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Books on the topic 'Cellular RNA'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Pitre, Liisa K. The application of RNA interference to cellular biotechnology. Sudbury, Ont: Laurentian University, 2003.

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2

Brasier, Allan R., Adolfo García-Sastre, and Stanley M. Lemon, eds. Cellular Signaling and Innate Immune Responses to RNA Virus Infections. Washington, DC, USA: ASM Press, 2008. http://dx.doi.org/10.1128/9781555815561.

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3

Kekkonen, Viktoria. Characterization of bacterial RNA and DNA signalling pathways that induce cellular dysfunction. Sudbury, Ont: Laurentian University, 2006.

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4

Post-transcriptional regulation by STAR proteins : control of RNA metabolism in development and disease. New York: Springer Science+Business Media, LLC, 2010.

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5

Wingender, Edgar. Gene regulation in eukaryotes. Weinheim: VCH, 1993.

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6

Mallick, Bibekanand. Regulatory RNAs: Basics, Methods and Applications. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012.

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7

Tax, Frans. Receptor-like Kinases in Plants: From Development to Defense. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012.

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8

Farrell, Robert E. RNA methodologies: A laboratory guide for isolation and characterization. 2nd ed. San Diego: Academic Press, 1998.

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9

Divan, Aysha, and Janice A. Royds. 3. RNA. Oxford University Press, 2016. http://dx.doi.org/10.1093/actrade/9780198723882.003.0003.

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The first RNA molecules to be discovered were those involved in protein synthesis, mRNA, transfer RNA (tRNA), and ribosomal RNA (rRNA). In recent years, a vast number of additional RNA molecules have been identified. ‘RNA’ explains that these are non-coding RNAs that are not involved in protein synthesis, but influence many normal cellular and disease processes by regulating gene expression. RNA interference (RNAi) as one of the main ways in which gene expression is regulated is described with applications to therapy. Classes of RNA, including long non-coding RNAs and catalytic RNAs, are expla

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10

Grimm, Dirk. Cellular RNA Interference Mechanisms. Elsevier Science & Technology Books, 2011.

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11

Grimm, Dirk. Cellular RNA Interference Mechanisms. Elsevier Science & Technology Books, 2011.

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12

Cellular RNA Interference Mechanisms. Elsevier, 2011. http://dx.doi.org/10.1016/c2011-0-04281-3.

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13

Masquida, Benoît, and Fabrice Leclerc. Looking at Ribozymes: From Atomic to Molecular and Cellular Scales. Elsevier, 2019.

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14

Masquida, Benoît, and Fabrice Leclerc. Looking at Ribozymes: From Atomic to Molecular and Cellular Scales. Elsevier, 2019.

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15

Gunaratne, Preethi, and Matthew L. Anderson. MicroRNAs in Health and Disease. Taylor & Francis Group, 2014.

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16

R, Brasier Allan, García-Sastre Adolfo, and Lemon Stanley M, eds. Cellular signaling and innate immune responses to RNA virus infections. Washington, D.C: ASM Press, 2009.

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17

Lemon, Stanley M., Allan R. Brasier, and Adolfo Garcia-Sastre. Cellular Signaling and Innate Immune Responses to RNA Virus Infections. Wiley & Sons, Limited, John, 2014.

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18

Replication of Viral and Cellular Genomes: Molecular events at the origins of replication and biosynthesis of viral and cellular genomes. Springer, 2011.

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19

Becker, Yechiel. Replication of Viral and Cellular Genomes: Molecular Events at the Origins of Replication and Biosynthesis of Viral and Cellular Genomes. Springer London, Limited, 2012.

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20

Becker, Yechiel. Replication of Viral and Cellular Genomes: Molecular Events at the Origins of Replication and Biosynthesis of Viral and Cellular Genomes. Springer, 2011.

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21

Reckman, Yolan J., and Yigal M. Pinto. The role of non-coding RNA/microRNAs in cardiac disease. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0031.

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In the past two decades, our knowledge about non-coding DNA has increased tremendously. While non-coding DNA was initially discarded as ‘junk DNA’, we are now aware of the important and often crucial roles of RNA transcripts that do not translate into protein. Non-coding RNAs (ncRNAs) play important functions in normal cellular homeostasis and also in many diseases across all organ systems. Among the different ncRNAs, microRNAs (miRNAs) and long non-coding RNAs (lncRNAs) have been studied the most. In this chapter we discuss the role of miRNAs and lncRNAs in cardiac disease. We present example

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22

Molecular biology of RNA: Proceedings of a Director's Sponsors-UCLA Symposium, held at Keystone, Colorado, April 4-10, 1988 (UCLA symposia on molecular and cellular biology). Liss, 1989.

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23

Erickson, Robert P. Gene Regulation: Biology of Antisense Rna and DNA (Raven Press Series on Molecular and Cellular Biology). Raven Pr, 1991.

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24

Becker, Yechiel. Molecular Evolution of Viruses - Past and Present: Evolution Of Viruses By Acquisition Of Cellular Rna And Dna. Springer, 2012.

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25

Yechiel, Becker, and Darai Gholamreza, eds. Molecular evolution of viruses-past and present: Evolution of viruses by acquisition of cellular RNA and DNA. Boston: Kluwer Academic Publishers, 2000.

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26

Becker, Yechiel, and Gholamreza Darai. Molecular Evolution of Viruses -- Past and Present: Evolution of Viruses by Acquisition of Cellular RNA and DNA. Springer London, Limited, 2012.

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27

(Editor), Y. Becker, and Gholamreza Darai (Editor), eds. Molecular Evolution of Viruses - Past and Present: Evolution of Viruses by Acquisition of Cellular RNA and DNA (VIRUS GENES). Springer, 2007.

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28

Jeffrey, Wilusz, ed. Post-transcriptional gene regulation. Totowa, N.J: Humana Press, 2008.

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29

Post-Transcriptional Gene Regulation. Humana Press, 2007.

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30

Wilusz, Jeffrey. Post-Transcriptional Gene Regulation. Humana Press, 2010.

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31

Short And Long Distance Signaling. Springer, 2011.

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32

Mallick, Bibekanand, and Zhumur Ghosh. Regulatory RNAs: Basics, Methods and Applications. Springer, 2012.

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33

Mallick, Bibekanand, and Zhumur Ghosh. Regulatory RNAs: Basics, Methods and Applications. Springer, 2014.

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34

Methylation, Hoffmann-LA Roche-UCLA Colloquium on Nucleic Acid, Dawn B. Willis, Arthur Weissbach, and Gary A. Clawson. Nucleic Acid Methylation: Proceedings of a Hoffman-LA Roche-UCLA Colloquium on Nucleic Acid Methylation Held at Frisco, Colorado, March 31-April 7, (Ucla ... and Cellular Biology, New Ser., V. 128). Wiley-Liss, 1989.

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35

Vaheri, Antti, James N. Mills, Christina F. Spiropoulou, and Brian Hjelle. Hantaviruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0035.

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Hantaviruses (genus Hantavirus, family Bunyaviridae) are rodent- and insectivore-borne zoonotic viruses. Several hantaviruses are human pathogens, some with 10-35% mortality, and cause two diseases: hemorrhagic fever with renal syndrome (HFRS) in Eurasia, and hantavirus cardiopulmonary syndrome (HCPS) in the Americas. Hantaviruses are enveloped and have a three-segmented, single-stranded, negative-sense RNA genome. The L gene encodes an RNA-dependent RNA polymerase, the M gene encodes two glycoproteins (Gn and Gc), and the S gene encodes a nucleocapsid protein. In addition, the S genes of some

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36

Signal Transduction: Pathways, Mechanisms and Diseases. Springer, 2009.

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37

Hughes, Alis, and Lesley Jones. Pathogenic Mechanisms. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0013.

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Huntington’s disease (HD) pathogenesis is complex. In the two decades since the gene and its mutation were discovered, there has been extensive exploration of how the expanded CAG repeat in HTT leads to neurodegeneration in HD. This chapter focuses on the mechanisms that potentially contribute to the dysfunction and death of cells in HD. These include repeat instability and RNA toxicity and the production, processing, modification, and degradation of mutant huntingtin. The effects of mutant HTT on cellular processes such as transcription, transport, neurotransmission, and protein clearance are

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38

Kemmerling, Birgit, and Frans Tax. Receptor-like Kinases in Plants: From Development to Defense. Springer, 2014.

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39

Choi, Sangdun. Encyclopedia of Signaling Molecules. Springer, 2017.

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40

Choi, Sangdun. Encyclopedia of Signaling Molecules. Springer, 2017.

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41

Encyclopedia Of Signaling Molecules. Springer, 2012.

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42

Choi, Sangdun. Encyclopedia of Signaling Molecules. Springer, 2013.

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43

Webster, Jessica Lynn, and Marco Vignuzzi. Viral evolution and impact for public health strategies in low-income countries. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0007.

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Viruses, the simplest organisms, obligate parasites that encode structural proteins and replicative functions requiring the cellular machinery of the host to ensure their propagation. Viruses are masters of evolution. An analysis of infectious diseases emerging since the 1980s revealed that most were caused by viruses, especially those with RNA genomes. New viral emergences are generally the result of intrinsic changes in the genetics of the virus to increase transmission, virulence or host range, and environmental or ecological changes that favor contacts between viruses and humans or other h

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44

Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.

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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only

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45

Dörner, Thomas, and Peter E. Lipsky. Cellular side of acquired immunity (B cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0050.

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B cells have gained interest in rheumatoid arthritis (RA) beyond being the precursors of antibody-producing plasma cells since they are also a broader component of the adaptive immune system. They are capable of functioning as antigen-presenting cells for T-cell activation and can produce an array of cytokines. Disturbances of peripheral B-cell homeostasis together with the formation of ectopic lymphoid neogenesis within the inflamed synovium appears to be a characteristic of patients with RA. Enhanced generation of memory B cells and autoreactive plasma cells producing IgM-RF and ACPA-IgG ant

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46

Pitzalis, Costantino, Frances Humby, and Michael P. Seed. Synovial pathology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or

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47

Simpson, A., E. Aarons, and R. Hewson. Marburg and Ebola viruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0038.

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Infection with Marburg and Ebola viruses cause haemorrhagic fevers that are characterized by organ malfunction, bleeding complications, and high mortality. The viruses are members of the family Filoviridae, a group of membrane-enveloped filamentous RNA viruses. Five distinct species of the genus Ebolavirus have been reported; the genus Marburgvirus contains only one species. Both Marburg and Ebola virus diseases are zoonotic infections whose primary hosts are thought to be bats. The initial human infection is acquired from wildlife and subsequent person-to-person spread propagates the outbreak

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48

Grant, Warren, and Martin Scott-Brown. Principles of oncogenesis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0322.

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It is obvious that the process of developing cancer—oncogenesis—is a multistep process. We know that smoking, obesity, and a family history are strong independent predictors of developing malignancy; yet, in clinics, we often see that some heavy smokers live into their nineties and that some people with close relatives affected by cancer spend many years worrying about a disease that, in the end, they never contract. For many centuries scientists have struggled to understand the process that make cancer cells different from normal cells. There were those in ancient times who believed that tumo

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49

Fleischmann, Roy. Signalling pathway inhibitors. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0081.

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Oral, small-molecule signalling pathway inhibitors, including ones that inhibit the JAK and SyK pathways, are currently in development for the treatment of rheumatoid arthritis (RA). Tofacitinib is an orally administered small-molecule inhibitor that targets the intracellular Janus kinase 3 and 1 (JAK1/3) molecules to a greater extent than JAK2 while baricitinib (formerly INCB028050) predominantly inhibits JAK1/2. Many of the proinflammatory cytokines implicated in the pathogenesis of RA utilize cell signalling that involves the JAK-STAT pathways and therefore inhibition of JAK-STAT signalling

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50

Rice-Evans, C. A., A. T. Diplock, and M. C. R. Symons. Techniques in Free Radical Research (Laboratory Techniques in Biochemistry and Molecular Biology). Elsevier Science, 1991.

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