Academic literature on the topic 'Cell spreading'

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Journal articles on the topic "Cell spreading"

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LeBrasseur, Nicole. "Spreading mitochondria." Journal of Cell Biology 172, no. 4 (2006): 482. http://dx.doi.org/10.1083/jcb1724rr4.

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Disatnik, Marie-Hélène, and Thomas A. Rando. "Integrin-mediated Muscle Cell Spreading." Journal of Biological Chemistry 274, no. 45 (1999): 32486–92. http://dx.doi.org/10.1074/jbc.274.45.32486.

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Lavine, Marc S. "Cell spreading affects energy consumption." Science 370, no. 6518 (2020): 806.2–806. http://dx.doi.org/10.1126/science.370.6518.806-b.

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Stewart, M. G., E. Moy, G. Chang, W. Zingg, and A. W. Neumann. "Thermodynamic model for cell spreading." Colloids and Surfaces 42, no. 2 (1989): 215–32. http://dx.doi.org/10.1016/0166-6622(89)80193-3.

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Stewart, M. G., E. Moy, G. Chang, W. Zingg, and A. W. Neumann. "Thermodynamic model for cell spreading." Colloids and Surfaces 42, no. 3-4 (1989): 215–32. http://dx.doi.org/10.1016/0166-6622(89)80342-7.

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Tsygankova, Oxana M., Changqing Ma, Waixing Tang, et al. "Downregulation of Rap1GAP in Human Tumor Cells Alters Cell/Matrix and Cell/Cell Adhesion." Molecular and Cellular Biology 30, no. 13 (2010): 3262–74. http://dx.doi.org/10.1128/mcb.01345-09.

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ABSTRACT Rap1GAP expression is decreased in human tumors. The significance of its downregulation is unknown. We show that Rap1GAP expression is decreased in primary colorectal carcinomas. To elucidate the advantages conferred on tumor cells by loss of Rap1GAP, Rap1GAP expression was silenced in human colon carcinoma cells. Suppressing Rap1GAP induced profound alterations in cell adhesion. Rap1GAP-depleted cells exhibited defects in cell/cell adhesion that included an aberrant distribution of adherens junction proteins. Depletion of Rap1GAP enhanced adhesion and spreading on collagen. Silencing
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Sadahira, Yoshito, Tadashi Yoshino, and Naoya Kojima. "B16 melanoma cell spreading on activated endothelial cells." In Vitro Cellular & Developmental Biology - Animal 30, no. 10 (1994): 648–50. http://dx.doi.org/10.1007/bf02631266.

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McInnes, C., P. Knox, and D. J. Winterbourne. "Cell spreading on serum is not identical to spreading on fibronectin." Journal of Cell Science 88, no. 5 (1987): 623–29. http://dx.doi.org/10.1242/jcs.88.5.623.

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Adhesion and spreading of cell lines on dishes coated with serum-derived proteins were studied after removal of cell-surface proteoglycans. A mixture of glycosaminoglycans lyases from heparin-induced Flavobacterium heparinum removed 80% of the [35S]sulphate-labelled glycosaminoglycans from the surface of attached cells within 30 min, but this had little effect on cell morphology. The rate of cell attachment to dishes coated with serum was unaffected by prior treatment of cells with this mixture of glycosaminoglycan lyases. While a heparan sulphate lyase preparation abolished cell spreading in
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Cramer, L. P., and T. J. Mitchison. "Myosin is involved in postmitotic cell spreading." Journal of Cell Biology 131, no. 1 (1995): 179–89. http://dx.doi.org/10.1083/jcb.131.1.179.

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We have investigated a role for myosin in postmitotic Potoroo tridactylis kidney (PtK2) cell spreading by inhibitor studies, time-lapse video microscopy, and immunofluorescence. We have also determined the spatial organization and polarity of actin filaments in postmitotic spreading cells. We show that butanedione monoxime (BDM), a known inhibitor of muscle myosin II, inhibits nonmuscle myosin II and myosin V adenosine triphosphatases. BDM reversibly inhibits PtK2 postmitotic cell spreading. Listeria motility is not affected by this drug. Electron microscopy studies show that some actin filame
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Wells, William A. "Exclusion is spreading." Journal of Cell Biology 168, no. 1 (2004): 11. http://dx.doi.org/10.1083/jcb1681rr3.

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Dissertations / Theses on the topic "Cell spreading"

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Treloar, Katrina K. "Mathematical models for collective cell spreading." Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/86960/1/Katrina_Treloar_Thesis.pdf.

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Collective cell spreading is frequently observed in development, tissue repair and disease progression. Mathematical modelling used in conjunction with experimental investigation can provide key insights into the mechanisms driving the spread of cell populations. In this study, we investigated how experimental and modelling frameworks can be used to identify several key features underlying collective cell spreading. In particular, we were able to independently quantify the roles of cell motility and cell proliferation in a spreading cell population, and investigate how these roles are influenc
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Promwikorn, Waraporn. "Regulation of gene expression and cell cycle progression by cell shape." Thesis, University of Liverpool, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250316.

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Jin, Hua. "The role of Abl tyrosine kinase in cell spreading." Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3274697.

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Thesis (Ph. D.)--University of California, San Diego, 2007.<br>Title from first page of PDF file (viewed October 5, 2007). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
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Streicher, Pia. "Studying integrin-mediated cell spreading using a biomimetic system." Paris 6, 2008. http://www.theses.fr/2008PA066668.

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L'adhésion cellulaire dépendant des intégrines a été étudiée grâce à un système modèle réaliste. Nous avons développé une méthode basée sur celle initialement mise au point pour d'autres protéines dans l'équipe de P. Bassereau. Elle consiste à reconstituer l'intégrine αIIbβ3 dans des protéoliposomes (0. 1 -0. 2 µm de diamètre), puis à électroformer les vésicules géantes à partir des protéoliposomes partiellement séchés. Le succès de la reconstitution a été vérifié en analysant l'incorporation de la protéine et son activité biologique. La dynamique de l'adhésion ces vésicules sur des surfaces c
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Vo, Brenda. "Novel likelihood-free Bayesian parameter estimation methods for stochastic models of collective cell spreading." Thesis, Queensland University of Technology, 2016. https://eprints.qut.edu.au/99588/1/Brenda_Vo_Thesis.pdf.

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Biological processes underlying skin cancer growth and wound healing are governed by various collective cell spreading mechanisms. This thesis develops new statistical methods to provide key insights into the mechanisms driving the spread of cell populations such as motility, proliferation and cell-to-cell adhesion, using experimental data. The new methods allow us to precisely estimate the parameters of such mechanisms, quantify the associated uncertainty and investigate how these mechanisms are influenced by various factors. The thesis provides a useful tool to measure the efficacy of medica
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Redmann, Anna-Lena. "Kinetics of cell attachment and spreading on hard and soft substrates." Thesis, University of Cambridge, 2019. https://www.repository.cam.ac.uk/handle/1810/290385.

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A very important aspect for the functioning of an organism is that cells adapt their behaviour to external stimuli. They continuously interact with their environment, and biochemical and physical cues can activate cellular signalling, which leads to changes in cell behaviour such as proliferation and shape. Understanding cells' interactions with their environment is also important for understanding diseases. For example mechanosensing, which is the sensing of the cell's mechanical environment, has been associated with cancer development. In order for a cell to be able to sense its mechanical e
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Moore, Edward Andrew. "Cell attachment and spreading on physical barriers used in periodontal guided tissue regeneration /." Oklahoma City : [s.n.], 2002. http://library.ouhsc.edu/epub/theses/Moore-William-A.pdf.

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Gill, Amritpal Singh. "Development of a Novel Single-Cell Attachment and Spreading Platform Utilizing Fused-Fiber Nanonets." Thesis, Virginia Tech, 2015. http://hdl.handle.net/10919/73504.

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Initial attachment to the extracellular matrix (ECM) and consequent spreading is a necessary process in the cell cycle of which little is known. Cell spreading has been well-recognized in 2D systems, however, the native fibrous ECM presents cells with 3D biophysical cues. Thus, using suspended fibers as model systems, we present the development of a novel platform (Cell-STEPs) capable of capturing cell attachment dynamics and forces from the moment a cell in suspension contacts the fiber. Cell-STEPs comprises of a custom glass-bottom petri dish with a lid to deliver a constant supply of CO2
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Messmer-Blust, Angela F. "Murine Guanylate-Binding Protein-2: An interferon-induced GTPase that inhibits cell adhesion, cell spreading and MMP-9 expression." University of Toledo / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1263394455.

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Tse, Kathy Wan-Kei. "The role of Pyk2 and FAK in B cell migration, adhesion, and spreading." Thesis, University of British Columbia, 2010. http://hdl.handle.net/2429/25041.

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The ability of the B cell receptor (BCR) to stimulate integrin-mediated adhesion, and induce cytoskeletal reorganization and cell spreading enhances the ability of B cells to bind and respond to antigens (Ag). The proper localization and trafficking of B cells in the secondary lymphoid organs are also critical for B cells to encounter Ags and to be activated. Proline-rich tyrosine kinase (Pyk2) and focal adhesion kinase (FAK) are related cytoplasmic tyrosine kinases that have been shown to regulate cell adhesion, morphology, and migration. However, their functions in B cells are not clear.
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Books on the topic "Cell spreading"

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L, Gauthier Mona. Calmodulin-binding proteins in dictyostelium chemotaxis and breast cancer cell spreading. 2002.

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Al-Anazi, KA, WK Al-Anazi, and AM Al-Jasser. Update on COVID-19 Infections and the Promising Role of Mesenchymal Stem Cell Therapies in their Management. Heighten Science Publications Inc., 2020. http://dx.doi.org/10.29328/ebook1002.

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The pandemic of COVID-19 has adversely affected almost every aspect of our lives but the world health and economic sectors suffer most of the repercussions of this disease. The search for a cure for this rapidly spreading virus which is causing massive life losses around the globe requires clear understanding of the immunopathogenesis of this virus as well as the mechanisms of actions of the various therapeutic modalities that are employed in the treatment of this life-threatening viral infection. Mesenchymal stem cells have antimicrobials effects in addition to their anti-inflammatory and imm
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Book chapters on the topic "Cell spreading"

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Margolis, Leonid B. "Cell Spreading and Intracellular pH in Mammalian Cells." In Mechanics of Swelling. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-84619-9_25.

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Borodich, Feodor M., and Stanislav N. Gorb. "Spreading of Red Caviar Cells: The Knife-Cell and the Cell-Cell Adhesive Interactions." In Biologically-Inspired Systems. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-85175-0_7.

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Sheetz, Michael P., Benjamin J. Dubin-Thaler, Gregory Giannone, Guoying Jiang, and Hans-Günther Döbereiner. "Functional Phases in Cell Attachment and Spreading." In Cell Migration in Development and Disease. Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527604669.ch1.

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Biber, Stephanie, and Lisa Wiesmüller. "Analysis of Replication Dynamics Using the Single-Molecule DNA Fiber Spreading Assay." In Cell Cycle Checkpoints. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1217-0_4.

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MacLeod, Matthew, Rachid Chreyh, and Gabriel Wainer. "Improved Cell-DEVS Models for Fire Spreading Analysis." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11861201_55.

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Shang, Hui, and Gabriel Wainer. "A Model of Virus Spreading Using Cell-DEVS." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/11428848_50.

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Kempski, O., H. Otsuka, T. Seiwert, and A. Heimann. "Spreading Depression Induces Permanent Cell Swelling Under Penumbra Conditions." In Brain Edema XI. Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6346-7_51.

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Timar, J., B. Liu, R. Bazaz, J. D. Taylor, and K. V. Honn. "Fatty Acid Modulation of Cancer Cell Spreading and Cytoskeleton Rearrangement." In Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation and Radiation Injury. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3520-1_125.

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Carey, Shawn P., Jonathan M. Charest, and Cynthia A. Reinhart-King. "Forces During Cell Adhesion and Spreading: Implications for Cellular Homeostasis." In Cellular and Biomolecular Mechanics and Mechanobiology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/8415_2010_22.

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Humphries, Martin J., Zohreh Mostafavi-Pour, Mark R. Morgan, Nicholas O. Deakin, Anthea J. Messent, and Mark D. Bass. "Integrin-Syndecan Cooperation Governs the Assembly of Signalling Complexes during Cell Spreading." In Signalling Networks in Cell Shape and Motility. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047001766x.ch14.

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Conference papers on the topic "Cell spreading"

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Weafer, Paul, Maarten H. van Es, Suzanne P. Jarvis, and Patrick J. McGarry. "Compression Force Measurement During Cell Spreading Using a Modified Atomic Force Microscope." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53741.

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Mechanical forces play a critical role in the regulation of cell spreading and cytoskeletal organization. It has been demonstrated that the force required to compress a spread cell is significantly higher than that required to compress a cell with a rounded morphology [1]. This increase in force can not be attributed to morphological changes alone [2], highlighting the importance of cytoskeletal remodeling during cell spreading in the resistance of cells to compressive forces.
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Yang, Ling, Qiang Zhou, and Xianlong Hong. "Congestion-Driven Placement Improvement Using Cell Spreading." In 2006 International Conference on Communications, Circuits and Systems. IEEE, 2006. http://dx.doi.org/10.1109/icccas.2006.285164.

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Barmawi, A. M. "Strengthening Dynamic Cell Spreading (DCS) Image Steganography." In 2013 International Conference on Information and Network Security (ICINS 2013). Institution of Engineering and Technology, 2013. http://dx.doi.org/10.1049/cp.2013.2467.

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Shi, Jian, Benlian Xu, Mingli Lu, Peiyi Zhu, and Jihong Zhu. "Multiple Cell Tracking by Ripple Spreading Optimization." In 2018 International Conference on Control, Automation and Information Sciences (ICCAIS). IEEE, 2018. http://dx.doi.org/10.1109/iccais.2018.8570642.

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Hu, Jia, and Yaling Liu. "Cell Adhesion on a Wavy Surface." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14059.

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The ability to control the position of cells in an organized pattern on a substrate has become increasingly important for biosensing and tissue engineering applications [1–3]. With the advent of nanofabrication techniques, a number of researchers have studied the effects of nano-scale grooves on cell spreading, migration, morphology, signaling and orientation [4–6]. Recent studies have shown that cell adhesion/spreading can be influenced by a nanostructured surface [7]. In most current studies, the pattern dimensions are much smaller than the size of a cell. In this paper, we focus on studying
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Vernerey, Franck J. "Biophysical Model of the Coupled Mechanisms of Cell Adhesion, Contraction and Spreading." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80309.

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Recent research has shown that cell spreading is highly dependent on the contractility of its cytoskeleton and the mechanical properties of its surrounding environment. This extended abstract introduces a mathematical formulation of cell spreading and contraction that couples the processes of stress fiber formation, protrusion growth through actin polymerization at the cell edge and dynamics of cross-membrane protein (integrins) enabling cell-substrate attachment. The evolving cell’s cytoskeleton is modeled as a mixture of fluid, proteins and filaments that can exchange mass and generate contr
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Li, Jianrong, Tianle Cheng, and Martin Y. M. Chiang. "Finite Element Modelling of Cell Adhesion Mediated by Receptor-Ligand Binding." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206297.

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The process of cell adhesion and spreading on the extracellular matrix (ECM) protein layer is mediated by the interaction of cell receptors and ECM ligands [1]. Receptors diffuse along the cell membrane surface and interact with ligands in ECM to form bonds. Cells spread and the adhesion zone grows as bond formation at the adhesion front increases to a critical level. This process involves coupling of reaction-diffusion and mechanical contact between cells and ECM. In this study, a novel numerical algorithm is developed to implement this coupling into the finite element method for modeling the
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Wang, Xueyan, Yici Cai, and Qiang Zhou. "Cell spreading optimization for force-directed global placers." In 2017 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2017. http://dx.doi.org/10.1109/iscas.2017.8050572.

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Wang, Sifei, Qiang Zhou, Xu Qian, Yici Cai, and Wenchao Gao. "GPCS: Global placement based on cell spreading method." In 2013 International Conference on Communications, Circuits and Systems (ICCCAS). IEEE, 2013. http://dx.doi.org/10.1109/icccas.2013.6765261.

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Robu, A., A. Neagu, and L. Stoicu-Tivadar. "The influence of cell-substrate and cell-medium interfacial tension on the cell spreading." In 2011 15th IEEE International Conference on Intelligent Engineering Systems (INES). IEEE, 2011. http://dx.doi.org/10.1109/ines.2011.5954714.

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Reports on the topic "Cell spreading"

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Ivanova, Svetlana I., Stoyan A. Chakarov, and Roumen G. Pankov. Formation of Fibrillar Adhesions Correlates with Spreading but Does not Depend on Cell Polarization. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2018. http://dx.doi.org/10.7546/crabs.2018.11.08.

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Research, Gratis. The Mystery behind Bacterial Retrons. Gratis Research, 2020. http://dx.doi.org/10.47496/gr.blog.05.

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Retron-mediated cell killing serves as a defensive strategy to prevent the spreading of phage infection in bacteria and the combined action of retron and CRISPR-based gene editing appear to be a potent gene-editing tool.
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