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Books on the topic 'Cerebral ischemia stroke apoplexy'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

United States. Agency for Health Care Policy and Research., ed. Extracranial-intracranial bypass to reduce the risk of ischemic stroke. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1990.

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2

Napoli, Mario Di. Neuroinflammation: An important role in the pathogenic pathways of cerebral ischemia. Nova Science Publishers, 2008.

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3

Hemorrhagic and ischemic stroke: Surgical, interventional, imaging, and medical approaches. Thieme Medical Publishers, 2011.

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4

Zoppo, Gregory J. Del. Innate inflammation and stroke. Edited by New York Academy of Sciences. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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5

Diagnostic Strategies In Cerebral Ischemia. Clinical Publishing Services, 2011.

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6

Gean, Alisa, and Pamela W. Schaefer. Stroke Imaging, an Issue of Neuroimaging Clinics. Elsevier - Health Sciences Division, 2011.

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7

Lin, Rick C. S., 1945-, ed. New concepts in cerebral ischemia. CRC Press, 2002.

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8

Y, Hsu Chung, ed. Ischemic stroke: From basic mechanisms to new drug development. Karger, 1998.

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9

N, Cohen Stanley, ed. Management of ischemic stroke. McGraw-Hill, Health Professions Division, 2000.

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10

Acute Ischemic Stroke: Imaging and Intervention. Springer, 2005.

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11

M, Roux Océane, ed. Brain hypoxia-ischemia research progress. Nova Biomedical Books, 2008.

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12

G, Somjen George, International Brain Research Organization. Congress, and Symposium on Cerebral Hypoxia and Stroke (1987 : Budapest, Hungary), eds. Mechanisms of cerebral hypoxia and stroke. Plenum Press, 1988.

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13

Kahn, Joseph H., Lauren M. Nentwich, and Brendan G. Magauran Jr. Acute Ischemic Stroke, an Issue of Emergency Medicine Clinics. Elsevier - Health Sciences Division, 2012.

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14

Ellis, Jason A., and E. Sander Connolly. Vascular Biology of Cerebral Ischemia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0107.

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Carotid stenosis may be treated by carotid endarterectomy (CEA) or carotid artery stenting (CAS). Moderate asymptomatic carotid stenosis (50%-70%) is associated with a low risk of ischemic stroke and does not warrant treatment. The severe stenosis (>70%) population sees a marginal benefit in seen with CEA. In the Asymptomatic Carotid Atherosclerosis Study, authors concluded that for patients with less than 60% stenosis, a 5.9% absolute risk reduction was obtained over 5 years with CEA compared with maximum medical management (11 vs. 5.1%).

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15

Dirnagl, Ulrich, and Bernd Elger. Neuroinflammation in Stroke. Springer, 2014.

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16

Greer, David M. Acute Ischemic Stroke: An Evidence-Based Approach. Wiley & Sons, Incorporated, John, 2008.

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17

(Editor), Sean I. Savitz, and Daniel M. Rosenbaum (Editor), eds. Stroke Recovery with Cellular Therapies (Current Clinical Neurology). Humana Press, 2007.

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18

1959-, Padovani Alessandro, and Pezzini Alessandro, eds. Cerebral ischemia in young adults: Pathogenic and clinical perspectives. Nova Science Publishers, 2009.

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19

M, Maier Carolina, and Steinberg Gary K, eds. Hypothermia and cerebral ischemia: Mechanisms and clinical applications. Humana Press, 2004.

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20

Hypothermia and cerebral ischemia: Mechanisms and clinical applications. Humana Press, 2003.

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21

M, Maier Carolina, and Steinberg Gary K, eds. Hypothermia and cerebral ischemia: Mechanisms and clinical applications. Humana Press, 2004.

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22

M, Maier Carolina, and Steinberg Gary K, eds. Hypothermia and cerebral ischemia: Mechanisms and clinical applications. Humana Press, 2004.

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23

(Editor), Carolina M. Maier, and Gary K. Steinberg (Editor), eds. Hypothermia and Cerebral Ischemia: Mechanisms and Clinical Applications. Humana Press, 2003.

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24

(Editor), Ulrich Dirnagl, and Bernd Elger (Editor), eds. Neuroinflammation in Stroke (Ernst Schering Research Foundation Workshop). Springer, 2004.

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25

Ch, Kessler, Rosengart Axel, and International Symposium on Hemostasis and Stroke (1993 : Lübeck, Germany), eds. Hemostasis and stroke. CRC Press, 1994.

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26

1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Birkhäuser, 2001.

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27

Acute ischemic stroke: An evidence-based approach. John Wiley & Sons, 2008.

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28

Acute Ischemic Stroke: An Evidence-based Approach. Wiley-Liss, 2007.

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29

Acute ischemic stroke: An evidence-based approach. Wiley-Liss, 2007.

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30

Eric, Kasner Scott, and Gorelick Philip B, eds. Prevention and treatment of ischemic stroke. Butterworth-Heinemann, 2004.

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31

Fieschi, Cesare, and Marc Fisher. Prevention of Ischemic Stroke. Informa Healthcare, 1999.

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32

Cesare, Fieschi, and Fisher Marc 1948-, eds. Prevention of ischemic stroke. Martin Dunitz, 2000.

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33

B, Goldstein Larry, ed. Restorative neurology: Advances in pharmacotherapy for recovery after stroke. Futura, 1998.

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34

Goldstein, Larry B. Restorative Neurology: Advances in Pharmacotherapy for Recovery After Stroke. Blackwell Publishing Limited, 1998.

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35

Felling, Ryan J. Targets for Neuroprotection in Ischemic Stroke. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0111.

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Cerebral ischemia or hypoxia-ischemia initiate a cascade of biochemical events including impaired reuptake of glutamate into perisynaptic glia causing glutamate flooding, calcium fluxing through NMDA glutamate channels, activation of neuronal nitric oxide synthetase, and impaired mitochondrial ATP production. In animal models it is possible to block these steps and protect the brain but the temporal window of protection after the insult lasts only a few hours. Recombinant TPA is clinically protective if given within 3 hours of stroke, but other agents have not been shown to protect brain tissu

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36

J, Del Zoppo Gregory, Mori Etsuro 1951-, and Hacke W. 1948-, eds. Thrombolytic therapy in acute ischemic stroke II. Springer-Verlag, 1993.

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37

Khouri, George, Shelly Ozark, and Bruce Ovbiagele. Common Risk factors for Stroke and Medical Prevention Therapies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0103.

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Stroke from thrombosis or emboli in cerebral vessels or hemorrhage is one of the most commonly encountered and most devastating neurological diseases. Rapid loss of function occurs due to an interruption of blood supply to the brain, leading to tissue ischemia and cell death. The risk of both ischemic and hemorrhagic stroke doubles for each successive decade after age 55, which is likely independent of other risk factors such as diabetes, hypertension, and hyperlipidemia. Lifestyle modifications, antiplatelet therapy and control of hyperlipidemia and hypertension are the mainstays of preventio

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38

Yang, Hui. Carotid Endarterectomy/Stenting. Edited by David E. Traul and Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0007.

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Carotid endarterectomy and carotid artery stenting have been performed worldwide to reduce neurologic symptoms and prevent stroke. Preoperative cardiovascular and neurologic evaluations are essential to optimize a patient for surgery and guide the intra- and postoperative management. The primary goals of anesthetic management are to maintain cerebral perfusion and prevent perioperative myocardial ischemia. A shunt may be placed to provide antegrade cerebral blood flow if signs of cerebral ischemia develop during carotid cross-clamping. An awake patient is the gold standard for monitoring the a

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39

Friedman, Deborah I., Shamin Masrour, and Susan Hutchinson. Headache. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0012.

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In most cases, women with headache disorders have normal pregnancy and delivery outcomes and should not be discouraged from becoming pregnant. Pre-pregnancy planning includes weaning of contraindicated medications. Most women with migraine without aura improve during pregnancy. Although there are limitations, various acute and preventive treatments may be employed, including non-pharmacologic options. Anti-epileptic medications should be avoided. For pseudotumor cerebri, the mainstay of treatment includes diuretics and therapeutic lumbar punctures, avoiding topiramate. Surgical treatment may b

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