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Journal articles on the topic 'Cells'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 May 2025

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1

Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (August 31, 2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted, regulated, and executed.

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2

MG, Bottone. "A Brief Communication on a Cell Line of Neural Stem Cells B50 Cells Treated With a New Cisplatin - Based Drug." Journal of Embryology & Stem Cell Research 2, no. 1 (2018): 1–4. http://dx.doi.org/10.23880/jes-16000108.

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3

CPK, Cheung. "T Cells, Endothelial Cell, Metabolism; A Therapeutic Target in Chronic Inflammation." Open Access Journal of Microbiology & Biotechnology 5, no. 2 (2020): 1–6. http://dx.doi.org/10.23880/oajmb-16000163.

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The role of metabolic reprogramming in the coordination of the immune response has gained increasing consideration in recent years. Indeed, it has become clear that changes in the metabolic status of immune cells can alter their functional properties. During inflammation, stimulated immune cells need to generate sufficient energy and biomolecules to support growth, proliferation and effector functions, including migration, cytotoxicity and production of cytokines. Thus, immune cells switch from oxidative phosphorylation to aerobic glycolysis, increasing their glucose uptake. A similar metabolic reprogramming has been described in endothelial cells which have the ability to interact with and modulate the function of immune cells and vice versa. Nonetheless, this complicated interplay between local environment, endothelial and immune cells metabolism, and immune functions remains incompletely understood. We analyze the metabolic reprogramming of endothelial and T cells during inflammation and we highlight some key components of this metabolic switch that can lead to the development of new therapeutics in chronic inflammatory disease.

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4

Gaddikeri1, Kavitha, and Deepak D. Bhorgonde2. "Assessment of role of mast cells in oral squamous cell carcinoma." Asian Pacific Journal of Health Sciences 3, Supplimentary 2016 (December 31, 2016): 63–66. http://dx.doi.org/10.21276/apjhs.2016.3.4s.9.

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5

Abramova, V. A., A. Kali, N. Abdolla, O. Yu Yurikova, Yu V. Perfilyeva, Ye O. Ostapchuk, R. T. Tleulieva, S. K. Madenova, and N. N. Belyaev. "Influence of tumor cells on natural killer cell phenotype and cytotoxicity." International Journal of Biology and Chemistry 8, no. 1 (2015): 9–14. http://dx.doi.org/10.26577/2218-7979-2015-8-1-9-14.

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6

YAMAMOTO, Takamitsu. "C207 DEVELOPMENT OF FUEL CELLS POWERED RAILWAY VEHICLE(Fuel Cell-1)." Proceedings of the International Conference on Power Engineering (ICOPE) 2009.2 (2009): _2–213_—_2–218_. http://dx.doi.org/10.1299/jsmeicope.2009.2._2-213_.

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7

Ahmed Elkammar, Hala. "Effect of human bone marrow derived mesenchymal stem cells on squamous cell carcinoma cell line." International Journal of Academic Research 6, no. 1 (January 30, 2014): 110–16. http://dx.doi.org/10.7813/2075-4124.2014/6-1/a.14.

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8

Deniz, Özdemir. "KAN0438757: A NOVEL PFKFB3 INHIBITOR THAT INDUCES PROGRAMMED CELL DEATH AND SUPPRESSES CELL MIGRATION IN NON-SMALL CELL LUNG CARCINOMA CELLS." Biotechnologia Acta 16, no. 5 (October 31, 2023): 34–44. http://dx.doi.org/10.15407/biotech16.05.034.

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Aim. PFKFB3 is glycolytic activators that is overexpressed in human lung cancer and plays a crucial role in multiple cellular functions including programmed cell death. Despite the many small molecules described as PFKFB3 inhibitors, some of them have shown disappointing results in vitro and in vivo. On the other hand KAN0438757, selective and potent, small molecule inhibitor has been developed. However, the effects of KAN0438757, in non-small cell lung carcinoma cells remain unknown. Herein, we sought to decipher the effect of KAN0438757 on proliferation, migration, DNA damage, and programmed cell death in non-small cell lung carcinoma cells. Methods. The effects of KAN0438757 on cell viability, proliferation, DNA damage, migration, apoptosis, and autophagy in in non-small cell lung carcinoma cells was tested by WST-1, real-time cell analysis, comet assay, wound-healing migration test, and MMP/JC-1 and AO/ER dual staining assays as well as western blot analysis. Results. Our results revealed that KAN0438757 significantly suppressed the viability and proliferation of A549 and H1299 cells and inhibited migration of A549 cells. More importantly, KAN0438757 caused DNA damage and triggered apoptosis and this was accompanied by the up-regulation of cleaved PARP in A549 cells. Furthermore, treatment with KAN0438757 resulted in increased LC3 II and Beclin1, which indicated that KAN0438757 stimulated autophagy. Conclusions. Overall, targeting PFKFB3 with KAN0438757 may be a promising effective treatment approach, requiring further in vitro and in vivo evaluation of KAN0438757 as a therapy in non-small cell lung carcinoma cells.

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J, Otsuka. "A Theoretical Study on the Cell Differentiation Forming Stem Cells in Higher Animals." Physical Science & Biophysics Journal 5, no. 2 (2021): 1–10. http://dx.doi.org/10.23880/psbj-16000191.

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The recent genome sequencing of multicellular diploid eukaryotes reveals an enlarged repertoire of protein genes for signal transmission but it is still difficult to elucidate the network of signal transmission to drive the life cycle of such an eukaryote only from biochemical and genetic studies. In the present paper, a theoretical study is carried out for the cell differentiation, the formation of stem cells and the growth from a child to the adult in the higher animal. With the intercellular and intracellular signal transmission in mind, the cell differentiation is theoretically derived from the process by the transition of proliferated cells from proliferation mode to differentiation mode and by both the long-range interaction between distinctive types of cells and the short-range interaction between the same types of cells. As the hierarchy of cell differentiation is advanced, the original types of self-reproducible cells are replaced by the self-reproducible cells returned from the cells differentiated already. The latter type of self-reproducible cells are marked with the signal specific to the preceding differentiation and become the stem cells for the next stage of cell differentiation. This situation is realized under the condition that the differentiation of cells occurs immediately after their proliferation in the development. The presence of stem cells in the respective lineages of differentiated cells strongly suggests another signal transmission for the growth of a child to a definite size of adult that the proliferation of stem cells in one lineage is activated by the signal from the differentiated cells in the other lineage(s) and is suppressed by the signal from the differentiated cells in its own lineage. This style of signal transmission also explains the metamorphosis and maturation of germ cells in higher animals.

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10

Fujimoto, Naohiro, Bin Han, Masayoshi Nomura, and Tetsuro Matsumoto. "WS1-1-1 Nitrogen-Containing Bisphosphonates Inhibit the Growth of Renal Cell Carcinoma Cells(Renal Cell Cancer)." Japanese Journal of Urology 99, no. 2 (2008): 142. http://dx.doi.org/10.5980/jpnjurol.99.142_1.

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11

S Deshmukh, M. "Fuel Cell Technology Evolution: Insights into Polymer, Solid Oxide, and Alkaline Fuel Cells." International Journal of Science and Research (IJSR) 13, no. 10 (October 5, 2024): 450–55. http://dx.doi.org/10.21275/sr241003221704.

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12

M. Baghdadi, Houry. "Effect of stem cells on genetic mutations and proliferation in squamous cell carcinoma." International Journal of Academic Research 6, no. 1 (January 30, 2014): 192–97. http://dx.doi.org/10.7813/2075-4124.2014/6-1/a.25.

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13

M, Aloysius Dhivya, Kishore A, and Bharathidevi SR. "Down-Regulation of NRF2 Signaling Triggers Cell Death in Y79 Cells upon Copper Chelation." Open Access Journal of Ophthalmology 10, no. 1 (2025): 1–10. https://doi.org/10.23880/oajo-16000329.

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Background: Retinoblastoma is a rare intraocular malignancy that leads to vision loss in children. While copper (Cu) chelation has been reported as a therapy in many cancers, its relevance to retinoblastoma has not yet been explored. Objectives: In this study, we have explored the role of penicillamine (a Cu chelator) as a therapeutic target for retinoblastoma using Y79 cells as a model. Methods: The effect of the Cu chelator on the viability of Y79 was assessed using the MTT assay. Additionally, we performed nuclear fractionation to assess Nuclear factor erythroid 2–related factor 2 (NRF2) activation, evaluated the downstream targets of NRF2 at transcript and protein levels, and measured SOD (superoxide dismutase) activity. Results: Penicillamine (2 mM) induced cell death in Y79 cells, inhibited NRF2 signaling, and reduced SOD activity. Furthermore, the downstream targets of NRF2 namely VEGF as well as PCNA (proliferating cell nuclear antigen) were decreased with penicillamine, which induced cell death in Y79 cells. We observed similar results in HeLa cells (positive control) comparable to Y79 cells. Conclusion: Therefore, we speculate that penicillamine could be a target for retinoblastoma as it induces cell death in Y79 cells by regulating NRF2 nuclear translocation and decreasing its downstream targets.

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14

F. Abdel Hamid, Mahmoud, Safaa M. Morsy, Mostafa Abou El Ela, Rehab A. Hegazy, Marwa M. Fawzy, Laila A. Rashed, Ahmed M. Omar, Eman R. Abdel Fattah, and Doaa M. Hany. "T helper-17 cells and T regulatory cells in vitiligo." International Journal of Academic Research 5, no. 6 (December 10, 2013): 273–78. http://dx.doi.org/10.7813/2075-4124.2013/5-6/a.34.

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15

Hananeh, W., R. Al Rukibat, and M. Daradka. "Primary splenic diffuse large B-cell lymphoma with multinucleated giant cells in a horse." Veterinární Medicína 66, No. 2 (February 2, 2021): 76–79. http://dx.doi.org/10.17221/61/2020-vetmed.

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A diagnosis of a diffuse splenic large B-cell lymphoma with multinucleated giant cells in a 5-year-old mare was made based upon the clinical, pathological, and immunohistochemical findings. The enormous primary splenic mass weighed 51.75 kg. To the best of our knowledge, this is the biggest reported splenic mass and the first case of an equine diffuse large B-cell lymphoma with multinucleated giant cells.

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16

Challa, Stalin Reddy, and Swathi Goli. "Differentiation of Human Embryonic Stem Cells into Engrafting Myogenic Precursor Cells." Stem cell Research and Therapeutics International 1, no. 1 (April 16, 2019): 01–05. http://dx.doi.org/10.31579/2643-1912/002.

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Degenerative muscle diseases affect muscle tissue integrity and function. Human embryonic stem cells (hESC) are an attractive source of cells to use in regenerative therapies due to their unlimited capacity to divide and ability to specialize into a wide variety of cell types. A practical way to derive therapeutic myogenic stem cells from hESC is lacking. In this study, we demonstrate the development of two serum-free conditions to direct the differentiation of hESC towards a myogenic precursor state. Using TGFß and PI3Kinase inhibitors in combination with bFGF we showed that one week of differentiation is sufficient for hESC to specialize into PAX3+/PAX7+ myogenic precursor cells. These cells also possess the capacity to further differentiate in vitro into more specialized myogenic cells that express MYOD, Myogenin, Desmin and MYHC, and showed engraftment in vivo upon transplantation in immunodeficient mice. Ex vivo myomechanical studies of dystrophic mouse hindlimb muscle showed functional improvement one month post-transplantation. In summary, this study describes a promising system to derive engrafting muscle precursor cells solely using chemical substances in serum-free conditions and without genetic manipulation.

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17

MAS, Bezerra, Ferreira LAM, Kawasaki-Oyama RS, Nascimento MCA, Cuzziol CI, Castanhole-Nunes MMU, Pavarino EC, Maniglia JM, and Goloni-Bertollo EM. "Effectiveness of Hypoxia-Induced Accumulation of Cancer Stem Cells in Head and Neck Squamous Cell Carcinoma." Cancer Medicine Journal 3, S1 (November 30, 2020): 13–23. http://dx.doi.org/10.46619/cmj.2020.3.s1-1003.

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INTRODUCTION: The small number of cancer stem cells, which correspond to only 0.01% - 0.1% of total tumor cells, has been the biggest obstacle in understanding their biology and role in the origin and maintenance of tumors, their metastatic and recurrence potentials, and resistance to radio-chemotherapy. Therefore, promoting its accumulation will enable further studies and future advances in the diagnosis and treatment of head and neck cancer squamous cell carcinoma. OBJECTIVE: To induce cancer stem cell accumulation in primary cell cultures of head and neck squamous cell carcinoma using a hypoxia chamber. METHODS: Head and neck squamous cell carcinoma samples were cultured and subjected to hypoxia. Oxygen deprivation aimed to induce cancer stem cell accumulation. RESULTS: Immediately after hypoxia, the percentage of O2-deprived cancer stem cells increased 2-fold as compared to control. Surprisingly, new phenotyping performed 45 days after hypoxia showed a 9-fold increase in cancer stem cell percentage in cells that suffered hypoxia. Hypoxic cells showed an increase in spheroid formation when compared to control cells, as well as enhanced abilities in invasion and migration. CONCLUSION: Hypoxia was efficient in cancer stem cell accumulation. As cancer stem cells are a small number of cells within the tumor, promoting their accumulation will enable further studies and future advances in the diagnosis and treatment of head and neck cancer.

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18

Zamponi, Mauricio O. "The Choanocyte Cells from Phylum Porifera, Nematocysts Cells from Phylun Cnidaria and Colloblast Cells from Ctenophora like Analogues Structures." International Journal of Zoology and Animal Biology 7, no. 5 (2024): 1–4. http://dx.doi.org/10.23880/izab-16000614.

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There are three basic structures belongs to Phylum Porifera, Phylum Cnidaria and Phylum Ctenophora very important because they have similar function like to catch food, but three of them have different origin and different structuration too. They are knowing like choanocyte (Porifera), nematocysts (Cnidaria) and colloblast (Ctenophora) so they are considered analogue structures and they have different positions in the animal; meanwhile the first one are localized in the general cavity (asconoid form, leuconoid form) of sponge, the second one are localized in different zones of the animal like tentacles, acontium , verrucae , vesicule, acrorargus and the last one is on tentacles.

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19

Y, Elshimali. "Chimeric Antigen Receptor T-Cell Therapy (Car T-Cells) in Solid Tumors, Resistance and Success." Bioequivalence & Bioavailability International Journal 6, no. 1 (2022): 1–6. http://dx.doi.org/10.23880/beba-16000163.

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CARs are chimeric synthetic antigen receptors that can be introduced into an immune cell to retarget its cytotoxicity toward a specific tumor antigen. CAR T-cells immunotherapy demonstrated significant success in the management of hematologic malignancies. Nevertheless, limited studies are present regarding its efficacy in solid and refractory tumors. It is well known that the major concerns regarding this technique include the risk of relapse and the resistance of tumor cells, in addition to high expenses and limited affordability. Several factors play a crucial role in improving the efficacy of immunotherapy, including tumor mutation burden (TMB), microsatellite instability (MSI), loss of heterozygosity (LOH), the APOBEC Protein Family, tumor microenvironment (TMI), and epigenetics. In this minireview, we address the current and future applications of CAR T-Cells against solid tumors and their measure for factors of resistance and success.

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20

Manimaran, K., Arun Kumar, AvinashGandi D, and S. Sankaranarayanan. "Interaction of Human Dental Pulp Stem Cells and Ameloblastic Cell In-vitro- A Preclinical Analysis." Annals of Oral Health and Dental Research 2, no. 1 (January 17, 2018): A1–5. http://dx.doi.org/10.21276/aohdr.1831.

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21

Guo, Y. M., J. Gong, Y. G. Zheng, B. L. Shi, X. Y. Guo, and S. M. Yan. "Protection of bovine mammary epithelial cells from hydrogen peroxide-induced oxidative cell damage by selenium." Czech Journal of Animal Science 63, No. 3 (February 9, 2018): 94–102. http://dx.doi.org/10.17221/76/2017-cjas.

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The uncontrolled release of arachidonic acid (ARA) and its metabolism by lipoxygenase (LOX) pathway can induce and aggravate cellular oxidative stress. Selenium (Se) is an integral part of some antioxidative selenoproteins and may protect cells from oxidative damage by modulating ARA release and metabolism. The present study aimed to investigate the protective response of Se against hydrogen peroxide (H<sub>2</sub>O<sub>2</sub>)-induced oxidative damage in bovine mammary epithelial cells (BMECs). The BMECs were incubated for 24 h in serum-free medium and then divided into four groups randomly. The cells in groups 1 and 2 were subsequently incubated for 30 h in serum-free medium containing 0 (control) and 50 nM Se (Se treatment group). The cells in groups 3 and 4 were incubated for 24 h in serum-free medium containing 0 and 50 nM Se, and then treated with 600 μM H<sub>2</sub>O<sub>2</sub> for 6 h (H<sub>2</sub>O<sub>2</sub> damage group and Se prevention group). The results showed that Se attenuated the H<sub>2</sub>O<sub>2</sub>-induced production of reactive oxygen species and the decrease of antioxidative enzymes as glutathione peroxidase (GPX), thioredoxin reductase (TrxR), selenoprotein P (SelP), superoxide dismutase, and catalase in BMECs. The preventive effects of Se on the decrease of selenoprotein activity were demonstrated further by the increase of mRNA expression for GPX1, TrxR1, and SelP, and protein expression for GPX1 and TrxR1. Pretreatment of cells with Se inhibited the H<sub>2</sub>O<sub>2</sub>-induced increase of mRNA expressions and activities for cytosolic phospholipase A2 and 5-lipoxygenase, ARA release, and 15-hydroperoxyeicosatetraenoic acid production. Se also blocked the H<sub>2</sub>O<sub>2</sub>-induced activation of p38 mitogen-activated protein kinase (MAPK) and c-Jun N-terminal kinase but not that of extracellular signal-regulated kinase. These results suggested that Se may protect BMECs against H<sub>2</sub>O<sub>2</sub>-induced oxidative damage by increasing selenoproteins synthesis, inhibiting MAPK pathway, and then decreasing ARA release and its metabolism by LOX pathway.

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22

Moorthy, Rajesh Kannan, Chandhru Srinivasan, Sridharan Jayamohan, Mahesh Kumar Kannan, Siva Sankari Thirugnanam, Janaki Sankar Ganesh, and Antony Joseph Velanganni Arockiam. "Knockdown of microRNA-375 suppresses cell proliferation and promotes apoptosis in human breast cancer cells." Indian Journal of Science and Technology 14, no. 43 (November 12, 2021): 3199–209. http://dx.doi.org/10.17485/ijst/v14i43.1719.

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23

PD, Gupta. "Liver Cells Can Dedifferentiate and Act as Progenitor Cells for Liver Growth." Journal of Embryology & Stem Cell Research 3, no. 2 (2019): 1–2. http://dx.doi.org/10.23880/jes-16000124.

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24

Sun, Hongyan, Guomin Zhang, Fulu Dong, Feng Wang, and Wenguang Cao. "Reprogramming Sertoli Cells into Pluripotent Stem Cells." Cellular Reprogramming 16, no. 3 (June 2014): 196–205. http://dx.doi.org/10.1089/cell.2013.0083.

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25

Sachenko, A. V. "Lateral multijunction photovoltaic cells." Semiconductor Physics Quantum Electronics and Optoelectronics 16, no. 1 (February 28, 2013): 1–17. http://dx.doi.org/10.15407/spqeo16.01.001.

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26

Jeon, Soo-Been, Bo-Gyeong Seo, Sang-Ki Baek, Hyeon-Geun Lee, Joon-Hong Shin, In-Won Lee, Hyo-Jin Kim, et al. "Endothelial Cells Differentiated from Porcine Epiblast Stem Cells." Cellular Reprogramming 23, no. 2 (April 1, 2021): 89–98. http://dx.doi.org/10.1089/cell.2020.0088.

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27

Tasneef Azam, Tasneef Azam, Fouzia Noreen Fouzia Noreen, Bina S. Siddiqui Bina S Siddiqui, Rahman M. Hafizur Rahman M Hafizur, and Sabira Begum Sabira Begum. "Suppression of β-Cell Apoptosis from H2O2-Induced Oxidative Stress in MIN6 cells using Methyl Gallate." Journal of the chemical society of pakistan 44, no. 1 (2022): 84. http://dx.doi.org/10.52568/000983/jcsp/44.01.2022.

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A major parameter for diabetic relevant diseases and hyperglycemia is the β-cell apoptosis. Anti diabetic drugs used widely these days chiefly target to lower hyperglycemia along with prevention of β-cells from apoptosis. In this study three natural products methyl gallate, syringic acid, and butanedioic acid from Myricaria germanica were analyzed for β-cell protection. Methyl gallate provided significant β-cell protection from H2O2-induced oxidative stress mediated apoptosis in MIN6 cells at 50 μM (95.5% and#177; 16.0 vs 57.6% and#177; 1.1) and at 100 μM (85.5% and#177; 7.0 vs 57.6% and#177; 1.1) concentrations.

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Kosaka, Yudai, and Tetsuhiko Ohba. "3P174 Study on membrane microfluidity of living cells using Muller Matrix microscopy(12. Cell biology,Poster)." Seibutsu Butsuri 53, supplement1-2 (2013): S240. http://dx.doi.org/10.2142/biophys.53.s240_5.

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RemyaV, RemyaV, Naveen Kumar, and Kutty M. V. H. Kutty M.V.H. "A Method for Cell Culture and RNA Extraction of Rabbit Bone Marrow Derived Mesenchymal Stem Cells." International Journal of Scientific Research 3, no. 7 (June 1, 2012): 31–33. http://dx.doi.org/10.15373/22778179/july2014/11.

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Robinson, Richard. "Cells within cells (within cells)." Journal of Cell Biology 179, no. 7 (December 18, 2007): 1328. http://dx.doi.org/10.1083/jcb.1797rr1.

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Bhatti, M. Tariq, Katherine E. Gres, Virginia B. Petitto, and Shelley Ann Cross. "Cells, Cells, and More Cells." Survey of Ophthalmology 52, no. 3 (May 2007): 315–24. http://dx.doi.org/10.1016/j.survophthal.2007.02.009.

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Chan, Anthony W. S., Pei-Hsun Cheng, Adam Neumann, and Jin-Jing Yang. "Reprogramming Huntington Monkey Skin Cells into Pluripotent Stem Cells." Cellular Reprogramming 12, no. 5 (October 2010): 509–17. http://dx.doi.org/10.1089/cell.2010.0019.

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Balch, Ying. "Subculture human skeletal muscle cells to produce the cells with different Culture medium compositions." Clinical Research and Clinical Trials 3, no. 4 (April 30, 2021): 01–03. http://dx.doi.org/10.31579/2693-4779/036.

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This study aimed to subculture human skeletal muscle cells (HSkMC) using a culture medium with different compositions to determine the most efficient medium for the growth of the human skeletal muscle cells. The culture media was divided into three groups: Group1. An HSkMC growth medium. Group 2. An HSkMC growth medium + with 10% high glucose (GH). Group 3. An HSkMC growth medium + 10% fetal bovine serum (FBS). HSkMC from groups 1 to 3 gradually became round in shape and gathered in clusters. These changes differed between the groups. In group 3, the HSkMC clusters were more in numbers and gathered as significantly more prominent than in the other groups under the EVOS-Microscope shown. We concluded that by manipulating the composition of the culture medium, it is possible to induce HSkMC to promote the best growth.

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Elgaly, Maher E., Mohamed E. El Ghareeb, and Farha El shennawy. "Cord Blood Mesenchymal Stem Cells Conditioned Media Suppress Epithelial Ovarian Cancer Cells in Vitro." International Journal of Trend in Scientific Research and Development Volume-2, Issue-5 (August 31, 2018): 1783–88. http://dx.doi.org/10.31142/ijtsrd18182.

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35

Gallon, Alex. "Auto Fluorescent Cells, Including Mesenchymal Stem Cells, are Indeed Amenable to Flow Cytometric Analyses." Stem Cells Research and Therapeutics International 1, no. 1 (April 16, 2019): 01. http://dx.doi.org/10.31579/2643-1912/001.

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36

SK, Deshmukh. "Immune Cells in the Tumor Microenvironment and Cancer Stem Cells: Interplay for Tumor Progression." Journal of Embryology & Stem Cell Research 2, no. 2 (2018): 1–2. http://dx.doi.org/10.23880/jes-16000109.

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Arai, Masataka, Shota Hori, Satoshi Miyamoto, Kazuhiro Nakashima, Toshihiro Sera, and Susumu Kudo. "OS18-5 Mechanical Stimulus Effects Diacylglycerol Distribution in Vascular Endothelial Cells(Cell and Tissue mechanics 2,OS18 Cell and tissue mechanics,BIOMECHANICS)." Abstracts of ATEM : International Conference on Advanced Technology in Experimental Mechanics : Asian Conference on Experimental Mechanics 2015.14 (2015): 239. http://dx.doi.org/10.1299/jsmeatem.2015.14.239.

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38

Marbán, Eduardo. "Big Cells, Little Cells, Stem Cells." Circulation Research 100, no. 4 (March 2, 2007): 445–46. http://dx.doi.org/10.1161/01.res.0000260271.33215.9b.

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Srinivas, Ampati. "The Constantly Highly Expression of Limbal Stromal Cells Compared to the Bone Marrow Mesenchymal Stromal Cells, Adipose-Derived Mesenchymal Stromal Cells and Foreskin Fibroblasts." Stem Cells Research and Therapeutics International 1, no. 1 (April 16, 2019): 01–06. http://dx.doi.org/10.31579/2643-1912/005.

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40

Nichat, Prachi Bhaurao, Kirti Chadha Kazi, Prem Punjabi, and Vikas Kavishwar. "Osteoclast-like Multinucleated Giant Cells." Annals of Pathology and Laboratory Medicine 5, no. 7 (July 29, 2018): C104–106. http://dx.doi.org/10.21276/apalm.1851.

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41

CR, Thambidorai. "Stem Cells in Urethral Replacement." Journal of Embryology & Stem Cell Research 4, no. 1 (2020): 1–2. http://dx.doi.org/10.23880/jes-16000139.

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Zamay, A. S., O. S. Kolovskaya, N. M. Titova, L. L. Chesnokova, E. A. Maltceva, and T. N. Zamay. "CALCIUM SIGNALING IN TUMOR CELLS." Siberian Medical Review, no. 3 (2016): 27–33. http://dx.doi.org/10.20333/25000136-2016-3-27-33.

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43

Baek, DS, TH Chung, YH Kim, SK Oh, KM So, and C. Park. "Changes in regulatory T cells in dogs with B-cell lymphoma and association with clinical tumour stage." Veterinární Medicína 62, No. 12 (December 4, 2017): 647–53. http://dx.doi.org/10.17221/7/2015-vetmed.

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Among several mechanisms that allow tumours to disarm the host immune system and thus to evade or suppress protective anti-tumour immunity, an important role for CD4<sup>+</sup>CD25<sup>+</sup>FoxP3<sup>+</sup> regulatory T cells (Tregs) has emerged. Numerous studies in humans have demonstrated increased Tregs in patients with carcinomas of the breast, lung, and pancreas, and this increased Treg has been correlated with poor prognosis. This study was performed (1) to investigate the percentage of Tregs in total lymphocytes of the peripheral blood in 12 canine patients with B cell lymphoma and (2) to investigate the change in the percentage of Tregs in canine lymphoma of different clinical tumour stages. On the flow cytometric analysis, the relative and absolute numbers of Tregs were significantly increased in 12 canine patients with B-cell lymphoma compared to five healthy beagles included in this study, and the greatest increases in the relative and absolute number of Tregs occurred in two dogs with more advanced World Health Organization clinical stages with bone marrow involvement compared to those in less advanced tumour stages without bone marrow involvement. This study provides basic information regarding the negative role of Treg recruitment in canine lymphoma patients and highlights the potential value of Treg levels as prognostic indicators in canine cancer patients.

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Z, Ding. "Concentration Polarization of Ox-LDL and Its Effect on Cell Proliferation and Apoptosis in Human Endothelial Cells." Journal of Cardiology and Cardiovascular Medicine 1, no. 1 (2016): 011–18. http://dx.doi.org/10.29328/journal.jccm.1001003.

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Kusuma, Riska Anggri, Linda Suyati, and Wasino Hadi Rahmanto. "Effect of Lactose Concentration as Lactobacillus bulgaricus Substrate on Potential Cells Produced in Microbial Fuel Cell Systems." Jurnal Kimia Sains dan Aplikasi 21, no. 3 (July 31, 2018): 144–48. http://dx.doi.org/10.14710/jksa.21.3.144-148.

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The effect of laxose concentration as Lactobacillus bulgaricus bacterial substrate on the cell potential produced in Microbial Fuel Cell System has been done. This study aims to determine the effect of lactose concentration as bacterial substrate, to generate electricity, maximum electric potential and determine the potential value of standard lactose (E ° Lactose.) Based on Nernst equation. The MFC system of two compartments and bridges of salt as a linkage is used in this study. Anode contains lactose with variation of concentration 3 - 7% and bacteria. The cathode contains a 1M KMO4. The electrodes used are graphite. MFC operational time is 14 days. The results showed that the lactose concentration had an effect on the cell potential produced in the MFC system. Maximum cell potential yielded at 4% lactose concentration, that is 710 mV then based on Nerst equation theory obtained E ° Lactose value in MFC system of + 0,236 V.

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Tavartkiladze, Alexandre, Revaz Turmanidze, Gaiane Simonia, Margalita Gogoladze, Pati Revazishvili, George Dundua, and Irine Andronikashvili. "Circulating Tumor Cells and cfDNA: Key Predictive Biomarkers in Non - Small Cell Lung Cancer Progression and Treatment." International Journal of Science and Research (IJSR) 12, no. 10 (October 5, 2023): 1078–81. http://dx.doi.org/10.21275/sr231013031834.

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O, CEVIK. "Exosomes Released from TRAIL -Resistance Glioblastoma Cells Influence the Migration and Growth of Healthy Cells." Cell & Cellular Life Sciences Journal 7, no. 2 (2022): 1–6. http://dx.doi.org/10.23880/cclsj-16000175.

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Exosomes are the smallest vesicles that can cross the blood-brain barrier and transport proteins, nucleic acids, and other cell elements to cells. The role of exosomes in drug resistance and their effects on healthy cells have been researched topics in recent years. This study investigated the effect of exosomes released from TRAIL resistance U87 glioblastoma cells on healthy L929 fibroblast cells. U87 cells were incubated with TRAIL (5ng/mL) for three weeks to develop drug resistance and exosomes were isolated with ultracentrifuge. The obtained exosomes were incubated at a concentration of 1 µg/mL-100 µg/ mL on L929 cells for 48 hours, and their cytotoxic effect was examined with MTT assay. After incubation, CXCR4 Protein and gene expression levels of CXCL-12, PAK4, and 14-3-3β were also examined. This study showed that exosomes released from TRAIL-resistant cells increase CXCR4, CXCL-12, and PAK4 levels in healthy cells and do not cause changes in 14-3-3 β oncogenic protein. While 100 µg/mL exosome affected L929 cells, no change was seen at a lower concentration. Exosomes affect the migration potential of other cells due to drug resistance. It may contribute to developing different resistance mechanisms in the long term by changing the proliferation and migration abilities of healthy cells around the tumor cell.

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Galende, Elisa, Ioannis Karakikes, Lisa Edelmann, Robert J. Desnick, Thomas Kerenyi, Georges Khoueiry, James Lafferty, et al. "Amniotic Fluid Cells Are More Efficiently Reprogrammed to Pluripotency Than Adult Cells." Cellular Reprogramming 12, no. 2 (April 2010): 117–25. http://dx.doi.org/10.1089/cell.2009.0077.

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Li, Danyang, Yiming Liu, Jinyan Qi, Xinhua Cui, Ying Guo, Dipanpan Wu, and Hui Liang. "Bone Marrow Mesenchymal Stem Cells Promote the Stemness of Hypopharyngeal Cancer Cells." Cellular Reprogramming 22, no. 5 (October 1, 2020): 269–76. http://dx.doi.org/10.1089/cell.2020.0004.

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E. Young, Henry, Mark O. Speight, and Asa C. Black Jr. "Functional Cells, Maintenance Cells, and Healing Cells." Stem Cell & Regenerative Medicine 1, no. 1 (June 30, 2017): 1–4. http://dx.doi.org/10.33425/2639-9512.1003.

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