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1

O’Rourke, Michael F. "Central aortic pressure." Journal of Hypertension 33, no. 1 (January 2015): 187–88. http://dx.doi.org/10.1097/hjh.0000000000000450.

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Narayan, Om, Anthony Dart, Ian T. Meredith, and James D. Cameron. "Central aortic pressure." Journal of Hypertension 33, no. 1 (January 2015): 188–89. http://dx.doi.org/10.1097/hjh.0000000000000451.

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3

Carlsen, Rasmus Kirkeskov, Simon Winther, Christian D. Peters, Esben Laugesen, Dinah S. Khatir, Hans E. Bøtker, Morten Bøttcher, Per Ivarsen, My Svensson, and Niels Henrik Buus. "Aortic Calcification Affects Noninvasive Estimates of Central Blood Pressure in Patients with Severe Chronic Kidney Disease." Kidney and Blood Pressure Research 44, no. 4 (2019): 704–14. http://dx.doi.org/10.1159/000501226.

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Background: Central blood pressure (BP) assessed noninvasively considerably underestimates true invasively measured aortic BP in chronic kidney disease (CKD) patients. The difference between the estimated and the true aortic BP increases with decreasing estimated glomerular filtration rates (eGFR). The present study investigated whether aortic calcification affects noninvasive estimates of central BP. Methods: Twenty-four patients with CKD stage 4–5 undergoing coronary angiography and an aortic computed tomography scan were included (63% males, age [mean ± SD ] 53 ± 11 years, and eGFR 9 ± 5 mL
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Buchnieva, Olha Volodymyrivna. "PROTECTION OF CENTRAL AND PERIPHERAL ORGANS IN AORTIC SURGERY." International Medical Journal, no. 3 (2020): 19–23. http://dx.doi.org/10.37436/2308-5274-2019-3-3.

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The introduction into clinical practice of hypothermic circulatory arrest, both in the non−perfusion version and with an artificial circulation, was the beginning of active use of systemic hypothermia as an effective element of cerebral and visceral protection during combined cardiac surgeries, including in aorta pathology. To evaluate ways of protecting visceral organs and spinal cord, namely the "no perfusion" technique with drainage of cerebrospinal fluid, lateral aortic compression, left−atrial−femoral bypass, deep hypothermia with cardiac arrest, i.e. hypothermic circulatory arest, bypass
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5

Adji, Audrey, and Michael F. O’Rourke. "Central aortic pressure calibration." Journal of Hypertension 35, no. 4 (April 2017): 893–94. http://dx.doi.org/10.1097/hjh.0000000000001246.

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6

Middeke, Martin. "Zentraler aortaler Blutdruck: Bedeutender Parameter für Diagnostik und Therapie." DMW - Deutsche Medizinische Wochenschrift 142, no. 19 (September 2017): 1430–36. http://dx.doi.org/10.1055/s-0043-113212.

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AbstractIn recent years great emphasis has been placed on the role of central aortic blood pressure as measured non invasively using pulse wave analysis in pathophysiology of cardiovascular diseases and clinical aspects of hypertension. The difference of blood pressure between the central aorta and the brachial artery (amplification) is not constant but varies according to physiological, pathological and pharmacological mechanisms. Central aortic blood pressure is more strongly related to cardiovascular organ damages than does brachial pressure. Several antihypertensive drugs have different ef
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O’Rourke, Michael F., and Kenji Takazawa. "Measurement of central aortic pressure." Journal of Hypertension 29, no. 10 (October 2011): 2038–39. http://dx.doi.org/10.1097/hjh.0b013e32834b17b4.

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8

Boutouyrie, Pierre. "Measurement of central aortic pressure." Journal of Hypertension 29, no. 10 (October 2011): 2040–41. http://dx.doi.org/10.1097/hjh.0b013e32834b17c7.

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9

Segers, Patrick, Jan G. Kips, Sebastian J. Vermeersch, and Luc M. Van Bortel. "Measurement of central aortic pressure." Journal of Hypertension 29, no. 10 (October 2011): 2039–40. http://dx.doi.org/10.1097/hjh.0b013e32834b22ff.

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10

Bulpitt, Christopher J., C. Rajkumar, and James D. Cameron. "Central aortic blood pressure measurements." Journal of Human Hypertension 14, no. 8 (August 2000): 531. http://dx.doi.org/10.1038/sj.jhh.1001066.

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11

V., Shantaram. "Importance of Central Aortic Pressure." Indian Journal of Cardiovascular Disease in Women WINCARS 02, no. 04 (December 2017): 065–66. http://dx.doi.org/10.1055/s-0038-1622967.

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12

Rajab, TK, JD Schmitto, and RP Gallegos. "Technique for central aortic cannulation in extensive aortic dissection." Annals of The Royal College of Surgeons of England 94, no. 6 (September 1, 2012): 439. http://dx.doi.org/10.1308/003588412x13373405386015c.

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13

J. Moon, S.-H. Lee, Y.-g. Ko, Y. Jang, W.-H. Shim, and D.-H. Choi. "Central aortic pressure in aortic aneurysm and aortic dissection: a novel prognostic marker." Acta Cardiologica 65, no. 3 (June 30, 2010): 303–8. http://dx.doi.org/10.2143/ac.65.3.2050346.

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14

Zhu, Yuanjia, Samir Kapadia, Amar Krishnaswamy, Lars G. Svensson, and Stephanie Mick. "Reoperative transapical transcatheter aortic valve replacement for central aortic regurgitation." Journal of Cardiac Surgery 31, no. 9 (July 12, 2016): 572–74. http://dx.doi.org/10.1111/jocs.12798.

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15

Forneris, Arianna, Miriam Nightingale, Alina Ismaguilova, Taisiya Sigaeva, Louise Neave, Amy Bromley, Randy D. Moore, and Elena S. Di Martino. "Heterogeneity of Ex Vivo and In Vivo Properties along the Length of the Abdominal Aortic Aneurysm." Applied Sciences 11, no. 8 (April 13, 2021): 3485. http://dx.doi.org/10.3390/app11083485.

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The current clinical guidelines for the management of aortic abdominal aneurysms (AAAs) overlook the structural and mechanical heterogeneity of the aortic tissue and its role in the regional weakening that drives disease progression. This study is a comprehensive investigation of the structural and biomechanical heterogeneity of AAA tissue along the length and circumference of the aorta, by means of regional ex vivo and in vivo properties. Biaxial testing and histological analysis were performed on ex vivo human aortic specimens systematically collected during open repair surgery. Wall-shear s
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16

Williams, Bryan, and Peter S. Lacy. "Central aortic pressure and clinical outcomes." Journal of Hypertension 27, no. 6 (June 2009): 1123–25. http://dx.doi.org/10.1097/hjh.0b013e32832b6566.

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17

Meijin, Zhang, Qing Liu, Zhuo You, and Jinxiu Lin. "A3170 Affecting central aortic systolic pressure." Journal of Hypertension 36 (October 2018): e143. http://dx.doi.org/10.1097/01.hjh.0000548579.06724.dd.

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18

O’Rourke, Michael F., Michel E. Safar, and Audrey Adji. "Resistant hypertension and central aortic pressure." Journal of Hypertension 32, no. 3 (March 2014): 699. http://dx.doi.org/10.1097/hjh.0000000000000088.

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19

Narayan, Om, Joshua Casan, Martin Szarski, Anthony M. Dart, Ian T. Meredith, and James D. Cameron. "Estimation of central aortic blood pressure." Journal of Hypertension 32, no. 9 (September 2014): 1727–40. http://dx.doi.org/10.1097/hjh.0000000000000249.

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20

O’Rourke, Michael F., and Audrey Adji. "Noninvasive Studies of Central Aortic Pressure." Current Hypertension Reports 14, no. 1 (November 15, 2011): 8–20. http://dx.doi.org/10.1007/s11906-011-0236-5.

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21

Clarenbach, Christian F., Giovanni Camen, Noriane A. Sievi, Christophe Wyss, John R. Stradling, and Malcolm Kohler. "Effect of simulated obstructive hypopnea and apnea on thoracic aortic wall transmural pressures." Journal of Applied Physiology 115, no. 5 (September 1, 2013): 613–17. http://dx.doi.org/10.1152/japplphysiol.00439.2013.

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Preliminary evidence supports an association between obstructive sleep apnea (OSA) and thoracic aortic dilatation, although potential causative mechanisms are incompletely understood; these may include an increase in aortic wall transmural pressures, induced by obstructive apneas and hypopneas. In patients undergoing cardiac catheterization, mean blood pressure (MBP) in the thoracic aorta and esophageal pressure was simultaneously recorded by an indwelling aortic pigtail catheter and a balloon-tipped esophageal catheter in randomized order during: normal breathing, simulated obstructive hypopn
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22

Williams, Bryan, Ewan McFarlane, Dawid Jedrzejewski, and Peter S. Lacy. "Identifying and treating high blood pressure in men under 55 years with grade 1 hypertension: the TREAT CASP study and RCT." Efficacy and Mechanism Evaluation 6, no. 13 (December 2019): 1–90. http://dx.doi.org/10.3310/eme06130.

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Background There is uncertainty regarding whether or not younger (i.e. aged < 55 years), low-risk patients with grade 1 hypertension (i.e. a clinic blood pressure of 140–159/90–99 mmHg) should be treated with blood pressure-lowering medication. This is a heterogeneous group of patients because of variation in systolic/pulse pressure amplification from the central aorta to the brachial artery. It is hypothesised that within grade 1 hypertension, patients can be divided into those with high central aortic systolic pressure and those with low central aortic systolic pressure. Objectives The ai
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23

Lantelme, Pierre, Anastase Dzudie, Hugues Milon, Giampiero Bricca, Liliana Legedz, Jean-Michel Chevalier, and Patrick Feugier. "Effect of abdominal aortic grafts on aortic stiffness and central hemodynamics." Journal of Hypertension 27, no. 6 (June 2009): 1268–76. http://dx.doi.org/10.1097/hjh.0b013e3283299b22.

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24

Hays, Brandon S., Michael Baker, Annie Laib, Wei Tan, Sebastian Udholm, Bryan H. Goldstein, Stephen P. Sanders, Alexander R. Opotowsky, and Gruschen R. Veldtman. "Histopathological abnormalities in the central arteries and veins of Fontan subjects." Heart 104, no. 4 (September 29, 2017): 324–31. http://dx.doi.org/10.1136/heartjnl-2017-311838.

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ObjectiveFontan circulations have obligatory venous hypertension, depressed cardiac output and abnormal arterial elastance. Ventriculovascular coupling is known to be abnormal, but the underlying mechanisms are poorly defined. We aim to describe the histopathological features of vascular remodelling encountered in the central arteries and veins in the Fontan circulation as a possible underlying pathological representation of abnormal ventriculovascular coupling.MethodsPostmortemvasculature (inferior vena cava (IVC), superior vena cava (SVC), pulmonary artery (PA), pulmonary vein (PV) and aorta
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25

Kuznetsov, A. A., E. E. Tsvetkova, D. V. Denisova, Yu I. Ragino, and M. I. Voevoda. "Central Aortic Pressure: Reference and Diagnostic Values." Kardiologiia 59, no. 3 (April 13, 2019): 11–17. http://dx.doi.org/10.18087/cardio.2019.3.10235.

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Objective. Practical application of central aortic pressure (CAP) parameters is limited by the absence of generally recognized reference and threshold diagnostic indices. The purpose of this work is to establish their values in the general population of Novosibirsk. Materials and Methods. A total of 327 people were examined: 155 men and 172 women aged 25–44 years from a representative sample from the general population of Novosibirsk. Applanation tonometry of the radial artery was performed by the SphygmoCor system. The reference values of CAP parameters were obtained by a nonparametric method
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26

Inoue, Yoshito, and Toshihiko Ueda. "Central cannulation in acute aortic dissection repair." Journal of Thoracic and Cardiovascular Surgery 134, no. 2 (August 2007): 545. http://dx.doi.org/10.1016/j.jtcvs.2007.02.047.

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27

O’Rourke, Michael F., Kenji Takazawa, and Nobuhiro Tanaka. "Validity of noninvasive central aortic pressure measurement." Journal of Hypertension 37, no. 11 (November 2019): 2300–2301. http://dx.doi.org/10.1097/hjh.0000000000002211.

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28

Avolio, Alberto. "Central Aortic Blood Pressure and Cardiovascular Risk." Hypertension 51, no. 6 (June 2008): 1470–71. http://dx.doi.org/10.1161/hypertensionaha.107.108910.

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29

Tsarenok, Svetlana Yu, Vladimir V. Gorbunov, and Tatiana A. Aksenova. "The central aortic blood pressure and arterial stiffness during the daily monitoring procedure in postmenopausal women with osteoporosis." Osteoporosis and Bone Diseases 20, no. 2 (December 15, 2017): 46–51. http://dx.doi.org/10.14341/osteo2017246-51.

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Aim: to evaluate the data of the central aortic pressure and arterial stiffness during the daily monitoring procedure in postmenopausal women with osteoporosis.
 Methods: 79 postmenopausal women (age from 57 to 78) were examined. All patients were divided into two groups: the first group consisted of 36 women with osteoporosis, the second group consisted of 43 women – control group. A daily monitoring of central aortic pressure and arterial stiffness were performed all women by the apparatus BPLab v.3.2.
 Results: increase of the mean daily of systolic, diastolic and mean aortic pres
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30

Skibitskiy, V. V., A. A. Kiselev, and A. V. Fendrikova. "Effectiveness of Chrono-Pharmacotherapy Depending on the Salt Sensitivity of Patients with Arterial Hypertension and Diabetes Mellitus Type 2." Rational Pharmacotherapy in Cardiology 14, no. 6 (January 5, 2019): 846–51. http://dx.doi.org/10.20996/1819-6446-2018-14-6-846-851.

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Aim. To study the effect of two regimens of combined antihypertensive therapy during the day on daily monitoring of arterial pressure, central aortic pressure, and arterial stiffness, depending on the salt sensitivity of hypertensive patients with diabetes mellitus type 2. Material and methods. 130 hypertensive patients with type 2 diabetes mellitus were included into the study. They were divided into 2 subgroups: salt-sensitive (group 1) and salt-resistant (group 2), and then randomized to subgroups A and B of ongoing therapy: in the morning ramipril and indapamide retard, bedtime – amlodipin
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31

Kawahito, Koji, Naoyuki Kimura, Atsushi Yamaguchi, and Kei Aizawa. "Malperfusion in type A aortic dissection: results of emergency central aortic repair." General Thoracic and Cardiovascular Surgery 67, no. 7 (February 7, 2019): 594–601. http://dx.doi.org/10.1007/s11748-019-01072-z.

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32

Matsukawa, Kanji, Kei Ishii, Akito Kadowaki, Nan Liang, and Tomoko Ishida. "Differential effect of central command on aortic and carotid sinus baroreceptor-heart rate reflexes at the onset of spontaneous, fictive motor activity." American Journal of Physiology-Heart and Circulatory Physiology 303, no. 4 (August 15, 2012): H464—H474. http://dx.doi.org/10.1152/ajpheart.01133.2011.

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Our laboratory has reported that central command blunts the sensitivity of the aortic baroreceptor-heart rate (HR) reflex at the onset of voluntary static exercise in conscious cats and spontaneous contraction in decerebrate cats. The purpose of this study was to examine whether central command attenuates the sensitivity of the carotid sinus baroreceptor-HR reflex at the onset of spontaneous, fictive motor activity in paralyzed, decerebrate cats. We confirmed that aortic nerve (AN)-stimulation-induced bradycardia was markedly blunted to 26 ± 4.4% of the control (21 ± 1.3 beats/min) at the onse
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33

Westerhof, Berend E., Ilja Guelen, Wim J. Stok, Han A. J. Lasance, Carl A. P. L. Ascoop, Karel H. Wesseling, Nico Westerhof, Willem Jan W. Bos, Nikos Stergiopulos, and Jos A. E. Spaan. "Individualization of transfer function in estimation of central aortic pressure from the peripheral pulse is not required in patients at rest." Journal of Applied Physiology 105, no. 6 (December 2008): 1858–63. http://dx.doi.org/10.1152/japplphysiol.91052.2008.

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Central aortic pressure gives better insight into ventriculo-arterial coupling and better prognosis of cardiovascular complications than peripheral pressures. Therefore transfer functions (TF), reconstructing aortic pressure from peripheral pressures, are of great interest. Generalized TFs (GTF) give useful results, especially in larger study populations, but detailed information on aortic pressure might be improved by individualization of the TF. We found earlier that the time delay, representing the travel time of the pressure wave between measurement site and aorta is the main determinant o
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34

Kaleda, Vasily I., Alexander P. Nissen, Anatoly V. Molochkov, Ivan A. Alekseev, Sergey Yu Boldyrev, and Tom C. Nguyen. "Simple Technique for Central Venous Cannulation with Cannula-Free Wound in Minimally Invasive Aortic Valve Surgery." Innovations: Technology and Techniques in Cardiothoracic and Vascular Surgery 15, no. 4 (May 22, 2020): 369–71. http://dx.doi.org/10.1177/1556984520925549.

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There are several approaches to venous cannulation in minimally invasive aortic valve surgery. Frequently used options include central dual-stage right atrial cannulation, or peripheral femoral venous cannulation. During minimally invasive aortic surgery via an upper hemisternotomy, central venous cannulas may obstruct the surgeon’s visualization of the aortic valve and root, or require extension of the skin incision, while femoral venous cannulation requires an additional incision, time and resources. Here we describe a technique for central venous cannulation during minimally invasive aortic
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35

Geltser, B. I., T. A. Brodskaya, V. A. Nevzorova, and E. V. Motkina. "Evaluation of the central arterial pressure in patients with bronchial asthma." PULMONOLOGIYA, no. 3 (June 28, 2008): 15–19. http://dx.doi.org/10.18093/0869-0189-2008-0-3-15-19.

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The purpose of this work was to study the central arterial pressure (CAP) and its correlation with peripheral arterial pressure (PAP) and aortic stiffness in patients with bronchial asthma (BА). We examined 45 asthmatic patients and 25 healthy volunteers using noninvasive arteriography (TensioClinic TL1 arteriograph, TensioMed, Hungary). Aortic stiffness parameters and aortic systolic arterial pressure (SAP) were measured. We estimated a difference between the central SAP and the peripheral SAP (ΔSAP) and calculated index of CAP to PAP conformity (IC). According to the indirect arteriography,
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36

Armstrong, Matthew K., Martin G. Schultz, Dean S. Picone, J. Andrew Black, Nathan Dwyer, Philip Roberts-Thomson, and James E. Sharman. "Associations of Reservoir-Excess Pressure Parameters Derived From Central and Peripheral Arteries With Kidney Function." American Journal of Hypertension 33, no. 4 (February 1, 2020): 325–30. http://dx.doi.org/10.1093/ajh/hpaa013.

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Abstract BACKGROUND Central artery reservoir-excess pressure parameters are clinically important but impractical to record directly. However, diastolic waveform morphology is consistent across central and peripheral arteries. Therefore, peripheral artery reservoir-excess pressure parameters related to diastolic waveform morphology may be representative of central parameters and share clinically important associations with end-organ damage. This has never been determined and was the aim of this study. METHODS Intra-arterial blood pressure (BP) waveforms were measured sequentially at the aorta,
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37

Gravlee, G. P., S. D. Brauer, M. F. O'Rourke, and A. P. Avolio. "A Comparison of Brachial, Femoral, and Aortic Intra-Arterial Pressures before and after Cardiopulmonary Bypass." Anaesthesia and Intensive Care 17, no. 3 (August 1989): 305–11. http://dx.doi.org/10.1177/0310057x8901700311.

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Following recent evidence that brachial and femoral artery pressures are more reliable than radial artery pressures after cardiopulmonary bypass, thirty-one adults had simultaneous pre and post-bypass measurements of brachial, femoral, and ascending aortic pressures. Two minutes after cardiopulmonary bypass, brachial artery systolic pressure and mean arterial pressure fell significantly below corresponding pressures in the femoral artery and aorta. Five minutes after cardiopulmonary bypass, only brachial artery systolic pressure was still less than femoral and aortic systolic pressures. By ten
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38

Matsukawa, Kanji, Kei Ishii, Akito Kadowaki, Tomoko Ishida, Mitsuhiro Idesako, and Nan Liang. "Signal transduction of aortic and carotid sinus baroreceptors is not modified by central command during spontaneous motor activity in decerebrate cats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 306, no. 10 (May 15, 2014): R735—R746. http://dx.doi.org/10.1152/ajpregu.00538.2013.

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Our laboratory has suggested that central command provides selective inhibition of the cardiomotor component of aortic baroreflex at the start of exercise, preserving carotid sinus baroreflex. It is postulated that central command may modify the signal transduction of aortic baroreceptors, so as to decrease aortic baroreceptor input to the cardiovascular centers, and, thereby, can cause the selective inhibition of aortic baroreflex. To test the hypothesis, we directly analyzed the responses in multifiber aortic nerve activity (AoNA) and carotid sinus nerve activity (CsNA) during spontaneous mo
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39

Bas, Ahmet, Deniz Goksedef, Sedat G. Kandemirli, Fatih Gulsen, and Furuzan Numan. "Central venous catheter insertion into the false lumen of a complicated aortic dissection." Scottish Medical Journal 62, no. 3 (June 20, 2017): 115–18. http://dx.doi.org/10.1177/0036933017715962.

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Thoracic endovascular repair is considered the first-line treatment in complicated acute type B dissection. Central venous catheters provide valuable vascular access during endovascular treatments. However, central venous catheters are not without complications. Herein, we report a case of central venous catheter insertion into the false lumen of a complicated acute type B aortic dissection by direct aortic puncture. The tip of the central venous catheter was in the false lumen. The central venous catheter was left in place initially and was removed after graft stent deployment. This case illu
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40

Edwards, David G., Amie L. Gauthier, Melissa A. Hayman, Jesse T. Lang, and Robert W. Kenefick. "Acute effects of cold exposure on central aortic wave reflection." Journal of Applied Physiology 100, no. 4 (April 2006): 1210–14. http://dx.doi.org/10.1152/japplphysiol.01154.2005.

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The purpose of this study was to determine the effects of acute cold exposure on the timing and amplitude of central aortic wave reflection and central pressure. We hypothesized that cold exposure would result in an early return of reflected pressure waves from the periphery and an increase in central aortic systolic pressure as a result of cold-induced vasoconstriction. Twelve apparently healthy men (age 27.8 ± 2.0 yr) were studied at random, in either temperate (24°C) or cold (4°C) conditions. Measurements of brachial artery blood pressure and the synthesis of a central aortic pressure wavef
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41

Michail, Michael, Alun D. Hughes, Andrea Comella, James N. Cameron, Robert P. Gooley, Liam M. McCormick, Anthony Mathur, Kim H. Parker, Adam J. Brown, and James D. Cameron. "Acute Effects of Transcatheter Aortic Valve Replacement on Central Aortic Hemodynamics in Patients With Severe Aortic Stenosis." Hypertension 75, no. 6 (June 2020): 1557–64. http://dx.doi.org/10.1161/hypertensionaha.119.14385.

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42

Boczar, Kevin E., Munir Boodhwani, Luc Beauchesne, Carole Dennie, Kwan Leung Chan, George A. Wells, and Thais Coutinho. "Aortic Stiffness, Central Blood Pressure, and Pulsatile Arterial Load Predict Future Thoracic Aortic Aneurysm Expansion." Hypertension 77, no. 1 (January 2021): 126–34. http://dx.doi.org/10.1161/hypertensionaha.120.16249.

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Thoracic aortic aneurysm is a disease associated with high morbidity and mortality. Clinically useful strategies for medical management of thoracic aortic aneurysm are critically needed. To address this need, we sought to determine the role of aortic stiffness and pulsatile arterial load on future aneurysm expansion. One hundred five consecutive, unoperated subjects with thoracic aortic aneurysm were recruited and prospectively followed. By combining arterial tonometry with echocardiography, we estimated measures of aortic stiffness, central blood pressure, steady, and pulsatile arterial load
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43

Bethencourt, Daniel M., Jennifer Le, Gabriela Rodriguez, Robert W. Kalayjian, and Gregory S. Thomas. "Minimally Invasive Aortic Valve Replacement via Right Anterior Minithoracotomy and Central Aortic Cannulation." Innovations: Technology and Techniques in Cardiothoracic and Vascular Surgery 12, no. 2 (March 2017): 87–94. http://dx.doi.org/10.1177/155698451701200203.

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44

Bethencourt, Daniel M., Jennifer Le, Gabriela Rodriguez, Robert W. Kalayjian, and Gregory S. Thomas. "Minimally Invasive Aortic Valve Replacement via Right Anterior Minithoracotomy and Central Aortic Cannulation." Innovations: Technology and Techniques in Cardiothoracic and Vascular Surgery 12, no. 2 (March 2017): 87–94. http://dx.doi.org/10.1097/imi.0000000000000358.

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Objective This study reports the evolution of a minimally invasive aortic valve replacement (mini-AVR) technique that uses a right anterior minithoracotomy approach with central cannulation, for a 13-year period. This technique has become our standard approach for isolated primary AVR in nearly all patients. Methods This observational study evaluated perioperative clinical outcomes of patients 18 years or older who underwent mini-AVR from November 2003 to June 2015. Results The mini-AVR technique was used in 202 patients during two periods of 2003 to 2009 (n = 65, “early”) and 2010 to 2015 (n
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45

Ruegg, Gion, Rebecca H. Mason, Maxine Hardinge, Jeremy Perkins, Marc Husmann, Erich W. Russi, Konrad E. Bloch, John R. Stradling, and Malcolm Kohler. "Augmentation index and central aortic blood pressure in patients with abdominal aortic aneurysms." Journal of Hypertension 28, no. 11 (November 2010): 2252–57. http://dx.doi.org/10.1097/hjh.0b013e32833e1187.

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46

Almousa, Ayman, and Ravi K. Ghanta. "Commentary: Straight into the heart of danger: Central aortic cannulation for aortic dissection." Journal of Thoracic and Cardiovascular Surgery 158, no. 1 (July 2019): 37–38. http://dx.doi.org/10.1016/j.jtcvs.2018.12.052.

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Rajani, Ronak, Phil Chowienczyk, Simon Redwood, Antoine Guilcher, and John B. Chambers. "The noninvasive estimation of central aortic blood pressure in patients with aortic stenosis." Journal of Hypertension 26, no. 12 (December 2008): 2381–88. http://dx.doi.org/10.1097/hjh.0b013e328313919f.

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Bulas, J., M. Potocarova, V. Kupcova, L. Gaspar, G. Wimmer, and J. Murin. "Central systolic blood pressure increases with aortic stiffness." Bratislava Medical Journal 120, no. 12 (2019): 894–98. http://dx.doi.org/10.4149/bll_2019_150.

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Ramasamy, S., J. M. Ravichandran, and Pradeep G. Nayar. "Central Aortic Blood Pressure: An Evidence-based Approach." Hypertension Journal 4, no. 4 (2018): 219–24. http://dx.doi.org/10.15713/ins.johtn.0138.

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Chen, Gailing, Kevin P. Bliden, Rahul Chaudhary, Fang Liu, Himabindu Kaza, Eliano P. Navarese, Udaya S. Tantry, and Paul A. Gurbel. "Central aortic pulse pressure, thrombogenicity and cardiovascular risk." Journal of Thrombosis and Thrombolysis 44, no. 2 (July 10, 2017): 223–33. http://dx.doi.org/10.1007/s11239-017-1524-y.

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