Relevant bibliographies by topics / Central-memory cells

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Central-memory cells'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Central-memory cells"

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Chiu, Bo-Chin, Brian E. Martin, Valerie R. Stolberg, and Stephen W. Chensue. "Cutting Edge: Central Memory CD8 T Cells in Aged Mice Are Virtual Memory Cells." Journal of Immunology 191, no. 12 (2013): 5793–96. http://dx.doi.org/10.4049/jimmunol.1302509.

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Huster, Katharina M, Martina Koffler, Christian Stemberger, Matthias Schiemann, Hermann Wagner, and Dirk H Busch. "Unidirectional development of CD8+ central memory T cells into protectiveListeria-specific effector memory T cells." European Journal of Immunology 36, no. 6 (2006): 1453–64. http://dx.doi.org/10.1002/eji.200635874.

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Li Causi, Eleonora, Suraj C. Parikh, Lindsey Chudley, et al. "Vaccination Expands Antigen-Specific CD4+ Memory T Cells and Mobilizes Bystander Central Memory T Cells." PLOS ONE 10, no. 9 (2015): e0136717. http://dx.doi.org/10.1371/journal.pone.0136717.

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Matos, T., A. Gehad, J. Teague, et al. "071 Human central memory T cells generate superior numbers of resident memory T cells in skin." Journal of Investigative Dermatology 138, no. 5 (2018): S12. http://dx.doi.org/10.1016/j.jid.2018.03.075.

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Maus, Marcela V., Birgit Kovacs, William W. Kwok, et al. "Extensive Replicative Capacity of Human Central Memory T Cells." Journal of Immunology 172, no. 11 (2004): 6675–83. http://dx.doi.org/10.4049/jimmunol.172.11.6675.

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Yi, Zuoan, Laura L. Stunz, Wai Wai Lin, and Gail A. Bishop. "TRAF3 Regulates Homeostasis of CD8+ Central Memory T Cells." PLoS ONE 9, no. 7 (2014): e102120. http://dx.doi.org/10.1371/journal.pone.0102120.

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Pepper, Marion, and Marc K. Jenkins. "Origins of CD4+ effector and central memory T cells." Nature Immunology 12, no. 6 (2011): 467–71. http://dx.doi.org/10.1038/ni.2038.

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Intlekofer, Andrew M., Naofumi Takemoto, Charlly Kao, et al. "Requirement for T-bet in the aberrant differentiation of unhelped memory CD8+ T cells." Journal of Experimental Medicine 204, no. 9 (2007): 2015–21. http://dx.doi.org/10.1084/jem.20070841.

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Immunity to intracellular pathogens requires dynamic balance between terminal differentiation of short-lived, cytotoxic effector CD8+ T cells and self-renewal of central–memory CD8+ T cells. We now show that T-bet represses transcription of IL-7Rα and drives differentiation of effector and effector–memory CD8+ T cells at the expense of central–memory cells. We also found T-bet to be overexpressed in CD8+ T cells that differentiated in the absence of CD4+ T cell help, a condition that is associated with defective central–memory formation. Finally, deletion of T-bet corrected the abnormal phenot
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Wan, Ni, Hehua Dai, Tao Wang, Yolonda Moore, Xin Xiao Zheng, and Zhenhua Dai. "Bystander Central Memory but Not Effector Memory CD8+ T Cells Suppress Allograft Rejection." Journal of Immunology 180, no. 1 (2007): 113–21. http://dx.doi.org/10.4049/jimmunol.180.1.113.

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Blander, J. Magarian, Derek B. Sant’Angelo, Daniela Metz, et al. "A Pool of Central Memory-Like CD4 T Cells Contains Effector Memory Precursors." Journal of Immunology 170, no. 6 (2003): 2940–48. http://dx.doi.org/10.4049/jimmunol.170.6.2940.

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Dissertations / Theses on the topic "Central-memory cells"

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Purushe, Janaki. "MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells." Diss., Temple University Libraries, 2018. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/489872.

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Infectious Disease & Immunity<br>Ph.D.<br>CAR-T cell immunotherapy is a highly efficacious treatment for CD19-positive hematological malignancies, however, some patients are non-responsive for reasons that are not well understood. Clinical efficacy has been correlated with long-term persistence, a propensity that can be predicted by the differentiation state of transplanted cells. Despite this, decades-old methods for expanding T cells have not been updated to prevent the deleterious effects of excessive differentiation in CAR-T cells. Uncoupling proliferation and differentiation is a long-hel
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Dobrowolski, Curtis Noel. "HISTONE LYSINE METHYLTRANSFERASES SELECTIVELY RESTRICT HIV-1 IN CENTRAL MEMORY T-CELLS." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1522842870401743.

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Gräf, Patricia [Verfasser]. "Serial transfer of single cell-derived immunocompetence reveals stemness of CD8+ central memory T cells / Patricia Gräf." München : Verlag Dr. Hut, 2015. http://d-nb.info/1070124389/34.

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Fukunaga, Akiko. "Altered Homeostasis of CD4+ Memory T cells in Allogeneic Hematopoietic Stem Cell Transplant Recipients: Chronic Graft-versus-Host Disease Enhances T cell Differentiation and Exhausts Central Memory T Cell Pool." Kyoto University, 2008. http://hdl.handle.net/2433/124214.

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Tanaka, Junya. "Human TSLP and TLR3 ligands promote differentiation of Th17 cells with a central memory phenotype under Th2-polarizing conditions." Kyoto University, 2010. http://hdl.handle.net/2433/120552.

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Bet, Jeannette [Verfasser], Dirk [Akademischer Betreuer] Busch, and Michael [Akademischer Betreuer] Groll. "Clinical multi-parameter purification of human central memory T cells for adoptive therapy / Jeannette Bet. Betreuer: Dirk Busch. Gutachter: Michael Groll ; Dirk Busch." München : Universitätsbibliothek der TU München, 2015. http://d-nb.info/1081216786/34.

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Lei, Hong. "Human natural regulatory T cells subsets." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2014. http://dx.doi.org/10.18452/16958.

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Regulatorische T-Zellen (Treg) eröffnen neue immuntherapeutische Wege zur Kontrolle unerwünschter Immunreaktionen, jedoch wirft die Heterogenität dieser Zellen die Frage auf, welche Treg-Population für die klinische Anwendung. Darauf basierend werden in dieser Arbeit drei Fragestellungen bearbeitet: i) Bestimmung der Häufigkeit von Tregs und deren Subpopulationen in verschiedenen Altersgruppen bei Empfängern einer Organtransplantation (Tx) und einer gesunden Kontrollgruppe; ii) Vergleich der Suppressorkapazität verschiedener Treg-Populationen und in vitro-Expansion der Zellen unter Erhaltung i
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Girard, Stephane. "Amnésie et thérapie cellulaire : Etude de l'écotropisme des cellules souches adultes de la lamina propria olfactive." Thesis, Aix-Marseille, 2012. http://www.theses.fr/2012AIXM4759.

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Les faibles capacités régénératives intrinsèques du système nerveux central, après la survenue de lésions traumatiques ou l'apparition de maladies neuro-dégénératives, ont orienté les recherches vers des thérapies basées sur l'utilisation de cellules souches dans le but de régénérer le tissu cérébral. Cependant, des limitations éthiques et techniques associées aux cellules souches embryonnaires, fœtales ou neurales chez l'adulte restreignent leur utilisation en clinique humaine. À la recherche d'une source alternative, nous nous sommes intéressés à des cellules souches adultes peu connues, pro
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Müller-Spahn, Christina [Verfasser], Stefan [Akademischer Betreuer] Burdach, Günther [Akademischer Betreuer] Richter, and Angela [Akademischer Betreuer] Krackhardt. "Interleukin-21 mediated generation of allorestricted central memory cytotoxic T cells directed against Ewing tumour-specific antigens / Christina Müller-Spahn. Gutachter: Stefan Burdach ; Angela Krackhardt. Betreuer: Stefan Burdach ; Günther Richter." München : Universitätsbibliothek der TU München, 2014. http://d-nb.info/1059872943/34.

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Hall, Charles. "Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence." VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/3176.

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Metastatic breast cancer treatment has seen few advances in recent years, yet treatment resistance continues to rise, causing disease recurrence. A pilot study was performed to determine the efficacy of ex vivo expansion and reprogramming of tumor-reactive immune cells from experimental metastatic tumor-sensitized mice. Also, phenotypic changes in tumors due to metastasis or tumor microenvironment influences were characterized. Metastatic neu+ mouse mammary carcinoma (mMMC) and its distant relapsing neu-antigen-negative variant (mANV) were investigated in FVBN202 mice. Tumor-reactive central m
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Books on the topic "Central-memory cells"

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Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.

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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only
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(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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Book chapters on the topic "Central-memory cells"

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Murray, Shannon, Rémi Fromentin, and Nicolas Chomont. "Central Memory CD4 T Cells." In Encyclopedia of AIDS. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9610-6_177-1.

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Murray, Shannon, Rémi Fromentin, and Nicolas Chomont. "Central Memory CD4 T Cells." In Encyclopedia of AIDS. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7101-5_177.

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Rajput, Rashi, Ramneek Kaur, Rishika Chadha, et al. "The Aging Brain." In Advances in Medical Diagnosis, Treatment, and Care. IGI Global, 2019. http://dx.doi.org/10.4018/978-1-5225-5282-6.ch001.

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Neurodegeneration is the progressive and gradual dysfunction and loss of axons in the central nervous system. It is the main pathological characteristic of chronic and acute neurodegenerative conditions like Alzheimer's disease (AD), Parkinson's disease (PD), and multiple sclerosis (MS). The usual aspects of pathogenesis of disease can be abridged with regards to the downstream implications of uncontrollable protein oligomerization and aggregation from postmitotic cells. The brain structure constantly changes in normal aging without any dysfunction accompanying the structural changes in brain. The decline in cognitive capabilities, for example, processing speed, memory, and functions related to decision making are the sign of healthy aging. The reduction in brain volume in healthy aging is possibly related to neuronal loss at some marginal extent. The following chapter discusses the structural and functional alterations in the brain in ageing and neurodegeneration.
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Ahlskog, J. Eric. "Symptoms, Related Brain Regions, and Diagnosis." In Dementia with Lewy Body and Parkinson's Disease Patients. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780199977567.003.0008.

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As a prelude to the treatment chapters that follow, we need to define and describe the types of problems and symptoms encountered in DLB and PDD. The clinical picture can be quite varied: problems encountered by one person may be quite different from those encountered by another person, and symptoms that are problematic in one individual may be minimal in another. In these disorders, the Lewy neurodegenerative process potentially affects certain nervous system regions but spares others. Affected areas include thinking and memory circuits, as well as movement (motor) function and the autonomic nervous system, which regulates primary functions such as bladder, bowel, and blood pressure control. Many other brain regions, by contrast, are spared or minimally involved, such as vision and sensation. The brain and spinal cord constitute the central nervous system. The interface between the brain and spinal cord is by way of the brain stem, as shown in Figure 4.1. Thought, memory, and reasoning are primarily organized in the thick layers of cortex overlying lower brain levels. Volitional movements, such as writing, throwing, or kicking, also emanate from the cortex and integrate with circuits just below, including those in the basal ganglia, shown in Figure 4.2. The basal ganglia includes the striatum, globus pallidus, subthalamic nucleus, and substantia nigra, as illustrated in Figure 4.2. Movement information is integrated and modulated in these basal ganglia nuclei and then transmitted down the brain stem to the spinal cord. At spinal cord levels the correct sequence of muscle activation that has been programmed is accomplished. Activated nerves from appropriate regions of the spinal cord relay the signals to the proper muscles. Sensory information from the periphery (limbs) travels in the opposite direction. How are these signals transmitted? Brain cells called neurons have long, wire-like extensions that interface with other neurons, effectively making up circuits that are slightly similar to computer circuits; this is illustrated in Figure 4.3. At the end of these wire-like extensions are tiny enlargements (terminals) that contain specific biological chemicals called neurotransmitters. Neurotransmitters are released when the electrical signal travels down that neuron to the end of that wire-like process.
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Dewing, Jan. "Understanding Dementia." In Adult Nursing Practice. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199697410.003.0017.

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This chapter presents a comprehensive understanding of dementia as a commonly encountered condition/syndrome in the nursing care of older adults and offers insights into the health challenges faced by people living with dementia. It will provide nurses with the knowledge to be able to assess, manage, and care for people with dementia in an evidence-based and person-centred way. After a comprehensive overview of the causes, risk factors, and impact of dementia, it will outline best practice to deliver care, as well as to prevent or minimize further ill-health. Nursing assessments and priorities are highlighted throughout, and the nursing management of the symptoms and common health problems associated with dementia can be found in Chapters 14 and 17, respectively. In the past, dementia was most often described in terms of mental disability. However, it is now more often described in terms of neurological disability (i.e. changes in the brain). For example, the Mental Health Foundation describes dementia as:…A decline in mental ability which affects memory, thinking, problem-solving, concentration and perception….The NHS Choices website states:…Dementia describes the effects of certain conditions and diseases on a person’s mental ability, personality and behaviour….Dementia is generally classified according to two international classification systems: the American Psychiatric Association Diagnostic and Statistical Manual of Mental Disorders fourth edition (DSM-IV); and the International Classification of Diseases tenth edition (ICD-10). Dementia can be defined as a syndrome whereby there is gradual death of brain cells, resulting in a loss of brain ability that is severe enough to interfere with normal activities of living for more than 6 months. Problems with brain function should not have been present at birth and it is not associated with a loss or alteration of consciousness. This latter point distinguishes dementia from delirium, which is a state of mental disorientation that can happen if you become medically unwell, also known as an ‘acute confusional state’ (Royal College of Psychiatrists, 2009). (See Chapter 11). It is vital that nurses hold central what dementia means for people living with it. For example, people will commonly experience changes to their perception, senses, memory, and the range of skills they need to carry out everyday activities.
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Sohrabji, Farida, Shameena Bake, and Amutha Selvamani. "Estrogenic Regulation of Neuroprotection and Inflammation in Ischemic Stroke and Aging." In Estrogens and Memory. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190645908.003.0025.

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Stroke is the fifth leading cause of mortality and the major cause of long-term disability in the United States. Epidemiological studies report sex differences in ischemic stroke occurrence, mortality and functional recovery. In younger demographics, the overall incidence of stroke is higher in men than younger women, but in the elderly population, stroke rates are higher in older women compared to age-matched men, indicating an interaction of age and sex as important modifiers of disease. The increased risk for stroke in older women is attributed to loss of ovarian hormones, principally estrogens. However, estrogen/estradiol therapy is not always neuroprotective for stroke, especially in aging populations. Age-related changes in central and peripheral immune cells and the blood–brain barrier may play a crucial role in modifying stroke outcomes and the effects of estrogens. This chapter discusses the role of estrogens as a stroke protectant in younger females in contrast to its anomalous effects in the aging brain. Furthermore, the chapter describes age-related changes in support cells in the brain and in the periphery and evaluates the evidence that age-associated inflammation underlies the switch in estrogens neuroprotective action in young females to its neurotoxic effects in older females.
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"Neuroanatomy and neurophysiology." In Oxford Handbook of Medical Sciences, edited by Robert Wilkins, Ian Megson, and David Meredith. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198789895.003.0011.

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‘Neuroanatomy and neurophysiology’ covers the anatomy and organization of the central nervous system, including the skull and cervical vertebrae, the meninges, the blood and lymphatic vessels, muscles and nerves of the head and neck, and the structures of the eye, ear, and central nervous system. At a cellular level, the different cell types and the mechanism of transmission across synapses are considered, including excitatory and inhibitory synapses. This is followed by a review of the major control and sensory systems (including movement, information processing, locomotion, reflexes, and the main five senses of sight, hearing, touch, taste, and smell). The integration of these processes into higher functions (such as sleep, consciousness and coma, emotion, memory, and ageing) is discussed, along with the causes and treatments of disorders of diseases such as depression, schizophrenia, epilepsy, addiction, and degenerative diseases.
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Davies, Jamie A. "5. Reacting and thinking." In Human Physiology: A Very Short Introduction. Oxford University Press, 2021. http://dx.doi.org/10.1093/actrade/9780198869887.003.0005.

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This chapter assesses the nervous system. In the trunk of the body and the neck, the central nervous system (CNS) is called the spinal cord; in the head, it is called the brain. The CNS is dominated by two cell types: neurons and glia. The neurons form a vast network in which information is split, combined, and somehow processed. Examples of this processing include reflex arcs, the ‘circuitry’ that detects features such as edges in images coming from the eyes, and simple types of learning and memory. However, most other things in the brain, especially thinking and feeling, are not yet understood at all well.
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D. Udovin, Lucas, Andrea Aguilar, Tamara Kobiec, et al. "Neuroprotective Properties of Cannabinoids in Cellular and Animal Models: Hypotheses and Facts." In Neuroprotection - New Approaches and Prospects. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.90761.

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Progressive neuronal loss is a typical characteristic of neurodegenerative diseases. In Parkinson’s disease, the loss of dopaminergic neurons in the basal ganglia results in impaired mobility and flawed muscle control. The loss of cholinergic neurons largely in the basal forebrain contributes to memory and attention deficits and the overall cognitive impairment in Alzheimer’s disease. This being said, neuroprotective drugs should be expected to preserve and/or restore the functions affected by neuronal loss, and substantially prevent cell death. The endocannabinoid system, comprising lipid mediators able to bind to and activate cannabinoid receptors, has emerged as a therapeutic target of potential interest in a variety of central nervous system diseases. Palmitoylethanolamide (PEA) is one of the most important endocannabinoids, which has a key role in modulating oxidative stress and inflammatory response with neuroprotective potential in neurological disorders. Neurodegenerative diseases undergo varied, progressive stages. The current therapeutical approaches are beginning to fall short when it comes to meet the expected results, urging to either develop or identify or develop new effective treatments. This chapter discusses the neuroprotective potential of new drugs, aiming to shed some light on their proposed mechanism of action and their effect in cellular and animal models of neurodegeneration.
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P. James, Steven, and Dena Bondugji. "Gamma-Aminobutyric Acid (GABA) and the Endocannabinoids: Understanding the Risks and Opportunities." In Gamma-Aminobutyric Acid - Neuropsychiatric and Therapeutic Implications [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99242.

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The Gamma-aminobutyric acid (GABA) system is the main inhibitory neurotransmitter system in the central nervous system (CNS) of vertebrates and is involved in critical cellular communication and brain function. The endocannabioid system (ECS) was only recenty discovered and quickly recognized to be abundantly expressed in GABA-rich areas of the brain. The strong relationship between the GABA system and ECS is supported both by studies of the neuraoanatomy of mammalian nervous systems and the chemical messaging between neurons. The ECS is currently known to consist of two endocannabinoids, Anandamide (AEA) and 2-Arachidonyl Glycerol (2-AG), that function as chemical messengers between neurons, at least two cannabinoid receptors (CB1 and CB2), and complex synthetic and degradative metabolic systems. The ECS differs from the GABA system and other neurotransmitter systems in multiple ways including retrograde communication from the activated post-synaptic neuron to the presynaptic cell. Together, this molecular conversation between the ECS and GABA systems regulate the homeostasis and the chemical messaging essential for higher cortical functions such as learning and memory and may play a role in several human pathologies. Phytocannabinoids are synthesized in the plant Cannabis sativa (C. sativa). Within the family of phytocannabinoids at least 100 different cannabinoid molecules or derivatives have been identified and share the properties of binding to the endogenous cannabinoid receptors CB1 and CB2. The well-known psychoactive phytocannabinoid Δ9-tetrahydrocannabinol (THC) and the non-psychoactive cannabidiol (CBD) are just two of the many substances synthesized within C. sativa that act on the body. Although the phytocannabinoids THC and CBD bind to these endogenous receptors in the mammalian CNS, these plant derived molecules have little in common with the endocannabinoids in structure, distribution and metabolism. This overlap in receptor binding is likely coincidental since phytocannabinoids evolved within the plant kingdom and the ECS including the endocannabinoids developed within animals. The GABA and ECS networks communicate through carefully orchestrated activities at localized synaptic level. When phytocannabinoids become available, the receptor affinities for CB1 and CB2 may compete with the naturally occurring endocannabinoid ligands and influence the GABA-ECS communication. In some instances this addition of phytocannabinoids may provide some therapeutic benefit while in other circumstances the presence of these plant derived ligands for the CB1 and CB2 receptors binding site may lead to disruption of important functions within the CNS. The regulatory approval of several THC products for nausea and vomiting and anorexia and CBD for rare pediatric seizure disorders are examples of some of the benefits of phytocannabinoids. Concerns regarding cannabis exposure in utero and in the child and adolescence are shrill warnings of the hazards associated with disrupting the normal maturation of the developing CNS.
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Conference papers on the topic "Central-memory cells"

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Trella, Emanuele, Evangelos Panoupolos, Swantje Heidtmann, Nermin Raafat, Giulio Cesare Spagnoli, and Paul Zajac. "Abstract 2883: Improved generation of central memory CD8+ T cells with CD40L expressing recombinant vaccinia virus." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2883.

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Xu, Jie, Yaroslav Kaminskiy, and Jan Joseph Melenhorst. "Abstract 1507: Chimeric antigen receptor T cells carrying IDH1 neomorph increase CD8 positive central memory proportion without interfering with cell growth." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1507.

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Hamilton, EP, AC Hobeika, HK Lyerly, et al. "P1-13-03: Zoledronic Acid Induces an Immune Response in Breast Cancer Patients through Stimulation of Central Memory and Effector Memory gamma/delta T-Cells." In Abstracts: Thirty-Fourth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 6‐10, 2011; San Antonio, TX. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/0008-5472.sabcs11-p1-13-03.

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Sodre, Andressa L., David M. Woods, Amod Sarnaik, Brian C. Betts, and Jeffrey S. Weber. "Abstract B109: Epigenetic reprogramming of T-cells from metastatic melanoma patients enhances central memory and decreases Th2/Treg phenotypes." In Abstracts: Second CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 25-28, 2016; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6066.imm2016-b109.

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Li, Kang, Lei Shi, Qing Wang, Oscar Onyema, Yizhan Guo, and Alexander Sasha Krupnick. "Abstract A47: Superior expansion of central memory CD8+ T cells using NKG2D-targeted delivery of IL-2: Implications for adoptive T cell immunotherapy." In Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; October 1-4, 2017; Boston, MA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/2326-6074.tumimm17-a47.

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Wang, Dongrui, Renate Starr, Brenda Aguilar, et al. "Abstract 3024: CD4+outperform CD8+central memory-derived CAR T cells, mediating persistent antitumor responses and long-term eradication of glioblastoma." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3024.

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Hanson, Amadna, Abha Daneshwar, Heather Cohen, et al. "Abstract 5536: ICOS hi CD4 T cells emerging on vopratelimab treatment have Th1 central memory characteristics and may contribute to durability of clinical responses." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-5536.

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Kim, Sojung, Lauren Suarez, Emily Lu, et al. "Abstract 1423: AIM ACT, a novel nanoparticle-based technology that generates therapeutic numbers of functional tumor-specific CD8+ T cells with T stem cell, central and effector memory phenotype in 14 days." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1423.

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Kim, Sojung, Lauren Suarez, Emily Lu, et al. "Abstract 1423: AIM ACT, a novel nanoparticle-based technology that generates therapeutic numbers of functional tumor-specific CD8+ T cells with T stem cell, central and effector memory phenotype in 14 days." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1423.

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Bae, Jooeun, Rao Prabhala, Ruben Carrasco, et al. "Abstract 638: Lenalidomide treatment enhances the anti-tumor activities of XBP1 specific cytotoxic T lymphocytes by increasing the frequency and tumor-specific response of central memory CD3+CD8+ T cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-638.

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