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1

Bassingthwaighte, James B. Whole Organ Approaches to Cellular Metabolism: Permeation, Cellular Uptake, and Product Formation. Springer New York, 1998.

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2

Farlinger, Christopher M. The influence of skeletal muscle cell volume on the regulation of carbohydrate uptake and muscle metabolism. Brock University, Faculty of Applied Health Sciences, 2007.

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3

Tarmohamed, Yasmin. The role of the cell wall in nickel and cadmium toxicity and uptake in two strains of chlamydomonas Reinhardtii. National Library of Canada, 1991.

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4

Kennedy, Ian M., and Abdul I. Barakat. Uptake and inflammatory effects of nanoparticles in a human vascular endothelial cell line: With a critique by the HEI Health Review Committee. Health Effects Institute, 2009.

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5

Sarabia, Vivian E. Calcium homeostasis and regulation of glucose uptake in human skeletal muscle cells in culture. National Library of Canada, 1990.

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6

Diver, Jonathan Michael. The uptake of quinolone antimicrobial agents into Escherichia coli: Transport mechanisms and physiological effects on cells. University of Birmingham, 1987.

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7

Tréhin, Rachel. Cell penetrating peptides: Uptake, permeation and metabolism in epithelial models. 2003.

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8

Arnold, Michael. Mobile Phone Uptake: A Review of the Literature and a Framework for Research. Heidelberg Press, 2003.

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9

Arnold, Michael. Mobile Phone Uptake: A Review of the Literature and a Framework for Research. Heidelberg Press, 2003.

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10

(Editor), James Bassingthwaighte, Carl A. Goresky (Editor), and John H. Linehan (Editor), eds. Whole Organ Approaches to Cellular Metabolism: Permeation, Cellular Uptake, and Product Formation. Springer, 1998.

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11

Chaudhuri, Srimoyee Ray. Heterogeneous uptake of atmospheric organic gas phase species by condensed organic film substrates: A low-pressure effusive cell study. 2006.

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12

Banjara, Manoj, and Damir Janigro. Effects of the Ketogenic Diet on the Blood-Brain Barrier. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0030.

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Ketone bodies (KBs) are always present in the blood, and their levels increase after high-fat diet intake, prolonged exercise, or extended fasting. Thus, one can predict effects on the brain capillary endothelium from high levels of ketones in the blood. Prolonged exposure of blood-brain barrier (BBB) endothelial cells to KBs induces expression of monocarboxylate transporters and enhances brain uptake of KBs. In addition, cell migration and expression of gap junction proteins are up-regulated by KBs. Thus, beneficial effects of the ketogenic diet may depend on increased brain uptake of KBs to
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13

Zelinka, Alena Ranata. Copper uptake by cultured human cells. 1985.

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14

Kirchman, David L. Predation and protists. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789406.003.0009.

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Protists are involved in many ecological roles in natural environments, including primary production, herbivory and carnivory, and parasitism. Microbial ecologists have been interested in these single-cell eukaryotes since Antonie van Leeuwenhoek saw them in his stool and scum from his teeth. This chapter focuses on the role of protozoa (purely heterotrophic protists) and other protists in grazing on other microbes. Heterotrophic nanoflagellates, 3–5 microns long, are the most important grazers of bacteria and small phytoplankton in aquatic environments. In soils, flagellates are also importan
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15

Koller-Lucae, Sibylle Klara Maria. N⁴-octadecyl-1-[beta]-D-arabinofuranosylcytosine (NOAC), a new anticancer drug: Pharmacokinetics and metabolism inmice, distribution in human blood, uptake and cytotoxicity in lymphoma cells. 1998.

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16

Hyde, Parker, Vincent J. Miller, and Jeff S. Volek. Keto-Adaptation in Health and Fitness. Edited by Dominic P. D’Agostino. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0038.

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When dietary carbohydrate is restricted and protein consumed in moderation, the evolutionarily-conserved ketogenic metabolic machinery awakens. After just a few days circulating ketones increase by an order of magnitude, and over several weeks there is a profound shift away from glucose as the primary energy substrate to the preferred use of fatty acids and ketones. This metabolic process is known as keto-adaptation. The deemphasis on insulin-dependent glucose uptake into cells and concomitant increase in fat oxidation has important implications in management of insulin resistance and its seco
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