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Journal articles on the topic 'Cell attachment'

Author: Grafiati

Published: 4 June 2021

Last updated: 22 February 2023

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1

Yamada, Kenneth. "Cell Attachment." Advances in Dental Research 9, no. 3_suppl (1995): 7. http://dx.doi.org/10.1177/0895937495009003s1901.

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2

Agladze, Konstantin, Xin Wang, and Tony Romeo. "Spatial Periodicity of Escherichia coli K-12 Biofilm Microstructure Initiates during a Reversible, Polar Attachment Phase of Development and Requires the Polysaccharide Adhesin PGA." Journal of Bacteriology 187, no. 24 (2005): 8237–46. http://dx.doi.org/10.1128/jb.187.24.8237-8246.2005.

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ABSTRACT Using fast Fourier transform (FFT) analysis, we previously observed that cells within Escherichia coli biofilm are organized in nonrandom or periodic spatial patterns (K. Agladze et al., J. Bacteriol. 185:5632-5638, 2003). Here, we developed a gravity displacement assay for examining cell adherence and used it to quantitatively monitor the formation of two distinct forms of cell attachment, temporary and permanent, during early biofilm development. Temporarily attached cells were mainly surface associated by a cell pole; permanent attachments were via the lateral cell surface. While t

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3

Yamamoto, Ayumu. "Shake It Off: The Elimination of Erroneous Kinetochore-Microtubule Attachments and Chromosome Oscillation." International Journal of Molecular Sciences 22, no. 6 (2021): 3174. http://dx.doi.org/10.3390/ijms22063174.

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Cell proliferation and sexual reproduction require the faithful segregation of chromosomes. Chromosome segregation is driven by the interaction of chromosomes with the spindle, and the attachment of chromosomes to the proper spindle poles is essential. Initial attachments are frequently erroneous due to the random nature of the attachment process; however, erroneous attachments are selectively eliminated. Proper attachment generates greater tension at the kinetochore than erroneous attachments, and it is thought that attachment selection is dependent on this tension. However, studies of meioti

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4

Joglekar, Ajit P., David Bouck, Ken Finley, et al. "Molecular architecture of the kinetochore-microtubule attachment site is conserved between point and regional centromeres." Journal of Cell Biology 181, no. 4 (2008): 587–94. http://dx.doi.org/10.1083/jcb.200803027.

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Point and regional centromeres specify a unique site on each chromosome for kinetochore assembly. The point centromere in budding yeast is a unique 150-bp DNA sequence, which supports a kinetochore with only one microtubule attachment. In contrast, regional centromeres are complex in architecture, can be up to 5 Mb in length, and typically support many kinetochore-microtubule attachments. We used quantitative fluorescence microscopy to count the number of core structural kinetochore protein complexes at the regional centromeres in fission yeast and Candida albicans. We find that the number of

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5

DeLuca, Jennifer G., Ben Moree, Jennifer M. Hickey, John V. Kilmartin, and E. D. Salmon. "hNuf2 inhibition blocks stable kinetochore–microtubule attachment and induces mitotic cell death in HeLa cells." Journal of Cell Biology 159, no. 4 (2002): 549–55. http://dx.doi.org/10.1083/jcb.200208159.

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Identification of proteins that couple kinetochores to spindle microtubules is critical for understanding how accurate chromosome segregation is achieved in mitosis. Here we show that the protein hNuf2 specifically functions at kinetochores for stable microtubule attachment in HeLa cells. When hNuf2 is depleted by RNA interference, spindle formation occurs normally as cells enter mitosis, but kinetochores fail to form their attachments to spindle microtubules and cells block in prometaphase with an active spindle checkpoint. Kinetochores depleted of hNuf2 retain the microtubule motors CENP-E a

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6

Knutton, Stuart, Tom Baldwin, and Peter Williams. "Actin in cell attachment." Nature 359, no. 6394 (1992): 369. http://dx.doi.org/10.1038/359369a0.

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7

Sanger, J., and J. Sanger. "Actin in cell attachment." Nature 359, no. 6394 (1992): 369. http://dx.doi.org/10.1038/359369b0.

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8

Venturato, Andrea, Gillian MacFarlane, Jin Geng, and Mark Bradley. "Understanding Polymer-Cell Attachment." Macromolecular Bioscience 16, no. 12 (2016): 1864–72. http://dx.doi.org/10.1002/mabi.201600253.

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9

Davydenko, Olga, Richard M. Schultz, and Michael A. Lampson. "Increased CDK1 activity determines the timing of kinetochore-microtubule attachments in meiosis I." Journal of Cell Biology 202, no. 2 (2013): 221–29. http://dx.doi.org/10.1083/jcb.201303019.

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Chromosome segregation during cell division depends on stable attachment of kinetochores to spindle microtubules. Mitotic spindle formation and kinetochore–microtubule (K-MT) capture typically occur within minutes of nuclear envelope breakdown. In contrast, during meiosis I in mouse oocytes, formation of the acentrosomal bipolar spindle takes 3–4 h, and stabilization of K-MT attachments is delayed an additional 3–4 h. The mechanism responsible for this delay, which likely prevents stabilization of erroneous attachments during spindle formation, is unknown. Here we show that during meiosis I, a

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10

Van Dellen, Katrina L., Laetitia Houot та Paula I. Watnick. "Genetic Analysis of Vibrio cholerae Monolayer Formation Reveals a Key Role for ΔΨ in the Transition to Permanent Attachment". Journal of Bacteriology 190, № 24 (2008): 8185–96. http://dx.doi.org/10.1128/jb.00948-08.

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ABSTRACT A bacterial monolayer biofilm is a collection of cells attached to a surface but not to each other. Monolayer formation is initiated when a bacterial cell forms a transient attachment to a surface. While some transient attachments are broken, others transition into the permanent attachments that define a monolayer biofilm. In this work, we describe the results of a large-scale, microscopy-based genetic screen for Vibrio cholerae mutants that are defective in formation of a monolayer biofilm. This screen identified mutations that alter both transient and permanent attachment. Transient

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11

Katchinskiy, Nir, Roseline Godbout, Helly R. Goez, and Abdulhakem Y. Elezzabi. "Femtosecond laser-induced cell-cell surgical attachment." Lasers in Surgery and Medicine 46, no. 4 (2014): 335–41. http://dx.doi.org/10.1002/lsm.22230.

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12

Kim, Il-gu. "Anglo-American Manic Narratives." Convergence English Language & Literature Association 7, no. 3 (2022): 1–34. http://dx.doi.org/10.55986/cell.2022.7.3.1.

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In the previous education it was taken for granted that the step-by-step progress in technology brought about development. Thus, a lot of time and effort were invested in the development of technology. Despite material progress, however, there is growing evidence that we are failing to create a sustainable future for humanity and a community where discrimination and conflict are minimized. If future generations do not have an intimate attachment to others by practicing environmental and social justice, there is a high possibility that humankind will gradually lose the light of hope and turn in

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13

Głuszek, A. Agata, C. Fiona Cullen, Wenjing Li, et al. "The microtubule catastrophe promoter Sentin delays stable kinetochore–microtubule attachment in oocytes." Journal of Cell Biology 211, no. 6 (2015): 1113–20. http://dx.doi.org/10.1083/jcb.201507006.

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The critical step in meiosis is to attach homologous chromosomes to the opposite poles. In mouse oocytes, stable microtubule end-on attachments to kinetochores are not established until hours after spindle assembly, and phosphorylation of kinetochore proteins by Aurora B/C is responsible for the delay. Here we demonstrated that microtubule ends are actively prevented from stable attachment to kinetochores until well after spindle formation in Drosophila melanogaster oocytes. We identified the microtubule catastrophe-promoting complex Sentin-EB1 as a major factor responsible for this delay. Wit

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14

Pottratz, S. T., T. D. Hall, W. M. Scribner, H. N. Jayaram, and V. Natarajan. "P-selectin-mediated attachment of small cell lung carcinoma to endothelial cells." American Journal of Physiology-Lung Cellular and Molecular Physiology 271, no. 6 (1996): L918—L923. http://dx.doi.org/10.1152/ajplung.1996.271.6.l918.

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Small cell lung carcinoma (SCLC) frequently metastasizes early in the course of the disease. P-selectin (P-sel), a cell adhesion molecule expressed on activated platelets and endothelial cells (EC), has previously been demonstrated to mediate binding of platelets to SCLC. We hypothesized that P-sel facilitates attachment of SCLC to EC, acting as an important factor in SCLC metastasis. To test this hypothesis, attachment of H82 cells (SCLC cell line) to EC was quantified. Attachment of H82 cells to 12-O-tetradecanoylphorbol-13-acetate (TPA)-activated EC was increased compared with control EC. I

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15

Turner, S. "Cell attachment on silicon nanostructures." Journal of Vacuum Science & Technology B: Microelectronics and Nanometer Structures 15, no. 6 (1997): 2848. http://dx.doi.org/10.1116/1.589742.

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16

Thompson, H. L., P. D. Burbelo, B. Segui-Real, Y. Yamada, and D. D. Metcalfe. "Laminin promotes mast cell attachment." Journal of Immunology 143, no. 7 (1989): 2323–27. http://dx.doi.org/10.4049/jimmunol.143.7.2323.

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Abstract Tissue mast cells often localize in close proximity to the basement membrane of endothelial cells and increase at sites of inflammation. The reason for this unique tissue distribution is unknown. We report here that both the murine mast cell line PT18 and mouse bone marrow-derived mast cells possess functional receptors for laminin, and exhibit adhesion, spreading and redistribution of histamine-containing granules on a laminin substratum. This adherence is enhanced in the presence of purified IL-3 and can be inhibited by antibodies to laminin and by antibodies to laminin receptors. N

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17

Jiang, Y., J. F. Zhu, F. W. Luscinskas, and D. T. Graves. "MCP-1-stimulated monocyte attachment to laminin is mediated by beta 2-integrins." American Journal of Physiology-Cell Physiology 267, no. 4 (1994): C1112—C1118. http://dx.doi.org/10.1152/ajpcell.1994.267.4.c1112.

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Migration of monocytes to sites of inflammation involves a series of attachments and detachments to extracellular matrix proteins. We examined the capacity of a chemokine, monocyte chemoattractant protein-1 (MCP-1), to regulate attachment of human monocytes to laminin, collagen I, collagen IV, or fibronectin. MCP-1 increased monocyte attachment to laminin in a dose- and time-dependent manner and stimulated a lesser increase to the other matrix proteins. Function-blocking monoclonal antibodies (MAbs) to the integrin beta 2-subunit (CD18), including Fab' fragments and alpha M (CD11b) blocked &gt

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18

Compton, Duane A. "Chromosome orientation." Journal of Cell Biology 179, no. 2 (2007): 179–81. http://dx.doi.org/10.1083/jcb.200709152.

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Precise chromosome segregation during cell division results from the attachment of chromosomes to microtubules emanating from both poles of the spindle apparatus. The molecular machinery involved in establishing and maintaining properly oriented microtubule attachments remains murky. Some clarity is now emerging with the identification of Bod1 (Biorientation Defective 1), a protein that promotes chromosome biorientation by unleashing chromosomes from improperly oriented microtubule attachments.

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19

Yu, Hong-Guo, Michael G. Muszynski, and R. Kelly Dawe. "The Maize Homologue of the Cell Cycle Checkpoint Protein MAD2 Reveals Kinetochore Substructure and Contrasting Mitotic and Meiotic Localization Patterns." Journal of Cell Biology 145, no. 3 (1999): 425–35. http://dx.doi.org/10.1083/jcb.145.3.425.

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We have identified a maize homologue of yeast MAD2, an essential component in the spindle checkpoint pathway that ensures metaphase is complete before anaphase begins. Combined immunolocalization of MAD2 and a recently cloned maize CENPC homologue indicates that MAD2 localizes to an outer domain of the prometaphase kinetochore. MAD2 staining was primarily observed on mitotic kinetochores that lacked attached microtubules; i.e., at prometaphase or when the microtubules were depolymerized with oryzalin. In contrast, the loss of MAD2 staining in meiosis was not correlated with initial microtubule

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20

Gay, Guillaume, Thibault Courtheoux, Céline Reyes, Sylvie Tournier, and Yannick Gachet. "A stochastic model of kinetochore–microtubule attachment accurately describes fission yeast chromosome segregation." Journal of Cell Biology 196, no. 6 (2012): 757–74. http://dx.doi.org/10.1083/jcb.201107124.

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In fission yeast, erroneous attachments of spindle microtubules to kinetochores are frequent in early mitosis. Most are corrected before anaphase onset by a mechanism involving the protein kinase Aurora B, which destabilizes kinetochore microtubules (ktMTs) in the absence of tension between sister chromatids. In this paper, we describe a minimal mathematical model of fission yeast chromosome segregation based on the stochastic attachment and detachment of ktMTs. The model accurately reproduces the timing of correct chromosome biorientation and segregation seen in fission yeast. Prevention of a

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21

Kim, Stella H., Zhigang Li, and David B. Sacks. "E-cadherin-mediated Cell-Cell Attachment Activates Cdc42." Journal of Biological Chemistry 275, no. 47 (2000): 36999–7005. http://dx.doi.org/10.1074/jbc.m003430200.

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22

Pugacheva, Elena N., Fabrice Roegiers, and Erica A. Golemis. "Interdependence of cell attachment and cell cycle signaling." Current Opinion in Cell Biology 18, no. 5 (2006): 507–15. http://dx.doi.org/10.1016/j.ceb.2006.08.014.

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23

DePhillipo, Nicholas N., Gilbert Moatshe, Jorge Chahla, et al. "Quantitative and Qualitative Assessment of the Posterior Medial Meniscus Anatomy: Defining Meniscal Ramp Lesions." American Journal of Sports Medicine 47, no. 2 (2018): 372–78. http://dx.doi.org/10.1177/0363546518814258.

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Background: Meniscal ramp lesions have been defined as a tear of the peripheral attachment of the posterior horn of the medial meniscus (PHMM) at the meniscocapsular junction or an injury to the meniscotibial attachment. Precise anatomic descriptions of these structures are limited in the current literature. Purpose: To quantitatively and qualitatively describe the PHMM and posteromedial capsule anatomy pertaining to the location of a meniscal ramp lesion with reference to surgically relevant landmarks. Study Design: Descriptive laboratory study. Methods: Fourteen male nonpaired fresh-frozen c

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24

Negash, Sewite, Hwai-Shi Wang, Chun Gao, Dolena Ledee, and Peggy Zelenka. "Cdk5 regulates cell-matrix and cell-cell adhesion in lens epithelial cells." Journal of Cell Science 115, no. 10 (2002): 2109–17. http://dx.doi.org/10.1242/jcs.115.10.2109.

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Cdk5 is a member of the cyclin-dependent kinase family, which is expressed predominantly in terminally differentiated neurons. Lower levels of Cdk5 are also found in a wide variety of cell types, including the lens. Although Cdk5 has been shown to play an important role in neuronal migration and neurite outgrowth, its function in non-neuronal cells is not known. Therefore, this study was undertaken to explore the role of Cdk5 in the lens. Results showed that, within the adult mouse lens, Cdk5 was localized to the cytoplasm,especially along the lateral membranes of differentiating primary fiber

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25

Geraghty, Zoë, Christina Barnard, Pelin Uluocak, and Ulrike Gruneberg. "The association of Plk1 with the astrin–kinastrin complex promotes formation and maintenance of a metaphase plate." Journal of Cell Science 134, no. 1 (2020): jcs251025. http://dx.doi.org/10.1242/jcs.251025.

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ABSTRACTErrors in mitotic chromosome segregation can lead to DNA damage and aneuploidy, both hallmarks of cancer. To achieve synchronous error-free segregation, mitotic chromosomes must align at the metaphase plate with stable amphitelic attachments to microtubules emanating from opposing spindle poles. The astrin–kinastrin (astrin is also known as SPAG5 and kinastrin as SKAP) complex, also containing DYNLL1 and MYCBP, is a spindle and kinetochore protein complex with important roles in bipolar spindle formation, chromosome alignment and microtubule–kinetochore attachment. However, the molecul

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HU, Jin-Song, Nai-Zheng DING, Jun-Lin TENG, and Jian-Guo CHEN. "FAAP is Involved in Cell Attachment*." PROGRESS IN BIOCHEMISTRY AND BIOPHYSICS 36, no. 1 (2009): 88–94. http://dx.doi.org/10.3724/sp.j.1206.2008.00344.

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27

Govind, Rakesh, and Juan Gonclaves. "Enhanced Biofiltration Using Cell Attachment Promoters." Proceedings of the Water Environment Federation 2008, no. 4 (2008): 702–28. http://dx.doi.org/10.2175/193864708788808177.

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28

Francis, R., and R. H. Waterston. "Muscle cell attachment in Caenorhabditis elegans." Journal of Cell Biology 114, no. 3 (1991): 465–79. http://dx.doi.org/10.1083/jcb.114.3.465.

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In the nematode Caenorhabditis elegans, the body wall muscles exert their force on the cuticle to generate locomotion. Interposed between the muscle cells and the cuticle are a basement membrane and a thin hypodermal cell. The latter contains bundles of filaments attached to dense plaques in the hypodermal cell membranes, which together we have called a fibrous organelle. In an effort to define the chain of molecules that anchor the muscle cells to the cuticle we have isolated five mAbs using preparations enriched in these components. Two antibodies define a 200-kD muscle antigen likely to be

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29

HIRANO, Yshiaki. "Cell Attachment Activity Peptide-Based Biomaterials." Journal of The Adhesion Society of Japan 38, no. 3 (2001): 97–103. http://dx.doi.org/10.11618/adhesion.38.97.

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30

Sakai, Hideaki, Yasuhiro Kobayashi, Eiko Sakai, and Yuzo Kato. "Cell attachment regulates survival of osteoclasts." Japanese Journal of Pharmacology 79 (1999): 277. http://dx.doi.org/10.1016/s0021-5198(19)35123-6.

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31

Goncalves, Juan J., and Rakesh Govind. "Enhanced Biofiltration Using Cell Attachment Promotors." Environmental Science & Technology 43, no. 4 (2009): 1049–54. http://dx.doi.org/10.1021/es801156x.

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32

Halperin, Stephen, and Jean-Michel Lemaire. "The fibre of a cell attachment." Proceedings of the Edinburgh Mathematical Society 38, no. 2 (1995): 295–311. http://dx.doi.org/10.1017/s001309150001909x.

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In view of understanding the Hopf algebra structure of the loop space homology in terms of H*(ΩE) and the map f, we consider the homotopy fibre F of the inclusion map In [15], the case when H*(Ωω) is surjective (the “inert” case) was studied, and in [11] a weaker condition, called “lazy”, was considered. Here we give several new characterizations of inert and lazy cell attachments in terms of properties of F. We also show how these results extend to the case of the mapping cone of an arbitrary map f: W→E.

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33

Custódio, C. A., V. San Miguel-Arranz, R. A. Gropeanu, et al. "Photopatterned Antibodies for Selective Cell Attachment." Langmuir 30, no. 33 (2014): 10066–71. http://dx.doi.org/10.1021/la502688h.

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34

Pierschbacher, M. D., E. G. Hayman, and E. Ruoslahti. "The cell attachment determinant in fibronectin." Journal of Cellular Biochemistry 28, no. 2 (1985): 115–26. http://dx.doi.org/10.1002/jcb.240280205.

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35

Cheng, Nan, and Xudong Cao. "Photosensitive chitosan to control cell attachment." Journal of Colloid and Interface Science 361, no. 1 (2011): 71–78. http://dx.doi.org/10.1016/j.jcis.2011.05.045.

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36

Jiang, Bo, Jian Yang, Nahla Rahoui, Nadia Taloub, and Yu Dong Huang. "Functional polymer materials affecting cell attachment." Advances in Colloid and Interface Science 250 (December 2017): 185–94. http://dx.doi.org/10.1016/j.cis.2017.09.002.

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37

Fuhrmann, Alexander, and Adam J. Engler. "The Cytoskeleton Regulates Cell Attachment Strength." Biophysical Journal 109, no. 1 (2015): 57–65. http://dx.doi.org/10.1016/j.bpj.2015.06.003.

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38

Adams, J. C., and J. Lawler. "Diverse mechanisms for cell attachment to platelet thrombospondin." Journal of Cell Science 104, no. 4 (1993): 1061–71. http://dx.doi.org/10.1242/jcs.104.4.1061.

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Thrombospondin-1 is a component of the extracellular matrix which is thought to play important roles in cell migration and proliferation, during embryogenesis and wound repair. To understand the basis for these activities, we are mapping the regions of the molecule with cell adhesive activity. Here, we use antagonists of specific cell binding sites, adhesion-perturbing thrombospondin monoclonal antibodies and proteolytic fragments of platelet thrombospondin, to investigate the adhesive mechanisms used by G361 melanoma cells, human intestinal smooth muscle cells (HISM), epidermal keratinocytes

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39

Tsugawa, Hitoshi, Humie Ito, Miho Ohshima, and Yoshio Okawa. "Cell adherence-promoted activity of Plesiomonas shigelloides GroEL." Journal of Medical Microbiology 56, no. 1 (2007): 23–29. http://dx.doi.org/10.1099/jmm.0.46766-0.

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Previously, it has been demonstrated that the invasion of Caco-2 cells by Plesiomonas shigelloides induces apoptotic cell death. Therefore, the attachment to and colonization of eukaryotic intestinal host cells by P. shigelloides are important steps in causing pathogenicity. In this study, the participation of P. shigelloides GroEL in the attachment of P. shigelloides was examined. The groESL operon of P. shigelloides was isolated by PCR. The nucleotide sequence of the groESL operon of P. shigelloides revealed two ORFs of 294 nucleotides for groES and 1647 nucleotides for groEL. Cell fractiona

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Campbell, AD, MW Long, and MS Wicha. "Developmental regulation of granulocytic cell binding to hemonectin." Blood 76, no. 9 (1990): 1758–64. http://dx.doi.org/10.1182/blood.v76.9.1758.1758.

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Abstract Hemonectin (HN), a component of the bone marrow (BM) extracellular matrix which promotes adhesion of cells in the granulocytic lineage, was purified to near homogeneity and tested for its ability to mediate attachment of normal and leukemic cells of granulocytic lineage. Purified HN immobilized on plastic substrates promoted serum-free attachment of normal granulocyte/macrophage progenitor cells (CFC-GM), using an in situ attachment assay in which cell attachment is inhibited by specific polyclonal antisera. When unfractionated BM cells were allowed to attach to purified HN and staine

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Campbell, AD, MW Long, and MS Wicha. "Developmental regulation of granulocytic cell binding to hemonectin." Blood 76, no. 9 (1990): 1758–64. http://dx.doi.org/10.1182/blood.v76.9.1758.bloodjournal7691758.

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Hemonectin (HN), a component of the bone marrow (BM) extracellular matrix which promotes adhesion of cells in the granulocytic lineage, was purified to near homogeneity and tested for its ability to mediate attachment of normal and leukemic cells of granulocytic lineage. Purified HN immobilized on plastic substrates promoted serum-free attachment of normal granulocyte/macrophage progenitor cells (CFC-GM), using an in situ attachment assay in which cell attachment is inhibited by specific polyclonal antisera. When unfractionated BM cells were allowed to attach to purified HN and stained in situ

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42

Watanabe, Yoshinori. "Monopolar attachment by Polo." Nature Cell Biology 5, no. 5 (2003): 379–82. http://dx.doi.org/10.1038/ncb0503-379.

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43

Deng, Hua, John B. Bell, and Andrew J. Simmonds. "Vestigial Is Required during Late-Stage Muscle Differentiation in Drosophila melanogaster Embryos." Molecular Biology of the Cell 21, no. 19 (2010): 3304–16. http://dx.doi.org/10.1091/mbc.e10-04-0364.

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The somatic muscles of Drosophila develop in a complex pattern that is repeated in each embryonic hemi-segment. During early development, progenitor cells fuse to form a syncytial muscle, which further differentiates via expression of muscle-specific factors that induce specific responses to external signals to regulate late-stage processes such as migration and attachment. Initial communication between somatic muscles and the epidermal tendon cells is critical for both of these processes. However, later establishment of attachments between longitudinal muscles at the segmental borders is larg

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Becker, S., G. Pasca, D. Strumpf, L. Min, and T. Volk. "Reciprocal signaling between Drosophila epidermal muscle attachment cells and their corresponding muscles." Development 124, no. 13 (1997): 2615–22. http://dx.doi.org/10.1242/dev.124.13.2615.

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Directed intercellular interactions between distinct cell types underlie the basis for organogenesis during embryonic development. This paper focuses on the establishment of the final somatic muscle pattern in Drosophila, and on the possible cross-talk between the myotubes and the epidermal muscle attachment cells, occurring while both cell types undergo distinct developmental programs. Our findings suggest that the stripe gene is necessary and sufficient to initiate the developmental program of epidermal muscle attachment cells. In stripe mutant embryos, these cells do not differentiate corre

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45

Andreozzi, Elisa, and Gaylen A. Uhlich. "PchE Regulation of Escherichia coli O157:H7 Flagella, Controlling the Transition to Host Cell Attachment." International Journal of Molecular Sciences 21, no. 13 (2020): 4592. http://dx.doi.org/10.3390/ijms21134592.

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Shiga toxins and intimate adhesion controlled by the locus of enterocyte effacement are major enterohemorrhagic Escherichia coli (EHEC) virulence factors. Curli fimbriae also contribute to cell adhesion and are essential biofilm components. The transcriptional regulator PchE represses the expression of curli and their adhesion to HEp-2 cells. Past studies indicate that pchE also represses additional adhesins that contribute to HEp-2 cell attachment. In this study, we tested for pchE regulation of several tissue adhesins and their regulators. Three adhesin-encoding genes (eae, lpfA1, fliC) and

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46

Schuldt, Alison. "Formin' an attachment." Nature Reviews Molecular Cell Biology 12, no. 5 (2011): 280. http://dx.doi.org/10.1038/nrm3110.

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47

Wimbish, Robert T., Keith F. DeLuca, Jeanne E. Mick, et al. "The Hec1/Ndc80 tail domain is required for force generation at kinetochores, but is dispensable for kinetochore–microtubule attachment formation and Ska complex recruitment." Molecular Biology of the Cell 31, no. 14 (2020): 1453–73. http://dx.doi.org/10.1091/mbc.e20-05-0286.

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This study demonstrates that the Ska complex and the Hec1 tail domain contribute to kinetochore–microtubule attachment regulation independently. The Hec1 tail is shown to be dispensable for Ska complex recruitment to kinetochores and for formation of kinetochore–microtubule attachments, but required for wild-type force generation at kinetochores.

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Roberts, D. D., J. A. Sherwood, and V. Ginsburg. "Platelet thrombospondin mediates attachment and spreading of human melanoma cells." Journal of Cell Biology 104, no. 1 (1987): 131–39. http://dx.doi.org/10.1083/jcb.104.1.131.

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Human platelet thrombospondin adsorbed on plastic promotes attachment and spreading of human G361 melanoma cells. Attachment is rapid, and spreading is maximal by 90 min with 60-90% of the attached cells spread. In contrast, thrombospondin promotes attachment but not spreading of human C32 melanoma cells, which attach and spread only on laminin substrates. The specificity of these interactions and the regions of the thrombospondin molecule involved in attachment and spreading were examined using proteolytic fragments of thrombospondin and by inhibition studies. The sulfated fucan, fucoidan, an

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Fong, Kimberly K., Krishna K. Sarangapani, Erik C. Yusko, et al. "Direct measurement of the strength of microtubule attachment to yeast centrosomes." Molecular Biology of the Cell 28, no. 14 (2017): 1853–61. http://dx.doi.org/10.1091/mbc.e17-01-0034.

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Centrosomes, or spindle pole bodies (SPBs) in yeast, are vital mechanical hubs that maintain load-bearing attachments to microtubules during mitotic spindle assembly, spindle positioning, and chromosome segregation. However, the strength of microtubule-centrosome attachments is unknown, and the possibility that mechanical force might regulate centrosome function has scarcely been explored. To uncover how centrosomes sustain and regulate force, we purified SPBs from budding yeast and used laser trapping to manipulate single attached microtubules in vitro. Our experiments reveal that SPB–microtu

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Belusa, Roger, Oleg Aizman, Ronnie M. Andersson, and Anita Aperia. "Changes in Na+-K+-ATPase activity influence cell attachment to fibronectin." American Journal of Physiology-Cell Physiology 282, no. 2 (2002): C302—C309. http://dx.doi.org/10.1152/ajpcell.00117.2001.

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Most vital cellular functions are dependent on a fine-tuned regulation of intracellular ion homeostasis. Here we have demonstrated, using COS cells that were untransfected or transfected with wild-type rat ouabain-resistant Na+-K+-ATPase, that partial inhibition of Na+-K+-ATPase has a dramatic influence on cell attachment to fibronectin. Ouabain dose-dependently decreased attachment in untransfected cells and in cells expressing wild-type Na+-K+-ATPase, but not in cells expressing ouabain-insensitive Na+-K+-ATPase, whereas inhibition of Na+-K+-ATPase by lowering extracellular K+ concentration

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