Relevant bibliographies by topics / Cell biology

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cell biology'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2025

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Journal articles on the topic "Cell biology"

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LAZARIDES, E. "Modern Cell Biology: Molecular Cell Biology." Science 234, no. 4782 (December 12, 1986): 1448. http://dx.doi.org/10.1126/science.234.4782.1448.

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Yamashita, Yukiko M. "Cell biology of stem cells: studying stem cells at the level of cell biology and studying cell biology using stem cells." Molecular Biology of the Cell 29, no. 24 (November 26, 2018): 2912. http://dx.doi.org/10.1091/mbc.e18-09-0596.

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Gordon, MY, F. Dazzi, SB Marley, JL Lewis, D. Nguyen, FH Grand, RJ Davidson, and JM Goldman. "Cell biology of CML cells." Leukemia 13, S1 (April 1999): S65—S71. http://dx.doi.org/10.1038/sj.leu.2401281.

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Purnell, B. A. "CELL BIOLOGY: Long-Lived Cells." Science 310, no. 5748 (October 28, 2005): 591c. http://dx.doi.org/10.1126/science.310.5748.591c.

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Jaffe, Eric A. "Cell biology of endothelial cells." Human Pathology 18, no. 3 (March 1987): 234–39. http://dx.doi.org/10.1016/s0046-8177(87)80005-9.

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Ogushi, Fumiko, and Hiroshi Kori. "3P277 Dependence of cell differentiation ratio on cell-cell interaction and noise(24. Mathematical biology,Poster)." Seibutsu Butsuri 53, supplement1-2 (2013): S257. http://dx.doi.org/10.2142/biophys.53.s257_6.

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BURGESS, D. R. "Cell Biology: Cell Motility." Science 234, no. 4775 (October 24, 1986): 492. http://dx.doi.org/10.1126/science.234.4775.492.

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Neff, Randi. "Cell Biology." American Biology Teacher 74, no. 9 (November 1, 2012): 656–57. http://dx.doi.org/10.1525/abt.2012.74.9.11c.

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Stankus, Tony. "Cell Biology." Serials Librarian 27, no. 2-3 (April 8, 1996): 79–86. http://dx.doi.org/10.1300/j123v27n02_06.

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Hashimoto, Takashi, and Dirk Inzé. "Cell biology." Current Opinion in Plant Biology 6, no. 6 (December 2003): 517–19. http://dx.doi.org/10.1016/j.pbi.2003.09.016.

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Dissertations / Theses on the topic "Cell biology"

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Einarsson, Elin. "Comparative Cell Biology in Diplomonads." Doctoral thesis, Uppsala universitet, Institutionen för cell- och molekylärbiologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-264541.

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The diplomonads are a diverse group of eukaryotic flagellates found in microaerophilic and anaerobic environments. The most studied diplomonad is the intestinal parasite Giardia intestinalis, which infects a variety of mammals and cause diarrheal disease. Less is known about Spironucleus salmonicida, a parasite of salmonid fish, known to cause systemic infections with high mortality. We created a transfection system for S. salmonicida to study cellular functions and virulence in detail (Paper I). The system was applied to explore the mitochondrion-related organelle (MRO) in S. salmonicida. We showed that S. salmonicida possesses a hydrogenosome (Paper II) with a higher metabolic capacity than the corresponding MRO of Giardia, the mitosome. Evolutionary analysis of key hydrogenosomal proteins showed ancient origin, indicating their presence in the ancestral diplomonad and subsequent loss in Giardia. Annexins are of evolutionary interest since these proteins are found across all kingdoms. Annexin-like proteins are intriguingly expanded into multigene families in Giardia and Spironucleus. The annexins of S. salmonicida were characterized (Paper III) with distinct localizations to various cellular structures, including a putative adhesion structure anterior in the cell. The disease-causing Giardia trophozoites differentiate into infectious cysts, a process essential for transmission and virulence of the parasite. Cysts are often spread via contaminated water and exposed to environmental stressors, such as UV irradiation. We studied the survival and transcriptional response to this stress factor (Paper IV) and results showed the importance of active DNA replication machinery for parasite survival after DNA damage. In addition, we studied transcriptional changes along the trajectory of encystation (Paper V), which revealed a coordinated cascade of gene regulation. This was observed for the entire transcriptome as well as putative regulators. Large transcriptional changes appeared late in the process with the majority of differentially regulated genes encoding hypothetical proteins. We studied the localizations of several of these to gain information of their possible function. To conclude, the diplomonads are complex eukaryotic microbes with cellular processes adjusted to match their life styles. The work in this thesis has provided insight of their adaptations, differences and similarities, but also new interesting leads for future studies of diplomonad biology and virulence.
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Lewis, Beverley Anne. "Cell biology of rat spermatozoa." Thesis, University of Edinburgh, 2001. http://hdl.handle.net/1842/23087.

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The purpose of the research presented in this thesis was to investigate the cell biology of rat spermatozoa. An additional aim was to utilise the knowledge obtained to aid the development of in vitro functional tests for the assessment of rat sperm fertility and identify potential markers of normal epididymal maturation. As mammalian spermatozoa migrate through the epididymis, they acquire the potential for fertilisation, characterised by the acquisition of the ability to express co-ordinated movement and the competence to undergo capacitance. The mechanisms by which epididymal maturation confers upon mammalian spermatozoa the potential to capacitate is poorly understood. These studies investigated the impact of epididymal maturation on the signal transduction pathways regulating tyrosine phosphorylation using the laboratory rat as an animal model, since this signal transduction pathway is thought to be central to the attainment of a capacitated state and expression of hyperactivated motility, both of which are prerequisites for fertilisation. Western Blot and immunocytochemical analysis demonstrated that epididymal maturation is associated with a progressive loss in phosphotyrosine expression located to the acrosomal domain. These differences in phosphotyrosine expression between caput and caudal epididymal spermatozoa appeared to reflect the normal in vivo situation. In addition, epididymal maturation of rat spermatozoa is also associated with an acquired competence to respond to high levels of intracellular cAMP by phoshorylating tyrosine residues on the sperm tail. Epididymal maturation also led to unique differences in the generation of reactive oxygen species (ROS) by spermatozoa obtained from the caput and caudal regions of the epididymis. Spermatozoa from both regions of the epididymis spontaneously generated equal levels of O2 whereas only mature caudal spermatozoa generated significant levels of H2O2. In contrast, although both caput and caudal spermatozoa generated increased O2-. in response to NADPH, induced levels were significantly greater in the immature caput cells.
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Yu, Yang. "Lipidomics Investigations in Cell Biology." Doctoral thesis, Università degli studi di Trento, 2014. https://hdl.handle.net/11572/368594.

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Cell membrane is the biological barrier serving as both territorial defense and the communication hinge for the interior of cell from its surroundings. As building blocks of cellular membranes and also precursor for second messengers, a variety of lipids play essential roles in cellular membrane dynamics as well as important functions such as cell proliferation, apoptosis, signal transduction and membrane trafficking modulation. Lipidomics, representing the systematic and integrative studies of diversified lipids (lipidome) in a biological system, is an emerging yet rapid developing field and hence requires advanced and complementary analytical techniques as well as multiple statistical tools. Our development of reliable analytical methodology (the advanced Mass Spectrometric and high-resolution NMR techniques) and application of multiple statitistical approaches (multivariate data analysis and univariate t-test) enable us to achieve these comprehensive understandings. We have investigated, first of all, the effects induced by hypoxia on cervical cancer derived cells (HeLa cells) to see how and how much the changes in phospholipids profile are able to get light into the targeted biological problem (hypoxia) and provide a preliminary insight into the underlying mechanisms. We found that hypoxia stimulation dramatically reduced the total amount of cellular phosphoinositols (PI) but prominently increased the amount of lyso phosphocholines (lyso-PC) and lyso phosphoethanolamines (lyso-PE). Moreover, our studies suggested the polyunsaturated phospholipids species as stronger biomarkers upon hypoxia treatment. The evaluation of changes in the average unsaturation index (UI) of the membrane lipids acyl chains revealed that UI slightly increased in several lipid classes, thus affecting membrane fluidity and further membrane-dependent functions. The plausible mechanisms by HeLa cells to adapt to hypoxia conditions are briefly reported as well. We have also conducted the comparative lipidomic studies of urothelial cancer cell line RT4 (a model system of a benign tumor) and T24 (a model system of a metastatic tumor) aiming to reveal probable roles and relevant differential changes of membrane lipids with respect to urinary bladder metastasis progress. Significant changes of lipids metabolism were found to correlate with urothelial nonmetastatic and metastatic cell models. The most remarkable finding was that the malignant cell type (T24) showed a strong decrease of ether PC species complemented by a sharp increase of the length and the average unsaturation number of lipids acyl chains. Ceramide-based sphinglipids also showed altered profiles in these two cell types. Such analyses suggest a certain significant re-organization of cellular membrane in malignant cell transformation, involving variations in compositional lipid structures and possible signaling transduction pathways. Observations of such reduction of the 1-alkyl PC species and the chain shortening of lipid species might serve as a tool in urinary bladder cancer intervention.
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Yu, Yang. "Lipidomics Investigations in Cell Biology." Doctoral thesis, University of Trento, 2014. http://eprints-phd.biblio.unitn.it/1294/1/Yang_Yu__Lipidomics_Investigations_in_Cell_Biology.pdf.

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Cell membrane is the biological barrier serving as both territorial defense and the communication hinge for the interior of cell from its surroundings. As building blocks of cellular membranes and also precursor for second messengers, a variety of lipids play essential roles in cellular membrane dynamics as well as important functions such as cell proliferation, apoptosis, signal transduction and membrane trafficking modulation. Lipidomics, representing the systematic and integrative studies of diversified lipids (lipidome) in a biological system, is an emerging yet rapid developing field and hence requires advanced and complementary analytical techniques as well as multiple statistical tools. Our development of reliable analytical methodology (the advanced Mass Spectrometric and high-resolution NMR techniques) and application of multiple statitistical approaches (multivariate data analysis and univariate t-test) enable us to achieve these comprehensive understandings. We have investigated, first of all, the effects induced by hypoxia on cervical cancer derived cells (HeLa cells) to see how and how much the changes in phospholipids profile are able to get light into the targeted biological problem (hypoxia) and provide a preliminary insight into the underlying mechanisms. We found that hypoxia stimulation dramatically reduced the total amount of cellular phosphoinositols (PI) but prominently increased the amount of lyso phosphocholines (lyso-PC) and lyso phosphoethanolamines (lyso-PE). Moreover, our studies suggested the polyunsaturated phospholipids species as stronger biomarkers upon hypoxia treatment. The evaluation of changes in the average unsaturation index (UI) of the membrane lipids acyl chains revealed that UI slightly increased in several lipid classes, thus affecting membrane fluidity and further membrane-dependent functions. The plausible mechanisms by HeLa cells to adapt to hypoxia conditions are briefly reported as well. We have also conducted the comparative lipidomic studies of urothelial cancer cell line RT4 (a model system of a benign tumor) and T24 (a model system of a metastatic tumor) aiming to reveal probable roles and relevant differential changes of membrane lipids with respect to urinary bladder metastasis progress. Significant changes of lipids metabolism were found to correlate with urothelial nonmetastatic and metastatic cell models. The most remarkable finding was that the malignant cell type (T24) showed a strong decrease of ether PC species complemented by a sharp increase of the length and the average unsaturation number of lipids acyl chains. Ceramide-based sphinglipids also showed altered profiles in these two cell types. Such analyses suggest a certain significant re-organization of cellular membrane in malignant cell transformation, involving variations in compositional lipid structures and possible signaling transduction pathways. Observations of such reduction of the 1-alkyl PC species and the chain shortening of lipid species might serve as a tool in urinary bladder cancer intervention.
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Bair, Elisabeth Laurine. "Cell-cell and cell-matrix interactions involved in cancer invasion." Diss., The University of Arizona, 2004. http://hdl.handle.net/10150/280673.

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In order for a cancer to metastasize, it must first invade through the basement membrane that surrounds it, invade blood vessels and travel through the bloodstream to a new location where it extravasates the vessel and begins growing at the new site. The mechanisms by which a cancer becomes able to invade and metastasize are currently under intense study. Interactions of the cell with its environment via cell-cell contacts, extracellular matrix (ECM) interactions, and circulating proteins are thought to play a major role in signaling for these invasive processes to occur. Upregulation of proteolytic enzymes, such as the matrix metalloproteases, is suspected of being involved in the metastatic process. Cell-cell and cell-matrix contacts via integrins and cadherins are necessary for upregulation of the matrix metalloprotease matrilysin in oral squamous cell carcinoma. In an effort to identify the factors involved in upregulation of matrilysin expression detected in a co-culture of oral squamous cell carcinoma (SCC) cells and fibroblast cells, a coculture model designed to represent the actual tumor environment, we show that inhibition of beta1 integrin, E-cadherin, and N-cadherin with blocking antibodies thoroughly decreases the induction of matrilysin in the co-culture model. This demonstrates that interactions between cancer cells and normal cells surrounding them may allow for invasion and metastasis. The protein 90K may also play a role in the invasive process of prostate cancer. It functions as an immune modulator upregulating cytokines that induce MMPs and we show that it can induce matrilysin expression in prostate cancer cells. It also functions in cell aggregation, which can help cells survive during metastasis. For this reason, expression of 90K in prostate cancer, which we examined, may be indicative of aggressive disease, making 90K a potentially useful tumor marker. Cell-matrix contacts are also important for the transmembrane matrix metalloprotease MT1-MMP cleavage of laminin-10. We demonstrate that recombinant MT1-MMP is able to cleave human laminin-10 into four distinct products. This allows for prostate cancer cell migration on laminin-10 coated substrates, which can be inhibited with the addition of MT1-MMP antisense oligonucleotides. Ln-10 cleavage also occurs in vivo in human prostate tissue, indicating that this cell-matrix interaction has in vivo relevance in human prostate cancer.
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Zhang, Yuan. "Cell-cell interactions and Gossypol's effects on cell functions of primary cultured Human Breast Epthelial, Stromal and Adipose Stromal cells /." The Ohio State University, 1999. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488187763845791.

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Agüera-González, Sonia. "Cell biology on NKG2D ligands and NK cell recognition." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609348.

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Camacho, Diogo Mayo. "In silico cell biology and biochemistry: a systems biology approach." Diss., Virginia Tech, 2007. http://hdl.handle.net/10919/27960.

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In the post-"omic" era the analysis of high-throughput data is regarded as one of the major challenges faced by researchers. One focus of this data analysis is uncovering biological network topologies and dynamics. It is believed that this kind of research will allow the development of new mathematical models of biological systems as well as aid in the improvement of already existing ones. The work that is presented in this dissertation addresses the problem of the analysis of highly complex data sets with the aim of developing a methodology that will enable the reconstruction of a biological network from time series data through an iterative process. The first part of this dissertation relates to the analysis of existing methodologies that aim at inferring network structures from experimental data. This spans the use of statistical tools such as correlations analysis (presented in Chapter 2) to more complex mathematical frameworks (presented in Chapter 3). A novel methodology that focuses on the inference of biological networks from time series data by least squares fitting will then be introduced. Using a set of carefully designed inference rules one can gain important information about the system which can aid in the inference process. The application of the method to a data set from the response of the yeast Saccharomyces cerevisiae to cumene hydroperoxide is explored in Chapter 5. The results show that this method can be used to generate a coarse-level mathematical model of the biological system at hand. Possible developments of this method are discussed in Chapter 6.
Ph. D.
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Buffa, Laura. "Cell Biology of the ICA69 protein family in Neurosecretory cells." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2007. http://nbn-resolving.de/urn:nbn:de:swb:14-1174057636463-96361.

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In type 1 diabetes (T1D), an autoimmune disease, autoantibodies are preferentially directed against proteins associated with Golgi and post-Golgi secretory vesicles, including insulin secretory granules and synaptic-like microvesicles. Thus, the study of beta-cell autoantigens with yet unknown function may provide novel insight into the secretory machinery of beta-cells and led to the discovery of novel pathways. Islet cell autoantigen of 69 kDa (ICA69) is a T1D autoantigen. It is a cytosolic protein of still unknown function. An impairment in neurotransmitter release upon mutation of its homologue in C. elegans suggests, however, an involvement of ICA69 in neurosecretion. Interestingly, ICA69 contains a BAR domain, present in several proteins involved in intracellular transport. The BAR domain functions as a dimerization motif, provides a general binding interface for different types of GTPases, and is a membrane binding/bending module. Its presence in ICA69 is a further hint supporting the putative involvement of ICA69 in intracellular membrane trafficking. The first part of this thesis was concerned with the characterization of ICA69, and the elucidation of its role in membrane traffic in pancreatic beta-cells. ICA69 was shown to be enriched in the perinuclear region, where also markers of the Golgi region are found. ICA69 was shown to interact with several membrane lipids, preferentially with PI(4)P, enriched on the Golgi complex. During the course of this thesis a combination of biochemical and imaging techniques were applied to investigate the interaction between ICA69 and Rab2, a small GTPase associated with the intermediate compartment and involved in the trafficking between the ER and the Golgi complex. ICA69 was shown to co-immunoprecipitate with Rab2 from INS-1 cells extracts. GST-pull down assays demonstrated that this interaction is GTP-dependent. Furthermore, confocal microscopy indicated that ICA69 and Rab2 extensively colocalize in particulate structures throughout the cytoplasm. Immunocytochemistry and subcellular fractionation experiments suggested that Rab2 recruits ICA69 to membranes. Functional studies indicated that ICA69 over-expression in INS-1 cells has effects that resemble, and in some cases amplify those observed upon Rab2 over-expression. Specifically, it impairs the trafficking between ER and Golgi, measured through the appearance and the conversion of the pro-form of ICA512 in the mature form of the protein. Moreover, it correlates with a redistribution of the beta-COP subunit of the coatomer, participating in the early secretory pathway, between membrane-bound compartments and the cytosol and it reduces stimulated insulin secretion. The data reported in this thesis conclusively point to ICA69 as a novel Rab2 effector, and may therefore contribute to the elucidation the yet poorly understood mechanism of action of Rab2 in the secretory pathway. The second part of the thesis was devoted to the study of an ICA69 paralogue gene, called ICA69-RP. Similarly to ICA69, ICA69-RP mRNA was shown to be primarily present in tissues such as brain and pancreatic islets, showing the expression pattern of a gene preferentially expressed in neuroendocrine cells. Unlike ICA69, however, and similar to other genes associated with the secretory machinery of beta-cells, ICA69-RP appeared to be glucose regulated, as shown by a 1.55 fold increase in mRNA levels upon stimulation of the cells with 25 mM glucose for two hours.Glucose stimulation of beta-cells prompts the activation of post-transcripional mechanisms which quickly up-regulate the expression of secretory granule genes and consequently renew granule stores. The increased expression of ICA69-RP upon glucose stimulation of cells may be part of this process. Unfortunately, all attempts to elucidate the intracellular localization of endogenous ICA69-RP failed, and it was not possible to obtain significant insights about its localization by over-expressing a fusion protein between ICA69-RP and GFP. Unlike other paralogues containing the BAR domain, such as amphiphysin 1 and 2 or Rvs167p and Rvs161p, ICA69 and ICA69-RP were shown not to form heterodimers. Furthermore, ICA69-RP did not show any interaction with Rab2 or Rab1, involved in the anterograde transport between ER and Golgi. Thus, its physiological role remains to be investigated.
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Edwards, T. L. "The molecular cell biology of spartin." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.598783.

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This thesis has focused on the protein spartin, which is mutated in a form of autosomal recessive hereditary spastic paraplegia (HSP) called Troyer syndrome. Upon commencement of this project little was known about the likely mechanism of axonopathy in spartin HSP, indeed there was considerable disagreement in the literature as to its precise subcellular localisation. However, a putative role for spartin in endocytosis was proposed due to sequence homology identified with several proteins known to function in this area. This suggested that spartin belonged to an enlarging group of HSP proteins with membrane traffic and transport-related roles. This thesis had three primary aims: to clarify the subcellular localisation of endogenous spartin, to identify proteins that interacted with spartin, and to investigate a possible functional role(s) for spartin. Spartin was found to localise predominantly in the cytoplasm, with a cystosolic pool that was recruited to endosomes. Additionally, a proportion of spartin was found in mitochondria. Furthermore, spartin was shown to undergo multiple monoubiquitination and to interact with ubiquitin, two ankyrin repeat domain-13 family members, and also with the E3 ubiquitin ligase AIP4. Functional assays suggest that spartin is an inhibitor of epidermal growth factor receptor degradation, and negatively regulates bone morphogenetic protein (BMP) signalling. A putative role in BMP signalling is interesting because this pathway is emerging as an important regulator of distal axonal morphology and function, and has been implicated in the pathogenesis of another endosomal HSP protein, NIPA1. This suggests that altered BMP signalling may be a key pathological component of spartin HSP.
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Books on the topic "Cell biology"

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Smith, C. A., and E. J. Wood. Cell Biology. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0441-8.

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Assmann, Sarah, and Bo Liu, eds. Cell Biology. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7881-2.

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Smith, C. A. Cell biology. London: Chapman & Hall, 1992.

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1947-, King B., ed. Cell biology. London: Allen & Unwin, 1986.

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C, Earnshaw William, and Lippincott-Schwartz Jennifer, eds. Cell biology. 2nd ed. Philadelphia: Saunders/Elsevier, 2008.

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J, Wood E., ed. Cell biology. 2nd ed. Cheltenham: Stanley Thornes, 1999.

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1941-, Wood Edward J., ed. Cell biology. London: Chapman & Hall, 1992.

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University of Toronto. Dept. of Biology., ed. Molecular cell biology: Biology 206F. [Toronto]: Erindale College, University of Toronto at Mississauga, 1999.

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Erindale College. Dept. of Biology., ed. Molecular cell biology: Biology 202S. [Mississauga]: Erindale College, University of Toronto, 1997.

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University of Toronto. Dept. of Biology., ed. Molecular cell biology: Biology 206F. [Toronto]: Erindale College, University of Toronto at Mississauga, 2000.

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Book chapters on the topic "Cell biology"

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Acconcia, Filippo, and Rakesh Kumar. "Cell Biology." In Encyclopedia of Cancer, 1–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_992-2.

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Boon, Mathilde E., and Johanna S. Drijver. "Cell Biology." In Routine Cytological Staining Techniques, 3–15. London: Macmillan Education UK, 1986. http://dx.doi.org/10.1007/978-1-349-18250-3_1.

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Kotyk, Arnošt. "Cell Biology." In Quantities, Symbols, Units, and Abbreviations in the Life Sciences, 63–64. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-206-7_9.

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Acconcia, Filippo, and Rakesh Kumar. "Cell Biology." In Encyclopedia of Cancer, 892–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_992.

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O’Keefe, Stephen J. D. "Cell Biology." In The Principles and Practice of Nutritional Support, 3–8. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1779-2_1.

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Hackstadt, Ted. "Cell Biology." In Chlamydia, 101–38. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555818203.ch5.

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McComas, William F. "Cell Biology." In Teaching Biology in Schools, 48–61. New York : Routledge, 2018. | Series: Teaching and learning in science series: Routledge, 2018. http://dx.doi.org/10.4324/9781315110158-5.

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Schmid, Amy K., and Nitin S. Baliga. "Prokaryotic Systems Biology." In Cell Engineering, 395–423. Dordrecht: Springer Netherlands, 2007. http://dx.doi.org/10.1007/1-4020-5252-9_12.

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Priyadarshini, Anjali, and Prerna Pandey. "Cell." In Molecular Biology, 1–28. Toronto ; New Jersey : Apple Academic Press, 2018.: Apple Academic Press, 2018. http://dx.doi.org/10.1201/b22354-1.

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Hexner, Elizabeth O., and Stephen G. Emerson. "Stem Cell Biology." In Hematopoietic Stem Cell Transplantation, 3–18. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-438-4_1.

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Conference papers on the topic "Cell biology"

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Inose, Tomoko. "Plasmonic nanowire based intracellular material delivery." In JSAP-Optica Joint Symposia, 16p_B4_1. Washington, D.C.: Optica Publishing Group, 2024. https://doi.org/10.1364/jsapo.2024.16p_b4_1.

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The technology for introducing biomolecules such as proteins and DNA into cells is widely used as a method to artificially control cell functions, ranging from basic biology to the pharmaceutical field. Methods employing liposomes, viral vectors, and the electroporation have been currently widely used, although these methods show low introduction efficiency or cell toxicity to some cell types. Another method for introducing biomolecules into cells is microinjection.1) This method physically introduces micro/nano needles directly into the cells, ensuring that biomolecules are reliably delivered within a cell.
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Bhatia, Sangeeta N., Martin L. Yarmush, and Mehmet Toner. "Engineered Substrates for Controlling Cell-Cell Interactions." In ASME 1997 International Mechanical Engineering Congress and Exposition, 99–103. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-1319.

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Abstract Biomaterials have been previously engineered to serve a variety of different functions: precise degradation in vivo (Kimura, 1993), modulation of cell physiology via binding to specific ligands (Hubbell et al, 1992), and selective permeability of certain solutes (Lysaght et al, 1994). However, in complex tissues, where cell-cell interactions strongly influence tissue function, biomaterials which modulate this fundamental parameter have not been available. In this study, we describe a technique which allows control over cell-cell interactions by using semiconductor-based microfabrication, silane derivatization of borosilicate, and immobilization of specific biomolecules. We have focused on liver tissues due to the clinical significance of liver disease and the potential utility of a highly functional in vitro substitute for the liver (Rozga et al, 1994). Previous studies have reported that co-culture of primary hepatocytes with mesenchymal cells induces up-regulation of liver-specific functions in the hepatocyte population (Guguen-Guillouzo et al, 1983). Using microfabricated techniques to pattern co-cultures, we have demonstrated the importance of local cell-cell interactions in overall tissue function. Specifically, we show that the level of heterotypic interaction modulates the kinetics of up-regulation of liver-specific functions when compared to unpatterned controls. This approach will have applications in many areas including tissue engineering, developmental biology, and transplantation.
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Nachman, R. L., R. L. Silverstein, and A. S. Asch. "THROMBOSPONDIN: CELL BIOLOGY OF AN ADHESIVE GLYCOPROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644653.

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Thrombospondin (TSP), a multifunctional 450 KD glycoprotein is a secretory product of thrombin stimulated platelets. It is a major component of the platelets alpha granule constituting approximately 3% of total platelet protein. Thrombospondin does not circulate in appreciable concentrations ∽0 100 ng/ml); however, the tissue distribution is broad. In addition to its expression on the membrane of activated platelets, the protein is synthesized by fibroblasts endothelial cells, glial cell smooth muscle cells alveolar pneumocytes mononuclear phagocytes and various tumor cells. TSP is a major constituent of the extracellular matrix and has been demonstrated in the vessel wall, basement membrane and glandular connective tissue. Fibroblasts, smooth muscle cells and endothelial cells in tissue culture incorporate TSP into the extracellular matrix. Matrix TSP is under cell-cycle regulatory control. Mesenchymal cells in the proliferative phase synthesize greater amounts of TSP than non growing cells. Platelet derived growth factor induces smooth muscle cell and glial cell synthesis of TSP. Atheromatous lesions contain increased amounts of TSP compared to normal vessels emphasizing the potential role of TSP in the interaction of proliferating cells with the matrix. TSP binds specifically, saturably, and reversibly to mouse peritoneal macrophages and to cells of the monocyte-like human cell line U937. Binding was time dependent and was optimal in the presence of both Ca++ and Mg++. PMA stimulated U937 cells and activated macrophages bound TSP to an equivalent extent as resting cells. The TSP binding site on the surface of U937 cells and peripheral blood monocytes mediates the adhesive interaction between these cells and thrombin-stimulated platelets. Using a sensitive rosetting assay we found that monocytes were not rosetted by resting platelets while >90% were rosetted by thrombin-stimulated platelets. Monoclonal and polyclonal anti-TSP antibodies markedly inhibited rosetting as did TSP itself. Antifibronectin or non-immune control antibodies did not inhibit rosetting, nor did fibronectin, fibrinogen, the fibronectinadhesion tetrapeptide arg-gly-asp-ser (RGDS), or heparin. The TSP membrane receptor, an 88 KD glycoprotein, formely known as GPIV has been identified in platelets, endothelial cells, monocytes and a variety of tumor cells. TSP may thus serve as a molecular bridge linking activated platelets with monocytes at sites of early vascular injury. Such interactions involving the TSP receptor complex may be of critical importance in the regulation of thrombosis and the initiation of atherosclerosis.
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Deraitus, Mary, and Kris Freeman. "Essentials of cell biology." In CHI '01 extended abstracts. New York, New York, USA: ACM Press, 2001. http://dx.doi.org/10.1145/634067.634339.

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Deraitus, Mary, and Kris Freeman. "Essentials of cell biology." In CHI '01 extended abstracts. New York, New York, USA: ACM Press, 2001. http://dx.doi.org/10.1145/634295.634339.

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ROEDER, INGO. "SYSTEMS STEM CELL BIOLOGY." In International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2007. http://dx.doi.org/10.1142/9789812708779_0001.

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Safitri, Nur Lina, Siti Zubaidah, Fatchur Rohman, and Sulisetijono Sulisetijono. "Online learning of cell biology from biology students’ perspectives." In THE 5TH INTERNATIONAL CONFERENCE ON MATHEMATICS AND SCIENCE EDUCATION (ICoMSE) 2021: Science and Mathematics Education Research: Current Challenges and Opportunities. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0112426.

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Jacobson, K. A. "Photobleaching methods in cell biology." In Conference on Lasers and Electro-Optics. Washington, D.C.: OSA, 1985. http://dx.doi.org/10.1364/cleo.1985.ws3.

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Wuertz-Kozak, Karin. "Cell Biology of the Spine." In eccElearning Postgraduate Diploma in Spine Surgery. eccElearning, 2017. http://dx.doi.org/10.28962/01.3.003.

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Jager, Edwin W. H. "Actuators, biomedicine, and cell-biology." In SPIE Smart Structures and Materials + Nondestructive Evaluation and Health Monitoring, edited by Yoseph Bar-Cohen. SPIE, 2012. http://dx.doi.org/10.1117/12.917428.

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Reports on the topic "Cell biology"

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Williams, Thomas. Cell Biology Board Game: Cell Survival (School Version). University of Dundee, 2022. http://dx.doi.org/10.20933/100001270.

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Cells are the smallest units of life. The environment around cells is always changing. Cells need to adapt to survive. This curriculum linked game and lesson plan introduces the world of cells to pupils 8-13. But can they keep their cells alive? This is a guide to how the cell survival resources can be used in a lesson and can be adapted as the teacher sees fit to do so. This lesson is aimed at 8-13 year olds, and fits into an hour long session. The Cell Survival Game has been adapted for both home use and for use in the classroom, and is accompanied by a series of videos. Learning Outcomes – Cells are the smallest unit of life – There are many different types of cells, and some examples of cell types – Cells experience many dangers, and some examples of dangers – How cells notice and defend themselves against dangers Links to the Curriculum – Health and Wellbeing: I am developing my understanding of the human body – Languages: I can find specific information in a straight forward text (book and instructions) to learn new things, I discover new words and phrases (relating to cells) – Mathematics: I am developing a sense of size and amount (by using the dice), I am exploring number processes (addition and subtraction) and understand they represent quantities (steps to finish line), I am learning about measurements (cell sizes) and am exploring patterns (of cell defences against dangers) – Science: I am learning about biodiversity (different types of microbes), body systems, cells and how they work. – Technology: I am learning about new technologies (used to understand how cells work).
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Williams, Thomas. Cell Biology Board Game: Cell Survival (Home Version). University of Dundee, 2022. http://dx.doi.org/10.20933/100001271.

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Williams, Thomas. Cell Biology Boardgame: Cell Survival: Transport. University of Dundee, March 2023. http://dx.doi.org/10.20933/100001281.

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Get your mRNAs from one side of the cell to the other so they can be turned into protein. Fastest wins! This game takes a fun approach to detail findings from real life research. Great for 2-5 people age 3+, lasts around 15 mins per game.
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Williams, Thomas. Cell Biology Board Game: Cell Life Cycle Top Trumps. University of Dundee, January 2023. http://dx.doi.org/10.20933/100001277.

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All living things from whole people to single cells and even viruses have life cycles. Explore the weird and wonderful world of life cycles at the level of the cell in this top trumps inspired game. Print and cut out the cards, then play anywhere you want!
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Williams, Thomas. Cell Biology Board Game: Cell Survival Drive. University of Dundee, 2023. http://dx.doi.org/10.20933/100001276.

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When dangers strike a cell, they are detected by sensors. Sensors tell messengers about the danger. Messengers tell the organiser. The organiser plans the cell defence, using responders and recyclers. Researchers in the MRC-PPU are figuring out how these different parts interact with each other.
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Nielsen, T. B., and J. L. Kidwell. Cell Biology of Hypoxia, 1996. Fort Belvoir, VA: Defense Technical Information Center, September 1996. http://dx.doi.org/10.21236/ada340589.

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Grego, Sonia, Edward R. Dougherty, Francis J. Alexander, Scott S. Auerbach, Brian R. Berridge, Michael L. Bittner, Warren Casey, et al. Systems Biology for Organotypic Cell Cultures. Office of Scientific and Technical Information (OSTI), August 2016. http://dx.doi.org/10.2172/1313549.

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Przekwas, Andrzej, Tom Friend, Rodrigo Teixeira, Z. J. Chen, and Patrick Wilkerson. Spatial Modeling Tools for Cell Biology. Fort Belvoir, VA: Defense Technical Information Center, October 2006. http://dx.doi.org/10.21236/ada460852.

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Drakakaki, Georgia, Marcela Rojas Pierce, and Marisa Otegui. Plant Cell Biology International Meeting 2022. Office of Scientific and Technical Information (OSTI), August 2023. http://dx.doi.org/10.2172/1993620.

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Zatz, Martin. Gordon Research Conference On Pineal Cell Biology. Fort Belvoir, VA: Defense Technical Information Center, July 1992. http://dx.doi.org/10.21236/ada264840.

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