Relevant bibliographies by topics / Cerebral collateral flow

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  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Cerebral collateral flow'

Author: Grafiati
Published: 5 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Cerebral collateral flow"

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Winship, Ian R., Glenn A. Armitage, Gomathi Ramakrishnan, Bin Dong, Kathryn G. Todd, and Ashfaq Shuaib. "Augmenting Collateral Blood Flow during Ischemic Stroke via Transient Aortic Occlusion." Journal of Cerebral Blood Flow & Metabolism 34, no. 1 (2013): 61–71. http://dx.doi.org/10.1038/jcbfm.2013.162.

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Collateral circulation provides an alternative route for blood flow to reach ischemic tissue during a stroke. Blood flow through the cerebral collaterals is a critical predictor of clinical prognosis after stroke and response to recanalization, but data on collateral dynamics and collateral therapeutics are lacking. Here, we investigate the efficacy of a novel approach to collateral blood flow augmentation to increase collateral circulation by optically recording blood flow in leptomeningeal collaterals in a clinically relevant model of ischemic stroke. Using high-resolution laser speckle cont
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Connolly, Florian, Jens E. Röhl, Javier Lopez-Prieto, Leon A. Danyel, Stephan J. Schreiber, and Jose M. Valdueza. "Pattern of Activated Pathways and Quality of Collateral Status in Patients with Symptomatic Internal Carotid Artery Occlusion." Cerebrovascular Diseases 48, no. 3-6 (2019): 244–50. http://dx.doi.org/10.1159/000504663.

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Background: Internal carotid artery occlusion (ICAO) is an important risk factor for stroke. Cerebral hemodynamics in patients with ICAO depends on the individual capacity to activate sufficient collateral pathways. Therefore, the assessment of intracranial collaterals is essential for the acute and long-term management of these patients and accurate estimation of further stroke risk. Methods: Acute stroke patients with unilateral ICAO were prospectively enrolled. We assessed the following collaterals by transcranial color-coded sonography (TCCS): the anterior and posterior communicating arter
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Liebeskind, David S., George A. Cotsonis, Jeffrey L. Saver, Michael J. Lynn, Harry J. Cloft, and Marc I. Chimowitz. "Collateral Circulation in Symptomatic Intracranial Atherosclerosis." Journal of Cerebral Blood Flow & Metabolism 31, no. 5 (2010): 1293–301. http://dx.doi.org/10.1038/jcbfm.2010.224.

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Collateral circulation in intracranial atherosclerosis has never been systematically characterized. We investigated collaterals in a multicenter trial of symptomatic intracranial atherosclerotic disease. Baseline angiography was reviewed for information on collaterals in stenoses of the internal carotid, middle cerebral, vertebral, and basilar arteries. A battery of angiographic scales was utilized to evaluate lesion site, arterial patency, antegrade flow, downstream territorial perfusion, and collateral circulation, blinded to all other data. Collateral circulation was adequately available fo
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Muhonen, Michael G., Christopher M. Loftus, and Donald D. Heistad. "Effects of Adenosine and 2-Chloroadenosine on Cerebral Collateral Vessels." Journal of Cerebral Blood Flow & Metabolism 15, no. 6 (1995): 1075–81. http://dx.doi.org/10.1038/jcbfm.1995.134.

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Adenosine is a potent cerebral vasodilator. We tested the hypothesis that dilatation of collateral vessels in cerebrum, in response to topical adenosine and 2-chloroadenosine (2-CAD), would increase blood flow to collateral-dependent cerebrum. In dogs anesthetized with halothane, a branch of the middle cerebral artery (MCA) was occluded proximally and cannulated distally. The collateral-dependent area at risk for infarction was perfused from a reservoir with microsphere-free blood, and blood flow to normal cerebrum and to cerebrum dependent on collateral flow was measured with radioactive micr
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Hartkamp, Nolan S., Esben T. Petersen, Michael A. Chappell, et al. "Relationship between haemodynamic impairment and collateral blood flow in carotid artery disease." Journal of Cerebral Blood Flow & Metabolism 38, no. 11 (2017): 2021–32. http://dx.doi.org/10.1177/0271678x17724027.

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Collateral blood flow plays a pivotal role in steno-occlusive internal carotid artery (ICA) disease to prevent irreversible ischaemic damage. Our aim was to investigate the effect of carotid artery disease upon cerebral perfusion and cerebrovascular reactivity and whether haemodynamic impairment is influenced at brain tissue level by the existence of primary and/or secondary collateral. Eighty-eight patients with steno-occlusive ICA disease and 29 healthy controls underwent MR examination. The presence of collaterals was determined with time-of-flight, two-dimensional phase contrast MRA and te
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Lan, Linfang, Xinyi Leng, Vincent Ip, et al. "Sustaining cerebral perfusion in intracranial atherosclerotic stenosis: The roles of antegrade residual flow and leptomeningeal collateral flow." Journal of Cerebral Blood Flow & Metabolism 40, no. 1 (2018): 126–34. http://dx.doi.org/10.1177/0271678x18805209.

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We aimed to investigate the roles of antegrade residual flow and leptomeningeal collateral flow in sustaining cerebral perfusion distal to an intracranial atherosclerotic stenosis (ICAS). Patients with apparently normal cerebral perfusion distal to a symptomatic middle cerebral artery (MCA)-M1 stenosis were enrolled. Computational fluid dynamics models were built based on CT angiography to obtain a translesional pressure ratio (PR) to gauge the residual antegrade flow. Leptomeningeal collaterals (LMCs) were scaled on CT angiography. Cerebral perfusion metrics were obtained in CT perfusion maps
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Kaspera, Wojciech, Henryk Majchrzak, Piotr Ładziński, and Witold Tomalski. "Color Doppler Sonographic Evaluation of Collateral Circulation in Patients with Cerebral Aneurysms and the Occlusion of the Brachiocephalic Vessels." Neurosurgery 57, no. 6 (2005): 1117–26. http://dx.doi.org/10.1227/01.neu.0000186009.62401.3c.

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Abstract OBJECTIVE: The objective of this study was to assess the collateral circulation and blood flow velocity in arteries forming collateral circulation in patients with cerebral aneurysms and the occlusion of the brachiocephalic vessels. METHODS: Between 1989 and 2004 we examined a group of seven consecutive patients with diagnosed cerebral aneurysm and occlusion of the extracranial artery by means of cerebral angiography, transcranial color-coded sonography (TCCS) and color Doppler sonography of extracranial arteries. The Doppler examination results achieved in this group were compared wi
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Beretta, Simone, Alessandro Versace, Davide Carone, et al. "Cerebral collateral therapeutics in acute ischemic stroke: A randomized preclinical trial of four modulation strategies." Journal of Cerebral Blood Flow & Metabolism 37, no. 10 (2017): 3344–54. http://dx.doi.org/10.1177/0271678x16688705.

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Cerebral collaterals are dynamically recruited after arterial occlusion and highly affect tissue outcome in acute ischemic stroke. We investigated the efficacy and safety of four pathophysiologically distinct strategies for acute modulation of collateral flow (collateral therapeutics) in the rat stroke model of transient middle cerebral artery (MCA) occlusion. A composed randomization design was used to assign rats (n = 118) to receive phenylephrine (induced hypertension), polygeline (intravascular volume load), acetazolamide (cerebral arteriolar vasodilation), head down tilt (HDT) 15° (cerebr
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Muhonen, Michael G., Scott C. Robertson, Jeffrey S. Gerdes, and Christopher M. Loftus. "Effects of serotonin on cerebral circulation after middle cerebral artery occlusion." Journal of Neurosurgery 87, no. 2 (1997): 301–6. http://dx.doi.org/10.3171/jns.1997.87.2.0301.

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✓ Serotonin (5-HT) produces constriction of peripheral collateral blood vessels. Using an animal model, the authors tested the hypothesis that 5-HT constricts collateral vessels in the cerebrum. A branch of the middle cerebral artery (MCA) was occluded proximally and cannulated distally in anesthetized dogs. Blood flow to the area at risk for infarction was detected by perfusing the cannulated MCA branch with microsphere-free blood during systemic injection of radioactive microspheres (shadow flow technique). Blood flow to collateral-dependent and normal cerebrum was measured during intravenou
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Seiler, Alexander, Annemarie Brandhofe, René-Maxime Gracien, et al. "DSC perfusion-based collateral imaging and quantitative T2 mapping to assess regional recruitment of leptomeningeal collaterals and microstructural cortical tissue damage in unilateral steno-occlusive vasculopathy." Journal of Cerebral Blood Flow & Metabolism 41, no. 1 (2020): 67–81. http://dx.doi.org/10.1177/0271678x19898148.

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Leptomeningeal collateral supply is considered pivotal in steno-occlusive vasculopathy to prevent chronic microstructural ischaemic tissue damage. The aim of this study was to assess the alleged protective role of leptomeningeal collaterals in patients with unilateral high-grade steno-occlusive vasculopathy using quantitative (q)T2 mapping and perfusion-weighted imaging (PWI)-based collateral abundance. High-resolution qT2 was used to estimate microstructural damage of the segmented normal-appearing cortex. Volumetric abundance of collaterals was assessed based on PWI source data. The ratio re
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Dissertations / Theses on the topic "Cerebral collateral flow"

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McConnell, Flora A. Kennedy. "Quantifying collateral flow pathways in the brain." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:2a0142ed-6161-4294-abd4-acd377ba6fed.

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Ischaemic stroke is a major cause of death and disability worldwide. Cerebral autoregulation, which can be impaired during acute stroke, and collateral flow to brain tissue through the circle of Willis, both play a role in preventing tissue infarction. The configuration of the arterial circle varies between individuals. Thus, personalised modelling of the cerebral arterial network, to determine the potential for collateral flow, can be of significant value in the clinical context of stroke. The interaction between autoregulation and collateral flow remains poorly understood. In this study, ste
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Peixoto, Daniel Escobar Bueno. "Análise do fluxo sanguíneo colateral em pacientes com AVC isquêmico por oclusão proximal da artéria cerebral média." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17161/tde-27082018-095709/.

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Sabe-se que um bom fluxo sanguíneo colateral (FSC) leptomeníngeo está associado a melhor desfecho funcional em paciente com oclusão vascular da circulação anterior. Entretanto, poucos estudos avaliaram os preditores de FSC e seu impacto prognósticos em países em desenvolvimento. O objetivo deste trabalho é avaliar os preditores e o impacto prognóstico do FSC leptomeníngeo nas oclusões do primeiro segmento (M1) da Artéria Cerebral Média (ACM). Métodos: Foi avaliado de maneira retrospectiva, uma coorte prospectiva de pacientes com acidente vascular cerebral (AVC), admitidos em um centro terciári
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Books on the topic "Cerebral collateral flow"

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Waje-Andreassen, Ulrike, and Nicola Logallo. Vascular imaging: Ultrasound. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198722366.003.0009.

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After computed tomography and computed tomography angiography or magnetic resonance imaging and magnetic resonance angiography at admission, ultrasound is the most important diagnostic tool to confirm angiographic findings and to closely follow-up patients until the clinical situation has stabilized. Thrombolysis and interventional therapy have given transcranial ultrasound a very important role in bedside monitoring of occlusions, collaterals, cerebral haemodynamics, and vasoreactivity. Detection of flow changes in sickle cell disease, circulating emboli, and right-to-left shunts may guide tr
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Book chapters on the topic "Cerebral collateral flow"

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Forsting, M., D. Krieger, R. von Kummer, W. Hacke, and K. Sartor. "The Prognostic Value of Collateral Blood Flow in Acute Middle Cerebral Artery Occlusion." In Thrombolytic Therapy in Acute Ischemic Stroke II. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78061-5_23.

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Kennedy McConnell, Flora A., and Stephen J. Payne. "Autoregulating Cerebral Tissue Selfishly Exploits Collateral Flow Routes Through the Circle of Willis." In Acta Neurochirurgica Supplement. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65798-1_54.

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Saqqur, Maher. "Transcranial Doppler (TCD) and Transcranial Color-Coded Duplex Sonography (TCCS): Defining Collateral Cerebral Blood Flow." In Neurosonology in Critical Care. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81419-9_32.

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Petito, Carol K. "Cerebrovascular Disease." In Principles And Practice Of Neuropathology. Oxford University PressNew York, NY, 2003. http://dx.doi.org/10.1093/oso/9780195125894.003.0008.

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Abstract Cerebrovascular diseases are a broad category of disorders that affect the cerebral circulation. They encompass not only diseases of the intraparenchymal and extraparenchymal cerebral blood vessels but also abnormalities in the systemic vasculature, especially the carotid arteries and heart, and systemic illnesses that alter blood coagulation or blood flow. The end result of these cerebrovascular abnormalities is cerebral hemorrhage or cerebral ischemia. The risk factors for cerebrovascular disease include environmental hazards such as cigarette smoking, metabolic disorders such as diabetes, and genetic factors such as protein S deficiency (Natowicz and Kelley, 1987). The following discussion reviews clinical disorders of cerebrovascular diseases (Whisnant, 1990), briefly discusses the pathology and etiology of infarcts and hemorrhage, and then describes specific disorders of the cerebral blood vessels. Clinical Disorders Cerebrovascular diseases are asymptomatic when an infarct or hemorrhage is in a “silent” region of the central nervous system or when arterial occlusion is compensated by adequate collateral flow. Such collateral pathways include the circle of Willis, communications between the external and internal carotid arteries via the ophthalmic artery, and reversed flow from the anterior spinal artery into the vertebral artery.
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Crum, Dr Brian A. "Dural arteriovenous fistula (DAVF) of the left medulla." In Fifty Neurologic Cases From Mayo Clinic. Oxford University PressNew York, NY, 2004. http://dx.doi.org/10.1093/oso/9780195177442.003.0027.

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Abstract The diagnosis was suggested in part by finding, on the cervical MRI study, dilated vessels within the upper cervical spinal canal extending from the level of the brainstem to C5 (Figure 2). Selective cerebral angiography identified, at the level of the left pontomedullary junction, a small high-flow DAVF supplied by the left vertebral artery, with left meningeal and left anterior inferior and posterior inferior cerebellar artery trunk feeder vessels and a small contribution through collateral vessels from the left external carotid artery.
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Conference papers on the topic "Cerebral collateral flow"

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Zhu, Guangyu, Qi Yuan, and Joon Hock Yeo. "Experimental Study of Hemodynamics in the Circle of Willis." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14162.

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The Circle of Willis (CoW) is a ring like structure located at the base of brain, which is composed of a single anterior communicating artery (ACoA), paired anterior cerebral arteries (ACAs), paired internal carotid arteries (ICAs), paired posterior communicating arteries (PCoAs), paired posterior cerebral arteries (PCAs), paired vertebral arteries (VAs) and a single basilar artery (BA). It is the main cerebral blood perfusion pathway and provides an important collateral channel in patients with severe carotid or vertebral artery disease. Over 50% of stroke cases are related to the stenosis of
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Luppi, Luca, Quan Long, Carola S. König, and Michael W. Collins. "Numerical Study of Blood Redistribution in the Circle of Willis of Patients With Carotid Artery Stenosis." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176429.

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Stroke is an acute neurological injury in which the blood supply to a part of the brain is interrupted due to either a diminution of cerebral blood flow or a sudden occlusion of a feeding artery. Blood perfusion depends not only on the status of the diseased vessels but also on the patency of collateral pathways. The primary collateral pathway is that provided by the Circle of Willis (CoW).
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Lima, Iana Campinho Braga de Araújo, Lavínia Flávia Xavier de Souza, Clara Wilma Fernandes Rosendo, et al. "Cerebral venous thrombosis simulating cerebral arterial thrombosis: Late complication of COVID-19?" In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.345.

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Context: Brain venous thrombosis (BVT) is uncommon and usually has a different clinic and treatment from cerebral arterial thrombosis. In this context, COVID-19 correlates with thrombogenesis with varied clinical repercussions. This report describes an unusual BVT case as a possible late complication of COVID-19. Case report: Male, 68 years old, athlete and healthy. April/2020: COVID-19 mild symptoms. February/2021: in road-running, he fell due to sudden left hemiparesis. Upon hospital admission: contacting, persistent headache. A week after, low level of consciousness and coma, when underwent
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Park, Dong Woo. "P003 Clinical relevance of leptomeningeal collateral blood flow on MRI in patients with acute middle cerebral artery occlusion." In 16th Congress of the European Society of Minimally Invasive Neurological Therapy (ESMINT) 2024. BMJ Publishing Group Ltd., 2024. http://dx.doi.org/10.1136/jnis-2024-esmint.40.

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Fahy, Paul, Patrick Delassus, Padraig O’Flynn, and Liam Morris. "An Experimental Study of the Effects Anatomical Variations Have on Collateral Flows Within the Circle of Willis." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53723.

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The circle of Willis (CoW) is a complex arterial network comprising of major cerebral arteries that converge to form a pentagonal arrangement as shown in Figure 1(A). This arterial network supplies oxygen-enriched blood to the brain. An incomplete CoW can exist in up to 50% of cases [1]. These missing vessels can be accommodated by the collateral flow feature within the CoW configuration. In certain circumstances, anatomical variations within the CoW can result in undesirable flow patterns [2–3]. It is unclear from the literature what effects these variations can have on blood flow collision p
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Bernardes, Leonardo de Sousa, Raphael Palomo Barreira, Marina Trombin Marques, Danyelle Sadala Reges, Vivian Dias Baptista Gagliardi, and Rubens José Gagliardi. "Internal Carotid Artery Dissection during Puerperium: A Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.456.

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Background: Carotid or vertebral artery dissection is a rare puerperium event, occurring in less than 6% of cases. Although physiopathology is not completely understood, it is probably multifactorial involving increased cardiac output, hypervolemia and hormonal changes remodeling endothelium and favoring dissection. Most patients present headache and neck pain, but it is also reported Horner syndrome, tinnitus, retroorbital pain and cranial nerve palsies. Case Report: Female, 39-year-old patient in the 20th day of puerperium presents a sudden headache with transitory left hemiparesis and dysar
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Rayz, V. L., G. Acevedo-Bolton, M. T. Lawton, V. Halbach, J. R. Leach, and D. Saloner. "Numerical Modeling of the Flow in Cerebral Aneurysms Can Predict Thrombus Deposition Regions Following Vascular Interventions." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14155.

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Giant intracranial aneurysms present a grave danger of hemorrhage, cerebral compression, and thromboembolism. Fusiform aneurysms present a particular challenge for interventional treatment since these lesions cannot be completely removed from the circulation by clipping or coiling without sacrificing flow to the distal vasculature. In some cases, these lesions can be treated by interventions eliminating pathological hemodynamics, such as indirect aneurysm occlusion or deployment of a flow diverter stent (FDS). The first approach consists of proximal occlusion, distal occlusion, or trapping, so
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Araújo, Maria Tereza Corrêa de, Amanda Virgínia Oliveira Leite, Ana Clara Sousa Leal, Daniel Jonatan de Aguiar Almeida, Iris Maria de Miranda Correia, and David Plácido Lopes. "AGenesis of the internal carotid artery (ICA agenesis): an integrative review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.308.

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Introduction: The ICA agenesis is a rare anomaly, an incidental finding whose incidence is lower than 0,01%. It is predominantly asymptomatic, because of the development of a collateral circulation, though it can present nonspecific symptoms, that result from insufficient blood flow, due to the development of atherosclerosis in vessels that previously supported the blood flow in the absence of the ICA, and by the association with brain aneurysm. Objectives: Conduct an integrative review about the ICA agenesis, describing the aspects and importance of vascular changes. Methods: It is an integra
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Sazonova, Olga B., Anna A. Ogurtsova, Elena Troshina, and Eugene Macherov. "THE POSSIBILITIES OF ELECTROENCEPHALOGRAPHY IN ASSESSING THE COLLATERAL BLOOD CIRCULATION OF THE HUMAN BRAIN." In NEW TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2021. http://dx.doi.org/10.47501/978-5-6044060-1-4.55.

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The purpose of this study was to show the role and capabilities of EEG in the assessment of collateral brain circulation (CBM). The paper presents the results of a retrospective EEG anal-ysis of 210 patients with large and giant cerebral aneurysms of various localization, who, in the order of neurosurgical treatment, had to turn off the vessel carrying the aneurysm. We used a test with compression of the carotid artery in the neck, leading to restriction of blood flow through the main vessels of the head. For EEG analysis, visual and mathematical methods were used with the calculation of power
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