Relevant bibliographies by topics / CGD (Constitutional Growth Delay)

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  2. Dissertations / Theses
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Academic literature on the topic 'CGD (Constitutional Growth Delay)'

Author: Grafiati
Published: 3 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "CGD (Constitutional Growth Delay)"

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Karim, Muhammad Rezaul, Kohinoor Jahan Shamaly, Baraka Badrudduja Tithi, Romana Akter, Ismat Jahan, and Suraiya Begum. "Etiology of short stature in children attending pediatric endocrinology clinic of a tertiary care hospital in Bangladesh." International Journal of Contemporary Pediatrics 7, no. 2 (2020): 363. http://dx.doi.org/10.18203/2349-3291.ijcp20200110.

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Background: Short stature is a common problem to practicing pediatricians. It results from various etiologies, which are categorized as normal variants and pathological causes. Normal variant short stature consists of Familial Short Stature (FSS) and Constitutional Growth Delay (CGD), while pathological causes are subdivided into endocrine diseases, clinically defined syndromes, chronic diseases, metabolic diseases and others. There are not so much data available in Bangladesh in this respect. So, present study was conducted to know the common causes of short stature.Methods: This cross-sectio
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Dr., Faryal Naveed Ahmed Dr. Asma Yasin Dr. Aatiqa Irshad. "SHORT STATURE ETIOLOGICAL PROFILE AND ITS CORRELATION TO FSS AND CGD IN BOTH MALE AND FEMALE: A CROSS-SECTIONAL RESEARCH." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 09 (2018): 8805–10. https://doi.org/10.5281/zenodo.1420171.

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<strong><em>Objective: </em></strong><em>We aimed to determine etiological factors frequency in the patients having short stature.</em> <strong><em>Methods: </em></strong><em>Sample size of this research was 100 children studied at Mayo Hospital, Lahore (February 2016 to March 2017). Design of the research was cross-sectional descriptive. Forty-eight boys and fifty-two girls were selected in the research sample in the age bracket of 3 &ndash; 15 years with a mean age of (9.9 &plusmn; 3.4). All the children were reported for their short stature. </em> <strong><em>Results: </em></strong><em>Most
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Loche, S., R. Corda, R. Puggioni, S. G. Cella, E. E. Müller, and C. Pintor. "Oxandrolone enhances growth hormone (GH) secretion and STIMULATES somatomedin C (SM-C) production in subjects with constitutional growth delay (CGD)." Journal of Steroid Biochemistry 25 (January 1986): 22. http://dx.doi.org/10.1016/0022-4731(86)90520-0.

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Hussein, Almontaser, Hekma Farghaly, Eman Askar, et al. "Etiological factors of short stature in children and adolescents: experience at a tertiary care hospital in Egypt." Therapeutic Advances in Endocrinology and Metabolism 8, no. 5 (2017): 75–80. http://dx.doi.org/10.1177/2042018817707464.

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Background: Accurate anthropometric measurements and critical analysis of growth data allow the clinician to promptly recognize children with short stature. The aim of this study was to determine the frequency of etiological factors causing short stature among children referred to the pediatric endocrinology clinic of Assiut University Children’s Hospital, the main tertiary care center in Upper Egypt. Methods: We conducted this descriptive observational study from May 2012 to December 2015, to analyze 637 children (boys 354, girls 283) with short stature. Evaluation included: detailed medical
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Rawat, Atul, Sumeet Gupta, and T. Joji Rao. "Risk analysis and mitigation for the city gas distribution projects." International Journal of Energy Sector Management 15, no. 5 (2021): 1007–29. http://dx.doi.org/10.1108/ijesm-10-2020-0001.

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Purpose This study aims to identify the operational and financial risks associated with the city gas distribution project in India and suggest appropriate strategies to mitigate them. Design/methodology/approach This study aims to identify the operational and financial risks associated with the city gas distribution project in India and suggest appropriate strategies to mitigate them. The survey data is evaluated using factor analysis to understand the latent structure of the critical risk factors. Second, the author uses Situation, Actor and Process–Learning, Action and Performance framework
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Frank, Graeme R. "Constitutional Delay of Growth and Puberty." Endocrinologist 13, no. 4 (2003): 341–46. http://dx.doi.org/10.1097/01.ten.0000084155.69073.d2.

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Lampit, Meir, and Ze’ev Hochberg. "Androgen Therapy in Constitutional Delay of Growth." Hormone Research in Paediatrics 59, no. 6 (2003): 270–75. http://dx.doi.org/10.1159/000070624.

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Bierich, Jürgen R. "6 Constitutional delay of growth and adolescence." Baillière's Clinical Endocrinology and Metabolism 6, no. 3 (1992): 573–88. http://dx.doi.org/10.1016/s0950-351x(05)80113-2.

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Stanhope, R., and M. A. Preece. "Management of constitutional delay of growth and puberty." Archives of Disease in Childhood 63, no. 9 (1988): 1104–10. http://dx.doi.org/10.1136/adc.63.9.1104.

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Tommiska, Johanna, Karoliina Wehkalampi, Kirsi Vaaralahti, Eeva-Maria Laitinen, Taneli Raivio, and Leo Dunkel. "LIN28B in Constitutional Delay of Growth and Puberty." Journal of Clinical Endocrinology & Metabolism 95, no. 6 (2010): 3063–66. http://dx.doi.org/10.1210/jc.2009-2344.

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Abstract Background: Recently variation in LIN28B, a human ortholog of the gene-regulating processing of micro-RNAs (miRNAs) controlling the timing of major developmental events in the nematode Caenorhabtidis elegans, was reported to be associated with timing of puberty in humans. In C. elegans, a gain-of-function allele of lin-28 causes a retarded phenotype. Objective: The objective of the study was to evaluate the variation in the LIN28B gene in 145 subjects with constitutional delay of growth and puberty (CDGP). Patients and Methods: For this study, 115 males and 30 females with CDGP were i
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Dissertations / Theses on the topic "CGD (Constitutional Growth Delay)"

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Tusset, Cíntia. "Pesquisa de mutações na neurocinina B e no seu receptor em pacientes com distúrbios puberais centrais idiopáticos." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-03092012-090655/.

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Mutações inativadoras nos genes TAC3 e TACR3, os quais codificam a neurocinina B (NKB) e o seu receptor NK3R, respectivamente, foram descritas em pacientes com hipogonadismo hipogonadotrófico isolado (HHI) normósmico. A partir desse achado, hipotetizamos que mutações ativadoras na NKB e/ou NK3R resultariam na secreção prematura de GnRH e, consequentemente, no desenvolvimento de puberdade precoce dependente de gonadotrofinas (PPDG). Nesse estudo, investigamos a presença de mutações ativadoras e/ou polimorfismos nos genes TAC3 e TACR3 em pacientes com PPDG, bem como mutações inativadoras e/ou po
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Books on the topic "CGD (Constitutional Growth Delay)"

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Mobbs, E. J. The Psychological Outcome of Constitutional Delay of Growth and Puberty (HORMONE RESEARCH). S. Karger A. G., 2005.

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Book chapters on the topic "CGD (Constitutional Growth Delay)"

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Bekx, M. Tracy, and Ellen Lancon Connor. "Constitutional Delay of Growth and Puberty." In Abnormal Female Puberty. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27225-2_3.

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Bierich, J. R. "Constitutional Delay of Growth and Adolescent Development." In Basic and Clinical Aspects of Growth Hormone. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5505-2_27.

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KIMMEL, S. "Short Child (Constitutional Growth Delay)." In Essential Family Medicine. Elsevier, 2006. http://dx.doi.org/10.1016/b978-1-4160-2377-7.50077-x.

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Hintz, Raymond L. "Growth, Normal Patterns and Constitutional Delay." In Encyclopedia of Endocrine Diseases. Elsevier, 2004. http://dx.doi.org/10.1016/b0-12-475570-4/00569-2.

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"Endocrinology: Short Stature (Constitutional Growth Delay)." In AM:STARs: Common Clinical Situations: A Resource for Practical Care and Exam Review, Vol. 28, No. 1. American Academy of Pediatrics, 2017. http://dx.doi.org/10.1542/9781610020732-19.

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Rotteveel, Joost, and Henriette A. Delemarre-Van de Waal. "Constitutional Delay of Growth and Puberty (CDGP)." In Encyclopedia of Endocrine Diseases. Elsevier, 2004. http://dx.doi.org/10.1016/b0-12-475570-4/00370-x.

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Rogol, Alan D., and John S. Fuqua. "Pubertal Delay and Hypogonadism." In Oxford Textbook of Endocrinology and Diabetes 3e, edited by John A. H. Wass, Wiebke Arlt, and Robert K. Semple. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198870197.003.0188.

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Delayed puberty occurs when no signs of pubertal maturation are seen by 14 years of age in boys or 13 years in girls. Although constitutional delay of growth and puberty is commonly seen and is often regarded as benign, there are many other causes of pubertal delay that ultimately result in permanent hypogonadism, and these must be differentiated from self-limited delays in maturation. Causes of hypogonadism include disorders affecting the gonads (primary hypogonadism) and abnormalities of pituitary or hypothalamic function (hypogonadotropic hypogonadism). Hypogonadotropic hypogonadism may be
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Butler, Gary. "Normal growth and its disorders." In Oxford Textbook of Medicine, edited by Mark Gurnell. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0255.

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Normal growth has three phases: rapid in infancy and adolescence, steady during mid childhood. Height should always be interpreted within the context of the family: short or tall stature is often familial; idiopathic short stature occurs when the height of a normal child is below their target range. Constitutional growth delay is a common normal variant, but poor growth and/or weight gain may be associated with recognized and unrecognized chronic disease, and also with psychosocial deprivation. Investigation must exclude conditions including hypothyroidism, coeliac disease, inflammatory bowel
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Ong, Ken, and Emile Hendriks. "Paediatric endocrinology." In Oxford Handbook of Endocrinology & Diabetes 4e. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198851899.003.0007.

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This chapter covers paediatric endocrinology. It starts with normal growth, then goes on to short stature, and constitutional delay of growth and puberty. Primary and secondary growth hormone deficiency are then explained, and treatment is outlined alongside GH resistance. It goes on to hypothyroidism, coeliac disease, skeletal dysplasias, and Turner syndrome. Small gestational age, and tall stature and rapid growth are all covered, alongside normal puberty, precocious puberty, and delayed or absent puberty. Normal sexual differentiation and disorders of sexual development and the assessment o
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Pugliese-Pires, P. N., J. P. Fortin, Y. Zhu, et al. "Novel Mutations in the Growth Hormone Secretagogue Receptor Gene (GHSR) Associated with Constitutional Delay in Growth and Puberty (CDGP)." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part3.or2.or35-3.

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