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Dissertations / Theses on the topic 'CEP104'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Rocha, Jaqueline Maria Matias da. "The role of CEP164 in ciliogenesis in Drosophila melanogaster." Master's thesis, Universidade de Aveiro, 2013. http://hdl.handle.net/10773/12494.

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Mestrado em Biotecnologia - Biotecnologia Industrial e Ambiental<br>Cilia/flagella are microtubule (MT)-based oraganelle emanating from the surface of many eukaryotic cells. They are involved in a variety of processes including cell motility, fluid flow, and sensing processes. The skeleton of cilium, called axoneme, is templated from the basal body, a modified mature centriole required for centrosome formation. Mutations in human genes disrupting the function or structure of these organelles cause several human disorders, including infertility, ciliopathies, and cancer. In Drosophila melanoga
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2

Lamla, Stefan. "Functional characterisation of the centrosomal protein Cep170." Diss., lmu, 2009. http://nbn-resolving.de/urn:nbn:de:bvb:19-97838.

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3

Anderhub, Simon [Verfasser]. "Charakterisierung der Proteine CEP164 und ppdpf sowie deren Einordnung in den mitotischen Kontext / Simon Anderhub." Mainz : Universitätsbibliothek Mainz, 2013. http://d-nb.info/1033734136/34.

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4

Chamling, Xitiz. "Identification and characterization of CEP131 as a novel BBSome interacting protein." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/4588.

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Bardet-Biedl syndrome (BBS) is a pleiotropic and genetically heterogeneous disorder, and a well-known ciliopathy. Nineteen different genes have been reported for BBS, mutations in which cause characteristic phenotypes including retinal degeneration, obesity, polydactyly, renal abnormalities, hypogenitalism and cognitive impairment. Protein products of eleven BBS genes are part of two major complexes: the BBSome complex and a CCT/CTRiC/BBS complex. The CCT/CTRiC/BBS complex assists in the formation of the BBSome complex, which in turn traffics numerous receptor proteins to the cilia. However, t
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5

Zanini, Marco. "Ciliogenesis Control Mechanisms in Cerebellar Neuron Progenitors." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS475/document.

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Pendant le développement du cervelet, les progéniteurs des neurones granulaires (PNG) nécessitent la présence du cil primaire pour proliférer en réponse à Sonic Hedgehog (SHH). En effet, la prolifération dérégulée des PNGs peut conduire à la formation d'une tumeur pédiatrique maligne appelée SHH-médulloblastome (MB), de ce fait comprendre comment le cil primaire est régulé dans les PNGs est crucial.Nous montrons que le facteur de transcription Atoh1 contrôle la présence du cil primaire dans les PNGs in vitro et in vivo. En particulier, la suppression du cil primaire par l’inactivation génétiqu
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6

Sir, Joo-Hee. "Characterisation of the autosomal recessive primary microcephaly complex, CEP63-CEP152 in the vertebrate centrosome." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608038.

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7

Schausten, Charlotte Verfasser], Rainer [Akademischer Betreuer] [Telle, and Georg [Akademischer Betreuer] Roth. "Untersuchungen zu Sinterverhalten und Gefügeentwicklung der Monazitkeramiken LaPO4, CePO4 und PrPO4 für die Endlagerung von Minoren Actinoiden / Charlotte Schausten ; Rainer Telle, Georg Roth." Aachen : Universitätsbibliothek der RWTH Aachen, 2020. http://d-nb.info/1227447086/34.

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8

Schausten, Charlotte [Verfasser], Rainer [Akademischer Betreuer] Telle, and Georg [Akademischer Betreuer] Roth. "Untersuchungen zu Sinterverhalten und Gefügeentwicklung der Monazitkeramiken LaPO4, CePO4 und PrPO4 für die Endlagerung von Minoren Actinoiden / Charlotte Schausten ; Rainer Telle, Georg Roth." Aachen : Universitätsbibliothek der RWTH Aachen, 2020. http://d-nb.info/1227447086/34.

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9

Huang, Han-Chiang, and 黃漢強. "Overexpressed Cep170 localizes to acentrosomal puncta and promotes neurite outgrowth." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/92897006679658144092.

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碩士<br>國立交通大學<br>生物科技學系<br>104<br>Microtubule is the major cytoskeleton in neuron, and regulates various crucial neuron developmental processes. To understand how microtubule cytoskeleton influences neuronal development, we have previously utilized quantitative proteomics to examine microtubule-associated proteomes in mouse embryonal carcinoma cells (P19 cells) before and after neuronal differentiation. One of the proteins on microtubules exhibiting substantial and significant increase upon neuronal differentiation is centrosomal protein 170 (Cep170). This suggests that it plays a role in regul
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10

Srour, Myriam. "Genetic Landscape of Joubert syndrome in French Canadians." Thèse, 2015. http://hdl.handle.net/1866/13027.

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Le syndrome de Joubert est une maladie récessive caractérisée par une malformation congénitale distincte du tronc cérébral et du cervelet, associée à une anomalie des mouvements oculaires (apraxie oculomotrice), une respiration irrégulière, un retard de développement, et une ataxie à la démarche. Au cours de la dernière décennie, plus de 20 gènes responsables ont été identifiés, tous ayant un rôle important dans la structure et la fonction des cils primaires. Ainsi, le syndrome de Joubert est considéré une ciliopathie. Bien que le Syndrome de Joubert ait été décrit pour la première fois dans
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11

Lamla, Stefan [Verfasser]. "Functional characterisation of the centrosomal protein Cep170 / vorgelegt von Stefan Lamla." 2008. http://d-nb.info/993276067/34.

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12

Chao, Nian-Hsin, and 趙年欣. "Functions of a novel lissencephaly gene CEP170 in neuronal migration during brain development." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/xd3hmq.

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碩士<br>國立陽明大學<br>腦科學研究所<br>107<br>Lissencephaly (smooth brain) is a human brain developmental disorder characterized by the lack of normal folding of the brain. Here we identified a nonsense mutation in the CEP170 gene, which results in the truncated of its product (p.R1259X), in a lissencephaly patient. CEP170 is localized on the subdistal appendage of the mother centriole and the centriole proximal end. In addition, CEP170 has also been reported to regulate the microtubule organization. However, the roles of CEP170 in brain development and how its p.R1259X mutation caused lissencephaly are st
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13

Goh, Angela, and 吳佳佳. "Cep170 localizes as non-centrosomal puncta along the neurite and promotes neurite elongation." Thesis, 2019. http://ndltd.ncl.edu.tw/handle/95n99n.

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碩士<br>國立交通大學<br>生物科技學系<br>108<br>Microtubule is the major cytoskeleton in neurons and involved in numerous developmental processes. In a previous study, quantitative proteomics has been used to examine microtubule-associated proteomes in mouse embryonal carcinoma cells before and after neuronal differentiation. The centrosome protein Cep170, which made up part of the mitotic centrosome, has been found to exhibit a significant increase on microtubules upon neuronal differentiation. Overexpressing Cep170 caused a significant increase in neurite length in both P19 cell-derived neurons and mouse h
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14

Wang, Chun-Hung, and 王竣弘. "The roles of Cep70 in ciliogenesis during postnatal cerebellar development." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/58kgh8.

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碩士<br>國立陽明大學<br>腦科學研究所<br>106<br>Abstract Background: During cerebellar development, granule neuron progenitors (GNPs) proliferate in the external granule layer (EGL) to expand the number of neurons. Abnormal GNP proliferation can give rise to medulloblastoma (MB), the most common malignant brain tumor in children. Interestingly, both GNP and MB cells proliferation requires primary cilia, short microtubule-based structures protruding from the cell membrane. Ablation of primary cilia in GNPs prevented MB formation. However, it is still not fully understood how primary cilia are regulated in GN
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15

Javadi, Esfehani Yalda. "CEP78, a novel centrosomal protein." Thèse, 2014. http://hdl.handle.net/1866/11029.

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Contexte: Le centrosome est un petit organite bien connu pour son rôle dans l'établissement du fuseau bipolaire pendant la division cellulaire. Les déficiences de la fonction du centrosome donnent souvent lieu à des maladies humaines, y compris le cancer et la formation de kystes rénaux. Nous sommes intéressés à étudier la fonction d'une nouvelle protéine centrosomale nommée CEP78, identifiée dans un criblage protéomique pour de nouveaux composants centrosomaux. Méthodes et résultats : Le traitement des cellules avec le nocodazole, un agent qui dépolymérise spécifiquement les
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