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Relevant bibliographies by topics / Cerebral ischemia / Dissertations / Theses

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Dissertations / Theses on the topic 'Cerebral ischemia'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Keasey, Matthew P. "MicroRNAs in Cerebral Ischemia." Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526014.

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2

Molnar, Maria. "Hyperglycemia in Experimental Cerebral Ischemia." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-247763.

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Cerebral ischemia is a life-threatening condition associated with a substantial morbidity and mortality. Hyperglycemia, a common coexisting phenomenon in both stroke and cardiac arrest (CA), may further aggravate ischemic brain injury. To date, the therapeutic possibilities are lim-ited and the search for new treatment modalities is warranted. One aspect of such a research could be to better understand the cerebral pathogenesis induced by hyperglycemic ischemia-reperfusion. We investigated the combination of ischemia and hyperglycemia in two experimental models of stroke and CA. The aims were

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3

Christensen, Thomas. "Experimental focal cerebral ischemia : pathophysiology, metabolism and pharmacology of the ischemic penumbra /." Copenhagen, 2007. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=016143698&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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4

Thorén, Anna. "Astrocyte metabolism following focal cerebral ischemia /." Göteborg : Institute of Neuroscience and Physiology, The Sahlgrenska Academy, Göteborg University, 2006. http://hdl.handle.net/2077/744.

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5

Thomas, Sunu Samuel. "Murine models of cerebral ischemia, development of a mouse model of global cerebral ischemia; response of GluR2 knockout mice in a model of permanent focal cerebral ischemia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0026/MQ50439.pdf.

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6

Li, Yan. "Inhibitory synpatic transmission in striatal neurons after transient cerebral ischemia." Connect to resource online, 2009. http://hdl.handle.net/1805/2021.

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Thesis (Ph.D.)--Indiana University, 2009.<br>Title from screen (viewed on December 1, 2009). Department of Anatomy and Cell Biology, Indiana University-Purdue University Indianapolis (IUPUI). Advisor(s): Zao C. Xu, Feng C. Zhou, Charles R. Yang, Theodore R. Cummins. Includes vitae. Includes bibliographical references (leaves 115-135).

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7

Karelina, Ekaterina. "MECHANISMS OF SOCIAL NEUROPROTECTION AFTER CEREBRAL ISCHEMIA." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1274922479.

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8

Adhami, Faisal. "Differential Adult and Neonatal Response to Cerebral Ischemia-Hypoxia." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1196054266.

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9

Edrissi, Hamidreza. "Blood Brain Barrier Dysfunction in Chronic Cerebral Ischemia." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/32531.

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Cerebral small vessel pathology is now known to be associated with the development of cognitive impairment and mild motor impairments such as gait disturbance in a variety of neurodegenerative diseases. This dissertation explores the hypothesis that blood brain barrier dysfunction is an early event in cerebral ischemia and contributes to the development of cerebral small vessel disease (CSVD). A common rodent model of CSVD is permanent bilateral common carotid artery occlusion in the rat. This model was used to study several aspects of the progression of CSVD including the timecourse of blood

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10

Ng, Kit-ying. "Neuroprotective effects of adiponectin in focal cerebral ischemia." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B39634371.

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11

Bogart, Robert William. "The effect of stress on global cerebral ischemia." Connect to resource, 2008. http://hdl.handle.net/1811/32235.

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12

Ng, Kit-ying, and 吳潔瑩. "Neuroprotective effects of adiponectin in focal cerebral ischemia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39634371.

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13

Raghavan, Aparna. "Neuroprotective Potential of Withania Somnifera in Cerebral Ischemia." University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1416570371.

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14

Schatlo, Bawarjan [Verfasser]. "Cerebral ischemia in experimental subarachnoid hemorrhage / Bawarjan Schatlo." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2011. http://d-nb.info/1026265347/34.

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15

Xu, Xingshun. "Novel Protective Agents against Cerebral Ischemia/Reperfusion Injury." Digital Commons @ East Tennessee State University, 2007. https://dc.etsu.edu/etd/2054.

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Stroke is the third leading cause of death and disability in the United States. At present, intravenous administration of tissue plasminogen activator (t-PA) is the only thrombolytic therapy approved by the FDA for the treatment of acute ischemic stroke. There are no other effective treatments available so far. The discovery of new drugs and new treatments for stroke to reduce mortality and disability is an urgent medical research priority. In this study, the protective effects and mechanisms of two novel agents Gly14 humanin (HNG) and necrostatin-1 (Nec-1) were examined. HNG, a highly potent

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16

Dowden, Jennifer. "Characterizing the neuroprotective efficacy of ischemic preconditioning (ischemic tolerance) : is age an important factor? /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0019/NQ54834.pdf.

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17

Wang, Yanxin. "Hypoxic-ischemic injury in the neonatal rat model prediction of irreversible infarction size by Diffusion Weighted MR Imaging /." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B35757577.

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18

Frykholm, Peter. "Cerebral ischemia studied with positron emission tomography and microdialysis." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2002. http://publications.uu.se/theses/91-554-5319-8/.

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19

Slack, Penelope Jean. "Dietary n-3 fatty acids and cerebral ischemia/reperfusion." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/3992.

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Many populations have low intakes of n-3 fatty acids, yet there is substantial evidence that the long chain n-3 fatty acid docosahexaenoic acid (DHA; 22:6n-3), found at high concentrations in the brain, is required for the proper development of the nervous system. However, less is known about requirements of long chain n-3 fatty acids for maintenance and function of the nervous system in later life. Several recent studies have reported that high amounts of long chain n-3 fatty acids reduce the extent of brain damage caused by cerebral ischemia in animals. However, whether or not a dietary defi

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20

Chaparro-Buitrago, Rafael Eduardo. "Neuroprotection with Anesthetics in Two Models of Cerebral Ischemia." Scholar Commons, 2010. http://scholarcommons.usf.edu/etd/3521.

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Neuroprotection with anesthetics has been studied for many decades; important advances in this field have modified the way Anesthesiologists treat patients in the operating room. Animal models have played an important role in the study of ischemia in the operating room. Recent studies have demonstrated that the effect of anesthetics seems to be different in different animal models. We decided to evaluate anesthetics in a well-known model of cerebral ischemia and also in hypotensive models designed by us. We used a model of cerebral ischemia (MCAO) to test anesthetics neuroprotective effect in

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21

Schweizer, Sophie [Verfasser]. "Histone methylation and neuroprotection in cerebral ischemia / Sophie Schweizer." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2015. http://d-nb.info/1076270808/34.

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22

Blazej, Katja [Verfasser]. "Role of inflammatory cells in cerebral ischemia / Katja Blazej." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2013. http://d-nb.info/1031097007/34.

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23

Ward, Nicholas M. "The assessment of behavioural deficits following focal cerebral ischemia." Thesis, University of St Andrews, 1997. http://hdl.handle.net/10023/14698.

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Evaluating the efficacy of neuroprotective drugs in rat models of focal cerebral ischemia has involved histological and behavioural batteries to examine pathology and sensorimotor function. However, the behavioural tests used provide little insight into the nature of the neurological impairments. In an effort to gain further insight into the behavioural impairment following ischemic lesions, a battery of tasks were used. The tasks included tests of sensorimotor, motor (paw use), motivation, sensory and attentional function. The use of the potent vasoconstrictor endothelin-1 has allowed cerebra

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24

Chan, Chu-fung. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B40687284.

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25

Gisselsson, Lars. "Ischemic brain damage the influence of hyperglycemia on tissue injury, cerebral circulation and edema formation /." Lund : Lund University, 1998. http://books.google.com/books?id=gMdrAAAAMAAJ.

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26

Li, Ping-An. "Mechanisms of acidosis-mediated ischemic brain damage histopathology and pathophysiology /." Lund : Lund University, 1996. http://catalog.hathitrust.org/api/volumes/oclc/38158955.html.

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27

Pereira, Claudia Figueiredo. "Carbon monoxide, autophagy and cytoprotection in response to cerebral ischemia." Master's thesis, Faculdade de Ciências e Tecnologia, 2013. http://hdl.handle.net/10362/10836.

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Dissertação para obtenção do Grau de Mestre em Genética Molecular e Biomedicina<br>There is an increasing need for promoting neuroprotection against cerebral ischemia, which is the main cause of brain damage in adults. Astrocytes are the most abundant cells inboard the central nervous system (CNS), being known as key glial cell for promoting neuronal survival and homeostasis. It is more established in nowdays that astrocytic dysfunction contributes to neurodegenerative processes. Although, carbon monoxide is a well renown as a lethal and toxic gas due to its high affinity to hemoglobin, CO ex

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28

BATTISTELLO, ENRICA. "PEMFs and cerebral ischemia: the pathways behind their beneficial effects." Doctoral thesis, Università degli studi di Ferrara, 2021. http://hdl.handle.net/11392/2488048.

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Low‐energy low‐frequency pulsed electromagnetic fields (PEMFs) exert numerous beneficial effects on different biological systems. In particular, several data have reported their neuroprotective role in cerebral ischemia, a disease that is a leading cause of mortality and disability worldwide. For these reasons, PEMFs treatment has been introduced in a clinical multicentre, prospective, randomized, placebo‐controlled, and double‐blind study, whose aim is to investigate the effectiveness of PEMFs in acute ischemic stroke, as non-invasive and safe tool to gain neuronal recovery. However, to date,

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29

Mokhberi, Shiva. "ELECTRICAL BIOIMPEDANCE CEREBRAL MONITORING : A Study of Cerebral Impedance Variation." Thesis, KTH, Skolan för teknik och hälsa (STH), 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-190876.

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Stroke is amongst the leading causes of death and disability worldwide. Today diagnosis of Stroke is restricted to fixed imaging facilities which do not provide a rapid diagnostic. A portable device which could provide a non invasive assessment of stroke would therefore decrease the time of diagnosis and increase the chance of survival. Recent studies have confirmed that Implementing Electrical Bioimpedance in a portable device could provide a reliable means for Stroke diagnostic. However in order to be able to use the brain impedance as an indicator of Stroke, the invariance of brain impedanc

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30

Chen, Xiaoqian. "The identification of 14-3-3[gamma] in astrocytes and its mechanism in protecting astrocytes from ischemia /." View Abstract or Full-Text, 2002. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202002%20CHEN.

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Thesis (Ph. D.)--Hong Kong University of Science and Technology, 2002.<br>Includes bibliographical references (leaves 180-202). Also available in electronic version. Access restricted to campus users.

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31

Silva, Matthew S. "NMR characterization of changes in the apparent diffusion coefficient of water following transient cerebral ischemia." Link to electronic thesis, 2002. http://www.wpi.edu/Pubs/ETD/Available/etd-0327102-221251.

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32

Tsang, Hing-wai, and 曾慶威. "In vitro studies of hypoxic ischemic down-regulated 1 (HID-1) protein encoded by a novel gene down-regulated in neonatal hypoxic-ischemicencephalopathy in different cell death paradigms." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45608192.

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33

Zou, Liangyu. "Role of cerebral ischemia in cognitive impairment clinical and experimental study /." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B32019889.

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34

Davis, Stephanie. "Leukemia Inhibitory Factor as a Neuroprotective Agent against Focal Cerebral Ischemia." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6218.

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Previous publications from this laboratory demonstrated that administration of leukemia inhibitory factor (LIF) (125 µg/kg) to young, male Sprague-Dawley rats at 6, 24, and 48 h after middle cerebral artery occlusion (MCAO) reduced infract volume, improved sensimotor skills, and alleviated damage to white matter at 72 h after the injury. In vitro studies using cultured oligodendrocytes (OLs) showed that LIF (200 ng/ml) also protects against 24 h of oxygen-glucose deprivation through activation of Akt signaling and upregulation of the antioxidant enzymes peroxiredoxin IV and metallothionein III

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35

Zou, Liangyu, and 鄒良玉. "Role of cerebral ischemia in cognitive impairment: clinical and experimental study." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B32019889.

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36

Vasylieva, N. V. "Chronic cerebral ischemia and cognitive impairment (an effect of complex therapy)." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/17642.

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37

Gaudier-Diaz, Monica M. "Sex-Specific Social Modulation of the Neuroinflammatory Response toGlobal Cerebral Ischemia." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1490713683841079.

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38

Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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39

Pogoryelova, Oksana. "A study of epigenetics in ischaemic stroke." Thesis, University of Aberdeen, 2013. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=201969.

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Ischaemic stroke rates are expected to rise significantly in the next decades due to an aging population. This increases the demand for new stroke biomarkers for early detection of patients at risk and new targets for treatment. It has been hypothesized that epigenetics may be important in the aetiology of stroke. The study consisted of three types of investigation: analysis of candidate gene polymorphism, candidate gene methylation analysis and epigenome-wide methylation analysis (EWAS) of pooled stroke and control samples. The stroke types studied were large vessel disease (LVD), small vesse

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40

Pennings, Frederik Anthonius. "The clinical value of assessing cerebral ischemia: emphasis on brain tissue oxygenation and function of the cerebral microcirculation." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2007. http://dare.uva.nl/document/45158.

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41

MARIANI, JACOPO. "MULTICENTRE AND MULTISPECIES PRECLINICAL TRIAL OF REMOTE ISCHEMIC CONDITIONING IN ANIMAL MODEL OF ACUTE ISCHEMIC STROKE (TRICS–BASIC)." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2023. https://hdl.handle.net/10281/403043.

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Il condizionamento ischemico remoto (RIC) risulta essere un candidato ideale per essere investigato in uno studio multicentrico volto al trattamento dell'ictus ischemico acuto (AIS). L’ efficacia terapeutica del RIC è stata dimostrata a livello preclinico, come riportato da precedenti studi ottenuti da singoli laboratori; ciò nonostante, gli studi clinici di fase II-III non hanno ancora fornito risultati soddisfacenti. TRICS Basic è definita come la fase preclinica del progetto TRICS, un Trial traslazionale multicentrico di Condizionamento Ischemico Remoto in modelli animali di Ictus Ischemic

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42

Li, Han-Dong, Minshu Li, Elaine Shi, et al. "A translocator protein 18 kDa agonist protects against cerebral ischemia/reperfusion injury." BIOMED CENTRAL LTD, 2017. http://hdl.handle.net/10150/625392.

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Background: Cerebral ischemia is a leading cause of death and disability with limited treatment options. Although inflammatory and immune responses participate in ischemic brain injury, the molecular regulators of neuroinflammation after ischemia remain to be defined. Translocator protein 18 kDa (TSPO) mainly localized to the mitochondrial outer membrane is predominantly expressed in glia within the central nervous system during inflammatory conditions. This study investigated the effect of a TSPO agonist, etifoxine, on neuroinflammation and brain injury after ischemia/reperfusion. Methods: We

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43

Sidhu, Ranjinder S. "Role of apoptosis following cerebral hypoxia-ischemia in immature and older rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0014/MQ32247.pdf.

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44

Kostulas, Nikolaos. "Studies on cytokines and chemokines in cerebrovascular diseases and experimental cerebral ischemia /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4701-5/.

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45

Zur, Nedden Stephanie. "Targeting the purine salvage pathway in in vitro models of cerebral ischemia." Thesis, University of Warwick, 2011. http://wrap.warwick.ac.uk/45926/.

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An interruption of the blood supply to the brain, as occurs during ischemic stroke, results in a rapid decline of ATP levels and a subsequent loss of neuronal function and viability. Under physiological conditions the brain reuses ATP degradation metabolites, such as hypoxanthine, via the purine salvage pathway, to restore its ATP pool. However, the massive degradation of ATP during ischemia results in the accumulation and loss of diffusible purine metabolites and thereby leads to a reduction in the post-ischemic ATP pool size, leaving the brain more vulnerable to secondary ischemic insults (r

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46

Xu, Mingjing, and 徐明婧. "Baicalin protects neural cells from cerebral ischemia reperfusion injury by scavenging peroxynitrite." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47753110.

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Ischemic stroke is the leading cause of death and disability in human diseases all around the world. As effective treatment for ischemic stroke is still absent, seeking for new therapy is of great interest. Currently, several key pathological cascades following cerebral ischemia have been explored to develop further therapies. Among them, reactive nitrogen species (RNS) has been indicated to play a critical role in cerebral ischemia reperfusion injury. As one of the RNS, peroxynitrite contributes to the neural cell death and subsequent brain dysfunction in the process. Thus, development of

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47

Syrett, Andrew J. "Bioactive Glycerophospholipids and Their Role in Modulating Neuronal Vulnerability Following Cerebral Ischemia." Thesis, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20536.

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Stroke is a devastating and debilitating condition resulting from a blockage or hemorrhage in the vasculature of the brain. Despite extensive research, the etiology and pathophysiology of the disease at the level of the cell membrane are poorly understood, and effective treatment has been elusive. Though much research has shown marked increases in lipid metabolism following stroke, the impact of these changes have often been overlooked given the technical challenges associated with identifying regionally specific changes in degenerating tissue. The advent of lipidomics – a systems biology appr

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48

Cordell, Ryan [Verfasser]. "The relationship between the blood-brain barrier and cerebral ischemia / Ryan Cordell." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2012. http://d-nb.info/1028496222/34.

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49

Queiroga, Cláudia Susana Fernandes. "Disclosing Carbon Monoxide Protection in Cerebral Ischemia: Insights into the cellular mechanisms." Doctoral thesis, Universidade Nova de Lisboa. Instituto de Tecnologia Química e Biológica, 2012. http://hdl.handle.net/10362/10869.

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Dissertation presented to obtain the PhD degree in Biochemistry, Neurosciences<br>The present work demonstrates the ability of carbon monoxide (CO) to prevent apoptosis in primary culture of astrocytes. For the first time, the anti-apoptotic behaviour can be clearly attributed to the inhibition of mitochondrial membrane permeabilisation (MMP), a key event in the intrinsic apoptotic pathway. In isolated non-synaptic mitochondria CO partially inhibits (i) loss of membrane potential, (ii) the opening of a non specific pore through the inner membrane, (iii) swelling and (iv) cytochrome c rel

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50

Padayachy, L. C. "The prevalence of cerebral hypoxia/ischemia in pediatric severe traumatic brain injury." Master's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/2887.

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