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Relevant bibliographies by topics / Cerebral arteries

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Academic literature on the topic 'Cerebral arteries'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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Journal articles on the topic "Cerebral arteries"

1

Mizutani, Tohru, Hideaki Kojima, and Yoshimasa Miki. "Arterial dissections of penetrating cerebral arteries causing hypertension-induced cerebral hemorrhage." Journal of Neurosurgery 93, no. 5 (November 2000): 859–62. http://dx.doi.org/10.3171/jns.2000.93.5.0859.

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Object. For the past 130 years, it has been believed that hypertension-induced cerebral hemorrhages are the result of ruptures of microaneurysms or ruptures of arteries that have degenerative changes. The majority of previous investigations have focused on autopsied brain. In this study, the authors attempted to verify the cause of hypertension-induced cerebral hemorrhage by using surgical specimens of the penetrating arteries responsible for the hemorrhages.Methods. Between 1997 and 1999, the authors performed pathological studies in surgical specimens of lenticulostriate arteries that had be

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Warnert, Esther AH, Kevin Murphy, Judith E. Hall, and Richard G. Wise. "Noninvasive Assessment of Arterial Compliance of Human Cerebral Arteries with Short Inversion Time Arterial Spin Labeling." Journal of Cerebral Blood Flow & Metabolism 35, no. 3 (March 2015): 461–68. http://dx.doi.org/10.1038/jcbfm.2014.219.

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A noninvasive method of assessing cerebral arterial compliance (AC) is introduced in which arterial spin labeling (ASL) is used to measure changes in arterial blood volume (aBV) occurring within the cardiac cycle. Short inversion time pulsed ASL (PASL) was performed in healthy volunteers with inversion times ranging from 250 to 850 ms. A model of the arterial input function was used to obtain the cerebral aBV. Results indicate that aBV depends on the cardiac phase of the arteries in the imaging volume. Cerebral AC, estimated from aBV and brachial blood pressure measured noninvasively in systol

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Dieguez, G., J. L. Garcia, N. Fernandez, A. L. Garcia-Villalon, L. Monge, and B. Gomez. "Cerebrovascular and coronary effects of endothelin-1 in the goat." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 263, no. 4 (October 1, 1992): R834—R839. http://dx.doi.org/10.1152/ajpregu.1992.263.4.r834.

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In vivo and in vitro effects of endothelin-1 (ET-1) on cerebral and coronary vasculature of goats were examined and compared. In six anesthetized goats intravenous injections of ET-1 (0.1-0.8 nmol) increased arterial pressure, did not change the middle cerebral (MCA) and left anterior descending or left circumflex coronary (LCC) arterial blood flows (electromagnetically measured), and increased cerebral and coronary vascular resistances. In four other anesthetized goats intra-arterial injections of ET-1 (0.01-0.3 nmol) decreased the MCA flow less than the LCC flow (maximal reduction was 20 and

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Chyatte, Douglas, Jeffrey Reilly, and David M. Tilson. "Morphometric Analysis of Reticular and Elastin Fibers in the Cerebral Arteries of Patients with Intracranial Aneurysms." Neurosurgery 26, no. 6 (June 1, 1990): 939–43. http://dx.doi.org/10.1227/00006123-199006000-00003.

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Abstract Elastin and reticular fibers were identified using standard histological stains in middle cerebral arteries taken from patients who had died from aneurysmal subarachnoid hemorrhage and control patients who did not have cerebral aneurysms, Examination of cerebral arteries from normal individuals revealed a dense network of fine reticular fibers in the arterial media that were uniformly distributed. Computerized morphometric analysis indicated that reticular fibers in the arterial media of cerebral arteries were significantly decreased in patients with aneurysms. In addition, these fibe

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Warnert, Esther AH, Emma C. Hart, Judith E. Hall, Kevin Murphy, and Richard G. Wise. "The major cerebral arteries proximal to the Circle of Willis contribute to cerebrovascular resistance in humans." Journal of Cerebral Blood Flow & Metabolism 36, no. 8 (November 20, 2015): 1384–95. http://dx.doi.org/10.1177/0271678x15617952.

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Cerebral autoregulation ensures constant cerebral blood flow during periods of increased blood pressure by increasing cerebrovascular resistance. However, whether this increase in resistance occurs at the level of major cerebral arteries as well as at the level of smaller pial arterioles is still unknown in humans. Here, we measure cerebral arterial compliance, a measure that is inversely related to cerebrovascular resistance, with our novel non-invasive magnetic resonance imaging-based measurement, which employs short inversion time pulsed arterial spin labelling to map arterial blood volume

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Sasaki, Tomio, Neal F. Kassell, Masanori Yamashita, Shigeru Fujiwara, and Mario Zuccarello. "Barrier disruption in the major cerebral arteries following experimental subarachnoid hemorrhage." Journal of Neurosurgery 63, no. 3 (September 1985): 433–40. http://dx.doi.org/10.3171/jns.1985.63.3.0433.

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✓ The effects of experimental subarachnoid hemorrhage (SAH) on the blood-arterial wall barrier in the major cerebral arteries were studied in 20 normotensive dogs. Horseradish peroxidase (HRP) was given intravenously before the animals were sacrificed to assess the integrity of the barrier. Transient elevation of intracranial pressure (ICP) produced by cisternal injection of saline solution resulted in HRP leakage at the branching points of the major cerebral arteries. Extensive disturbance of the blood-arterial wall barrier was consistently observed in the major cerebral arteries after SAH, w

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Ayajiki, Kazuhide, та Noboru Toda. "Regional Difference in the Response Mediated by β1-Adrenoceptor Subtype in Bovine Cerebral Arteries". Journal of Cerebral Blood Flow & Metabolism 12, № 3 (травень 1992): 507–13. http://dx.doi.org/10.1038/jcbfm.1992.69.

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Helical strips of bovine rostral cerebral arteries (anterior cerebral, middle cerebral, and internal carotid artery) responded to norepinephrine with contractions, whereas the caudal cerebral arteries (posterior communicating, posterior cerebral, and basilar artery) relaxed in response to the amine. After blockade of α-adrenoceptors, norepinephrine-induced rostral artery contractions were reversed to relaxations, which were smaller than those in the caudal arteries. Isoproterenol, dobutamine, and terbutaline produced greater relaxations in caudal than in rostral arteries, but forskolin relaxed

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Sabec-Pereira, Dayane Kelly, Fabiano C. Lima, Fabiano R. Melo, Fabiana Cristina S. A. Melo, Kleber Fernando Pereira, and Valcinir Aloisio S. Vulcani. "Vascularization of the Alouatta belzebul brain base." Pesquisa Veterinária Brasileira 40, no. 4 (April 2020): 315–23. http://dx.doi.org/10.1590/1678-5150-pvb-6536.

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ABSTRACT: We studied the arterial circle in the brain of five specimens of the Alouatta belzebul primate. The material had the arterial system perfused (water at 40°C), injected with stained latex (Neoprene 650), fixed in aqueous formaldehyde solution (10%) and dissected for vessel verification. The arterial circle of this primate is composed of two vascular systems: the vertebra-basilar and the carotid ones, which anastomose to close the arterial circuit. In the caudal portion of the arterial circle, there are the vertebral arteries and their branches: the rostral spinal artery and the caudal

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Mangiarua, Elsa I., and Robert M. K. W. Lee. "Increased sympathetic innervation in the cerebral and mesenteric arteries of hypertensive rats." Canadian Journal of Physiology and Pharmacology 68, no. 4 (April 1, 1990): 492–99. http://dx.doi.org/10.1139/y90-070.

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The density of catecholamine-containing nerve fibers was studied in the cerebral and mesenteric arteries from normotensive Wistar–Kyoto rats (WKY), spontaneously hypertensive rats (SHR), and stroke-prone SHR (SHRSP) in the growing (SHR, WKY) and adult (SHR, SHRSP, WKY) animals. Cerebral arteries from SHR showed an increased adrenergic innervation from day 1. The nerve plexuses reached an adult pattern earlier in SHR than in WKY. The arteries from adult SHR and SHRSP (22 weeks old) showed a markedly higher nerve density than WKY. There was a positive linear correlation between blood pressure an

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Amin, Faisal Mohammad, Mohammad Sohail Asghar, Julie W. Ravneberg, Patrick JH de Koning, Henrik BW Larsson, Jes Olesen, and Messoud Ashina. "The effect of sumatriptan on cephalic arteries: A 3T MR-angiography study in healthy volunteers." Cephalalgia 33, no. 12 (April 15, 2013): 1009–16. http://dx.doi.org/10.1177/0333102413483374.

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Aim To explore a possible differential effect of sumatriptan on extracerebral versus cerebral arteries, we examined the superficial temporal (STA), middle meningeal (MMA), extracranial internal carotid (ICAextra), intracranial internal carotid (ICAintra), middle cerebral (MCA) and basilar arteries (BA). Methods The arterial circumferences were recorded blindly using high-resolution magnetic resonance angiography before and after subcutaneous sumatriptan injection (6 mg) in 18 healthy volunteers. Results We found significant constrictions of MMA (16.5%), STA (16.4%) and ICAextra (15.2%) ( p ≤ 0

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Dissertations / Theses on the topic "Cerebral arteries"

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Pierre, Lisa Natalie. "Endothelin receptors in human small coronary and cerebral arteries." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624843.

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Storer, Kingsley Paul School of Medicine UNSW. "Cerebral arteriovenous malformations: molecular biology and enhancement of radiosurgical treatment." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/31942.

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Object Rupture of intracranial arteriovenous malformations is a leading cause of stroke in children and young adults. Treatment options include surgery and highly focused radiation (stereotactic radiosurgery). For large and deep seated lesions, the risks of surgery may be prohibitively high, while radiosurgery has a disappointingly low efficacy and long latency. Radiosurgery carries the most promise for significant advances, however the process by which radiosurgery achieves obliteration is incompletely understood. Inflammation and thrombosis are likely to be important in the radiation res

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Corrêa, José Fernando Guedes. ""Dificuldades no tratamento microcirúrgico dos aneurismas gigantes e complexos da circulação anterior do polígono de Willis: proposta de escala técnica prognóstica"." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/5/5132/tde-27092005-145748/.

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Para desenvolver e avaliar a aplicabilidade de uma escala técnica prognostica das dificuldades no tratamento microcirúrgico dos aneurismas gigantes e complexos da circulação anterior do polígono de Willis, 50 lesões foram operadas. Um valor numérico foi dado a cada uma das 8 variáveis da escala. Somando-se os valores para cada variável, uma nota (de 1 a 14) foi obtida, para cada uma das 50 cirurgias. Dois grupos, portanto, foram definidos: cirurgia difícil (nota de 1 a 8) e cirurgia extremamente difícil (nota de 9 a 14). Foi feita análise estatística comparando-se os 2 grupos em relação a dive

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Almeida, Lygia Maria de. "Sistematização das artérias da base do encéfalo, distribuição e territórios das artérias cerebrais rostral, média e caudal e da artéria mesencefálica na superfície do encéfalo em jacaré do papo-amarelo (Cayman latirostris)." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2010. http://hdl.handle.net/10183/22679.

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Foram utilizados 30 encéfalos de jacaré do papo-amarelo (Cayman latirostris), injetados com látex, corado em vermelho, com objetivo de sistematizar e descrever a distribuição e territórios das artérias carótidas internas e suas principais ramificações na superfície do encéfalo. As artérias carótidas internas apresentaram uma anastomose intercarótica e a artéria oftálmica interna. Na altura da hipófise estas se dividiram num ramo rostral e num curto ramo caudal, que continuou naturalmente como artéria cerebral caudal. O ramo rostral formou a rede da artéria cerebral média, a artéria cerebral ro

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Hoiland, Ryan Leo. "Carbon dioxide mediated vasomotion of extra-cranial cerebral arteries : a role for prostaglandins?" Thesis, University of British Columbia, 2015. http://hdl.handle.net/2429/54108.

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Cerebrovascular regulation during perturbations in arterial CO₂ is thought to occur solely at the level of the pial vessels. However, recent evidence implicates large extra-cranial cerebral blood vessels in this regulatory process. Although the mechanisms governing CO₂ mediated vasomotion remain unclear, animal and human studies support a large role of prostaglandins. Thus, we examined two hypotheses: 1) vasomotion of the internal carotid artery (ICA) would occur in response to both hyper and hypocapnia; and 2) pharmacological inhibition of prostaglandin synthesis with Indomethacin (INDO; a no

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Dhital, Kumud Kumar. "Perivascular innervation of cerebral arteries and vasa nervorum : changes in development and disease." Thesis, University College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322304.

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Mathewson, Alastair Munro. "The modulation of no-mediated in porcine cerebral arteries by inducible nitric oxide synthase." Thesis, University of Strathclyde, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269936.

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Mughal, Amreen. "Regulation of Vascular Tone in Cerebral and Coronary Arteries by Apelin/APJ Receptor Mechanisms." Diss., North Dakota State University, 2018. https://hdl.handle.net/10365/27847.

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The peptide, apelin, is expressed in fat cells, endothelial cells, and CNS neurons. Increasing evidence (e.g. inotropic and vasomotor effects) supports a role for apelin in the regulation of the cardiovascular system. This research aimed to understand vascular effects of apelin and bridge gaps in the knowledge about apelin-induced effects on different vascular beds i.e. cerebral and coronary arteries. My first objective was to assess apelin-induced vascular effects in cerebral arteries. Based on current data, one could conclude that apelin by itself has no effects on vasomotor tone of cerebral

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Miller, Alastair Leslie. "Investigation of the mechanisms underlying the myogenic response in resistance blood vessels from the rat cerebral vasculature." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322245.

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Sutherland, Brad Alexander, and n/a. "Heme oxygenase and the use of tin protoporphyrin in hypoxia-ischaemia-induced brain damage : mechanisms of action." University of Otago. Department of Pharmacology & Toxicology, 2009. http://adt.otago.ac.nz./public/adt-NZDU20090119.150318.

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Stroke is the third largest cause of death, and the leading cause of disability worldwide. Treatments are sought to reduce mortality, and increase survival time following an ischaemic stroke. Hypoxia-ischaemia (HI) is the combination of cerebral ischaemia and global hypoxia that can lead to neuronal damage, particularly perinatally. The complex neurodegenerative cascade following ischaemic stroke and HI activates many stress pathways, including heme oxygenase (HO). HO metabolises free heme to release iron, carbon monoxide, and biliverdin, which is subsequently metabolised to bilirubin. This th

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Books on the topic "Cerebral arteries"

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Robert, Courbier, ed. Basis for a classification of cerebral arterial diseases: Proceedings of a symposium held in Marseilles, 28-29 September 1984. Amsterdam: Excerpta Medica, 1985.

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W, Baumgartner R., ed. Handbook on cerebral artery dissection. Basel: Karger, 2005.

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Berguer, Ramon. Surgery of the arteries to the head. New York: Springer-Verlag, 1992.

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Takahashi, Shōki. Neurovascular imaging: MRI & microangiography. Dordrecht: Springer, 2010.

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K, Perktold, ed. Computer Simulation lokaler arterieller Strömungsformen unter besonderer Beachtung der cerebralen Gefässe. Graz: Forschungsgesellschaft Joanneum, 1987.

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Hajime, Handa, Kikuchi Haruhiko, Yonekawa Yasuhiro 1939-, and International Symposium on Microvascular Anastomoses for Cerebral Ischemia (6th : 1982 : Kyoto, Japan), eds. Microsurgical anastomoses for cerebral ischemia. New York: Igaku-Shoin, 1985.

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Noé, Battistini, ed. Acute brain ischemia: Medical and surgical therapy. New York: Raven Press, 1986.

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U, Sliwka, and United States. National Aeronautics and Space Administration., eds. Effects of sustained low-level elevations of carbon dioxide on cerebral blood flow and autoregulation of the intracerebral arteries in humans. [Washington, DC: National Aeronautics and Space Administration, 1996.

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U, Sliwka, and United States. National Aeronautics and Space Administration., eds. Effects of sustained low-level elevations of carbon dioxide on cerebral blood flow and autoregulation of the intracerebral arteries in humans. [Washington, DC: National Aeronautics and Space Administration, 1996.

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N, Tulenko Thomas, and Cox Robert H, eds. Recent advances in arterial diseases: Atherosclerosis, hypertension, and vasospasm : proceedings of the A.N. Richards Symposium, held in Philadelphia, Pennsylvania, May 10-11, 1984. New York: Liss, 1986.

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Book chapters on the topic "Cerebral arteries"

1

Schäberle, Wilhelm. "Extracranial Cerebral Arteries." In Ultrasonography in Vascular Diagnosis, 291–375. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-02509-9_5.

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Schäberle, Wilhelm. "Extracranial Cerebral Arteries." In Ultrasonography in Vascular Diagnosis, 291–387. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-64997-9_5.

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Hacke, Werner, Herman J. Gelmers, Michael Hennerici, and Günter Krämer. "Applied Anatomy of the Cerebral Arteries." In Cerebral Ischemia, 1–16. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75548-4_1.

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Antochi, Florina, and Athena Mergeani. "Cervico-cerebral Arteries Dissection." In Arterial Revascularization of the Head and Neck, 301–23. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-34193-4_14.

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Bradac, Gianni Boris. "Spontaneous Dissection of Carotid and Vertebral Arteries." In Cerebral Angiography, 289–304. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54404-0_16.

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Bradac, Gianni Boris. "Spontaneous Dissection of Carotid and Vertebral Arteries." In Cerebral Angiography, 255–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-15678-6_16.

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Widder, Bernhard, and Gerhard F. Hamann. "Cerebral Vasospasms." In Duplex sonography of the brain-supplying arteries, 263–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65566-5_22.

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Bradac, Gianni Boris. "Aortic Arch and Origin of the Cranial Cerebral Arteries." In Cerebral Angiography, 1–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54404-0_1.

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Bradac, Gianni Boris. "Aortic Arch and Origin of the Cranial Cerebral Arteries." In Cerebral Angiography, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-15678-6_1.

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Bradac, Gianni Boris. "Spontaneous Dissection of Carotid and Vertebral Arteries." In Applied Cerebral Angiography, 371–92. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57228-4_16.

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Conference papers on the topic "Cerebral arteries"

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Fonck, E., G. G. Feigl, J. Fasel, D. Sage, M. Unser, D. A. Rüfenacht, and N. Stergiopulos. "Effect of Ageing on Elastin Functionality in Human Cerebral Arteries." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192727.

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The ageing process affects elastin, a key component of the arterial wall integrity and functionality. Elastin may play an important role in cerebral vessels because elastin degradation is linked to cerebrovascular disease [1]. The goal of this study is to assess the biomechanical properties of human cerebral arteries, their composition and geometry, with particular focus on the functional alterations of elastin in cerebral arteries due to ageing.

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Fabbri, Dario, Quan Long, Saroj Das, and Michele Pinelli. "Study of Embolic Particle Migration in Cerebral Arteries by Computational Modelling." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80314.

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As known, embolism is one of the major causes of stroke, which represents the rapid loss of brain functions. Two major sources of emboli which may cause ischemic attack were emboli formed in heart and from a ruptured arterial plaque in carotid arties. Due to the different characteristics of emboli formed from different mechanisms, the migration route of specific emboli in cerebral arteries may be different, so does the territory of the ischemic attack caused by them. Therefore, a good understanding of emboli migration in the complex cerebral arterial network may provide a good guidance for the

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Jin, Kazuyoshi, Ko Kitamura, Shunji Mugikura, Naoko Mori, Makoto Ohta, and Hitomi Anzai. "Evaluation of Normalization Methods in a Cerebral Artery Atlas for Automatic Labeling." In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-71097.

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Abstract An existence probability atlas has been used for automatic labeling of cerebral arteries. However, the number of arteries varies frequently because of image quality and individual variation of the artery structure. To moderate the influence of number imbalance on labeling accuracy, we propose a new normalized atlas for automatic labeling of cerebral artery centerlines. The number of arteries, which was obtained from magnetic resonance angiography, varies from 11 to 46 among the artery sites. Based on the centerline and diameter, the arterial volume was reconstructed into a voxel space

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Northcutt, Avione, and Hai-Chao Han. "Finite Element Analysis of Buckling of Arteries With Aneurysms." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206275.

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Tortuosity of arteries occurs when a normally straight artery starts to take on a twisted path. This is often referred to as tortuosity and kinking of arteries. This phenomenon occurs in arteries throughout the body including the aorta and cerebral [1, 2]. Arterial tortuosity is a potentially dangerous condition that can lead to cerebrovascular symptoms, ischemia, and stroke [3].

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Harazawa, M., and T. Yamaguchi. "Computational Fluid Dynamics Simulation of the Blood Flow in the Circle of Willis." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32516.

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The blood supply for the brain is born by four arteries, that is, two internal carotid arteries and two vertebral arteries. They are mutually connected at the cerebral base, and form a closed arterial circle, called the circle of Willis, so that the safety of the brain blood supply is increased. However their anastomoses show a very wide variety of atypism. If some of anastomses are very thin, or even do not exist, the safety of the blood supply is not secured. This is particularly important when some diseases such as cerebral thrombosis occurs and the blood flow supply stops unilaterally. Red

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Zhu, Guangyu, Qi Yuan, and Joon Hock Yeo. "Experimental Study of Hemodynamics in the Circle of Willis." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14162.

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The Circle of Willis (CoW) is a ring like structure located at the base of brain, which is composed of a single anterior communicating artery (ACoA), paired anterior cerebral arteries (ACAs), paired internal carotid arteries (ICAs), paired posterior communicating arteries (PCoAs), paired posterior cerebral arteries (PCAs), paired vertebral arteries (VAs) and a single basilar artery (BA). It is the main cerebral blood perfusion pathway and provides an important collateral channel in patients with severe carotid or vertebral artery disease. Over 50% of stroke cases are related to the stenosis of

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Yuan, Qi, Zhen Chen, and Guangyu Zhu. "Blood Flow Dynamic and Fluid-Structure Interaction in Patient-Specific Circle of Willis." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206230.

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Over 50% stroke is related to the cerebral artery stenosis. The most common position is in the Circle of Willis (CoW), which is composed of a single anterior communicating artery (ACoA), paired anterior cerebral arteries (ACA), paired middle cerebral arteries (MCA), internal carotid arteries (ICA), posterior communicating arteries (PCoA) and posterior cerebral arteries (PCA). Detailed knowledge of the cerebral hemodynamics is important for a variety of clinical applications [1]. There has been a significant body of research performed on blood flow in the CoW [1,2] treating the cerebral vascula

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Reymond, Philippe, Fabrice Merenda, Fabienne Perren, Daniel Rüfenacht, and Nikos Stergiopulos. "Validation of 1D Model of the Systemic Arterial Tree Including the Cerebral Circulation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192529.

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The aim of this study is to develop a distributed model of the entire systemic arterial tree, coupled to a heart model and including a detailed description of the cerebral arteries. Distributed models of the arterial tree have been studied extensively in the past (Avolio [1], Stergiopulos et al [2], Westerhof et al [3]), however, no model has been developed so far that offers a physiologically relevant coupling to the heart and includes the entire cerebral arterial tree.

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Reymond, Philippe, Fabrice Merenda, Fabienne Perren, Daniel Rüfenacht, and Nikos Stergiopulos. "One Dimensional Model of the Systemic Arterial Tree Including Cerebral Circulation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176452.

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The aim of this study is to develop a distributed model of the entire systemic arterial tree, coupled to a heart model and including a detailed description of the cerebral arteries. Distributed models of the arterial tree have been studied extensively in the past (Avolio [1]; Cassot et al [2]; Meister [3]; Schaaf and Abbrecht [4]; Stergiopulos et al [5]; Westerhof et al [6]; Zagzoule and Marc-Vergnes [7]), however, no model has been developed so far that offers a physiologically relevant coupling to the heart and includes the entire cerebral artery network.

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Fonck, E., G. Feigl, L. Augsburger, D. A. Rüfenacht, and N. Stergiopulos. "Structural Properties of Human Cerebral Arteries as Assessed by a Constituent-Based Biomechanical Model." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176122.

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Human cerebral arteries may develop aneurysms, as a result of geometrical and structural modifications in the arterial wall resulting from a variety of genetic, epigenetic and biomechanical factors. Aneurysms exhibit a markedly different wall composition and structure, characterized by relatively low elastin content as shown in Figure 1.

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Reports on the topic "Cerebral arteries"

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Alshammari, Mohammed Kanan. Efficacy of Complementary and Alternative Medicine in Peripheral Arterial Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0001.

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Review question / Objective: To explore various CAM therapies available and to generate evidence that these therapies are effective for managing the disease. Condition being studied: Peripheral arterial disease (PAD) is described as the atherosclerotic process of arteries other than cerebral and coronary arteries i.e. the abdominal aorta, iliac, and arteries of the lower limb which leads to the narrowing and blocking of arteries. Information sources: An online systematic literature search will be done from the time of database inception from 5 electronic databases namely PubMed, Cochrane Centr

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