Relevant bibliographies by topics / Cerebral aqueduct

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Academic literature on the topic 'Cerebral aqueduct'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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Journal articles on the topic "Cerebral aqueduct"

1

Longatti, Pierluigi, Alessandro Fiorindi, Alessandro Perin, and Andrea Martinuzzi. "Endoscopic Anatomy of the Cerebral Aqueduct." Operative Neurosurgery 61, suppl_3 (September 1, 2007): ONS—1—ONS—6. http://dx.doi.org/10.1227/01.neu.0000289705.64931.0c.

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Abstract Objective: What is known about the cerebral aqueduct is derived mainly from the legacy of classic histology and from the most recent advanced neuroimaging technologies. In fact, although this important structure is frequently glimpsed by neurosurgeons, only limited anatomic contributions have been added by microsurgery to its direct in vivo description. A review of our surgical experience in navigating the fourth ventricle prompted us to revisit the classical anatomic descriptions of the aqueduct and compare them using the novel perspective of neuroendoscopy. Methods: We reviewed video recordings of 65 transaqueductal explorations of the fourth ventricle using flexible endoscopes, which were performed in our center to treat various pathological conditions. Forty-one patients were selected as being more informative for anatomic description. They include 21 patients with communicating normal pressure hydrocephalus, 6 patients with intraventricular hemorrhage, 5 patients with membranous obstruction of the foramen of Magendie, 5 patients with trapped fourth ventricle as evidenced after aqueductoplasty, 3 patients with colloid cysts, and 1 patient with craniopharyngioma with apparently normal aqueduct, which was navigated to aspirate small fragments of colloid and tiny clots. Results: Patients with normal-sized third ventricles confirmed the typical triangular shape of the aqueductal adytum, whereas all pathological aqueducts invariably had an oval contour. The posterior commissure, a faint trace of the median sulcus, and the rubral eminences were the structures invariably noticed. Five segments of the aqueduct were always identifiable: the adytum, first constriction, ampulla, second constriction, and posterior part or egressus. Conclusion: Neuroendoscopy provides a novel perspective into the inner aqueductal wall and supplies an incomparable view of the intracanalicular anatomic structures.
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Stankovic, Gordana, Valentina Nikolic, Laslo Puskas, Branislav Filipovic, Ljubica Stojsic-Dzunja, and Dragan Krivokuca. "A histological study of cerebral aqueduct." Medical review 58, no. 11-12 (2005): 534–40. http://dx.doi.org/10.2298/mpns0512534s.

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Cerebral (sylvian) aqueduct is a narrow channel in the mesencephalon. It lies between the tectum and the tegmentum of the mesencephalon and is surrounded by the periaqueductal gray matter. The aim of this study was to determine the shape of the . aqueduct of sylvius and the structure of its walls in a series of transverse histological sections. Serial transverse sections of the mesencephalon were examined in twenty adult brains of both sexes. Six sections were stained by the hematoxyiin-eosin method. The rostral part of the the aqueduct has a triangular shape with dorsal concavity caused by retrocommissural fossae. In the middle, its shape is oval to irregular, the rostral part has a T shape due to isthmic recess on the floor. Walls of the aqueduct are coated with a layer of prismatic cells. Determination of the morphological and histological features of the mesencephalic aqueduct is important for differentiation between physiological and pathological processes in this region.
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Hamilton, Robert, Kevin Baldwin, Jennifer Fuller, Paul Vespa, Xiao Hu, and Marvin Bergsneider. "Intracranial pressure pulse waveform correlates with aqueductal cerebrospinal fluid stroke volume." Journal of Applied Physiology 113, no. 10 (November 15, 2012): 1560–66. http://dx.doi.org/10.1152/japplphysiol.00357.2012.

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This study identifies a novel relationship between cerebrospinal fluid (CSF) stroke volume through the cerebral aqueduct and the characteristic peaks of the intracranial pulse (ICP) waveform. ICP waveform analysis has become much more advanced in recent years; however, clinical practice remains restricted to mean ICP, mainly due to the lack of physiological understanding of the ICP waveform. Therefore, the present study set out to shed some light on the physiological meaning of ICP morphological metrics derived by the morphological clustering and analysis of continuous intracranial pulse (MOCAIP) algorithm by investigating their relationships with a well defined physiological variable, i.e., the stroke volume of CSF through the cerebral aqueduct. Seven patients received both overnight ICP monitoring along with a phase-contrast MRI (PC-MRI) of the cerebral aqueduct to quantify aqueductal stroke volume (ASV). Waveform morphological analysis of the ICP signal was performed by the MOCAIP algorithm. Following extraction of morphological metrics from the ICP signal, nine temporal ICP metrics and two amplitude-based metrics were compared with the ASV via Spearman's rank correlation. Of the nine temporal metrics correlated with the ASV, only the width of the P2 region (ICP-Wi2) reached significance. Furthermore, both ICP pulse pressure amplitude and mean ICP did not reach significance. In this study, we showed the width of the second peak (ICP-Wi2) of an ICP pulse wave is positively related to the volume of CSF movement through the cerebral aqueduct. This finding is an initial step in bridging the gap between ICP waveform morphology research and clinical practice.
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Sola, Joaquin, Isabel Arcas, Juan F. Martinez-Lage, Miguel Martinez Perez, Juan A. Esteban, and M�ximo Poza. "Astrocytoma of the cerebral aqueduct." Child's Nervous System 3, no. 5 (December 1987): 294–96. http://dx.doi.org/10.1007/bf00271827.

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5

Kramer, Larry A., Khader M. Hasan, Ashot E. Sargsyan, Karina Marshall-Goebel, Jörn Rittweger, Dorit Donoviel, Saki Higashi, Benson Mwangi, Darius A. Gerlach, and Eric M. Bershad. "Quantitative MRI volumetry, diffusivity, cerebrovascular flow, and cranial hydrodynamics during head-down tilt and hypercapnia: the SPACECOT study." Journal of Applied Physiology 122, no. 5 (May 1, 2017): 1155–66. http://dx.doi.org/10.1152/japplphysiol.00887.2016.

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To improve the pathophysiological understanding of visual changes observed in astronauts, we aimed to use quantitative MRI to measure anatomic and physiological responses during a ground-based spaceflight analog (head-down tilt, HDT) combined with increased ambient carbon dioxide (CO2). Six healthy, male subjects participated in the double-blinded, randomized crossover design study with two conditions: 26.5 h of −12° HDT with ambient air and with 0.5% CO2, both followed by 2.5-h exposure to 3% CO2. Volume and mean diffusivity quantification of the lateral ventricle and phase-contrast flow sequences of the internal carotid arteries and cerebral aqueduct were acquired at 3 T. Compared with supine baseline, HDT (ambient air) resulted in an increase in lateral ventricular volume ( P = 0.03). Cerebral blood flow, however, decreased with HDT in the presence of either ambient air or 0.5% CO2( P = 0.002 and P = 0.01, respectively); this was partially reversed by acute 3% CO2exposure. Following HDT (ambient air), exposure to 3% CO2increased aqueductal cerebral spinal fluid velocity amplitude ( P = 0.01) and lateral ventricle cerebrospinal fluid (CSF) mean diffusivity ( P = 0.001). We concluded that HDT causes alterations in cranial anatomy and physiology that are associated with decreased craniospinal compliance. Brief exposure to 3% CO2augments CSF pulsatility within the cerebral aqueduct and lateral ventricles.NEW & NOTEWORTHY Head-down tilt causes increased lateral ventricular volume and decreased cerebrovascular flow after 26.5 h. Additional short exposure to 3% ambient carbon dioxide levels causes increased cerebrovascular flow associated with increased cerebrospinal fluid pulsatility at the cerebral aqueduct. Head-down tilt with chronically elevated 0.5% ambient carbon dioxide and acutely elevated 3% ambient carbon dioxide causes increased mean diffusivity of cerebral spinal fluid within the lateral ventricles.
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Lucic, Milos, Katarina Koprivsek, Viktor Till, and Zoran Vesic. "Dynamic magnetic resonance imaging of the cerebrospinal fluid flow within the cerebral aqueduct by different FISIP 2D sequences." Vojnosanitetski pregled 67, no. 5 (2010): 357–63. http://dx.doi.org/10.2298/vsp1005357l.

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Background/Aim. A vast majority of current radiogical techniques, such as computerized tomography (CT) and magnetic resonance imaging (MRI) have great potential of visualization and delineation of cerebrospinal fluid spaces morphology within cerebral aqueduct. The aim of this study was to determine the possibilities of two differently acquired FISP (Fast Imaging with Steady State Precession) 2D MR sequences in the estimation of the pulsatile cerebrospinal fluid (CSF) flow intensity through the normal cerebral aqueduct. Methods. Sixty eight volunteers underwent brain MRI on 1.5T MR imager with additionally performed ECG retrospectively gated FISP 2D sequences (first one, as the part of the standard software package, with following technical parameters: TR 40, TE 12, FA 17, Matrix: 192 ? 256, Acq 1, and the second one, experimentally developed by our investigation team: TR 30, TE 12, FA 70, Matrix: 192 ? 256, Acq 1) respectively at two fixed slice positions - midsagittal and perpendicular to cerebral aqueduct, displayed and evaluated by multiplegated images in a closed-loop cinematographic (CINE) format. Results. Normal brain morphology with preserved patency of the cerebral aqueduct in all of 68 healthy volunteers was demonstrated on MRI examination. Cerebrospinal fluid flow within the cerebral aqueduct was distinguishable on both CINE MRI studies in midsagittal plane, but the estimation of intraaqueductal CSF flow in perpendicular plane was possible on CINE MRI studies acquired with experimentally improved FISP 2D (TR 30, FA 70) sequence only. Conclusion. Due to the changes of technical parameters CINE MRI study acquired with FISP 2D (TR 30, FA 70) in perpendicular plane demonstrated significantly higher capability in the estimation of the CSF pulsation intensity within the cerebral aqueduct. .
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Markenroth Bloch, Karin, Johannes Töger, and Freddy Ståhlberg. "Investigation of cerebrospinal fluid flow in the cerebral aqueduct using high-resolution phase contrast measurements at 7T MRI." Acta Radiologica 59, no. 8 (November 15, 2017): 988–96. http://dx.doi.org/10.1177/0284185117740762.

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Background The cerebral aqueduct is a central conduit for cerebrospinal fluid (CSF), and non-invasive quantification of CSF flow in the aqueduct may be an important tool for diagnosis and follow-up of treatment. Magnetic resonance (MR) methods at clinical field strengths are limited by low spatial resolution. Purpose To investigate the feasibility of high-resolution through-plane MR flow measurements (2D-PC) in the cerebral aqueduct at high field strength (7T). Material and Methods 2D-PC measurements in the aqueduct were performed in nine healthy individuals at 7T. Measurement accuracy was determined using a phantom. Aqueduct area, mean velocity, maximum velocity, minimum velocity, net flow, and mean flow were determined using in-plane resolutions 0.8 × 0.8, 0.5 × 0.5, 0.3 × 0.3, and 0.2 × 0.2 mm2. Feasibility criteria were defined based on scan time and spatial and temporal resolution. Results Phantom validation of 2D-PC MR showed good accuracy. In vivo, stroke volume was −8.2 ± 4.4, −4.7 ± 2.8, −6.0 ± 3.8, and −3.7 ± 2.1 µL for 0.8 × 0.8, 0.5 × 0.5, 0.3 × 0.3, and 0.2 × 0.2 mm2, respectively. The scan with 0.3 × 0.3 mm2 resolution fulfilled the feasibility criteria for a wide range of heart rates and aqueduct diameters. Conclusion 7T MR enables non-invasive quantification of CSF flow and velocity in the cerebral aqueduct with high spatial resolution.
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Feletti, Alberto, Stavros Dimitriadis, and Giacomo Pavesi. "Cavernous Angioma of the Cerebral Aqueduct." World Neurosurgery 98 (February 2017): 876.e15–876.e22. http://dx.doi.org/10.1016/j.wneu.2016.11.096.

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Jacobson, Erica E., David F. Fletcher, Michael K. Morgan, and Ian H. Johnston. "Fluid Dynamics of the Cerebral Aqueduct." Pediatric Neurosurgery 24, no. 5 (1996): 229–36. http://dx.doi.org/10.1159/000121044.

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Feletti, Alberto, Alessandro Fiorindi, and Pierluigi Longatti. "Split cerebral aqueduct: a neuroendoscopic illustration." Child's Nervous System 32, no. 1 (August 1, 2015): 199–203. http://dx.doi.org/10.1007/s00381-015-2827-y.

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Dissertations / Theses on the topic "Cerebral aqueduct"

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Beggs, Clive B., C. R. Magnano, Simon J. Shepherd, K. Marr, V. Valnarov, D. Hojnacki, N. Bergsland, et al. "Aqueductal cerebrospinal fluid pulsatility in healthy individuals is affected by impaired cerebral venous outflow." 2013. http://hdl.handle.net/10454/11801.

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To investigate cerebrospinal fluid (CSF) dynamics in the aqueduct of Sylvius (AoS) in chronic cerebrospinal venous insufficiency (CCSVI)-positive and -negative healthy individuals using cine phase contrast imaging. Materials and Methods Fifty-one healthy individuals (32 CCSVI-negative and 19 age-matched CCSVI-positive subjects) were examined using Doppler sonography (DS). Diagnosis of CCSVI was established if subjects fulfilled ≥2 venous hemodynamic criteria on DS. CSF flow and velocity measures were quantified using a semiautomated method and compared with clinical and routine 3T MRI outcomes. Results CCSVI was associated with increased CSF pulsatility in the AoS. Net positive CSF flow was 32% greater in the CCSVI-positive group compared with the CCSVI-negative group (P = 0.008). This was accompanied by a 28% increase in the mean aqueductal characteristic signal (ie, the AoS cross-sectional area over the cardiac cycle) in the CCSVI-positive group compared with the CCSVI-negative group (P = 0.021). Conclusion CSF dynamics are altered in CCSVI-positive healthy individuals, as demonstrated by increased pulsatility. This is accompanied by enlargement of the AoS, suggesting that structural changes may be occurring in the brain parenchyma of CCSVI-positive healthy individuals
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Beggs, Clive B., Simon J. Shepherd, and P. Zamboni. "Cerebral venous outflow resistance and interpretation of cervical plethysmography data with respect to the diagnosis of chronic cerebrospinal venous insufficiency." 2014. http://hdl.handle.net/10454/10606.

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PURPOSE: To investigate cerebrospinal fluid (CSF) dynamics in the aqueduct of Sylvius (AoS) in chronic cerebrospinal venous insufficiency (CCSVI)-positive and -negative healthy individuals using cine phase contrast imaging. MATERIALS AND METHODS: Fifty-one healthy individuals (32 CCSVI-negative and 19 age-matched CCSVI-positive subjects) were examined using Doppler sonography (DS). Diagnosis of CCSVI was established if subjects fulfilled >/=2 venous hemodynamic criteria on DS. CSF flow and velocity measures were quantified using a semiautomated method and compared with clinical and routine 3T MRI outcomes. RESULTS: CCSVI was associated with increased CSF pulsatility in the AoS. Net positive CSF flow was 32% greater in the CCSVI-positive group compared with the CCSVI-negative group (P = 0.008). This was accompanied by a 28% increase in the mean aqueductal characteristic signal (ie, the AoS cross-sectional area over the cardiac cycle) in the CCSVI-positive group compared with the CCSVI-negative group (P = 0.021). CONCLUSION: CSF dynamics are altered in CCSVI-positive healthy individuals, as demonstrated by increased pulsatility. This is accompanied by enlargement of the AoS, suggesting that structural changes may be occurring in the brain parenchyma of CCSVI-positive healthy individuals.
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Magnano, C. R., C. V. Schirda, B. Weinstock-Guttman, D. S. Wack, E. Lindzen, D. Hojnacki, N. Bergsland, et al. "Cine cerebrospinal fluid imaging in multiple sclerosis." 2012. http://hdl.handle.net/10454/6076.

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PURPOSE: To investigate cerebrospinal fluid (CSF) dynamics in the aqueduct of Sylvius in multiple sclerosis (MS) patients and healthy controls (HC) using cine phase contrast imaging. MATERIALS AND METHODS: In all, 67 MS patients (48 relapsing-remitting [RR] and 19 secondary-progressive [SP]), nine patients with clinically isolated syndrome (CIS), and 35 age- and sex-matched HC were examined. CSF flow and velocity measures were quantified using a semiautomated method and compared with clinical and magnetic resonance imaging (MRI) disease outcomes. RESULTS: Significantly decreased CSF net flow was detected in MS patients compared to HC (-3.7 vs. -7.1 muL/beat, P = 0.005). There was a trend for increased net positive flow between SP, RR, and CIS patients. Altered CSF flow and velocity measures were associated with more severe T1 and T2 lesion volumes, lateral and fourth ventricular volumes, and third ventricular width in MS and CIS patients (P < 0.01 for all). In CIS patients, conversion to clinically definite MS in the following year was related to decreased CSF net flow (P = 0.007). There was a trend between increased annual relapse rate and altered CSF flow/velocity measures in RRMS patients (P < 0.05). CONCLUSION: CSF flow dynamics are altered in MS patients. More severe clinical and MRI outcomes in RRMS and CIS patients relate to altered CSF flow and velocity measures.
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Book chapters on the topic "Cerebral aqueduct"

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Sola, Joaquin, Isabel Arcas, Juan F. Martinez-Lage, Miguel Martinez, Juan A. Esteban, and Máximo Poza. "Astrocytoma of the Cerebral Aqueduct: Case report." In Annual Review of Hydrocephalus, 139. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-662-11152-9_99.

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Kemp, S. S., R. A. Zimmerman, L. T. Bilaniuk, D. B. Hackney, H. I. Goldberg, and R. I. Grossman. "Magnetic Resonance Imaging of the Cerebral Aqueduct." In Annual Review of Hydrocephalus, 58–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-662-11152-9_38.

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Chrissicopoulos, Christos, S. Mourgela, N. Ampertos, A. Sakellaropoulos, K. Kirgiannis, K. Petritsis, and A. Spanos. "Benign Cerebral Aqueductal Stenosis in an Adult." In Acta Neurochirurgica Supplementum, 141–42. Vienna: Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0923-6_28.

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Czosnyka, Zofia, Dong-Joo Kim, Olivier Balédent, Eric A. Schmidt, Peter Smielewski, and Marek Czosnyka. "Mathematical Modelling of CSF Pulsatile Flow in Aqueduct Cerebri." In Acta Neurochirurgica Supplement, 233–36. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65798-1_47.

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Jhaveri, Miral D., Karen L. Salzman, Jeffrey S. Ross, Kevin R. Moore, Anne G. Osborn, and Chang Yueh Ho. "Cerebral Aqueduct/Periaqueductal Lesion." In Expertddx: Brain and Spine, 110–15. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-44308-1.50252-x.

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"5 Cerebral Aqueduct and Fourth Ventricle Anatomy." In Neuroendoscopic Surgery, edited by Jaime Gerardo Torres-Corzo, Leonardo Rangel-Castilla, and Peter Nakaji. Stuttgart: Georg Thieme Verlag, 2016. http://dx.doi.org/10.1055/b-0036-141980.

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Han, Rowland, and David D. Limbrick. "Communicating Hydrocephalus." In Pediatric Neurosurgery, 1–9. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190617073.003.0001.

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Communicating hydrocephalus results from an imbalance between cerebrospinal fluid (CSF) production and reabsorption. It is attributed to impaired absorption of CSF through arachnoid villi, elevated cranial venous sinus pressure, or excessive CSF production. Communicating hydrocephalus is radiologically characterized by dilation of the entire ventricular system and patency of the foramina of Monro, cerebral aqueduct, and fourth ventricular outlets. Surgical intervention should be considered in the setting of clinical or radiographic progression in order to optimize neurologic outcome, provide symptomatic relief, and avoid neurologic deficits. Treatment options include definitive CSF diversion with a CSF shunt or endoscopic third ventriculostomy with or without choroid plexus cauterization. There is a need for standardized care pathways for children with communicating hydrocephalus.
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Wijdicks, Eelco F. M., and William D. Freeman. "Intracranial Pressure." In Mayo Clinic Critical and Neurocritical Care Board Review, edited by Eelco F. M. Wijdicks, James Y. Findlay, William D. Freeman, and Ayan Sen, 69–73. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190862923.003.0008.

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Cerebrospinal fluid (CSF) fills the subarachnoid space, spinal canal, and ventricles of the brain. CSF is enclosed within the brain by the pial layer, ependymal cells lining the ventricles, and the epithelial surface of the choroid plexus, where it is largely produced. Choroid plexus is present throughout the ventricular system with the exception of the frontal and occipital horns of the lateral ventricle and the cerebral aqueduct. The vascular smooth muscle and the epithelium of the choroid plexus receive both sympathetic and parasympathetic input. In an adult, CSF is normally acellular. A normal spinal sample may contain up to 5 white blood cells (WBCs) or red blood cells (RBCs). CSF allows for a route of delivery and removal of nutrients, hormones, and transmitters for the brain.
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Conference papers on the topic "Cerebral aqueduct"

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Hamilton, R. B., K. Baldwin, P. Vespa, M. Bergsneider, and Xiao Hu. "Subpeak regional analysis of intracranial pressure waveform morphology based on cerebrospinal fluid hydrodynamics in the cerebral aqueduct and prepontine cistern." In 2012 34th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2012. http://dx.doi.org/10.1109/embc.2012.6346827.

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Lefever, Joel A., José Jaime García, and Joshua H. Smith. "A Large Deformation Finite Element Model for Non-Communicating Hydrocephalus." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80179.

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In a healthy brain, a continuous flow of cerebrospinal fluid (CSF) is produced in the choroid plexus, located in the lateral ventricles. Most of the CSF drains via the Sylvius aqueduct into the subarachnoid space around the brain, but a small amount flows directly through the cerebrum into the subarachnoid space inside the skull. Non-communicating hydrocephalus occurs when an obstruction blocks the Sylvius aqueduct. Because the cerebrum has only limited capacity for CSF to flow through it, CSF accumulates in the ventricles, yielding a significant increase in ventricular volume and deformation of the cerebrum, which may lead to tissue damage.
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