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Books on the topic 'Cerebellar nuclei'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

1929-, Cohen Bernard, Tomko David L, and Guedry Fred E, eds. Sensing and controlling motion: Vestibular and sensorimotor function. New York, N.Y: New York Academy of Sciences, 1992.

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2

Fenili, Daniela. Same-cell, high-resolution tracking of nuclear and cytoplasmic fluorescent signals in live, transfected cerebellar neurons. Ottawa: National Library of Canada, 2003.

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3

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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4

DeFelipe, Javier. Cerebellum and Deep Cerebellar Nuclei. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190842833.003.0006.

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5

Safiulina, Dzahmilja. The studies of mitochondria in cultured cerebellar granule neurons: Characterization of mitochondrial function, volume homeostasis and interaction with neurosteroids. 2006.

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6

Mason, Peggy. Cerebellum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0024.

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The cerebellum uses sensory feedback and information about intended actions to ensure coordinated and smooth movements despite changing conditions. An analogy between the cerebellum and an orchestral conductor is elaborated. The cerebellum’s involvement in forming and executing motor memories is presented. Cerebellar circuits through the cerebellar cortex and deep nuclei and the dependence of cerebellar learning on climbing fiber input to Purkinje cells are briefly described. Sensory reafference and motor efference copy are defined and their roles in coordinating movement introduced. Cerebellar symptoms including ataxia, dysmetria and dysdiadochokinesia, are discussed and a possible model for dysmetria is considered. The specific inputs to and outputs from the vermis, paravermis, and lateral lobes are detailed in a description of canonical cerebellar loops. Finally, evidence that the cerebellum is involved in modulating nonmotor functions such as language, affect, social cognition, and visceral control is presented for the reader’s consideration.

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7

Gould, B. Brown. Organization of Afferents from the Brain Stem Nuclei to the Cerebellar Cortex in the Cat. Springer London, Limited, 2012.

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8

Mason, Peggy. The Versatile Brainstem. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0006.

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The role of the brainstem in life is detailed from both medical and legal points of view. This chapter describes how the brainstem divides up the fundamental processes of human life, with the most automatic and basic ones supported most caudally and progressively more expressive functions depending on more rostral brainstem regions. The text then steps through the internal anatomy of the brainstem with a focus on cranial nerve nuclei. The location of the three long tracts is followed for the length of the brainstem, and the course of the corticobulbar tract is presented. A primer on the anatomy of the cerebellum is capped by introducing ataxia, the classic symptom stemming from ipsilateral cerebellar damage.

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9

Cohen, Bernard, and David L. Tomko. Sensing and Controlling Motion: Vestibular and Sensorimotor Function (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1992.

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10

Cohen, Bernard, and David L. Tomko. Sensing and Controlling Motion: Vestibular and Sensorimotor Function (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1992.

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11

Trocello, Jean-Marc, and France Woimant. Disorders of Copper and Iron Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0044.

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Both copper and iron are essential metals that have a critical function in a series of biochemical pathways. This chapter describes the disorders associated with genetic abnormalities in copper and iron metabolic pathways and their manifestations in adult patients. Mutations in the genes of the copper transporting P-type ATPases, ATP7A and ATP7B are associated with Wilson disease, Menkes disease, occipital horn syndrome and ATP7A-related distal motor neuropathy. Neurodegeneration with brain iron accumulation (NBIA) is a group of disorders characterized by excess iron deposition in globus pallidus, substantia nigra pars reticulata, striata and cerebellar dentate nuclei. Several genes associated with NBIA have been identified.

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12

Fisch, Adam. Cerebellum. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199845712.003.0208.

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Chapter 15 discusses the cerebellum, including lobes, zones, and modules, somatotopy and lobules, peduncles, midline structures, arteries, nuclei and circuitry, and the corticopontocerebellar pathway.

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13

Chan-Palay, V. Cerebellar Dentate Nucleus: Organization, Cytology and Transmitters. Springer London, Limited, 2013.

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14

Chan-Palay, V. Cerebellar Dentate Nucleus: Organization, Cytology and Transmitters. Springer, 2014.

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15

Chan-Palay, V. Cerebellar Dentate Nucleus: Organization, Cytology and Transmitters. Springer, 2014.

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16

Montgomery, Erwin B. Approach to DBS in the Vicinity of the Ventral Intermediate Nucleus of the Thalamus DBS. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190259600.003.0013.

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The regional anatomy around the DBS lead in the ventral intermediate nucleus of the thalamus (Vim) determines efficacy and adverse effects. Understanding the regional anatomy allows the programmer to adjust the stimulation to provide optimal benefit and the absence of adverse effects. Vim is the target of therapeutic DBS. The ventrocaudal nucleus of the thalamus (Vc) lies posterior to the Vim. Electrical stimulation of Vc can cause treatment-limiting paresthesias. The corticospinal and cortical bulbar tracts in the internal capsule lie lateral and ventral to the Vim. Electrical stimulation of the internal capsule can cause tonic muscle contractions. There are multiple nomenclatures of the subnuclei of the thalamus. Although the term ventrolateral thalamus (VL) is commonly used in the physiology literature, ventral intermediate thalamus (Vim), is used in the DBS literature. Technically, the VL refers to both regions of the thalamus that receive inputs from GPi and cerebellum, whereas Vim refers to the cerebellar-receiving area of the thalamus and is thus a subdivision of the VL and is the target of DBS for tremor-related disorders.

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17

Nuclear topology of murine, cerebellar purkinje neurons: Changes as a function of postnatal development. Ottawa: National Library of Canada, 2000.

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18

Averhage, Karl. Quantitative Histochemische Bestimmung von Enzymaktivitäten in Nucleus Vetibularis Medialis und Pedunculus Cerebellaris Inferior der Erwachsenen Albinoratte zu Verschiedenen Tageszeiten. 1987.

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19

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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