Relevant bibliographies by topics / Cell Line, Tumor

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cell Line, Tumor'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Cell Line, Tumor"

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Lichtenauer, Urs D., Igor Shapiro, Klaus Geiger, et al. "Side Population Does Not Define Stem Cell-Like Cancer Cells in the Adrenocortical Carcinoma Cell Line NCI h295R." Endocrinology 149, no. 3 (2007): 1314–22. http://dx.doi.org/10.1210/en.2007-1001.

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Recent evidence suggests the existence of a stem cell-like subpopulation of cells in hematological and solid tumor entities, which determine the malignant phenotype of a given tumor through their proliferative potential and chemotherapy resistance. A recently used technique for the isolation of this cell population is through exclusion of the vital dye Hoechst 33342, which defines the so-called side population (SP). Herein we demonstrate the presence of SP cells in a variety of adrenal specimens, including primary cultures of human adrenocortical tumors and normal adrenal glands as well as est
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Crawford, Sarah Adelaide. "Microvesicles Secreted by Glioblastoma Multiforme DBTRG-05MG Tumor Cell Line Contain Proteins Involved in Tumor Invasion, Stemness and Immunosuppression." Cancer Research and Cellular Therapeutics 4, no. 1 (2020): 01–09. http://dx.doi.org/10.31579/2640-1053/067.

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Lapointe, Henry, Howard Lampe, and Diponkar Banerjee. "Head and Neck Squamous Cell Carcinoma Cell Line-Induced Suppression of in vitro Lymphocyte Proliferative Responses." Otolaryngology–Head and Neck Surgery 106, no. 2 (1992): 149–58. http://dx.doi.org/10.1177/019459989210600205.

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Tumour-infiltrating lymphocytes (TILS) are often difficult to expand in vitro. In some cases this has been attributable to immunosuppression mediated by the elaboration of prostaglandins by either tumor cells or tumor-infiltrating monocytes. In this laboratory, freshly prepared TILs containing single-cell suspensions of head and neck tumors displayed both poor proliferation as well as minimal responsiveness to indomethacin-mediated reversal of immunosuppression. In order to investigate tumor-mediated immunosuppression further, a system was developed whereby a new cell line of head and neck squ
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Magatti, M., S. De Munari, E. Vertua, S. Acali, and O. Parolini. "Amniotic mesenchymal tissue cells inhibit tumor cell line proliferation." Placenta 32 (October 2011): S336. http://dx.doi.org/10.1016/j.placenta.2011.07.063.

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Bagriacik, Emin Umit, Mustafa Kemali Baykaner, Melek Yaman, et al. "Establishment of a Primary Pleomorphic Xanthoastrocytoma Cell Line." Neurosurgery 70, no. 1 (2011): 188–97. http://dx.doi.org/10.1227/neu.0b013e3182262c5b.

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Abstract BACKGROUND Anaplastic pleomorphic xanthoastrocytoma is an aggressively growing, malignant, and eventually fatal tumor of the central nervous system. Testing chemotherapeutic drug sensitivity under in vitro conditions would be a useful strategy to determine sensitive or resistant drugs for fatal brain cancers. OBJECTIVE To establish primary cell cultures of excised tumor tissue from pleomorphic xanthoastrocytoma–bearing patients and to test their sensitivity against various anticancer chemotherapy drugs. METHODS Prepared suspensions of the excised tumor tissue from a patient who had a
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Bremmer, Felix, Hanibal Bohnenberger, Stefan Küffer, et al. "Proteomic Comparison of Malignant Human Germ Cell Tumor Cell Lines." Disease Markers 2019 (September 3, 2019): 1–14. http://dx.doi.org/10.1155/2019/8298524.

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Malignant germ cell tumors (GCT) are the most common malignant tumors in young men between 18 and 40 years. The correct identification of histological subtypes, in difficult cases supported by immunohistochemistry, is essential for therapeutic management. Furthermore, biomarkers may help to understand pathophysiological processes in these tumor types. Two GCT cell lines, TCam-2 with seminoma-like characteristics, and NTERA-2, an embryonal carcinoma-like cell line, were compared by a quantitative proteomic approach using high-resolution mass spectrometry (MS) in combination with stable isotope
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Chang, Inyoub, Takbum Ohn, Daeun Moon, Young Hee Maeng, Bo Gun Jang, and Sang-Pil Yoon. "SNU-333 Cells as an Appropriate Cell Line for the Orthotopic Renal Cell Carcinoma Model." Technology in Cancer Research & Treatment 20 (January 2021): 153303382110384. http://dx.doi.org/10.1177/15330338211038487.

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Objective: To investigate a feasible candidate for an appropriate cell line for the orthotopic renal cell carcinoma (RCC) model. Methods: Normal human proximal tubule cells (HK-2) and RCC cells were used for MTT assay, Western blotting, sphere-forming assay, and orthotopic injection of BALB/c-nude mice. Immunohistochemistry was adopted in tissue arrays and orthotopic tumors. Results: Primary RCC cells showed resistance to a GPX4 inhibitor compared to HK-2 and to metastatic RCC cells, Caki-1. Caki-2 and SNU-333 cells showed resistance to ferroptosis via increased GPX4 and FTH1, respectively. RC
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Ross, Douglas T., and Charles M. Perou. "A Comparison of Gene Expression Signatures from Breast Tumors and Breast Tissue Derived Cell Lines." Disease Markers 17, no. 2 (2001): 99–109. http://dx.doi.org/10.1155/2001/850531.

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Cell lines derived from human tumors have historically served as the primary experimental model system for exploration of tumor cell biology and pharmacology. Cell line studies, however, must be interpreted in the context of artifacts introduced by selection and establishment of cell linesin vitro. This complication has led to difficulty in the extrapolation of biology observed in cell lines to tumor biologyin vivo. Modern genomic analysis tool like DNA microarrays and gene expression profiling now provide a platform for the systematic characterization and classification of both cell lines and
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Shamova, T. V., A. O. Sitkovskaya, E. E. Rostorguev, N. S. Kuznetsova, and S. E. Kavitsky. "Preparation of primary glial tumor cell lines." Perm Medical Journal 37, no. 5 (2021): 79–89. http://dx.doi.org/10.17816/pmj37579-89.

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Objective. The aim of this work was to obtain the primary cell lines of brain malignant tumors using the explant method.
 Materials and methods. Thirteen patients of both sexes, aged 22 to 66, were recruited. The tumor material of the patients was fragmented and placed in flasks with complete nutrient medium for glial tumor cells. Subsequently, the material was photographed at various stages of cultivation, the cell morphology was determined, and the rate of monolayer formation at the zero and first passages was assessed.
 Results. As a result, thirteen primary human cell lines of gl
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Shamova, T. V., A. O. Sitkovskaya, E. E. Rostorguev, N. S. Kuznetsova, and S. E. Kavitsky. "Preparation of primary glial tumor cell lines." Perm Medical Journal 37, no. 5 (2021): 79–89. http://dx.doi.org/10.17816/pmj37579-89.

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Objective. The aim of this work was to obtain the primary cell lines of brain malignant tumors using the explant method.
 Materials and methods. Thirteen patients of both sexes, aged 22 to 66, were recruited. The tumor material of the patients was fragmented and placed in flasks with complete nutrient medium for glial tumor cells. Subsequently, the material was photographed at various stages of cultivation, the cell morphology was determined, and the rate of monolayer formation at the zero and first passages was assessed.
 Results. As a result, thirteen primary human cell lines of gl
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Dissertations / Theses on the topic "Cell Line, Tumor"

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Justiniano, Steven E. "IN VITRO SCREEN FOR ROLES OF Drosophila melanogaster TUMOR SUPPRESSORS IN CELL LINE ESTABLISHMENT AND DIFFERENTIAL EXPRESSION ANALYSIS OF Drosophila melanogaster CELL LINES." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1221919360.

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HOSHINO, MUNEMITSU, MUTSUSHI MATSUYAMA, OSAMU TAGUCHI, et al. "Establishment and Characterization of Immortalized Non-Transplantable Mouse Mammary Cell Lines Cloned from a MMTV-induced Tumor Cell Line Cultured for A Long Duration." Nagoya University School of Medicine, 1991. http://hdl.handle.net/2237/17515.

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Patrick, James Lambert. "Computer Aided Analysis of Restriction Landmark Genomic Scanning Images from Tumor and Cell Line Models." University of Toledo Health Science Campus / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=mco1196365469.

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Liao, Nan. "The unfolded protein response increases production of pro-angiogenic factors by tumor cell lines." View the abstract Download the full-text PDF version (on campus access only), 2008. http://etd.utmem.edu/ABSTRACTS/2008-008-Liao-index.html.

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Thesis (M.S. )--University of Tennessee Health Science Center, 2008.<br>Title from title page screen (viewed on July 17, 2008). Research advisor: Linda M. Hendershot, Ph.D. Document formatted into pages (x, 57 p. : ill.). Vita. Abstract. Includes bibliographical references (p. 47-57).
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Ciesiolka, Malgorzata. "Studies of tax-mediated activation of BLV expression in silently infected ovine tumor B cell line." Doctoral thesis, Universite Libre de Bruxelles, 1998. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/212024.

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Bérubé, Nathalie Guylaine. "Tumor suppression of the DU 145 prostate cancer cell line: Implication of chromosomes 8 and 12." Thesis, University of Ottawa (Canada), 1996. http://hdl.handle.net/10393/9782.

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Tumorigenesis results from the accumulation of genetic events which give rise to inappropriate behaviour of cells. Mutations which lead to cancer progression are generally classified in two major categories, that of oncogenes and tumor suppressor genes. The latter have been more difficult to identify due to their recessive nature. The application of somatic cell fusion and microcell fusion technologies presents a means to identify genes inactivated in tumor cells. We have used these methodologies to identify specific chromosomes that, once introduced into the DU 145 prostate cancer cell line,
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Liu, Zhiwen. "Matrix metalloproteases and cell motility in malignant mesothelioma /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-061-3/.

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Kim, Ji-Eun 1974. "Regulation of tumor necrosis factor-alpha induced apoptosis via posttranslational modifications in a human colon adenocarcinoma cell line." Thesis, Massachusetts Institute of Technology, 2004. http://hdl.handle.net/1721.1/28865.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Biological Engineering Division, 2004.<br>Includes bibliographical references.<br>(cont.) phosphoproteomics technology, IMAC/LC/MS/MS, [approximately] 200 phosphosites were identified from HT-29 cells, some of which were detected only from insulin-treated cells. Our phosphoproteomics approach also enabled us to detect alteration of both known and unknown phosphorylation states of apoptosis-related proteins at two time points during early apoptosis induced by tumor necrosis factor-α<br>Apoptosis, a physiologically regulated cell death, pla
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Athanassiou-Papaefthymiou, Maria G. "Functional activation of the p53 tumor suppressor in non-tumorigenic variants of the HeLa cervical carcinoma cell line." Thesis, Massachusetts Institute of Technology, 1996. http://hdl.handle.net/1721.1/39376.

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Okamoto, Shinya. "Overexpression of Fas Ligand Dose Not Confer Immune Privilege to a Pancreatic β Tumor cell Line(βTC-3)". Kyoto University, 2000. http://hdl.handle.net/2433/180854.

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Books on the topic "Cell Line, Tumor"

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Brail, Leslie Harris. Heterogeneity of plasminogen activator activity produced by two murine tumour cell lines. National Library of Canada, 1994.

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Weinstein, Ellen Michele. Inhibition of CA 125, a novel high molecular weight glycoprotein expressed by an ovarian carcinoma cell line: 0VCA 433, is related to glucocorticoid effects of altered cell growth, morphology, and growth pattern. s.n.], 1988.

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Turnbull, Heather. Biochemical and physiological characterization of newly established drug-resistant MCF-7 breast tumour cell lines. Laurentian University, 2000.

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Al-Hakim, Abdallah. A role for the type 4 adenylyl cyclase isoform in forskolin-response pathway of Y1 mouse adrencortical tumor cell lines. National Library of Canada, 2003.

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service), SpringerLink (Online, ed. Insulin-like Growth Factors and Cancer: From Basic Biology to Therapeutics. Springer Science+Business Media, LLC, 2012.

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1938-, Hay Robert, Park Jae-Gahb, and Gazdar Adi F, eds. Atlas of human tumor cell lines. Academic Press, 1994.

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Drexler, Hans G. The Leukemia-Lymphoma Cell Line Factsbook. Academic Press, 2000.

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Drexler, Hans G. The Leukemia-Lymphoma Cell Line Factsbook. Academic Press, 2000.

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B, Roninson Igor, ed. Molecular and cellular biology of multidrug resistance in tumor cells. Plenum Press, 1991.

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Moerdler, Scott, and Xingxing Zang. PD-1/PDL-1 Inhibitors as Immunotherapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0010.

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Programmed death 1 (PD-1), a member of the B7-CD28 immunoglobulin superfamily, and its ligands PD-L1/PD-L2 inhibit T-cell activation. They also play a key role in the tumor microenvironment, allowing for cancer immune escape. PD-1 is induced on a variety of immune cells, including tumor-infiltrating lymphocytes (TILs), while PD-L1 is found on many types of solid tumors including ovarian cancer and some TILs. The use of immunocheckpoint inhibitors like anti-PD-1 and anti-PD-L1 therapies has been shown to reactivate the immune system to attack tumor cells. Ovarian cancers have been shown to be r
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Book chapters on the topic "Cell Line, Tumor"

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Meditz, Katharina, and Beate Rinner. "Establishment of Tumor Cell Lines: From Primary Tumor Cells to a Tumor Cell Line." In Cell Culture Technology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-74854-2_4.

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Boyd, Michael R. "The NCI Human Tumor Cell Line (60-Cell) Screen." In Anticancer Drug Development Guide. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-739-0_3.

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Offner, F. A., G. Ott, L. Füzesi, et al. "Analysis of Tumor Heterogeneity in Monolayers and Tumor Spheroids of a Transitional Cell Carcinoma Cell Line." In Investigative Urology 4. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75972-7_18.

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Rahman, Mohsen, Herman Yeger, and Laurence E. Becker. "In vivo characterization of a human neuroectodermal tumor cell line." In Biology of Brain Tumour. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_15.

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Vaupel, P., and W. Mueller-Klieser. "Cell Line and Growth Site as Relevant Parameters Governing Tumor Tissue Oxygenation." In Oxygen Transport to Tissue VIII. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5188-7_76.

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Chang, Y. S., S. Y. Lin, P. F. Lee, H. C. Chung, and M. S. Tsai. "Establishment and Characterization of a Tumor Cell Line from Nasopharyngeal Carcinoma Tissue." In Epstein-Barr Virus and Human Disease • 1988. Humana Press, 1989. http://dx.doi.org/10.1007/978-1-4612-4508-7_11.

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Campbell, James, Colm J. Ryan, and Christopher J. Lord. "Identifying Genetic Dependencies in Cancer by Analyzing siRNA Screens in Tumor Cell Line Panels." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7493-1_5.

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Kaneko, Junichi, Yoshinori Inagaki, Takeaki Ishizawa, and Norihiro Kokudo. "Near-Infrared Laser Photodynamic Therapy for Human Hepatocellular Carcinoma Cell Line Tumor with Indocyanine Green Fluorescence." In Fluorescence Imaging for Surgeons. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-15678-1_19.

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Afzal, S. M. J., and R. P. Liburdy. "Magnetic Fields Reduce the Growth Inhibitory Effects of Tamoxifen in a Human Brain Tumor Cell Line." In Electricity and Magnetism in Biology and Medicine. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4867-6_111.

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Kalil, Ricardo K. "Langerhans Cell Histiocytosis." In Tumors and Tumor-Like Lesions of Bone. Springer London, 2015. http://dx.doi.org/10.1007/978-1-4471-6578-1_58.

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Conference papers on the topic "Cell Line, Tumor"

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Esumi, N., S. Todo, and S. Imashuku. "INTERACTION BETWEEN HEMOSTATIC COMPONENTS AND TUMOR CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643202.

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Involvement of platelets and coagulation systems in the hematogenous metastasis of tumor cells has been suggested from in vivo and in vitro studies, however, there is still controversy about the exact role of hemostasis in metastasis. To date, at least three types of platelet aggregating mechanisms and three types of tumor cell procoagulants have been reported in different tumor cells.We investigated platelet aggregating activity (PAA), procoagulant activity (PCA) and the relationship between these two activities, using eight human neuroblastoma cell lines, three human leukemia cell lines and
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Laug, Walter E. "HETEROGENOUS EXPRESSION OF PLASMINOGEN ACTIVATOR (PA) GENES IN THE HUMAN SARCOMA CELL LINE HT1080." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644395.

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Tumor cell derived PA activities are of crucial importance for tissue invasion and destruction by tumor cells. Therefore, we studied the expression of the PA genes in HT1080 cells using immunoenzymatic methods and specific PA gene probes.Immunenzymatic methods allowed only for the detection of urokinase like PA (u-PA) activities in HT1080 cells which was suppressed by treatment of the cells with dexamethasone (10-7 m). Despite the lack of u-PA activities, the cells still degraded extracellular tissue glycoproteins. Northern blot analysis with specific PA gene probe showed that HT1080 cells exp
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O'Day, Christine, Yulia Ovechkina, Karen Marcoe, et al. "240 human tumor cell line profiling to evaluate relationships between tumor genotypes and cancer cell sensitivity." In AACR International Conference: Molecular Diagnostics in Cancer Therapeutic Development– Sep 27-30, 2010; Denver, CO. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/diag-10-a1.

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Stuelten, Christina H., Susan D. Mertins, Johanna I. Busch, et al. "Abstract 3372: Analysis of putative tumor stem cell markers in the NCI-60 tumor cell line panel." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3372.

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Jamieson, G. A., and G. Grignani. "GENERATION OF ADP BY HUMAN AND MURINE TUMOR CELLS IS SPECIFIC BUT IS UNRELATED TO METASTATIC POTENTIAL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643204.

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The ability of tumor cells to activate platelets may facilitate the metastatic process. It has been generally assumed that the production of ADP by tumor cells is due to non-specific damage during harvesting in vitro or, in vivo, by frictional interactions with the capillary wall. The present work shows that tumor cell ADP arises not from cell damage but by a specific process under metabolic control. The human 253J urinary carcinoma cell line activated heparinized human platelets by an ADP-dependent mechanism based on inhibition by CP/CPK and the identification of aggregating concentrations (1
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Ovechkina, Yulia Y., Christine O'Day, Karen Marcoe, et al. "Abstract 4845: Tumor cell line profiling of eighteen cancer therapeutics to evaluate relationships between tumor genotypes and cancer cell sensitivities." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4845.

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Sung, Pi-Lin, Chi-Ying F. Huang, Michael Hsiao, Kuo-Chang Wen, Ming-Shyen Yen, and Mong-Hong Lee. "Abstract 1922: Target mutation comparasion of WGA circulating tumor cell and primary tumor in epithelial ovarian cancer cell line using NGS." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1922.

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Arnoux, D., B. Boutière, N. Pourreau-Schneider, P. Martin, and J. Sampol. "PLASMINOGEN ACTIVATORS (t-PA and u-PA) IN HUMAN NEOPLASTIC CELL LINES AND THEIR MODULATION BY BASEMENT MEMBRANE COMPONENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643190.

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Plasminogen activators (PA)may play an important role in the regulation of enzyme activation relative to basement membrane degradation associated with the invasive growth of tumors. In order to acquire a better understanding of the complex cascade reactions leading to the formation of plasmin, we have undertaken a comparative study of urokinase-type (u-PA) and tissue-type (t-PA) plasminogen activators in cellular extracts of 20 human cancer cell lines (13 malignant melanomas, 6 breast adenocarcinomas and 1 vulvar carcinoma). Four malignant cell lines,showing various t-PA or u-PA activity level
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Western, Laura T., Kuldeepsinh Rana, and Michael R. King. "Flow-Based Isolation and Neutralization of Circulating Tumor Cells." In ASME 2009 7th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2009. http://dx.doi.org/10.1115/icnmm2009-82137.

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Circulating tumor cells (CTC) have the potential to be used clinically as a diagnostic tool and a treatment tool in the field of oncology. As a diagnostic tool, CTC may be used to indicate the presence of a tumor before the tumor is large enough to cause noticeable symptoms. As a treatment tool, CTC isolated from patients may be used to test the efficacy of chemotherapy options to personalize patient treatment. One way for tumors to spread is through metastasis via the circulatory system. CTC are able to exploit the natural leukocyte recruitment process that is initially mediated by rolling on
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Bastida, E., and L. Almirall. "EFFECTS OF 13-H0DE AND HETEs ON TUMOR CELL/ENDOTHELIAL CELL INTERACTIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643947.

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We and others reported that endothelial cells (ECs) convert linoleic acid into 13-hydroxyoctadecadienoic acid (13-H0DE) under basal conditions, and arachidonic acid into 15-hydroxyeicosatet-raenoic acid (15-HETE) following stimulation (1,2). We also reported that lipoxygenase metabolism influenced platelet (PLT) interactions with ECs, tumor cells (TCs) and extracellular matrix (BM) (1,3,4). Thus, we performed studies to determine i) if TCs also produce 13-H0DE and HETEs, and ii) the effect of TC and EC 13-H0DE and HETEs synthesis on TC/EC adhesion. We measured i) the ratios of 13-H0DE:HETE in
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Reports on the topic "Cell Line, Tumor"

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Condeelis, John. Isolation of Motile Tumor Cells From Live Breast Tumors. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada395259.

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Condeelis, John. Isolation of Motile Tumor Cells from Live Breast Tumors. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada412991.

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Furbert-Harris, Paulette. Growth Inhibition of Breast Tumor Cells by Hypodense and Normodense Eosinophilic Cell Lines. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada394003.

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Furbert-Harris, Paulette M. Growth Inhibition of Breast Tumor Cells by Hypodense and Normodense Eosinophilic Cell Lines. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383068.

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Vogel, Kristine S. Cell Motility and Invasiveness of Neurofibromin-Deficient Neural Crest Cells and Malignant Triton Tumor Lines. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada439284.

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Vogel, Kristine S. Cell Motility and Invasiveness of Neurotibromin-Deficient Neural Crest Cells and Malignant Triton Tumor Lines. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411714.

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Vogel, Kristine S. Cell Motility and Invasiveness of Neurofibromin-Deficient Neural Crest Cells and Malignant Triton Tumor Lines. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada422403.

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Collart, F. R., C. B. Chubb, B. L. Mirkin, and E. Huberman. Increased IMP dehydrogenase gene expression in solid tumor tissues and tumor cell lines. Office of Scientific and Technical Information (OSTI), 1992. http://dx.doi.org/10.2172/10148922.

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Vogel, Kristine S. Cell Motility and Invasiveness of Neurofibromin-Deficient Neural Crest Cells and Malignant Triton Tumor Lines. Addendum. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada458421.

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Griend, Donald Vander. Isolation and Growth of Prostate Stem Cells and Establishing Cancer Cell Lines from Human Prostate Tumors. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada511041.

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