Academic literature on the topic 'Cellular signal transduction'

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Journal articles on the topic "Cellular signal transduction"

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Bae, Yun Soo, and June Seung Lee. "Cellular Signal Transduction." Journal of the Korean Medical Association 44, no. 7 (2001): 716. http://dx.doi.org/10.5124/jkma.2001.44.7.716.

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Macara, I. G. "Oncogenes and cellular signal transduction." Physiological Reviews 69, no. 3 (July 1, 1989): 797–820. http://dx.doi.org/10.1152/physrev.1989.69.3.797.

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Ball, A. "Introduction to Cellular Signal Transduction." Cell Biology International 24, no. 11 (November 2000): 855. http://dx.doi.org/10.1006/cbir.2000.0590.

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Marks, F., and P. Angel. "Signal Transduction into the Nucleus: Fifth Colloquium on Cellular Signal Transduction." Journal of Cancer Research and Clinical Oncology 122, no. 10 (October 1996): 638–42. http://dx.doi.org/10.1007/bf01221198.

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Marks, F., and G. F�rstenberger. "Fourth colloquium on cellular signal transduction. Lipid mediators: signal transduction and transport." Journal of Cancer Research and Clinical Oncology 121, no. 7 (July 1995): 434–38. http://dx.doi.org/10.1007/bf01212952.

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Wurthner, Jens U., Amal K. Mukhopadhyay, and Claus-Jürgen Peimann. "A cellular automaton model of cellular signal transduction." Computers in Biology and Medicine 30, no. 1 (January 2000): 1–21. http://dx.doi.org/10.1016/s0010-4825(99)00020-7.

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Wetzel, C. H. "Cellular Mechanisms of Olfactory Signal Transduction." Chemical Senses 30, Supplement 1 (January 1, 2005): i321—i322. http://dx.doi.org/10.1093/chemse/bjh244.

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Schmidt-Ullrich, Rupert K., Paul Dent, Steven Grant, Ross B. Mikkelsen, and Kristoffer Valerie. "Signal Transduction and Cellular Radiation Responses." Radiation Research 153, no. 3 (March 2000): 245–57. http://dx.doi.org/10.1667/0033-7587(2000)153[0245:stacrr]2.0.co;2.

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Lin, James C. A., Jimmy K. Li, and Walter H. Chang. "Signal Transduction Pathway of Ultrasound Stimulation on Osteoblasts(Cellular & Tissue Engineering)." Proceedings of the Asian Pacific Conference on Biomechanics : emerging science and technology in biomechanics 2004.1 (2004): 87–88. http://dx.doi.org/10.1299/jsmeapbio.2004.1.87.

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Mattson, Mark P. "Cerebral Signal Transduction." Journal of Molecular Neuroscience 14, no. 3 (2000): 206–8. http://dx.doi.org/10.1385/jmn:14:3:206.

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Dissertations / Theses on the topic "Cellular signal transduction"

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Pat, Betty Kila. "Signal transduction pathways in renal fibrosis /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17739.pdf.

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Haugh, Jason Michael 1972. "Cellular compartmentation effects in receptor-mediated signal transduction." Thesis, Massachusetts Institute of Technology, 1999. http://hdl.handle.net/1721.1/85364.

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Leahy, Rachel A. "Signal Transduction and Cellular Differentiation in Airway Epithelium." Kent State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=kent1352673026.

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Kim, Hyun Ji. "Development and signal transduction in Dictyostelium." Thesis, University of Oxford, 1999. http://ora.ox.ac.uk/objects/uuid:4ed80c6e-adc8-46d6-aeaf-c853cef7af77.

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Dictyostelium, is a simple eukaryote that multiplies as separate amoebae. However when nutrients are no longer available it embarks on a developmental programme in which the amoebae collect together by chemotaxis and the resulting aggregates eventually transform into fruiting bodies consisting of a cluster of spores held up on a cellular stalk. The entire process of development normally takes about 24 hours. However there are mutants, termed rapidly developing mutants (rde) which complete development in about two-thirds of this time. RdeA null mutants have been reported to have elevated levels
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Chang, Wen-Tsan. "Molecular studies of signal transduction and development." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360212.

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Brownlie, Zoe. "Regulation of signal transduction by RGS4." Connect to e-thesis, 2007. http://theses.gla.ac.uk/124/.

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Thesis (Ph.D.) - University of Glasgow, 2007.<br>Ph.D. thesis submitted to the Division of Biochemistry and Molecular Biology, Institute of Biomedical and Life Sciences, University of Glasgow, 2007. Includes bibliographical references.
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Gammon, Benjamin Matthew. "Signal transduction in the cellular slime mould Dictyostelium discoideum." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279872.

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Manne, Bhanu Kanth. "CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION." Diss., Temple University Libraries, 2015. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/340495.

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Physiology<br>Ph.D.<br>Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis. Nevertheless, the primary physiological function of platelets is to act as essential mediators in maintaining homeostasis of the circulatory system by forming hemostatic thrombi that prevent blood loss and maintain vascular integrity. CLEC-2 is a C-type lectin-like receptor that is highly expressed in platelets and lesser extent, in other cell types such as activated dendritic cells and B cells. Rhodocytin was the
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Hung, Hiu Wai. "Signal transduction mechanism in xenopus presynaptic differentiation /." View Abstract or Full-Text, 2003. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202003%20HUNG.

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Gong, Yunchen 1965. "Analyses of alternative cell signal transduction pathways." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85552.

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Living cells keep sensing the changes in their environments, mostly, via cell surface receptors for different ligands. Attachment-dependent cells are sensitive to alterations in extracellular matrix (ECM). ECM is not only required for cell survival, but also prerequisite for epidermal growth factor (EGF) to stimulate cell proliferation. The receptors for the majority of ECM components are integrins and the receptor for EGF is EGF receptor (EGFR). When bound by their ligands, integrins and EGFR induce signal transduction cascades composed of alternative pathways. A quantitative assessmen
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Books on the topic "Cellular signal transduction"

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Sitaramayya, Ari, ed. Introduction to Cellular Signal Transduction. Boston, MA: Birkhäuser Boston, 1999. http://dx.doi.org/10.1007/978-1-4612-1990-3.

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Ari, Sitaramayya, ed. Introduction to cellular signal transduction. Boston: Birkhauser, 1999.

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Dennis, Edward A. Transduction mechanisms in cellular signaling. Amsterdam: Elsevier/AP, 2011.

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T, Leeds Dorothy, ed. Focus on cellular signalling. New York: Nova Science Publishers, 2006.

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Klaus, Palme, ed. Signals and signal transduction pathways in plants. Dordrecht: Kluwer Academic Publishers, 1994.

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Yehuda, Gutman, and Lazarovici Philip, eds. Toxins and signal transduction. Australia: Harwood Academic Pub., 1997.

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F, Greco Lorenzo, and Martino Alessandro L, eds. Signal transduction: New research. New York: Nova Science Publishers, 2008.

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R, Tatham Peter E., Kramer Ijsbrand M, Knovel (Firm), and ScienceDirect (Online service), eds. Signal transduction. 2nd ed. Amsterdam: Elsevier/Academic Press, 2009.

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J, Van Eldik Linda, and Watterson D. Martin, eds. Calmodulin and signal transduction. San Diego: Academic Press, 1998.

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Krauss, Gerhard. Biochemistry of signal transduction and regulation. 2nd ed. Weinheim: Wiley-VCH, 2001.

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Book chapters on the topic "Cellular signal transduction"

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Marks, Friedrich, Ursula Klingmüller, and Karin Müller-Decker. "Signal Transduction by Ions." In Cellular Signal Processing, 485–541. Second edition. | New York, NY: Garland Science, 2017.: Garland Science, 2017. http://dx.doi.org/10.4324/9781315165479-14.

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Park, Gyungsoon, Carol A. Jones, and Katherine A. Borkovich. "Signal Transduction Pathways." In Cellular and Molecular Biology of Filamentous Fungi, 50–59. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816636.ch5.

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Lee, Joanna Y., and Lina M. Obeid. "Ceramide, Aging and Cellular Senescence." In Sphingolipid-Mediated Signal Transduction, 61–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-22425-0_5.

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Marks, Friedrich, Ursula Klingmüller, and Karin Müller-Decker. "Signal Transduction by Proteolysis, and Programmed Cell Death." In Cellular Signal Processing, 453–83. Second edition. | New York, NY: Garland Science, 2017.: Garland Science, 2017. http://dx.doi.org/10.4324/9781315165479-13.

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Marks, Friedrich, Ursula Klingmüller, and Karin Müller-Decker. "Signal Transduction by Receptors with Seven Transmembrane Domains." In Cellular Signal Processing, 191–227. Second edition. | New York, NY: Garland Science, 2017.: Garland Science, 2017. http://dx.doi.org/10.4324/9781315165479-5.

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Marks, Friedrich, Ursula Klingmüller, and Karin Müller-Decker. "Signal Transduction by Serine/Threonine Kinase-Coupled Receptors." In Cellular Signal Processing, 229–47. Second edition. | New York, NY: Garland Science, 2017.: Garland Science, 2017. http://dx.doi.org/10.4324/9781315165479-6.

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Boyd, Jonathan W., Richard R. Neubig, Alice Han, and Maren Prediger. "Introduction to Cellular Signal Transduction." In Cellular Signal Transduction in Toxicology and Pharmacology, 1–19. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2019. http://dx.doi.org/10.1002/9781119060208.ch1.

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Neubig, Richard R., Jonathan W. Boyd, Julia A. Mouch, and Nicole Prince. "Mechanisms of Cellular Signal Transduction." In Cellular Signal Transduction in Toxicology and Pharmacology, 21–48. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2019. http://dx.doi.org/10.1002/9781119060208.ch2.

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Niederfellner, Gerhard. "Signal Transduction and Cellular Communication." In Biochemical Pathways, 286–324. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118657072.ch7.

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Ganey, Patricia E., and Sean A. Misek. "Signal Transduction in Disease." In Cellular Signal Transduction in Toxicology and Pharmacology, 73–111. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2019. http://dx.doi.org/10.1002/9781119060208.ch4.

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Conference papers on the topic "Cellular signal transduction"

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Liu, Yuan-Wei, and Chun-Liang Lin. "Idea of Control Design for Cellular Signal Transduction Pathway of Ras." In International Conference on Computational Intelligence and Multimedia Applications (ICCIMA 2007). IEEE, 2007. http://dx.doi.org/10.1109/iccima.2007.267.

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Uzgare, Rajneesh, Thomas Hundley, Dhanrajan Tiruchinapalli, Anna Solomon, Cheryl Horton, and Hao Chen. "Abstract 236: Characterizing cellular signal transduction cross-talk using in-cell kinase screen." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-236.

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Mednieks, M. I. "Secretory proteins characteristic of environmental changes in cellular signal transduction: Expression in oral fluid." In HADRONS AND NUCLEI: First International Symposium. AIP, 2000. http://dx.doi.org/10.1063/1.1302484.

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Rattanakul, Chontita, and Yongwimon Lenbury. "Cellular automata simulation of signal transduction and calcium dynamics with healthy and faulty receptor trafficking." In 2016 Annual IEEE Systems Conference (SysCon). IEEE, 2016. http://dx.doi.org/10.1109/syscon.2016.7490536.

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KYODA, KOJI M., MICHIKO MURAKI, and HIROAKI KITANO. "CONSTRUCTION OF A GENERALIZED SIMULATOR FOR MULTI-CELLULAR ORGANISMS AND ITS APPLICATION TO SMAD SIGNAL TRANSDUCTION." In Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447331_0030.

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Takai, Erica, Clark T. Hung, Aurea Tucay, Djordje Djukic, Mary L. Linde, Kevin D. Costa, James T. Yardley, and X. Edward Guo. "Design of a Microfluidic System for 3D Culture of Osteocytes In Vitro." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-33229.

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Bone adapts to its mechanical environment so that its form follows function, a mechanism known as Wolff’s law, or bone adaptation. Although the basic concepts of Wolff’s law have been generally accepted, the regulatory signals and the underlying cellular and molecular pathways, which mediate this adaptive process, are unknown. Failure of normal bone adaptation plays a significant role in the etiology of metabolic bone diseases such as osteoporosis and osteopetrosis, bone loss in space flight and failure of total joint replacements. During the past three decades, there have been extensive in vi
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LeDuc, Philip. "Linking Molecular to Cellular Biomechanics With Nano- and Micro-Technology." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43987.

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The link between mechanics and biochemistry has been implicated in a myriad of scientific and medical problem, from orthopedics and cardiovascular medicine, to cell motility and division, to signal transduction and gene expression. Most of these studies have been focused on organ-level issues, yet cellular and molecular level research has become essential over the last decade in this field thanks to the revolutionary developments in genetics, molecular biology, fabrication processes, and biotechnology. Developing the link between molecular and cellular biomechanics through subcellular studies
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HATTA-OHASHI, Y., T. TAKAHASHI, and H. SUZUKI. "VISUALIZATION OF SEQUENTIAL RESPONSE IN INTRA CELLULAR SIGNAL TRANSDUCTION CASCADE BY FLUORESCENCE AND LUMINESCENCE IMAGING IN THE SAME LIVING CELL." In Proceedings of the 15th International Symposium. WORLD SCIENTIFIC, 2008. http://dx.doi.org/10.1142/9789812839589_0081.

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Yang, Jui-Ming, and Philip R. LeDuc. "Three-Dimensional Laminar Flow for Localized Cellular Stimulation." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61643.

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Stimulation of living mammalian cells is primarily accomplished by the delivery of chemical agents to single cells or cell populations. Due to the fast response time of diffusion for these agents over the small size scale of individual cells, localized stimulation is limited. Currently, there are alternate techniques that can produce localized gradients of chemical stimulants over single cells, but they lack the ability for long time scale events that are requisite for many cellular processes because of this diffusion limitation. We have developed a device that is able to create chemical agent
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Guilak, Farshid, and H. Ping Ting-Beall. "The Effects of Osmotic Pressure on the Viscoelastic and Physical Properties of Articular Chondrocytes." In ASME 1999 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/imece1999-0398.

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Abstract Mechanical stress is an important factor in the regulation of chondrocyte metabolism and the maintenance of the cartilage extracellular matrix. Previous studies have shown that compression of cartilage explants alters cellular metabolism in a time- and spatially-varying manner which is correlated with the mechanical environment within the extracellular matrix [1]. Furthermore, cellular response has been shown to be influenced by mechanical, electrical, and physicochemical events which are coupled to deformation of the cells and the extracellular matrix. Therefore, detailed information
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Reports on the topic "Cellular signal transduction"

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Merrill, Alfred H., and Jr. Subcellular Signal Transduction Systems in the Cellular Trauma of Ischemia. Fort Belvoir, VA: Defense Technical Information Center, November 1990. http://dx.doi.org/10.21236/ada229876.

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Naim, Michael, Andrew Spielman, Shlomo Nir, and Ann Noble. Bitter Taste Transduction: Cellular Pathways, Inhibition and Implications for Human Acceptance of Agricultural Food Products. United States Department of Agriculture, February 2000. http://dx.doi.org/10.32747/2000.7695839.bard.

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Historically, the aversive response of humans and other mammals to bitter-taste substances has been useful for survival, since many toxic constituents taste bitter. Today, the range of foods available is more diverse. Many bitter foods are not only safe for consumption but contain bitter constituents that provide nutritional benefits. Despite this, these foods are often eliminated from our current diets because of their unacceptable bitterness. Extensive technology has been developed to remove or mask bitterness in foods, but a lack of understanding of the mechanisms of bitterness perception a
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Philosoph-Hadas, Sonia, Peter B. Kaufman, Shimon Meir, and Abraham H. Halevy. Inhibition of the Gravitropic Shoot Bending in Stored Cut Flowers Through Control of Their Graviperception: Involvement of the Cytoskeleton and Cytosolic Calcium. United States Department of Agriculture, December 2005. http://dx.doi.org/10.32747/2005.7586533.bard.

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Original objectives: The basic goal of the present project was to study the mechanism involved in shoot graviperception and early transduction, in order to determine the sequence of events operating in this process. This will enable to control the entire process of gravity-induced differential growth without affecting vertical growth processes essential for development. Thus, several new postulated interactions, operating at the perception and early transduction stages of the signaling cascade leading to auxin-mediated bending, were proposed to be examined in snapdragon spikes and oat shoot pu
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O'Neill, Sharman, Abraham Halevy, and Amihud Borochov. Molecular Genetic Analysis of Pollination-Induced Senescence in Phalaenopsis Orchids. United States Department of Agriculture, 1991. http://dx.doi.org/10.32747/1991.7612837.bard.

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The project investigated the molecular genetic and biochemical basis of pollination-induced senescence of Phalaenopsis flowers. This experimental system offered unique advantages in that senescence is strictly regulated by pollination, providing the basis to experimentally initiate and synchronize senescence in populations of flowers. The postpollination syndrome in the Phalaenopsis orchid system was dissected by investigating the temporal and spatial regulation of ACC synthase gene expression. In the stigma, pollen-borne auxin induces the expression of the auxin-regulated ACC synthase (PS-ACS
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Philosoph-Hadas, Sonia, Peter Kaufman, Shimon Meir, and Abraham Halevy. Signal Transduction Pathway of Hormonal Action in Control and Regulation of the Gravitropic Response of Cut Flowering Stems during Storage and Transport. United States Department of Agriculture, October 1999. http://dx.doi.org/10.32747/1999.7695838.bard.

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Original objectives: The basic goal of the present project was to increase our understanding of the cellular mechanisms operating during the gravitropic response of cut flowers, for solving their bending problem without affecting flower quality. Thus, several elements operating at the 3 levels o the gravity-induced signal transduction pathway, were proposed to be examined in snapdragon stems according to the following research goals: 1) Signaling: characterize the signal transduction pathway leading to the gravitropic response, regarding the involvement of [Ca2+]cyt as a mediator of IAA moveme
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Avni, Adi, and Kirankumar S. Mysore. Functional Genomics Approach to Identify Signaling Components Involved in Defense Responses Induced by the Ethylene Inducing Xyalanase Elicitor. United States Department of Agriculture, December 2009. http://dx.doi.org/10.32747/2009.7697100.bard.

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Plant-microbe interactions involve a large number of global regulatory systems, which are essential for plants to protect themselves against pathogen attack. An ethylene-inducing xylanase (EIX) of Trichoderma viride is a potent elicitor of plant defense responses, like hypersensitive response (HR), in specific cultivars of tobacco (Nicotiana tabacum) and tomato (Lycopersicon esculentum). The central goal of this proposal was to investigate the molecular mechanisms that allow plants to specifically activate defense responses after EIX treatment. We proposed to identify cellular signaling compon
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Olszewski, Neil, and David Weiss. Role of Serine/Threonine O-GlcNAc Modifications in Signaling Networks. United States Department of Agriculture, September 2010. http://dx.doi.org/10.32747/2010.7696544.bard.

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Significant evidence suggests that serine/threonine-O-linked N-acetyl glucosamine0-(GlcNAc) modifications play a central role in the regulation of plant signaling networks. Forexample, mutations in SPINDLY,) SPY (an O-GlcNAc transferase,) OGT (promote gibberellin GA) (signal transduction and inhibit cytokinin responses. In addition, mutating both Arabidopsis OGTsSEC (and SPY) causes embryo lethality. The long-term goal of this research is to elucidate the mechanism by which Arabidopsis OGTs regulate signaling networks. This project investigated the mechanisms of O-GlcNAc regulation of cytokini
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