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Relevant bibliographies by topics / Cerebral ischemia stroke apoplexy

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Academic literature on the topic 'Cerebral ischemia stroke apoplexy'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Cerebral ischemia stroke apoplexy"

1

Ahn, Jae-Min, Hyuk-Jin Oh, Jae-Sang Oh, and Seok-Mann Yoon. "Pituitary apoplexy causing acute ischemic stroke: Which treatment should be given priority." Surgical Neurology International 11 (May 16, 2020): 113. http://dx.doi.org/10.25259/sni_82_2020.

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Background: Pituitary apoplexy is syndrome of sudden onset of headache, visual loss, pituitary dysfunction, and altered consciousness. Pituitary apoplexy followed by acute cerebral ischemia is extremely rare. Here, we introduced the case of successful surgical resection of pituitary adenoma which induced acute cerebral ischemia. Case Description: A 78-year-old man with a known pituitary macroadenoma presented with decreased consciousness and left hemiparesis. Magnetic resonance image (MRI) and computed tomography (CT) showed large pituitary macroadenoma with hemorrhage and diffusion-perfusion

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Azizov, Miralim M. "Acute stroke in pituitary adenomas complicated by pituitary apoplexy." Russian Journal of Oncology 21, no. 3 (2016): 136–39. http://dx.doi.org/10.18821/1028-9984-2016-21-3-136-139.

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Pituitary apoplexy is a clinical syndrome that is manifested by headache, visual disturbances, ophthalmoplegia or impaired consciousness. It can develop as a result of necrosis or hemorrhage in the pituitary gland or in cases of pituitary tumors. A favorable prognosis is possible if early diagnosis and timely surgical treatment. Pituitary apoplexy complicated by the disorder of the cerebral circulation occurs relatively rare. We observed the female patient aged of 51 year with pituitary adenoma, clinical signs of which were sudden depression of consciousness, right hemiparesis and left-sided p

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Hu, Beilei, Songfang Chen, Ming Zou, Zhiyong He, Shengmin Shao, and Baohua Liu. "Effect of Extracellular Vesicles on Neural Functional Recovery and Immunologic Suppression after Rat Cerebral Apoplexy." Cellular Physiology and Biochemistry 40, no. 1-2 (2016): 155–62. http://dx.doi.org/10.1159/000452533.

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Background: Previous studies have demonstrated that mesenchymal stem cells (MSCs) can promote the recovery of neural function after cerebral apoplexy by secreting multiple cytokines. In addition, cell factor-derived extracellular vesicles play an important role in recovery of neural function. The aim of this study was to determine the effect of extracellular vesicles on neural functional recovery and brain tissue remodeling after cerebral apoplexy in a rat model. Methods: The rat models with local ischemic stroke was established and three random groups were created. In groups A and B, human bo

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Kornilova, A. A., O. V. Lagoda, and M. M. Tanashyan. "Cerebral Amyloid Angiopathy in Combination with Paroxysmal Atrial Fibrillation." Russian neurological journal 25, no. 4 (2020): 31–37. http://dx.doi.org/10.30629/2658-7947-2020-25-4-31-37.

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The present article addresses the definition of cerebral amyloid angiopathy (CAA) and its symptoms based on the analysis of the medical case; the issues of diagnosis and treatment of this pathology are discussed. The Boston criteria, which became the basis for diagnosis, study of clinical manifestations and progression of CAA and approaches to its therapy, are presented. Methods and modes of neuroimaging, including magnetic resonance imaging (MRI), which verify micro cerebral haemorrhage, are described. At the same time, the role and significance of cardiac arrhythmias in the genesis of ischem

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Wortham, Joy, Brenda Sandoval, Maureen Koops, Ramona Granda-Rodriguez, and Jan M. Bruder. "Giant Prolactinoma Causing Proptosis and Stroke." Journal of the Endocrine Society 5, Supplement_1 (2021): A584. http://dx.doi.org/10.1210/jendso/bvab048.1191.

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Abstract Background: Although suprasellar and cavernous sinus invasion are common in giant prolactinomas, intra-orbital extension is extremely uncommon [1]. Even less reported are cases of giant prolactinomas causing cerebral ischemia or death. Clinical Case: A 51-year old woman presented to the ED with confusion, right-sided weakness and severe left eye proptosis with loss of vision. Five years prior, she underwent a partial transphenoidal resection for a macroprolactinoma due to acute vision changes with compression of the optic chiasm. Prior to surgery, prolactin level was elevated to 2,106

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Zou, Mingyang, Junjie Liao, Yurong Zeng, Qianwen Guan, and Bowen Lan. "Diagnostic Value of CT Angiography Combined with High-Resolution Magnetic Resonance Angiography in Vascular Lesions in Acute Stroke." Scientific Programming 2021 (September 11, 2021): 1–7. http://dx.doi.org/10.1155/2021/2274443.

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Cerebrovascular disease is increasing rapidly because of its high morbidity and high mortality, which is a serious threat to human health. For the early diagnosis and treatment of diseases, the CT vascular noise combined with high-resolution magnetic resonance angiography in acute cerebral apoplexy vascular disease is adopted. 150 patients with ischemic stroke were selected, which were admitted to the Department of Radiology, Huizhou Central People’s Hospital, from January 2020 to December 2020. All patients accepted digital subtraction angiography (DSA), magnetic resonance angiography (MRA),

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7

Ben-Nakhi, A., T. J. E. Muttikkal, V. N. K. Chavan, A. Yt Al-Turkomani, and R. Gupta. "Pituitary Apoplexy: A Rare Cause of Cerebral Infarction." Neuroradiology Journal 21, no. 5 (2008): 661–65. http://dx.doi.org/10.1177/197140090802100509.

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Pituitary apoplexy is usually the result of hemorrhagic infarction in pituitary adenoma. The clinical presentation of pituitary apoplexy varies widely and includes asymptomatic hemorrhage, classical pituitary apoplexy and even sudden death. Few cases of cerebral infarction associated with pituitary apoplexy have been reported in the literature. Pituitary apoplexy can cause narrowing of intracranial vessels by mechanical obstruction due to mass effect or by vasospasm resulting in cerebral ischemia. We report a case of pituitary apoplexy associated with cerebral infarction and the putative mecha

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Bettag, Christoph, Christoph Strasilla, Andreas Steinbrecher, and Rüdiger Gerlach. "Unilateral Tuberothalamic Artery Ischemia Caused by Pituitary Apoplexy." Journal of Neurological Surgery Part A: Central European Neurosurgery 79, no. 06 (2018): 550–54. http://dx.doi.org/10.1055/s-0038-1656514.

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Background and Importance Pituitary apoplexy (PA) occasionally occurs in patients with pituitary adenoma and may cause severe functional deficits. Headache, pituitary insufficiency, visual impairment, and cranial nerve palsies are the most frequent symptoms in patients with PA. Secondary cerebral ischemia develops in only a limited number of PA patients. Two pathogenic mechanisms were previously proposed. One states that ischemia may be due to major vessel encasement or to vessel compression, as a result of extended tumor growth. The second states that cerebral vasospasm following PA may cause

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Wang, Yang, Yongting Wang, and Guo-Yuan Yang. "MicroRNAs in Cerebral Ischemia." Stroke Research and Treatment 2013 (2013): 1–6. http://dx.doi.org/10.1155/2013/276540.

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The risk of ischemic stroke increases substantially with age, making it the third leading cause of death and the leading cause of long-term disability in the world. Numerous studies demonstrated that genes, RNAs, and proteins are involved in the occurrence and development of stroke. Current studies found that microRNAs (miRNAs or miRs) are also closely related to the pathological process of stroke. miRNAs are a group of short, noncoding RNA molecules playing important role in posttranscriptional regulation of gene expression and they have emerged as regulators of ischemic preconditioning and i

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Simard, J. Marc, Kevin N. Sheth, W. Taylor Kimberly, et al. "Glibenclamide in Cerebral Ischemia and Stroke." Neurocritical Care 20, no. 2 (2013): 319–33. http://dx.doi.org/10.1007/s12028-013-9923-1.

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Dissertations / Theses on the topic "Cerebral ischemia stroke apoplexy"

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Karelina, Ekaterina. "MECHANISMS OF SOCIAL NEUROPROTECTION AFTER CEREBRAL ISCHEMIA." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1274922479.

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Pogoryelova, Oksana. "A study of epigenetics in ischaemic stroke." Thesis, University of Aberdeen, 2013. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=201969.

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Ischaemic stroke rates are expected to rise significantly in the next decades due to an aging population. This increases the demand for new stroke biomarkers for early detection of patients at risk and new targets for treatment. It has been hypothesized that epigenetics may be important in the aetiology of stroke. The study consisted of three types of investigation: analysis of candidate gene polymorphism, candidate gene methylation analysis and epigenome-wide methylation analysis (EWAS) of pooled stroke and control samples. The stroke types studied were large vessel disease (LVD), small vesse

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Chan, Chu-fung. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B40687284.

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Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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Raghavan, Aparna. "Neuroprotective Potential of Withania Somnifera in Cerebral Ischemia." University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1416570371.

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Slack, Penelope Jean. "Dietary n-3 fatty acids and cerebral ischemia/reperfusion." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/3992.

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Many populations have low intakes of n-3 fatty acids, yet there is substantial evidence that the long chain n-3 fatty acid docosahexaenoic acid (DHA; 22:6n-3), found at high concentrations in the brain, is required for the proper development of the nervous system. However, less is known about requirements of long chain n-3 fatty acids for maintenance and function of the nervous system in later life. Several recent studies have reported that high amounts of long chain n-3 fatty acids reduce the extent of brain damage caused by cerebral ischemia in animals. However, whether or not a dietary defi

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Kostulas, Nikolaos. "Studies on cytokines and chemokines in cerebrovascular diseases and experimental cerebral ischemia /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4701-5/.

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Adhami, Faisal. "Differential Adult and Neonatal Response to Cerebral Ischemia-Hypoxia." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1196054266.

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Chaparro-Buitrago, Rafael Eduardo. "Neuroprotection with Anesthetics in Two Models of Cerebral Ischemia." Scholar Commons, 2010. http://scholarcommons.usf.edu/etd/3521.

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Neuroprotection with anesthetics has been studied for many decades; important advances in this field have modified the way Anesthesiologists treat patients in the operating room. Animal models have played an important role in the study of ischemia in the operating room. Recent studies have demonstrated that the effect of anesthetics seems to be different in different animal models. We decided to evaluate anesthetics in a well-known model of cerebral ischemia and also in hypotensive models designed by us. We used a model of cerebral ischemia (MCAO) to test anesthetics neuroprotective effect in

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Mokhberi, Shiva. "ELECTRICAL BIOIMPEDANCE CEREBRAL MONITORING : A Study of Cerebral Impedance Variation." Thesis, KTH, Skolan för teknik och hälsa (STH), 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-190876.

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Stroke is amongst the leading causes of death and disability worldwide. Today diagnosis of Stroke is restricted to fixed imaging facilities which do not provide a rapid diagnostic. A portable device which could provide a non invasive assessment of stroke would therefore decrease the time of diagnosis and increase the chance of survival. Recent studies have confirmed that Implementing Electrical Bioimpedance in a portable device could provide a reliable means for Stroke diagnostic. However in order to be able to use the brain impedance as an indicator of Stroke, the invariance of brain impedanc

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Books on the topic "Cerebral ischemia stroke apoplexy"

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United States. Agency for Health Care Policy and Research., ed. Extracranial-intracranial bypass to reduce the risk of ischemic stroke. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1990.

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2

Napoli, Mario Di. Neuroinflammation: An important role in the pathogenic pathways of cerebral ischemia. Nova Science Publishers, 2008.

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Hemorrhagic and ischemic stroke: Surgical, interventional, imaging, and medical approaches. Thieme Medical Publishers, 2011.

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Zoppo, Gregory J. Del. Innate inflammation and stroke. Edited by New York Academy of Sciences. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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Diagnostic Strategies In Cerebral Ischemia. Clinical Publishing Services, 2011.

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6

Gean, Alisa, and Pamela W. Schaefer. Stroke Imaging, an Issue of Neuroimaging Clinics. Elsevier - Health Sciences Division, 2011.

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7

Lin, Rick C. S., 1945-, ed. New concepts in cerebral ischemia. CRC Press, 2002.

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8

Y, Hsu Chung, ed. Ischemic stroke: From basic mechanisms to new drug development. Karger, 1998.

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9

N, Cohen Stanley, ed. Management of ischemic stroke. McGraw-Hill, Health Professions Division, 2000.

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10

Acute Ischemic Stroke: Imaging and Intervention. Springer, 2005.

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Book chapters on the topic "Cerebral ischemia stroke apoplexy"

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Ter Horst, Gert J., and Antonio Postigo. "Stroke." In Clinical Pharmacology of Cerebral Ischemia. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-59259-472-6_1.

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Todua, Fridon, and Dudana Gachechiladze. "Cerebral Ischemia and Stroke." In Noninvasive Radiologic Diagnosis of Extracranial Vascular Pathologies. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91367-4_8.

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Tatemichi, T. K., M. A. Foulkes, J. P. Mohr, et al. "Dementia in Stroke Survivors." In Cerebral Ischemia and Dementia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76208-6_4.

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Stevens, Henk, Hugo M. L. Jansen, Jacques DeReuck, and Jakob Korf. "Imaging of Stroke." In Clinical Pharmacology of Cerebral Ischemia. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-59259-472-6_2.

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Caplan, L. R. "Cerebral Ischemia and Infarction in Blacks." In Stroke in Blacks. KARGER, 1998. http://dx.doi.org/10.1159/000062916.

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Inzitari, D., G. Pracucci, M. Cadelo, M. Mascalchi, and L. Amaducci. "The Relation of Stroke to Leukoaraiosis." In Cerebral Ischemia and Dementia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76208-6_29.

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Kushner, M., C. Fieschi, M. Reivich, et al. "Focal and Remote Dysmetabolism in Stroke." In Cerebral Ischemia and Hemorheology. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_36.

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Obrenovitch, Tihomir P. "Glutamate Neurotoxicity and Stroke." In Clinical Pharmacology of Cerebral Ischemia. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-59259-472-6_5.

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Wu, K. K. "Defective Serum PGI2 Binding in Thrombotic Stroke: A Potential Mechanism for Platelet Hyperaggregability in Stroke." In Cerebral Ischemia and Hemorheology. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_39.

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Gelmers, H. J. "Calcium Blocker Therapy in Acute Ischemic Stroke." In Cerebral Ischemia and Calcium. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5_46.

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Conference papers on the topic "Cerebral ischemia stroke apoplexy"

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Гиганова, Анастасия Александровна, Татьяна Викторовна Ананьина, Алёна Андреевна Кисель, and Марина Юрьевна Ходанович. "MICROGLYOCYTES MORPHOLOGY AFTER FOCAL ISCHEMIC STROKE IN A MOUSE BRAIN." In Высокие технологии и инновации в науке: сборник избранных статей Международной научной конференции (Санкт-Петербург, Сентябрь 2020). Crossref, 2020. http://dx.doi.org/10.37539/vt187.2020.56.18.005.

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Проведен цитологический анализ изменения морфологии микроглиоцитов в мозге мышей, перенесших локальную ишемию головного мозга посредством окклюзии средней мозговой артерии (MCAO). Описана специфика изменения морфологии микроглиоцитов в белом и сером веществе головного мозга в ипсилатеральном полушарии. It was performed the cytological analysis of microglia morphology in mice underwent focal ischemia by means of middle cerebral artery occlusion (MCAO). The features of morphological microglyocyte changes in a white and gray matter of ipsilateral lesioned hemisphere was described.

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Strano, A., G. Davi, I. Catalano, G. Francavilla, and A. Notarbartolo. "IS BETATHROMBOGLOBULIN (BTG) OF PROGNOSTIC VALUE IN PATIENTS WITH TRANSIENT CEREBRAL ISCHEMIA (TIA) ?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643055.

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TIA and stroke have for a long time been recognized as being associated with various abnormalities in platelet function.Plasma levels of BTG, a marker of platelet release reaction, have been found raised in TIA patients. However plasma measurements can be influenced by venipuncture or handling of the samples Therefore we measured urinary BTG levels and compared plasma and urinary levels of this protein in 18 patients with TIA and 18 controls of equivalent age (41-68 years) and sex.Plasma BTG levels were not different between TIA subjects (24.1 ± 8.6 ng/ml) and controls (20.2 ± 6.4 ng/ml). Urin

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Northcutt, Avione, and Hai-Chao Han. "Finite Element Analysis of Buckling of Arteries With Aneurysms." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206275.

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Tortuosity of arteries occurs when a normally straight artery starts to take on a twisted path. This is often referred to as tortuosity and kinking of arteries. This phenomenon occurs in arteries throughout the body including the aorta and cerebral [1, 2]. Arterial tortuosity is a potentially dangerous condition that can lead to cerebrovascular symptoms, ischemia, and stroke [3].

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Coulson, Rebecca J., Marilyn J. Cipolla, Lisa Vitullo, and Naomi C. Chesler. "Mechanical Properties of Active and Passive Rat Middle Cerebral Arteries." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32508.

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Cerebral arteries play an important role in the regulation of cerebral blood flow through autoregulation, a well established phenomenon which is caused by a combination of myogenic, neuronal and metabolic mechanisms [1]. Myogenic reactivity is the ability of the vascular smooth muscle cells (SMC) to contract in response to stretch or to an increase in transmural pressure (TMP), and to dilate in response to a decrease in TMP [2]. It is this active constriction of arteries within the autoregulatory range that prompts studies of not just passive mechanical properties, but also active mechanical p

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Alessandri, C., F. Violi, M. Rasura, C. Caliendo, and P. Pelaia. "BEHAVIOUR OF ADREN0CHR0ME PATHWAY IN PATIENTS WITH CEREBROVASCULAR DISEASES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643169.

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Histopathological studies in segments of cerebral ischaemia show local inflammation with leucocytes infiltration.This event has been confirmed in vivo by means of radiolabelled leucocytes. This inflammatory response could be of detriment to cerebral tissue since leucocytes release toxic substances such as oxygen free radicals.A free radical mechanism,in fact,has been supposed as an event worsening the evolution of ischemia.Evidence of neutrophil activation in stroke patients was shown by us in previous reports, where we have described that the plasma of these patients contained an excess of a

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Attaluri, Anilchandra, Liang Zhu, and Zhongping Huang. "Targeted Brain Hypothermia Induced by an Interstitial Cooling Device in Human Neck: An Experimental Study." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205558.

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In recent years, mild or moderate hypothermia during which brain temperature is reduced to 30–35°C, has been proposed for clinical use as an adjunct for achieving protection from cerebral ischemia during cardiac bypass injury [Nussmeier 2002], carotid endarterectomy [Jamieson et al., 2003] and resection of extra-cranial aneurysm [Wagner and Zuccarello 2005], as well as stroke and traumatic brain injury [Marion et al., 1996; Marion 1997]. It has been shown that a reduction in brain temperature as small as 2°C substantially reduced ischemic cell damage [Clark et al., 1996], or improved significa

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