente nos gustaría traer a discusión la importancia que tiene la enfermedad celiaca (EC), también conocida como esprúe celiaco o enteropatía sensible al gluten, la cual corresponde a una enfermedad autoinmune desencadenada por la ingesta de gluten en individuos predispuestos genéticamente (1). La EC se caracteriza por una amplia variabilidad de manifestaciones clínicas, pero su forma clásica es la más habitual en el adulto; aquí los síntomas guía son de naturaleza gastrointestinal (diarrea, esteatorrea y dolor abdominal inespecífico). Por otro lado, las formas no clásicas y silentes son cada vez más frecuentes y se caracterizan por un amplio abanico de signos y síntomas como osteoporosis, hipovitaminosis D, anemia ferropénica, dermatitis herpetiforme, ataxia, polineuropatía sensitiva periférica, migrañas y alteración de las pruebas de función hepática (2).

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26

Lemos, Andréa dos Reis, Vanessa Dias Capriles, Maria Elisabeth Machado Pinto e Silva, and José Alfredo Gomes Arêas. "Effect of incorporation of amaranth on the physical properties and nutritional value of cheese bread." Food Science and Technology 32, no. 3 (July 31, 2012): 427–31. http://dx.doi.org/10.1590/s0101-20612012005000079.

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At the present celiac disease has no known cure, and its only treatment is a strict lifelong adherence to a gluten-free diet. Cheese bread is a traditional Brazilian product and a safe option for celiacs. However, like other gluten-free breads, it has inherent low levels of fibers and minerals. The objective of this study was to evaluate the effect of incorporation of whole amaranth flour on the physical properties and nutritional value of cheese bread. Amaranth flour was incorporated at 10, 15, and 20% proportions in different formulations. The increasing amaranth levels darkened the product, reduced specific volume, and increased compression force. Ten percent amaranth-content cheese breads exhibited slight differences in physical properties compared with the controls. These results demonstrated the possibility of incorporating 10% of whole amaranth flour in the formulation of cheese bread resulting in a product with higher dietary fiber and iron contents and the same level of acceptance as that of the conventional formulation. The aim of this approach is to increase the availability of gluten-free bakery products with added nutritional value contributing to increase the variety of the diet of celiac patients.

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Szaflarska-Popławska, Anna. "Liver injury in coeliac disease – own study and review of the literature." Gastroenterology Review 4 (2011): 259–66. http://dx.doi.org/10.5114/pg.2011.24309.

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Sánchez-Iglesias, Sofía, Antía Fernández-Pombo, Silvia Cobelo-Gómez, Álvaro Hermida-Ameijeiras, Helena Alarcón-Martínez, Rosario Domingo-Jiménez, Alejandro Iván Ruíz Riquelme, Jesús R. Requena, and David Araújo-Vilar. "Celia’s Encephalopathy (BSCL2-Gene-Related): Current Understanding." Journal of Clinical Medicine 10, no. 7 (April 1, 2021): 1435. http://dx.doi.org/10.3390/jcm10071435.

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Seipin, encoded by the BSCL2 gene, is a protein that in humans is expressed mainly in the central nervous system. Uniquely, certain variants in BSCL2 can cause both generalized congenital lipodystrophy type 2, upper and/or lower motor neuron diseases, or progressive encephalopathy, with a poor prognosis during childhood. The latter, Celia’s encephalopathy, which may or may not be associated with generalized lipodystrophy, is caused by the c.985C >T variant. This cytosine to thymine transition creates a cryptic splicing zone that leads to intronization of exon 7, resulting in an aberrant form of seipin, Celia seipin. It has been proposed that the accumulation of this protein, both in the endoplasmic reticulum and in the nucleus of neurons, might be the pathogenetic mechanism of this neurodegenerative condition. In recent years, other variants in BSCL2 associated with generalized lipodystrophy and progressive epileptic encephalopathy have been reported. Interestingly, most of these variants could also lead to the loss of exon 7. In this review, we analyzed the molecular bases of Celia’s encephalopathy and its pathogenic mechanisms, the clinical features of the different variants, and a therapeutic approach in order to slow down the progression of this fatal neurological disorder.

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Zwolińska-Wcisło, Małgorzata, Romana Tomaszewska, Piotr Rozpondek, Magdalena Przybylska, and Tomasz Mach. "Mucosal lesions of the gastric mucosa in adult patients with coeliac disease." Gastroenterology Review 5 (2012): 291–98. http://dx.doi.org/10.5114/pg.2012.32068.

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Nowak-Oczkowska, Aleksandra, Anna Szaflarska-Popławska, and Anetta Soroczyńska-Wrzyszcz. "Are Down’s syndrome patients a risk group for celiac disease?" Gastroenterology Review 2 (2013): 77–85. http://dx.doi.org/10.5114/pg.2013.34832.

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Szaflarska-Popławska, Anna, and Anna Krakowska. "Influence of specific dietary interventions on clinical manifestation of coeliac disease." Gastroenterology Review 1 (2010): 24–30. http://dx.doi.org/10.5114/pg.2010.13944.

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Ciobanu, Daniela, Lorena Elena Meliț, Ana-Maria Șimon, Nicoleta Tomșa, and Cristina Oana Mărginean. "Criza celiacă, formă atipică de debut al bolii celiace la adolescent – prezentare de caz." Romanian Journal of Pediatrics 69, no. 4 (December 31, 2020): 335–38. http://dx.doi.org/10.37897/rjp.2020.4.14.

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MUCENICA, Irina Manuela, Ancuţa IGNAT, Gabriela PĂDURARU, Andrada DRUICĂ, Valeriu Vasile LUPU, and Marin BURLEA. "Celiac disease: diagnosis and dilemmas." Romanian Journal of Medical Practice 11, no. 2 (June 30, 2016): 204–7. http://dx.doi.org/10.37897/rjmp.2016.2.17.

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Celiac disease is an autoimmune disorder of the gastrointestinal tract. The condition may develop at any age, triggered by exposure to dietary gluten in genetically susceptible individuals. The most frequent intestinal manifestations include diarrhea and weight loss. Common extradigestive manifestations include iron deficiency anemia, dermatitis herpetiformis. The authors presents a clinical case diagnosed with celiac disease expressed with gastrointestinal and extradigestive symptoms, such as abdominal pain, flatulence, anemia, impaired hepatic function and dermatitis herpetiformis.

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Hoffmanová, Iva. "Extraintestinální projevy celiakie." Vnitřní lékařství 66, no. 2 (April 2, 2020): e6-e7. http://dx.doi.org/10.36290/vnl.2020.033.

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Deus, Elielson Silva Gomes de. "DOENÇA CELÍACA (DC) – Diagnóstico, tratamento, hábitos e práticas alimentares." Revista Científica Multidisciplinar Núcleo do Conhecimento 04, no. 01 (January 15, 2019): 37–49. http://dx.doi.org/10.32749/nucleodoconhecimento.com.br/saude/doenca-celiaca.

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Rato, Beatriz. "Doença Celíaca no Idoso." Medicina Interna 28, no. 2 (June 18, 2021): 175–83. http://dx.doi.org/10.24950/r/293/20/2/2021.

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A doença celíaca é um distúrbio crónico e autoimune que pode surgir em qualquer idade. Atualmente, um quarto dos novos diagnósticos ocorre depois dos 60 anos de idade. A apresentação clínica subtil com défices de micronutrientes e osteopenia ou osteoporose, associada à subvalorização médica desta hipótese, leva ao seu subdiagnóstico neste grupo etário. A doença celíaca na idade avançada pode ter maior risco de associação a doenças autoimunes, linfoproliferativas e cardiovasculares, com impacto na morbimortalidade. A adesão à dieta sem glúten não parece ser problemática e a qualidade de vida aparenta não diminuir. É essencial um seguimento multidisciplinar adaptado ao doente. Nesta revisão, sistematiza-se a evidência atual da doença celíaca no idoso incluindo epidemiologia, fisiopatologia, manifestações clínicas, diagnóstico e abordagem terapêutica, com o objetivo de consciencializar e reduzir o atraso no seu diagnóstico.

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Mohan, Neelam, Sakshi Karkra, and Rajeev Khanna. "Is Atypical Celiac Disease on a Rise in India? Is HLA Expression Different in Atypical Celiacs?" American Journal of Gastroenterology 104 (October 2009): S110. http://dx.doi.org/10.14309/00000434-200910003-00290.

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Neuhausen, Susan L., Linda Steele, Sarah Ryan, Maryam Mousavi, Marie Pinto, Kathryn E. Osann, Pamela Flodman, and John J. Zone. "Co-occurrence of celiac disease and other autoimmune diseases in celiacs and their first-degree relatives." Journal of Autoimmunity 31, no. 2 (September 2008): 160–65. http://dx.doi.org/10.1016/j.jaut.2008.06.001.

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Ferster, Magdalena, Anna Obuchowicz, Beata Jarecka, Jolanta Pietrzak, and Krystyna Karczewska. "Difficulties related to compliance with gluten-free diet by patients with coeliac disease living in Upper Silesia." Pediatria i Medycyna Rodzinna 11, no. 4 (December 30, 2015): 410–18. http://dx.doi.org/10.15557/pimr.2015.0039.

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de Vicente Ortega, A., B. Tercero Lozano, and S. Jamal Ismail. "Celiac disease diagnosed by video capsule endoscopy." Revista Andaluza de Patología Digestiva 43, no. 5 (November 4, 2020): 200–202. http://dx.doi.org/10.37352/2020435.8.

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Resumen La celiaquía es una enfermedad multiorgánica autoinmune crónica, que afecta al intestino delgado en individuos genéticamente predispuestos, cuya prevalencia se está viendo aumentada. Exponemos un caso de celiaquía, en el que gracias a la obtención de una imagen típica por videocapsula endoscópica, se consigue una alta sospecha que permite completar el diagnóstico. Como conclusión, queremos resaltar la importancia de la imagen típica de atrofia vellositaria para la cual la videocápsula endoscópica es muy útil, aunque deba confirmarse histopatológicamente.

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Olteanu, Dan, Alexandru Diaconescu, Radu Voiosu, Andrei Voiosu, and Cristina Olariu. "PRESENT DATA IN THE DIAGNOSIS AND TREATMENT OF COELIAC DISEASE (2)." Romanian Medical Journal 64, no. 1 (March 31, 2017): 25–33. http://dx.doi.org/10.37897/rmj.2017.1.5.

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Coeliac disease incidence rised during the last 50 years and represents a concern by diagnostic problems and costs. The recent data regarding etiology, pathogeny, comparative diagnostic value of serology and small intestinal biopsy are summarised. The new data about refractory celiac disease to gluten free diet and therapeutic perspectives are also presented (glutenases, larazotide acetate, genetic alteration of cereals, tissulary transglutaminase inhibitors etc).

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Socha, Jerzy, and Bożena Cukrowska. "Letter to Editor Screening tests for celiac disease in children with autistic spectrum disorders." Gastroenterology Review 1 (2011): 55–56. http://dx.doi.org/10.5114/pg.2011.20109.

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Olteanu, Dan, Alexandru Diaconescu, Radu Voiosu, Andrei Voiosu, and Cristina Olariu. "Present data in the diagnosis and treatment of coeliac disease (Part I)." Romanian Medical Journal 63, no. 4 (December 31, 2016): 272–79. http://dx.doi.org/10.37897/rmj.2016.4.2.

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Coeliac disease incidence raised during the last 50 years and represents a concern by diagnostic problems and costs. The recent data regarding etiology, pathogeny, comparative diagnostic value of serology and small intestinal biopsy are summarised. New data about refractory celiac disease to gluten free diet and therapeutic perspectives are also presented (glutenases, larazotide acetate, genetic alteration of cereals, tissulary transglutaminase inhibitors etc).

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Ferreira, Silvia, María Elena Chamorro, José Masi, Diana Sanabria, Sara Benegas, María Mercedes Carpinelli, Vivian Giménez, and Patricia Langjahr. "Niveles de IgA en adultos con enfermedad celíaca." Memorias del Instituto de Investigaciones en Ciencias de la Salud 17, no. 1 (April 15, 2019): 54–58. http://dx.doi.org/10.18004/mem.iics/1812-9528/2019.017(01)54-058.

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Berioli, Maria Giulia, Giulia Mancini, Nicola Principi, Elisa Santi, Martina Ascenzi, Francesco Rogari, Giulia Ceccarini, Ursula Grohmann, and Susanna Esposito. "Growth and glycemic control in children with type 1 diabetes and asymptomatic celiac disease treated with a gluten -free diet for 1 year." European Journal of Inflammation 17 (January 2019): 205873921985557. http://dx.doi.org/10.1177/2058739219855574.

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To compare growth and glycemic control in children with type 1 diabetes and silent celiac disease treated with a gluten-free diet for 1 year with those of similar age and gender with type 1 diabetes but without celiac disease, 16 type 1 diabetes patients with silent celiac disease were enrolled and each celiac disease-positive case was matched for age, sex, and duration of diabetes with two type 1 diabetes controls with negative serologic markers of celiac disease. All 16 children with positive celiac disease serology had histologic features consistent with celiac disease despite the absence of symptoms. The mean growth and metabolic control values were similar between children with type 1 diabetes and celiac disease and those with type 1 diabetes but without celiac disease. This study seems to suggest that the early diagnosis of celiac disease and initiation of a gluten-free diet may prevent further deterioration in the nutritional status of children with type 1 diabetes and celiac disease and may reduce the prospect of celiac disease complications without any impact on type 1 diabetes control.

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Limanowska, Paulina, Justyna Kubiak, Katarzyna Napieraj, and Dorota Różańska. "Evaluation of selected aspects of daily life and factors determining adherence to a gluten-free diet in patients with celiac disease." Medycyna Ogólna i Nauki o Zdrowiu 20, no. 4 (December 15, 2014): 356–59. http://dx.doi.org/10.5604/20834543.1132035.

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Chishty, Sadia, Monika Monika, and Nimali Singh. "Nutritional status of celiac and non-celiac children from Rajasthan, India." Nutrition & Food Science 47, no. 2 (March 13, 2017): 240–53. http://dx.doi.org/10.1108/nfs-05-2016-0056.

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Purpose The aim of the study was to compare the nutritional status of children having celiac disease (CD) with those not having the disease in the age group of 7-12 years. Children not having CD were from first- and second-degree siblings of the children affected to match for family and environment. In Indian celiac pediatric population, studies on nutritional status of celiac children and comparisons with their siblings as reference have not been reported. Design/methodology/approach Children with CD (n = 50) and without CD (n = 25) were matched for age and were purposively selected. Nutritional assessment included anthropometry, biochemical, clinical and nutrient intake. Weight and height measurements were recorded as per the standardized techniques. Biochemical investigations were done by skilled technicians. A two-day 24-h dietary recall method was used for calculation of nutrient intake. The observations were categorized as celiac and non-celiac children and pre-adolescents in age group of 7-9 years and 10-12 years. Findings The issues of being underweight and of low height according to age among celiac children were rampant. The non-celiac children were comparatively taller than children with CD. The hemoglobin and serum iron were significantly lower in celiac than non-celiac group (p < 0.01). Energy intake was significantly higher (p < 0.05) in celiac pre-adolescents as compared to non-celiac children (7-9 years). The protein intake was higher in non-celiac children, and the difference was highly significant (p < 0.01). The fat consumption was higher in celiac pre-adolescents. The nutritional status of celiac children was poor as compared to non-celiac children. Originality/value The present study is an attempt to compare the nutritional status of celiac children with their siblings.

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González, Teba, Idoia Larretxi, Juan Vitoria, Luis Castaño, Edurne Simón, Itziar Churruca, Virginia Navarro, and Arrate Lasa. "Celiac Male’s Gluten-Free Diet Profile: Comparison to that of the Control Population and Celiac Women." Nutrients 10, no. 11 (November 8, 2018): 1713. http://dx.doi.org/10.3390/nu10111713.

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The aim of the present work was to analyze the body composition and dietary profile of Spanish celiac men and to compare them to control men and celiac women from our previous studies. Forty-two celiac men (31.5 ± 11.9 years) were recruited and anthropometric measurements were taken. Analysis of energy consumption, macro- and micronutrient intake and food frequency consumption was carried out. Celiac men were more overweight and obese than celiac women, but less than the control population, reporting the same energy intake and macronutrient distribution. Most micronutrient deficiencies in celiac men were not directly related to a gluten free diet; these were also observed for the entire population. The least adherence to Dietary Reference Intakes in women was reported for iron, iodine, potassium and selenium, whereas magnesium intake was higher than in men. Among celiac participants (both genders), cereal, vegetable and legume consumption was poor and meat intake was contrastingly excessive. In conclusion, the dietary profile of celiac men is as unbalanced as that of control men but slightly more than that of celiac women. General nutritional education should be given to both general and celiac populations, and specific advices to celiac men, in order to decrease the risk of celiac disease-related pathologies.

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CECILIO, Lucila Arantes, and Mauro W. BONATTO. "THE PREVALENCE OF HLA DQ2 AND DQ8 IN PATIENTS WITH CELIAC DISEASE, IN FAMILY AND IN GENERAL POPULATION." ABCD. Arquivos Brasileiros de Cirurgia Digestiva (São Paulo) 28, no. 3 (September 2015): 183–85. http://dx.doi.org/10.1590/s0102-67202015000300009.

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Background: Celiac disease is an enteropathy characterized by gluten sensitivity and broad clinical aspect. Has a multifactorial cause and depends on genetic, immunological and environmental factors for its development. The genetic influence is given mostly by the human leukocyte antigens HLA DQ2 and DQ8. Aim: To evaluate the prevalence of human leukocyte antigens DQ2 and DQ8 in three different groups: patients with celiac disease, first-degree relatives and the general population. Method: Retrospective analysis that evaluated serologic and endoscopic data of 74 patients with celiac disease and 109 non-celiac, which were subdivided into two subgroups: non-celiac who had first-degree relatives with celiac and non-celiac who did not. All patients underwent laboratory examination for screening genetic sensitivity given by HLA DQ2 and HLA DQ8 by. Results: The presence of HLA DQ2 and DQ8 was identified in 98,4% of 74 celiac patients, of which 79,7% had only HLA DQ2; 8,1% had only HLA DQ8 and 10,8% had both antigens histocompatibility. In the group of relatives of celiac patients, were included 29 patients; among them, 89,6% had HLA DQ2 and/or DQ8; 76% only the HLA DQ2, 10,3% only HLA DQ8 and 3,4% presented both human leukocyte antigens (HLA). Conclusion: HLA DQ2/DQ8 was present in 98,4% of celiac patients; 89,6% relatives of celiac family and in 55,4% of people from the general population without family celiac.

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Otsuka, Hiroyuki, Toshiki Sato, Hiromichi Aoki, Yoshihide Nakagawa, and Sadaki Inokuchi. "Optimal Treatment for Ruptured Pancreaticoduodenal Artery Aneurysm Caused by Celiac Artery Obstruction Due to Celiac Artery Dissection." Vascular and Endovascular Surgery 52, no. 8 (June 25, 2018): 648–52. http://dx.doi.org/10.1177/1538574418784691.

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A pancreaticoduodenal artery (PDA) aneurysm develops due to increased flow through the pancreaticoduodenal arcade in the setting of celiac or superior mesenteric artery occlusion. Additionally, there is no evidence on the computed tomography scan or angiography images that the dissection process extends to the PDA arcade. Moreover, the optimal treatment protocols for PDA aneurysms with celiac artery obstruction and for celiac artery dissection are controversial. We report 2 cases of ruptured PDA aneurysms caused by celiac artery obstruction due to celiac artery dissection in which the aneurysm was excluded, but celiac artery revascularization was not performed successfully. Our cases indicate that endovascular management for ruptured PDA aneurysms and conservative management for celiac artery obstruction due to celiac artery dissection are feasible as first-line treatment in such cases.

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