Relevant bibliographies by topics / Cell organelle

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Cell organelle'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Cell organelle"

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Ouellet, Jimmy, and Yves Barral. "Organelle segregation during mitosis: Lessons from asymmetrically dividing cells." Journal of Cell Biology 196, no. 3 (February 6, 2012): 305–13. http://dx.doi.org/10.1083/jcb.201102078.

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Studies on cell division traditionally focus on the mechanisms of chromosome segregation and cytokinesis, yet we know comparatively little about how organelles segregate. Analysis of organelle partitioning in asymmetrically dividing cells has provided insights into the mechanisms through which cells control organelle distribution. Interestingly, these studies have revealed that segregation mechanisms frequently link organelle distribution to organelle growth and formation. Furthermore, in many cases, cells use organelles, such as the endoplasmic reticulum and P granules, as vectors for the segregation of information. Together, these emerging data suggest that the coordination between organelle growth, division, and segregation plays an important role in the control of cell fate inheritance, cellular aging, and rejuvenation, i.e., the resetting of age in immortal lineages.
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TANAKA, Arowu, Fumi KANO, and Masayuki MURATA. "Organelle Inheritance-Cell Cycle Dependent Dynamics of Organelles in Mammalian Cells." Seibutsu Butsuri 42, no. 3 (2002): 116–21. http://dx.doi.org/10.2142/biophys.42.116.

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Evans, David E., and Chris Hawes. "Organelle Biogenesis and Positioning in Plants." Biochemical Society Transactions 38, no. 3 (May 24, 2010): 729–32. http://dx.doi.org/10.1042/bst0380729.

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The biogenesis and positioning of organelles involves complex interacting processes and precise control. Progress in our understanding is being made rapidly as advances in analysing the nuclear and organellar genome and proteome combine with developments in live-cell microscopy and manipulation at the subcellular level. This paper introduces the collected papers resulting from Organelle Biogenesis and Positioning in Plants, the 2009 Biochemical Society Annual Symposium. Including papers on the nuclear envelope and all major organelles, it considers current knowledge and progress towards unifying themes that will elucidate the mechanisms by which cells generate the correct complement of organelles and adapt and change it in response to environmental and developmental signals.
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Mallo, Natalia, Justin Fellows, Carla Johnson, and Lilach Sheiner. "Protein Import into the Endosymbiotic Organelles of Apicomplexan Parasites." Genes 9, no. 8 (August 14, 2018): 412. http://dx.doi.org/10.3390/genes9080412.

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: The organelles of endosymbiotic origin, plastids, and mitochondria, evolved through the serial acquisition of endosymbionts by a host cell. These events were accompanied by gene transfer from the symbionts to the host, resulting in most of the organellar proteins being encoded in the cell nuclear genome and trafficked into the organelle via a series of translocation complexes. Much of what is known about organelle protein translocation mechanisms is based on studies performed in common model organisms; e.g., yeast and humans or Arabidopsis. However, studies performed in divergent organisms are gradually accumulating. These studies provide insights into universally conserved traits, while discovering traits that are specific to organisms or clades. Apicomplexan parasites feature two organelles of endosymbiotic origin: a secondary plastid named the apicoplast and a mitochondrion. In the context of the diseases caused by apicomplexan parasites, the essential roles and divergent features of both organelles make them prime targets for drug discovery. This potential and the amenability of the apicomplexan Toxoplasma gondii to genetic manipulation motivated research about the mechanisms controlling both organelles’ biogenesis. Here we provide an overview of what is known about apicomplexan organelle protein import. We focus on work done mainly in T. gondii and provide a comparison to model organisms.
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Pelham, R. J., J. J. Lin, and Y. L. Wang. "A high molecular mass non-muscle tropomyosin isoform stimulates retrograde organelle transport." Journal of Cell Science 109, no. 5 (May 1, 1996): 981–89. http://dx.doi.org/10.1242/jcs.109.5.981.

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Although non-muscle tropomyosins (TM) have been implicated in various cellular functions, such as stabilization of actin filaments and possibly regulation of organelle transport, their physiological role is still poorly understood. We have probed the role of a high molecular mass isoform of human fibroblast TM, hTM3, in regulating organelle transport by microinjecting an excess amount of bacterially-expressed protein into normal rat kidney (NRK) epithelial cells. The microinjection induced the dramatic retrograde translocation of organelles into the perinuclear area. Microinjection of hTM5, a low molecular mass isoform had no effect on organelle distribution. Fluorescent staining indicated that hTM3 injection stimulated the retrograde movement of both mitochondria and lysosomes. Moreover, both myosin I and cytoplasmic dynein were found to redistribute with the translocated organelles to the perinuclear area, indicating that these organelles were able to move along both microtubules and actin filaments. The involvement of microtubules was further suggested by the partial inhibition of hTM3-induced organelle movement by the microtubule-depolymerizing drug nocodazole. Our results, along with previous genetic and antibody microinjection studies, suggest that hTM3 may be involved in the regulation of organelle transport.
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Esch, Nicholas, Seokwon Jo, Mackenzie Moore, and Emilyn U. Alejandro. "Nutrient Sensor mTOR and OGT: Orchestrators of Organelle Homeostasis in Pancreatic β-Cells." Journal of Diabetes Research 2020 (December 15, 2020): 1–24. http://dx.doi.org/10.1155/2020/8872639.

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The purpose of this review is to integrate the role of nutrient-sensing pathways into β-cell organelle dysfunction prompted by nutrient excess during type 2 diabetes (T2D). T2D encompasses chronic hyperglycemia, hyperlipidemia, and inflammation, which each contribute to β-cell failure. These factors can disrupt the function of critical β-cell organelles, namely, the ER, mitochondria, lysosomes, and autophagosomes. Dysfunctional organelles cause defects in insulin synthesis and secretion and activate apoptotic pathways if homeostasis is not restored. In this review, we will focus on mTORC1 and OGT, two major anabolic nutrient sensors with important roles in β-cell physiology. Though acute stimulation of these sensors frequently improves β-cell function and promotes adaptation to cell stress, chronic and sustained activity disturbs organelle homeostasis. mTORC1 and OGT regulate organelle function by influencing the expression and activities of key proteins, enzymes, and transcription factors, as well as by modulating autophagy to influence clearance of defective organelles. In addition, mTORC1 and OGT activity influence islet inflammation during T2D, which can further disrupt organelle and β-cell function. Therapies for T2D that fine-tune the activity of these nutrient sensors have yet to be developed, but the important role of mTORC1 and OGT in organelle homeostasis makes them promising targets to improve β-cell function and survival.
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Hertle, Alexander P., Benedikt Haberl, and Ralph Bock. "Horizontal genome transfer by cell-to-cell travel of whole organelles." Science Advances 7, no. 1 (January 2021): eabd8215. http://dx.doi.org/10.1126/sciadv.abd8215.

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Recent work has revealed that both plants and animals transfer genomes between cells. In plants, horizontal transfer of entire plastid, mitochondrial, or nuclear genomes between species generates new combinations of nuclear and organellar genomes, or produces novel species that are allopolyploid. The mechanisms of genome transfer between cells are unknown. Here, we used grafting to identify the mechanisms involved in plastid genome transfer from plant to plant. We show that during proliferation of wound-induced callus, plastids dedifferentiate into small, highly motile, amoeboid organelles. Simultaneously, new intercellular connections emerge by localized cell wall disintegration, forming connective pores through which amoeboid plastids move into neighboring cells. Our work uncovers a pathway of organelle movement from cell to cell and provides a mechanistic framework for horizontal genome transfer.
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Okamoto, Koji. "Organellophagy: Eliminating cellular building blocks via selective autophagy." Journal of Cell Biology 205, no. 4 (May 26, 2014): 435–45. http://dx.doi.org/10.1083/jcb.201402054.

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Maintenance of organellar quality and quantity is critical for cellular homeostasis and adaptation to variable environments. Emerging evidence demonstrates that this kind of control is achieved by selective elimination of organelles via autophagy, termed organellophagy. Organellophagy consists of three key steps: induction, cargo tagging, and sequestration, which involve signaling pathways, organellar landmark molecules, and core autophagy-related proteins, respectively. In addition, posttranslational modifications such as phosphorylation and ubiquitination play important roles in recruiting and tailoring the autophagy machinery to each organelle. The basic principles underlying organellophagy are conserved from yeast to mammals, highlighting its biological relevance in eukaryotic cells.
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Walsh, J. B. "Intracellular selection, conversion bias, and the expected substitution rate of organelle genes." Genetics 130, no. 4 (April 1, 1992): 939–46. http://dx.doi.org/10.1093/genetics/130.4.939.

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Abstract A key step in the substitution of a new organelle mutant throughout a population is the generation of germ-line cells homoplasmic for that mutant. Given that each cell typically contains multiple copies of organelles, each of which in turn contains multiple copies of the organelle genome, processes akin to drift and selection in a population are responsible for producing homoplasmic cells. This paper examines the expected substitution rate of new mutants by obtaining the probability that a new mutant is fixed throughout a cell, allowing for arbitrary rates of genome turnover within an organelle and organelle turnover within the cell, as well as (possibly biased) gene conversion and genetic differences in genome and/or organelle replication rates. Analysis is based on a variation of Moran's model for drift in a haploid population. One interesting result is that if the rate of unbiased conversion is sufficiently strong, it creates enough intracellular drift to overcome even strong differences in the replication rates of wild-type and mutant genomes. Thus, organelles with very high conversion rates are more resistant to intracellular selection based on differences in genome replication and/or degradation rates. It is found that the amount of genetic exchange between organelles within the cell greatly influences the probability of fixation. In the absence of exchange, biased gene conversion and/or differences in genome replication rates do not influence the probability of fixation beyond the initial fixation within a single organelle. With exchange, both these processes influence the probability of fixation throughout the entire cell. Generally speaking, exchange between organelles accentuates the effects of directional intracellular forces.(ABSTRACT TRUNCATED AT 250 WORDS)
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Steinberg, G., and M. Schliwa. "Organelle movements in the wild type and wall-less fz;sg;os-1 mutants of Neurospora crassa are mediated by cytoplasmic microtubules." Journal of Cell Science 106, no. 2 (October 1, 1993): 555–64. http://dx.doi.org/10.1242/jcs.106.2.555.

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The cellular basis of organelle transport in filamentous fungi is still unresolved. Here we have studied the intracellular movement of mitochondria and other organelles in the fungus Neurospora crassa. Four different model systems were employed: hyphae, protoplasts, a cell wallless mutant, and experimentally generated small, flattened cell fragments of the mutant cells. Organelle movements were visualized by DIC optics and computer-enhanced video microscopy. In all cell models the transport of organelles was vectorial and saltatory in nature. The mean velocities for mitochondria, particles and nuclei were 1.4, 2.0, and 0.9 microns/s, respectively. Treatment with 10 microM nocodazole for 30 minutes caused a complete disappearance of microtubules and reversibly blocked directed transport of virtually all organelles, whereas cytochalasin D up to 20 microM was without effect. Correlative video and immunofluorescence microscopy of small fragments of wall-less mutant cells revealed a clear match between microtubule distribution and the tracks of moving organelles. We conclude that organelle movement in the filamentous fungus Neurospora crassa is a microtubule-dependent process.
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Dissertations / Theses on the topic "Cell organelle"

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Mayer, Jürgen. "Investigation of the biophysical basis for cell organelle morphology." Master's thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2010. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-26600.

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It is known that fission yeast Schizosaccharomyces pombe maintains its nuclear envelope during mitosis and it undergoes an interesting shape change during cell division - from a spherical via an ellipsoidal and a peanut-like to a dumb-bell shape. However, the biomechanical system behind this amazing transformation is still not understood. What we know is, that the shape must change due to forces acting on the membrane surrounding the nucleus and the microtubule based mitotic spindle is thought to play a key role. To estimate the locations and directions of the forces, the shape of the nucleus was recorded by confocal light microscopy. But such data is often inhomogeneously labeled with gaps in the boundary, making classical segmentation impractical. In order to accurately determine the shape we developed a global parametric shape description method, based on a Fourier coordinate expansion. The method implicitly assumes a closed and smooth surface. We will calculate the geometrical properties of the 2-dimensional shape and extend it to 3-dimensional properties, assuming rotational symmetry. Using a mechanical model for the lipid bilayer and the so called Helfrich-Canham free energy we want to calculate the minimum energy shape while respecting system-specific constraints to the surface and the enclosed volume. Comparing it with the observed shape leads to the forces. This provides the needed research tools to study forces based on images.
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Mayer, Jürgen. "Investigation of the biophysical basis for cell organelle morphology." Master's thesis, Max-Planck-Institut für Molekulare Zellbiologie und Genetik, 2008. https://tud.qucosa.de/id/qucosa%3A25225.

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It is known that fission yeast Schizosaccharomyces pombe maintains its nuclear envelope during mitosis and it undergoes an interesting shape change during cell division - from a spherical via an ellipsoidal and a peanut-like to a dumb-bell shape. However, the biomechanical system behind this amazing transformation is still not understood. What we know is, that the shape must change due to forces acting on the membrane surrounding the nucleus and the microtubule based mitotic spindle is thought to play a key role. To estimate the locations and directions of the forces, the shape of the nucleus was recorded by confocal light microscopy. But such data is often inhomogeneously labeled with gaps in the boundary, making classical segmentation impractical. In order to accurately determine the shape we developed a global parametric shape description method, based on a Fourier coordinate expansion. The method implicitly assumes a closed and smooth surface. We will calculate the geometrical properties of the 2-dimensional shape and extend it to 3-dimensional properties, assuming rotational symmetry. Using a mechanical model for the lipid bilayer and the so called Helfrich-Canham free energy we want to calculate the minimum energy shape while respecting system-specific constraints to the surface and the enclosed volume. Comparing it with the observed shape leads to the forces. This provides the needed research tools to study forces based on images.
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Dolman, Nicholas James. "Polarised signalling and organelle distribution in the pancreatic acinar cell." Thesis, University of Liverpool, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406669.

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Malchus, Nina Isabelle [Verfasser], and Michael [Akademischer Betreuer] Hausmann. "On the spatial organization of cell organelles and diffusion of proteins in organelle membranes / Nina Isabelle Malchus ; Betreuer: Michael Hausmann." Heidelberg : Universitätsbibliothek Heidelberg, 2011. http://d-nb.info/1179230477/34.

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Fujisawa, Alma. "Development of chemical labeling methods for organelle molecule analysis." Kyoto University, 2019. http://hdl.handle.net/2433/243315.

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Nilsson, Harriet. "The role of nitric oxide in cytoskeleton-mediated organelle transport and cell adhesion /." Linköping : Univ, 2001. http://www.bibl.liu.se/liupubl/disp/disp2001/med660s.pdf.

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Ahmadi, Shiva [Verfasser]. "Application of BioID to in vitro organelle and in vivo cell-type-specific proteomics / Shiva Ahmadi." Bonn : Universitäts- und Landesbibliothek Bonn, 2020. http://nbn-resolving.de/urn:nbn:de:hbz:5-59196.

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Hatchel, Jennifer M. "Structure and Function of the Electron-dense Core in Mycoplasma pneumoniae and its Relatives." Miami University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=miami1248183957.

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Matsuzaki, Satoshi. "Hole Burning Imaging Studies of Cancerous and Analogous Normal Ovarian Tissues Utilizing Organelle Specific Dyes." Ames, Iowa : Oak Ridge, Tenn. : Ames Laboratory ; distributed by the Office of Scientific and Technical Information, U.S. Dept. of Energy, 2004. http://www.osti.gov/servlets/purl/837275-3aN4nd/webviewable/.

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Thesis (Ph.D.); Submitted to Iowa State Univ., Ames, IA (US); 19 Dec 2004.
Published through the Information Bridge: DOE Scientific and Technical Information. "IS-T 2692" Satoshi Matsuzaki. US Department of Energy 12/19/2004. Report is also available in paper and microfiche from NTIS.
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Buks, Ralfs. "Impact of JAK2V617F on Terminal Erythroid Differentiation and Red Blood Cell Electrophysiology in Polycythemia Vera." Thesis, Université de Paris (2019-....), 2020. http://www.theses.fr/2020UNIP7096.

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Les néoplasies myéloprolifératives (NMP) regroupent des pathologies caractérisées par une prolifération et une différenciation anormales des cellules souches hématopoïétiques. La polyglobulie de Vaquez (PV) est un type de NMP caractérisé par un nombre de globules rouges (GR) élevé. Plus de 95% des patients atteints de PV présentent la mutation V617F de la tyrosine kinase JAK2 conduisant à une prolifération érythroïde incontrôlée entraînant un risque élevé de thrombose. Les patients présentant un risque pronostique élevé sont traités par des thérapies cytoréductrices, dont le ruxolitinib, un inhibiteur de JAK1/JAK2. Nous avons analysé le protéome de la membrane des GR PV par spectrométrie de masse et avons montré des niveaux élevés de protéines de liaison au Ca2+ ainsi que la présence anormale de protéines du réticulum endoplasmique.Dans cette thèse, nous avons étudié (1) l'impact de JAK2V617F sur l'expression et le tri des protéines pendant la différenciation érythroïde terminale, l'énucléation et la maturation ; (2) l'impact de JAK2V617F sur l'homéostasie du calcium et l'électrophysiologie des GR dans la PV ; (3) l'import du ruxolitinib dans le compartiment intracellulaire.Nos données montrent que JAK2V617F jouerait un rôle dans la rétention des organelles pendant la phase d'énucléation des érythroblastes, ce qui pourrait perturber la maturation des réticulocytes circulants chez la souris et l'homme. Nos données obtenues par patch-clamp automatisé montrent une homéostasie calcique modifiée dans les GR PV et des lignées cellulaires exprimant JAK2V617F, avec un impact fonctionnel sur l'activité du canal Gárdos, ce qui pourrait contribuer à une déshydratation cellulaire dans un contexte JAK2V617F. Enfin, nous avons étudié le transport du ruxolitinib à l’aide de tests de cytotoxicité et d'apoptose dans des lignées cellulaires humaines, des GR et des progéniteurs érythroïdes humains différenciés in vitro. Nos résultats suggèrent que le ruxolitinib est importé dans le compartiment intracellulaire par la protéine ABCG2, un membre de la superfamille des transporteurs à « ATP binding cassette » (ABC).Étant donné le rôle central du calcium dans la régulation des voies de signalisation, notre étude ouvre de nouvelles perspectives pour explorer la relation entre JAK2V617F, l'homéostasie du calcium et les anomalies cellulaires dans les NMP telles que les interactions cellulaires dans la circulation sanguine en relation avec les événements thrombotiques. Ces interactions pourraient également être déclenchées par les propriétés anormales des GR suite à des défauts de maturation réticulocytaire. En ce qui concerne le traitement des patients, notre étude montre pour la première fois le rôle d'un transporteur spécifique dans l’import intracellulaire du ruxolitinib. Elle ouvre de nouvelles perspectives de recherche sur l'efficacité du ruxolitinib en analysant le polymorphisme d'ABCG2 et son expression sur les cellules cibles des patients traités
Myeloproliferative neoplasms (MPNs) are a group of disorders characterised by abnormal proliferation and differentiation of hematopoietic stem cells in the bone marrow. Polycythemia Vera (PV) is a type of MPN characterised by overproduction of red blood cells (RBCs). Over 95 % of PV patients carry the V617F mutation in the tyrosine kinase Janus kinase 2 (JAK2) resulting in uncontrolled erythroid proliferation and high risk of thrombosis. Patients with high prognostic risk scores are treated with cytoreductive therapies, including ruxolitinib, a JAK1/JAK2 inhibitor. Using mass spectrometry, we analysed the RBC membrane proteome and showed elevated levels of multiple Ca2+ binding proteins as well as endoplasmic reticulum residing proteins in PV RBC membranes compared to RBC membranes from healthy individuals. In this thesis we investigated:(1) the impact of JAK2V617F on protein expression and sorting during terminal erythroid differentiation, enucleation and maturation;(2) the impact of JAK2V617F on calcium homeostasis and RBC electrophysiology in PV;(3) ruxolitinib import in the intracellular compartment.Our data shows that JAK2V617F could play a role in organelle retention during the enucleation step of erythroid differentiation, which may affect maturation of circulating reticulocytes in mouse and human. Our data from automated patch-clamp shows modified calcium homeostasis in PV RBCs and cell lines expressing JAK2V617F, with a functional impact on the activity of the Gárdos channel that could contribute to cellular dehydration. Finally, we investigated ruxolitinib transport using cytotoxicity and apoptosis assays in human cell lines, RBCs and in vitro differentiated human erythroid progenitors. Our findings suggest that ruxolitinib is imported in the intracellular compartment by ABCG2, a member of the ATP-binding cassette (ABC) transmembrane superfamily.Given the central role that calcium plays in the regulation of signalling pathways, our study opens new perspectives to explore the relationship between JAK2V617F, calcium homeostasis and cellular abnormalities in MPNs, including cellular interactions in the bloodstream in relation to thrombotic events. These interactions could also be triggered by the abnormal properties of circulating RBCs secondary to the presence of organelle remnants in their membrane and cytoplasm. Regarding patients’ treatment, our study shows for the first time the role of a specific transporter in ruxolitinib cellular influx. It opens new perspectives in ruxolitinib efficacy research targeting cell types depending on ABCG2 expression and polymorphisms among patients
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Books on the topic "Cell organelle"

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Sadava, David. Cell biology: Organelle structure and function. Boston, Mass: Jones and Bartlett, 1993.

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Tazawa, M. Cell Dynamics: Cytoplasmic Streaming Cell Movement-Contraction and Migration Cell and Organelle Division Phototaxis of Cell and Cell Organelle. Vienna: Springer Vienna, 1989.

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Sadava, David E. Cell biology: Organelle structure and function. Boston: Jones and Bartlett Publishers, 1993.

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Weissig, Volkmar, and Gerard G. M. D'Souza. Organelle-specific pharmaceutical nanotechnology. Hoboken, NJ: Wiley, 2010.

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Bradshaw, Ralph A. Regulation of organelle and cell compartment signaling. Amsterdam: Elsevier/Academic Press, 2011.

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Evans, D. E. (David E.), Hawes C. R, and Biochemical Society (Great Britain), eds. Organelle biogenesis and positioning in plants. London: Portland Press, 2010.

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Herrmann, Reinhold G., ed. Cell Organelles. Vienna: Springer Vienna, 1992. http://dx.doi.org/10.1007/978-3-7091-9138-5.

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Organelles. New York: Guilford Press, 1989.

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Deutsche Gesellschaft für Zellbiologie. Meeting. Annual meeting of the Deutsche Gesellschaft für Zellbiologie: Biogenesis of organelles, ion transport, cell polarity, cell proliferation : Heidelberg, 16-20 March 1987 : abstracts. Stuttgart: Wissenschaftliche Verlagsgesellschaft, 1987.

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Deutsche Gesellschaft für Zellbiologie. Meeting. Annual meeting of the Deutsche Gesellschaft für Zellbiologie: Halle (Saale), March 16-20, 1997 : abstracts. Stuttgart: Wissenschaftliche Verlagsgesellschaft, 1997.

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Book chapters on the topic "Cell organelle"

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Bhattacharya, Anjanabha, Anish Kumar, Nirali Desai, and Seema Parikh. "Organelle Transformation." In Plant Cell Culture Protocols, 401–6. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-818-4_29.

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Nebenführ, Andreas. "Organelle Dynamics During Cell Division." In Plant Cell Monographs, 195–206. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/7089_2007_129.

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Da Costa, Leandro Silva, and Damien Arnoult. "Organelle Separation and Cell Signaling." In Methods in Molecular Biology, 111–15. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6780-3_11.

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Foged, Camilla, Xiaona Jing, and Hanne Moerck Nielsen. "Cell-Penetrating Peptides for Cytosolic Delivery of Biomacromolecules." In Organelle-Specific Pharmaceutical Nanotechnology, 403–32. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9780470875780.ch22.

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Robertson, Alasdair M., and Viki J. Allan. "Cell cycle regulation of organelle transport." In Progress in Cell Cycle Research, 59–75. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5371-7_6.

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Jones, Arwyn T. "Uptake and Intracellular Dynamics of Proteins Internalized by Cell-Penetrating Peptides." In Organelle-Specific Pharmaceutical Nanotechnology, 263–88. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9780470875780.ch15.

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Link, Gerhard. "Plastid Differentiation: Organelle Promoters and Transcription Factors." In Results and Problems in Cell Differentiation, 65–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-540-48037-2_3.

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Kloc, Malgorzata, and Jacek Z. Kubiak. "Exogenous Molecule and Organelle Delivery in Oogenesis." In Results and Problems in Cell Differentiation, 3–16. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60855-6_1.

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Maruyama, Jun-ichi, and Katsuhiko Kitamoto. "The Woronin Body: A Fungal Organelle Regulating Multicellularity." In Biology of the Fungal Cell, 3–14. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-05448-9_1.

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Musnick, David, Larissa Severson, and Sarah Brennan. "Structure: From Organelle and Cell Membrane to Tissue." In Integrative and Functional Medical Nutrition Therapy, 173–90. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-30730-1_12.

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Conference papers on the topic "Cell organelle"

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Nawa, Yasunori, Wataru Inami, Atsushi Ono, Sheng Lin, Yoshimasa Kawata, and Susumu Terakawa. "Label-free cell organelle imaging by D-EXA microscopy." In JSAP-OSA Joint Symposia. Washington, D.C.: OSA, 2014. http://dx.doi.org/10.1364/jsap.2014.19a_c4_8.

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Guirguis, Mark, Katelyn Rimkunas, Michael Raymond, and Leo Q. Wan. "Cell Organelle Positioning of Micropatterned Single C2C12 Mouse Myoblasts." In 2013 39th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2013. http://dx.doi.org/10.1109/nebec.2013.168.

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Wang, Ling. "Organelle topology is a new breast cancer cell classifier." In European Light Microscopy Initiative 2021. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.elmi2021.72.

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Plotnikov, E. Y., V. A. Babenko, D. N. Silachev, I. B. Pevzner, L. D. Zorova, G. T. Sukhikh, and D. B. Zorov. "COULD STEM CELLS ACT AS ORGANELLES DONORS: CELL-TO-CELL MITOCHONDRIA TRANSPORT." In The Second All-Russian Scientific Conference with international participation "Regulation Mechanisms of Eukariotic Cell Organelle Functions". SIPPB SB RAS, 2018. http://dx.doi.org/10.31255/978-5-94797-318-1-92-93.

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Degtyarev, Michael, Mike Reichelt, and Kui Lin. "Abstract 328: Novel quantitative autophagy analysis by organelle flow cytometry after cell sonication." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-328.

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Stepanov, A. V. "THE INVESTIGATION OF PLANT CELL ORGANELLS USING OF FLUORESCENCE MICROSCOPY." In The Second All-Russian Scientific Conference with international participation "Regulation Mechanisms of Eukariotic Cell Organelle Functions". SIPPB SB RAS, 2018. http://dx.doi.org/10.31255/978-5-94797-318-1-125-126.

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Moran, Emma C., Pedro M. Baptista, Kenichiro Nishii, David Wasnick, Shay Soker, and Jessica L. Sparks. "Expression of Primary Cilia on Liver Stem and Progenitor Cells: Potential Role for Mechanosensing in Liver Development." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14122.

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The primary cilium is a non-motile organelle that projects out from the plasma membrane of many cell types in the body. It consists of an axoneme with microtubules arranged in a 9+0 arrangement that extends from the mother centriole contained within the basal body. Once thought to be a non-essential organelle, it is now known that primary cilia have an important role in embryonic and post-natal development, as well as maintenance of adult tissues. Mutations affecting primary ciliary development result in a class of serious diseases known as ciliopathies [1, 2]. Recent research suggests that the primary cilia/ centrosomes might play a role in embryonic stem cell differentiation through cell cycle regulation and their association with the Hedgehog signaling pathway [3, 4].
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Saviz, Mehrdad, and Reza Faraji-Dana. "Realistic cell and organelle shape modeling for computational bioengineering: A new open-source toolbox." In 2014 22nd Iranian Conference on Electrical Engineering (ICEE). IEEE, 2014. http://dx.doi.org/10.1109/iraniancee.2014.6999842.

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Slomka, Noa, and Amit Gefen. "Cell-to-Cell Variability in Tensile Strains Occurring in the Plasma Membrane and Nuclear Surface Area of Compressed Myoblasts." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53234.

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Confocal-microscopy-based three-dimensional (3D) cell-specific finite element (FE) modeling has recently been introduced by our group as a method to simulate the structural behavior of realistic cell geometries under external loading, while considering details of intracellular organelle [1,2]. This method provides comprehensive knowledge regarding cellular mechanics problems, for example, it is useful in the context of understanding the aetiology of deep tissue injury (DTI) — a type of a serious pressure ulcer associated with sustained cellular deformations [3–6]. In this regard, we previously postulated that sustained deformations of soft tissues near bony prominences could cause cell death by a mechanism of locally stretching cells, the consequence of which being that the permeability of the plasma membrane and nuclear surface area (NSA) in the affected cells increases. This, in turn, pathologically changes cell-matrix and intracellular transport profiles and eventually disrupts cellular homeostasis [1,7]. We hypothesize that tensile strains in the plasma membrane and NSA might differ in magnitude and pattern across externally-loaded individual cells of the same cell type, due to cell-to-cell morphological differences. Hence, in this study, we utilize confocal-based cell-specific 3D modeling to analyze tensile strain states in the plasma membrane and NSA of 3 different skeletal muscle cells (myoblasts) subjected to compression. We were specifically interested in chacterizing cell-to-cell variability in magnitudes and patterns of the localized strains.
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Ozolina, N. V. "THE ROLE MEMBRANE CONTACT SITES IN CELL LIFE." In The Second All-Russian Scientific Conference with international participation "Regulation Mechanisms of Eukariotic Cell Organelle Functions". SIPPB SB RAS, 2018. http://dx.doi.org/10.31255/978-5-94797-318-1-77-79.

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