Relevant bibliographies by topics / Chemoattractants

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Chemoattractants'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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Journal articles on the topic "Chemoattractants"

1

Heit, Bryan, Samantha Tavener, Eko Raharjo, and Paul Kubes. "An intracellular signaling hierarchy determines direction of migration in opposing chemotactic gradients." Journal of Cell Biology 159, no. 1 (October 7, 2002): 91–102. http://dx.doi.org/10.1083/jcb.200202114.

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Neutrophils must follow both endogenous and bacterial chemoattractant signals out of the vasculature and through the interstitium to arrive at a site of infection. By necessity, in the setting of multiple chemoattractants, the neutrophils must prioritize, favoring end target chemoattractants (e.g., fMLP and C5a) emanating from the site of infection over intermediary endogenous chemoattractants (e.g., IL-8 and LTB4) encountered en route to sites of infection. In this study, we propose a hierarchical model of two signaling pathways mediating the decision-making process of the neutrophils, which allows end target molecules to dominate over intermediary chemoattractants. In an under agarose assay, neutrophils predominantly migrated toward end target chemoattractants via p38 MAPK, whereas intermediary chemoattractant-induced migration was phosphoinositide 3-kinase (PI3K)/Akt dependent. When faced with competing gradients of end target and intermediary chemoattractants, Akt activation was significantly reduced within neutrophils, and the cells migrated preferentially toward end target chemoattractants even at 1/1,000th that of intermediary chemoattractants. End target molecules did not require chemotactic properties, since the p38 MAPK activator, LPS, also inhibited Akt and prevented migration to intermediary chemoattractants. p38 MAPK inhibitors not only reversed this hierarchy, such that neutrophils migrated preferentially toward intermediary chemoattractants, but also allowed neutrophils to be drawn out of a local end target chemoattractant environment and toward intermediary chemoattractants unexpectedly in an exaggerated (two- to fivefold) fashion. This was entirely related to significantly increased magnitude and duration of Akt activation. Finally, end target chemoattractant responses were predominantly Mac-1 dependent, whereas nondominant chemoattractants used primarily LFA-1. These data provide support for a two pathway signaling model wherein the end target chemoattractants activate p38 MAPK, which inhibits intermediary chemoattractant-induced PI3K/Akt pathway, establishing an intracellular signaling hierarchy.
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King, Danielle, Hakan Başağaoğlu, Hoa Nguyen, Frank Healy, Melissa Whitman, and Sauro Succi. "Effects of Advective-Diffusive Transport of Multiple Chemoattractants on Motility of Engineered Chemosensory Particles in Fluidic Environments." Entropy 21, no. 5 (May 4, 2019): 465. http://dx.doi.org/10.3390/e21050465.

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Motility behavior of an engineered chemosensory particle (ECP) in fluidic environments is driven by its responses to chemical stimuli. One of the challenges to understanding such behaviors lies in tracking changes in chemical signal gradients of chemoattractants and ECP-fluid dynamics as the fluid is continuously disturbed by ECP motion. To address this challenge, we introduce a new multiscale numerical model to simulate chemotactic swimming of an ECP in confined fluidic environments by accounting for motility-induced disturbances in spatiotemporal chemoattractant distributions. The model accommodates advective-diffusive transport of unmixed chemoattractants, ECP-fluid hydrodynamics at the ECP-fluid interface, and spatiotemporal disturbances in the chemoattractant concentrations due to particle motion. Demonstrative simulations are presented with an ECP, mimicking Escherichia coli (E. coli) chemotaxis, released into initially quiescent fluids with different source configurations of the chemoattractants N-methyl-L-aspartate and L-serine. Simulations demonstrate that initial distributions and temporal evolution of chemoattractants and their release modes (instantaneous vs. continuous, point source vs. distributed) dictate time histories of chemotactic motility of an ECP. Chemotactic motility is shown to be largely determined by spatiotemporal variation in chemoattractant concentration gradients due to transient disturbances imposed by ECP-fluid hydrodynamics, an observation not captured in previous numerical studies that relied on static chemoattractant concentration fields.
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Mahadeo, Dana C., Mirkka Janka-Junttila, Rory L. Smoot, Pavla Roselova, and Carole A. Parent. "A Chemoattractant-mediated Gi-coupled Pathway Activates Adenylyl Cyclase in Human Neutrophils." Molecular Biology of the Cell 18, no. 2 (February 2007): 512–22. http://dx.doi.org/10.1091/mbc.e06-05-0418.

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Neutrophils and Dictyostelium use conserved signal transduction pathways to decipher chemoattractant gradients and migrate directionally. In both cell types, addition of chemoattractants stimulates the production of cAMP, which has been suggested to regulate chemotaxis. We set out to define the mechanism by which chemoattractants increase cAMP levels in human neutrophils. We show that chemoattractants elicit a rapid and transient activation of adenylyl cyclase (AC). This activation is sensitive to pertussis toxin treatment but independent of phosphoinositide-3 kinase activity and an intact cytoskeleton. Remarkably, and in sharp contrast to Gαs-mediated activation, chemoattractant-induced AC activation is lost in cell lysates. Of the nine, differentially regulated transmembrane AC isoforms in the human genome, we find that isoforms III, IV, VII, and IX are expressed in human neutrophils. We conclude that the signal transduction cascade used by chemoattractants to activate AC is conserved in Dictyostelium and human neutrophils and is markedly different from the canonical Gαs-meditated pathway.
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Geiger, Jeremy, Deborah Wessels, Shawn R. Lockhart, and David R. Soll. "Release of a Potent Polymorphonuclear Leukocyte Chemoattractant Is Regulated by White-Opaque Switching in Candida albicans." Infection and Immunity 72, no. 2 (February 2004): 667–77. http://dx.doi.org/10.1128/iai.72.2.667-677.2004.

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ABSTRACT Previous studies employing transmembrane assays suggested that Candida albicans and related species, as well as Saccharomyces cerevisiae, release chemoattractants for human polymorphonuclear leukocytes (PMNs). Because transmembrane assays do not definitively distinguish between chemokinesis and chemotaxis, single-cell chemotaxis assays were used to confirm these findings and test whether mating-type or white-opaque switching affects the release of attractant. Our results demonstrate that C. albicans, C. dubliniensis, C. tropicalis, C. parapsilosis, and C. glabrata release bona fide chemoattractants for PMNs. S. cerevisiae, however, releases a chemokinetic factor but not a chemoattractant. Characterization of the C. albicans chemoattractant revealed that it is a peptide of approximately 1 kDa. Whereas the mating type of C. albicans did not affect the release of chemoattractant, switching did. White-phase cells released chemoattractant, but opaque-phase cells did not. Since the opaque phase of C. albicans represents the mating-competent phenotype, it may be that opaque-phase cells selectively suppress the release of chemoattractant to facilitate mating.
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Goldman, G., R. Welbourn, J. M. Klausner, L. Kobzik, C. R. Valeri, D. Shepro, and H. B. Hechtman. "Intravascular chemoattractants inhibit diapedesis by selective receptor occupancy." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 2 (February 1, 1991): H465—H472. http://dx.doi.org/10.1152/ajpheart.1991.260.2.h465.

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An extravascular chemoattractant leads to migration of polymorphonuclear neutrophils (PMN) to that site, whereas intravascular administration leads to PMN oxidative activity and sequestration in microvessels but no diapedesis. This study examines the inhibitory role of intravascular chemoattractants. Rabbits (n = 37) were pretreated with zymosan-activated plasma (ZAP), leukotriene (LT) B4, or thromboxane (Tx) mimic. These agents were given intra-arterially, topically into plastic chambers taped atop sites of dermabrasion on the back, or into a lobar bronchus (n = 35). Intra-arterial injection of each chemoattractant resulted, 10 min later, in a 29-42% increase in intracellular PMN H2O2. In saline-infused animals, topical administration of the chemoattractants into dermabrasion chambers resulted in PMN accumulation per cubic millimeter after 3 h of 600 with ZAP, 536 with LTB4, and 643 with Tx mimic; all values higher than 46 with saline and 63 with normal plasma (all P less than 0.05). In other saline-infused animals, lobar lung aspiration of chemoattractants led to diapedesis as measured in bronchoalveolar lavage (BAL) fluid (PMN x 10(4)/ml) after 3 h: 19.0 with ZAP, 11.2 with LTB4 and 14.5 with Tx mimic, all greater than aspiration with saline or normal plasma 4.0 and 4.9, respectively (all P less than 0.05). Intra-arterial chemotactic administration inhibited subsequent PMN diapedesis in response to that same chemoattractant, both in dermabrasion chambers and in BAL fluid. When different intra- and extra-vascular chemoattractants were used diapedesis was promoted. Thus Tx infused intra-arterially and ZAP applied to a blister or lobar bronchus led to rapid cell migration and increased cell numbers.(ABSTRACT TRUNCATED AT 250 WORDS)
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Ho, Y. S., W. M. Lee, and R. Snyderman. "Chemoattractant-induced activation of c-fos gene expression in human monocytes." Journal of Experimental Medicine 165, no. 6 (June 1, 1987): 1524–38. http://dx.doi.org/10.1084/jem.165.6.1524.

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Human monocytes use the products of phosphoinositide hydrolysis (1,2-diacylglycerol and inositol 1,4,5-triphosphate) as second messengers to trigger rapid cellular activation during the occupancy of chemoattractant receptors. The effect of chemoattractants on modulation of gene expression in monocytes was examined in this study. The chemoattractants FMLP and platelet-activating factor induced the progressive increase of c-fos RNA to 6-15-fold over those of control within 30 min after treatment. Similar kinetics of c-fos gene activation was also observed when cells were treated with PMA or sn-1,2-dioctanoylglycerol, but not with the calcium mobilizer ionomycin, suggesting a role for protein kinase C in gene regulation by chemoattractant receptors. Activation of c-fos gene expression by FMLP is mediated through a pertussis toxin-sensitive G protein, since pertussis toxin treatment of the cells blocked the induction of the c-fos gene by FMLP but not PMA. The level of c-myc RNA was slightly decreased after 1 h of treatment with chemoattractants, but not with PMA or diacylglycerol. This implies that chemoattractant receptor occupancy generates signals beyond protein kinase C activation that are capable of selectively downregulating monocyte gene expression. The effect of FMLP and PMA on the accumulation of c-fos RNA appears to result from altering both the rate of transcription and message stability. These observations indicate that signals generated through chemoattractant receptor occupancy may regulate monocyte function at the genetic level.
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Shutt, D. C., L. M. Jenkins, E. J. Carolan, J. Stapleton, K. J. Daniels, R. C. Kennedy, and D. R. Soll. "T cell syncytia induced by HIV release. T cell chemoattractants: demonstration with a newly developed single cell chemotaxis chamber." Journal of Cell Science 111, no. 1 (January 1, 1998): 99–109. http://dx.doi.org/10.1242/jcs.111.1.99.

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A chemotaxis chamber has been developed to analyze both the velocity and the directionality of individual T cells in gradients of high molecular mass molecules over long periods of time. Employing this chamber, it is demonstrated that syncytia induced by HIV in SUP-T1 cell cultures release two T cell chemoattractants with approximate molecular masses of 30 and 120 kDa. Neither uninfected single cells nor polyethylene glycol-induced syncytia release detectable chemoattractant, suggesting that these chemoattractants are linked to HIV infection. Soluble gp120 functions as a T cell chemoattractant and the addition of anti-gp120 antibody to syncytium-conditioned medium blocks the high molecular mass chemoattractant activity but not the low molecular mass activity. The addition of anti-CD4 antibody to syncytium-conditioned medium also blocks the high molecular mass chemoattractant activity but not the low molecular mass activity. These results demonstrate that HIV-induced T cell syncytia release a low and a high molecular mass T cell chemoattractant, and suggest that the high molecular mass factor is gp120 and that it functions through the CD4 receptor.
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Takeyama, Kiyoshi, Carlos Agustí, Iris Ueki, James Lausier, Lars Olaf Cardell, and Jay A. Nadel. "Neutrophil-dependent goblet cell degranulation: role of membrane-bound elastase and adhesion molecules." American Journal of Physiology-Lung Cellular and Molecular Physiology 275, no. 2 (August 1, 1998): L294—L302. http://dx.doi.org/10.1152/ajplung.1998.275.2.l294.

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We examined the effect of the neutrophil chemoattractants interleukin (IL)-8 and N-formyl-methionyl-leucyl-phenylalanine on goblet cell (GC) degranulation in guinea pigs. Chemoattractants caused time-dependent neutrophil recruitment and GC degranulation in vivo. NPC 15669 (an inhibitor of leukocyte infiltration) prevented both responses, implicating neutrophils. ICI 200,355 (an inhibitor of neutrophil elastase and proteinase-3) or secretory leukocyte protease inhibitor (an inhibitor of elastase but not of proteinase-3) abolished IL-8-induced GC degranulation, implicating elastase. Incubating tracheal segments with IL-8 plus neutrophils caused GC degranulation in vitro, an effect due to activation of the neutrophils themselves (and not an effect present in the supernatant). Chemoattractant increased surface staining of elastase and the cleavage of elastase-specific fluorogenic substrate by neutrophils. Pretreatment with anti-intercellular adhesion molecule-1, anti-CD18, or anti-CD11b antibody inhibited the chemoattractant-induced GC degranulation in vitro, implicating adhesion molecules. These studies suggest that chemoattractants cause neutrophil-dependent GC degranulation involving adhesive interactions between cells, with elastase activity occurring at the cell interface, causing GC secretion. The findings, reproduced in human airways, suggest novel methods of therapeutic intervention.
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Kim, Chang H., and Hal E. Broxmeyer. "In Vitro Behavior of Hematopoietic Progenitor Cells Under the Influence of Chemoattractants: Stromal Cell–Derived Factor-1, Steel Factor, and the Bone Marrow Environment." Blood 91, no. 1 (January 1, 1998): 100–110. http://dx.doi.org/10.1182/blood.v91.1.100.

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Abstract How multiple chemoattractants cooperate in directing the migration of hematopoietic progenitor cells (HPC) for homing and peripheral blood mobilization has not yet been established. We report here the behavior of HPC under the influence of two different chemoattractants, stromal cell-derived factor (SDF)-1 and steel factor (SLF), and the chemotactic nature of the bone marrow (BM) environment using a two-chamber in vitro migration system. Various formulae were adopted to quantitate these effects. Based on these quantitations, SDF-1 showed only chemotactic activity, while SLF showed both chemotactic and chemokinetic activities on factor-dependent MO7e cells. SLF, like SDF-1, attracted human HPC from a population of CD34+ cells and induced actin polymerization in MO7e cells. SLF and SDF-1 cooperated in attracting MO7e cells, as well as cord blood (CB) and BM CD34+cells. A negative concentration gradient of SLF and SDF-1, formed by the presence of chemoattractants in the upper chamber, showed potent inhibitory effects on MO7e cell migration induced by either of these chemoattractants in the lower chamber, and SDF-1 and SLF were synergistic in mobilizing cells to the lower chamber from this negative chemoattractant gradient. Plasma obtained from BM aspirates, but not CB or peripheral blood, showed strong chemotactic effects on BM and CB CD34+ cells, and an inhibitory effect in a negative gradient on SDF-1–dependent CD34+ cell migration. These in vitro migration experiments suggest that chemoattractants such as SDF-1 and SLF with other unidentified BM chemoattractants may be involved cooperatively in the migration of HPC to the BM and in preventing spontaneous mobilization of HPC out of the BM.
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Kim, Chang H., and Hal E. Broxmeyer. "In Vitro Behavior of Hematopoietic Progenitor Cells Under the Influence of Chemoattractants: Stromal Cell–Derived Factor-1, Steel Factor, and the Bone Marrow Environment." Blood 91, no. 1 (January 1, 1998): 100–110. http://dx.doi.org/10.1182/blood.v91.1.100.100_100_110.

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How multiple chemoattractants cooperate in directing the migration of hematopoietic progenitor cells (HPC) for homing and peripheral blood mobilization has not yet been established. We report here the behavior of HPC under the influence of two different chemoattractants, stromal cell-derived factor (SDF)-1 and steel factor (SLF), and the chemotactic nature of the bone marrow (BM) environment using a two-chamber in vitro migration system. Various formulae were adopted to quantitate these effects. Based on these quantitations, SDF-1 showed only chemotactic activity, while SLF showed both chemotactic and chemokinetic activities on factor-dependent MO7e cells. SLF, like SDF-1, attracted human HPC from a population of CD34+ cells and induced actin polymerization in MO7e cells. SLF and SDF-1 cooperated in attracting MO7e cells, as well as cord blood (CB) and BM CD34+cells. A negative concentration gradient of SLF and SDF-1, formed by the presence of chemoattractants in the upper chamber, showed potent inhibitory effects on MO7e cell migration induced by either of these chemoattractants in the lower chamber, and SDF-1 and SLF were synergistic in mobilizing cells to the lower chamber from this negative chemoattractant gradient. Plasma obtained from BM aspirates, but not CB or peripheral blood, showed strong chemotactic effects on BM and CB CD34+ cells, and an inhibitory effect in a negative gradient on SDF-1–dependent CD34+ cell migration. These in vitro migration experiments suggest that chemoattractants such as SDF-1 and SLF with other unidentified BM chemoattractants may be involved cooperatively in the migration of HPC to the BM and in preventing spontaneous mobilization of HPC out of the BM.
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Dissertations / Theses on the topic "Chemoattractants"

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Salib, Rami Jean. "Mast cell chemoattractants in allergic rhinitis." Thesis, University of Southampton, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420254.

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Collington, Sarah J. "Chemoattractants involved in mast cell trafficking." Thesis, Imperial College London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.501471.

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Morales, Garcia Auden Andres. "The response of human spermatozoa to chemoattractants." Thesis, University of Birmingham, 2010. http://etheses.bham.ac.uk//id/eprint/630/.

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The effect of the chemoattractant bourgeonal on [Ca\(^{2+}\)]i and chemotaxis in human sperm was investigated. Burgeonal induced a dose-dependent, slowly-developing tonic elevation in [Ca\(^{2+}\)]i, The response was dependent on capacitation. In low-Ca\(^{2+}\) or EGTA-buffered saline the response to bourgeonal was inhibited. Pretreating spermatozoa with bis-phenol (20μM) to release stored Ca\(^{2+}\) did not alter the response. Thus bourgeonal acts primarily by inducing Ca\(^{2+}\) influx. Treatment of sperm with bourgeonal caused an increase in [cAMP]. When cells were pretreted with bourgeonal in low-Ca\(^{2+}\)saline, subsequent introduction of Ca\(^{2+}\) resulted in a single, large [Ca\(^{2+}\)]i transient in >75% of the cells, indicating that sudden influx of Ca\(^{2+}\) caused closure of the bourgeonal-sensitive Ca\(^{2+}\)- channel. This negative feedback was not modulated by IBMX (1mM) or dbcAMP (1mM), indicating that cAMP was not involved and that a direct action Ca\(^{2+}\) was more likely. Both Ni\(^{2+}\) (10μM) and La\(^{3+}\) (100μM) inhibited the action of bourgeonal on [Ca\(^{2+}\)]i, suggesting a possible role of CNG channels. Exposing sperm to a temporal bourgeonal gradient caused a series of transient [Ca\(^{2+}\)]i elevations in >20% of the cells. A gradient of progesterone (another characterised chemoattractant for human sperm) induced similar Ca\(^{2+}\) oscillations (in >20% of the cells), which increased in amplitude and frequency in response to the increasing progesterone concentration. Human spermatozoa responded chemotactically to a 1nM bourgeonal gradient, Chemotaxis was dependent on capacitation. The response was inhibited in low [Ca\(^{2+}\)]o but was unaltered by TMB-8 (an inhibitor of stored Ca\(^{2+}\) store release), thus showing a dependence on Ca\(^{2+}\) influx similar to the [Ca\(^{2+}\)]i signal.
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Stubbs, Victoria Emma Lucy. "The regulation of human cosinophil function by chemoattractants." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407128.

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Tan, Peter. "The role of chemoattractants in modulating neutrophil-endothelial adhesion." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302345.

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Sheikine, Yuri. "Chemoattractants as causative agents, biomarkers and therapeutic targets in vascular pathology /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-884-3/.

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Lin, Hao-jan. "Studies of chemoattractants from pea border cells and the release of pea (Pisum sativum) root border cells." Thesis, The University of Arizona, 1991. http://hdl.handle.net/10150/144632.

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Some plants release thousands of viable cells from root caps into the soil. These cells can be technically defined as Root Border Cells (BRD cells) and may play a role in the regulation of microbial populations in the rhizosphere. Chemoattractants released from pea (Pisam sativum) to Agrobacterium tumefaciens were characterized by using lectin and chemical analysis for heat-stability, size, and solubility. To understand the process of BRD cell release, a relationship was established between pectolytic enzyme activity and the release of pea BRD cells.
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Ivey, Claire Louise. "Investigation of the generation of leukocyte chemoattractants in a model of ischaemia and reperfusion in the anaesthetised rabbit." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309436.

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Newman, Mary B. "Human umbilical cord blood cells migration to stroke cns tissue extracts and the potential cytokines and chemokines involved." [Tampa, Fla.] : University of South Florida, 2005. http://purl.fcla.edu/fcla/etd/SFE0001206.

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Hasan, Anwar Matar. "Intracellular signalling in eosinophils : differential roles for phosphoinositide 3-kinase and p38 MAP kinase in cellular responses to chemoattractants." Thesis, Keele University, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.505655.

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Eosinophils are characteristic infiltrating cells in asthmatic airways and may contribute to allergic and inflammatory diseases pathology through the ability of their products to produce chronic inflammatory and remodelling processes. One focus of efforts to define new therapeutic targets for chronic inflammatory diseases has been the study of mechanisms through which eosinophils are recruited to tissues and become activated, leading to secretion of reactive oxygen species, inflammatory mediators, cationic proteins and cytokines. The signalling pathways responsible for evoking migratory and secretory response in human eosinophils are incompletely understood.
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Books on the topic "Chemoattractants"

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Bernardini, Giovanni, and Brian A. Zabel, eds. The Role of Chemoattractants in the Tumor Microenvironment. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-338-8.

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Moser, Bernhard, ed. History of Chemoattractant Research. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-701-9.

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Richard, Horuk, ed. Chemoattractant ligands and their receptors. Boca Raton: CRC Press, 1996.

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Horuk, Richard. Chemoattractant Ligands and Their Receptors. Edited by Richard Horuk. CRC Press, 2020. http://dx.doi.org/10.1201/9780367812539.

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Rodríguez-Fernández, José Luis, Mario Mellado, Marcus Thelen, and Philip Murphy, eds. Atypical Functions of Leukocyte Chemoattractant Receptors. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88966-205-0.

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Book chapters on the topic "Chemoattractants"

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Grotendorst, G. R., L. Paglia, C. Mcivor, S. Barsky, Y. Martinet, and D. Pencev. "Chemoattractants in Fibrotic Disorders." In Ciba Foundation Symposium 114 - Fibrosis, 150–63. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720950.ch10.

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Williams, T. J., D. A. Griffiths-Johnson, P. J. Jose, and P. D. Collins. "Eosinophil Chemoattractants Generated in Vivo." In Novel Molecular Approaches to Anti-Inflammatory Therapy, 1–9. Basel: Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7276-8_1.

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Opdenakker, Ghislain, and Jo Van Damme. "Novel Monocyte Chemoattractants in Cancer." In Chemokines and Cancer, 51–69. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-701-7_4.

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Vertuani, G., M. Boggian, and A. Scatturin. "SPECTROSCOPIC STUDIES OF SYNTHETIC PEPTIDE CHEMOATTRACTANTS." In Porto Carras, Chalkidiki, Greece, Aug. 31–Sept. 5, 1986, edited by Dimitrios Theodoropoulos, 353–56. Berlin, Boston: De Gruyter, 1987. http://dx.doi.org/10.1515/9783110864243-081.

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Parales, Rebecca E., and Jayna L. Ditty. "Chemotaxis to Atypical Chemoattractants by Soil Bacteria." In Methods in Molecular Biology, 255–80. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7577-8_21.

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Formaggio, Fernando, Monica Pantano, Marco Crisma, Gian Maria Bonora, Giovanni Valle, Claudio Toniolo, Elmer L. Becker, Wilhelmus H. J. Boesten, Hans E. Schoemaker, and Johan Kamphuis. "Formyl-methionyl tripeptide chemoattractants containing Cα-methylated chiral amino acids." In Peptides 1992, 615–16. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1470-7_278.

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Liao, Xin-Hua, and Alan R. Kimmel. "Biochemical Responses to Chemoattractants in Dictyostelium: Ligand-Receptor Interactions and Downstream Kinase Activation." In Methods in Molecular Biology, 271–81. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-198-1_18.

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Meena, Netra Pal, and Alan R. Kimmel. "Biochemical Responses to Chemically Distinct Chemoattractants During the Growth and Development of Dictyostelium." In Methods in Molecular Biology, 141–51. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3480-5_11.

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Ruff, Michael R., and Candace B. Pert. "Neuropeptides are Chemoattractants for Human Monocytes and Tumor Cells: A Basis for Mind-Body Communication." In Enkephalins and Endorphins, 387–98. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4899-0557-4_28.

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Smith, Charles D., Margarith W. Verghese, and Ralph Snyderman. "Regulation of Leukocyte Responses to Chemoattractants: Role of Receptors, Guanine Nucleotide Regulatory (N) Proteins and Phospholipase C." In Molecular Mechanisms of Desensitization to Signal Molecules, 277–89. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71782-6_18.

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Conference papers on the topic "Chemoattractants"

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Chukwurah, Kengelle Q., Yaping Yang, Jian Wang, Yajun Yan, and Eric C. Freeman. "Incorporating Stimuli-Responsive Bacteria in Microfluidic Droplets." In ASME 2015 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/smasis2015-9041.

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Model cellular membranes respond to chemical and electrical stimuli, regulating transport and exchange between two neighboring aqueous droplets. This regulated exchange may prove useful for controlling aqueous micro-environments for studying stimuli-responsive encapsulated bacteria. This concept is explored in this work, focusing on characterizing the bacterial response within a synthetic cellular environment. In the droplet interface bilayer (DIB) approach, aqueous micro-droplets deposited in an oil reservoir with dissolved lipids are coated with lipid monolayers and arranged into artificial cellular networks. This approach has been explored for potential use as a biologically-inspired smart material, but new material transduction pathways are necessary. This may be accomplished by combining this bottom-up approach to synthetic biology with living organisms such as stimuli-responsive bacteria. Bacteria encapsulation within the microfluidic droplets begins with a strain of Escherichia coli (E. coli), XL1-Blue. These flagellated bacteria naturally respond and move towards chemoattractants such as casamino acids, and their motion may be tracked through differential interference contrast (DIC) and fluorescent microscopy. Chemotaxis of XL1-Blue was assessed through low-flow perfusion of the chemoattractant (casamino acids) into a buffer solution containing the bacteria through a tailored capillary tube. Next, the response of bacteria within asymmetric DIB networks separating the bacteria and the chemoattractant were studied.
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Farahat, Waleed A., and H. Harry Asada. "Control of Eukaryotic Cell Migration Through Modulation of Extracellular Chemoattractant Gradients." In ASME 2010 Dynamic Systems and Control Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/dscc2010-4190.

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Cell migration is fundamental to a wide range of biological and physiological functions including: wound healing, immune defense, cancer metastasis, as well as the formation and development of biological structures such as vascular and neural networks. In these diverse processes, cell migration is influenced by a broad set of external mechanical and biochemical cues, particularly the presence of (time dependent) spatial gradients of soluble chemoattractants in the extracellular domain. Many biological models have been proposed to explain the mechanisms leading to the migratory response of cells as a function of these external cues. Based on such models, here we propose approaches to controlling the chemotactic response of eukaryotic cells by modulating their micro-environments in vitro (for example, using a microfluidic chemotaxis chamber). By explicitly modeling i) chemoattractant-receptor binding kinetics, ii) diffusion dynamics in the extracellular domain, and iii) the chemotactic response of cells, models for the migration processes arise. Based on those models, optimal control formulations are derived. We present simulation results, and suggest experimental approaches to controlling cellular motility in vitro, which can be used as a basis for cellular manipulation and control.
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Rao, Smitha M. N., Uday Tata, Victor K. Lin, Jer-Tsong Hsieh, Kytai Nguyen, and J. C. Chiao. "A Microfluidic Assay for Metastasis Potential Analysis." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13300.

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We have designed and characterized a poly-dimethyl-siloxane (PDMS) based microfluidic device called MiMiC™ that enables time-lapse study of cell migration. Cell migration is a key step of malignant metastasis during cancer progression. The device mimics the narrow confines the cells need to traverse and the microenvironments that are similar to the ones inside human body. Photolithography and soft lithography processes were used to fabricate the microfluidic devices. The device consists of two separate chambers connected by microfluidic channels allowing introduction of cells in one chamber and chemoattractants in the other. The response of lung-metastasized prostate cancer (PC-3-ML) cells and their migration response to chemoattractants were observed and analyzed. The numbers of cells under migration were determined from time-lapse images and compared to control groups. Our microfluidic assays provide advantages over the traditional Boyden chambers such as time-lapse observation, use of smaller amounts of reagents and direct assessment of cells under migration.
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Sambrano, Jesus, Yelena Smagley, Alexandre Chigaev, Larry A. Sklar, and Jessica P. Houston. "Using FRET to quantify changes in integrin structures in human leukocytes induced by chemoattractants with multi-frequency flow cytometry." In SPIE BiOS, edited by Wei R. Chen. SPIE, 2017. http://dx.doi.org/10.1117/12.2253340.

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Shih, Po-Chen, Yi-Jr Su, Ke-Min Chen, Lin-Ni Jen, Cheng-tzu Liu, and Long Hsu. "Using optical tweezers to examine the chemotactic force to a single inflammatory cell--eosinophil stimulated by chemoattractants prepared from Toxocara Canis larvae." In Optics & Photonics 2005, edited by Kishan Dholakia and Gabriel C. Spalding. SPIE, 2005. http://dx.doi.org/10.1117/12.616826.

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Pachynski, Russell K., Brian A. Zabel, Nicole Tejeda, and Eugene C. Butcher. "Abstract 2939: Leukocyte chemoattractant chemerin as a novel immunotherapeutic agent." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2939.

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Kim, Min Jun, and Kenneth S. Breuer. "A Selective Mixing in Microfluidic Systems Using Bacterial Chemotaxis." In ASME 2005 International Mechanical Engineering Congress and Exposition. ASMEDC, 2005. http://dx.doi.org/10.1115/imece2005-80497.

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We demonstrate the use of Escherichia coli and their chemotactic characteristics to enhance mixing in a microchannel in a controlled and bi-directional manner. The presence of a chemoattractant in one arm of a three-junction microchannel results in an asymmetric increase in the effective diffusion coefficient of Dextran, which rises linearly with the concentration of attractant from a baseline value of 8 μm2/s to 42 μm2/s at a concentration of 0.1 M. The response to repellent is similar, with the opposite bias.
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O'Reilly, Philip, Matthew Hardison, Patricia L. Jackson, F. Shawn Galin, Xin Xu, Robert Snelgrove, Amit Gaggar, and James E. Blalock. "Neutrophils Contain Prolyl Endopeptidase And Generate The Neutrophil Chemoattractant, PGP, From Collagen." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3886.

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Meng, Ke, Hao Yang, Yong Wang, and Dong Sun. "Active disturbance rejection control of single cell migration induced by chemoattractant-loaded microbead." In 2016 IEEE International Conference on Real-time Computing and Robotics (RCAR). IEEE, 2016. http://dx.doi.org/10.1109/rcar.2016.7784062.

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Hao Yang, Xue Gou, Henry Chu, Yong Wang, and Dong Sun. "Quantitative analysis of chemoattractant gradient induced cell migration velocity and automatic controller design." In 2014 IEEE 14th International Conference on Nanotechnology (IEEE-NANO). IEEE, 2014. http://dx.doi.org/10.1109/nano.2014.6968077.

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