Relevant bibliographies by topics / Cell proliferation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cell proliferation'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Cell proliferation"

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Fraser, Hamish M., Helen Wilson, Audrey Silvestri, Keith D. Morris, and Stanley J. Wiegand. "The Role of Vascular Endothelial Growth Factor and Estradiol in the Regulation of Endometrial Angiogenesis and Cell Proliferation in the Marmoset." Endocrinology 149, no. 9 (2008): 4413–20. http://dx.doi.org/10.1210/en.2008-0325.

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The present studies explore the roles of vascular endothelial growth factor (VEGF) and estradiol on angiogenesis and stromal and epithelial cell proliferation in the marmoset endometrium during the proliferative phase of the ovulatory cycle. At the start of the proliferative phase, marmosets were 1) treated with vehicle, 2) treated with a VEGF inhibitor (VEGF Trap, aflibercept), 3) ovariectomized, 4) ovariectomized and given replacement estradiol, or 5) treated with VEGF Trap and given replacement estradiol. The uterus was examined 10 d later in the late proliferative phase. Changes in endothe
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M. Baghdadi, Houry. "Effect of stem cells on genetic mutations and proliferation in squamous cell carcinoma." International Journal of Academic Research 6, no. 1 (2014): 192–97. http://dx.doi.org/10.7813/2075-4124.2014/6-1/a.25.

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Robey, H. L., P. S. Hiscott, and I. Grierson. "Cytokeratins and retinal epithelial cell behaviour." Journal of Cell Science 102, no. 2 (1992): 329–40. http://dx.doi.org/10.1242/jcs.102.2.329.

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The expression of cytokeratins 18 and 19 by human retinal pigment epithelial cells (HRPE) has been suspected of being associated with HRPE proliferation. We have investigated the involvement of these cytokeratin subtypes in the proliferative and migratory behaviour of cultured HRPE. Cell proliferation markers (bromodeoxyuridine and proliferating cell nuclear antigen) and the cytokeratins were identified using immunohistochemical techniques. In vitro, cytokeratins 18 and 19, as detected by the monoclonal antibodies RGE 53 and K4.62, were expressed in a subset of HRPE and this subset was signifi
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Chan, Ming Liang, Janka Petravic, Alexandra M. Ortiz, et al. "Limited CD4+ T cell proliferation leads to preservation of CD4+ T cell counts in SIV-infected sooty mangabeys." Proceedings of the Royal Society B: Biological Sciences 277, no. 1701 (2010): 3773–81. http://dx.doi.org/10.1098/rspb.2010.0972.

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Human immunodeficiency virus (HIV) and simian immunodeficiency virus (SIV) infections result in chronic virus replication and progressive depletion of CD4+ T cells, leading to immunodeficiency and death. In contrast, ‘natural hosts’ of SIV experience persistent infection with high virus replication but no severe CD4+ T cell depletion, and remain AIDS-free. One important difference between pathogenic and non-pathogenic infections is the level of activation and proliferation of CD4+ T cells. We analysed the relationship between CD4+ T cell number and proliferation in HIV, pathogenic SIV in macaq
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Kabraji, Sheheryar Kairas, Giorgio Gaglia, Danae Argyropoulou, et al. "Temporal and spatial topography of cell proliferation in cancer." Journal of Clinical Oncology 39, no. 15_suppl (2021): 3122. http://dx.doi.org/10.1200/jco.2021.39.15_suppl.3122.

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3122 Background: Tumors are complex ecosystems where exogenous and endogenous cues are integrated to either stimulate or inhibit cancer cell proliferation. However, the nature of these complex cell cycle states, their spatial organization, response to perturbation, and implications for clinical outcomes, are poorly characterized in tumor tissues. Methods: We used multiplexed tissue imaging to develop a robust classifier of proliferation, the multivariate proliferation index (MPI), using 513 unique tumors across five cancer types. Next, we used dimensionality reduction analysis to assess how th
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Tsunoda, Mikiya, Hiroyasu Aoki, Munetomo Takahashi, et al. "Abstract 5180: T cell receptor repertoire analysis revealed tissue tropism of tumor-reactive T-cell clones in cell cycle reporter mice." Cancer Research 83, no. 7_Supplement (2023): 5180. http://dx.doi.org/10.1158/1538-7445.am2023-5180.

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Abstract Tumor-reactive T cells are composed of clones with various TCRs, and each clone has different in vivo kinetics. By analyzing TCR repertoire of tumor and tumor-draining lymph node (dLN), we have demonstrated that Tumor-reactive CD8+ T cells can be classified into “dLN Major”, “Tumor Major”, and “Double Major” clones, which exhibited high frequency in the dLN, tumor, or both tissues. To investigate whether this classification was related to the tissue tropism in the proliferation of each clone, we here employed tumor-bearing Fucci transgenic mice expressing a fluorescent cell-cycle indi
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Levine, Richard, Magnus S. Agren, and Patricia M. Mertz. "Effect of Occlusion on Cell Proliferation during Epidermal Healing." Journal of Cutaneous Medicine and Surgery 2, no. 4 (1998): 193–98. http://dx.doi.org/10.1177/120347549800200403.

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Background: Occlusive dressings influence epithelization of superficial wounds by some unknown mechanism(s). Objective: The effects of occlusion on epidermal cell proliferation in two types of wounds were examined. Methods: Partial-thickness wounds and tape-stripped skin wounds were compared. An immunohistochemical technique, employing PC10 — a monoclonal antibody against proliferating cell nuclear antigen (PCNA) — was applied to formalin-fixed, paraffin-embedded porcine tissue sections. Results: The number of PC10-positive cells was low during the migratory phase, then increased to a peak of
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Deniz, Özdemir. "KAN0438757: A NOVEL PFKFB3 INHIBITOR THAT INDUCES PROGRAMMED CELL DEATH AND SUPPRESSES CELL MIGRATION IN NON-SMALL CELL LUNG CARCINOMA CELLS." Biotechnologia Acta 16, no. 5 (2023): 34–44. http://dx.doi.org/10.15407/biotech16.05.034.

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Aim. PFKFB3 is glycolytic activators that is overexpressed in human lung cancer and plays a crucial role in multiple cellular functions including programmed cell death. Despite the many small molecules described as PFKFB3 inhibitors, some of them have shown disappointing results in vitro and in vivo. On the other hand KAN0438757, selective and potent, small molecule inhibitor has been developed. However, the effects of KAN0438757, in non-small cell lung carcinoma cells remain unknown. Herein, we sought to decipher the effect of KAN0438757 on proliferation, migration, DNA damage, and programmed
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Chung, Hyunju, and Seungjoon Park. "Ghrelin regulates cell cycle-related gene expression in cultured hippocampal neural stem cells." Journal of Endocrinology 230, no. 2 (2016): 239–50. http://dx.doi.org/10.1530/joe-16-0126.

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We have previously demonstrated that ghrelin stimulates the cellular proliferation of cultured adult rat hippocampal neural stem cells (NSCs). However, little is known about the molecular mechanisms by which ghrelin regulates cell cycle progression. The purpose of this study was to investigate the potential effects of ghrelin on cell cycle regulatory molecules in cultured hippocampal NSCs. Ghrelin treatment increased proliferation assessed by CCK-8 proliferation assay. The expression levels of proliferating cell nuclear antigen and cell division control 2, well-known cell-proliferating markers
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Hall, Peter A. "Cell proliferation." Journal of Pathology 165, no. 4 (1991): 349–54. http://dx.doi.org/10.1002/path.1711650412.

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Dissertations / Theses on the topic "Cell proliferation"

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Falk, Anna. "Stem cells : proliferation, differentiation, migration /." Stockholm, 2005. http://diss.kib.ki.se/2006/91-7140-497-X/.

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Cheng, Wai. "The relationship between peroxisome proliferator-activated receptors (PPARs) and cell proliferation /." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36433937.

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Cheng, Wai, and 鄭蔚. "The relationship between peroxisome proliferator-activated receptors (PPARs) and cell proliferation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B45010614.

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Ashagbley, Anthony J. "Ethanolamine requirement and cell proliferation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq23203.pdf.

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Hooper, Nigel I. "Methylglyoxal, glyoxalases and cell proliferation." Thesis, Aston University, 1987. http://publications.aston.ac.uk/12548/.

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The metabolic function of the glyoxalase system was investigated in (a) the differentiation and proliferation of human tumour cells in vitro, (b) the cell-free assembly of microtubules and (c) in the red blood cells during hyperglycaemia associated with Diabetes Mellitus. Chemically-induced differentiation of human promyelocytic HL60 leukaemia cells to neutrophils, and K562 erythroleukaemia cells, was accompanied by a decrease and an increase in the activity of glyoxalase I, respectively. Growth-arrest of Burkitt's lymphoma Raji cells and GM892 lymphoblastoid cells was accompanied by an increa
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Ellison, David William. "Cell proliferation, cell death, and differentiation in gliomas." Thesis, University of Southampton, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295912.

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Petersen, Cecilia. "Paracrine regulation of Sertoli cell proliferation /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-443-7/.

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Zhang, Jiao, and 张姣. "Regulation of cell proliferation and modulation of differentiation in human induced pluripotent stem cell-derived mesenchumal stem cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B49617503.

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Functional mesenchymal stem cells (MSCs) derived from human induced pluripotent stem cells (iPSCs) may represent an unlimited cell source with superior therapeutic benefits for tissue regeneration to somatic tissue, such as bone marrow (BM)-derived MSC. In the first part of this project, I investigated whether the differential expression of ion channels in iPSC-MSCs was responsible for their higher proliferation capacity than that of BM-MSCs. The expression of ion channels for K+, Na+, Ca2+ and Cl- currents was assessed by reverse transcription-polymerase chain reaction (RT-PCR). The function
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Yamak, Fatimah Abir. "GATA4 Partners in Cardiac Cell Proliferation." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23802.

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Cardiovascular diseases are the leading cause of death in humans throughout the world and “congenital heart defects” (CHDs) are the major cause of infant mortality and morbidity. GATA4 is one of the most critical and intensely studied cardiac transcription factor. It is important for proliferation of cardiomyocytes as well as their survival and adaptive response. The focus of the following thesis was to identify GATA4 mediators and cofactors in cardiac growth. The first part focused on cyclin D2 (CycD2), a growth inducible cell cycle protein. We identified Ccnd2 (gene encoding CycD2) as a dire
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Cheung, Man-keung, and 張文強. "FBI-1 and choriocarcinoma cell proliferation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193565.

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Gestational trophoblastic disease (GTD) includes a spectrum of diseases that involve abnormal growth of trophoblastic cells inside the uterus. It can range from benign hydatidiform moles (HM) to frankly malignant choriocarcinoma, placental site trophoblastic tumor (PSTT) or epithelioid trophoblastic tumour (ETT).GTD are considered curable if the patient is correctly diagnosed and receive appropriate treatment during the early stage of the disease. About 15% -30% of hydatidiform moles will develop persistent GTD, but majority of them can usually resolved by surgical intervention and post-ope
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Books on the topic "Cell proliferation"

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1950-, Hughes David, and Mehmet H, eds. Cell proliferation & apoptosis. BIOS Scientific, 2003.

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P, Briggs Andre, and Coburn Jacob A, eds. Handbook of cell proliferation. Nova Science Publishers, 2009.

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Enders, Greg H. Cell cycle deregulation in cancer. Springer, 2010.

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L, Boynton Alton, and Leffert H. L, eds. Control of animal cell proliferation. Academic Press, 1985.

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John, Crocker, ed. Cell Proliferation in Lymphomas. Blackwell Scientific Publications, 1993.

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Heath, John K., ed. Principles of Cell Proliferation. Blackwell Publishing Ltd, 2001. http://dx.doi.org/10.1002/9780470757086.

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Heath, John K. Principles of Cell Proliferation. John Wiley & Sons, Ltd., 2008.

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Sonnenschein, C. The society of cells: Cancer and control of cell proliferation. Bios Scientific Publishers, 1999.

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Dondua, A. K. Kletochnai͡a reprodukt͡sii͡a i prot͡sessy different͡siat͡sii: Sbornik nauchnykh trudov. Izd-vo "Nauka" Leningradskoe otd-nie, 1990.

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Avner, Friedman, and Aguda B, eds. Cell cycle, proliferation, and cancer. Springer, 2006.

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Book chapters on the topic "Cell proliferation"

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Wardle, E. Nigel. "Cell Proliferation." In Guide to Signal Pathways in Immune Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-538-5_5.

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Roy, Priti Kumar. "T Cell Proliferation." In Mathematical Models for Therapeutic Approaches to Control HIV Disease Transmission. Springer Singapore, 2015. http://dx.doi.org/10.1007/978-981-287-852-6_3.

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Lovicu, F. J., L. Iyengar, L. J. Dawes, and J. W. McAvoy. "Lens Epithelial Cell Proliferation." In Lens Epithelium and Posterior Capsular Opacification. Springer Japan, 2014. http://dx.doi.org/10.1007/978-4-431-54300-8_4.

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Kuwayama, Hajime. "Sucralfate and Cell Proliferation." In Sucralfate. Springer US, 1995. http://dx.doi.org/10.1007/978-0-585-32154-7_14.

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Serrano, Manuel. "Proliferation: the Cell Cycle." In Advances in Experimental Medicine and Biology. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0081-0_2.

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Schwarz, Michael, Albrecht Buchmann, Larry W. Robertson, and Werner Kunz. "Cell Proliferation and Hepatocarcinogenesis." In Scientific Issues in Quantitative Cancer Risk Assessment. Birkhäuser Boston, 1990. http://dx.doi.org/10.1007/978-1-4684-9218-7_6.

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Lee, Sun-Hwa, Stacy Lee, and Jae Ung Jung. "Virus-Mediated Cell Proliferation." In Cancer Associated Viruses. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0016-5_3.

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Thiriet, Marc. "Cell Growth and Proliferation." In Control of Cell Fate in the Circulatory and Ventilatory Systems. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0329-6_2.

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Wang, Zhihui, and Thomas S. Deisboeck. "Multilevel Modeling, Cell Proliferation." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_50.

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Watanabe, Nobumoto, and Hiroyuki Osada. "Cell Proliferation and Differentiation." In Bioprobes. Springer Japan, 2017. http://dx.doi.org/10.1007/978-4-431-56529-1_2.

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Conference papers on the topic "Cell proliferation"

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Udalamaththa, Vindya, Praneeth Ratnayake, and Preethi Udagama. "Use of Human Primary Stem Cell Cultures for Screening Cell Proliferation Stimulatory and Inhibitory Effects of Botanical Preparations: an Alternative to Animal Models." In SLIIT INTERNATIONAL CONFERENCE ON ADVANCEMENTS IN SCIENCES AND HUMANITIES. Faculty of Humanities & Sciences, SLIIT, 2024. https://doi.org/10.54389/qeyi9401.

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Animals are widely used in scientific research as highly specific ‘models’ of humans. Primary cell culture is a widely used model to reduce and replace the use of animal models, as per the 3R concept. We investigated cell stimulatory effects of selected botanical preparations on primary human stem cell cultures, in place of animal models. Primary human fibroblast stem cell (hFSC), human mesenchymal stem cell (hMSC), and human haematopoietic stem cell (hHSC) cultures, were established in-house, and characterized by immunophenotyping. Varying concentrations of selected botanical preparations (ma
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Previtera, Michelle L., Mason Hui, Malav Desai, Devendra Verma, Rene Schloss, and Noshir A. Langrana. "Neuronal Precursor Cell Proliferation on Elastic Substrates." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53246.

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Numerous stem cells therapies have been studied for the replacement of damaged neurons due to spinal cord injury. Our laboratory’s goal is to design an implantable platform for spinal cord neuron (SCN) proliferation and differentiation in order to replace damaged neurons in the injured spinal cord. Based on previous literature, we suspect we can promote neuronal precursor cell (NPC) proliferation and differentiation utilizing elastic matrices.
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Xu, F., A. E. Emre, E. S. Turali, et al. "Cell proliferation in bioprinted cell-laden collagen droplets." In 2009 IEEE 35th Annual Northeast Bioengineering Conference. IEEE, 2009. http://dx.doi.org/10.1109/nebc.2009.4967727.

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"Does Progesterone Influence Hela Cell Proliferation?" In 2016 International Conference on Biological and Environmental Science. Universal Researchers, 2016. http://dx.doi.org/10.17758/ur.u0616232.

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Ghaffari, A., S. Ghanizadeh Chenarbon, and P. Rahmani Vahid. "Mathematical and Optimization for a Non-Linear Yeast Cell Proliferation Problem Using Genetic Algorithm." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-68480.

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We consider an age-maturity structured model arising from a yeast cell proliferation problem. This model is a new study in the filed of analysis of cell kinetics and cell division using mathematical modeling and optimized by Genetic Algorithm. We use our mathematical analysis in conjunction with experimental data from the division analysis of primitive cells to characterize the maturation/proliferation process.
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Gu, Ying, Shanxiang Jiang, Elahe Mahdavian, and Shile Huang. "Abstract 4566: Fusarochromanone inhibits cell proliferation and induces cell death in COS7 cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4566.

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Liote, F., M. P. Wautier, E. Savariau, H. Setiadi, and J. L. Wautier. "INHIBITION OF ENDOTHELIAL CELL PROLIFERATION BY NORMAL HUMAN MONOCYTES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643170.

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Human peripheral blood monocytes and macrophages possess factors which are capable of inhibiting or stimulating endothelial cell proliferation. We have further explored if such activity is due to cytotoxic effects of monocytes. Normal mononuclear cells were isolated first by density gradient. Monocytes were then purified by three different techniques: 1) counter centrifugation elutriation (CCE) (Beckman) 2) selective adhesion to gelatin-plasma (GPI) 3) selective adhesion to fibronectin (Fn). Cytotoxicity was estimated by counting the release of 51cr used to label the human umbilical vein endot
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Nobile, Marco S., Thalia Vlachou, Simone Spolaor, et al. "ProCell: Investigating cell proliferation with Swarm Intelligence." In 2019 IEEE Conference on Computational Intelligence in Bioinformatics and Computational Biology (CIBCB). IEEE, 2019. http://dx.doi.org/10.1109/cibcb.2019.8791468.

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Vinjimore Kesavan, S., C. P. Allier, F. Navarro, F. Mittler, B. Chalmond, and J. M. Dinten. "Lensless imaging system to quantify cell proliferation." In SPIE BiOS, edited by Daniel L. Farkas, Dan V. Nicolau, and Robert C. Leif. SPIE, 2013. http://dx.doi.org/10.1117/12.2001826.

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Wang, Hao, Xiaoyi Lv, Guohua Wu, Guodong Lv, and Xiangxiang Zheng. "Cell proliferation detection based on deep learning." In 2020 2nd International Conference on Information Technology and Computer Application (ITCA). IEEE, 2020. http://dx.doi.org/10.1109/itca52113.2020.00051.

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Reports on the topic "Cell proliferation"

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Baker, Nicholas E. Cell Proliferation, Cell Death, and Size Regulation. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/adb248354.

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Band, Hamid. Compartmentalized Signaling and Breast Cancer Cell Proliferation. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada400191.

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Band, Hamid. Compartmentalized Signaling and Breast Cancer Cell Proliferation. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada431079.

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada407274.

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada390950.

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DeVries, George H. Molecular Mechanisms of Schwann Cell Proliferation in NF1. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada392306.

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Eckert, Richard L. TIG3-A Novel Inhibitor of Breast Cancer Cell Proliferation. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada420065.

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Eckert, Richard L. TIG3 - A Novel Inhibitor of Breast Cancer Cell Proliferation. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada391127.

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จันทร์เจ้า, จันทร์เพ็ญ. องค์ประกอบทางเคมีและแอกทิวิตีทางชีวภาพของผลิตภัณฑ์ผึ้งจากผึ้งโพรง (Apis cerana) และชันโรง (Tetragonula laeviceps). จุฬาลงกรณ์มหาวิทยาลัย, 2013. https://doi.org/10.58837/chula.res.2013.42.

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In this research, it was focused on the anti-proliferation of cancer cells and the anti-agglutination of human blood cell infected by H1 N1 virus (AHB) of honey and AHB of propolis from Tetragonula laeviceps. Honey (90 g) was extracted by 96% EtOH and H20 resulting in CHE and CHW, respectively, Later, it was tested against the proliferation of 5 cancer cell lines (BT474, Chago, Hep-G2, KATO-III, and SW620) and CH-liver as normal cell by MTT assay. The data was estimated from the average of percentage of cell viability (PS) and statistically analysed by SPSS statistics 17.0. The result showed t
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Fletterick, Robert J. Inhibition of Pancreatic Cancer Cell Proliferation by LRH-1 Inhibitors. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada599687.

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