Relevant bibliographies by topics / Cell death pathways

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cell death pathways'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Cell death pathways"

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Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted,
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Stekovic, Slaven, and Frank Madeo. "Cell death pathways." Biochimica et Biophysica Acta (BBA) - Molecular Cell Research 1833, no. 12 (2013): 3447. http://dx.doi.org/10.1016/j.bbamcr.2013.09.016.

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Fulda, Simone. "Alternative Cell Death Pathways and Cell Metabolism." International Journal of Cell Biology 2013 (2013): 1–4. http://dx.doi.org/10.1155/2013/463637.

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While necroptosis has for long been viewed as an accidental mode of cell death triggered by physical or chemical damage, it has become clear over the last years that necroptosis can also represent a programmed form of cell death in mammalian cells. Key discoveries in the field of cell death research, including the identification of critical components of the necroptotic machinery, led to a revised concept of cell death signaling programs. Several regulatory check and balances are in place in order to ensure that necroptosis is tightly controlled according to environmental cues and cellular nee
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Choi, Soo Youn, Whaseon Lee-Kwon, Hwan Hee Lee, Jun Ho Lee, Satoru Sanada, and Hyug Moo Kwon. "Multiple cell death pathways are independently activated by lethal hypertonicity in renal epithelial cells." American Journal of Physiology-Cell Physiology 305, no. 10 (2013): C1011—C1020. http://dx.doi.org/10.1152/ajpcell.00384.2012.

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When hypertonicity is imposed with sufficient intensity and acuteness, cells die. Here we investigated the cellular pathways involved in death using a cell line derived from renal epithelium. We found that hypertonicity rapidly induced activation of an intrinsic cell death pathway—release of cytochrome c and activation of caspase-3 and caspase-9—and an extrinsic pathway—activation of caspase-8. Likewise, a lysosomal pathway of cell death characterized by partial lysosomal rupture and release of cathepsin B from lysosomes to the cytosol was also activated. Relationships among the pathways were
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Shymanskyy, I. O., O. O. Lisakovska, A. O. Mazanova, D. O. Labudzynskyi, A. V. Khomenko, and M. M. Veliky. "Prednisolone and vitamin D(3) modulate oxidative metabolism and cell death pathways in blood and bone marrow mononuclear cells." Ukrainian Biochemical Journal 88, no. 5 (2016): 38–47. http://dx.doi.org/10.15407/ubj88.05.038.

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Wong, Brian, and Yongwon Choi. "Pathways leading to cell death in T cells." Current Opinion in Immunology 9, no. 3 (1997): 358–64. http://dx.doi.org/10.1016/s0952-7915(97)80082-9.

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Mansilla, Sylvia, Laia Llovera, and Jose Portugal. "Chemotherapeutic Targeting of Cell Death Pathways." Anti-Cancer Agents in Medicinal Chemistry 12, no. 3 (2012): 226–38. http://dx.doi.org/10.2174/187152012800228805.

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Jin, Zhaoyu, and Wafik S. El-Deiry. "Overview of cell death signaling pathways." Cancer Biology & Therapy 4, no. 2 (2005): 147–71. http://dx.doi.org/10.4161/cbt.4.2.1508.

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Horowitz, Stuart. "Pathways to Cell Death in Hyperoxia." Chest 116 (July 1999): 64S—67S. http://dx.doi.org/10.1378/chest.116.suppl_1.64s.

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MacFarlane, M. "Cell death pathways – potential therapeutic targets." Xenobiotica 39, no. 8 (2009): 616–24. http://dx.doi.org/10.1080/00498250903137990.

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Dissertations / Theses on the topic "Cell death pathways"

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McComb, Scott. "The Paradoxical Roles of Cell Death Pathways in Immune Cells." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/24331.

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Cell death plays a vital role throughout the immune response, from the onset of inflammation to the elimination of primed T cells. Understanding the regulation of cell death within immune cells is of vital importance to understanding the immune system and developing therapies against various immune-disorders. In this thesis I have investigated the regulation of cell death and its functional role in of the innate and adaptive arms of the immune system. The mechanisms that govern expansion and contraction of antigen stimulated CD8+ T cells are not well understood. In the first section of this
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Yung, Hong Wa. "Regulation of astrocyte cell death by kinase signalling pathways." Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.620576.

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Mässenhausen, Anne von, Wulf Tonnus, and Andreas Linkermann. "Cell Death Pathways Drive Necroinflammation during Acute Kidney Injury." Karger, 2018. https://tud.qucosa.de/id/qucosa%3A71624.

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Renal tubules represent an intercellular unit and function as a syncytium. When acute tubular necrosis was first visualized to occur through a process of synchronized regulated necrosis (SRN) in handpicked primary renal tubules, it became obvious that SRN actually promotes nephron loss. This realization adds to our current understanding of acute kidney injury (AKI)-chronic kidney disease (CKD) transition and argues for the prevention of AKI episodes to prevent CKD progression. Because SRN is triggered by necroptosis and executed by ferroptosis, 2 recently identified signaling pathways of regul
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Guo, Jing. "Studying the signaling pathways in ROS-induced neuronal cell death /." View abstract or full-text, 2005. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202005%20GUO.

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Benford, Helena L. "Molecular pathways of bisphosphonate-induced apoptosis." Thesis, University of Aberdeen, 2000. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU602025.

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Recent studies have proposed that non-nitrogen-containing and nitrogen- containing bisphosphonate drugs inhibit osteoclastic bone resorption by different molecular mechanisms. The aim of this thesis was to investigate the molecular mechanisms of action of bisphosphonates in macrophages and osteoclasts and, in particular, the activation of caspase proteases and their role in apoptotic cell death. Apoptosis of J774 macrophages induced by nitrogen-containing bisphosphonates was found to involve the activation of caspase-3. By contrast, non-nitrogen- containing bisphosphonates did not cause caspas
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Zhang, Tongli. "Mathematical Models of Some Signaling Pathways Regulating Cell Survival and Death." Diss., Virginia Tech, 2008. http://hdl.handle.net/10919/29443.

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In a multi-cellular organism, cells constantly receive signals on their internal condition and surrounding environment. In response to various signals, cells proliferate, move around or even undergo suicide. The signal-response is controlled by complex molecular machinery, understanding of which is an important goal of basic molecular biological research. Such understanding is also valuable for clinical application, since lethal diseases like cancer show maladaptive responses to growth-regulating signals. Because the multiple feedbacks in the molecular regulatory machinery obscure cause-effect
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Yatim, Nader. "Coordinated activation of cell death and inflammatory pathways in dying cells regulate adaptive immunity." Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCC233.

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Les Cellules mourantes initient l'immunité adaptative en fournissant des stimuli inflammatoires et des antigènes pour les cellules dendritiques (CD), qui à leurs tour activent les cellules T CD8 + par un processus appelé la présentation-croisée. Pour définir comment les différentes formes programmées de la mort cellulaire influencent l'immunité, nous avions établi des modèles de nécroptose et apoptose, par l'activation spécifique de RIPK3 ou CASP8, que nous avons utilisé pour évaluer in vitro et in vivo la réponse immunitaire. Nous avons alors trouvé que les cellules nécroptotiques exprimant l
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Janson, Veronica. "Cisplatin-resistance and cell death in malignant pleural mesothelioma cells." Doctoral thesis, Umeå universitet, Medicinsk biovetenskap, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1680.

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Malignant pleural mesothelioma (MPM) is an aggressive, treatment-resistant tumour. Cisplatin (cis-diamminedichloroplatinum (II)) is the best single-agent chemotherapy for MPM, but platinum-based combination therapies give the best overall response rates. However, cisplatin use is limited by resistance and severe side effects. This thesis has increased the knowledge concerning cisplatin-induced cell death in MPM by describing a novel potential therapeutic target, and three novel phenotypes of cisplatin-resistance in a human MPM cell line (P31) and its cisplatin-resistant sub-line (P31res1.2). T
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Davies, Carwyn Children's Cancer Institute Australia for Medical Research UNSW. "The influence of p21WAF1 on cell death pathways in acute lymphoblastic leukaemia." Publisher:University of New South Wales. Children's Cancer Institute Australia for Medical Research, 2009. http://handle.unsw.edu.au/1959.4/44416.

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The p53 protein is a primary mediator of apoptosis and growth arrest after exposure to DNA-damaging agents. Previous work has categorised a wild type p53 gene in the majority of childhood acute lymphoblastic leukaemia (ALL) cases, in which instance the p53 protein functions as a modulator of chemotherapy-induced cell death. In contrast, certain p53-induced proteins, such as p21WAF1, can act in an anti-apoptotic manner, and bestow resistance to chemotherapy. Previous studies of the p53 pathway in ALL have utilised cell lines and primary material. In this study a model of ALL was utilised that h
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Zhang, Tejia. "Discovery of bioactive lipids and lipid pathways in cell death and disease." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11483.

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Apoptosis is an intricately regulated cellular process required for the health and homeostasis of living systems. The mitochondrial apoptotic pathway depends on the BCL-2 family of pro- and anti-apoptotic members whose interactions regulate cell fate. BAX and BAK are key pro-apoptotic proteins required for mitochondrial permeabilization during apoptosis. While the mitochondrial death program relies heavily on its protein components, evidences support equally crucial roles for lipids and lipid metabolism in promoting or hindering apoptosis at the mitochondria. To gain insight into the inter
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Books on the topic "Cell death pathways"

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Bromfield, Gillian. Cell death pathways in irradiated prostate cells. National Library of Canada, 2002.

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Aft, Rebecca, ed. Targeting New Pathways and Cell Death in Breast Cancer. InTech, 2012. http://dx.doi.org/10.5772/1744.

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(Editor), Jochen Schacht, Arthur N. Popper (Editor), and Richard R. Fay (Editor), eds. Auditory Trauma, Protection, and Repair (Springer Handbook of Auditory Research). Springer, 2008.

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Phosphoinositide 3-kinase Signalling Pathway: The Key to Cell Proliferation And Death. Imperial College Press, 2006.

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Goligorsky, Michael S., Julien Maizel, Radovan Vasko, May M. Rabadi, and Brian B. Ratliff. Pathophysiology of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0221.

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In the intricate maze of proposed mechanisms, modifiers, modulators, and sensitizers for acute kidney injury (AKI) and diverse causes inducing it, this chapter focuses on several common and undisputable strands which do exist.Structurally, the loss of the brush border, desquamation of tubular epithelial cells, and obstruction of the tubular lumen are commonly observed, albeit to various degrees. These morphologic hallmarks of AKI are accompanied by functional defects, most consistently reflected in the decreased glomerular filtration rate and variable degree of reduction in renal blood flow, a
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Anning, Lin, ed. The JNK signaling pathway. Landes Bioscience, 2006.

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Plutynski, Anya. Causation, Causal Selection, and Causal Parity. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780199967452.003.0004.

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It is typical to refer to cancer as a “genetic” or “genomic” disease. This claim is ambiguous; one of the central goals of this chapter is to disambiguate this claim. I first distinguish different types of causal claims: claims about causal relevance, causal role, and causal specificity. As a backdrop to this discussion, I introduce what I call the “mechanistic research program” in cancer, according to which progression to cancer involves breakdowns in regulatory controls on gene expression in ways that affect cell birth and death. While this research program has been successful, it has downpl
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Pleniceanu, Oren, and Benjamin Dekel. Kidney stem cells. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0344.

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End-stage renal failure is a major cause of death with currently only dialysis and transplantation available as therapeutic options, each with its own limitations and drawbacks. To allow regenerative medicine-based kidney replacement therapies and due to the fact that neither haematopoietic stem cells nor mesenchymal stem cells, the most accessible human stem cells, can be used to derive genuine nephron progenitors, much attention has been given to finding adult renal stem cells. Several candidates for this have been described, but their true identity as stem or progenitor cells and their pote
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Kriz, Wilhelm. Podocyte loss as a common pathway to chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0139.

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Experimental studies show that podocyte death first causes focal scars, but beyond approximately 40% loss is lethal to a glomerulus. Podocytes have limited ability to regenerate, although some degree of replacement may occur from stem cells located near the urinary pole of Bowman’s capsule. It is not yet known whether this plays a significant part in ameliorating damage in disease processes. In one interpretation, foot process effacement may be seen as an adaptation by the podocyte to remain attached to the glomerular basement membrane after injury, at the expense of proteinuria. Podocyte dysf
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Noordenbos, Troy, and Dominique Baeten. Immune mechanisms: innate immunity. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0007.

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Innate immune mechanisms are strongly implied in the pathophysiology of spondyloarthritis (SpA). This chapter discusses available data on the role of the innate immune system in relation to HLA-B27, genetic associations, and the cellular and molecular characteristics of disease target tissue. Regarding the linkage with MCH-class I molecule HLA-B27, the chapter discusses the arthritogenic peptide hypothesis and three popular antigen-independent theories. The genetic architecture of the disease argues against a role for the adaptive immune system and identifies cytokine pathways, such as IL-1, T
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Book chapters on the topic "Cell death pathways"

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McDonald, E. Robert, and Wafik S. El-Deiry. "Mammalian Cell Death Pathways." In Death Receptors in Cancer Therapy. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-851-x:001.

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Horowitz, Stuart. "Pathways to Oxidative Cell Death." In Acute Respiratory Distress Syndrome. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4419-8634-4_26.

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Marín-García, José. "Cell-Death Pathways and Mitochondria." In Mitochondria and Their Role in Cardiovascular Disease. Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4599-9_11.

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Lin, Yong. "RIP1-Mediated Signaling Pathways in Cell Survival and Death Control." In Necrotic Cell Death. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8220-8_2.

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Wardle, E. Nigel. "Programmed Cell Death: Apoptosis." In Guide to Signal Pathways in Immune Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-538-5_8.

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Morley, Simon J. "The Regulation of eIF4F During Cell Growth and Cell Death." In Signaling Pathways for Translation. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-662-09889-9_1.

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Meuer, Katrin, Mathias Bähr, and Jochen H. Weishaupt. "CDK5 and Mitochondrial Cell Death Pathways." In Cyclin Dependent Kinase 5 (Cdk5). Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-78887-6_7.

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Kessel, David, and Nancy L. Oleinick. "Photodynamic Therapy and Cell Death Pathways." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-697-9_3.

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Singh, Gurmit, Omar Alqawi, and Myrna Espiritu. "Metronomic PDT and Cell Death Pathways." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-697-9_5.

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Andersen, Joshua L., and Jeffrey C. Rathmell. "Cell Death Pathways in Toxicological Response." In Mammalian Toxicology. John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781118683484.ch4.

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Conference papers on the topic "Cell death pathways"

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Kessel, David, and John J. Reiners, Jr. "Cell death pathways associated with PDT." In Biomedical Optics 2006, edited by David Kessel. SPIE, 2006. http://dx.doi.org/10.1117/12.639925.

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Wright, Neil T. "Parameter Correlation in Models of Hyperthermic Cell Death." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53933.

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A number of mathematical models have been developed to predict the survival of cells after heating. Some of these models have been based on first principle arguments, while others have been empirically motivated. Some models have been inspired by analogs of damage to cells by ionizing radiation. Evidence exists for multiple targets leading to cell death, although precise definition of the pathways for the various temperature ranges and environmental conditions remains in question. For reviews of the cellular targets of heating, see [1], [2], or [3].
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Belashov, A. V., A. A. Zhikhoreva, D. A. Rogova, et al. "Holographic monitoring of cell death pathways induced by reactive oxygen species." In 2018 International Conference Laser Optics (ICLO). IEEE, 2018. http://dx.doi.org/10.1109/lo.2018.8435688.

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Arumugam, Arunkumar, and Rajkumar Lakshmanaswamy. "Abstract 5361: Progesterone alters cell survival and cell death pathways in estrogen-induced mammary carcinogenesis." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5361.

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Barbee, Kenneth A., Gulyeter Serbest, and Joel Horwitz. "Membrane Integrity as a Therapeutic Target in Neural Cell Injury." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61566.

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The importance of cell membrane integrity for normal cell function and indeed survival is well established, yet the role of membrane disruption in cellular pathology is seldom considered except as a prelude to, or indication of, cell death. However, evidence from diverse fields strongly implicates membrane disruption as a key precipitating event in the pathological responses to various stimuli. Dynamic mechanical loading of neural cells produces an acute disruption of the plasma membrane as indicated by a rapid and transient release of LDH from the cytoplasm of injured cells. In this report, w
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Turubanova, Victoria, Iuliia Efimova, Tatiana Mishchenko, Irina Balalaeva, Maria Vedunova, and Dmitri Krysko. "IMMUNOGENIC PATHWAYS OF CONTROLLED CELL DEATH IN THE TREATMENT OF NEURO-ONCOLOGICAL DISEASES." In XV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2019. http://dx.doi.org/10.29003/m584.sudak.ns2019-15/415.

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Shah, Pooja A., Tuhina Mazumdar, Reid T. Powell, et al. "Abstract 369: Identification of pathways that enhance cell death in NOTCH1-mutant HNSCC." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-369.

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Abdi, A. "Computational modeling of molecular pathways regulating cell survival and death by the SigFlux algorithm." In 2017 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2017. http://dx.doi.org/10.1109/spmb.2017.8257047.

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Turubanova, Victoria, Iuliia Efimova, Irina Balalaeva, et al. "IMMUNOGENIC PATHWAYS OF CONTROLLED CELL DEATH IN THE TREATMENT OF NEURO-ONCOLOGICAL DISEASES USING PORPHYRAZINES." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1291.sudak.ns2020-16/464-465.

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Oliver, David J., Anusha Chaparala, Emeka Okafor, et al. "Abstract 5104: Identification of cancer-specific COPI inhibitors and their associated apoptotic cell death pathways." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5104.

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Reports on the topic "Cell death pathways"

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Vincenz, Claudius. Identification of Components of the Cell Death Pathway. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada372429.

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Reed, John C. Yeast Genetics for Delineating Bax/Bc1 Pathway of Cell Death Regulation. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada329121.

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Xu, Qunli. Training in Support of Research Project Entitled Genetic Regulation of the Bcl-2/Bax Cell Death Pathway".". Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada368475.

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