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Journal articles on the topic 'Charkov'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Dąbrowska, Magdalena. "IWAN ŁOBOJKO W POLSKIM ŚRODOWISKU NAUKOWYM (DWA PODEJŚCIA DO TEMATU NA PODSTAWIE WSPOMNIEŃ)." Acta Neophilologica 2, no. XX (December 1, 2019): 41–52. http://dx.doi.org/10.31648/an.3633.

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This paper consists of three parts: 1. The current state of research on Ivan Lobojko’s memoirs (comments of Samuel Fishman, Abram Reytblat, Marya Prussak, Reda Gruskaite); 2. The biography of Ivan Lobojko (born 1786 in Charkov, died 1861 in Mitau) and his contacts with Polish scientists (the community of Vilnius University, Samuel Bogumił Linde, Joachim Lelewel and others); 3. Two approaches to the subject “Ivan Lobojko in the Polish scientific community on the basis of his memoirs” (I. Lobojko, Moi vospominaniya. Moi zapiski, ed. A.I. Rejtblat, Moscow 2013): a) the “external” method – “The Poles about Lobojko”, a comparison of Lobojko’s memoirs and Poles’ memoires (A.H. Kirkor, A. Malinowski, T. Dobszewicz), b) the “internal” method – “Lobojko about the Poles”, a reconstruction of the profiles of Polish scientists in Lobojko’s memoirs (Zorian Dołęga Chodakowski and others).

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I., Lazaretov, and Polyakov A. "Investigations of the Uibat Charkov Burial Ground and New Data on the Early Stage of the Ocunevskaya Culture Development." Teoriya i praktika arkheologicheskikh issledovaniy, no. 3 (2018): 41–69. http://dx.doi.org/10.14258/tpai(2018)3(23).-03.

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3

Chernov, Oleg Alexandrovich. "The «Roman question» in N.V. Charykov’s assessment." Samara Journal of Science 6, no. 2 (June 1, 2017): 153–57. http://dx.doi.org/10.17816/snv201762213.

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N.V. Charykov paid much attention to the Roman question. His interest to this issue was likely to have been formed when he was a Russian diplomatic representative at the Vatican. After his diplomatic service in the Vatican N.V. Charykov published its greatest scientific work that was dedicated mainly to the relations of Russia and the Vatican in the seventeenth century. The problem discussed in this paper is connected with Charykovs views on the Roman question rather than his diplomatic service. The Roman question was covered in two articles The Pope and Caliph and The Roman question, Leo XIII, and signor Mussolini. In these papers N.V. Charykov revealed the origins of the problem, its development and a solution of the Roman question, as well as potential problems. In his opinion, the problem arose during the unification of Italy and a solution was found under the Government of Mussolini who was interested in the support of the Pope. The author pays attention to the relations between Russia and the Vatican after their recovery, referring to his own diplomatic practice.

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4

Chernov, Oleg Alexandrovich. "N.V. Charykov’s activities on reforming the ministry of foreign affairs of Russia." Samara Journal of Science 5, no. 2 (June 1, 2016): 105–10. http://dx.doi.org/10.17816/snv20162208.

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The problem of reforming the Ministry of Foreign Affairs of the Russian Empire in the beginning of XX century was widely highlighted in historical literature. However, the role of N.V. Charykov is covered very briefly. Since he was the chairman of the council on the reform of the Ministry of Foreign Affairs, it seemed necessary to explore his ideas and role in this transformative direction. He became the head of the council on the reform of the Ministry of Foreign Affairs after he had an appointment to a deputy minister of Foreign Affairs. It happened after A.P. Izvolsky (N.V. Charykovs friend and classmate at Imperial Alexander Lyceum) had become a foreign minister. N.V. Charykov denotes that A.P. Izvolsky invited him to become the deputy minister of Foreign Affairs. All the legal affairs of the Ministry of Foreign Affairs were transferred under authority of N.V. Charykov by A.P. Izvolsky. N.V. Charykov took up his post as the deputy minister of Foreign Affairs and was the head of the council on the reform of the Ministry of Foreign Affairs, implemented thoughtful and meticulous work. He could establish coordination among the actions of all the departments from which the reform was dependent on. Furthermore, he believed in the necessity of changing the structure of the Ministry of Foreign Affairs, linking it to the need to increase funding. Besides, he stood out for maintaining diplomatic correspondence in Russian instead of French. The important point was a democratization of the diplomatic exam on the one hand and an increase of the level of educational requirements for candidates on the other.

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5

Gordon, Rae Beth. "From Charcot to Charlot: Unconscious Imitation and Spectatorship in French Cabaret and Early Cinema." Critical Inquiry 27, no. 3 (April 2001): 515–49. http://dx.doi.org/10.1086/449019.

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6

Dr Munde D. D, Dr Munde D. D., Dr Sampada Karne, and Dr Nakul Shah. "Charcot Joints: A Case Study." Indian Journal of Applied Research 3, no. 1 (October 1, 2011): 129–30. http://dx.doi.org/10.15373/2249555x/jan2013/48.

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7

Chernov, Oleg Alexandrovich. "Contemporaries’ memoirs about N.V. Charykov’s diplomatic activity." Samara Journal of Science 5, no. 1 (March 1, 2016): 102–6. http://dx.doi.org/10.17816/snv20161206.

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The article focuses on N.V. Charykov, who was an outstanding Russian diplomat who played an important role in Russias foreign policy. His activity is reflected in many official documents. However, they not fully represent his interaction with other civil servants of the Russian empire, and, consequently, do not reflect in full the atmosphere in which the diplomat worked. At the same time, autobiographical sources containing a subjective approach by definition are not capable of giving objective characteristic of his activity. We do not consider the memoirs of the diplomat himself, as our objective is to find out his contemporariess opinion of him. The considered memoirs can be classified in two basic types - diaries and memoirs. Diaries can be divided into two types - business and personal. The latter are much less informative than the former. A personal diary has an advantage over a business one from the point of view of the emotional colouring and to a certain degree reproduces attitudes of the individuals described. The memoirists line of activity is important. Diplomats memoirs contain a better weighed appraisal of the diplomats activity. The authors, who were not diplomats themselves, display their incompetence. The memoirs contain different, sometimes opposite assessment of the diplomats activity, that is another proof of their subjectivity. At the same time, they help to better understand the motives of N.V. Charykovs activity and the attitude of the milieu to him.

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8

Agarwal, Lakshmi, Ila Sharma, and Naresh Rai. "Charcot Leyden Crystals in Liver FNAC." Annals of Advance Medical Sciecnes 1, no. 1 (December 9, 2017): L1—L2. http://dx.doi.org/10.21276/aams.1758.

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9

Goetz, Christopher G. "Poor Beard!!" Neurology 57, no. 3 (August 14, 2001): 510.1–514. http://dx.doi.org/10.1212/wnl.57.3.510.

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Objective: To analyze the role of the seminal 19th-century neurologist, Jean-Martin Charcot, in the internationalization of neurasthenia, previously known as “the American disease.”Background: The New York neurologist, George Beard, first described neurasthenia in 1869 and considered it a disorder related to the particular stress of modern civilization, most typified in the United States.Methods: Charcot’s personal files on neurasthenia from the Bibliothèque Charcot, Salpêtrière Hospital, Paris, were studied and his teaching lessons and lectures were examined.Results: Charcot presented numerous cases of neurasthenia, always crediting Beard with the original name and description. Calling Beard’s 1880 work a “remarkable monograph,” Charcot emphasized that patients with neurasthenia also comprised the bulk of his own private practice. Focusing on the signs of sexual impotency, fatigue, and a tight band of pressure around the head, Charcot categorically distinguished patients with neurasthenica from patients with hysteria. Like Beard, Charcot concluded that the origin of neurasthenia was psychological stress and felt the European society also fostered the environment to precipitate the disease. Charcot adamantly opposed extrapolations that called for early childhood educational reforms to reduce current classroom stress. Charcot sympathized more with the treating physician than the patient, calling neurasthenics insufferable (insupportables). On the front sheet of his neurasthenia file, he wrote in large script, “Poor Beard!!”Conclusion: By emphasizing the prevalence of neurasthenia and extending Beard’s observations, Charcot internationally legitimized the new diagnosis. Adding neurasthenia to the other neurologic descriptions from the United States by Hammond, Mitchell, and Dana, Charcot helped to foster the recognition of the American Neurologic School.

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10

Fishco, William D. "Surgically Induced Charcot’s Foot." Journal of the American Podiatric Medical Association 91, no. 8 (September 1, 2001): 388–93. http://dx.doi.org/10.7547/87507315-91-8-388.

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The neuropathic foot has the potential to develop Charcot arthropathy. This study describes multiple cases of Charcot’s foot following surgery. Of all the cases described, only one patient had any preexisting Charcot deformity or acute Charcot event. The study concludes that alterations of mechanical forces in the foot play an important role in triggering an acute Charcot episode. (J Am Podiatr Med Assoc 91(8): 388-393, 2001)

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11

Ogbaa, Maurine. "Chariot." Callaloo 36, no. 1 (2013): 70–79. http://dx.doi.org/10.1353/cal.2013.0058.

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12

Pradhan, Subhav, Abhishek Dubey, Shweta Khare, Saideep Nannapaneni, Aniruddha Gokhale, Sankaran Mahadevan, Douglas C. Schmidt, and Martin Lehofer. "CHARIOT." ACM Transactions on Cyber-Physical Systems 2, no. 3 (July 18, 2018): 1–37. http://dx.doi.org/10.1145/3134844.

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13

Meshram, Pankaj, and N. P. Awate N. P. Awate. "Design, Analysis of Mechanism of Amber Charkha." Indian Journal of Applied Research 3, no. 6 (October 1, 2011): 213–15. http://dx.doi.org/10.15373/2249555x/june2013/70.

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14

R, Sivakami. "Shoulder Charcot- a Presage of Cervical Syrinx." Journal of Medical Science And clinical Research 04, no. 10 (October 27, 2016): 13367–70. http://dx.doi.org/10.18535/jmscr/v4i10.94.

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15

Stephens, Sébastien, and Simon Platt. "Charcot Foot and the Osteoclast: More Than Just Cytokines." Foot & Ankle Orthopaedics 4, no. 4 (October 1, 2019): 2473011419S0041. http://dx.doi.org/10.1177/2473011419s00412.

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Category: Basic Sciences/Biologics Introduction/Purpose: Charcot foot is a serious complication of diabetes present in up to 13% carrying with high morbidity (dislocations, fractures, deformities) and up to 14% mortality. In Charcot, osteoclasts are found in increased numbers and with increased activity3,4 while C-telopeptide of type I collagen (CTX) is upregulated as opposed to ALP suggesting this is indeed an osteoclast and not an osteoblast disease. Methods: The scientific literature was reviewed to identify relevant osteoclastic activators and their role in the hyper activated state. We will also show videos of our current micromanipulation research techniques used to study osteoclast inhibitors in the lab. Results: The blood of Charcot patients is different at the nano level. Micro-nano-particles are extracellular vesicles derived from cells into plasma. Microparticles are found in increased quantity in Charcot;which contain more cytokines; known osteoclast activators. MicroRNAs – Epigenetic changes also are in Charcot; 16 different circulating microRNA known osteoclast activators are associated with Charcot. Advanced glycation end products (AGE). The hyperglycemic state in Charcot is a driving force in pathogenesis. AGEs are normal proteins turned dud after hyperglycemic induced glycation, a post translational modification which is irreversible affects both intracellular and extracellular proteins. AGEs may bind receptor RAGE, increasing osteoclast activity . RANKL is the main osteoclast differentiator and activator. RANKL has been the target of osteoclast control almost since it was found in 1998 . Conclusion: Charcot mediated bony destruction and remodeling; both of which are mediated by the osteoclast has traditionally had a tremendous cytokine focus. Our review suggests the mechanism for modifying a Charcot response on a local and systemic level. It is interesting to note that osteoclast inhibitors Denosumab, Bisphosphonates, Calcitonin and osteoblcast activators rPTH are not completely effective in switching off the Charcot response, implying there is a far more complex interaction between the inflammatory response, osteoclastic stimulators and osteoblastic inhibitors.

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16

Babazadeh, Sina, James D. Stoney, Keith Lim, and Peter F. M. Choong. "Arthroplasty of a Charcot knee." Orthopedic Reviews 2, no. 2 (August 6, 2010): 17. http://dx.doi.org/10.4081/or.2010.e17.

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<p class="MsoNormal" style="line-height: normal; margin: 0cm 0cm 10pt;"><span style="font-family: "><span style="font-size: small;">The Charcot knee - or neuropathic arthropathy - presents a considerable challenge to the orthopaedic surgeon. Caused by a combination of sensory, motor and autonomic neuropathy, it was originally described as an arthritic sequelae of neurosyphilis. In today’s western orthopaedics it is more often caused by diabetes. A Charcot knee is often symptomatically painful and unstable. Traditional management has usually been conservative or arthrodesis, with limited success. Arthroplasty of a Charcot joint has commonly been avoided at all costs. However, in the right patient, using the right technique, arthroplasty can significantly improve the symptoms of a Charcot joint. This article explores the evidence surrounding the role of arthroplasty in the management of a Charcot knee. Arthroplasty is compared to other forms of treatment and specific patient demographics and surgical techniques are explored in an attempt to define the role of arthroplasty in the management of a Charcot knee.</span></span></p>

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17

Saloky, Kaitlin, Michael Aynardi, Kempland Walley, Paul Juliano, Heather Matchik, and Thomas Abraham. "Quantitative Assessment of Type-1 Collagen in Charcot Neuroarthropathy with Use of Harmonic Generation and Multiphoton Microscopy." Foot & Ankle Orthopaedics 3, no. 3 (July 1, 2018): 2473011418S0041. http://dx.doi.org/10.1177/2473011418s00414.

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Category: Diabetes Introduction/Purpose: Diabetic Charcot neuroarthropathy is characterized by rapid loss of bone with accompanied disruption to the surrounding joints and is often in the absence of trauma or infection. Previous research has demonstrated neuropeptide release and osteoclast activation in addition to increased collagen turnover. Currently, there is limited understanding on the impact of the disease on type-1 collagen within the joint synovium. The purpose of this study was to analyze qualitative and quantitative differences in type-1 collagen between diabetic, neuropathic patient controls and those with Charcot neuroarthropathy. Methods: A prospective case-control study using routinely discarded surgical waste synovial tissue from diabetic, neuropathic patients with and without Charcot. The tissue was prepared in the laboratory for analysis. Microscopy was performed using multiphoton excitation fluorescence (MPEF) and second harmonic generation (SHG) microscopy techniques, utilizing a mode locked femto-second i: Sapphire Tsunami (Spectra-Physics, Mountain View, CA) synchronously pumped by a Millenia Xs J (Spectra-Physics) diode-pumped solid state laser. Qualitative and quantitative microscopic analysis was performed on Charcot specimens and controls. A statistical analysis was performed to detect a quantitative difference in type-1 collagen. Results: An analysis was performed on 8 samples; 4 neuropathic controls and 4 Charcot neuroarthropathy samples. There was a statistically significant difference between the amount, the quality and organization of type-1 collagen (p<0.0001). The Charcot group had significantly less type-1 collagen when compared to the control (p<0.0001). Qualitative analysis was performed using a mean intensity value. The control group’s mean intensity was 2561.4 nm while the Charcot group’s was 884.1 nm. The Charcot group had a significantly smaller mean intensity indicating poorer quality of collagen (p<0.0001). Orientation index was used to assess the organization of type-1 collagen in the synovium. A smaller orientation index signifies an increase in disorganization. The Charcot group had a significantly smaller orientation index of 16.5 compared to the control group’s 27.8 (p<0.0001). Conclusion: Charcot neuroarthropathy leads to significant abnormalities in type-1 collagen within the joint synovium when compared to the diabetic controls. Further research will determine if these differences are attributable to neuropeptide release and osteoclastic activation.

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18

Schmidt, Brian M., and Crystal M. Holmes. "Influence of Patient Setting and Dedicated Limb Salvage Efforts on Outcomes in Charcot-Related Foot Ulcer." International Journal of Lower Extremity Wounds 18, no. 4 (July 15, 2019): 362–66. http://dx.doi.org/10.1177/1534734619861571.

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Charcot neuroarthropathy is a devastating consequence of diabetes mellitus and peripheral neuropathy. Because of its rarity, the condition is often misdiagnosed or poorly managed. When misadventure occurs, patients with Charcot neuroarthropathy can suffer ulceration, infection, amputation, and death. When patients have Charcot-related foot ulcers, the risks are amplified. Utilizing advanced electronic medical record analysis, a 30-month investigation was undertaken to determine if patients with diabetes mellitus and a concomitant diagnosis of Charcot-related foot ulcer were at greater risk of complications because of location setting of initial treatment for their condition. Charcot foot–related ulcers that are diagnosed in the outpatient setting had established foot specialist care. The outpatient management of the condition lead to a significant reduction in the amount of admissions to a higher acuity setting. However, patient outcomes did not vary once established and dedicated limb salvage efforts were employed. In this large contemporary population managed in a tertiary health system, patients with Charcot-related foot ulcer had negative outcomes when they were initially treated in an inpatient setting and had a significantly greater likelihood of readmission as compared with individuals who had established focused limb salvage care.

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Pakarinen, T. K., H. J. Laine, S. E. Honkonen, J. Peltonen, H. Oksala, and J. Lahtela. "Charcot Arthropathy of the Diabetic Foot. Current Concepts and Review of 36 Cases." Scandinavian Journal of Surgery 91, no. 2 (June 2002): 195–201. http://dx.doi.org/10.1177/145749690209100212.

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Background and Aims: The incidence of diabetic Charcot neuroarthropathy has increased. The purpose here was to study the current diagnostics and treatment of the Charcot foot. Materials and Methods: During a time period from 1994 to 2000, a total of 36 feet were diagnosed as cases of diabetic Charcot neuroarthropathies. A retrospective analysis of patient records and radiographs was undertaken. A review of the recent literature is presented. Results: 29 cases were diagnosed in the dissolution stage, 2 in coalascence, and 5 in the resolution stage. The diagnostic delay averaged 29 weeks. Treatment with cast immobilisation ranged from 4 to 37 weeks (mean 11 weeks). A total of 14 surgical procedures were carried out on 10 patients: six exostectomies, four midfoot arthrodeses, one triple arthrodesis, one tibiocalcaneal arthrodesis and two below-knee amputations. radiological fusion was achieved in two thirds of the attempted arthrodeses. Conclusions: A physician should always consider the Charcot neuroarthropathy when a diabetic patient has an inflamed foot. In the absence of fever, elevated CRP or ESR, infection is a highly unlikely diagnosis, and a Charcot process should primarily be considered. The initial treatment of an inflamed Charcot foot consists in sufficiently long non-weightbearing with a cast, which should start immediately after the diagnosis. The prerequisites of successful reconstructive surgery are correct timing, adequate fixation and a long postoperative non-weightbearing period. In the resolution stage most Charcot foot patients need custom-molded footwear.

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Teive, Hélio A. Ghizoni, Sérgio M. Almeida, Walter Oleschko Arruda, Daniel S. Sá, and Lineu C. Werneck. "Charcot and Brazil." Arquivos de Neuro-Psiquiatria 59, no. 2A (June 2001): 295–99. http://dx.doi.org/10.1590/s0004-282x2001000200032.

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OBJECTIVE: To describe the relationship between Professor Charcot and Brazil. BACKGROUND: During the XIX century, French Neurology and its most prominent figure, Professor Charcot, dominated the area of nervous system diseases in the world. METHOD: We have reviewed some of the main publications about Charcot's life, the biography of Dom Pedro II, Emperor of Brazil and the development of Neurology in Brazil. RESULTS: Among the most important patients in Charcot's practice was the Emperor of Brazil. Dom Pedro II became a close friend of Charcot and he was a distinguished guest at Charcot's house, particularly at Tuesday soirées on boulevard St. Germain. In 1887, during the visit of Dom Pedro II to France, Charcot evaluated him and made the diagnosis of surmenage. In 1889, Dom Pedro II was deposed and went to Paris, where he lived until his death in 1891. Charcot signed the death certificate and gave the diagnosis of pneumonitis. Charcot had a passionate affection for animals, a feeling shared by Dom Pedro II. Dom Pedro II was affiliated to the French Society for the Protection of Animals. It is conceivable that Charcot's little monkey, from South America, was given to him by Dom Pedro II. The Brazilian Neurological School was founded by Professor A . Austregésilo in 1911, in Rio de Janeiro. At the time, of Charcot's death in 1893, his influence was still very important in the whole world. He and his pupils played a major role in the development of Brazilian Neurology. CONCLUSION: Professor Charcot had a close relationship with the Emperor of Brazil, Dom Pedro II. He was his private physician and they were close friends. The neurological school, created by professor Charcot, contributed significantly, albeit in an indirect way, to the development of Brazilian Neurology, starting in 1911, in Rio de Janeiro, by Professor A . Austregésilo.

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Shnayder, N. A., Ye V. Gluschenko, and Ye A. Kozulina. "Evaluation of life quality in patients with hereditary neuropathy Charcot—Marie—Tooth in the Krasnoyarsk Region." Bulletin of Siberian Medicine 10, no. 2 (April 28, 2011): 57–62. http://dx.doi.org/10.20538/1682-0363-2011-2-57-62.

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To assess the life quality in patients with hereditary neuropathy Charcot—Marie—Tooth in the Krasnoyarsk Region. Life quality was assessed in 28 patients with confirmed diagnosis of hereditary neuropathy Charcot—Marie—Tooth, using the «EuroQuality of Life — 5 Dimention», Rankin scale. A significant decline in the quality of life in patients with hereditary neuropathy Charcot—Marie—Tooth suggests the need to develop strategies for their physical, medical and social rehabilitation.

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Nakagawa, Masanori. "Diagnosis and care of Charcot-Marie-Tooth disease." Rinsho Shinkeigaku 54, no. 12 (2014): 950–52. http://dx.doi.org/10.5692/clinicalneurol.54.950.

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23

Płaza, Mateusz, Anna Nowakowska-Płaza, Marta Walentowska-Janowicz, Marek Chojnowski, and Iwona Sudoł-Szopińska. "Artropatia Charcota w obrazie ultrasonograficznym – opis przypadku." Journal of Ultrasonography 16, no. 65 (April 30, 2016): 210–15. http://dx.doi.org/10.15557/jou.2016.0022.

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24

Su, Jiyong. "A Brief History of Charcot-Leyden Crystal Protein/Galectin-10 Research." Molecules 23, no. 11 (November 9, 2018): 2931. http://dx.doi.org/10.3390/molecules23112931.

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Eosinophils are present in tissues, such as the respiratory tract, spleen, lymph nodes and blood vessels. The significant presence of eosinophils in these tissues are associated with various diseases, including asthma, allergies, acute myeloid leukemia, etc. Charcot-Leyden crystal protein/galectin-10 is overexpressed in eosinophils and has also been identified in basophils and macrophages. In human body, this protein could spontaneously form Charcot-Leyden crystal in lymphocytes or in the lysates of lymphocytes. At present, the role of Charcot-Leyden crystal protein/galectin-10 in lymphocytes is not fully understood. This review summarizes research progress on Charcot-Leyden crystal protein/galectin-10, with emphasis on its history, cellular distributions, relations to diseases, structures and ligand binding specificity.

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Carter, Stephen, Fraser Hunter, Andrea Smith, Mhairi Hastie, Steven Lancaster, Magnar Dalland, Robert Hurford, Edward Bailey, Gerry McDonnell, and Tony Swiss. "A 5th CenturybcIron Age Chariot Burial from Newbridge, Edinburgh." Proceedings of the Prehistoric Society 76 (2010): 31–74. http://dx.doi.org/10.1017/s0079497x0000044x.

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The remains of the first Iron Age chariot burial in Britain outside Yorkshire were discovered during the winter of 2000–1, near the Bronze Age burial mound of Huly Hill, at Newbridge, Edinburgh. Excavated by Headland Archaeology (UK) Ltd with the assistance of the National Museums Scotland, the chariot proved unique beyond just its burial location. The Newbridge chariot was buried intact, a method consistent with the burial practices of Continental Europe rather than Yorkshire, where they were predominantly buried disassembled. Detailed post-excavation analysis revealed a history of repair and reuse, and construction techniques that indicate links with the chariot building traditions of both Yorkshire and Continental Europe. Fifth centurybcradiocarbon dates for the burial place it firmly within La Tène A, consistent with its similarities to European examples and indicating that it pre-dates the known Yorkshire examples. This indicates the emergence of a British tradition of chariot construction by this time, familiar with Continental habits and technology but distinctively different in such areas as wheel construction and suspension fittings. Based on the findings, a reconstruction of the chariot was built for display in the National Museums Scotland, providing an opportunity further to understand and confirm the construction techniques observed or hypothesised during excavation.

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El Oraby, Hussein A., Mona M. Abdelsalam, Yara M. Eid, Rana El Hilaly, and Heba A. Marzouk. "Bone Mineral Density in Type 2 Diabetes Patients with Charcot Arthropathy." Current Diabetes Reviews 15, no. 5 (August 20, 2019): 395–401. http://dx.doi.org/10.2174/1573399814666180711115845.

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Introduction: Charcot arthropathy is one of the disabling diabetes complications. There are enigmatic areas concerning its underlying pathophysiology and risk predictors. Osteoporosis and local osteopenia have been postulated to have a role in Charcot arthropathy development, but it is still controversial. Background: The study aims to compare bone mineral density among type 2 diabetics with and without Charcot arthropathy. Methods: Two groups with type 2 diabetes participated in this study; Group I [30] patients with Charcot arthropathy while Group II [30] patients without charcot arthropathy. All patients underwent full clinical examination and complete history taking with special emphasis on foot problems. Laboratory investigations were done that included fasting blood sugar, postprandial blood sugar, glycosylated hemoglobin, serum calcium, serum phosphorus, and alkaline phosphatase. All patients underwent MRI for both feet and dual energy X-ray absorptiometry scan of the lumbar spine and femur. The demographic data, clinical data, the presence or absence of comorbidities and bone mineral density were compared for both groups. Results: Bone mineral density was significantly lower in Group I than Group II with median lumber T score (-0.15, 1.99 p <0.001), median Femur T score (0.050, 2.400, p <0.001). Group I showed higher propensity for hypertension, neuropathy, micro-albuminuria with peripheral arterial disease (23.33 %) compared to Group II (p <0.001). Multiple logistic regression analysis revealed that female gender and low femur bone mineral density can be risk predictors of the condition. Conclusion: Bone mineral density is lower in patients with Charcot arthropathy with female gender and Femur T score as risk predictors. Peripheral arterial disease shows greater incidence in Charcot patients than was previously reported.

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Ayele, J., B. Fabre, and M. Mazet. "Influence de l'origine et de la nature des substances humiques sur l'adsorption de l'atrazine sur charbon actif en poudre." Revue des sciences de l'eau 6, no. 4 (April 12, 2005): 381–94. http://dx.doi.org/10.7202/705181ar.

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Les essais d'adsorption sur charbon actif en poudre, de sept acides humiques (H) ou fulviques (F) et pour trois pH (5,6 ; 7,0 ; 8,8) marquent que ces acides ont un comportement différent suivant leur origine. Lorsque le pH de la suspension de charbon est basique, l'adsorption des différentes fractions humiques diminue, ce qui est en relation avec l'augmentation de leur solubilité. Pour un même pH de la suspension, les acides humiques et fulviques d'origine aqueuse s'adsorbent mieux que ceux d'origine commerciale ou que ceux extraits de sol. La capacité maximale d'adsorption de l'atrazine sur charbon actif en poudre : 260 mg.g-1 (Langmuir), est plus fortement diminuée en présence d'acides fulviques que d'acides humiques d'une part, et plus inhibée (jusqu'à 40 %) par les acides fulviques extraits d'eau (F1, F2) que par ceux extraits de sol (F3) d'autre part. Par contre, les acides humiques provenant de substances commerciales (H4, H5) influencent moins l'adsorption de l'atrazine. L'ensemble des essais montre que l'origine et la nature des substances humiques joue un rôle prépondérant sur l'adsorption de l'atrazine sur charbon actif, le pH ayant une influence limitée. Enfin l'étude de l'influence de l'atrazine sur l'adsorption des substances humiques sur charbon actif en poudre indique également une différence de comportement entre les acides fulviques, pour lesquels on note peu de variation, et les acides humiques, pour lesquels on note à la fois une promotion de l'adsorption et une meilleure affinité d'adsorption sur le charbon actif en poudre en présence d'atrazine. Ce travail met en évidence l'importance du choix des substances humiques pour des essais d'évaluation des interactions substances humiques - pesticides lors de l'adsorption sur charbon actif en poudre.

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Mortada, M. A., N. Ezz El_din, and M. Hammad. "AB1120 A TRIAD OF ULTRASOUND DETECTED HIGH-GRADE SYNOVITIS (MID TARSAL & ANKLE JOINTS), HIGH-GRADE TENOSYNOVITIS IN (TIBIALIS POSTERIOR & PERONEAL TENDONS) AND EROSIONS OF FIBULAR DISTAL END IS HIGHLY SUGGESTIVE OF CHARCOT ARTHROPATHY." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 1849.2–1849. http://dx.doi.org/10.1136/annrheumdis-2020-eular.2936.

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Background:Charcot arthropathy poses many clinical challenges in its diagnosis and management. Ultrasonography (US) may be able to identify pathological changes in cases of Charcot arthropathy (1).Objectives:to characterize the ultrasonographicfeatures of patients in early pre-radiographic stages of neuropathic (Charcot) arthropathy.Methods:This is an extension of our previous study of ultrasonographic features Charcot ankle and foot (1).an observational studywhere 42 patients with neuropathic (Charcot) arthropathy of the footbetween January 2013 and october 2019 were enrolled. Inclusion criteria included: 1- Foot pain and swelling. 2- Typical MRI findings of Charcot arthropathy in the form of subcondral bone marrow edema with or without sub-chondralmicrofractures, joint effusion or soft-tissue inflammation. 3- Grade 0 Modified Eichenholtz classification system which means normal radiography of the affected foot. Patients with other forms of arthropathy that may mimic Charcot arthropathy e.g. gouty arthritis and rheumatoid arthritis, were excluded from the study.Ultrasonographic (grey scale and Doppler US) examination of mid-tarsal and ankle joints was performed according to the EULAR guidelines.Results:Synovitis and effusionatmid tarsal joints were found in all patients and to a lesser extent in the ankle joints. High degree Doppler activity at both ankle and mid tarsal joints could be observed in most patients. Bony erosions were also common as well as tendonitis. A triad of active synovitis (in mid tarsal joints and ankle joints), active tendonitis (of tibialis posterior and peroneal tendons) and erosions in the distal end of fibula was present in 40 (95.2%) cases.Conclusion:our study confirms the ability of ultrasonography to detect inflammatory lesions in early stages of Charcot arthropathy.References:[1]Mortada M, Hammad M, Ezzeldin N..Annals of the Rheumatic Diseases 2016;75:1231-1232.Disclosure of Interests:None declared

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Banks, Alan S., and E. Dalton McGlamry. "Charcot Foot." Journal of the American Podiatric Medical Association 97, no. 4 (July 1, 2007): 325–48. http://dx.doi.org/10.7547/0970325.

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Banks, AS, and ED McGlamry. "Charcot foot." Journal of the American Podiatric Medical Association 79, no. 5 (May 1, 1989): 213–35. http://dx.doi.org/10.7547/87507315-79-5-213.

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Successful management of the Charcot foot is one of the most challenging undertakings faced by physicians. However, many times such patients undergo prolonged and attentive care only to develop further deformity, and in many cases succumb to amputation. Research in the past few years has yielded a new understanding of the Charcot process that should serve as the basis for improved therapeutic measures. The authors discuss these more recent developments and how this knowledge may be applied to better serve the patient. In addition, surgical reconstruction of the diabetic Charcot foot is introduced and discussed.

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Hamel, O., K. Buffenoir, M. Lefort, B. Perrouin-Verbe, and R. Robert. "Charcot-Spine." Annals of Physical and Rehabilitation Medicine 54 (October 2011): e115. http://dx.doi.org/10.1016/j.rehab.2011.07.656.

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Hansmann, Y., V. Remy, and D. Christmann. "Le charbon." Médecine et Maladies Infectieuses 34 (June 2004): S127—S129. http://dx.doi.org/10.1016/s0399-077x(04)90095-9.

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Houston, Dorothy S., and Janette Curran. "Charcot Foot." Orthopaedic Nursing 20, no. 1 (January 2001): 11–15. http://dx.doi.org/10.1097/00006416-200101000-00003.

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Pearce, J. M. "Charcot joints." Journal of Neurology, Neurosurgery & Psychiatry 58, no. 6 (June 1, 1995): 680. http://dx.doi.org/10.1136/jnnp.58.6.680.

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Levin, P. "The Chariot." Literary Imagination 8, no. 1 (January 1, 2006): 154–56. http://dx.doi.org/10.1093/litimag/8.1.154.

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Zgonis, Thomas, Thomas S. Roukis, John J. Stapleton, and Douglas T. Cromack. "Charcot foot." OR Nurse 4, no. 1 (January 2010): 36–41. http://dx.doi.org/10.1097/01.orn.0000366023.23313.81.

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Mumoli, N., and A. Camaiti. "Charcot foot." Canadian Medical Association Journal 184, no. 12 (March 19, 2012): 1392. http://dx.doi.org/10.1503/cmaj.111972.

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Giza, Eric, Christopher F. Hyer, Enzo J. Sella, and Thomas Zgonis. "Charcot Neuroarthropathy." Foot & Ankle Specialist 1, no. 4 (August 2008): 243–46. http://dx.doi.org/10.1177/1938640008321388.

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Landsman, Adam, Thomas Zgonis, Steven Neufeld, and Enzo J. Sella. "Hindfoot Charcot." Foot & Ankle Specialist 2, no. 2 (February 12, 2009): 83–88. http://dx.doi.org/10.1177/1938640009332252.

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Frykberg, Robert G., Ronald A. Sage, Dane K. Wukich, Michael S. Pinzur, and John M. Schuberth. "Charcot Arthropathy." Foot & Ankle Specialist 5, no. 4 (July 26, 2012): 262–71. http://dx.doi.org/10.1177/1938640012451234.

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Hyer, Christopher F., Michael S. Pinzur, J. Kent Ellington, W. Hodges Davis, and Carroll P. Jones. "Charcot Arthropathy." Foot & Ankle Specialist 7, no. 4 (July 15, 2014): 286–90. http://dx.doi.org/10.1177/1938640014542216.

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Lawson, Kay. "Jean Chariot." Modern & Contemporary France 5, no. 2 (May 1997): 251. http://dx.doi.org/10.1080/09639489708456376.

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Aubert, Geneviève. "Charcot Revisited." Archives of Neurology 62, no. 1 (January 1, 2005): 155. http://dx.doi.org/10.1001/archneur.62.1.155.

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Dattani, Rupen, Surendra Patnaik, and Mohan Lal. "Charcot foot." British Journal of Hospital Medicine 69, no. 6 (June 2008): 357. http://dx.doi.org/10.12968/hmed.2008.69.6.29630.

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Marx, J. S. "Exercice Chardon." Journal Européen des Urgences et de Réanimation 32, no. 1 (March 2020): 45–46. http://dx.doi.org/10.1016/j.jeurea.2020.02.004.

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Staloch, Michael A., and Stephen F. Hatem. "Charcot spine." Emergency Radiology 14, no. 4 (June 7, 2007): 265–69. http://dx.doi.org/10.1007/s10140-007-0615-z.

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Mittlmeier, T., K. Klaue, P. Haar, and M. Beck. "Charcot-Fuß." Der Unfallchirurg 111, no. 4 (March 29, 2008): 218–31. http://dx.doi.org/10.1007/s00113-008-1431-y.

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Kwaadu, Kwasi Y. "Charcot Reconstruction." Clinics in Podiatric Medicine and Surgery 37, no. 2 (April 2020): 247–61. http://dx.doi.org/10.1016/j.cpm.2019.12.002.

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Zwipp, H., S. Rammelt, C. Dahlen, and H. Reichmann. "Charcot Foot." Der Orthopäde 28, no. 6 (June 1999): 550–58. http://dx.doi.org/10.1007/pl00003640.

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Supriyani, Dwi, Imam Baehaqie, and Mulyono Mulyono. "ISTILAH-ISTILAH SESAJI RITUAL JAMASAN KERETA KANJENG NYAI JIMAT DI MUSEUM KERETA KERATON YOGYAKARTA." Jurnal Sastra Indonesia 8, no. 1 (April 16, 2019): 6–11. http://dx.doi.org/10.15294/jsi.v8i1.29852.

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Penelitian ini bertujuan untuk mendeskripsikan istilah sesaji ritual jamasan kereta Kanjeng Nyai Jimat di museum kereta Keraton Yogyakarta, makna dalam istilah sesaji ritual jamasan kereta Kanjeng Nyai Jimat, serta fungsi istilah-istilah sesaji sesaji ritual jamasan kereta Kanjeng Nyai Jimat di museum kereta Keraton Yogyakarta. Penelitian ini menggunakan pendekatan etnolinguistik dan pendekatan deskriptif kualitatif. Data dalam penelitian ini dijaring dengan menggunakan metode simak dan cakap. Analisis data menggunakan metode agih dengan teknik BUL, metode padan dengan teknik pilih unsur penentu, dan metode interaktif. Hasil penelitian menunjukkan sebagai berikut. Pertama, istilah-istilah sesaji ritual jamasan kereta Kanjeng Nyai Jimat terdiri atas bentuk satuan lingual berupa kata dan frasa. Kedua, istilah-istilah sesaji ritual Jamasan kereta Kanjeng Nyai Jimat diklasifikasi berdasarkan makna leksikal dan makna kultural. Ketiga, fungsi istilah-istilah sesaji ritual jamasan kereta Kanjeng Nyai Jimat terdiri atas sebagai alat komunikasi serta sebagai penghubung dunia gaib dan dunia nyata. This study aims to describe the term offerings in the ritual cleanse chariot Kanjeng Nyai Jimat at chariot museum of Yogyakarta Palace, meaning terms offerings in the ritual cleanse chariot Kanjeng Nyai Jimat, as well as the function of the terms of offerings in the ritual cleanse chariot Kanjeng Nyai Jimat at chariot museum of Yogyakarta Palace. This study uses an ethnolinguistic approach and a qualitative descriptive approach. Data was captured by hear and speak methods. Analysis of the data using agih method by BUL technique, method of padan with technique of choosing determinant element, and interactive method. The results showed as follows. First, the terms of offerings ritual cleanse chariot Kanjeng Nyai Jimat consists of a lingual unit form of words and phrases. Second, the terms of offerings ritual cleanse chariot Kanjeng Nyai Jimat classified based on the meaning of lexical and cultural meaning. Third, the function of the terms offerings ritual cleanse chariot Kanjeng Nyai Jimat consists of as a means of communication as well as a connector of the unseen world and the real world.

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