Relevant bibliographies by topics / Chemokine inhibitor

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Chemokine inhibitor'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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Journal articles on the topic "Chemokine inhibitor"

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Calkins, Casey M., Denis D. Bensard, Julie K. Heimbach, et al. "l-Arginine attenuates lipopolysaccharide-induced lung chemokine production." American Journal of Physiology-Lung Cellular and Molecular Physiology 280, no. 3 (2001): L400—L408. http://dx.doi.org/10.1152/ajplung.2001.280.3.l400.

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Chemokines stimulate the influx of leukocytes into tissues. Their production is regulated by nuclear factor-κB (NF-κB), an inducible transcription factor under the control of inhibitory factor κB-α (IκB-α). We have previously demonstrated that l-arginine (l-Arg) attenuates neutrophil accumulation and pulmonary vascular injury after administration of lipopolysaccharide (LPS). We hypothesized thatl-Arg would attenuate the production of lung chemokines by stabilizing IκB-α and preventing NF-κB DNA binding. We examined the effect of l-Arg on chemokine production, IκB-α degradation, and NF-κB DNA b
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Gewirtz, AM, J. Zhang, J. Ratajczak, et al. "Chemokine regulation of human megakaryocytopoiesis." Blood 86, no. 7 (1995): 2559–67. http://dx.doi.org/10.1182/blood.v86.7.2559.2559.

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Abstract We have previously shown that platelet factor 4 (PF4), a platelet-specific CXC chemokine, can directly and specifically inhibit human megakaryocyte colony formation. We therefore hypothesized that PF4 might function as a negative autocrine regulator of megakaryocytopoiesis. Herein we present additional studies characterizing the inhibitory effect of CXC chemokines on human megakaryocyte development. We first corroborated our initial studies by showing that recombinant human (rH) PF4, like the native protein, inhibited megakaryocytopoiesis. We then examined the inhibitory properties of
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Gewirtz, AM, J. Zhang, J. Ratajczak, et al. "Chemokine regulation of human megakaryocytopoiesis." Blood 86, no. 7 (1995): 2559–67. http://dx.doi.org/10.1182/blood.v86.7.2559.bloodjournal8672559.

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We have previously shown that platelet factor 4 (PF4), a platelet-specific CXC chemokine, can directly and specifically inhibit human megakaryocyte colony formation. We therefore hypothesized that PF4 might function as a negative autocrine regulator of megakaryocytopoiesis. Herein we present additional studies characterizing the inhibitory effect of CXC chemokines on human megakaryocyte development. We first corroborated our initial studies by showing that recombinant human (rH) PF4, like the native protein, inhibited megakaryocytopoiesis. We then examined the inhibitory properties of other CX
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RECKLESS, Jill, and David J. GRAINGER. "Identification of oligopeptide sequences which inhibit migration induced by a wide range of chemokines." Biochemical Journal 340, no. 3 (1999): 803–11. http://dx.doi.org/10.1042/bj3400803.

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We have identified an amino acid sequence, termed peptide 3, corresponding to amino acids 51-62 of the mature human monocyte chemoattractant protein-1 (MCP-1), which inhibits human mononuclear-cell and THP-1-cell migration induced by a wide range of chemokines. For example, peptide 3 inhibited MCP-1-induced THP-1 migration in a transwell assay with an ED50 of approx. 8 μM. Peptide 3 binds directly to THP-1 cells with an association constant of approx. 10 μM, and is therefore likely to be a direct receptor antagonist for CC and CXC chemokine receptors. By performing a structure-function analysi
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Wu, X., G. J. Dolecki, and J. B. Lefkowith. "GRO chemokines: a transduction, integration, and amplification mechanism in acute renal inflammation." American Journal of Physiology-Renal Physiology 269, no. 2 (1995): F248—F256. http://dx.doi.org/10.1152/ajprenal.1995.269.2.f248.

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We recently observed that cytokine-induced neutrophil chemoattractant (CINC), a GRO chemokine, contributes to neutrophil migration into the inflamed glomerulus in rat. Therefore, we sought to clarify how expression of the GRO chemokines, CINC and macrophage inflammatory protein-2 (MIP-2), is regulated in mesangial cells in vitro and the kidney in vivo. Mesangial cells expressed both GRO chemokine mRNAs in response to mediators of acute renal inflammation [interleukin-1 beta (IL-1 beta), tumor necrosis factor-alpha (TNF-alpha), and lipopolysaccharides (LPS)], but not chronic renal inflammation
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Ramnath, Raina Devi, Jia Sun, Sharmila Adhikari, Liang Zhi та Madhav Bhatia. "Role of PKC-δ on substance P-induced chemokine synthesis in pancreatic acinar cells". American Journal of Physiology-Cell Physiology 294, № 3 (2008): C683—C692. http://dx.doi.org/10.1152/ajpcell.00360.2007.

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Interaction of the neuropeptide substance P (SP) with its high-affinity neurokinin-1 receptor (NK1R) plays an important role in the pathophysiology of acute pancreatitis. SP is known to stimulate the production of chemokines monocyte chemoattractant protein-1 (MCP-1), macrophage inflammatory protein (MIP)-1α, and MIP-2 in pancreatic acinar cells via the activation of NF-κB. However, the signaling mechanisms by which the SP-NK1R interaction induces NF-κB activation and chemokine production remain unclear. To that end, in the present study, we investigated the participation of PKC in SP-induced
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Sun, Jia, Raina Devi Ramnath, and Madhav Bhatia. "Neuropeptide substance P upregulates chemokine and chemokine receptor expression in primary mouse neutrophils." American Journal of Physiology-Cell Physiology 293, no. 2 (2007): C696—C704. http://dx.doi.org/10.1152/ajpcell.00060.2007.

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Neuropeptides play an important role in the active communication between the nervous and immune systems. Substance P (SP) is a prominent neuropeptide involved in neurogenic inflammation and has been reported to exert various proinflammatory actions on inflammatory leukocytes including neutrophils. The present study further investigated the modulatory effect of SP (1 μM) on chemokine production and chemokine receptor expression in primary mouse neutrophils. Our results showed that SP primed neutrophils for chemotactic responses not only to the CXC chemokine macrophage inflammatory protein (MIP)
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Alejo, Alí, Carolina Sánchez, Sylvie Amu, Padraic G. Fallon, and Antonio Alcamí. "Addition of a Viral Immunomodulatory Domain to Etanercept Generates a Bifunctional Chemokine and TNF Inhibitor." Journal of Clinical Medicine 9, no. 1 (2019): 25. http://dx.doi.org/10.3390/jcm9010025.

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The inhibition of tumor necrosis factor (TNF) through the use of either antibodies or soluble receptors is a highly effective strategy for the clinical control of chronic inflammatory conditions such as rheumatoid arthritis. Different viruses have similarly exploited this concept by expressing a set of specifically tailored secreted TNF decoy receptors to block host inflammatory responses. Poxviruses have been shown to encode at least two distinct molecules, termed Cytokine response modifier D (CrmD) and CrmB, in which a TNF inhibitor is combined with a chemokine inhibitor on the same molecule
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Lateef, Zabeen, Margaret A. Baird, Lyn M. Wise, Andrew A. Mercer, and Stephen B. Fleming. "Orf virus-encoded chemokine-binding protein is a potent inhibitor of inflammatory monocyte recruitment in a mouse skin model." Journal of General Virology 90, no. 6 (2009): 1477–82. http://dx.doi.org/10.1099/vir.0.009589-0.

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The parapoxvirus orf virus causes pustular dermatitis in sheep and is transmissible to humans. The virus encodes a secreted chemokine-binding protein (CBP). We examined the ability of this protein to inhibit migration of murine monocytes in response to CC inflammatory chemokines, using chemotaxis assays, and its effects on monocyte recruitment into the skin, using a mouse model in which inflammation was induced with bacterial lipopolysaccharide. CBP was shown to bind murine chemokines CCL2, CCL3 and CCL5 with high affinity by surface plasmon resonance and it completely inhibited chemokine-indu
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Chen, Xin, Lu Yang, Ning Zhang, et al. "Shikonin, a Component of Chinese Herbal Medicine, Inhibits Chemokine Receptor Function and Suppresses Human Immunodeficiency Virus Type 1." Antimicrobial Agents and Chemotherapy 47, no. 9 (2003): 2810–16. http://dx.doi.org/10.1128/aac.47.9.2810-2816.2003.

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ABSTRACT Shikonin is a major component of zicao (purple gromwell, the dried root of Lithospermum erythrorhizon), a Chinese herbal medicine with various biological activities, including inhibition of human immunodeficiency virus (HIV) type 1 (HIV-1). G protein-coupled chemokine receptors are used by HIV-1 as coreceptors to enter the host cells. In this study, we assessed the effects of shikonin on chemokine receptor function and HIV-1 replication. The results showed that, at nanomolar concentrations, shikonin inhibited monocyte chemotaxis and calcium flux in response to a variety of CC chemokin
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Dissertations / Theses on the topic "Chemokine inhibitor"

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Zhang, Li. "Structural study of the interaction between poxvirus-encoded cc chemokine inhibitor vcci and human mip-1beta." Diss., Texas A&M University, 2008. http://hdl.handle.net/1969.1/85901.

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Chemokines (chemotactic cytokines) comprise a large family of proteins that recruit and activate leukocytes, giving chemokines a major role in both immune response and inflammation-related diseases. Viral CC chemokine inhibitor (vCCI) is a poxvirus encoded protein that has been shown to bind tightly and inhibit the action of many CC chemokines. This function suggests that vCCI could be explored as an antiinflammatory therapeutic, a possibility that has been supported in mouse studies. The structure of vCCI in unbound form was determined by others, but to date no structure has been reported of
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Simmons, Graham. "Human immunodeficiency syndrome virus type 1 cell tropism and inhibition by chemokines and chemokine analogues." Thesis, Institute of Cancer Research (University Of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368041.

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Montero, Rosa Maria. "Chemokines and macrophage migration inhibitory factor in diabetic nephropathy." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/29851.

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Introduction: Diabetic nephropathy (DN) is the leading cause of end-stage renal disease in the Western world. Aim: To determine whether macrophage migration inhibitory factor (MIF), monocyte chemoattractant protein-1 (MCP-1) or CC chemokine ligand 18 (CCL18) have a causative role in the development of renal inflammation and fibrosis in DN and are useful biomarkers of disease progression. Methods: Urine and plasma samples were collected from 115 DM and 116 Non-DM at baseline, previously analysed for MCP-1 and CCL18 ELISA by Dr Qureshi. I measured MIF in these samples and collected 107 DM and 11
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Bono, Johann S. de. "Macrophage inflammatory protein-1#alpha# : an inhibitor of clonogenic epidermal keratinocyte proliferation?" Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298214.

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Kok, Tsz-wai. "Blockade of chemokine (C-X-C motif) receptor 4 for the inhibition of hepatocellular carcinoma metastasis." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B4068765X.

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Kok, Tsz-wai, and 郭梓瑋. "Blockade of chemokine (C-X-C motif) receptor 4 for the inhibition of hepatocellular carcinoma metastasis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B4068765X.

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Brahm, Kevin [Verfasser], Katja [Akademischer Betreuer] Schmitz, and Harald [Akademischer Betreuer] Kolmar. "Untersuchung von Peptidomimetika als Inhibitoren für das Chemokin CXCL8 / Kevin Brahm ; Katja Schmitz, Harald Kolmar." Darmstadt : Universitäts- und Landesbibliothek Darmstadt, 2020. http://d-nb.info/1211726185/34.

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Jöst, Marina [Verfasser], Katja [Akademischer Betreuer] Schmitz, and Harald [Akademischer Betreuer] Kolmar. "Etablierung von zellbasierten Assays zur Identifizierung von Inhibitoren des Chemokins CXCL8 / Marina Jöst. Betreuer: Katja Schmitz ; Harald Kolmar." Darmstadt : Universitäts- und Landesbibliothek Darmstadt, 2016. http://d-nb.info/1112333231/34.

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Amati, Anca-Laura [Verfasser]. "Chemokines (CCL3, CCL4, CCL5) inhibit ATP-induced release of IL-1beta by monocytic cells / Anca-Laura Amati." Gießen : Universitätsbibliothek, 2019. http://d-nb.info/1198109173/34.

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Eriksson, Catharina. "Immunological mechanisms in systemic autoimmunity : autoantibodies and chemokines in systemic lupus erythematosus and during treatment with TNF inhibitors in rheumatoid arthritis." Doctoral thesis, Umeå universitet, Klinisk immunologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-42954.

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Background. Rheumatoid Arthritis (RA) is an autoimmune inflammatory disease that, without powerful treatment, may lead to irreversible joint damage. During the past decade, anti-cytokine therapy has become available, e.g., infliximab, a chimeric antibody targeting the pro-inflammatory cytokine TNF that has a central role in the inflammatory process in RA patients. Systemic Lupus Erythematosus (SLE) is a systemic autoimmune disease that may affect all organs and is characterized by a massive antibody production. Chemokines, chemokine receptors and lipoprotein receptor-related protein 1(CD91) ar
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Books on the topic "Chemokine inhibitor"

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Mease, Philip. Neurobiology of pain in osteoarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0013.

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Significant advances in our understanding of the neurobiology of pain in osteoarthritis (OA) have occurred in the last decade and are herein summarized. Pain is the predominant symptom of OA and occurs at multiple levels from non-cartilage peripheral tissues to spinal cord, and brain and back. At each level, nerve function is regulated by complex ionic channels, neuropeptide expression, and cytokine and chemokine activity. Previously considered a non-inflammatory condition, it is now recognized that cell proliferation and inflammatory cytokine production occurs in OA synovium, contributing to
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Book chapters on the topic "Chemokine inhibitor"

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Hippe, Andreas, Bernhard Homey, and Anja Mueller-Homey. "Chemokines." In Angiogenesis Inhibition. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-78281-0_4.

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Graham, Gerard J., and Robert J. B. Nibbs. "Macrophage Inflammatory Protein-1α and Stem Cell Inhibition." In Chemokines and Cancer. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-701-7_16.

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Su, S. B., H. Ueda, O. M. Z. Howard, et al. "Inhibition of the Expression and Function of Chemokine Receptors on Human CD4+ Leukocytes by HIV-1 Envelope Protein gp120." In Chemical Immunology and Allergy. KARGER, 1999. http://dx.doi.org/10.1159/000058731.

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Zhang, W., C. Smith, R. Monette, J. Hutchison, and D. B. Stanimirovic. "Indomethacin and Cyclosporin a Inhibit in Vitro Ischemia-Induced Expression of ICAM-1 and Chemokines in Human Brain Endothelial Cells." In Brain Edema XI. Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6346-7_10.

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Trkola, Alexandra, Timothy Wells, and Amanda Proudfoot-Fichard. "Chemokine Receptors." In Cytokine Inhibitors. CRC Press, 2000. http://dx.doi.org/10.1201/9780203904244.ch7.

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Cairns, J. Scott, and M. Patricia D’Souza. "Therapies to Prevent or Inhibit Chemokine Receptor Expression." In Chemokine Receptors and AIDS. CRC Press, 2019. http://dx.doi.org/10.1201/9780429074974-12.

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Lüscher, Thomas F., and Paul M. Ridker. "Anti-inflammatory therapies for cardiovascular disease." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0272.

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Inflammation involves many blood cells and the molecules released by them such as cytokines, chemokines, and antibodies. In particular, cells derived from monocyte/macrophage lines are involved in atherogenesis, as are numerous chemokines, cytokines, and adhesion molecules expressed in the vasculature or adipose tissue. Different inflammatory pathways have been considered in the prevention and treatment of cardiovascular disease. Specifically, as outlined previously in a review of the authors, several lines of evidence support the concept that inhibition of the central immune pathway linking interleukin 1, tumour necrosis factor alpha, and interleukin 6 might be novel targets.
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Barnes, Debra A., Steven W. Jones, and H. Daniel Perez. "High throughput screening for identification of RANTES chemokine expression inhibitors." In Methods in Enzymology. Elsevier, 1997. http://dx.doi.org/10.1016/s0076-6879(97)87021-8.

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Mukherjee, Sitabja, and Santosh K. Kar. "Curcuminoids: The Novel Molecules of Nature." In Herbs and Spices - New Processing Technologies [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99201.

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Curcuminoids inactivate Nuclear Factor-Kappa B (NF-κB), a key pro-inflammatory transcription factor which is involved in inflammation and immune response in diseases like cancer. NF-κB activation is necessary to determine tumor microenvironment which controls migration and metastatis of cancer cells through chemokines and their receptors and involvement of some cell adhesion molecules. Therefore inhibition of NF-κB by curcuminoids could be a new approach in treatment of cancer by immune modulation. Curcuminoids are not bioavailable and therefore there were problems in efficacy. Now by using bioavailable curcuminoid formulations the problem has been resolved to a great extent. Out of 49 placebo controlled double blind clinical trials using curcuminoids, 17 have been found to be successful. Therefore curcuminoids could be developed as an adjunct therapy for diseases like cancer to save human life.
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Kuang, Yi-Qun. "The Development and Clinical Progress on Chemokine Receptor-Based HIV Entry Inhibitors." In Frontiers in Clinical Drug Research - HIV. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/9781681085265119040006.

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Conference papers on the topic "Chemokine inhibitor"

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Renner, Andreas, Katharina Marth, Helmut Schmutz, Julia Romanova, Boris Ferko, and Wolfgang Pohl. "Phase 1 trial of a novel chemokine inhibitor." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3712.

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Seitz, S., C. Stange, T. Dreyer, J. Nikonov, M. Kiechle, and H. Bronger. "Der PARP-Inhibitor Olaparib induziert die tumor-suppressiven Chemokine CXCL9 und CX3CL1 im Ovarialkarzinom." In Kongressabstracts zur Tagung 2020 der Deutschen Gesellschaft für Gynäkologie und Geburtshilfe (DGGG). © 2020. Thieme. All rights reserved., 2020. http://dx.doi.org/10.1055/s-0040-1718124.

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Williford, John-Michael, Jun Ishihara, Ako Ishihara, Aslan Mansurov, Melody A. Swartz, and Jeffrey A. Hubbell. "Abstract 2270: Recruitment of CD103+DCs via tumor-targeted chemokine delivery enhances efficacy of checkpoint inhibitor immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2270.

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Williford, John-Michael, Jun Ishihara, Ako Ishihara, Aslan Mansurov, Melody A. Swartz, and Jeffrey A. Hubbell. "Abstract 2270: Recruitment of CD103+DCs via tumor-targeted chemokine delivery enhances efficacy of checkpoint inhibitor immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2270.

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"Impact of Heparan Sulphate Binding Domain of Chemokine CCL21 to Migration of Breast Cancer Cells." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0132.

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Lymph node metastasis constitutes a key event in breast cancer progression. Chemokines are small proteins, which can promote metastatic spread by inducing cancer cell migration and invasion. Chemokine function is dependant upon their binding to both cell surface heparan sulphate (HS) molecules and to their specific receptor. Our group has demonstrated a significant increase in chemokine receptor CCR7 expression in cancerous breast epithelia compared to healthy controls. This study is designed to test the hypothesis that a non-HS binding forms of chemokine CCL21 can disrupt the normal response
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Zhang, Jihui, Jon Richardson, Jie Chen, et al. "A Novel Der P 1 Inhibitor ADZ 51,457 Inhibits Eosinophil Recruitment And Chemokine Release Following House Dust Mite Allergen Challenge In Brown Norway Rats." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2792.

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Dong, Yuanlin, Syeda M. Kabir, Eunsook Lee, and Deok-Soo Son. "Abstract 527: Proinflammatory chemokine receptor CXCR2 promotes cellular proliferation through suppression of cell cycle inhibitor p21 protein in ovarian cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-527.

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Pyo, Kyung-Ho, Hee Kyu Lee, Ha Ni Jo, et al. "Abstract 2755: Single-cell RNA sequencing reveals priming professional antigen-presenting macrophages and chemokine expressing T cells in tumor microenvironment by AXL inhibitor, SKI-G-801." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-2755.

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Ju, Donghong, Cecelia Speyer, David Gorski, and Mary A. Kosir. "Abstract 3008: Effects of HSPG on inhibition of chemokine-induced angiogenesis." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3008.

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Mir, Hina, Neeraj Kapur, Rajesh Singh, Guru Sonpavde, James W. Lillard, and Shailesh Singh. "Abstract 5362: Andrographolide inhibits prostate cancer by modulating chemokine and cytokines." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-5362.

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