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Relevant bibliographies by topics / Chicken cholera

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Academic literature on the topic 'Chicken cholera'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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Journal articles on the topic "Chicken cholera"

1

Harper, Marina, John D. Boyce, Ian W. Wilkie, and Ben Adler. "Signature-Tagged Mutagenesis of Pasteurella multocida Identifies Mutants Displaying Differential Virulence Characteristics in Mice and Chickens." Infection and Immunity 71, no. 9 (September 2003): 5440–46. http://dx.doi.org/10.1128/iai.71.9.5440-5446.2003.

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ABSTRACT Pasteurella multocida is the causative agent of fowl cholera in birds. Signature-tagged mutagenesis (STM) was used to identify potential virulence factors in a mouse septicemia disease model and a chicken fowl cholera model. A library of P. multocida mutants was constructed with a modified Tn916 and screened for attenuation in both animal models. Mutants identified by the STM screening were confirmed as attenuated by competitive growth assays in both chickens and mice. Of the 15 mutants identified in the chicken model, only 5 were also attenuated in mice, showing for the first time the presence of host-specific virulence factors and indicating the importance of screening for attenuation in the natural host.

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Sabrin, M. S., S. Saha, and M. M. Amin. "IMMUNE RESPONSE CAPABILITY OF `SHUVRA CHICKEN." Bangladesh Journal of Veterinary Medicine 10, no. 1-2 (July 9, 2013): 1–7. http://dx.doi.org/10.3329/bjvm.v10i1-2.15639.

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The study was carried out to determine the humoral immune response to Newcastle Disease Virus (NDV) and Bangladesh Agricultural University Fowl cholera (BAUFC) vaccines in Shuvra chicken, a newly develop chicken strain by BLRI (Bangladesh Livestock Research Institute). Ten Shuvra chickens were vaccinated with Baby Chick Ranikhet Disease Vaccine (BCRDV) at day 7 through intra ocular route (i/o) and with Ranikhet Disease Vaccine (RDV) at day 35 through intramuscular (i/m) route. Vaccine induced serum Haemagglunination Inhibition (HI) antibodies were measured by HI test. Two weeks after final immunization all vaccinated and control Shuvra chickens were challenged with virulent field isolates of NDV where all the vaccinated birds survived without showing any typical signs of NDV during the period of ten days observation period and all the control chickens died. Another 10 Shuvra were vaccinated twice with BAUFC vaccine through intramuscular route at day 42 and 70, and 10 Shuvra chickens were kept as unvaccinated control. This vaccine also induced significantly higher level of antibody titre as determined by Passive Haemagglutination (PHA) test. Vaccinated chickens showed significantly higher survival (80%) following challenge with virulent fowl cholera isolate and all the control birds died within 10 days of observation period. DOI: http://dx.doi.org/10.3329/bjvm.v10i1-2.15639Bangl. J. Vet. Med. (2012). 10 (1&2): 1-7

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3

Shampa, Chamak Nahar, Suma Akter, Sukumar Saha, Md Hadiuzzaman, Azhar Ul Alam, and Muhammad Tofazzal Hossain. "Comparative efficacy of BAU-fowl cholera and DLS-fowl cholera vaccines in indigenous chicken." Research in Agriculture Livestock and Fisheries 5, no. 2 (September 9, 2018): 193–99. http://dx.doi.org/10.3329/ralf.v5i2.38108.

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The present study was conducted to determine the immune response induced in indigenous chicken produced against BAU-FC and DLS-FC vaccines with their efficacy study against Pasteurella multocida. A total of forty (40) chickens were selected and divided into Group A (15), Group B (15) and Group C (10). Group A and B were vaccinated with BAU-FCV and DLS-FCV, respectively at the dose rate of 0.5 ml through SC at six weeks of age followed by boostering at 10 weeks of age while Group C was kept as unvaccinated control. Sera samples were collected after primary and booster vaccination and antibody titre was determined by Passive hemagglutination (PHA) test. The mean PHA titres recorded at 4 weeks after primary vaccination was 51.20 ± 7.84 in birds of group A and 38.40 ± 6.40 in birds of Group B. After booster vaccination, mean PHA titer was found 140.80 ± 31.35 at 16 weeks of age in case of BAU-FC vaccinated group and 115.20 ± 12.80 in case of DLS-FC vaccinated group. The mean PHA titer was 204.80 ± 31.35 and 179.20 ± 31.35 at 19 weeks of age in birds of BAU-FC and DLS-FC vaccinated group, respectively. Birds of all groups were challenged with virulent P. multocida at 17 weeks of age. It was observed that vaccinated chickens showed maximal resistance (100%) following challenge with virulent whereas unvaccinated control birds failed to resist the challenge infection. It can be assumed from the findings of present research work that both BAU-FCV and DLS-FCV are able to protect indigenous chicken from the outbreak of avian pasteurellosis and BAU-FV vaccine showed relatively higher immuno-protective titre than that of DLS-FC vaccine.Res. Agric., Livest. Fish.5(2): 193-199, August 2018

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Rozhdestvenskaya, T. N., L. Karimova, S. V. Pankratov, A. V. Ruzina, and E. V. Tomina. "Modern approaches to the production of inactivated vaccines against chicken cholera." Agrarian science 1, no. 7-8 (October 7, 2022): 68–73. http://dx.doi.org/10.32634/0869-8155-2022-361-7-8-68-73.

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Introduction. Chicken cholera is one of the most dangerous avian infectious diseases, causing significant economic damage to the industrial poultry production. Chicken cholera usually occurs in septic form, and causes high morbidity and mortality (60–80%), but recently it has become chronic, subclinical and associated. Inactivated emulsion vaccines are used worldwide to prevent chicken cholera and provide high and long-term immunity. However, there is a problem with residual reactogenicity of inactivated vaccines, particularly of the bacterial variants. This problem can be solved by using safer, next-generation adjuvants. The aim of the article is to study the physical and biological properties and determine the optimal inoculation volume and method of administration of inactivated vaccines against chicken cholera , based on different adjuvants.Materials and methods. Formaldehyde inactivated culture of P. multocida st. 115and a number of adjuvants (“Montanide GEL-02” and oil adjuvants “Montanide ISA 70 VG” and “Montanide ISA 78 VG”) were used for vaccine production. The vaccine samples were tested for sterility, stability and viscosity by conventional methods. Determination of reactogenicity and antigenic activity of the vaccines was carried out on young 30-days old chickensof egg-laying type.Results. The vaccine sample based on the adjuvant “Montanide ISA 70 VG” containing 1.5 billion P. Multocida microbial cells in a single immunizing dose of 0.3 cm3 was found to be the best among the tested preparations. When assessing the reactogenicity, it was obvious that all samples, regardless of the type of adjuvant, showed more pronounced residual reactogenic properties when injected intramuscularly into the chest muscle than when injected subcutaneously into the middle third of the neck.

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5

Chung, Jing Yeng, Ian Wilkie, John D. Boyce, Kirsty M. Townsend, Alan J. Frost, Majid Ghoddusi, and Ben Adler. "Role of Capsule in the Pathogenesis of Fowl Cholera Caused by Pasteurella multocida Serogroup A." Infection and Immunity 69, no. 4 (April 1, 2001): 2487–92. http://dx.doi.org/10.1128/iai.69.4.2487-2492.2001.

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ABSTRACT We have constructed a defined acapsular mutant inPasteurella multocida X-73 (serogroup A:1) by disrupting the hexA gene through the insertion of a tetracycline resistance cassette. The genotype of thehexA::tet(M) strain was confirmed by PCR and Southern hybridization, and the acapsular phenotype of this strain was confirmed by electron microscopy. ThehexA::tet(M) strain was attenuated in both mice and chickens. Complementation of the mutant with an intact hexAB fragment restored lethality in mice but not in chickens. In contrast to the results described previously for P. multocida serogroup B (J. D. Boyce and B. Adler, Infect. Immun. 68:3463–3468, 2000), thehexA::tet(M) strain was sensitive to the bactericidal action of chicken serum, whereas the wild-type and complemented strains were both resistant. Following inoculation into chicken muscle, the bacterial count of thehexA::tet(M) strain decreased significantly, while the wild-type and complemented strains both grew rapidly over 4 h. The capsule is thus an essential virulence determinant in the pathogenesis of fowl cholera.

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6

Ismail, Eman M., Mona Kadry, Esraa A. Elshafiee, Eman Ragab, Eman A. Morsy, Omar Rizk, and Manal M. Zaki. "Ecoepidemiology and Potential Transmission of Vibrio cholerae among Different Environmental Niches: An Upcoming Threat in Egypt." Pathogens 10, no. 2 (February 10, 2021): 190. http://dx.doi.org/10.3390/pathogens10020190.

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Cholera is a negative public health event caused by Vibrio cholerae. Although V. cholerae is abundant in natural environments, its pattern and transmission between different niches remain puzzling and interrelated. Our study aimed to investigate the occurrence of nonpathogenic V. cholerae in the natural environment during endemicity periods. It also aimed to highlight the role of molecular ecoepidemiology in mapping the routes of spread, transmission, and prevention of possible future cholera outbreaks. V. cholerae was detected in different aquatic environments, waterfowl, and poultry farms located along the length of the Nile River in Giza, Cairo, and Delta provinces, Egypt. After polymerase chain reaction amplification of the specific target outer membrane gene (Omp W) of suspected isolates, we performed sequence analysis, eventually using phylogenetic tree analysis to illustrate the possible epidemiological relationships between different sequences. Data revealed a significant variation in the physicochemical conditions of the examined Nile districts related to temporal, spatial, and anthropogenic activities. Moreover, data showed an evident association between V. cholerae and the clinically diseased Synodontis schall fish. We found that the environmental distress triggered by the salinity shift and elevated temperature in the Middle Delta of the Nile River affects the pathogenesis of V. cholerae, in addition to the characteristics of fish host inhabiting the Rosetta Branch at Kafr El-Zayat, El-Gharbia province, Egypt. In addition, we noted a significant relationship between V. cholerae and poultry sources that feed on the Nile dikes close to the examined districts. Sequence analysis revealed clustering of the waterfowl and broiler chicken isolates with human and aquatic isolated sequences retrieved from the GenBank databases. From the obtained data, we hypothesized that waterfowl act as a potential vector for the intermediate transmission of cholera. Therefore, continuous monitoring of Nile water quality and mitigation of Nile River pollution, in addition to following good managemental practices (GMPs), general hygienic guidelines, and biosecurity in the field of animal production and industry, might be the way to break this cyclic transmission between human, aquatic, and animal sectors.

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Victor, Atere, Bamikole Mathew, Oluyege Adekemi, Ajurojo Ayo, and Alo Odunayo. "Prevalence and antibiotic resistance of Pasteurella multocida isolated from chicken in Ado-Ekiti metropolis." International Journal of Scientific World 4, no. 2 (June 27, 2016): 40. http://dx.doi.org/10.14419/ijsw.v4i2.6273.

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Pasteurella multocida is a poultry bacterial pathogen causing fowl cholera in chicken. The prevalence and antibiotic susceptibility of P. multocida isolates from freshly dead chicken were determined. Ninety seven (97) freshly dead chicken from 23 different farms were analyzed for the presence of P. multocida. Swabs of the trachea and the liver of the necropsied chicken were activated on buffered peptone water and later cultured on blood agar and MacConkey agar. Pure culture of organisms were subjected to cultural and biochemical characterization. In vitro susceptibility of the pure isolates of P. multocida against 12 antimicrobial agents was determined using disk diffusion method. Twelve isolates of P. multocida were recovered from the chicken, with a prevalence of 12.4%. Nine of the isolates were recovered from the trachea and three from the liver. All the 12 isolates recovered from the birds were multi-resistant to the antibiotics used in this research. The antibiogram showed that all the isolates resisted ampicillin, amoxicillin/clavulinate, doxycycline and tylosine. Nitrofuratoin and gentamycin had the best antimicrobial activity with 25% and 50% resistance respectively. The resistance of other antibiotics are: Ofloxacin 75%, Ciprofloxacin 83.3%, Enrofloxacin 75%, Furasol 66.7%, Ceftazidime 91.7% and Cefuroxime 66.7%. This result showed that there is an emergence of multi- resistance in P. multocida, therefore it is important to carry out sensitivity test before administration of antibiotics in order to control fowl cholera.

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Quéré, Pascale, and Fabienne Girard. "Systemic adjuvant effect of cholera toxin in the chicken." Veterinary Immunology and Immunopathology 70, no. 1-2 (September 1999): 135–41. http://dx.doi.org/10.1016/s0165-2427(99)00060-4.

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9

Harper, Marina, Andrew D. Cox, Frank St. Michael, Ian W. Wilkie, John D. Boyce, and Ben Adler. "A Heptosyltransferase Mutant of Pasteurella multocida Produces a Truncated Lipopolysaccharide Structure and Is Attenuated in Virulence." Infection and Immunity 72, no. 6 (June 2004): 3436–43. http://dx.doi.org/10.1128/iai.72.6.3436-3443.2004.

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ABSTRACT Pasteurella multocida is the causative agent of fowl cholera in birds. In a previous study using signature-tagged mutagenesis, we identified a mutant, AL251, which was attenuated for virulence in mice and in the natural chicken host. Sequence analysis indicated that AL251 had an insertional inactivation of the gene waaQPM , encoding a putative heptosyl transferase, required for the addition of heptose to lipopolysaccharide (LPS) (M. Harper, J. D. Boyce, I. W. Wilkie, and B. Adler, Infect. Immun. 71:5440-5446, 2003). In the present study, using mass spectrometry and nuclear magnetic resonance, we have confirmed the identity of the enzyme encoded by waaQPM as a heptosyl transferase III and demonstrated that the predominant LPS glycoforms isolated from this mutant are severely truncated. Complementation experiments demonstrated that providing a functional waaQPM gene in trans can restore both the LPS to its full length and growth in mice to wild-type levels. Furthermore, we have shown that mutant AL251 is unable to cause fowl cholera in chickens and that the attenuation observed is not due to increased serum sensitivity.

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10

Boyce, John D., Ian Wilkie, Marina Harper, Mike L. Paustian, Vivek Kapur, and Ben Adler. "Genomic Scale Analysis of Pasteurella multocida Gene Expression during Growth within the Natural Chicken Host." Infection and Immunity 70, no. 12 (December 2002): 6871–79. http://dx.doi.org/10.1128/iai.70.12.6871-6879.2002.

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ABSTRACT Little is known about the genomic-scale transcriptional responses of bacteria during natural infections. We used whole-genome microarray analysis to assess the transcriptional state of the gram-negative pathogen Pasteurella multocida, the causative agent of fowl cholera, during infection in the natural chicken host. We compared the expression profiles of bacteria harvested from the blood of septicemic chickens experiencing late-stage fowl cholera with those from bacteria grown in rich medium. Independent analysis of bacterial expression profiles from the infection of three individual chickens indicated that 40 genes were differentially expressed in all three individuals, 126 were differentially expressed in two of the three individuals, and another 372 were differentially expressed in one individual. Real-time reverse transcription-PCR assays were used to confirm the expression ratios for a number of genes. Of the 40 genes differentially expressed in all three individuals, 17 were up-regulated and 23 were down-regulated in the host compared with those grown in rich medium. The majority (10 of 17) of the up-regulated genes were involved in amino acid transport and metabolism and energy production and conversion, clearly indicating how P. multocida alters its biosynthetic and energy production pathways to cope with the host environment. In contrast, the majority (15 of 23) of down-regulated genes were of unknown or poorly characterized functions. There were clear differences in gene expression between the bacteria isolated from each of the three chickens, a finding consistent with individual host variation being an important factor in determining pathogen gene expression. Interestingly, bacteria from only two of the three infected animals had a gene expression profile highly similar to that observed during growth under iron-limiting conditions, suggesting that severe iron starvation may not always occur during P. multocida infection.

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Dissertations / Theses on the topic "Chicken cholera"

1

Hopkins, Brett A. "Fowl cholera in turkeys : vaccination, pathogenicity, and DNA analysis /." free to MU campus, to others for purchase, 1999. http://wwwlib.umi.com/cr/mo/fullcit?p9962544.

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2

Jablonski, Lynn McGonagle. "Development of a cloning system for gene expression in Pasteurella multocida." Diss., This resource online, 1993. http://scholar.lib.vt.edu/theses/available/etd-05042006-164516/.

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3

Gunawardana, Gnanalatha Abeywickramasinghe. "Pasteurellosis in chickens : studies on the humoral response of chickens to Paseurelle multocida and the genetic analysis of causative strains of fowl cholera /." [St. Lucia, Qld.], 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17050.pdf.

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4

Harper, Marina. "Virulence determinants of Pasteurella multocida." Monash University, Dept. of Microbiology, 2003. http://arrow.monash.edu.au/hdl/1959.1/9341.

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Lo, Miranda. "Characterisation of in vivo expressed proteins of Pasteurella multocida." Monash University, Dept. of Microbiology, 2003. http://arrow.monash.edu.au/hdl/1959.1/9429.

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Books on the topic "Chicken cholera"

1

Friend, Milton. Avian cholera: A major new cause of waterfowl mortality. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1989.

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2

M, Mulcahy Daniel, and U.S. Fish and Wildlife Service. Research and Development., eds. Avian cholera and related topics: An annotated bibliography. Washington, DC: U.S. Dept. of the Interior, Fish and Wildlife Service, Research and Development, 1988.

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3

Friend, Milton. Waterfowl management handbook.: A major new cause of waterfowl mortality. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1989.

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Friend, Milton. Waterfowl management handbook.: A major new cause of waterfowl mortality. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1989.

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Friend, Milton. Waterfowl management handbook.: The invisible disease. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1989.

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Friend, Milton. Waterfowl management handbook.: The invisible disease. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, 1989.

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Khosraviani, Mehraban. Monoclonal antibodies against P-1059 strain of Pasteurella Multocida. 1989.

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Book chapters on the topic "Chicken cholera"

1

Ndlovu, Wiseman, Nyambeni Ronald Mudimeli, Marizvikuru Mwale, Tshianeo Mellda Ndou, Ola Segun Obadire, and Joseph Francis. "Ethnoveterinary Practices for Indigenous Poultry Health Management by Smallholder Farmers." In Herbs and Spices - New Advances [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.108912.

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Most resource-constrained smallholder farmers rely on ethnoveterinary medicine to treat village chicken diseases. An in-depth literature review and empirical study was conducted to establish ethnoveterinary practices used in indigenous chicken health management. The study showed that most village chicken farmers were female (70%). Most farmers kept chickens for socio-economic purposes; food, status and income. Common birds’ ailments treated and controlled using EVPs include Fowl Cholera; Pullorum; Diarrhea; Gumboro; Avian Influenza; Ngorok & Snot (Infectious Coryza); Bloody & watery diarrhea (Coccidiosis). Variety of materials were used to treat and control chicken diseases either as purely indigenous methods or in combination with conventional medicines. Among the practices, medicinal plants like Aloe vera emerged as the most commonly used botanical plant. More so, both literature and the empirical study, showed that farmers prepared and applied A. vera uniquely depending on the region and type of A. vera. Majority of remedies are used to treat more than one ailment. It is recommended that these practices be preserved and considered for new drug advancement and commercialization to promote cheaper and environmentally friendly options for poultry health management.

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2

Fagan, Brian. "Tourists Along the Nile." In From Stonehenge to Samarkand. Oxford University Press, 2006. http://dx.doi.org/10.1093/oso/9780195160918.003.0011.

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Until 1830, the traveler to India faced a long, and often stormy, passage around the Cape of Good Hope. The advent of the steamship changed everything. Now you could take a steamer from England or Marseilles to Alexandria, then spend a few days or weeks in Cairo waiting for news that the ship for India was approaching Suez. You then took a camel, horse, or wagon across the desert to meet the vessel at what was then a small village. Hotels opened in Suez and Cairo to accommodate transit passengers. The British Hotel in Cairo, soon to be renamed Shepheard’s Hotel after its manager, welcomed its first guests in 1841. This magnificent Victorian institution became world famous, especially after the opening of the Suez Canal in 1869, when it became the hotel of choice for the British Raj on its way to and from India. The hotel also catered to a new breed, the archaeological tourist. Bubonic plague epidemics periodically claimed thousands of lives in Egypt until 1844, when it suddenly and mysteriously disappeared. Cholera arrived from India to take its place, but despite this scourge, Egypt became a recommended destination for travelers wishing to escape damp European winters. By this time, a journey up the Nile to the First Cataract was routine, although one had to endure long quarantines on account of the plague. Nile travel became so popular that the London publisher John Murray commissioned the Egyptologist John Gardner Wilkinson to write a guide, one of a series aimed at a new audience of middle-class tourists.Wilkinson traveled in style, his baggage requiring a small army of porters. The contents of his baggage included an iron bedstead, a sword and other oddities, and “much more,” including a chicken coop, ample biscuits (cookies), and potted meats. He lamented the high cost of living in Egypt and the changes brought by a rising tide of visitors. “The travelers who go up the Nile will I fear soon be like Rhine tourists. & Cheapside will pour out its Legions upon Egypt.” His Handbook for Travellers in Egypt first appeared in 1847, went through multiple editions until 1873, and was still in common use half a century after its first appearance.

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Gratzer, Walter. "The use of vacations." In Eurekas and euphorias, 277–78. Oxford University PressNew York, NY, 2002. http://dx.doi.org/10.1093/oso/9780192804037.003.0173.

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Abstract Here is how Louis Pasteur (1822-95) came upon one of the main tenets of vaccination. It illustrates perfectly his famous maxim that fortune favours the prepared mind: he had been studying fowl cholera in chickens and interrupted his work by a vacation. when he returned he tested the cultures of the cholera bacteria and found that they had become inactive, in fact had died: subcultures (growth media seeded with bacteria from the original cultures) did not grow and the birds into which they were injected developed no sign of disease. Pasteur prepared to start over with new cultures, but instead of merely abandoning the abortive experiment, he decided, for no reason that he could articulate, to re inject the same birds with a new, active culture, One of his colleagues reported the outcome: To the surprise of all, and perhaps even of Pasteur, who was not expecting such success, nearly all these fowls withstood the inoculation, although fresh fowls succumbed after the usual incubation period.

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