Relevant bibliographies by topics / Cellules HL60

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Cellules HL60'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Cellules HL60"

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Ul-Haq, R., and C. R. Chitambar. "Modulation of transferrin receptor mRNA by transferrin-gallium in human myeloid HL60 and lymphoid CCRF-CEM leukaemic cells." Biochemical Journal 294, no. 3 (1993): 873–77. http://dx.doi.org/10.1042/bj2940873.

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Gallium binds to the iron transport protein transferrin (Tf), is incorporated into cells through transferrin receptors (TfR) and inhibits iron-dependent DNA synthesis. Since cellular TfR expression is tightly regulated by the availability of iron, we investigated the effects of transferrin-gallium (Tf-Ga) on TfR mRNA levels in myeloid HL60 and lymphoid CCRF-CEM cells. In HL60 cells, Tf-Ga increased TfR mRNA levels in a dose-dependent fashion. This increase in TfR mRNA was blocked by Tf-Fe and by cycloheximide. Analysis of the rate of mRNA decay in the presence of actinomycin D revealed that th
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Rudin, Deborah, Maurice Schmutz, Noëmi Johanna Roos, Jamal Bouitbir, and Stephan Krähenbühl. "Reactive Metamizole Metabolites Enhance the Toxicity of Hemin on the ATP Pool in HL60 Cells by Inhibition of Glycolysis." Biomedicines 8, no. 7 (2020): 212. http://dx.doi.org/10.3390/biomedicines8070212.

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Metamizole is an analgesic, whose pharmacological and toxicological properties are attributed to N-methyl-aminoantipyrine (MAA), its major metabolite. In the presence of heme iron, MAA forms reactive metabolites, which are toxic for granulocyte precursors. Since decreased cellular ATP is characteristic for MAA-associated toxicity, we studied the effect of MAA with and without hemin on energy metabolism of HL60 cells, a granulocyte precursor cell line. The combination MAA/hemin depleted the cellular ATP stronger than hemin alone, whereas MAA alone was not toxic. This decrease in cellular ATP wa
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Hu, Yumin, Gang Chen, Yu Jia, et al. "The Combination of PEITC (Phenehyl Isothiocyanate) with a Histone Deacetylase Inhibitor (HDACi) Has Synergistic Antileukemia Activity by Overcoming a Redox-Mediated Resistance Pathway." Blood 114, no. 22 (2009): 1739. http://dx.doi.org/10.1182/blood.v114.22.1739.1739.

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Abstract Abstract 1739 Poster Board I-765 Introduction Histone deacetylase inhibitors (HDACI) have limited but well established clinical activity in human leukemia. Results of a phase 1 trial of vorinostat in AML indicate that a gene signature composed mainly of antioxidants was associated with clinical resistance to vorinostat (Blood 2008;111:1060-60). This study suggested that generation of reactive oxygen species (ROS) appears to be a mechanism of action of vorinostat whereas increase of antioxidants may correlate with vorinostat resistance. The aims of this study were to further investigat
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Halada, Petr, Petr Man, Dana Grebeňová, Zbyněk Hrkal, and Vladimír Havlíček. "Identification of HL60 Proteins Affected by 5-Aminolevulinic Acid-Based Photodynamic Therapy Using Mass Spectrometric Approach." Collection of Czechoslovak Chemical Communications 66, no. 11 (2001): 1720–28. http://dx.doi.org/10.1135/cccc20011720.

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A combination of mass spectrometric techniques was used for identification of HL60 leukemia cell proteins affected by 5-aminolevulinic acid-based photodynamic therapy (ALA-PDT). We compared two-dimensional electrophoresis (2-DE) protein maps of ALA-treated non-irradiated and irradiated cells and found extensive changes in the proteome of HL60 cells. The silver-stained 2-DE pattern of HL60 proteins contained more than 1 350 spots. Matrix-assisted laser desorption/ionisation mass spectrometry and microcapillary liquid chromatography/tandem mass spectrometry have identified twelve proteins differ
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Liu, Liqiong, Ping Wang, Xingbin Wang, Junxia Gu, Xiaoqing Li, and Shiang Huang. "Trans-Cinnamaldehyde Induces Granulocytic Differentiation and Impairs Survival and Migration of Acute Myeloid Leukemic Cells." Blood 112, no. 11 (2008): 5035. http://dx.doi.org/10.1182/blood.v112.11.5035.5035.

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Abstract Trans-cinnamaldehyde (TCA), the major active component of oil isolated from the stem bark of Cinnamomum cassia traditionally used to treat dyspepsia, gastritis and inflammatory disease world widely, has been shown to inhibit proliferation and promote apoptosis in a number of cancer cells. However, the functional roles TCA plays in hematopoietic system have not been fully investigated. In this study, we show the effects of TCA on acute promyelocytic leukemia (APL) cell line HL60 and primary bone marrow mononuclear cells (BMMNC) as well as bone marrow stromal cells (BMSC) from acute mye
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Amirkhosravi, Ali, Sonja Loges, Todd Meyer, et al. "An in vitro study on the mechanisms of coagulation activation in acute myelogenous leukemia (AML): role of tissue factor regulation by cytotoxic drugs and GM-CSF." Thrombosis and Haemostasis 92, no. 11 (2004): 1136–46. http://dx.doi.org/10.1160/th04-04-0215.

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SummaryAML patients may suffer from a disseminated coagulopathy, which can aggravate a pre-existing bleeding tendency due to thrombocytopenia and platelet dysfunction. The cellular and molecular mechanisms underlying this coagulopathy, however, are not completely understood. Indeed, the broad and increasing therapeutic use of cytotoxic drugs and growth factors is likely to contribute to the complexity of hemostatic abnormalities encountered in this hematologic malignancy. The nature of coagulation activation in AML was therefore investigated in vitro using the human leukemic cell line, HL60. T
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Bouvy, Céline, Adeline Wannez, Christian J. Chatelain, and Jean-Michel Dogné. "Transfer of Multidrug Resistance Among Acute Myeloid Leukemia Cells Via Extracellular Vesicles and Their Micrornas Cargo." Blood 128, no. 22 (2016): 2864. http://dx.doi.org/10.1182/blood.v128.22.2864.2864.

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Abstract Resistance of cancer cells to chemotherapy is a major issue in acute myeloid leukemia (AML) and is usually due to a clonal selection of resistant leukemic cells. Recently, a horizontal transfer of chemoresistance among tumor cells has been reported via extracellular vesicles (EVs). EVs are vesicles ranging from 0.03 to 1µm generated by almost all cell types. They carry cellular components such as nucleic acids, membrane and cytosolic proteins. By interacting with cells and by transferring their content, these vesicles could modify target cells' phenotype. Because of their presence in
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Campos, Lydia, Pascale Flandrin, Daniela Olaru, and Denis Guyotat. "Hsp90 Inhibitors Induce Apoptosis in Human Leukemia Cells." Blood 104, no. 11 (2004): 93. http://dx.doi.org/10.1182/blood.v104.11.93.93.

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Abstract The 90-Kda heat shock protein (Hsp90), together with a number of other chaperones are involved in normal cellular differentiation and cancerogenesis. Hsp90 is implicated in the conformational maturation of a large variety of protein kinases. We have shown its overexpression in a series of 116 acute myeloid leukemias (AML). Geldanamycin and its analogue 17-AAG selectively block the activities of Hsp90, but do not interact with other members of the Hsp family. The objective of the study was to test the effect of 17-AAG on cell lines (Jurkat, U-937, HL60, HL60R : resistant to daunorubici
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Noble, S. L., L. E. Wendel, M. M. Donahue, G. T. Buzzard, and A. E. Rundell. "Sparse-Grid-Based Adaptive Model Predictive Control of HL60 Cellular Differentiation." IEEE Transactions on Biomedical Engineering 59, no. 2 (2012): 456–63. http://dx.doi.org/10.1109/tbme.2011.2174361.

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Sinclair, J., D. McClain, and R. Taetle. "Effects of insulin and insulin-like growth factor I on growth of human leukemia cells in serum-free and protein-free medium." Blood 72, no. 1 (1988): 66–72. http://dx.doi.org/10.1182/blood.v72.1.66.66.

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Abstract Human myeloid leukemia cells (HL60) and malignant lymphocytes (Namalwa) were grown in protein-free, Fe-supplemented media and used to study growth responses to insulin and insulin-like growth factor 1 (IGF-I). HL60 cells previously grown in serum-free medium containing microgram quantities of insulin showed an 18-fold reduction in cumulative cell production when grown without insulin. However, the same cells showed reduced or absent growth stimulation with 1 to 100 ng/mL insulin or IGF- I for at least four days following insulin deprivation, indicating that culture conditions modified
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Dissertations / Theses on the topic "Cellules HL60"

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Félin, Murielle. "Interactions glycoproteine-lectine dans le noyau des cellules de la lignee myeloblastique leucemique humaine hl60." Paris 5, 1996. http://www.theses.fr/1996PA05S012.

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La regulation de certaines activites nucleaires par des interactions glycoproteine-lectine est supposee depuis la decouverte de ces proteines dans le noyau cellulaire. Toutefois, l'existence de telles interactions restait hypothetique. En effet, les lectines nucleaires isolees jusqu'alors se fixaient, soit au glucose, soit aux galactosides, tandis que les glycoproteines nucleaires les mieux caracterisees comportaient des residus n-acetylglucosamine. Notre travail, realise sur la lignee myeloblastique leucemique humaine hl60, a donc consiste a rechercher des lectines nucleaires et leurs ligands
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Nanfah, Woda Murielle Patricia. "Étude de la différenciation des HL60 en cellules de type ostéoclastes et rôle des facteurs rhumatoïdes sur la résorption osseuse des ostéoclastes." Master's thesis, Université Laval, 2013. http://hdl.handle.net/20.500.11794/24255.

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La polyarthrite rhumatoïde est une maladie auto-immune qui touche environ 1% de la population mondiale. Elle entraîne une inflammation synoviale et une destruction de l’architecture articulaire. L’articulation rhumatoïde est un milieu très inflammatoire qui est infiltré par des cellules telles que les neutrophiles, les macrophages, les monocytes, les lymphocytes B et T ; par des protéines telles que des cytokines, des chimiokines, des molécules d’adhésion, par des complexes immuns, et par des auto-anticorps comme les facteurs rhumatoïdes qui sont un marqueur spécifique de la maladie et qui per
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Jegham, Hajer. "Étude de l'activité biologique et du mécanisme d'action de dérivés stéroïdiens à potentiel anticancéreux." Thesis, Université Laval, 2009. http://www.theses.ulaval.ca/2009/26956/26956.pdf.

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Nanfah, Woda Murielle Patricia. "Etude de la différenciation des HL60 en cellules de type ostéoclastes et rôle des facteurs rhumatoïdes sur la résorption osseuse des ostéoclastes." Thesis, Université Laval, 2013. http://www.theses.ulaval.ca/2013/29880/29880.pdf.

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La polyarthrite rhumatoïde est une maladie auto-immune qui touche environ 1% de la population mondiale. Elle entraîne une inflammation synoviale et une destruction de l’architecture articulaire. L’articulation rhumatoïde est un milieu très inflammatoire qui est infiltré par des cellules telles que les neutrophiles, les macrophages, les monocytes, les lymphocytes B et T ; par des protéines telles que des cytokines, des chimiokines, des molécules d’adhésion, par des complexes immuns, et par des auto-anticorps comme les facteurs rhumatoïdes qui sont un marqueur spécifique de la maladie et qui per
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5

Thibeault, Dominic. "Synthèse chimique et évaluation biologique d'une nouvelle famille de 2[bêta]-amino-5[alpha]-androstane -3[alpha], 17[bêta]-diols comme agents antileucémiques potentiels /." 2003. http://proquest.umi.com/pqdweb?did=766801401&sid=18&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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Book chapters on the topic "Cellules HL60"

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Horiguchi-Yamada, Junko, and Hisashi Yamada. "Differing Responses of G2-Related Genes During Differentiation of HL60 Cells Induced by TPA or DMSO." In Cellular Function and Metabolism. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-3078-7_5.

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