Relevant bibliographies by topics / Cellular Proliferation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cellular Proliferation'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Cellular Proliferation"

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Yao, Guang. "Modelling mammalian cellular quiescence." Interface Focus 4, no. 3 (2014): 20130074. http://dx.doi.org/10.1098/rsfs.2013.0074.

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Cellular quiescence is a reversible non-proliferating state. The reactivation of ‘sleep-like’ quiescent cells (e.g. fibroblasts, lymphocytes and stem cells) into proliferation is crucial for tissue repair and regeneration and a key to the growth, development and health of higher multicellular organisms, such as mammals. Quiescence has been a primarily phenotypic description (i.e. non-permanent cell cycle arrest) and poorly studied. However, contrary to the earlier thinking that quiescence is simply a passive and dormant state lacking proliferating activities, recent studies have revealed that
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Hatchell, D. L., T. McAdoo, S. Sheta, R. T. King, and J. V. Bartolome. "Quantification of Cellular Proliferation in Experimental Proliferative Vitreoretinopathy." Archives of Ophthalmology 106, no. 5 (1988): 669–72. http://dx.doi.org/10.1001/archopht.1988.01060130731033.

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Rubtsova, Maria P., Denis A. Nikishin, Mikhail Y. Vyssokikh, Maria S. Koriagina, Andrey V. Vasiliev, and Olga A. Dontsova. "Telomere Reprogramming and Cellular Metabolism: Is There a Link?" International Journal of Molecular Sciences 25, no. 19 (2024): 10500. http://dx.doi.org/10.3390/ijms251910500.

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Telomeres—special DNA–protein structures at the ends of linear eukaryotic chromosomes—define the proliferation potential of cells. Extremely short telomeres promote a DNA damage response and cell death to eliminate cells that may have accumulated mutations after multiple divisions. However, telomere elongation is associated with the increased proliferative potential of specific cell types, such as stem and germ cells. This elongation can be permanent in these cells and is activated temporally during immune response activation and regeneration processes. The activation of telomere lengthening m
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Zhang, Jian Chun, Howard E. Savage, Peter G. Sacks, et al. "Innate cellular fluorescence reflects alterations in cellular proliferation." Lasers in Surgery and Medicine 20, no. 3 (1997): 319–31. http://dx.doi.org/10.1002/(sici)1096-9101(1997)20:3<319::aid-lsm11>3.0.co;2-8.

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Calderón, Christian G., and Francisco Arvelo. "Kca3.1-Related Cellular Signalling Involved in Cancer Proliferation." Cellular Physiology and Biochemistry 58, no. 1 (2024): 107–27. http://dx.doi.org/10.33594/000000688.

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Anomalous expression of potassium channels in cancer tissues is associated with several cancer hallmarks that support deregulated proliferation and tumor progression. Ion channels seem to influence cell proliferation; however, the crucial molecular mechanisms involved remain elusive. Some results show how extracellular mitogenic signals modulate ion channel activity through intracellular secondary messengers. It is relevant because we are beginning to understand how potassium channels can affect the proliferative capacity of cells, either in normal mitogen-dependent proliferation or in mitogen
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CLARKE, CHRISTINE L., and ROBERT L. SUTHERLAND. "Progestin Regulation of Cellular Proliferation*." Endocrine Reviews 11, no. 2 (1990): 266–301. http://dx.doi.org/10.1210/edrv-11-2-266.

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Lenkala, Divya, Eric R. Gamazon, Bonnie LaCroix, Hae Kyung Im, and R. Stephanie Huang. "MicroRNA biogenesis and cellular proliferation." Translational Research 166, no. 2 (2015): 145–51. http://dx.doi.org/10.1016/j.trsl.2015.01.012.

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Mankoff, David A., Anthony F. Shields, and Kenneth A. Krohn. "PET imaging of cellular proliferation." Radiologic Clinics of North America 43, no. 1 (2005): 153–67. http://dx.doi.org/10.1016/j.rcl.2004.09.005.

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VINCENT, P. C. "Leukemic Cellular Proliferation: A Perspective." Annals of the New York Academy of Sciences 459, no. 1 Hematopoietic (1985): 308–27. http://dx.doi.org/10.1111/j.1749-6632.1985.tb20839.x.

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Zlotorynski, Eitan, and Reuven Agami. "A PASport to Cellular Proliferation." Cell 134, no. 2 (2008): 208–10. http://dx.doi.org/10.1016/j.cell.2008.07.003.

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Dissertations / Theses on the topic "Cellular Proliferation"

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Gan, Lisha. "Corneal cellular proliferation and wound healing /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4505-5/.

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Kranc, Kamil. "The role of Cited2 in cellular proliferation." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398233.

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PIUNTI, ANDREA. "POLYCOMB ROLE IN CELLULAR PROLIFERATION AND TRANSFORMATION." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/696485.

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The Polycomb Group proteins (PcGs) are present in cells nuclei as two main repressive complexes named Polycomb Repressive Complex 1 (PRC1) and 2 (PRC2). Both have been involved in several cellular functions among which the ability to promote cellular proliferation is the main PcG feature that links their activity to cancer development. Both complexes are directly involved in repressing the transcription of the Ink4aArf locus that encodes for the tumor suppressive proteins p16 and p19/Arf (p14/Arf in humans), potent inhibitors of cell growth via the positive regulation of pRb and p53 functions.
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Sangfelt, Olle. "Effects of interferon on cellular proliferation and apoptosis /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19981014sang.

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Stacy, Andrew Jared. "Regulation of ΔNp63α by TIP60 promotes cellular proliferation". Wright State University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1596151919161674.

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Chakravarthy, Usha. "The effect of gamma radiation on intraocular cellular proliferation." Thesis, Queen's University Belfast, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317046.

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Maiti, Baidehi. "E2F and survivin - key players in cellular proliferation and transformation." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1173801044.

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Khav, Eddie. "Visualizing an RB-E2F Cellular Switch that Controls Cell Proliferation." Thesis, The University of Arizona, 2013. http://hdl.handle.net/10150/297627.

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Mammalian cell proliferation is regulated by an Rb-E2F gene network. The input node of this network, Cyclin D, receives graded growth signals; the output node, E2F, generates an all-or-none response. That is, the Rb-E2F gene network functions as a cellular switch, converting analog growth signals into digital E2F activities. The On or Off of this Rb-E2F switch determines the On or Off of cell proliferation. To help better understand the analog/digital conversion mechanism, we constructed a reporter cell line to visualize the dynamic expression of Cyclin D and E2F genes by red and green fluores
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Simmons, Ambrosia. "The Role of Polarity Complex Proteins in Neural Progenitor Proliferation." Diss., Temple University Libraries, 2019. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/552083.

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Biomedical Sciences<br>Ph.D.<br>Cortical malformations arise from defects in any stage of brain development and often result in life-long disability ranging from epilepsy to developmental delay and even perinatal lethality. The neuroepithelium of the emergent cortex lays the foundation on which the future cortex will develop, and as such, neuroepithelial tissue and the neural progenitor cells (NPCs) which comprise it are critical to the proper growth and development of the cortex. Here I demonstrate the significance of neuroepithelial cell polarity determinants in cortical development and how
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Reed, Jennifer. "Interferon-gamma increases CD4+ T cell survival and proliferation." Click here for download, 2006. http://wwwlib.umi.com/cr/villanova/fullcit?p1432655.

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Books on the topic "Cellular Proliferation"

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Guest, Simon Sean. Strathmin is an intracellular regulator of cellular proliferation. University of Birmingham, 1996.

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Jones, Neil Austin. The role of a major cytosolic protein in cellular proliferation. University of Birmingham, 1992.

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L, Boynton Alton, and Leffert H. L, eds. Control of animal cell proliferation. Academic Press, 1985.

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Renato, Baserga, ed. Biological regulation of cell proliferation. Raven Press, 1986.

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S, Stein Gary, and Lian Jane B, eds. Molecular and cellular approaches to the control of proliferation and differentiation. Academic Press, 1992.

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P, Cronkite Eugene, Bond Victor P, Chandra Pradeep, et al., eds. Hematopoietic cellular proliferation: An international conference in honor of Eugene P. Cronkite. New York Academy of Sciences, 1985.

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riazi, Sheila. Pathophysiological links between impaired elastogenesis and increased cellular proliferation in development of cardiovascular disorders. National Library of Canada, 2002.

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Burton, Jean. A study of cellular proliferation rates in squamous cell carcinomas of the lung, with relation to p53 status. The Author], 1994.

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Wei, Dai, ed. Checkpoint responses in cancer therapy. Humana Press, 2008.

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International Conference on Gene Regulation, Oncogenesis, and AIDS (1st 1989 Loutráki, Greece). Oncogenesis: Oncogenes in signal transduction and cell proliferation : papers delivered at the First International Conference on Gene Regulation, Oncogenesis, and AIDS, Loutraki, Greece, September 15-21, 1989. Edited by Papas Takis S. Portfolio Pub. Co., 1990.

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Book chapters on the topic "Cellular Proliferation"

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Brockhoff, Gero. "DNA and Proliferation Analysis by Flow Cytometry." In Cellular Diagnostics. KARGER, 2008. http://dx.doi.org/10.1159/000209173.

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Matatall, Katie A., Claudine S. Kadmon, and Katherine Y. King. "Detecting Hematopoietic Stem Cell Proliferation Using BrdU Incorporation." In Cellular Quiescence. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_7.

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Jalbert, Emilie, and Eric M. Pietras. "Analysis of Murine Hematopoietic Stem Cell Proliferation During Inflammation." In Cellular Quiescence. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7371-2_14.

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Mierke, Claudia Tanja. "Cell Proliferation, Survival, Necrosis and Apoptosis." In Cellular Mechanics and Biophysics. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58532-7_16.

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Dover, R. "Basic Methods for Assessing Cellular Proliferation." In Assessment of Cell Proliferation in Clinical Practice. Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68287-5_4.

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Dover, R. "Basic Methods for Assessing Cellular Proliferation." In Assessment of Cell Proliferation in Clinical Practice. Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-3190-8_4.

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Horan, Paul Karl, Sue E. Slezak, and Bruce D. Jensen. "Cellular Proliferation History by Fluorescent Analysis." In Flow Cytometry. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84616-8_8.

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Guerrieri, Ferruccio. "The F0F1-ATP Synthase in Cell Proliferation and Aging." In Frontiers of Cellular Bioenergetics. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4843-0_27.

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Herbig, A. Katherine, Sameh Girgis, and Patrick J. Stover. "Effects of Cellular Glycine on Cell Proliferation." In Chemistry and Biology of Pteridines and Folates. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0945-5_83.

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Macieira-Coelho, Alvaro. "Slowing Down of the Cell Cycle During Fibroblast Proliferation." In Cellular Ageing and Replicative Senescence. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26239-0_3.

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Conference papers on the topic "Cellular Proliferation"

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Hung, Clark T., Eric D. Johnston, Fred Allen, Mitchell Litt, Solomon R. Pollack, and David F. Meaney. "Changes in Osteoblasts Under Time Averaged Microgravity Conditions." In ASME 1997 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-1314.

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Abstract Subtle alterations in mechanical stimuli can promote changes in mammalian cells ranging from cell realignment and intracellular signaling to immediate early gene expression. In this study, we examined the cellular growth and phenotypic activity of primary rat osteoblasts exposed to a simulated microgravity environment. We developed a method to produce time averaged microgravity conditions in tissue culture flasks and compared the response of cells in this environment to cells grown in conditions of unit gravity, both with and without hydrostatic pressure load. Three responses were mea
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Qian, Xu, He Hujun, Yang Guangtao, and Yang Xu. "Effect of Formaldehyde on Cellular Proliferation of HEK293 Cells." In 2007 1st International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/icbbe.2007.122.

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Dho, So Hee, Ji Young Kim, Chang-Jin Kim, et al. "Abstract 2916: NOXX: Friend or foe for cellular proliferation." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2916.

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Blahna, Matthew T., Matthew R. Jones, Lee J. Quinton, and Joseph P. Mizgerd. "Zcchc11 Enhances Cellular Proliferation Independent Of Its Uridyltransferase Activity." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2124.

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"The Effect of Hydroalcoholic Extract of Junipers communis on Proliferation BHK Cells." In International Conference on Cellular & Molecular Biology and Medical Sciences. Universal Researchers (UAE), 2016. http://dx.doi.org/10.17758/uruae.ae0916411.

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Savage, Howard E., Venkateswara Kolli, Jian C. Zhang, Robert R. Alfano, Peter G. Sacks, and Stimson P. Schantz. "Tissue autofluorescence spectroscopy: in-vivo alterations may reflect cellular proliferation." In OE/LASE '94, edited by Robert R. Alfano. SPIE, 1994. http://dx.doi.org/10.1117/12.175991.

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Chung, Eunna, and M. N. Rylander. "Thermal Preconditioning Protocols for Cartilage Tissue Engineering." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193107.

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Successful creation of cartilaginous engineered tissues is often limited by insufficient cellular proliferation and formation of extracellular matrix. Stress conditioning protocols using heat have been shown to induce up-regulation of molecular chaperones called heat shock proteins (HSP) [1]. These proteins have been linked to enhanced cell proliferation and collagen synthesis which is critical for formation of the extracellular matrix. Therefore, identification of effective thermal stress preconditioning protocols that enhance HSP expression could substantially advance development of replacem
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Solarte, Efrain, Hernan Urrea, William Criollo, and Oscar Gutierrez. "LED illumination effects on proliferation and survival of meningioma cellular cultures." In BiOS, edited by Valery V. Tuchin, Donald D. Duncan, and Kirill V. Larin. SPIE, 2010. http://dx.doi.org/10.1117/12.843060.

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Blahna, Matthew T., Matthew R. Jones, Lee J. Quinton, and Joseph P. Mizgerd. "The Uridyl-Transferase Enzyme Zcchc11 Prevents Senescence And Promotes Cellular Proliferation." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4926.

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Shi, Caleb, Robert Chang, and Donna Leonardi. "The Effects of Mechanical Vibration on Cellular Health in Differentiated Neuroblastoma Cells." In ASME 2018 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/detc2018-86280.

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The effects of mechanical impact forces on neurological health is a critical concern, likely due to issues of traumatic brain injury (TBI) in sports and brain damage stemming from the potential of “sonic terrorism.” The quantitative analysis and evaluation of such forces on brain tissue function is very difficult. To address this issue, this research proposes a novel approach of using a cellular model subjected to mechanical vibration for analysis. Here, neuron-like differentiated neuroblastoma cells were subjected to vibration at frequencies of 20, 200, 2000, and 20000 Hz for a period of 24 h
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Reports on the topic "Cellular Proliferation"

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Eldar, Avigdor, and Donald L. Evans. Streptococcus iniae Infections in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Toward the Pathogen and Vaccine Formulation. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575286.bard.

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In Israel and in the U.S., Streptococcus iniae is responsible for considerable losses in various fish species. Poor understanding of its virulence factors and limited know-how-to of vaccine formulation and administration are the main reasons for the limited efficacy of vaccines. Our strategy was that in order to Improve control measures, both aspects should be equally addressed. Our proposal included the following objectives: (i) construction of host-pathogen interaction models; (ii) characterization of virulence factors and immunodominant antigens, with assessment of their relative importance
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Sun, Lina, Yanan Han, Hua Wang, et al. MicroRNAs as Potential Biomarkers for the Diagnosis of Inflammatory Bowel Disease: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.2.0027.

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Review question / Objective: The purpose of this systematic review was to systematically review the clinical studies regarding miRNAs as diagnostic biomarkers for inflammatory bowel disease and assess the overall diagnostic accuracy of miRNAs. Condition being studied: The symptoms of inflammatory bowel disease (IBD) are highly variable. The diagnosis of IBD must be made through medical history, physical, laboratory, radiologic, endoscopic, and histological examinations. However, these diagnostic techniques are not specific and sometimes even equivocal. Therefore, reliable biomarkers are urgent
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Kartika, Wardhani, Levina Aviva, Liam Stephens, et al. Neutral rhenium(i) tricarbonyl complexes with sulfur-donor ligands: anti-proliferative activity and cellular localization. Office of Scientific and Technical Information (OSTI), 2024. http://dx.doi.org/10.2172/2372653.

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Zhou, Ting, Roni Shapira, Peter Pauls, Nachman Paster, and Mark Pines. Biological Detoxification of the Mycotoxin Deoxynivalenol (DON) to Improve Safety of Animal Feed and Food. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7613885.bard.

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The trichothecene deoxynivalenol (DON, vomitoxin), one of the most common mycotoxin contaminants of grains, is produced by members of the Fusarium genus. DON poses a health risk to consumers and impairs livestock performance because it causes feed refusal, nausea, vomiting, diarrhea, hemolytic effects and cellular injury. The occurrence of trichothecenes contamination is global and they are very resistant to physical or chemical detoxification techniques. Trichothecenes are absorbed in the small intestine into the blood stream. The overall objective of this project was to develop a protecting
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