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Journal articles on the topic 'Cell interaction'

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

Brückner, David B., Nicolas Arlt, Alexandra Fink, Pierre Ronceray, Joachim O. Rädler, and Chase P. Broedersz. "Learning the dynamics of cell–cell interactions in confined cell migration." Proceedings of the National Academy of Sciences 118, no. 7 (February 12, 2021): e2016602118. http://dx.doi.org/10.1073/pnas.2016602118.

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The migratory dynamics of cells in physiological processes, ranging from wound healing to cancer metastasis, rely on contact-mediated cell–cell interactions. These interactions play a key role in shaping the stochastic trajectories of migrating cells. While data-driven physical formalisms for the stochastic migration dynamics of single cells have been developed, such a framework for the behavioral dynamics of interacting cells still remains elusive. Here, we monitor stochastic cell trajectories in a minimal experimental cell collider: a dumbbell-shaped micropattern on which pairs of cells perf

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2

Ogushi, Fumiko, and Hiroshi Kori. "3P277 Dependence of cell differentiation ratio on cell-cell interaction and noise(24. Mathematical biology,Poster)." Seibutsu Butsuri 53, supplement1-2 (2013): S257. http://dx.doi.org/10.2142/biophys.53.s257_6.

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3

Chesterton, C. J. "Cell to cell interaction." FEBS Letters 293, no. 1-2 (November 1, 1991): 229. http://dx.doi.org/10.1016/0014-5793(91)81201-i.

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4

Manimaran, K., Arun Kumar, AvinashGandi D, and S. Sankaranarayanan. "Interaction of Human Dental Pulp Stem Cells and Ameloblastic Cell In-vitro- A Preclinical Analysis." Annals of Oral Health and Dental Research 2, no. 1 (January 17, 2018): A1–5. http://dx.doi.org/10.21276/aohdr.1831.

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5

Oropesa-Nuñez, Reinier, Andrea Mescola, Massimo Vassalli, and Claudio Canale. "Impact of Experimental Parameters on Cell–Cell Force Spectroscopy Signature." Sensors 21, no. 4 (February 4, 2021): 1069. http://dx.doi.org/10.3390/s21041069.

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Atomic force microscopy is an extremely versatile technique, featuring atomic-scale imaging resolution, and also offering the possibility to probe interaction forces down to few pN. Recently, this technique has been specialized to study the interaction between single living cells, one on the substrate, and a second being adhered on the cantilever. Cell–cell force spectroscopy offers a unique tool to investigate in fine detail intra-cellular interactions, and it holds great promise to elucidate elusive phenomena in physiology and pathology. Here we present a systematic study of the effect of th

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6

Hwang, Inkyu, Jing-Feng Huang, Hidehiro Kishimoto, Anders Brunmark, Per A. Peterson, Michael R. Jackson, Charles D. Surh, Zeling Cai, and Jonathan Sprent. "T Cells Can Use Either T Cell Receptor or Cd28 Receptors to Absorb and Internalize Cell Surface Molecules Derived from Antigen-Presenting Cells." Journal of Experimental Medicine 191, no. 7 (March 27, 2000): 1137–48. http://dx.doi.org/10.1084/jem.191.7.1137.

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At the site of contact between T cells and antigen-presenting cells (APCs), T cell receptor (TCR)–peptide–major histocompatibility complex (MHC) interaction is intensified by interactions between other molecules, notably by CD28 and lymphocyte function-associated antigen 1 (LFA-1) on T cells interacting with B7 (B7-1 and B7-2), and intracellular adhesion molecule 1 (ICAM-1), respectively, on APCs. Here, we show that during T cell–APC interaction, T cells rapidly absorb various molecules from APCs onto the cell membrane and then internalize these molecules. This process is dictated by at least

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Yuan, Ye, Carlos Cosme, Taylor Sterling Adams, Jonas Schupp, Koji Sakamoto, Nikos Xylourgidis, Matthew Ruffalo, Jiachen Li, Naftali Kaminski, and Ziv Bar-Joseph. "CINS: Cell Interaction Network inference from Single cell expression data." PLOS Computational Biology 18, no. 9 (September 12, 2022): e1010468. http://dx.doi.org/10.1371/journal.pcbi.1010468.

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Studies comparing single cell RNA-Seq (scRNA-Seq) data between conditions mainly focus on differences in the proportion of cell types or on differentially expressed genes. In many cases these differences are driven by changes in cell interactions which are challenging to infer without spatial information. To determine cell-cell interactions that differ between conditions we developed the Cell Interaction Network Inference (CINS) pipeline. CINS combines Bayesian network analysis with regression-based modeling to identify differential cell type interactions and the proteins that underlie them. W

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8

Okuyama, Akihiko. "INTRATESTICULAR CELL TO CELL INTERACTION." Japanese Journal of Urology 83, no. 7 (1992): 1027–35. http://dx.doi.org/10.5980/jpnjurol1989.83.1027.

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9

Yamasaki, Hiroshi. "Cell-Cell Interaction and Carcinogenesis." Toxicologic Pathology 14, no. 3 (April 1986): 363–69. http://dx.doi.org/10.1177/019262338601400313.

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10

Lin, Yingxin, Lipin Loo, Andy Tran, David M. Lin, Cesar Moreno, Daniel Hesselson, G. Gregory Neely, and Jean Y. H. Yang. "Scalable workflow for characterization of cell-cell communication in COVID-19 patients." PLOS Computational Biology 18, no. 10 (October 5, 2022): e1010495. http://dx.doi.org/10.1371/journal.pcbi.1010495.

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COVID-19 patients display a wide range of disease severity, ranging from asymptomatic to critical symptoms with high mortality risk. Our ability to understand the interaction of SARS-CoV-2 infected cells within the lung, and of protective or dysfunctional immune responses to the virus, is critical to effectively treat these patients. Currently, our understanding of cell-cell interactions across different disease states, and how such interactions may drive pathogenic outcomes, is incomplete. Here, we developed a generalizable and scalable workflow for identifying cells that are differentially i

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11

Guo, Chutian. "Cell Type Specific Gene Interaction between Microbiota and Antidepressant Drugs." International Journal of Bioscience, Biochemistry and Bioinformatics 11, no. 2 (April 2021): 14–21. http://dx.doi.org/10.17706/ijbbb.2021.11.2.14-21.

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12

Pasqual, Giulia. "Characterising cell-cell interactions." EU Research 32, Autumn 2022 (2022): 24–25. http://dx.doi.org/10.56181/qvsz2211.

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T-cells are an important part of the immune system, but they need to interact with dendritic cells before they can be activated and acquire specific functions. We spoke to Dr Giulia Pasqual about her research into the interactions between dendritic- and T-cells, and how this interaction influences the subsequent fate of T-cells.

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13

Van De Water, Thomas R. "Tissue interactions and cell differentiation: neurone–sensory cell interaction during otic development." Development 103, Supplement (September 1, 1988): 185–93. http://dx.doi.org/10.1242/dev.103.supplement.185.

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Statoacoustic ganglion neurones (SAG) are produced by the same group of cells (otic placode) that produce all of the receptor cells that populate the sensory areas of the inner ear. The observation that ingrowth of SAG neurites to presumptive sensory areas of the inner ear preceded cytodifferentiation of those receptor cells suggested a causal relationship. Results from in vivo, in ovo and in vitro studies do not support a causal relationship. These studies support the hypothesis that the programme for labyrinthine sensory cell differentiation is intrinsic and does not require the extrinsic st

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14

van Vliet, Simon, Christoph Hauert, Kyle Fridberg, Martin Ackermann, and Alma Dal Co. "Global dynamics of microbial communities emerge from local interaction rules." PLOS Computational Biology 18, no. 3 (March 4, 2022): e1009877. http://dx.doi.org/10.1371/journal.pcbi.1009877.

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Most microbes live in spatially structured communities (e.g., biofilms) in which they interact with their neighbors through the local exchange of diffusible molecules. To understand the functioning of these communities, it is essential to uncover how these local interactions shape community-level properties, such as the community composition, spatial arrangement, and growth rate. Here, we present a mathematical framework to derive community-level properties from the molecular mechanisms underlying the cell-cell interactions for systems consisting of two cell types. Our framework consists of tw

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15

Parekkadan, Biju, Yevgeny Berdichevsky, Daniel Irimia, Avrum Leeder, Gabriel Yarmush, Mehmet Toner, John B. Levine, and Martin L. Yarmush. "Cell–cell interaction modulates neuroectodermal specification of embryonic stem cells." Neuroscience Letters 438, no. 2 (June 2008): 190–95. http://dx.doi.org/10.1016/j.neulet.2008.03.094.

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16

Nadel, Jay A. "Cell-to-Cell Interaction in Airways." Chest 93, no. 6 (June 1988): 1281–82. http://dx.doi.org/10.1378/chest.93.6.1281.

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17

Canipari, Rita. "Cell–cell interaction and oocyte growth." Zygote 2, no. 4 (November 1994): 343–45. http://dx.doi.org/10.1017/s0967199400002173.

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In most mammals, oocytes initiate meiosis in late fetal life; by the time of birthe they have already entered the diplotene stage of prophase I of meiosis and becaome arrested thereafter at the dictyate state(Baker, 1972). At this stage they became surrounded by a few nonproliferating flat follicle cells forming a unit called the resting or primordial follicle.

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18

Hoshino, Takashi, Kazuya Shimizu, Tomoyuki Honda, Tomomi Kawakatsu, Taihei Fukuyama, Takeshi Nakamura, Michiyuki Matsuda, and Yoshimi Takai. "A Novel Role of Nectins in Inhibition of the E-Cadherin–induced Activation of Rac and Formation of Cell-Cell Adherens Junctions." Molecular Biology of the Cell 15, no. 3 (March 2004): 1077–88. http://dx.doi.org/10.1091/mbc.e03-05-0321.

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Nectins are Ca2+-independent immunoglobulin (Ig)-like cell-cell adhesion molecules. The trans-interactions of nectins recruit cadherins to the nectin-based cell-cell adhesion, resulting in formation of cell-cell adherens junctions (AJs) in epithelial cells and fibroblasts. The trans-interaction of E-cadherin induces activation of Rac small G protein, whereas the trans-interactions of nectins induce activation of not only Rac but also Cdc42 small G protein. We showed by the fluorescent resonance energy transfer (FRET) imaging that the trans-interaction of E-cadherin induced dynamic activation a

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19

Pancheva, Alexandrina, Helen Wheadon, Simon Rogers, and Thomas D. Otto. "Using topic modeling to detect cellular crosstalk in scRNA-seq." PLOS Computational Biology 18, no. 4 (April 8, 2022): e1009975. http://dx.doi.org/10.1371/journal.pcbi.1009975.

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Cell-cell interactions are vital for numerous biological processes including development, differentiation, and response to inflammation. Currently, most methods for studying interactions on scRNA-seq level are based on curated databases of ligands and receptors. While those methods are useful, they are limited to our current biological knowledge. Recent advances in single cell protocols have allowed for physically interacting cells to be captured, and as such we have the potential to study interactions in a complimentary way without relying on prior knowledge. We introduce a new method based o

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20

Tang, Xiaoqing, Xiaoming Liu, Pengyun Li, Yuqing Lin, Kojima Masaru, Qiang Huang, and Tatsuo Arai. "Efficient Cell Trapping for Cell-Cell Interaction Analysis." Proceedings of JSME annual Conference on Robotics and Mechatronics (Robomec) 2019 (2019): 2A2—S07. http://dx.doi.org/10.1299/jsmermd.2019.2a2-s07.

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21

Hara, Kodai, Masayuki Uchida, Risa Tagata, Hideshi Yokoyama, Yoshinobu Ishikawa, Asami Hishiki, and Hiroshi Hashimoto. "Structure of proliferating cell nuclear antigen (PCNA) bound to an APIM peptide reveals the universality of PCNA interaction." Acta Crystallographica Section F Structural Biology Communications 74, no. 4 (March 22, 2018): 214–21. http://dx.doi.org/10.1107/s2053230x18003242.

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Proliferating cell nuclear antigen (PCNA) provides a molecular platform for numerous protein–protein interactions in DNA metabolism. A large number of proteins associated with PCNA have a well characterized sequence termed the PCNA-interacting protein box motif (PIPM). Another PCNA-interacting sequence termed the AlkB homologue 2 PCNA-interacting motif (APIM), comprising the five consensus residues (K/R)-(F/Y/W)-(L/I/V/A)-(L/I/V/A)-(K/R), has also been identified in various proteins. In contrast to that with PIPM, the PCNA–APIM interaction is less well understood. Here, the crystal structure o

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22

Chatterjee, Sharmila, and Ulhas P. Naik. "Pericyte-endothelial cell interaction." Cell Adhesion & Migration 6, no. 3 (May 2012): 157–59. http://dx.doi.org/10.4161/cam.20252.

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23

LANGUINO, L., S. COLELLA, N. POLENTARUTTI, L. MAES, A. MANTOVANI, G. MARGUERIE, and E. DEJANA. "Fibrinogen-endothelial cell interaction." Cell Biology International Reports 10, no. 6 (June 1986): 491. http://dx.doi.org/10.1016/0309-1651(86)90066-4.

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24

Owens, Trevor, and Rana Zeine. "The cell biology of T-dependent B cell activation." Biochemistry and Cell Biology 67, no. 9 (September 1, 1989): 481–89. http://dx.doi.org/10.1139/o89-078.

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The requirement that CD4+ helper T cells recognize antigen in association with class II Major Histocompatibility Complex (MHC) encoded molecules constrains T cells to activation through intercellular interaction. The cell biology of the interactions between CD4+ T cells and antigen-presenting cells includes multipoint intermolecular interactions that probably involve aggregation of both polymorphic and monomorphic T cell surface molecules. Such aggregations have been shown in vitro to markedly enhance and, in some cases, induce T cell activation. The production of T-derived lymphokines that ha

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25

Doyle, Andrew D., Shayan S. Nazari, and Kenneth M. Yamada. "Cell–extracellular matrix dynamics." Physical Biology 19, no. 2 (January 12, 2022): 021002. http://dx.doi.org/10.1088/1478-3975/ac4390.

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Abstract The sites of interaction between a cell and its surrounding microenvironment serve as dynamic signaling hubs that regulate cellular adaptations during developmental processes, immune functions, wound healing, cell migration, cancer invasion and metastasis, as well as in many other disease states. For most cell types, these interactions are established by integrin receptors binding directly to extracellular matrix proteins, such as the numerous collagens or fibronectin. For the cell, these points of contact provide vital cues by sampling environmental conditions, both chemical and phys

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26

Santoso, Sentot, Valeria V. Orlova, Kaimei Song, Ulrich J. Sachs, Cornelia L. Andrei-Selmer, and Triantafyllos Chavakis. "The Homophilic Binding of Junctional Adhesion Molecule-C Mediates Tumor Cell-Endothelial Cell Interactions." Journal of Biological Chemistry 280, no. 43 (August 23, 2005): 36326–33. http://dx.doi.org/10.1074/jbc.m505059200.

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The junctional adhesion molecule C (JAM-C) was recently shown to undergo a heterophilic interaction with the leukocyte β2 integrin Mac-1, thereby mediating interactions between vascular cells in inflammatory cell recruitment. Here, the homophilic interaction of JAM-C is presented and functionally characterized to mediate tumor cell-endothelial cell interactions. Recombinant soluble JAM-C in fluid phase bound to immobilized JAM-C as assessed in a purified system; moreover, JAM-C-transfected Chinese hamster ovary (CHO) cells adhered to immobilized JAM-C. The homophilic interaction of JAM-C was m

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27

Kneitz, C., M. Goller, M. Wilhelm, C. Mehringer, G. Wohlleben, A. Schimpl, and H.-P. Tony. "Inhibition of T cell/B cell interaction by B-CLL cells." Leukemia 13, no. 1 (January 1999): 98–104. http://dx.doi.org/10.1038/sj.leu.2401235.

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28

Onoda, Makoto. "Cell-to-cell interaction in the testis." Newsletter of Japan Society for Comparative Endocrinology, no. 65 (1992): 8–12. http://dx.doi.org/10.5983/nl2001jsce.18.8.

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29

IMURA, Katsuaki, Katsuko FURUKAWA, Takashi USHIDA, and Tetsuya TATEISHI. "Induction of chondrogenesis by cell-cell interaction." Proceedings of the Bioengineering Conference Annual Meeting of BED/JSME 2003.15 (2003): 289–90. http://dx.doi.org/10.1299/jsmebio.2003.15.289.

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30

Lamponi, Stefania, Clara Dl Canio, and Rolando Barbucci. "Heterotypic Cell-Cell Interaction on Micropatterned Surfaces." International Journal of Artificial Organs 32, no. 8 (August 2009): 507–16. http://dx.doi.org/10.1177/039139880903200805.

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Purpose The aim of this paper was to study the influence of chemical and topographical signals on cell behavior and to obtain a heterotypic cell-cell interaction on microstructured domains. Methods The polysaccharide hyaluronic acid (Hyal) was photoimmobilized on glass surfaces in order to obtain a pattern with squares and rectangles of different dimensions and chemistry. The microstructured surfaces were characterized by Scanning Electron Microscopy (SEM) and Atomic Force Microscopy (AFM). The behavior of Human Coronary Artery Endothelial Cells (HCAEC) and human tumoral dermal fibroblasts (C5

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31

Häussinger, D., and F. Lang. "Interaction of Cell Volume and Cell Function." Kidney and Blood Pressure Research 13, no. 3 (1990): 162–79. http://dx.doi.org/10.1159/000173362.

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32

Danese, Silvio, and Claudio Fiocchi. "Endothelial Cell-Immune Cell Interaction in IBD." Digestive Diseases 34, no. 1-2 (2016): 43–50. http://dx.doi.org/10.1159/000442925.

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The proper delivery of immune cells throughout the host's various tissues and organs is essential to health, and abnormalities in the type and quantity of leukocyte distribution is usually associated with disease. Because of its size and presence of a very large amount of immunocytes in the mucosa and mesenteric lymph nodes, the gut is the recipient of a constant influx of leukocytes, a process tightly regulated by multiple factors. These include cell adhesion molecules on the leukocytes and their counter-receptors on the microvascular endothelial cells in the bowel wall, a number of chemokine

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33

Chappell, Dale B., and Nicholas P. Restifo. "T cell-tumor cell: a fatal interaction?" Cancer Immunology, Immunotherapy 47, no. 2 (October 15, 1998): 65–71. http://dx.doi.org/10.1007/s002620050505.

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34

Inooka, Shoshi, and Tatsushi Toyokuni. "Sphingosine Transfer in Cell-to-Cell Interaction." Biochemical and Biophysical Research Communications 218, no. 3 (January 1996): 872–76. http://dx.doi.org/10.1006/bbrc.1996.0155.

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35

Murillo, Gonzalo, Andreu Blanquer, Carolina Vargas-Estevez, Lleonard Barrios, Elena Ibáñez, Carme Nogués, and Jaume Esteve. "Electromechanical Nanogenerator-Cell Interaction Modulates Cell Activity." Advanced Materials 29, no. 24 (April 24, 2017): 1605048. http://dx.doi.org/10.1002/adma.201605048.

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36

Septiadi, Dedy, Federica Crippa, Thomas Lee Moore, Barbara Rothen-Rutishauser, and Alke Petri-Fink. "Nanoparticle-Cell Interaction: A Cell Mechanics Perspective." Advanced Materials 30, no. 19 (January 9, 2018): 1704463. http://dx.doi.org/10.1002/adma.201704463.

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37

Van Der Donk, J. A., A. L. De Ruiter-Bootsma, A. M. Ultee-Van Gessel, and P. J. J. Wauben-Penris. "Cell-cell interaction between rat sertoli cells and mouse germ cells in vitro." Experimental Cell Research 164, no. 1 (May 1986): 191–98. http://dx.doi.org/10.1016/0014-4827(86)90466-0.

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38

Miang-Lon Ng, Patricia, and Thomas Lufkin. "Embryonic stem cells: protein interaction networks." BioMolecular Concepts 2, no. 1-2 (April 1, 2011): 13–25. http://dx.doi.org/10.1515/bmc.2011.008.

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AbstractEmbryonic stem cells have the ability to differentiate into nearly all cell types. However, the molecular mechanism of its pluripotency is still unclear. Oct3/4, Sox2 and Nanog are important factors of pluripotency. Oct3/4 (hereafter referred to as Oct4), in particular, has been an irreplaceable factor in the induction of pluripotency in adult cells. Proteins interacting with Oct4 and Nanog have been identified via affinity purification and mass spectrometry. These data, together with iterative purifications of interacting proteins allowed a protein interaction network to be constructe

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Noe, Remi, Sothea Touch, Christine Gaboriaud, Jitka Fučíková, Veronique Fremeaux-Bacchi, Guido Kroemer, Oliver Kepp, and Lubka T. Roumenina. "Complement interaction with cells undergoing immunogenic cell death." Molecular Immunology 89 (September 2017): 122. http://dx.doi.org/10.1016/j.molimm.2017.06.054.

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40

van Wamel, Annemieke, Ayache Bouakaz, Michel Versluis, and Nico de Jong. "Micromanipulation of endothelial cells: Ultrasound-microbubble-cell interaction." Ultrasound in Medicine & Biology 30, no. 9 (September 2004): 1255–58. http://dx.doi.org/10.1016/j.ultrasmedbio.2004.07.015.

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Levine, J. F., and F. E. Stockdale. "Cell-cell interactions promote mammary epithelial cell differentiation." Journal of Cell Biology 100, no. 5 (May 1, 1985): 1415–22. http://dx.doi.org/10.1083/jcb.100.5.1415.

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Mammary epithelium differentiates in a stromal milieu of adipocytes and fibroblasts. To investigate cell-cell interactions that may influence mammary epithelial cell differentiation, we developed a co-culture system of murine mammary epithelium and adipocytes and other fibroblasts. Insofar as caseins are specific molecular markers of mammary epithelial differentiation, rat anti-mouse casein monoclonal antibodies were raised against the three major mouse casein components to study this interaction. Mammary epithelium from mid-pregnant mice was plated on confluent irradiated monolayers of 3T3-L1

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Kulharia, Mahesh. "Geometrical and electro-static determinants of protein-protein interactions." Bioinformation 17, no. 10 (October 31, 2021): 851–60. http://dx.doi.org/10.6026/97320630017851.

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Protein-protein interactions (PPI) are pivotal to the numerous processes in the cell. Therefore, it is of interest to document the analysis of these interactions in terms of binding sites, topology of the interacting structures and physiochemical properties of interacting interfaces and the of forces interactions. The interaction interface of obligatory protein-protein complexes differs from that of the transient interactions. We have created a large database of protein-protein interactions containing over100 thousand interfaces. The structural redundancy was eliminated to obtain a non-redunda

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Purushothaman, Anurag, Shyam Kumar Bandari, Jian Liu, James A. Mobley, Elizabeth E. Brown, and Ralph D. Sanderson. "Fibronectin on the Surface of Myeloma Cell-derived Exosomes Mediates Exosome-Cell Interactions." Journal of Biological Chemistry 291, no. 4 (November 24, 2015): 1652–63. http://dx.doi.org/10.1074/jbc.m115.686295.

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Exosomes regulate cell behavior by binding to and delivering their cargo to target cells; however, the mechanisms mediating exosome-cell interactions are poorly understood. Heparan sulfates on target cell surfaces can act as receptors for exosome uptake, but the ligand for heparan sulfate on exosomes has not been identified. Using exosomes isolated from myeloma cell lines and from myeloma patients, we identify exosomal fibronectin as a key heparan sulfate-binding ligand and mediator of exosome-cell interactions. We discovered that heparan sulfate plays a dual role in exosome-cell interaction;

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Jacobs, Evan S., Thaddeus C. George, Sukhwinder Singh, Richard Wnek, Paul Fischer, Shila K. Nordone, Gregg A. Dean, and Patricia Fitzgerald-Bocarsly. "Plasmacytoid dendritic cell interaction with HIV-infected T cells: Live cell nibbling vs cell–cell fusion." Cytokine 48, no. 1-2 (October 2009): 66–67. http://dx.doi.org/10.1016/j.cyto.2009.07.214.

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Milicevic, Katarina D., Danijela B. Bataveljic, Jelena J. Bogdanovic Pristov, Pavle R. Andjus та Ljiljana M. Nikolic. "Astroglial Cell-to-Cell Interaction with Autoreactive Immune Cells in Experimental Autoimmune Encephalomyelitis Involves P2X7 Receptor, β3-Integrin, and Connexin-43". Cells 12, № 13 (5 липня 2023): 1786. http://dx.doi.org/10.3390/cells12131786.

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In multiple sclerosis (MS), glial cells astrocytes interact with the autoreactive immune cells that attack the central nervous system (CNS), which causes and sustains neuroinflammation. However, little is known about the direct interaction between these cells when they are in close proximity in the inflamed CNS. By using an experimental autoimmune encephalomyelitis (EAE) model of MS, we previously found that in the proximity of autoreactive CNS-infiltrated immune cells (CNS-IICs), astrocytes respond with a rapid calcium increase that is mediated by the autocrine P2X7 receptor (P2X7R) activatio

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Fathima, Samreen, Swati Sinha, and Sainitin Donakonda. "Network Analysis Identifies Drug Targets and Small Molecules to Modulate Apoptosis Resistant Cancers." Cancers 13, no. 4 (February 18, 2021): 851. http://dx.doi.org/10.3390/cancers13040851.

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Programed cell death or apoptosis fails to induce cell death in many recalcitrant cancers. Thus, there is an emerging need to activate the alternate cell death pathways in such cancers. In this study, we analyzed the apoptosis-resistant colon adenocarcinoma, glioblastoma multiforme, and small cell lung cancers transcriptome profiles. We extracted clusters of non-apoptotic cell death genes from each cancer to understand functional networks affected by these genes and their role in the induction of cell death when apoptosis fails. We identified transcription factors regulating cell death genes a

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Wang, Jun, Douglas Tham, Wei Wei, Young Shik Shin, Chao Ma, Habib Ahmad, Qihui Shi, Jenkan Yu, Raphael D. Levine, and James R. Heath. "Quantitating Cell–Cell Interaction Functions with Applications to Glioblastoma Multiforme Cancer Cells." Nano Letters 12, no. 12 (November 7, 2012): 6101–6. http://dx.doi.org/10.1021/nl302748q.

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Tay, Neil Q., Tiffany Juan, Joseph J. Muldoon, Justin Eyquem, and Michael T. McManus. "Abstract 6633: Tracking cell-cell interactions using intercellular barcode transfer." Cancer Research 84, no. 6_Supplement (March 22, 2024): 6633. http://dx.doi.org/10.1158/1538-7445.am2024-6633.

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Abstract Cell-cell interaction is one of the fundamental biological mechanisms by which cells communicate and is a key modality by which cancer cells interact with other cells in their microenvironment. Current methods, such as enzymatic labeling and proximity-based tagging, allow the experimental tracking of these interactions but are not compatible with sequencing-based readouts, which are essential for high-throughput analyses. We have developed a highly scalable intercellular barcode transfer technology that bridges this gap. Our novel “Relay” technology offers a scalable solution to monit

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Inagaki, Naoki, Hirokazu Kawasaki, and Hiroichi Nagai. "Regulation of mast cell activation through cell to cell interaction." Japanese Journal of Pharmacology 67 (1995): 72. http://dx.doi.org/10.1016/s0021-5198(19)46258-6.

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Bowerman, B., F. E. Tax, J. H. Thomas, and J. R. Priess. "Cell interactions involved in development of the bilaterally symmetrical intestinal valve cells during embryogenesis in Caenorhabditis elegans." Development 116, no. 4 (December 1, 1992): 1113–22. http://dx.doi.org/10.1242/dev.116.4.1113.

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We describe two different cell interactions that appear to be required for the proper development of a pair of bilaterally symmetrical cells in Caenorhabditis elegans called the intestinal valve cells. Previous experiments have shown that at the beginning of the 4-cell stage of embryogenesis, two sister blastomeres called ABa and ABp are equivalent in development potential. We show that cell interactions between ABp and a neighboring 4-cell-stage blastomere called P2 distinguish the fates of ABa and ABp by inducing descendants of ABp to produce the intestinal valve cells, a cell type not made

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