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Journal articles on the topic 'Cell transdifferentiation'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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1

OKADA, T. S. "Transdifferentiation in Animal Cells: Fact or Artifact?. (cell commitment/transdifferentiation/cell type conversion)." Development, Growth and Differentiation 28, no. 3 (1986): 213–21. http://dx.doi.org/10.1111/j.1440-169x.1986.00213.x.

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2

Zhao, Zhiliang, Mengyao Xu, Meng Wu, Xiaocheng Tian, Cuiping Zhang, and Xiaobing Fu. "Transdifferentiation of Fibroblasts by Defined Factors." Cellular Reprogramming 17, no. 3 (2015): 151–59. http://dx.doi.org/10.1089/cell.2014.0089.

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3

Maclean, Norman. "Transdifferentiation: Flexibility in cell differentiation." Trends in Biochemical Sciences 17, no. 8 (1992): 322. http://dx.doi.org/10.1016/0968-0004(92)90447-h.

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4

Mitashov, V. I. "Genetic Mechanisms of Cell Transdifferentiation." Russian Journal of Developmental Biology 36, no. 4 (2005): 240–46. http://dx.doi.org/10.1007/s11174-005-0039-1.

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5

Lee, Tsong-Hai, Pei-Shan Liu, Su-Jane Wang, Ming-Ming Tsai, Velayuthaprabhu Shanmugam, and Hsi-Lung Hsieh. "Bradykinin, as a Reprogramming Factor, Induces Transdifferentiation of Brain Astrocytes into Neuron-like Cells." Biomedicines 9, no. 8 (2021): 923. http://dx.doi.org/10.3390/biomedicines9080923.

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Kinins are endogenous, biologically active peptides released into the plasma and tissues via the kallikrein-kinin system in several pathophysiological events. Among kinins, bradykinin (BK) is widely distributed in the periphery and brain. Several studies on the neuro-modulatory actions of BK by the B2BK receptor (B2BKR) indicate that this neuropeptide also functions during neural fate determination. Previously, BK has been shown to induce differentiation of nerve-related stem cells into neuron cells, but the response in mature brain astrocytes is unknown. Herein, we used rat brain astrocyte (R

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6

English, Denis. "Transdifferentiation Wars." Stem Cells and Development 14, no. 6 (2005): 605–7. http://dx.doi.org/10.1089/scd.2005.14.605.

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7

Eisenberg, Leonard M., and Carol A. Eisenberg. "Stem cell plasticity, cell fusion, and transdifferentiation." Birth Defects Research Part C: Embryo Today: Reviews 69, no. 3 (2003): 209–18. http://dx.doi.org/10.1002/bdrc.10017.

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8

Luo, Liang, Da-Hai Hu, James Q. Yin, and Ru-Xiang Xu. "Molecular Mechanisms of Transdifferentiation of Adipose-Derived Stem Cells into Neural Cells: Current Status and Perspectives." Stem Cells International 2018 (September 13, 2018): 1–14. http://dx.doi.org/10.1155/2018/5630802.

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Neurological diseases can severely compromise both physical and psychological health. Recently, adult mesenchymal stem cell- (MSC-) based cell transplantation has become a potential therapeutic strategy. However, most studies related to the transdifferentiation of MSCs into neural cells have had disappointing outcomes. Better understanding of the mechanisms underlying MSC transdifferentiation is necessary to make adult stem cells more applicable to treating neurological diseases. Several studies have focused on adipose-derived stromal/stem cell (ADSC) transdifferentiation. The purpose of this

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9

Huang, Shian, Xiulong Zhu, Wenjun Huang, et al. "Quercetin Inhibits Pulmonary Arterial Endothelial Cell Transdifferentiation Possibly by Akt and Erk1/2 Pathways." BioMed Research International 2017 (2017): 1–8. http://dx.doi.org/10.1155/2017/6147294.

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This study aimed to investigate the effects and mechanisms of quercetin on pulmonary arterial endothelial cell (PAEC) transdifferentiation into smooth muscle-like cells. TGF-β1-induced PAEC transdifferentiation models were applied to evaluate the pharmacological actions of quercetin. PAEC proliferation was detected with CCK8 method and BurdU immunocytochemistry. Meanwhile, the identification and transdifferentiation of PAECs were determined by FVIII immunofluorescence staining andα-SMA protein expression. The related mechanism was elucidated based on the levels of Akt and Erk1/2 signal pathway

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10

Corbett, James L., and David Tosh. "Conversion of one cell type into another: implications for understanding organ development, pathogenesis of cancer and generating cells for therapy." Biochemical Society Transactions 42, no. 3 (2014): 609–16. http://dx.doi.org/10.1042/bst20140058.

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Metaplasia is the irreversible conversion of one differentiated cell or tissue type into another. Metaplasia usually occurs in tissues that undergo regeneration, and may, in a pathological context, predispose to an increased risk of disease. Studying the conditions leading to the development of metaplasia is therefore of significant clinical interest. In contrast, transdifferentiation (or cellular reprogramming) is a subset of metaplasia that describes the permanent conversion of one differentiated cell type into another, and generally occurs between cells that arise from neighbouring regions

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11

Rogers, Ian. "Transdifferentiation of endogenous cells: Cell therapy without the cells." Cell Cycle 8, no. 24 (2009): 4023–28. http://dx.doi.org/10.4161/cc.8.24.10512.

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12

Moimas, Silvia, Francesco Salton, Beata Kosmider, et al. "miR-200 family members reduce senescence and restore idiopathic pulmonary fibrosis type II alveolar epithelial cell transdifferentiation." ERJ Open Research 5, no. 4 (2019): 00138–2019. http://dx.doi.org/10.1183/23120541.00138-2019.

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RationaleAlveolar type II (ATII) cells act as adult stem cells contributing to alveolar type I (ATI) cell renewal and play a major role in idiopathic pulmonary fibrosis (IPF), as supported by familial cases harbouring mutations in genes specifically expressed by these cells. During IPF, ATII cells lose their regenerative potential and aberrantly express pathways contributing to epithelial–mesenchymal transition (EMT). The microRNA miR-200 family is downregulated in IPF, but its effect on human IPF ATII cells remains unproven. We wanted to 1) evaluate the characteristics and transdifferentiatin

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13

Wells, William A. "Is transdifferentiation in trouble?" Journal of Cell Biology 157, no. 1 (2002): 15–18. http://dx.doi.org/10.1083/jcb.200203037.

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Spectacular examples of transdifferentiation—such as brain cells turning to blood and blood to brain—have given way to sneaking suspicions about artifacts in culture, fusion, and clonality. Could cell fates be relatively fixed after all?

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14

Slack, Jonathan M. W., and David Tosh. "Transdifferentiation and metaplasia — switching cell types." Current Opinion in Genetics & Development 11, no. 5 (2001): 581–86. http://dx.doi.org/10.1016/s0959-437x(00)00236-7.

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15

Lipton, Bruce H., Klaus G. Bensch, and Marvin A. Karasek. "Microvessel endothelial cell transdifferentiation: phenotypic characterization." Differentiation 46, no. 2 (1991): 117–33. http://dx.doi.org/10.1111/j.1432-0436.1991.tb00872.x.

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16

Purnell, B. A. "Epigenetics Direct Transdifferentiation." Science Signaling 7, no. 339 (2014): ec222-ec222. http://dx.doi.org/10.1126/scisignal.2005804.

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17

Cordeiro-Rudnisky, Fernanda, Yue Sun, and Rayan Saade. "Prostate Carcinoma With Overlapping Features of Small Cell and Acinar Adenocarcinoma: A Case Report." American Journal of Clinical Pathology 152, Supplement_1 (2019): S66—S67. http://dx.doi.org/10.1093/ajcp/aqz113.072.

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Abstract Introduction Prostate neuroendocrine (NE) cells can stimulate prostate adenocarcinoma (PA) cell growth, but occasionally adenocarcinoma cells themselves acquire NE characteristics, a phenomenon known as NE transdifferentiation of prostate adenocarcinoma. During this process, tumor cells acquire small cell-like morphology and become positive for neuroendocrine markers. NE transdifferentiation is associated with decreased androgen receptor (AR) signaling, a mechanism of resistance to AR-targeted treatments. Case A 74-year-old male with a history of cirrhosis, splenomegaly, and thrombocy

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18

Xie, Chao, William Donelan, Shun Lu, and Li Jun Yang. "Developing a Sensitive Reporter System for Monitoring of Pancreatic and Duodenal Homeobox Gene 1 (Pdx1) and Neurogenin 3 (Ngn3) – Mediated Transdifferentiation from Human Hepatic Cells into Insulin-Producing Beta-Like Cells." Advanced Materials Research 989-994 (July 2014): 1003–6. http://dx.doi.org/10.4028/www.scientific.net/amr.989-994.1003.

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It is well known that cellular differentiation is not a terminal process. Transdifferentiation is the conversion of one differentiated cell type to another. There are many examples of induced transdifferentiation between cell types by expression of ectopic transcription factors. Here we show that combined lentiviral expression of Pdx1 or Pdx1-VP16 fusion protein and Ngn3 can direct the transdifferentiation of hepatic cells into insulin producing cells. We showed that the Pdx1 or Pdx1-VP16 fusion protein and Ngn3 together synergistically increased transactivation for the insulin gene. This prov

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19

Chua, Shawn J., Robert F. Casper, and Ian M. Rogers. "Toward Transgene-Free Induced Pluripotent Stem Cells: Lessons from Transdifferentiation Studies." Cellular Reprogramming 13, no. 4 (2011): 273–80. http://dx.doi.org/10.1089/cell.2010.0108.

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20

Kaur, Keerat, Jinpu Yang, Carol A. Eisenberg, and Leonard M. Eisenberg. "5-Azacytidine Promotes the Transdifferentiation of Cardiac Cells to Skeletal Myocytes." Cellular Reprogramming 16, no. 5 (2014): 324–30. http://dx.doi.org/10.1089/cell.2014.0021.

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21

Yuan, Zhao-Di, Wei-Ning Zhu, Ke-Zhi Liu, Zhan-Peng Huang, and Yan-Chuang Han. "Small Molecule Epigenetic Modulators in Pure Chemical Cell Fate Conversion." Stem Cells International 2020 (October 20, 2020): 1–12. http://dx.doi.org/10.1155/2020/8890917.

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Although innovative technologies for somatic cell reprogramming and transdifferentiation provide new strategies for the research of translational medicine, including disease modeling, drug screening, artificial organ development, and cell therapy, recipient safety remains a concern due to the use of exogenous transcription factors during induction. To resolve this problem, new induction approaches containing clinically applicable small molecules have been explored. Small molecule epigenetic modulators such as DNA methylation writer inhibitors, histone methylation writer inhibitors, histone acy

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22

Perán, Macarena, Juan Antonio Marchal, Fernando Rodríguez‑Serrano, Pablo Álvarez, and Antonia Aránega. "Transdifferentiation: why and how?" Cell Biology International 35, no. 4 (2011): 373–79. http://dx.doi.org/10.1042/cbi20100445.

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23

Miettinen, P. J., R. Ebner, A. R. Lopez, and R. Derynck. "TGF-beta induced transdifferentiation of mammary epithelial cells to mesenchymal cells: involvement of type I receptors." Journal of Cell Biology 127, no. 6 (1994): 2021–36. http://dx.doi.org/10.1083/jcb.127.6.2021.

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The secreted polypeptide transforming growth factor-beta (TGF-beta) exerts its multiple activities through type I and II cell surface receptors. In epithelial cells, activation of the TGF-beta signal transduction pathways leads to inhibition of cell proliferation and an increase in extracellular matrix production. TGF-beta is widely expressed during development and its biological activity has been implicated in epithelial-mesenchymal interactions, e.g., in branching morphogenesis of the lung, kidney, and mammary gland, and in inductive events between mammary epithelium and stroma. In the prese

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24

Ha, Seon-Ah, Hyun K. Kim, JinAh Yoo, et al. "Transdifferentiation-inducing HCCR-1 oncogene." BMC Cell Biology 11, no. 1 (2010): 49. http://dx.doi.org/10.1186/1471-2121-11-49.

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25

Di Tullio, Alessandro, та Thomas Graf. "C/EBPαbypasses cell cycle-dependency during immune cell transdifferentiation". Cell Cycle 11, № 14 (2012): 2739–46. http://dx.doi.org/10.4161/cc.21119.

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26

Grove, Lisa M., Maradumane L. Mohan, Susamma Abraham та ін. "Translocation of TRPV4-PI3Kγ complexes to the plasma membrane drives myofibroblast transdifferentiation". Science Signaling 12, № 607 (2019): eaau1533. http://dx.doi.org/10.1126/scisignal.aau1533.

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Myofibroblasts are key contributors to pathological fibrotic conditions of several major organs. The transdifferentiation of fibroblasts into myofibroblasts requires both a mechanical signal and transforming growth factor–β (TGF-β) signaling. The cation channel transient receptor potential vanilloid 4 (TRPV4) is a critical mediator of myofibroblast transdifferentiation and in vivo fibrosis through its mechanosensitivity to extracellular matrix stiffness. Here, we showed that TRPV4 promoted the transdifferentiation of human and mouse lung fibroblasts through its interaction with phosphoinositid

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27

Krishnamurthy, Akilan, A. Jimmy Ytterberg, Meng Sun, et al. "Citrullination Controls Dendritic Cell Transdifferentiation into Osteoclasts." Journal of Immunology 202, no. 11 (2019): 3143–50. http://dx.doi.org/10.4049/jimmunol.1800534.

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28

Beresford, William A. "Transdifferentiation: Flexibility in Cell Differentiation.T. S. Okada." Quarterly Review of Biology 68, no. 1 (1993): 110–11. http://dx.doi.org/10.1086/417954.

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29

Xie, Xin, Chenwen Huang, Yanbin Fu, Long Yuan, and Quan Wang. "Chemical-Mediated Somatic Cell Reprogramming and Transdifferentiation." Proceedings for Annual Meeting of The Japanese Pharmacological Society WCP2018 (2018): OR19–1. http://dx.doi.org/10.1254/jpssuppl.wcp2018.0_or19-1.

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30

Cantaluppi, Vincenzo, Stefania Bruno та Giovanni Camussi. "Pancreatic ductal transdifferentiation for β-cell neogenesis". Expert Opinion on Therapeutic Patents 18, № 8 (2008): 963–67. http://dx.doi.org/10.1517/13543776.18.8.963.

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31

Yi, Fei, Guang-Hui Liu, and Juan Carlos Izpisua Belmonte. "Rejuvenating liver and pancreas through cell transdifferentiation." Cell Research 22, no. 4 (2012): 616–19. http://dx.doi.org/10.1038/cr.2012.33.

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32

Sisakhtnezhad, Sajjad, and Maryam M. Matin. "Transdifferentiation: a cell and molecular reprogramming process." Cell and Tissue Research 348, no. 3 (2012): 379–96. http://dx.doi.org/10.1007/s00441-012-1403-y.

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33

Yan, Jiawei, Ruoning Wang, and Tiffany Horng. "mTOR Is Key to T Cell Transdifferentiation." Cell Metabolism 29, no. 2 (2019): 241–42. http://dx.doi.org/10.1016/j.cmet.2019.01.008.

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34

Kalra, Rajkumar Singh, Jaspreet Kaur Dhanjal, Mriganko Das, Birbal Singh, and Rajesh Naithani. "Cell Transdifferentiation and Reprogramming in Disease Modeling: Insights into the Neuronal and Cardiac Disease Models and Current Translational Strategies." Cells 10, no. 10 (2021): 2558. http://dx.doi.org/10.3390/cells10102558.

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Cell transdifferentiation and reprogramming approaches in recent times have enabled the manipulation of cell fate by enrolling exogenous/artificial controls. The chemical/small molecule and regulatory components of transcription machinery serve as potential tools to execute cell transdifferentiation and have thereby uncovered new avenues for disease modeling and drug discovery. At the advanced stage, one can believe these methods can pave the way to develop efficient and sensitive gene therapy and regenerative medicine approaches. As we are beginning to learn about the utility of cell transdif

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35

O'Neill, Kathy E., Shifaan Thowfeequ, Wan-Chun Li та ін. "Hepatocyte-Ductal Transdifferentiation Is Mediated by Reciprocal Repression of SOX9 and C/EBPα". Cellular Reprogramming 16, № 5 (2014): 314–23. http://dx.doi.org/10.1089/cell.2014.0032.

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36

Feng, Jian. "Kinetic barriers in transdifferentiation." Cell Cycle 15, no. 8 (2016): 1019–20. http://dx.doi.org/10.1080/15384101.2016.1151730.

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37

Keilhoff, Gerburg, Alexander Goihl, Kristina Langnäse, Hisham Fansa, and Gerald Wolf. "Transdifferentiation of mesenchymal stem cells into Schwann cell-like myelinating cells." European Journal of Cell Biology 85, no. 1 (2006): 11–24. http://dx.doi.org/10.1016/j.ejcb.2005.09.021.

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38

Li, Wan-Chun, Wei-Yuan Yu, Jonathan M. Quinlan, Zoë D. Burke, and David Tosh. "The molecular basis of transdifferentiation." Journal of Cellular and Molecular Medicine 9, no. 3 (2005): 569–82. http://dx.doi.org/10.1111/j.1582-4934.2005.tb00489.x.

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39

Fu, Lina, Xiping Zhu, Fei Yi, Guang-Hui Liu, and Juan Carlos Izpisua Belmonte. "Regenerative medicine: Transdifferentiation in vivo." Cell Research 24, no. 2 (2013): 141–42. http://dx.doi.org/10.1038/cr.2013.165.

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40

Zhang, Jingxue, Shen Wu, Zi-Bing Jin, and Ningli Wang. "Stem Cell-Based Regeneration and Restoration for Retinal Ganglion Cell: Recent Advancements and Current Challenges." Biomolecules 11, no. 7 (2021): 987. http://dx.doi.org/10.3390/biom11070987.

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Glaucoma is a group of irreversible blinding eye diseases characterized by the progressive loss of retinal ganglion cells (RGCs) and their axons. Currently, there is no effective method to fundamentally resolve the issue of RGC degeneration. Recent advances have revealed that visual function recovery could be achieved with stem cell-based therapy by replacing damaged RGCs with cell transplantation, providing nutritional factors for damaged RGCs, and supplying healthy mitochondria and other cellular components to exert neuroprotective effects and mediate transdifferentiation of autologous retin

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41

Zhao, Lan, Min Yee та Michael A. O'Reilly. "Transdifferentiation of alveolar epithelial type II to type I cells is controlled by opposing TGF-β and BMP signaling". American Journal of Physiology-Lung Cellular and Molecular Physiology 305, № 6 (2013): L409—L418. http://dx.doi.org/10.1152/ajplung.00032.2013.

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Alveolar epithelial type II (ATII) cells are essential for maintaining normal lung homeostasis because they produce surfactant, express innate immune proteins, and can function as progenitors for alveolar epithelial type I (ATI) cells. Although autocrine production of transforming growth factor (TGF)-β1 has been shown to promote the transdifferentiation of primary rat ATII to ATI cells in vitro, mechanisms controlling this process still remain poorly defined. Here, evidence is provided that Tgf-β1, - 2, - 3 mRNA and phosphorylated SMAD2 and SMAD3 significantly increase as primary cultures of m

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42

Huang, Gui-Lin, Ni-Ni Zhang, Jun-Sheng Wang, Li Yao, Yu-Jie Zhao, and Yu-Ying Wang. "Transdifferentiation of Human Amniotic Epithelial Cells into Acinar Cells Using a Double-Chamber System." Cellular Reprogramming 14, no. 4 (2012): 377–83. http://dx.doi.org/10.1089/cell.2011.0096.

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43

Zhang, Nian, Liru Hu, Jiyuan Liu, Wenbin Yang, Ye Li, and Jian Pan. "Wnt Signaling Regulates the Lymphatic Endothelial Transdifferentiation of Adipose-Derived Stromal Cells In Vitro." Cellular Reprogramming 23, no. 2 (2021): 117–26. http://dx.doi.org/10.1089/cell.2020.0058.

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44

Kim, Seung Hyun L., Seunghun S. Lee, Inseon Kim, et al. "Ectopic transient overexpression of OCT-4 facilitates BMP4-induced osteogenic transdifferentiation of human umbilical vein endothelial cells." Journal of Tissue Engineering 11 (January 2020): 204173142090920. http://dx.doi.org/10.1177/2041731420909208.

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Limitation in cell sources for autologous cell therapy has been a recent focus in stem cell therapy and tissue engineering. Among various research advances, direct conversion, or transdifferentiation, is a notable and feasible strategy for the generation and acquirement of wanted cell source. So far, utilizing cell transdifferentiation technology in tissue engineering was mainly restricted at achieving single wanted cell type from diverse cell types with high efficiency. However, regeneration of a complete tissue always requires multiple cell types which poses an intrinsic complexity. In this

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45

Wild, Sebastian L., and David Tosh. "Molecular mechanisms of transcription factor mediated cell reprogramming: conversion of liver to pancreas." Biochemical Society Transactions 49, no. 2 (2021): 579–90. http://dx.doi.org/10.1042/bst20200219.

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Transdifferentiation is a type of cellular reprogramming involving the conversion of one differentiated cell type to another. This remarkable phenomenon holds enormous promise for the field of regenerative medicine. Over the last 20 years techniques used to reprogram cells to alternative identities have advanced dramatically. Cellular identity is determined by the transcriptional profile which comprises the subset of mRNAs, and therefore proteins, being expressed by a cell at a given point in time. A better understanding of the levers governing transcription factor activity benefits our abilit

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46

Minami, Kohtaro. "Pancreatic acinar-to-beta cell transdifferentiation in vitro." Frontiers in Bioscience Volume, no. 13 (2008): 5824. http://dx.doi.org/10.2741/3119.

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47

Sharma, Anup D., Jayme Wiederin, Metin Uz, et al. "Proteomic analysis of mesenchymal to Schwann cell transdifferentiation." Journal of Proteomics 165 (August 2017): 93–101. http://dx.doi.org/10.1016/j.jprot.2017.06.011.

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48

SHE, H., S. HAZRA, S. XIONG, J. WANG, C. SUNG, and H. TSUKAMOTO. "211 Adipogenic regulation of hepatic stellate cell transdifferentiation." Hepatology 38 (2003): 258. http://dx.doi.org/10.1016/s0270-9139(03)80254-9.

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49

Ibrahim, Michael, and Pavan Atluri. "Transdifferentiation: A new frontier in cardiovascular cell therapy." Journal of Thoracic and Cardiovascular Surgery 153, no. 1 (2017): 130–31. http://dx.doi.org/10.1016/j.jtcvs.2016.09.007.

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50

Mann, D. A., and J. Mann. "479 EPIGENETIC REGULATION OF HEPATIC STELLATE CELL TRANSDIFFERENTIATION." Journal of Hepatology 48 (January 2008): S182. http://dx.doi.org/10.1016/s0168-8278(08)60481-x.

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