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Relevant bibliographies by topics / Cerebral ischemia – Treatment / Dissertations / Theses

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Dissertations / Theses on the topic 'Cerebral ischemia – Treatment'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

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1

Yang, Di, and 楊荻. "Neuroprotective effects of lycium barbarum extracts in cerebral and retinal ischemia/reperfusion injury." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206738.

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Ischemic stroke is a devastating cerebrovascular disease resulting in high mortality rate and distressing sequelae such as hemiplegia, ataxia and even visual impairment. Retinal ischemia refers to a common pathological feature shared by many blinding diseases including retinal vascular occlusions, diabetic retinopathy, glaucoma, and retinopathy of prematurity. Ischemia/reperfusion injury is implicated in both of these pathological conditions, which greatly impact on one’s daily life. The eventual consequence of the insult is irreversible neuronal cell death and functional deterioration. Apart

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2

Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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3

Xu, Mingjing, and 徐明婧. "Baicalin protects neural cells from cerebral ischemia reperfusion injury by scavenging peroxynitrite." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47753110.

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Ischemic stroke is the leading cause of death and disability in human diseases all around the world. As effective treatment for ischemic stroke is still absent, seeking for new therapy is of great interest. Currently, several key pathological cascades following cerebral ischemia have been explored to develop further therapies. Among them, reactive nitrogen species (RNS) has been indicated to play a critical role in cerebral ischemia reperfusion injury. As one of the RNS, peroxynitrite contributes to the neural cell death and subsequent brain dysfunction in the process. Thus, development of

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4

Khanasari, Parto S. "An investigation of the neuroprotective properties of fenamate NSAIDs, against experimental model of ischemic stroke." Scholarly Commons, 2007. https://scholarlycommons.pacific.edu/uop_etds/671.

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Stroke is a devastating neurological disease with limited treatment opportunities. Recent advances in understanding the underlying pathogenesis of cerebral ischemia support the involvement of multiple biochemical pathways in the development of the ischemic injury. The work reported in this thesis was undertaken to investigate the hypothesis that fenamate NSAIDs have neuroprotective properties against ischemic stroke and to explore the underlying mechanisms for any efficacy. Fenamates are non-selective inhibitors of cyclooxygenases. In addition, fenamates are antagonists of non-selective cation

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5

Weiss, Miriam [Verfasser], Gerrit Alexander [Akademischer Betreuer] Schubert, and Martin [Akademischer Betreuer] Wiesmann. "Endovascular rescue treatment for delayed cerebral ischemia after subarachnoid hemorrhage is safe and effective / Miriam Weiss ; Gerrit Alexander Schubert, Martin Wiesmann." Aachen : Universitätsbibliothek der RWTH Aachen, 2019. http://d-nb.info/1215927614/34.

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6

Boulos, Sherif. "Identification and characterisation of potential neuroprotective proteins induced by erythropoietin (EPO) preconditioning of cortical neuronal cultures." University of Western Australia. School of Biomedical and Chemical Sciences, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0128.

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[Truncated abstract] Clinical therapeutic agents to directly inhibit ischaemic neuronal death are presently unavailable. One approach to developing therapeutics is based upon the identification of proteins up-regulated by 'preconditioning', a natural adaptive response utilised by the neural cells to counter damaging insults, such as ischaemia. Thus, my project aimed to firstly identify proteins differentially expressed following erythropoietin (EPO) mediated neuronal preconditioning and secondly to assess whether any of these proteins possessed neuroprotective activity using in vitro ischaemia

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7

Rahman, Rosanna, and n/a. "Potential causes of the delayed neural damage observed post-stroke & the effects of epigallocatechin gallate administration." University of Otago. Department of Pharmacology & Toxicology, 2006. http://adt.otago.ac.nz./public/adt-NZDU20070508.122246.

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Stroke is the 3rd leading cause of death and the leading cause of major disability worldwide. Currently, there are no neuroprotective drugs approved for the acute treatment of ischaemic stroke. The vast majority of stroke therapeutics failed in clinical trials due to toxic side effects and/or a clinically irrelevant therapeutic window. This thesis is focused on exploiting the delayed neurodegeneration that occurs in the compromised penumbra, as these cells may be capable of being saved by therapeutic intervention in a clinically obtainable window. In order to investigate the ischaemic cascade

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8

Pütz, Volker, Matthias Weise, Rüdiger von Kummer, and Georg Gahn. "Effective Treatment with Abciximab for Consecutive Bilateral Middle Cerebral Artery Occlusion." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135290.

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9

Pütz, Volker, Matthias Weise, Rüdiger von Kummer, and Georg Gahn. "Effective Treatment with Abciximab for Consecutive Bilateral Middle Cerebral Artery Occlusion." Karger, 2006. https://tud.qucosa.de/id/qucosa%3A27636.

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10

Brodin, Camille. "De la paillasse au lit du patient, surmonter les problèmes de translation dans le domaine de l'AVC ischémique Single- and two- chain tissue plasminogen activator treatment differentially influence cerebral recovery after stroke Single- and two- chain tissue plasminogen activator treatment differentially influence cerebral recovery after stroke Cerebral blood flow correlates with ischemic brain lesion only when Stroke occurs awake: a preclinical model to bypass the translational roadblocks to clinic." Thesis, Normandie, 2019. http://www.theses.fr/2019NORMC427.

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Les défauts de translation des études précliniques vers les essais cliniques dans le domaine des AVC ischémiques et l'échec des développements thérapeutiques pourraient être expliqués par trois aspects : (1) le manque de compréhension des mécanismes des deux formes de rtPA, le traitement pharmacologique de l'AVC; (2) le manque d'outils adaptés d'imagerie de perfusion chez le petit animal et (3) l'influence de l'anesthésie sur l’effet des traitements testés dans les modèles animaux.Le tPA utilisé en clinique (Actilyse®) est un mélange de deux formes de tPA: une forme chaîne simple (sc-rtPA) et

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11

Poignet, Hervé. "Activites pharmacologiques des antagonistes du calcium sur differents modeles physiopathologiques utilises dans l'ischemie cerebrale experimentale : effets sur les atteintes fonctionnelles et neuronales." Clermont-Ferrand 2, 1988. http://www.theses.fr/1988CLF21111.

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12

Huang, Chien-Chih, and 黃千芝. "Delayed Treatment with Cinnamophilin Improves Electrophysiological and Neurobehavioral Outcomes in Rats Subjected to Transient Focal Cerebral Ischemia." Thesis, 2006. http://ndltd.ncl.edu.tw/handle/51816525239392927642.

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碩士<br>國立成功大學<br>醫學工程研究所碩博士班<br>94<br>The neuroprotective properties of cinnamophilin, a strong natural antioxidant and free radical scavenger which may enhance cerebral hemodynamics, decrease platelet aggregation and lipid peroxidation, and prevent energy depletion during ischemia, will be evaluated in Sprague-Dawley rats subjected to 1-hour transient middle cerebral artery (MCA) occlusion. A series of experiments with delayed treatment of CINN was employed to examine whether CINN is neuroprotective following a short period of recovery using a model of reversible MCA occlusion which includes i

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13

Chen, Li-Hsuan, and 陳俐璇. "Delayed Treatment with Magnesium Improves Electrophysiological and Neurobehavioral Outcomes in Rats Subjected to Transient Focal Cerebral Ischemia." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/90184549099176769182.

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碩士<br>國立成功大學<br>醫學工程研究所<br>93<br>In the current study, magnesium sulfate (750μmol/kg) was given via intracarotid infusion at the beginning of reperfusion in a rat model of transient focal cerebral ischemia. Much evidence implicated neuroprotective mechanisms of Mg2+ to include inhibition of excitatory neurotransmitter release, blockade of voltage-dependent NMDA channels, and blockade of L- and N-type voltage-operated calcium channels. In addition to reduced brain infarction, this study emphasized the importance of electrophysiological and neurobehavioral outcomes. Improvement of functional out

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14

Tsai, Yi-Yin, and 蔡宜殷. "Delayed Treatment with Melatonin Improves Electrophysiological and Neurobehavioral Outcomes in Rats Subjected to Transient Focal Cerebral Ischemia." Thesis, 2003. http://ndltd.ncl.edu.tw/handle/43528059838035888218.

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碩士<br>國立成功大學<br>醫學工程研究所<br>91<br>In general, cerebral ischemia elicits calcium accumulation and production of free radicals leading to irreversible cell damage and neurobehavioral dysfunction. Thus an effective neuroprotective agent is needed to protect ischemic brain after thrombolytic therapies. Melatonin is well known a direct free radical scavenger and an indirect antioxidant, and was reported to reduce infarct volume recently. In the current study, we investigate weather delayed treatment with melatonin (5mg/kg iv) could improve electrophysiology and neurobehavioral function in

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15

"Diagnosis, microemboli detection and hemodynamic monitoring of intracranial atherosclerosis by transcranial Doppler in the ischemic stroke." Thesis, 2008. http://library.cuhk.edu.hk/record=b6074600.

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Early deterioration and long-term recurrence were common after stroke or transient ischemic attach (TIA), however, it is unclear whether they were correlated with active embolization and the consequent new cerebral infarct in acute phase. By employing TCD and diffusion weighted imaging (DWI), we studied the significance of the progression of MES and infarcts during acute phase on the clinical outcomes. We found that the disappearance of MES was correlated with better improvement on day 7 of recruitment; for the long-term outcome, occurrence of exacerbating infarct tended to predict recurrent s

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16

Hsieh, Fu-Shun, and 謝復順. "Transplantation of low-energy laser stimulation on adipose tissue-derived stem cells treatment of focal cerebral ischemia in rats." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/38450605186460127492.

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碩士<br>中臺科技大學<br>醫學影像暨放射科學系暨研究所<br>100<br>This study investigated the influence of a large-area irradiation of low-level laser on the proliferation and differentiation of inductions of adipose-derived stem cells (i-ADSCs) into nerve cells. We then applied this approach to treat ischemic stroke in rats in the evaluation of various treatment mechanisms. DAPI staining showed that the number of cells did not increase as a result of irradiation; MTT assays indicated no significant difference between the number of laser-treated and non-laser-treated cells. Our results demonstrate that wide-area irrad

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17

"External counterpulsation (ECP): a new, non-invasive method to enhance cerebral blood flow and its application in ischemic stroke." Thesis, 2007. http://library.cuhk.edu.hk/record=b6074448.

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Han, Jinghao.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2007.<br>Includes bibliographical references (p. 182-204).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Abstracts in English and Chinese.

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18

Turner, Renée Jade. "Characterising the role of substance P in acute ischaemic stroke." 2007. http://hdl.handle.net/2440/56839.

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More than 15 million people worldwide will suffer a stroke each year two thirds will die or be left permanently disabled. Accordingly, stroke represents an enormous financial burden on the community, due to the cost of hospitalisation, treatment and rehabilitation of stroke patients. Despite the significance of this public health problem, a safe and widely applicable stroke therapeutic remains elusive. Cerebral oedema is widely recognised as a common and often fatal complication of stroke that is associated with worsened outcome. However, the exact mechanisms of oedema formation remain unclear

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19

Turner, Renée Jade. "Characterising the role of substance P in acute ischaemic stroke." Thesis, 2007. http://hdl.handle.net/2440/56839.

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More than 15 million people worldwide will suffer a stroke each year two thirds will die or be left permanently disabled. Accordingly, stroke represents an enormous financial burden on the community, due to the cost of hospitalisation, treatment and rehabilitation of stroke patients. Despite the significance of this public health problem, a safe and widely applicable stroke therapeutic remains elusive. Cerebral oedema is widely recognised as a common and often fatal complication of stroke that is associated with worsened outcome. However, the exact mechanisms of oedema formation remain unclear

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20

"The hemodynamic effects of external counterpulsation in patients with recent stroke." Thesis, 2011. http://library.cuhk.edu.hk/record=b6075504.

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Lin, Wenhua.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2011.<br>Includes bibliographical references (leaves 162-190).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Abstract also in Chinese.

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21

Chang, Chia-Yu, and 張嘉祐. "Therapeutic Treatment with Ascorbate Rescues Mice from Heat Stroke-induced Death and Reduces Focal Cerebral Ischemia-induced Brain Infarction in Rats." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/aj93gy.

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博士<br>國立中興大學<br>食品暨應用生物科技學系所<br>105<br>The impact of ascorbate (vitamin C, ascorbic acid) on oxidative stress-related diseases is moderate because of its limited oral bioavailability and rapid clearance. However, parenteral administration can increase the benefit of ascorbate supplementation as is evident in critically ill patients. Heatstroke is defined as a form of excessive hyperthermia associated with a systemic inflammatory response that results in multiple organ dysfunctions in which central nervous system disorders such as delirium, convulsions, and coma are predominant. The thermoreg

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22

Liao, Wen-Yen, and 廖文彥. "Evaluating the role of Toll-like receptors 2 and 4 in treatment of Moutan Cortex Radicis and Paeonol on cerebral ischemia-reperfusion injured rat." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/j2e3mr.

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23

"In vivo and in vitro studies of the anti-oxidative, anti-inflammatory and anti-apoptotic effects of Gastrodiae Rhizoma water extract on ischemic stroke." 2013. http://library.cuhk.edu.hk/record=b5884430.

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Hung, Sze Man.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2013.<br>Includes bibliographical references (leaves 186-192).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Abstracts also in Chinese.

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24

"Potential of serotonin in stem cell technology and therapy in a mouse ischemic stroke model." 2012. http://library.cuhk.edu.hk/record=b5549580.

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Serotonin (5-hydroxytryptamine, 5-HT) is a neurotransmitter involved in the embryonic neural development and adult neurogenesis. But the effects of 5-HT on stem cells are not fully known. In this study, the effects and underlying signal pathways of 5- HT on proliferation and neural differentiation of mouse embryonic stem (ES) cells, neural progenitor (NP) cell line C 17.2 and embryonic neural stem (NS) cells were explored. Molecular analysis, immunostaining and western blotting revealed that NP/NB cells expressed the rate-limiting enzyme tryptophan hydroxylase (TPH) and produced endogenous 5-H

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25

"Baicalin-mediated neuronal induction of neural stem cells and improvement of cognitive function in a mouse stroke model." Thesis, 2009. http://library.cuhk.edu.hk/record=b6074973.

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Baicalin, which is a flavonoid, was previously shown to exert neuroprotective effects against ischemic injury and oxidative insults. In this study, baicalin was found to induce neuronal differentiation on both C17.2 NSC and primary mouse NSC originated from hippocampuses of E14.5 mouse embryos. The baicalin-mediated differentiation of C17.2 NSC was noted in dose- and time-dependent manners. Baicalin-treated NSC displayed long processes of neurites. The gene expression of neuronal markers, NF-L, TUBB3 and MAP2 was also significantly increased after treated with 20 to 50 muM baicalin on C17.2 NS

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26

"The effects of hematopoietic growth factors and tanshinone IIA on neuro-protection." Thesis, 2005. http://library.cuhk.edu.hk/record=b6073980.

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Neonatal hypoxic-ischemic encephalopathy (HIE) is a common clinical problem. Tanshinone IIA is a compound purified from the Chinese herb Danshen ( Radix Salviae Miltiorrhiza Bge). Thrombopoietin (TPO) and Erythropoietin (Epo) are hematopoietic growth factors. The effects of tanshinone IIA, EPO and TPO on hypoxia-ischemia brain injury were investigated in this study, using in vitro model of neural cell culture and an in vivo model of hypoxic-ischemic brain damage.<br>Our observation provided the first evidence showing the expression of functional TPO receptor c-mpl in central nervous system. It

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27

Chen, Chun-Hao, and 陳君豪. "Antioxidant Defenses of Astrocytes Activation after Hyperbaric Oxygen Treatment in Transient Focal Cerebral Ischemic Rats." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/78103140084657078491.

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碩士<br>國立陽明大學<br>物理治療學系暨研究所<br>95<br>Background and Purpose: Brain following transient focal cerebral ischemia develops a complex series of pathophysiological events. As a consequence of ischemia and subsequent reperfusion, the reactive free radicals react with cellular components (carbohydrates, amino acids, DNA, and phospholipids) and damage them. Hyperbaric oxygenation (HBO) has a characteristic in dramatically increasing the oxygen content of blood by physically dissolving oxygen and theoretically penetrates to deeper and poorly perfused ischemic areas. Thus, HBO appears to be a neuroprotec

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